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Journal articles on the topic 'Progressive hearing loss'

Author: Grafiati

Published: 25 May 2024

Last updated: 31 July 2025

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1

Hayes, Deborah, and Susan Dreith. "Catastrophic Progressive Hearing Loss in Childhood." Journal of the American Academy of Audiology 11, no. 06 (2000): 300–308. http://dx.doi.org/10.1055/s-0042-1748058.

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AbstractWe present a report on a 5-year-old child with a complex medical and audiologic history who exhibited catastrophic progression in hearing loss. Hearing loss was initially attributed to bacterial meningitis at age 3 months; progression was apparently related to perilymph fistula at age 8 years. Etiologies associated with progressive hearing loss in children as well as signs of progression and monitoring protocols for children at risk for progressive hearing loss are discussed. Abbreviations: ABR = auditory brainstem response, CMV = cytomegalovirus, JCIH = Joint Committee on Infant Heari

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2

Fitzpatrick, Elizabeth M., Flora Nassrallah, Bénédicte Vos, JoAnne Whittingham, and Jessica Fitzpatrick. "Progressive Hearing Loss in Children With Mild Bilateral Hearing Loss." Language, Speech, and Hearing Services in Schools 51, no. 1 (2020): 5–16. http://dx.doi.org/10.1044/2019_lshss-ochl-19-0013.

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Purpose This study investigated progressive hearing loss in a cohort of children who were identified with permanent mild bilateral hearing loss. Method This population-based study included 207 children with permanent mild bilateral hearing loss, diagnosed and followed from 2003 to 2016 in 1 region of Canada. Clinical characteristics and initial audiologic results were collected prospectively at diagnosis, and audiologic information was updated. Changes in hearing levels between the 1st and most recent assessment were analyzed to determine progressive hearing loss. Clinical characteristics were

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3

Vrabec, Jeffrey T. "Hydrocodone Use and Sensorineural Hearing Loss." Pain Physician 3;10, no. 5;3 (2007): 467–72. http://dx.doi.org/10.36076/ppj.2007/10/467.

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Background: The hydrocodone/acetaminophen combination is one of the most commonly used analgesic preparations. Isolated incidences of suspected association between hydrocodone abuse and rapidly progressive hearing loss have been reported. In this study, we describe the clinical characteristics of 5 patients presenting with progressive hearing loss and a history of hydrocodone use. Methods: Patients presenting with rapidly progressive bilateral hearing loss who had a documented history of hydrocodone use were selected for the study. The presentation, audiologic findings, associated comorbiditie

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4

Roberts, Deanne M., Matthew L. Bush, and Raleigh O. Jones. "Adult Progressive Sensorineural Hearing Loss." Otology & Neurotology 35, no. 2 (2014): 241–45. http://dx.doi.org/10.1097/mao.0b013e3182a437b3.

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5

Whelan, Alison, and Anne Hing. "Genetics of Progressive Hearing Loss." Seminars in Hearing 16, no. 03 (1995): 246–56. http://dx.doi.org/10.1055/s-0028-1083722.

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6

Levi, Haya, Lilly Tell, and Moshe Feinmesser. "Progressive Hearing Loss in Hard-of-Hearing Children." International Journal of Audiology 32, no. 2 (1993): 132–36. http://dx.doi.org/10.3109/00206099309071862.

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7

de Wolf, M. J. F., J. Honings, F. B. M. Joosten, L. Hoefsloot, E. A. M. Mylanus, and C. W. R. J. Cremers. "Two siblings with progressive, fluctuating hearing loss after head trauma, treated with cochlear implantation." Journal of Laryngology & Otology 124, no. 1 (2009): 86–89. http://dx.doi.org/10.1017/s0022215109990296.

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AbstractObjective:Description of two siblings with unexplained, progressive, perceptive hearing loss after head trauma.Design:Case report.Subjects:Two siblings aged six and eight years old with bilateral, intermittent but progressive hearing loss.Results:These patients had a c.1172G>A (p.Ser391Asn) mutation in the SLC26A4 gene, which has not previously been reported and which caused Pendred or enlarged vestibular aqueduct syndrome. The diagnosis was based on the perceptive hearing loss, computed tomography findings and mutation analysis. The patients were each fitted with a cochlear implant

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8

Reisser, Christoph F. V., William J. Kimberling, and Christian R. Otterstedde. "Hearing Loss in Usher Syndrome Type II is Nonprogressive." Annals of Otology, Rhinology & Laryngology 111, no. 12 (2002): 1108–11. http://dx.doi.org/10.1177/000348940211101208.

