Relevant bibliographies by topics / Progress of the disease curve

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Academic literature on the topic 'Progress of the disease curve'

Author: Grafiati
Published: 4 June 2021
Last updated: 2 February 2022

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Journal articles on the topic "Progress of the disease curve"

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Knudsen, G. R., C. S. Johnson, and H. W. Spurr. "Use Of a Simulation Model to Explore Fungicide Strategies for Control of Cercospora Leafspot of Peanut1." Peanut Science 15, no. 1 (January 1, 1988): 39–43. http://dx.doi.org/10.3146/i0095-3679-15-1-11.

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Abstract A sub-model describing persistence and efficacy of chlorothalonil fungicide was incorporated into a computer simulation model of Cercospora leafspot of peanut. The resultant model was validated using independent data sets from field trials over a two-year period. Predicted disease progress curves and area under the disease progress curve for different fungicide application schedules and rates were compared with field observations. The model was then used to compare predicted disease severity and area under the disease progress curve (AUDPC) for a calendar spray schedule vs a leafspot advisory program under different weather conditions. Predicted disease severity levels and area under disease progress curves were similar for advisory and calendar spray schedules. Results were insensitive to changes in parameters describing fungicide persistence or efficacy. The model described herein is a good estimator of the combined effects of weather and chlorothalonil treatments on disease progress, effectively ranks treatments or environmental conditions in terms of their effect on leafspot, and provides a basis for comparison of fungicide scheduling strategies. The simulation model predicted AUDPC more accurately than end-of-season disease, and AUDPC is a more reliable indicator of the effect of peanut leafspot disease on yield loss. Simulation experiments will be useful in optimizing fungicide or biocontrol strategies for long-term financial benefit to growers.
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GILLIGAN, C. A. "Comparison of disease progress curves." New Phytologist 115, no. 2 (June 1990): 223–42. http://dx.doi.org/10.1111/j.1469-8137.1990.tb00448.x.

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Godoy, Cláudia V., Lílian Amorim, Armando Bergamin Filho, Herbert P. Silva, Willian J. Silva, and Richard D. Berger. "Temporal progress of southern rust in maize under different environmental conditions." Fitopatologia Brasileira 28, no. 3 (June 2003): 273–78. http://dx.doi.org/10.1590/s0100-41582003000300008.

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The progress of the severity of southern rust in maize (Zea mays) caused by Puccinia polysora was quantified in staggered plantings in different geographical areas in Brazil, from October to May, over two years (1995-1996 and 1996-1997). The logistic model, fitted to the data, better described the disease progress curves than the Gompertz model. Four components of the disease progress curves (maximum disease severity; area under the disease progress curve, AUDPC; area under the disease progress curve around the inflection point, AUDPCi; and epidemic rate) were used to compare the epidemics in different areas and at different times of planting. The AUDPC, AUDPCi, and the epidemic rate were analyzed in relation to the weather (temperature, relative humidity, hours of relative humidity >90%, and rainfall) and recorded during the trials. Disease severity reached levels greater than 30% in Piracicaba and Guaíra in the plantings between December and January. Lower values of AUDPC occurred in later plantings at both locations. The epidemic rate was positively correlated (P < 0.05) with the mean daily temperatures and negatively correlated with hours of relative humidity >90%. The AUDPC was not correlated with any weather variable. The AUDPCi was negatively related to both variables connected to humidity, but not to rain. Long periods (mostly >13 h day-1) of relative humidity >90% (that corresponded to leaf wetness) occurred in Castro. Severity of southern rust in maize has always been low in Castro, thus the negative correlations between disease and the two humidity variables.
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Navas-Cortés, Juan A., Bernhard Hau, and Rafael M. Jiménez-Díaz. "Effect of Sowing Date, Host Cultivar, and Race of Fusarium oxysporum f. sp. ciceris on Development of Fusarium Wilt of Chickpea." Phytopathology® 88, no. 12 (December 1998): 1338–46. http://dx.doi.org/10.1094/phyto.1998.88.12.1338.

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Microplots experiments were carried out at Córdoba, southern Spain, from 1986 to 1989 to determine the effects of sowing date in the management of Fusarium wilt of chickpea as influenced by virulence of the pathogen race and by cultivar susceptibility. A total of 108 epidemics of the disease were described, analyzed, and compared to assess the degree of disease control. The epidemics were characterized by five curve elements: final disease intensity index (DII), standardized area under DII progress curve, time to epidemic onset, time to inflection point (tip), and the DII value at tip, the last two parameters being estimates from the Richards function adjusted by nonlinear regression analysis. The structure of Fusarium wilt epidemics was examined by conducting multivariate principal components and cluster analyses. From these analyses, three factors accounting for 98 to 99% of the total variance characterized the DII progress curves and provided plausible epidemiological interpretations. The first factor included the tip and the time to disease onset and can be interpreted as a positional factor over time. This factor accounted for the largest proportion of the total variance and may, therefore, be considered as the main factor for analysis of Fusarium wilt epidemics. The second factor concerns the standardized area under DII progress curves and the final DII of the epidemics. The third factor identified the uniqueness of the estimated value for the point of inflection of the DII progress curve over time. Our results indicate that for each year of experiment epidemic development was related mainly to the date of sowing. Thus, for chickpea crops in southern Spain, advancing the sowing date from early spring to early winter can slow down the development of Fusarium wilt epidemics, delay the epidemic onset, and minimize the final amount of disease. However, the net effect of this disease management practice may also be influenced, though to a lesser extent, by the susceptibility of the chickpea cultivar and the virulence and inoculum density of the Fusarium oxysporum f. sp. ciceris race.
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MUKHERJEE, A. K., N. K. MOHAPATRA, A. V. SURIYA RAO, and P. NAYAK. "Effect of nitrogen fertilization on the expression of slow-blasting resistance in rice." Journal of Agricultural Science 143, no. 5 (September 30, 2005): 385–93. http://dx.doi.org/10.1017/s0021859605005551.

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The development of rice blast disease in four slow-blasting (SB) genotypes was compared with that in the fast-blasting (FB) genotype Karuna, under natural field epidemics over a period of 3 years at five levels of nitrogen, in order to determine if the application of high doses of nitrogen influenced the expression of disease progress in the SB types. The treatment effects were compared through estimation of nine parameters viz. (i) lesion number (LN); (ii) area under disease progress curve (AUDPC); (iii) relative area under disease progress curve (RAUDPC); (iv) logistic apparent infection rate (r); (v) Gompertz apparent infection rate (k); (vi) logit line intercept (logit-a); (vii) gompit line intercept (gompit-a); (viii) time required for the disease to reach 0·25 severity in logistic (T25r); and (ix) Gompertz (T25k) models. There was a significant increase in LN, AUDPC, RAUDPC, r and k with increased levels of nitrogen application in all genotypes, but the rate of increase in disease severity was much lower in SB genotypes than the FB one and did not lead to breakdown of resistance in the SB genotypes, since severity level was much below the economic injury level. Among the nine derived parameters for evaluation of resistance LN, AUDPC, RAUDPC, r and k were best. The AUDPC and RAUDPC had lower degrees of error variance compared with the other parameters and hence were considered superior measures for characterization of disease progress curves.
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Fernández-Campos, M., C. Góngora-Canul, S. Das, M. R. Kabir, B. Valent, and C. D. Cruz. "Epidemiological Criteria to Support Breeding Tactics Against the Emerging, High-Consequence Wheat Blast Disease." Plant Disease 104, no. 8 (August 2020): 2252–61. http://dx.doi.org/10.1094/pdis-12-19-2672-re.

