Relevant bibliographies by topics / Pq4305

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Academic literature on the topic 'Pq4305'

Author: Grafiati
Published: 25 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "Pq4305"

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Vasyagin, Egor A., Anna A. Zykova, Eugenia S. Mardanova, et al. "Influenza A Vaccine Candidates Based on Virus-like Particles Formed by Coat Proteins of Single-Stranded RNA Phages Beihai32 and PQ465." Vaccines 12, no. 9 (2024): 1033. http://dx.doi.org/10.3390/vaccines12091033.

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Efficient control of influenza A infection can potentially be achieved through the development of broad-spectrum recombinant vaccines based on conserved antigens. The extracellular domain of the transmembrane protein M2 of influenza A virus (M2e) is highly conserved but poorly immunogenic and needs to be fused to an adjuvant protein or carrier virus-like particles (VLPs) to increase immunogenicity and provide protection against infection. In this study, we obtained VLPs based on capsid proteins (CPs) of single-stranded RNA phages Beihai32 and PQ465 bearing the M2e peptides. Four copies of the
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2

Engdahl, Taylor B., F. Graeme Frost, Amanda J. Schech, and Samantha L. Elliott. "Viability and migratory effects of IGF-1R inhibitor, PQ401, on triple negative breast cancer cells." BIOS 90, no. 1 (2019): 14. http://dx.doi.org/10.1893/0005-3155-90.1.14.

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Zhou, Xiang, Xinli Zhao, Xiangsheng Li, et al. "PQ401, an IGF-1R inhibitor, induces apoptosis and inhibits growth, proliferation and migration of glioma cells." Journal of Chemotherapy 28, no. 1 (2016): 44–49. http://dx.doi.org/10.1179/1973947815y.0000000026.

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Maher, Lynn M., and Anastasia M. Raymer. "Management of Anomia." Topics in Stroke Rehabilitation 11, no. 1 (2004): 10–21. http://dx.doi.org/10.1310/318r-rmd5-055j-pq40.

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Ghosh, Manik C., Vijay Gorantla, Patrudu S. Makena, et al. "Insulin-like growth factor-I stimulates differentiation of ATII cells to ATI-like cells through activation of Wnt5a." American Journal of Physiology-Lung Cellular and Molecular Physiology 305, no. 3 (2013): L222—L228. http://dx.doi.org/10.1152/ajplung.00014.2013.

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Alveolar type II (ATII) epithelial cells play a crucial role in the repair and remodeling of the lung following injury. ATII cells have the capability to proliferate and differentiate into alveolar type I (ATI) cells in vivo and into an ATI-like phenotype in vitro. While previous reports indicate that the differentiation of ATII cells into ATI cells is a complex biological process, the underlying mechanism responsible for differentiation is not fully understood. To investigate factors involved in this differentiation in culture, we used a PCR array and identified several genes that were either
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Huang, Binbin, Long Ge, Dong Xiang, et al. "Isolation, characterization, and genomic analysis of a lytic bacteriophage, PQ43W, with the potential of controlling bacterial wilt." Frontiers in Microbiology 15 (August 1, 2024). http://dx.doi.org/10.3389/fmicb.2024.1396213.

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Bacterial wilt (BW) is a devastating plant disease caused by the soil-borne bacterium Ralstonia solanacearum species complex (Rssc). Numerous efforts have been exerted to control BW, but effective, economical, and environmentally friendly approaches are still not available. Bacteriophages are a promising resource for the control of bacterial diseases, including BW. So, in this study, a crop BW pathogen of lytic bacteriophage was isolated and named PQ43W. Biological characterization revealed PQ43W had a short latent period of 15 min, 74 PFU/cell of brust sizes, and good stability at a wide rang
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Kim, Wooseong, Guijin Zou, Wen Pan, et al. "The Neutrally Charged Diarylurea Compound PQ401 Kills Antibiotic-Resistant and Antibiotic-Tolerant Staphylococcus aureus." mBio 11, no. 3 (2020). http://dx.doi.org/10.1128/mbio.01140-20.

