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Relevant bibliographies by topics / PPAR gamma

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Academic literature on the topic 'PPAR gamma'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "PPAR gamma"

1

Yoshikawa, Keiji. "PPAR^|^gamma;." Japanese Journal of SURGICAL METABOLISM and NUTRITION 48, no. 2 (2014): 81–85. http://dx.doi.org/10.11638/jssmn.48.2_81.

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Konger, Raymond L., Xiaoling Xuei, Ethel Derr-Yellin, Fang Fang, Hongyu Gao та Yunlong Liu. "The Loss of PPARγ Expression and Signaling Is a Key Feature of Cutaneous Actinic Disease and Squamous Cell Carcinoma: Association with Tumor Stromal Inflammation". Cells 13, № 16 (2024): 1356. http://dx.doi.org/10.3390/cells13161356.

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Given the importance of peroxisome proliferator-activated receptor (PPAR)-gamma in epidermal inflammation and carcinogenesis, we analyzed the transcriptomic changes observed in epidermal PPARγ-deficient mice (Pparg-/-epi). A gene set enrichment analysis revealed a close association with epithelial malignancy, inflammatory cell chemotaxis, and cell survival. Single-cell sequencing of Pparg-/-epi mice verified changes to the stromal compartment, including increased inflammatory cell infiltrates, particularly neutrophils, and an increase in fibroblasts expressing myofibroblast marker genes. A com

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Fajas, L., MB Debril, and J. Auwerx. "Peroxisome proliferator-activated receptor-gamma: from adipogenesis to carcinogenesis." Journal of Molecular Endocrinology 27, no. 1 (2001): 1–9. http://dx.doi.org/10.1677/jme.0.0270001.

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Peroxisome proliferator-activated receptors (PPARs) are nuclear hormone receptors, initially described as molecular targets for synthetic compounds inducing peroxisome proliferation. PPAR-gamma, the best characterized of the PPARs, plays a crucial role in adipogenesis and insulin sensitization. Furthermore, PPAR-gamma has been reported to affect cell proliferation/differentiation pathways in various malignancies. We discuss in the present review recent advances in the understanding of the function of PPAR-gamma in both cell proliferation and adipocyte differentiation.

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Ferreira, Silvana R., Leandro M. Vélez, Maria F. Heber, Giselle A. Abruzzese, and Alicia B. Motta. "Prenatal androgen excess alters the uterine peroxisome proliferator-activated receptor (PPAR) system." Reproduction, Fertility and Development 31, no. 8 (2019): 1401. http://dx.doi.org/10.1071/rd18432.

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It is known that androgen excess induces changes in fetal programming that affect several physiological pathways. Peroxisome proliferator-activated receptors (PPARs) α, δ and γ are key mediators of female reproductive functions, in particular in uterine tissues. Thus, we aimed to study the effect of prenatal hyperandrogenisation on the uterine PPAR system. Rats were treated with 2mg testosterone from Day 16 to 19 of pregnancy. Female offspring (PH group) were followed until 90 days of life, when they were killed. The PH group exhibited an anovulatory phenotype. We quantified uterine mRNA level

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Triana, Rina, Nurrani Mustika Dewi, Siska Darmayanti, et al. "PPAR-gamma Signaling in Metabolic Homeostasis." Indonesian Biomedical Journal 8, no. 3 (2016): 147. http://dx.doi.org/10.18585/inabj.v8i3.209.

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BACKGROUND: Peroxisome proliferator-activated receptor (PPAR)-γ, or also known as nuclear receptor subfamily 1 group C member 3 (NR1C3), is a PPAR which serves as master regulator of adipocytes differentiation, and plays an important role in lipid metabolism or adipogenesis. Recent study showed that PPAR-γ is expressed in most tissue and also has critical impact in many metabolic homeostasis disorders.CONTENT: Dysregulation of PPAR-γ is correlated to the development of obesity, type 2 diabetes, atherosclerosis, cardiovascular disease, acute kidney injury, autoimmune disease, gastrointestinal d

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Wagner, Nicole, and Kay-Dietrich Wagner. "Peroxisome Proliferator-Activated Receptors and the Hallmarks of Cancer." Cells 11, no. 15 (2022): 2432. http://dx.doi.org/10.3390/cells11152432.