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Plant disease epidemiology can make a significant contribution for cultivar selection by elucidating the principles of an epidemic under different levels of resistance. For emerging diseases as wheat blast (WB), epidemiological parameters can provide support for better selection of genetic resources. Field experiments were conducted at two locations in Bolivia in 2018–2019 to characterize the temporal dynamics of the disease on 10 cultivars with different levels of reaction to WB. Logistic models best (R2 = 0.70–0.96) fit the disease progress curve in all cultivars followed by Gompertz (R2 = 0.64–0.94), providing additional evidence of a polycyclic disease. Total area under disease progress curve (tAUDPC), final disease severity (Ymax), and logistic apparent infection rates (rL*) were shown to be appropriate epidemiological parameters for describing resistance and cultivar selection. Cultivars that showed a high spike AUDPC (sAUDPC) showed a high leaf AUDPC (lAUDPC). tAUPDC, Ymax, and rL* were positively correlated among them (P < 0.01) and all were negatively correlated with grain weight (P < 0.01). Based on the epidemiological parameters used, cultivars that showed resistance to WB were Urubó, San Pablo, and AN-120, which were previously reported to have effective resistance against the disease under field conditions. The information generated could help breeding programs to make technical decisions about relevant epidemiological parameters to consider prior to cultivar release.
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Pereira, Renata C. M., Maria A. Ferreira, Thaissa P. F. Soares, Mario F. C. M. Andrade, Cézar A. L. Filho, Edson A. Pozza, Maria L. M. Avelar, and Lucas A. Melo. "Temporal Progress of Candeia Rust Caused by Puccinia velata in Clonal Candeia Plantation (Eremanthus erythropappus (DC.) McLeisch)." Forests 11, no. 7 (June 29, 2020): 720. http://dx.doi.org/10.3390/f11070720.

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The temporal progress of candeia rust, caused by the fungus Puccinia velata, was monitored in an experimental field at Lavras municipality, Southern Minas Gerais state, Brazil. A plantation with 17 Eremanthus erythropappus clones was set at the site, and the temporal disease progress was analyzed based on visual assessments of disease severity on leaves. The disease was monitored monthly between September 2016 and August 2017. Progress curves based on disease severity were constructed and empirical models were fitted. The area under the disease progress curve (AUDPC) was calculated, and the means test was applied to select clones resistant to the disease. The Pearson coefficient was used to assess correlations between disease severity and environmental variables. The model that best described disease progress over the assessment period was the Gompertz model. The mean AUDPC values were grouped into four groups of resistance levels according to the Scott–Knott test. There was a negative correlation between air temperature and disease severity. Considering that the disease occurred in all clones and that the climatic conditions of Southern Minas Gerais are favorable to the candeia rust, it is important to adopt measures for the selection of clones resistant to this disease.
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Madden, L. V., G. Hughes, and M. E. Irwin. "Coupling Disease-Progress-Curve and Time-of-Infection Functions for Predicting Yield Loss of Crops." Phytopathology® 90, no. 8 (August 2000): 788–800. http://dx.doi.org/10.1094/phyto.2000.90.8.788.

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A general approach was developed to predict the yield loss of crops in relation to infection by systemic diseases. The approach was based on two premises: (i) disease incidence in a population of plants over time can be described by a nonlinear disease progress model, such as the logistic or monomolecular; and (ii) yield of a plant is a function of time of infection (t) that can be represented by the (negative) exponential or similar model (ζ(t)). Yield loss of a population of plants on a proportional scale (L) can be written as the product of the proportion of the plant population newly infected during a very short time interval (X′(t)dt) and ζ(t), integrated over the time duration of the epidemic. L in the model can be expressed in relation to directly interpretable parameters: maximum per-plant yield loss (α, typically occurring at t = 0); the decline in per-plant loss as time of infection is delayed (γ; units of time-1); and the parameters that characterize disease progress over time, namely, initial disease incidence (X0), rate of disease increase (r; units of time-1), and maximum (or asymptotic) value of disease incidence (K). Based on the model formulation, L ranges from αX0 to αK and increases with increasing X0, r, K, α, and γ-1. The exact effects of these parameters on L were determined with numerical solutions of the model. The model was expanded to predict L when there was spatial heterogeneity in disease incidence among sites within a field and when maximum per-plant yield loss occurred at a time other than the beginning of the epidemic (t > 0). However, the latter two situations had a major impact on L only at high values of r. The modeling approach was demonstrated by analyzing data on soybean yield loss in relation to infection by Soybean mosaic virus, a member of the genus Potyvirus. Based on model solutions, strategies to reduce or minimize yield losses from a given disease can be evaluated.
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LAL AHAMED, M., S. S. SINGH, J. B. SHARMA, and R. B. RAM. "Evaluation of inheritance to leaf rust in wheat using area under disease progress curve." Hereditas 141, no. 3 (February 8, 2005): 323–27. http://dx.doi.org/10.1111/j.1601-5223.2004.01794.x.

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Simko, Ivan, and Hans-Peter Piepho. "The Area Under the Disease Progress Stairs: Calculation, Advantage, and Application." Phytopathology® 102, no. 4 (April 2012): 381–89. http://dx.doi.org/10.1094/phyto-07-11-0216.

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The area under the disease progress curve (AUDPC) is frequently used to combine multiple observations of disease progress into a single value. However, our analysis shows that this approach severely underestimates the effect of the first and last observation. To get a better estimate of disease progress, we have developed a new formula termed the area under the disease progress stairs (AUDPS). The AUDPS approach improves the estimation of disease progress by giving a weight closer to optimal to the first and last observations. Analysis of real data indicates that AUDPS outperforms AUDPC in most of the tested trials and may be less precise than AUDPC only when assessments in the first or last observations have a comparatively large variance. We propose using AUDPS and its standardized (sAUDPS) and relative (rAUDPS) forms when combining multiple observations from disease progress experiments into a single value.
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Dissertations / Theses on the topic "Progress of the disease curve"

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Moreira, Renato Cesar. "ReaÃÃo de clones de cajueiro comum à resinose." Universidade Federal do CearÃ, 2011. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=7925.