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ABSTRACT Resistance or tolerance to traditional antibiotics is a challenging issue in antimicrobial chemotherapy. Moreover, traditional bactericidal antibiotics kill only actively growing bacterial cells, whereas nongrowing metabolically inactive cells are tolerant to and therefore “persist” in the presence of legacy antibiotics. Here, we report that the diarylurea derivative PQ401, previously characterized as an inhibitor of the insulin-like growth factor I receptor, kills both antibiotic-resistant and nongrowing antibiotic-tolerant methicillin-resistant Staphylococcus aureus (MRSA) by lipid
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8

Li, Junhong, Xin Zhou, Jialin Chen, Pernilla Eliasson, Paul J. Kingham, and Ludvig J. Backman. "Secretome from myoblasts statically loaded at low intensity promotes tenocyte proliferation via the IGF‐1 receptor pathway." FASEB Journal 37, no. 10 (2023). http://dx.doi.org/10.1096/fj.202301097r.

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AbstractExercise is widely recognized as beneficial for tendon healing. Recently, it has been described that muscle‐derived molecules secreted in response to static exercise influence tendon healing. In this study, the optimal static loading intensity for tendon healing and the composition of secretome released by myoblasts in response to different intensities of static strain were investigated. In an in vitro coculture model, myoblasts were mechanically loaded using a Flexcell Tension System. Tenocytes were seeded on transwell inserts that allowed communication between the tenocytes and myobl
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Vong, Chi Teng, Dechao Tan, Fengyun Liao та ін. "Ginsenoside Rk1 Ameliorates ER Stress-Induced Apoptosis through Directly Activating IGF-1R in Mouse Pancreatic β-Cells and Diabetic Pancreas". American Journal of Chinese Medicine, 27 травня 2024, 1–17. http://dx.doi.org/10.1142/s0192415x24500484.

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Hyperglycemia induces chronic stresses, such as oxidative stress and endoplasmic reticulum (ER) stress, which can result in [Formula: see text]-cell dysfunction and development of Type 2 Diabetes Mellitus (T2DM). Ginsenoside Rk1 is a minor ginsenoside isolated from Ginseng. It has been shown to exert anti-cancer, anti-inflammatory, anti-oxidant, and neuroprotective effects; however, its effects on pancreatic cells in T2DM have never been studied. This study aims to examine the novel effects of Ginsenoside Rk1 on ER stress-induced apoptosis in a pancreatic [Formula: see text]-cell line MIN6 and
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Small, Theodore W., and J. Geoffrey Pickering. "Abstract 270: Insulin-Like Growth Factor-1 Promotes Vascular Smooth Muscle Survival By 26S Proteasome-Mediated Degradation of Wilms’ Tumor 1-Associating Protein." Circulation 116, suppl_16 (2007). http://dx.doi.org/10.1161/circ.116.suppl_16.ii_34-a.

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Apoptosis of vascular smooth muscle cells (SMC) is a culprit event in atherosclerotic plaque destabilization. We recently discovered that Wilms’ tumor 1-associating protein (WTAP) is a dynamically expressed transcriptional regulator that can be pro-apoptotic for human SMCs (Circ Res, 2006). To identify upstream regulators of this nuclear protein, we screened growth factors for their capacity to impact WTAP expression and found that insulin-like growth factor-1 (IGF-1), a potent survival factor for SMCs, stimulated a striking decline in WTAP protein abundance, to 10% at 12 h. We further determi
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Dissertations / Theses on the topic "Pq4305"

1

Giuliana, Chiara. "Negotiating home spaces : spatial practices in Italian postcolonial literature." Thesis, University of St Andrews, 2015. http://hdl.handle.net/10023/9764.

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Books on the topic "Pq4305"

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Alighieri, Dante. Dantis Alagherii Comedia. SISMEL edizioni del Galluzzo, 2001.

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Alighieri, Dante. La Divina commedia. Salerno editrice, 2021.

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Alighieri, Dante. La Divina commedia. Classici Rusconi, 2003.

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Alighieri, Dante. Dantes Divina Commedia mit den Illustrationen von Sandro Botticelli: Entstanden Ende des 15. Jahrhunderts. Belser Verlag, 1986.

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Alighieri, Dante. Commedia: Secondo il Ms. XIII C 2 della Biblioteca Nazionale di Napoli. Bibliopolis, 1988.

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Alighieri, Dante. Divina Commedia: Paradiso. Rizzoli, 1992.

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Alighieri, Dante. La Commedìa: Nuovo testo critico secondo i più antichi manoscritti fiorentini. De Rubeis, 1995.

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Alighieri, Dante. Div ina commedia: Inferno; Purgatorio; Paradiso. Tascabili Economici Newton, 1993.

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Alighieri, Dante. The divine comedy. Oxford University Press, 2008.

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Alighieri, Dante. An choiméide dhiaga. Clóchomhar, 1997.

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