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Peroxisome proliferator-activated receptors (PPARs) function as nuclear transcription factors upon the binding of physiological or pharmacological ligands and heterodimerization with retinoic X receptors. Physiological ligands include fatty acids and fatty-acid-derived compounds with low specificity for the different PPAR subtypes (alpha, beta/delta, and gamma). For each of the PPAR subtypes, specific pharmacological agonists and antagonists, as well as pan-agonists, are available. In agreement with their natural ligands, PPARs are mainly focused on as targets for the treatment of metabolic sy

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Abdulazeez, Azeemat, Zaharadeen Yusuf, and Sulaimon Folami. "Peroxisome Proliferator Activated Receptors." Journal of Biological Research and Reviews 1, no. 2 (2024): 97. http://dx.doi.org/10.5455/jbrr.20240414015408.

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Peroxisome proliferator-activated receptors (PPARs) are nuclear receptors that regulate key physiological processes, including lipid metabolism, glucose homeostasis, and inflammation. The three subtypes, PPAR-alpha, PPAR-beta/delta, and PPAR-gamma, have distinct roles and tissue distributions, making them promising targets for metabolic and inflammatory disease therapies. Experimental methods encompassed animal models, genetically modified mice, and pharmacological interventions with PPAR agonists and antagonists. Cell culture studies utilized specific PPAR ligands, along with molecular biolog

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Mansour, Mahmoud M., Hari O. Goyal, Tim D. Braden, et al. "Activation of Penile Proadipogenic Peroxisome Proliferator-Activated Receptor with an Estrogen: Interaction with Estrogen Receptor Alpha during Postnatal Development." PPAR Research 2008 (2008): 1–10. http://dx.doi.org/10.1155/2008/651419.

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Exposure to the estrogen receptor alpha (ER) ligand diethylstilbesterol (DES) between neonatal days 2 to 12 induces penile adipogenesis and adult infertility in rats. The objective of this study was to investigate the in vivo interaction between DES-activated ER and the proadipogenic transcription factor peroxisome proliferator-activated receptor gamma (PPAR). Transcripts for PPARs , , and and 1a splice variant were detected in Sprague-Dawley normal rat penis with PPAR predominating. In addition, PPAR1b and PPAR2 were newly induced by DES. The PPAR transcripts were significantly upregulated wi

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Wagner, Nicole, and Kay-Dietrich Wagner. "PPAR Beta/Delta and the Hallmarks of Cancer." Cells 9, no. 5 (2020): 1133. http://dx.doi.org/10.3390/cells9051133.

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Peroxisome proliferator-activated receptors (PPARs) belong to the nuclear hormone receptor family. Three different isoforms, PPAR alpha, PPAR beta/delta and PPAR gamma have been identified. They all form heterodimers with retinoic X receptors to activate or repress downstream target genes dependent on the presence/absence of ligands and coactivators or corepressors. PPARs differ in their tissue expression profile, ligands and specific agonists and antagonists. PPARs attract attention as potential therapeutic targets for a variety of diseases. PPAR alpha and gamma agonists are in clinical use f

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SONI, DEEPA, and ROBERT M. FRIEDLANDER. "PPAR-gamma." Neurosurgery 52, no. 4 (2003): NA. http://dx.doi.org/10.1227/01.neu.0000309176.04106.fe.

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Dissertations / Theses on the topic "PPAR gamma"

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Olson, Peter. "PPAR gamma and PPAR delta in glucose and lipid homoeostasis /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2004. http://wwwlib.umi.com/cr/ucsd/fullcit?p3153704.

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Michel, Ursula. "Assoziation der Variante Pro115Gln im PPAR[gamma]2-Gen [PPAR-gamma-2-Gen] mit Adipositas und Diabetes mellitus Typ II." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=969118597.