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Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico
O cajueiro (Anacardium occidentale) no nordeste brasileiro caracterizando-se como uma das mais importantes fontes de emprego e renda das populaÃÃes. O lanÃamento de clones de cajueiro tipo anÃo-precoce aumentou a produtividade e qualidade das castanhas e pendÃculos, permitindo uma exploraÃÃo comercial mais vantajosa aos produtores, porem a uniformidade genÃtica deixou os pomares mais vulnerÃveis. A resinose hoje à considerada a principal doenÃa do cajueiro no semiÃrido nordestino. Esse estudo teve por objetivo avaliar a reaÃÃo de genÃtipos de cajueiro do tipo comum à resinose sob condiÃÃes de elevada incidÃncia no semiÃrido. Vinte clones de cajueiro-comum previamente selecionados pelo programa de melhoramento genÃtico da Embrapa AgroindÃstria Tropical foram usados no estudo e dessa forma foi possÃvel concluir que hà fontes de resistÃncia para uso em programas de melhoramento. Os clones CNPAT 06, CNPAT 08, CNPAT 11, CNPAT 12 e CNPAT 13 se apresentara como os mais resistentes a resinose, enquanto que os clones CNPAT 07, CNPAT 09, CNPAT 14, COMUM 05 e COMUM 31 forma os que obtiveram maiores notas de severidade de resinose. Outro importante resultado obtido foi que os clones CNPAT 08, CNPAT 11 e CNPAT 15 apresentaram capacidade de recuperaÃÃo da resinose. Por fim, sugere-se que com a avaliaÃÃo da incidÃncia à possÃvel estimar a severidade de resinose em pomares de cajueiro.
The cashew nut (Anacardium occidentale) represents a social and economic tradition in Brazilian northeastern as one of the most important source of income and labor for its people. The releasing of early-dwarf clones contributed to increase yield and nut and apple quality, allowing gain in profit by small growers, in spite of increase genetic vulnerability of orchards. Cashew gummosis is presently the most important disease of this species in semi-arid regions of northeast. This study aimed to evaluate reactions of common selected clones of cashew to gummosis under high disease pressure of semi-arid region. Twenty clones selected by the cashew breeding program of Embrapa AgroindÃstria Tropical were evaluated for three consecutive years. Clones named CNPAT 06, CNPAT 08, CNPAT 11, CNPAT 12 and CNPAT 13 attained the highest levels of resistance, while CNPAT 07, CNPAT 09, CNPAT 14, COMUM 05 and COMUM 31 otherwise were the most susceptible ones. It was also found that clones CNPAT 08, CNPAT 11 and CNPAT 15 are able to heal gummosis cankers, which points to a new kind of resistance to be considered. Finally, it was shown that monitoring gummosis incidence it is possible to estimate disease severity.
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AGUIAR, Renata Alves de. "Manejo do mofo branco (Sclerotinia sclerotiorum L.) em tomateiro industrial." Universidade Federal de Goiás, 2011. http://repositorio.bc.ufg.br/tede/handle/tde/444.

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Brazil ranks ninth in the world production of tomato (Solanum esculentum L.), while much of this production is destined for processing industries. The added value obtained with the fruit processing endorses this species as the vegetable with the greatest economic importance in the Cerrado region of Brazil, where the State of Goiás stands out as the largest producer. Despite the favorable soil and climatic conditions, several factors have hindered its production, especially diseases caused by soilborne pathogens, which had increased their importance with the adoption of intensive production systems. Among them, white mold caused by Sclerotinia sclerotiorum (Lib.) De Bary has caused serious problems in infested soils, under mild temperatures and high humidity. Concerning the large number of S. sclerotiorum hosts and the lack of resistant hybrids, chemical control has been chosen as the most common method for disease management, despite not always efficient, due to the difficulties to reach the pathogen s resistance structures in the soil. Therefore, the study aimed to evaluate the disease escape on different tomato hybrids, to compare the effectiveness of synthetic fungicides mixed or not to potassium silicate; evaluate the biological control of white mold, with different commercial products based on Trichoderma spp. associated or not to a synthetic fungicide, and to evaluate the biological control with or without chemical fungicides, applied through chemigation. Tests were carried out in soil of medium texture at Unilever experimental farm, in Goiânia (GO), from 2008 to 2010. The experimental area was previously infested with sclerotia of the pathogen, obtained in pre-cleaning wastes of soybean. Drip irrigation was used in all tests, which had 4 plants m-1 with 1.5 meters between rows. The hybrids used in the disease escape tests were: U232, U2006 (Unilever), H9992, H7155 (Heinz), N877 (Nunhems) and H108 (Hypeel). In all other essays, Heinz 9780 was the chosen hybrid. All experiments were conducted under randomized blocks design with three replications, and had weekly assessments of disease incidence to estimate the area under the disease progress curve (AUDPC). We also evaluated the productivity and its components, as well as acidity, soluble solids content and industrial yield. The results were submitted to ANOVA and to the Scott Knott or Tukey tests at 5%, using the statistical program Sisvar. It was shown that: Hybrids H9992 and Hp108 had lower AUDPCs, suggesting a partial escape to white mold and that hybrid choice can be added to the disease management cultural practices, despite there was no difference on their yield. In 2008, under higher disease pressure, potassium silicate in plots without fungicide application showed disease incidence, AUDPC, productivity and industrial yield equivalent to treatments with fluazinam and procymidone, and superior to results with benzalkonium chloride. There was no difference between treatments with Trichoderma spp. associated or not to fluazinam, in any of the assessed traits. Regarding industrial yield, there was an interaction between Trichoderma, fluazinam and years, with higher pulp yield under higher disease incidence and fluazinam sprayed alone. It was found that biological control with Trichoderma spp. via chemigation as a single measure or in mixture with the synthetic fungicides procymidone fluazinam reduced the AUDPC and increased the productivity of processing tomatoes in 25 tons on average, compared to the control. Therefore, this study showed new options for the integrated management of white mold in processing tomatoes.
O Brasil ocupa o nono lugar na produção mundial de tomate (Solanum esculentum L.), sendo que grande parte desta produção é destinada a indústrias de processamento. A agregação de valor obtida com o processamento torna esta espécie a hortaliça de maior importância econômica na região do Cerrado do Brasil, onde o Estado de Goiás se destaca como maior produtor. Apesar das condições edafo-climáticas favoráveis, vários fatores têm dificultado sua produção, principalmente as doenças provocadas por patógenos habitantes do solo, que tem aumentado sua importância em sistemas de produção intensivos. Dentre elas, o mofo branco provocado por Sclerotinia sclerotiorum (Lib.) De Bary causa sérios problemas em solos infestados, sob condições de temperatura amena e alta umidade. Por ser um patógeno polífago e não existirem híbridos resistentes, o controle químico tem sido o método mais utilizado no manejo da doença, apesar de nem sempre ser eficiente pela dificuldade de atingir as estruturas de resistência do patógeno no solo. Dessa forma, este trabalho teve como objetivo avaliar diferentes híbridos quanto à arquitetura da planta para escape da doença; comparar a aplicação de fungicidas sintéticos com e sem a utilização de silício; avaliar o controle biológico do mofo branco por meio de diferentes produtos comerciais elaborados a partir de isolados do fungo Trichoderma spp., associados ou não com a aplicação de fungicida sintético, via barra de pulverização ou via fungigação. Foram conduzidos ensaios na fazenda experimental da Unilever, em Goiânia (GO), nos anos de 2008 a 2010, em solo de textura média. A área experimental foi previamente infestada com escleródios do patógeno, obtidos em resíduos de pré-limpeza de soja. A irrigação em todos os experimentos foi realizada por gotejamento em parcelas com estande de 4 plantas m-1 com 1,5 metro entre linhas. Os híbridos utilizados para avaliação quanto à arquitetura da planta foram: U232, U2006 (Unilever), H9992, H7155 (Heinz), N877 (Nunhems) e Hp108 (Hypeel). Já o híbrido utilizado nos demais ensaios foi o Heinz 9780. Todos os experimentos foram conduzidos sob delineamento experimental de blocos ao acaso, com três repetições, e tiveram avaliações semanais da incidência da doença para obtenção da área abaixo da curva de progresso da doença (AACPD). Avaliou-se ainda a produtividade e seus componentes, além de pH, teor de sólidos solúveis e rendimento de polpa. Os resultados foram submetidos à ANOVA e ao teste de Scott-Knott ou Tukey (5%) com auxílio do programa estatístico Sisvar. Verificou-se que: Em ambos os anos, a menor AACPD nos híbridos H9992 e Hp108 (Scott-Knott 5%), é creditada ao escape parcial da doença, devido ao porte das plantas e concentração de maturação, demonstrando que a escolha do híbrido pode ser adicionada às práticas culturais já utilizadas para o manejo da doença. Apesar de não ter havido diferenças entre os híbridos quanto à sua produtividade, as perdas na produção foram correlacionadas à incidência da doença e à AACPD. Em ano com elevada incidência os fungicidas (fluazinam e procimidona) são eficientes e silicato de potássio, sem aplicação de fluazinam ou procimidona, foi igualmente eficiente, podendo ser utilizado na agricultura orgânica. No controle do mofo branco, via barra de pulverização, na cultura do tomate para processamento industrial, o uso do Trichodermax e Trichodermil não diferiram do padrão de controle em relação a incidência da doença, produtividades e rendimentos. Não há influência do fluazinam via barra de pulverização no controle do mofo branco. Já para o rendimento de polpa há interação entre Trichoderma, ano e fluazinam, sendo que ocorreu um maior rendimento de polpa no ano de maior incidência com o uso do fluazinam. Verificou-se que o controle biológico com utilização do Trichoderma sp., via fungigação, para o mofo branco, isolado ou em combinação com os fungicidas sintéticos fluazinam e procimidona, reduz a AACPD e incrementa a produtividade do tomate para processamento industrial em até 25 toneladas ha-1 em média. O rendimento de polpa nos tratamentos com controle biológico foi aumentado em cerca de 1,0 e 7,0 t ha-1, respectivamente, em 2009 e 2010.
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Lima, Lonjoré Leocádio de [UNESP]. "Reação de cultivares de algodoeiro a Ramularia areola Atk." Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/96913.

