Relevant bibliographies by topics / Pramipexole/6-OHDA

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'Pramipexole/6-OHDA'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Pramipexole/6-OHDA"

1

Gao, Hua, Dan Wang, Yu-Ling Wang, Jie-Ping Mao, Sen Jiang та Xin-Ling Yang. "Pramipexole attenuates 6-OHDA-induced Parkinson’s disease by mediating the Nurr1/NF-κB pathway". Molecular Biology Reports 48, № 4 (2021): 3079–87. http://dx.doi.org/10.1007/s11033-021-06343-8.

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2

Cao, Lei-Fang, Xiao-Yan Peng, Ya Huang, et al. "Restoring Spinal Noradrenergic Inhibitory Tone Attenuates Pain Hypersensitivity in a Rat Model of Parkinson’s Disease." Neural Plasticity 2016 (2016): 1–16. http://dx.doi.org/10.1155/2016/6383240.

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In the present study, we investigated whether restoring descending noradrenergic inhibitory tone can attenuate pain in a PD rat model, which was established by stereotaxic infusion of 6-hydroxydopamine (6-OHDA) into the bilateral striatum (CPu). PD rats developed thermal and mechanical hypersensitivity at the 4th week after surgery. HPLC analysis showed that NE content, but not dopamine or 5-HT, significantly decreased in lumbar spinal cord in PD rats. Additional noradrenergic depletion by injection of N-(2-chloroethyl)-N-ethyl-2-bromobenzylamine (DSP-4) aggravated pain hypersensitivity in PD
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3

Marin, C., M. Bonastre, G. Mengod, et al. "Early L-dopa, but not pramipexole, restores basal ganglia activity in partially 6-OHDA-lesioned rats." Neurobiology of Disease 64 (April 2014): 36–47. http://dx.doi.org/10.1016/j.nbd.2013.12.009.

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4

Kemmerer, Elyse S., Timothy J. Desmond, Roger L. Albin, Michael R. Kilbourn, and Kirk A. Frey. "Treatment effects on nigrostriatal projection integrity in partial 6-OHDA lesions: comparison of l -DOPA and pramipexole." Experimental Neurology 183, no. 1 (2003): 81–86. http://dx.doi.org/10.1016/s0014-4886(03)00096-7.

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5

Dardou, D., L. Reyrolle, C. Chassain, and F. Durif. "Chronic pramipexole treatment induces compulsive behavior in rats with 6-OHDA lesions of the substantia nigra and ventral tegmental area." Behavioural Brain Research 332 (August 2017): 327–36. http://dx.doi.org/10.1016/j.bbr.2017.06.016.

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Dissertations / Theses on the topic "Pramipexole/6-OHDA"

1

Magnard, Robin. "Mécanismes physiopathologiques des comportements impulsifs associés à la maladie de Parkinson : approches expérimentales chez le rat." Thesis, Université Grenoble Alpes (ComUE), 2019. http://www.theses.fr/2019GREAV006/document.

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Au-delà des symptômes moteurs, la maladie de Parkinson (MP) est également caractérisée par une myriade de symptômes neuropsychiatriques allant de l’apathie et la dépression aux troubles du contrôle des impulsions (TCI). Les TCI représentent un groupe d’addictions comportementales incluant le jeu pathologique, l’hypersexualité et les achats faits de manière compulsive. Observés chez 10 à 14 % des patients parkinsoniens sous traitement dopaminergique, ils affectent fortement leur qualité de vie. L’impulsivité cognitive reflétant notamment l’incapacité à tolérer les délais de renforcements, est a
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2

Aleksandrova, Lily R. "Preclinical Modeling of Treatment-induced Impulsivity in Parkinson's Disease." Thesis, 2013. http://hdl.handle.net/1807/42665.

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Dopamine agonist therapy and deep brain stimulation (DBS) are both linked to increased impulsivity in Parkinson’s disease (PD), but the underlying mechanisms remain unclear. We trained intact and PD-like rats on a rat gambling task (rGT) measuring impulsive choice and premature responding. Animals were then retested with/without treatment, pramipexole (PPX) or DBS, administered chronically prior to rGT testing. Early PD-like rats did not exhibit major differences in rGT performance or treatment response. Our work suggests that DBS and PPX are not intrinsically linked with increases in impulsi
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