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Shiau, Chung-Wai. "Thiazolidinediones: from peroxisome-proliferator-activated receptor [gamma] (PPAR[gamma]) to anticancer agents." Columbus, Ohio : Ohio State University, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1128111032.

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Takahama, Carina Harumi. "Controle da Anexina 1 sobre a expressão do receptor nuclear proliferador de peroxissomos em diferentes tipos celulares." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/9/9141/tde-26092016-125014/.

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A proteína Anexina A1 (ANXA1), sintetizada e liberada por fagócitos pela ação de glicocorticóides, é uma proteína anti-inflamatória, pois inibe o influxo de neutrófilos para o foco da inflamação, e induz os mecanismos de eferocitose em neutrófilos e macrófagos. Nosso grupo mostrou que a ANXA1 regula a expressão do receptor ativado por proliferadores de peroxissomos (PPAR) em macrófagos. Em continuidade, o presente trabalho investigou o mecanismo da ANXA1 sobre a expressão de PPARγ em macrófagos, e se este controle ocorre em demais leucócitos e tecido adiposo. Para tanto, macrófagos, neutr

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Gomes, Emerson Rodrigo Machi 1977. "Caracterização bioquímica e celular da glutaminase isoforma Kidney-type com seus parceiros de interação." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311950.

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Orientador: Sandra Martha Gomes Dias<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-23T00:13:51Z (GMT). No. of bitstreams: 1 Gomes_EmersonRodrigoMachi_M.pdf: 4424058 bytes, checksum: 4547741a163b53b4836c32f103096cd3 (MD5) Previous issue date: 2013<br>Resumo: Células tumorais apresentam uma autonomia metabólica aumentada em comparação a células não-transformadas, incorporando nutrientes e metabolizando-os através de vias que suportam o seu crescimento e proliferação. O foco deste trabalho foi a enzima glutami

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Tremblay, Hugo. "Identification et caractérisation d'agonistes mixtes des récepteurs GPR40 et PPAR[gamma]." Mémoire, Université de Sherbrooke, 2011. http://savoirs.usherbrooke.ca/handle/11143/4930.

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Le diabète de type II est une maladie impliquant deux déficiences majeures : la résistance à l'insuline et la diminution de sécrétion d'insuline. Afin d'identifier de nouvelles pistes de traitement potentiel pour cette maladie nous avons utilisé une approche polypharmacologique ciblant simultanément ces deux déficiences. Pour ce faire nous avons conçu et synthétisé une librairie de 29 composés et caractérisé leur activité biologique sur les récepteurs GPR40 et PPAR[gamma], dans le but d'identifier des agonistes mixtes. Ce travail a mené l'identification de deux composés ayant une activité sur

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Kim, Dasom. "PPAR-gamma Regulates T Cell Responses in Air Pollutant-associated Inflammation." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1522414820700163.

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Evans, Kyle William. "PPAR gamma AND eNOS CONTRIBUTE TO THE RESOLUTION OF CHRONIC INFLAMMATION." Diss., Temple University Libraries, 2011. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/197871.

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Microbiology and Immunology<br>Ph.D.<br>Chronic inflammation follows defined phases of induction, inflammation, and resolution. The resolution phase requires cycloxygenase-2 (COX-2) activity. This study aims to address what other molecules are required for a functional resolution phase. We demonstrated that in murine collagen-induced arthritis the transcription factor, PPARgamma plays a role in the resolution phase. Inhibition of COX-2 activity results in fewer PPARgamma positive cells in the arthritic synovium. Treatment with a PPARgamma antagonist, SR202, alone, also disrupts the process of

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Baker, Amelia Rachel Haas. "Environmental PPAR-gamma agonists accelerate aging of bone and impair lymphopoiesis." Thesis, Boston University, 2012. https://hdl.handle.net/2144/12274.