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Dentre as doenças causadoras de manchas foliares em algodoeiro, a mancha de ramulária tem se tornado importante em virtude das condições climáticas favoráveis para o desenvolvimento do patógeno, aliadas ao uso de cultivares suscetíveis, plantio consecutivo e extenso. Neste trabalho avaliaram-se as cultivares BRS Antares, BRS Araçá, BRS Aroeira, BRS Buriti, BRS Cedro, BRS Facual, BRS Ipê, BRS Sucupira, BRS 11612, Campo Verde 02, Campo Verde 05, Coodetec 409, Delta Opal, Deltapine Acala 90, Fiber Max 966, FMT 501, FMT 701, CNPA Ita 90, BRS Ita 96 e SL 502, quanto a resistência a Ramularia areola, em condições de campo na FCAV-UNESP no ano de 2006/2007. A severidade da doença, que ocorreu por infecção natural das plantas pelo fungo, foi avaliada semanalmente em trinta plantas previamente marcadas, utilizando uma escala descritiva de notas: 1 = 0%, 2 = Até 5%, 3 = de 5,1 a 25%, 4 = de 25,1 a 50% e 5 = Acima de 50% de área foliar com sintomas. O modelo de Gompertz foi o que melhor se ajustou aos dados de severidade de mancha de Ramulária em algodoeiro nas condições climáticas de Jaboticabal – SP no ano 2007. As cultivares Campo Verde 05, BRS 11612 e Deltapine Acala 90 foram as mais resistentes à doença. As cultivares BRS Ita 96, Campo Verde 02, BRS Facual, CNPA Ita 90 e BRS Buriti que apresentaram uma suscetibilidade intermediária à doença. As cultivares BRS Antares, BRS Aroeira, Delta Opal, SL 502, BRS Araçá, Coodetec 409, BRS Ipê, FMT 701, BRS Sucupira, BRS Cedro, Fiber Max 966 e FMT 501, mostraram – se mais suscetíveis.
Among the disease causing leaves spots in cotton plant, the gray mildew (Ramularia areola Atk.) has become important an disease in virtue of the environmental conditions favorable for development of the pathogens allread to the use of susceptible cultivars, consecutive and extensive plantation In this work the following cultivars were evaluated for the resistence to R. areola: BRS Antares, BRS Araçá, BRS Aroeira, BRS Buriti, BRS Cedro, BRS Facual, BRS Ipê, BRS Sucupira, BRS 11612, Campo Verde 02, Campo Verde 05, Coodetec 409, Delta Opal, Deltapine Acala 90, Fiber Max 966, FMT 501, FMT 701, CNPA Ita 90, BRS Ita 96 e SL 502. The experiment was carried out at the UNESP/FCAV experimental area in field conditions during 2006/2007. The disease severity occurred for natural infection and, the evolutions were made weekly in thirty marked plants using the scale: 1 = 0%, 2 = up to 5%, 3 = 5,1 to 25%, 4 = 25,1 to 50% and 5 = over of 50% of foliar area with symptoms. The Gompertz model was the better for the adjustment of the data of disease severity related to environmental conditions of Jaboticabal for 2006/2007. The cultivars Campo Verde 05, BRS 11612 and Deltapine Acala 90 were the most resistant to the disease. The cultivars BRS Ita 96, Campo Verde 02, BRS Facual, CNPA Ita 90 and BRS Buriti were moderately susceptible, while BRS Antares, BRS Aroeira, Delta Opal, SL 502, BRS Araçá, Coodetec 409, BRS Ipê, FMT 701, BRS Sucupira, BRS Cedro, Fiber Max 966 and FMT 501 were the most susceptible.
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Lima, Lonjoré Leocádio de. "Reação de cultivares de algodoeiro a Ramularia areola Atk /." Jaboticabal : [s.n.], 2007. http://hdl.handle.net/11449/96913.