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Usher syndrome is an autosomal recessive disorder characterized by sensorineural hearing loss and progressive visual loss secondary to retinitis pigmentosa. In the literature, a possible progression of the moderate to severe hearing loss in Usher syndrome type II (Usher II) is controversial. We studied the development of the hearing loss of 125 patients with a clinical diagnosis of Usher syndrome type II intraindividually and interindividually by repeatedly performing complete audiological and neuro-otologic examinations. Our data show a very characteristic slope of the hearing curve in all Us

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9

Lahiri, Asish Kumar, Anandita Gupta, Rakesh Vohra, Shalabh Sharma, and Satinder Singh. "Cochlear Otosclerosis: An Undiagnosed Cause of Progressive Sensorineural Hearing Loss." Bengal Journal of Otolaryngology and Head Neck Surgery 28, no. 2 (2020): 127–37. http://dx.doi.org/10.47210/bjohns.2020.v28i2.329.

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Introduction Otosclerosis presents as conductive or mixed hearing loss depending upon the stage of the disease. Isolated sensorineural hearing loss though known to occur has been rarely reported. Pure cochlear otosclerosis presenting as sensorineural hearing loss should be considered as a differential diagnosis in young patients presenting with progressive hearing loss. The aim of this article is to note the prevalence of cochlear otosclerosis in young patients presenting with progressive sensorineural hearing loss.  Materials and Methods Retrospective chart analysis of 19 pa

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10

Hashimoto, Sho, and Harold F. Schuknecht. "Progressive Hearing Loss from Strial Dysplasia." Annals of Otology, Rhinology & Laryngology 96, no. 2 (1987): 229–31. http://dx.doi.org/10.1177/000348948709600219.

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An otherwise healthy male patient had a bilateral, slowly progressive hearing loss first noticed in early childhood and possibly present at birth. Audiometric studies at the age of 32 showed a moderately severe, bilateral, mixed type hearing loss. He died of unrelated causes at the age of 34. Studies of the temporal bones showed bilateral hypoplasia and atrophy of the striae vascularis as the cause of hearing loss. The history and findings are consistent with a genetically determined hypoplasia and atrophy of this structure.

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11

Marcos Alonso, Susana, Cristina Nicole Almeida Ayerve, Sofía Pacheco López, et al. "Hearing loss in vestibular schwannoma." Revista ORL 13, no. 1 (2022): e27266. http://dx.doi.org/10.14201/orl.27266.

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Purpose: The most common reason for consultation in patients suffering from Vestibular Schwannoma (VS) is progressive hearing loss. The main objective of this study is analyzing the hearing loss in patients with VS and determining the extent to which the tumor grade and the hearing loss are related. Methods: An observational retrospective study was conducted with a sample of 291 patients diagnosed with VS between 1995 and 2017 in the Department of Otorhinolaryngology and Head and Neck Surgery of the Tertiary Care Primary Center of Salamanca. We analyzed preoperative and postoperative dat

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12

Lee, Jennifer W., and Manohar L. Bance. "Hearing loss." Practical Neurology 19, no. 1 (2018): 28–35. http://dx.doi.org/10.1136/practneurol-2018-001926.

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Hearing loss affects one in six people in the UK and is a significant disease burden. In addition to communication problems, there is also an association with depression and dementia. Clinical assessment with targeted history and examination can identify the characteristics and cause of hearing loss, and complementary audiological testing can confirm its type and severity. Retrocochlear screening is recommended for sudden, rapidly progressive or asymmetric sensorineural hearing loss. Medical or surgical therapies may be indicated in cases of conductive hearing loss, while hearing assistive dev

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13

Karjalainen, Seppo, Leena Pakarinen, Helena Kääriäinen, Markku Teräsvirta, and Eero Vartiainen. "Progressive Hearing Loss in Usher's Syndrome." Annals of Otology, Rhinology & Laryngology 98, no. 11 (1989): 863–66. http://dx.doi.org/10.1177/000348948909801106.

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In 18 patients with Usher's syndrome, progressive hearing loss was verified audiologically in eight cases. Despite poor auditory threshold values and low speech discrimination scores, there was only one patient who could not communicate with speech. The possibility of hearing impairment being mainly progressive in Usher's syndrome is discussed.

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14

Newton, V. E., and V. J. Rowson. "Progressive sensorineural hearing loss in childhood." British Journal of Audiology 22, no. 4 (1988): 287–95. http://dx.doi.org/10.3109/03005368809076466.

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15

Gray, R. F. "Cochlear implantation for progressive hearing loss." Archives of Disease in Childhood 88, no. 8 (2003): 708–11. http://dx.doi.org/10.1136/adc.88.8.708.