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Thesis (Ph.D.)--Boston University PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.<br>A growing number of environmental contaminants, including phthalates and organotins, are being recognized for their ability to activate peroxisome proliferator-activated receptor γ (PPARγ) and promote adipogene

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Beuzelin, Diane. "Rôle des récepteurs nucléaires PPAR gamma et PPAR alpha dans la conversion d'adipocytes blancs humains en adipocytes bruns/brites." Thesis, Toulouse 3, 2015. http://www.theses.fr/2015TOU30346/document.

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Chez les mammifères, deux types de tissu adipeux (TA) sont présents: le TA blanc, qui est l'organe de stockage et de libération des lipides, et le TA brun, qui est un organe spécialisé dans la production de chaleur grâce à l'expression de la protéine découplante mitochondriale UCP1.Chez l'homme, la présence d'un TA brun métaboliquement actif est inversement corrélée à l'obésité et au diabète de type 2. Ce TA brun est composé de deux types distincts de cellules thermogéniques, les adipocytes bruns classiques présents dans des dépôts spécifiques et les adipocytes "brite " (brown-in-white). Chez

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Books on the topic "PPAR gamma"

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Sauma, Lilian. Transcriptional activity of PPAR[gamma] in primary human adipocytes. 2008.

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Betz, Matthias Johannes Maximilian. Effekte von PPAR[gamma]-Agonisten auf Proliferation und Differenzierung humaner Nebennierenrindenkarzinomzellen. 2006.

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Petscher, Kerstin Patricia. Peroxisome proliferator-activated receptor-γ [receptor-gamma] (PPARγ) [PPAR-gamma] und ihre Auswirkungen auf die f-Aktin-Fraktion CD4-positiver Zellen. 2010.

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Book chapters on the topic "PPAR gamma"

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Toffoli, Barbara, and Béatrice Desvergne. "PPAR Gamma Receptor, Skin Lipids and Hair." In Lipids and Skin Health. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09943-9_18.

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Chawla, Ajay, Chih-Hao Lee, Yaacov Barak, Debbie Liao, and Ronald M. Evans. "Functions of PPAR Gamma in Macrophages and Atherosclerosis." In Medical Science Symposia Series. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-1171-7_3.

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Sakharkar, Meena Kishore. "Integrative Approaches for Drug Discovery – PPAR Gamma as a Case Study." In Advances in Biomedical Infrastructure 2013. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-37137-0_1.

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Harris, Sarah G., and Richard P. Phipps. "induction of apoptosis in mouse t cells upon peroxisome proliferator-activated receptor gamma (ppar-γ) binding." In Advances in Experimental Medicine and Biology. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0193-0_65.

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Brown, Robert Andrew. "The Crucial Relevance of ALA and LA as Primary Peroxisomal Beta-Oxidation Substrates, of Oxidised LA as the Primary Endogenous Activator of PPAR Gamma, and Energy Deficit as the Primary Activator of PPAR Alpha." In Omega-3 Fatty Acids. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40458-5_32.

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Brown, Robert Andrew. "The Roles of Linoleic and Alpha-linolenic Acid, Their Oxylipins and the PPAR Alpha-, Delta- and Gamma-Related Peroxisomal Pathways on Obesity in the Context of a “Western” Diet." In Omega-3 Fatty Acids. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40458-5_31.

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Brown, Robert Andrew. "In a Western Dietary Context Excess Oxidised Linoleic Acid of Dietary and Endogenous Origin by Over-Activation of PPAR Gamma so Immune and Inflammatory Pathways, and through Cardiolipin Damage, Increases Cardiovascular Risk." In Omega-3 Fatty Acids. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40458-5_29.

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Farmer, Stephen R. "Regulation of the Cell Cycle by Peroxisome Proliferator — Activated Receptor Gamma (PPARγ)." In Steroid Hormones and Cell Cycle Regulation. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0965-3_11.

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Sahin, Ali. "Insulin-Sensitizing Antihyperglycemic Drugs." In Current Perspective on Diabetes Mellitus in Clinical Sciences. Nobel Tip Kitabevleri, 2023. http://dx.doi.org/10.69860/nobel.9786053359111.10.