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Orientador: Modesto Barreto
Banca: Margarete Camargo
Banca: Érika Auxiliadora Giacheto Scaloppi
Resumo: Dentre as doenças causadoras de manchas foliares em algodoeiro, a mancha de ramulária tem se tornado importante em virtude das condições climáticas favoráveis para o desenvolvimento do patógeno, aliadas ao uso de cultivares suscetíveis, plantio consecutivo e extenso. Neste trabalho avaliaram-se as cultivares BRS Antares, BRS Araçá, BRS Aroeira, BRS Buriti, BRS Cedro, BRS Facual, BRS Ipê, BRS Sucupira, BRS 11612, Campo Verde 02, Campo Verde 05, Coodetec 409, Delta Opal, Deltapine Acala 90, Fiber Max 966, FMT 501, FMT 701, CNPA Ita 90, BRS Ita 96 e SL 502, quanto a resistência a Ramularia areola, em condições de campo na FCAV-UNESP no ano de 2006/2007. A severidade da doença, que ocorreu por infecção natural das plantas pelo fungo, foi avaliada semanalmente em trinta plantas previamente marcadas, utilizando uma escala descritiva de notas: 1 = 0%, 2 = Até 5%, 3 = de 5,1 a 25%, 4 = de 25,1 a 50% e 5 = Acima de 50% de área foliar com sintomas. O modelo de Gompertz foi o que melhor se ajustou aos dados de severidade de mancha de Ramulária em algodoeiro nas condições climáticas de Jaboticabal - SP no ano 2007. As cultivares Campo Verde 05, BRS 11612 e Deltapine Acala 90 foram as mais resistentes à doença. As cultivares BRS Ita 96, Campo Verde 02, BRS Facual, CNPA Ita 90 e BRS Buriti que apresentaram uma suscetibilidade intermediária à doença. As cultivares BRS Antares, BRS Aroeira, Delta Opal, SL 502, BRS Araçá, Coodetec 409, BRS Ipê, FMT 701, BRS Sucupira, BRS Cedro, Fiber Max 966 e FMT 501, mostraram - se mais suscetíveis.
Abstract: Among the disease causing leaves spots in cotton plant, the gray mildew (Ramularia areola Atk.) has become important an disease in virtue of the environmental conditions favorable for development of the pathogens allread to the use of susceptible cultivars, consecutive and extensive plantation In this work the following cultivars were evaluated for the resistence to R. areola: BRS Antares, BRS Araçá, BRS Aroeira, BRS Buriti, BRS Cedro, BRS Facual, BRS Ipê, BRS Sucupira, BRS 11612, Campo Verde 02, Campo Verde 05, Coodetec 409, Delta Opal, Deltapine Acala 90, Fiber Max 966, FMT 501, FMT 701, CNPA Ita 90, BRS Ita 96 e SL 502. The experiment was carried out at the UNESP/FCAV experimental area in field conditions during 2006/2007. The disease severity occurred for natural infection and, the evolutions were made weekly in thirty marked plants using the scale: 1 = 0%, 2 = up to 5%, 3 = 5,1 to 25%, 4 = 25,1 to 50% and 5 = over of 50% of foliar area with symptoms. The Gompertz model was the better for the adjustment of the data of disease severity related to environmental conditions of Jaboticabal for 2006/2007. The cultivars Campo Verde 05, BRS 11612 and Deltapine Acala 90 were the most resistant to the disease. The cultivars BRS Ita 96, Campo Verde 02, BRS Facual, CNPA Ita 90 and BRS Buriti were moderately susceptible, while BRS Antares, BRS Aroeira, Delta Opal, SL 502, BRS Araçá, Coodetec 409, BRS Ipê, FMT 701, BRS Sucupira, BRS Cedro, Fiber Max 966 and FMT 501 were the most susceptible.
Mestre
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Sposito, Marcel Bellato. "Dinâmica temporal e espacial da mancha preta (Guignardia citricarpa) e quantificação dos danos causados à cultura dos citros." Universidade de São Paulo, 2004. http://www.teses.usp.br/teses/disponiveis/11/11135/tde-19042004-133413/.

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A mancha preta dos citros (MPC), incitada pelo fungo Guignardia citricarpa, causa lesões em frutos depreciando-os para a comercialização no mercado interno de fruta fresca, restringindo-os para a exportação e pode causar queda prematura. Para auxiliar nos estudos epidemiológicos, foi desenvolvida uma escala diagramática para a severidade da MPC que contempla os dois tipos de sintomas usualmente observados no campo, “mancha dura”, cujos valores variam entre 0,5 e 49,0%, e “falsa melanose”, cujos valores variam entre 1,1 e 68%. O grau de suscetibilidade das variedades cítricas ‘Hamlin’, ‘Pera’ e ‘Valência’ à MPC foi avaliado em pomar comercial, sob infecção natural. O modelo monomolecular ajustou-se às curvas de progresso da incidência e da severidade da doença para as três variedades. Não houve diferença estatística significativa entre as taxas de progresso da MPC, concluindo-se que as três variedades possuem o mesmo grau de suscetibilidade à doença. O agente causal da MPC em sua fase epidêmica forma ascósporos e conídios. Os ascósporos, formados em folhas em decomposição no solo, são disseminados a curtas e longas distâncias pela ação do vento. Os conídios, formados em ramos e frutos fixados à planta, são disseminados a curtas distâncias pela ação da água. O padrão de distribuição espacial de plantas com MPC foi avaliado pelo índice de dispersão e pela função K de Ripley. As plantas doentes mostraram-se agregadas independentemente da incidência da doença, indicando elevada importância da dispersão do inóculo a curtas distâncias. Avaliou-se, também, o padrão de distribuição de frutos sintomáticos dentro da planta, pelo índice de dispersão e pela forma binária da lei de Taylor. Pelo índice de dispersão 84% das plantas avaliadas mostraram agregação de frutos sintomáticos. Pela lei de Taylor concluiu-se que a agregação de frutos sintomáticos independe da incidência da doença na planta, sugerindo a participação dos conídios no incremento da doença em condições de campo. Estudou-se o efeito da supressão de ascósporos, pela retirada periódica de folhas cítricas em decomposição do campo e o de conídios, pela colheita antecipada de frutos de maturação tardia, na intensidade da MPC, por dois anos, em área com alta pressão de inóculo. Esses tratamentos reduziram a severidade da doença, no segundo ano de avaliação, entretanto não foram suficientes para controlá-la adequadamente. Estas medidas de sanitização são importantes no controle da MPC, devendo ser adotadas em conjunto com o controle químico. Danos causados pela redução na produtividade e perdas medidas pelo retorno financeiro foram avaliados em dois experimentos. Tratamentos cujo incremento na produção por hectare que, em comparação com a isenção de tratamento, excederam 100 caixas de 40,8 kg, foram rentáveis. A amostragem de áreas para diagnosticar a presença da MPC depende da finalidade da produção. Em áreas para exportação e mercado interno de fruta fresca deve-se avaliar todas as plant as do talhão, uma vez que esta doença é quarentenária e causa danos qualitativos. Em áreas para a indústria de suco cítrico concentrado, 285 plantas são suficientes para amostrar incidências superiores a 15% de MPC, em pomares médios de 2200 plantas.
The fungus Guignardia citricarpa is the causal agent of citrus black spot (CBS), which is a disease that become the fruits unsightly and unsuitable for the fresh fruit market. Besides, premature fruit drop may occur, reducing the productivity. A diagrammatic scale for the two symptoms of CBS (hard spot and false melanose) was developed to evaluate the severity and help in epidemiological studies of this disease. The susceptibility level of ‘Hamlin’, ‘Pera’ and ‘Valência’ sweet orange to CBS was evaluated in commercial orchard, under natural infection. The monomolecular model was fitted to the incidence and severity progress curves of the disease for the three sweet oranges. According to the progress curves, there was not significant difference among cultivars, showing that ‘Hamlin’, ‘Pera’ and ‘Valência’ sweet orange have similar susceptibility level to CBS. G. citricarpa in the epidemic phase produce ascospores and conidia. The ascospores, produced in leaves on soil, are disseminated by wind, while conidia, produced in plant twigs and fruits, are disseminated by water. The pattern of spatial distribution of CBS plants was evaluated by dispersion index and Ripley K function. The groups of symptomatic plants were distributed independently of disease incidence, suggesting that short distance dispersion of the inocullum could be the most important factor in distribution of the disease. The binary form of Taylor´s power law and dispersion index were used to evaluate the distribution of symptomatic fruits in the plant. According to dispersion index 84% o f the evaluated plants showed aggregation of symptomatic fruits, while by the Taylor´s power law the symptomatic fruits aggregation occurred independently of the incidence of plant disease, suggesting that the increase of disease in field conditions is related to conidia production. The effect of ascospore suppression on intensity of CBS was evaluated by removing citrus leaves from the orchard soil surface, while the conidia suppression was evaluated by early harvesting of late maturation fruits. These treatments, carried out during two years in a high inocullum incidence area, reduced the disease severity in the second year, but the satisfactory control was not observed. However, these treatments could be important in CBS control whether used in association with chemical control. Damages caused by yield reduction and losses of financial return were evaluated in two experiments. In relation to non-treated orchards, those treated orchards where the production/ha increased more than hundred boxes (40.8 kg each box) were considered profitable. The destination of the orange production (juice of fresh fruit market) are important for determine the size of samples to assess the CBS incidence. Since the G. citricarpa fungus is considered a quarentine organism that causes qualitative losses, in areas used for production of fresh fruit market whole orchard has to be evaluated. However, in areas used for production of fruits to concentrated orange juice, 285 plants sampled are enough to determinate the CBS incidence superior to 15%, in average orchards containing 2200 plants.
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Araujo, Maria Nilsa Martins de. "Análise de sobrevivência do tomateiro a Phytophthora infestans." Universidade Federal de Viçosa, 2008. http://locus.ufv.br/handle/123456789/4014.