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16

Barreira-Nielsen, Carmen, Elizabeth Fitzpatrick, Serena Hashem, JoAnne Whittingham, Nicholas Barrowman, and Mary Aglipay. "Progressive Hearing Loss in Early Childhood." Ear and Hearing 37, no. 5 (2016): e311-e321. http://dx.doi.org/10.1097/aud.0000000000000325.

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17

Neely, J. "Overview of Progressive Sensorineural Hearing Loss." Seminars in Hearing 16, no. 03 (1995): 209–13. http://dx.doi.org/10.1055/s-0028-1083717.

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18

Berrettini, Stefano, Francesca Ravecca, Stefano Sellari-Franceschini, Fabio Matteucci, Gabriele Siciliano, and Francesco Ursino. "Progressive sensorineural hearing loss in childhood." Pediatric Neurology 20, no. 2 (1999): 130–36. http://dx.doi.org/10.1016/s0887-8994(98)00123-4.

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19

Meyerhoff, William L., Steven Cass, Mitchell K. Schwaber, Nancy Sculerati, and William H. Slattery. "Progressive Sensorineural Hearing Loss in Children." Otolaryngology–Head and Neck Surgery 110, no. 6 (1994): 569–79. http://dx.doi.org/10.1177/019459989411000617.

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20

Nishimura, Tadashi, and Hiroshi Hosoi. "Progressive hearing loss in intracochlear schwannoma." European Archives of Oto-Rhino-Laryngology 265, no. 4 (2007): 489–92. http://dx.doi.org/10.1007/s00405-007-0483-x.

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21

Jenkinson, P. W., M. I. Syed, and L. Mcclymont. "Progressive, reversible sensorineural hearing loss caused by azathioprine." Journal of Laryngology & Otology 128, no. 9 (2014): 838–40. http://dx.doi.org/10.1017/s0022215114001807.

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AbstractObjective:This paper describes the first reported case of progressive sensorineural hearing loss caused by azathioprine, which was reversed on stoppage of the drug.Case report:A female patient with previously normal hearing presented with progressive sensorineural hearing loss after being started on azathioprine. Otological and neurological examination findings were unremarkable. After stopping the drug, the patient reported an improvement in hearing, which was confirmed on pure tone audiometry.Conclusion:This previously unreported side effect of azathioprine is highlighted in order to

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22

Battelino, S., B. Repič Lampret, M. Žargi, and K. Trebušak Podkrajšek. "Novel connexin 30 and connexin 26 mutational spectrum in patients with progressive sensorineural hearing loss." Journal of Laryngology & Otology 126, no. 8 (2012): 763–69. http://dx.doi.org/10.1017/s0022215112001119.

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AbstractObjective:Mutations in the gap junction protein beta-2 gene (‘GJB2’) are known to be responsible for mild to profound congenital and late-onset hearing loss. This study aimed to investigate the molecular basis of progressive hearing loss compared with non-progressive hearing loss.Methods:Following clinical otorhinolaryngological evaluation, a genetic analysis was performed in a cohort of 72 patients with progressive sensorineural hearing loss.Results:Pathological genotypes were established in 16 patients (22.2 per cent). Six different gap junction protein beta-2 gene mutations were det

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23

Potts, Lisa, and Michael Valente. "Programmable Hearing Aids: Applications for Progressive and Fluctuating Hearing Loss." Seminars in Hearing 16, no. 03 (1995): 266–79. http://dx.doi.org/10.1055/s-0028-1083724.

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24

Thalmann, Isolde, and J. Neely. "Progressive Hearing Loss and the Perilymphatic Fistula." Seminars in Hearing 16, no. 03 (1995): 214–18. http://dx.doi.org/10.1055/s-0028-1083718.

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25

Savastano, Marina, Marilena Savini, and Claudio Andreoli. "Idiopathic progressive sensorineural hearing loss in children." International Journal of Pediatric Otorhinolaryngology 26, no. 3 (1993): 225–33. http://dx.doi.org/10.1016/0165-5876(93)90092-h.

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26

Silkes, Ellen D., and Janie Chabot. "Progressive hearing loss following Haemophilus influenzae meningitis." International Journal of Pediatric Otorhinolaryngology 9, no. 3 (1985): 249–56. http://dx.doi.org/10.1016/s0165-5876(85)80041-0.

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27

Terayama, Yoshihiko, Yuhko Ishibe, and Jun-Ichi Matsushima. "Rapidly Progressive Sensorineural Hearing Loss (Rapid Deafness)." Acta Oto-Laryngologica 105, sup456 (1988): 43–48. http://dx.doi.org/10.3109/00016488809125076.