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Insulin-sensitizing antihyperglycemic drugs are a class of medications used in the management of diabetes mellitus, particularly type 2 diabetes, by improving the body’s response to insulin. One prominent group within this category is the thiazolidinediones (TZDs), which include drugs like pioglitazone and rosiglitazone. These medications act primarily by activating peroxisome proliferator-activated receptor gamma (PPARγ) receptors, which play a key role in regulating glucose and lipid metabolism. By enhancing insulin sensitivity in peripheral tissues such as muscle, adipose tissue, and liver,

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Meyer, Stefanie, Thomas Vogt, Michael Landthaler, et al. "Cyclooxygenase 2 (COX2) and Peroxisome Proliferator-Activated Receptor Gamma (PPARG) Are Stage-Dependent Prognostic Markers of Malignant Melanoma." In From Molecular to Modular Tumor Therapy. Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-9531-2_22.

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Conference papers on the topic "PPAR gamma"

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Jiang, WG, X. Li, and RE Mansel. "P2-05-02: PGC1, Peroxisome Proliferator Activated Receptor-gamma (PPAR-gamma) Coactivator-1, Is Necessary in PPAR-gamma Modulated Angiogenesis." In Abstracts: Thirty-Fourth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 6‐10, 2011; San Antonio, TX. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/0008-5472.sabcs11-p2-05-02.

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Guan, Yaxin, Jingwei Lin, Glen M. Borchert, et al. "FGF21 mediates corticosteroid-related bone mass loss through PPAR-$\gamma$." In 2019 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2019. http://dx.doi.org/10.1109/bibm47256.2019.8983042.

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Park, Yong S., and Kwang C. Kim. "The Anti-inflammatory Role Of PPAR-gamma Requires MUC1 Mucin." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1416.

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Oldenburg, Peter J., Elizabeth B. Klein, Sandra M. Wells, and Joseph H. Sisson. "Ethanol Increases PPAR-Gamma Activation In Cultured Lung Epithelial Cells." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2820.

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Church, Colin, Andrew Peacock, and David Welsh. "Effect Of PPAR-gamma Agonists On Rat Pulmonary Artery Fibroblasts." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6288.

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Koutroulis, I., C. E. Hoptay, and R. J. Freishtat. "PPAR-Gamma Agonism Can Alleviate the Hyperimmune Response in Pediatric Sepsis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1704.

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Kusampudi, S., V. Meganathan, P. Nsiah та V. Boggaram. "Peroxisome Proliferator-Activated Receptor Gamma (PPAR-γ) Activator 15d-PGJ2 Suppresses Organic Dust Induction of Lung Inflammatory Mediators Independently of PPAR-γ". У American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4414.

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Weidman, Elizabeth K., Beverly R. Wuertz, Patrick M. Gaffney, and Frank G. Ondrey. "Abstract 3802: PPAR gamma mediated induction of transglutaminase in aerodigestive preneoplasia in vitro." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3802.

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Keith, Robert L., Patrick J. Blackford, John K. Kittelson, et al. "Abstract ED01-03: Chemoprevention of lung cancer with prostacyclins or PPAR gamma agonists." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 12-16, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1535-7163.targ-11-ed01-03.

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Lian, Xuemei, Huimin Li, Lori Stanton, Cong Yan, and Hong Du. "The Role Of Respiratory Epithelial Cells In The Pathophysiological Function Of PPAR-gamma." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1363.

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Reports on the topic "PPAR gamma"

1

Hayward, Simon W. Modulation of PPAR-Gamma Signaling in Prostatic Carcinogenesis. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada525561.

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Hayward, Simon W. Modulation of PPAR-Gamma Signaling in Prostatic Carcinogenesis. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada547687.

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ZHANG, Zhuoya, YAN Qi, Wenhao WU, Yuan ZHAO, and Hua ZHANG. PPAR-alpha/gamma agonists, glucagon-like peptide-1 receptor agonists and metformin for non-alcoholic fatty liver disease: A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.4.0066.

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