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Reburning caused by Phytophthora infestansis is characterized as an aggressive disease of great destructive impact, capable of limiting or even hindering the economic cultivation of the tomato plant under conditions of high humidity and low temperatures. In view of the problems reburning can cause to tomato plant crops, this work aimed to: 1) fit models to describe the progress of the disease and form groups of tomato accesses with similar curves; 2) estimate data referring to the number of days to reach 5% severity of the disease, by means of inverse regression; 3) fit survival curves by means of the Kaplan-Meier estimator for the access groups and compare them by means of the Logrank test;4)fit survival curves by means of probabilistic models and compare these curves with Kaplan Meir´s non-parametric technique. Using tomato reburning real data, it was possible to fit the exponential model (Y = y0 exp (rX)) to describe the disease s progress. The means of the parameter estimates were submitted to grouping analysis using the centroid method, generating 10 access groups. Time up to 5% of the disease was calculated via inverse regression. Non-parametric techniques were used to estimate survival function by means of the Kaplan-Meier´s estimator to compare the survival curves by the Logrank test .The survival function was also fit using the probabilistic models, exponential Weibull and Log-normal, respectively, which were compared by means of the verisimilitude ratio test (VRT), considering the generalized Gamma model, as a general case for these models. The methodology applied allowed fitting the exponential model to describe tomato plant reburning progress and to regroup the accesses studied in the 10 groups. The access BGH-6 obtained a smaller disease progress than the others, thus characterizing its higher resistance to the disease; An inverse regression allowed time estimation up to the occurrence of 5% of the severity of the tomato plant reburning. The Kaplan-Meier ´s non-parametric technique allowed estimating the survival curves of the tomato plant accesses belonging to the groups 1, 2, 4, 6 and 8. Utilizing the Logrank test, it could be concluded that most two-by-two comparisons were significant (p<0.05), except in the comparisons of groups 2x4, 4x8 and 6x8. The use of the probabilistic models, exponential Weibull and Log-normal allowed estimating the survival curves of groups 2, 4, 6 and 8, except for group 4, to which the Weibull model was not adequate. Comparing the probabilistic models with the non-parametric technique, the curves of the probabilistic models of groups 2 and 4 presented satisfactory results, compared to the curve estimated by Kaplan-Meier.
A requeima causada por Phytophthora infestans caracteriza-se por ser uma doença agressiva e de grande impacto destrutivo, podendo limitar ou até mesmo impedir o cultivo econômico do tomateiro sob condições de alta umidade e baixas temperaturas. Diante dos problemas que a requeima pode provocar às lavouras de tomate, este trabalho teve por objetivos: 1) ajustar modelos para descrever o progresso da doença e formar grupos de acessos de tomateiro com curvas semelhantes; 2) estimar dados referentes ao número de dias até atingir 5% de severidade da doença, por meio de regressão inversa; 3) ajustar curvas de sobrevivência por meio do estimador de Kaplan-Meier para grupos de acessos e compará-las mediante o uso do teste Logrank; 4) ajustar curvas de sobrevivência por meio de modelos probabilísticos e compará-las com a técnica não-paramétrica de Kaplan-Meier. Utilizando dados reais sobre a requeima do tomateiro, foi possível ajustar o modelo exponencial (Y = y0 exp (rX)) para descrever o progresso da doença. As médias das estimativas dos parâmetros foram submetidas à análise de agrupamento pelo método Centróide, o que gerou 10 grupos de acessos, sendo o tempo até a incidência de 5% da doença calculado via regressão inversa. Foram utilizadas técnicas não-paramétricas para estimar a função de sobrevivência por meio do estimador de Kaplan-Meier e para comparar as curvas de sobrevivência pelo teste Logrank. Foi também ajustada a função de sobrevivência, empregando-se os modelos probabilísticos Exponencial, Weibull e Log-normal, os quais foram comparados por meio do Teste da Razão da Verossimilhança (TRV), considerando-se o modelo Gama generalizado por ser caso geral para esses modelos. A metodologia utilizada permitiu ajustar o modelo Exponencial para descrever o progresso da requeima do tomateiro e agrupar os acessos estudados em 10 grupos. O acesso BGH-6 sofreu um progresso de doença menor que os demais, caracterizando-se, assim, sua maior resistência à enfermidade. A regressão inversa possibilitou estimar o tempo até a ocorrência de 5% da severidade da requeima do tomateiro. Pela técnica não-paramétrica de Kaplan-Meier, foi possível estimar as curvas de sobrevivência dos acessos de tomateiro pertencentes aos grupos 1, 2, 4, 6 e 8. Utilizando o teste Logrank, pode-se concluir que a maioria das comparações duas a duas foi significativa (p<0,05), exceto nas comparações dos grupos 2x4, 4x8 e 6x8. O uso dos modelos probabilísticos Exponencial, Weibull e Log-normal possibilitou a estimação das curvas de sobrevivência nos grupos 2, 4, 6 e 8, exceto no grupo 4, em que o modelo Weibull não foi adequado. Comparando os modelos probabilísticos com a técnica não-paramétrica, as curvas dos modelos probabilísticos dos grupos 2 e 4 apresentaram ajustes satisfatórios com relação à curva estimada por Kaplan-Meier.
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IJzerman, E. P. F. "Progress in diagnostics and prevention of Legionnaires' disease." [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2009. http://irs.ub.rug.nl/ppn/315954442.

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Baird, Simon Herbert. "Progress in diagnosis and treatment of acute coronary syndromes." Thesis, Queen's University Belfast, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326416.

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Harjunpää, Vesa. "Enzymes hydrolysing wood polysaccharides : a progress curve study of oligosaccharide hydrolysis by two cellobiohydrolases and three ß-mannanases /." Espoo : Technical Research Centre of Finland, 1998. http://www.vtt.fi/inf/pdf/publications/1998/P372.pdf.

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Ahnert, Peter, Petra Creutz, Markus Scholz, Hartwig Schütte, Christoph Engel, Hamid Hossain, Trinad Chakraborty, et al. "PROGRESS – prospective observational study on hospitalized community acquired pneumonia." Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-209571.

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Background: Community acquired pneumonia (CAP) is a high incidence disease resulting in about 260,000 hospital admissions per year in Germany, more than myocardial infarction or stroke. Worldwide, CAP is the most frequent infectious disease with high lethality ranging from 1.2 % in those 20–29 years old to over 10 % in patients older than 70 years, even in industrial nations. CAP poses numerous medical challenges, which the PROGRESS (Pneumonia Research Network on Genetic Resistance and Susceptibility for the Evolution of Severe Sepsis) network aims to tackle: Operationalization of disease severity throughout the course of disease, outcome prediction for hospitalized patients and prediction of transitions from uncomplicated CAP to severe CAP, and finally, to CAP with sepsis and organ failure as a life-threatening condition. It is a major aim of PROGRESS to understand and predict patient heterogeneity regarding outcome in the hospital and to develop novel treatment concepts. Methods: PROGRESS was designed as a clinical, observational, multi-center study of patients with CAP requiring hospitalization. More than 1600 patients selected for low burden of co-morbidities have been enrolled, aiming at a total of 3000. Course of disease, along with therapy, was closely monitored by daily assessments and long-term follow-up. Daily blood samples allow in depth molecular-genetic characterization of patients. We established a well-organized workflow for sample logistics and a comprehensive data management system to collect and manage data from more than 50 study centers in Germany and Austria. Samples are stored in a central biobank and clinical data are stored in a central data base which also integrates all data from molecular assessments. Discussion: With the PROGRESS study, we established a comprehensive data base of high quality clinical and molecular data allowing investigation of pressing research questions regarding CAP. In-depth molecular characterization will contribute to the discovery of disease mechanisms and establishment of diagnostic and predictive biomarkers. A strength of PROGRESS is the focus on younger patients with low burden of co-morbidities, allowing a more direct look at host biology with less confounding. As a resulting limitation, insights from PROGRESS will require validation in representative patient cohorts to assess clinical utility. Trial registration: The PROGRESS study was retrospectively registered on May 24th, 2016 with ClinicalTrials.gov: NCT02782013
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Books on the topic "Progress of the disease curve"

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Shulman, Leslie Sara. Progress curve analysis of enzyme catalyzed reactions. Ottawa: National Library of Canada, 1996.

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O'Shea, Lester. A cure worse than the disease: Fighting discrimination through government control. Tampa, Fla: Hallberg Publ., 1999.

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R, Vining Aidan, and University of Toronto. Centre for Public Management, eds. Cure or disease?: Private health insurance in Canada. Toronto, Ont: Centre for Public Management, Faculty of Management, University of Toronto, 1996.

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Globerman, Steven. Cure or disease?: Private health insurance in Canada. Toronto: Centre for Public Management, Faculty of Management, University of Toronto, 1996.

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UNDP/World Bank/WHO Special Programme for Research and Training in Tropical Diseases. Tropical disease research: Progress 1997-98. Geneva: World Health Organization, 1998.

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Diseases, UNDP/World Bank/WHO Special Programme for Research and Training in Tropical. Tropical disease research: Progress 1999-2000. Geneva: World Health Organization, 2000.

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Organization, World Health, World Bank, and United Nations Development Programme, eds. Tropical disease research: Progress 1995-96. Geneva: World Health Organization, 1997.

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Rodgers, Anne Brown. 2001-2002 Alzheimer's disease progress report. Silver Spring, MD: Alzheimer's Disease Education and Referral Center, National Institute on Aging, 2003.

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UNDP/World Bank/WHO Special Programme for Research and Training in Tropical Diseases. Tropical disease research: Progress 2001-2002. Geneva: World Health Organization, 2003.

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Diseases, UNDP/World Bank/WHO Special Programme for Research and Training in Tropical. Tropical disease research: Progress 1999-2000. Geneva: World Health Organization, 2001.

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Book chapters on the topic "Progress of the disease curve"

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Madden, L. V., and C. L. Campbell. "Nonlinear Disease Progress Curves." In Epidemics of Plant Diseases, 181–229. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75398-5_6.

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Kranz, Jürgen. "Comparison of Temporal Aspects of Epidemics: The Disease Progress Curves." In Comparative Epidemiology of Plant Diseases, 93–134. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05261-7_5.

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Böttcher, Albrecht, and Yuri I. Karlovich. "Interaction between curve and weight." In Progress in Mathematics, 71–115. Basel: Birkhäuser Basel, 1997. http://dx.doi.org/10.1007/978-3-0348-8922-3_3.

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Caldow, Marissa K., and David Cameron-Smith. "J-Shaped Curve." In Encyclopedia of Exercise Medicine in Health and Disease, 498. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2574.

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Johnson, C. D. "Progress and Prospects." In Pancreatic Disease, 369–71. London: Springer London, 1991. http://dx.doi.org/10.1007/978-1-4471-3356-8_31.

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Kang, Yuan-Yuan, and Ji-Guang Wang. "The J-Curve Phenomenon in Hypertension." In Hypertension and Cardiovascular Disease, 249–60. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39599-9_16.

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Sutton, Robert, Deborah D. Stocken, Janet A. Dunn, Helen Hickey, Michael G. T. Raraty, Paula Ghaneh, John Buckels, et al. "Progress by Collaboration: ESPAC Studies." In Pancreatic Disease, 57–69. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-85233-904-3_5.

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Thangaraj, Parimelazhagan. "Inflammatory Bowel Disease." In Progress in Drug Research, 117–22. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-26811-8_18.

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Karniš, J., and V. Pechoč. "Master flow curve for fibre-forming polypropylenes." In Progress and Trends in Rheology II, 358–60. Heidelberg: Steinkopff, 1988. http://dx.doi.org/10.1007/978-3-642-49337-9_124.

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Segawa, Masaya. "Progress in Segawa’s Disease." In Mapping the Progress of Alzheimer’s and Parkinson’s Disease, 353–59. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-0-306-47593-1_61.

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Conference papers on the topic "Progress of the disease curve"

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Chang, Albert Ling Sheng, Chong Khim Phin, and Ho Chong Mun. "Comparing nonlinear models in describing disease progress curve of cocoa black pod." In PROCEEDING OF THE 25TH NATIONAL SYMPOSIUM ON MATHEMATICAL SCIENCES (SKSM25): Mathematical Sciences as the Core of Intellectual Excellence. Author(s), 2018. http://dx.doi.org/10.1063/1.5041682.

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Ivanov, Zhanna A., Robert C. Scott, Jenna Rosano, Barbara Krynska, and Mohammad F. Kiani. "Engineering Cardiac Tissue Using Stem Cell Therapy to Mend the Broken Heart." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-203624.

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Myocardial infarction (MI) is one of the most severe forms of coronary artery disease and is the leading cause of death in the United States [1]. Current treatments for an MI are either highly invasive, such as coronary artery bypass grafting and stent angioplasty, or might have undesirable long-term effects as is the case with pharmacological interventions. However, newly emerging methodologies, such as a less invasive stem cell therapy, aim to cure the disease rather than just alleviate its symptoms. This new tissue engineering technology has shown promise in restoring the homeostasis of the heart muscle after MI in preclinical and clinical studies [2]. However, controversies regarding inconsistent methodologies and a lack of mechanistic understanding of its actions have hampered progress in this field [3].
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Baniya, A., S. Thapa, E. Borquist, D. Bailey, D. Wood, G. Dutta, P. Arumugam, J. Glawe, C. Kevil, and L. Weiss. "Lab-on-a-Chip Device for Hydrogen Sulfide Sensing in Biomedical and Environmental Applications Using Electrochemical Approach." In ASME 2015 International Technical Conference and Exhibition on Packaging and Integration of Electronic and Photonic Microsystems collocated with the ASME 2015 13th International Conference on Nanochannels, Microchannels, and Minichannels. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/ipack2015-48491.

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Hydrogen sulfide (H2S) is rapidly emerging as a biologically significant signaling molecule. In recent studies, sulfide level in blood or plasma has been reported to be in the concentration between 10 and 300 μM suggesting it acts in various diseases. This work reports progress on a new Lab-on-a-Chip (LOC) device for these applications. The uniquely designed, hand-held device uses advanced liberation chemistry that releases H2S from liquid sample and an electrochemical approach to detect sulfide concentration from the aqueous solution. The device itself consists of three distinct layers of Polydimethylsiloxane (PDMS) structures and a three electrode system for direct and rapid H2S concentration measurement. In this work specifically, the oxidation of sulfide at the gold (Au) and platinum (Pt.) electrodes has been examined. This is the first known application of electrochemical H2S sensing in an LOC application. The analytical utility and performance of the device has been assessed through direct detection using chronoamperometry (CA) scan and cyclic voltammetry (CV). An electrocatalytic sulfide oxidation signal has been recorded for sulfide concentration range vs, Ag/AgCl at different pH buffers at the trapping chamber. The calibration curve in the range 1 μM to 1 M was obtained using this electrode setup. The detection limit was found to be 0.1 μM. This device shows promise for providing fast and inexpensive determination of H2S concentration in aqueous samples.
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Jastrzebski, Radoslaw, and Krzysztof Chwastek. "Analytical expressions for magnetization curve." In 2017 Progress in Applied Electrical Engineering (PAEE). IEEE, 2017. http://dx.doi.org/10.1109/paee.2017.8009019.

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Liu, Xiaohua, Han Chen, Shi Chen, Shengqiang Shen, and Xinyu Zhao. "Research progress of droplet impact on dry curve surfaces." In INTERNATIONAL CONFERENCE ON THERMAL SCIENCE AND TECHNOLOGY (ICTST) 2017. Author(s), 2018. http://dx.doi.org/10.1063/1.5046620.

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Hawkins, Richard H., Timothy J. Ward, Donald E. Woodward, and Joseph A. VanMullem. "Progress Report: ASCE Task Committee on Curve Number Hydrology." In Watershed Management Conference 2005. Reston, VA: American Society of Civil Engineers, 2005. http://dx.doi.org/10.1061/40763(178)150.

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Boyle, Justin, Mohan Karunanithi, Tim Wark, Wilbur Chan, and Christine Colavitti. "Quantifying Functional Mobility Progress for Chronic Disease Management." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.260426.

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Boyle, Justin, Mohan Karunanithi, Tim Wark, Wilbur Chan, and Christine Colavitti. "Quantifying Functional Mobility Progress for Chronic Disease Management." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.4398804.

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Yang, Yan-ling. "Statistical Study on Disease-Related ncRNAs Using Z-curve Method." In 2009 International Conference on Computational Intelligence and Natural Computing (CINC). IEEE, 2009. http://dx.doi.org/10.1109/cinc.2009.80.

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Cyrus, Kaveh M., and Ali Maaleki. "Presenting Countermeasures against Deviations in Projects Progress, Using Improved S-Curve." In The 2nd World Congress on Mechanical, Chemical, and Material Engineering. Avestia Publishing, 2016. http://dx.doi.org/10.11159/icmie16.127.

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Reports on the topic "Progress of the disease curve"

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Hutchison, Michael. A Cure Worse Than the Disease? Currency Crises and the Output Costs of IMF-Supported Stabilization Programs. Cambridge, MA: National Bureau of Economic Research, May 2001. http://dx.doi.org/10.3386/w8305.

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Michelmore, R. Transposon tagging of disease resistance genes. Progress report, May 1, 1988--1992. Office of Scientific and Technical Information (OSTI), June 1994. http://dx.doi.org/10.2172/10179207.

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Imre, K. Growth curve analysis for plasma profiles using smoothing splines. Annual progress report, June 1992--June 1993. Office of Scientific and Technical Information (OSTI), May 1993. http://dx.doi.org/10.2172/10159533.

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Amano, K. I., A. O. Anderson, C. L. Bailey, M. Balady, and R. F. Berendt. U.S. Army Medical Research Institute of Infectious Disease Annual Progress Report, Fiscal Year 1985. Fort Belvoir, VA: Defense Technical Information Center, October 1985. http://dx.doi.org/10.21236/ada230449.

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Grossman, L. The role of aging and DNA repair in chronic disease. Final progress report, December 1, 1985--September 29, 1993. Office of Scientific and Technical Information (OSTI), November 1993. http://dx.doi.org/10.2172/10186975.

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Absolon, Randall F. Changes in Gas Bubble Disease Signs for Migrating Juvenile Salmonids Experimentally Exposed to Supersaturated Gasses, 1996-1997 Progress Report. Office of Scientific and Technical Information (OSTI), March 1999. http://dx.doi.org/10.2172/751544.

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Absolon, Randall F. Changes in Gas Bubble Disease Signs and Survival of Migrating Juvenile Salmonids Experimentally Exposed to Supersaturated Gasses, 1995-1996 Progress Report. Office of Scientific and Technical Information (OSTI), November 1997. http://dx.doi.org/10.2172/14086.

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