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28

Steel, K. P. "HUMAN GENETICS: Progress in Progressive Hearing Loss." Science 279, no. 5358 (1998): 1870–71. http://dx.doi.org/10.1126/science.279.5358.1870.

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29

Branch, Matthew P., and Barry E. Hirsch. "Management of Unilateral Acoustic Neuroma in a 72-Year-Old Patient with Contralateral Congenital Deafness." Otolaryngology–Head and Neck Surgery 127, no. 5 (2002): 483–86. http://dx.doi.org/10.1067/mhn.2002.129809.

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Patients with asymmetric or progressive hearing impairment warrant evaluation for the cause of their loss. We present a 72–year–old woman with near life–long hearing loss in one ear and a history of contralateral slowly progressive hearing loss. The audiometric and radiologic findings are demonstrated along with discussion of the management of acoustic neuroma in an only hearing ear.

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30

Hiraki, N., T. Udaka, H. Yamamoto, Y. Kadokawa, J. Ohkubo, and H. Suzuki. "Mitochondrial neurogastrointestinal encephalomyopathy associated with progressive hearing loss." Journal of Laryngology & Otology 124, no. 9 (2010): 1007–9. http://dx.doi.org/10.1017/s0022215110001477.

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AbstractObjective:We report a rare case of mitochondrial neurogastrointestinal encephalomyopathy with hearing loss.Case report:A 46-year-old woman presented with a three-year history of progressive, bilateral hearing loss and tinnitus. She had been suffering from unexplained abdominal pain and diarrhoea for 20 years. When first seen, her otoscopic findings were normal, and pure tone audiometry showed mild and moderate hearing loss in her right and left ears, respectively. She also had: bilateral ophthalmoparesis, neck and limb muscle weakness, and hypoactive deep tendon reflexes on neurologica

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31

Oike, Hideaki, Kyoko Yamasaki, Kenji Hashiguchi, and Honoka Uehira. "Evaluation of intake of aged garlic extract and organosulfur compounds on progressive hearing loss in DBA/2J mice." PLOS ONE 20, no. 4 (2025): e0322105. https://doi.org/10.1371/journal.pone.0322105.

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Garlic is rich in organosulfur compounds with high antioxidant capacity and is known to have various health benefits. Aged garlic is a particularly effective source of these active compounds because it contains fewer toxic components. This study evaluated the effects of S-allyl cysteine (SAC), a major functional organosulfur compound in garlic, and aged garlic extract (AGE) on the suppression of progressive hearing loss. SAC and AGE were dissolved in drinking water at 1% (w/w) and administered to DBA/2J mice, a model of early progressive hearing loss, for 12 weeks, starting at 4 weeks of age.

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32

Mori, Kentaro, Ikuyo Miyanohara, Hideaki Moteki, Shin-ya Nishio, Yuichi Kurono, and Shin-ichi Usami. "Novel Mutations in GRXCR1 at DFNB25 Lead to Progressive Hearing Loss and Dizziness." Annals of Otology, Rhinology & Laryngology 124, no. 1_suppl (2015): 129S—134S. http://dx.doi.org/10.1177/0003489415575061.

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Objective: We identified 2 patients in 1 family who had novel mutations in GRXCR1, which caused progressive hearing loss. Methods: One thousand one hundred twenty Japanese hearing loss patients with sensorineural hearing loss from unrelated families were enrolled in this study. Targeted genomic enrichment with massively parallel sequencing of all known nonsyndromic hearing loss genes was used to identify the genetic causes of hearing loss. Results: In this study, 2 affected individuals with compound heterozygous mutations—c.439C>T (p.R147C) and c.784C>T (p.R262X)—in GRXCR1 were identifie

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33

Muino, Juan, Roger Carreras, Muriel Ocampo, et al. "La importancia de los auto- anticuerpos IGG específicos tipo II en la enfermedad de MENIERE y pérdida progresiva de la audición." Revista de la Facultad de Ciencias Médicas de Córdoba 56, no. 2 (2023): 71–91. http://dx.doi.org/10.31053/1853.0605.v56.n2.40240.

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The Meniere's Disease and Progressive Hearing Loss were considered idiopathic. Both entities were produced by endo lymphatic hydrops and disruption of the membrane which contain type II collagen. The inner ear presented widely expression of type II collagen. These pathologies were probably autoimmune diseases.The aim of this work was to study the relationship of specific IgO to type II collagen in Meniere's disease, Progressive hearing loss, and compared with Sudden hearing loss patients, vascular vertigo patients and normal controls. Patients were divided by clinical findings in: 1° Meniere's

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34

Fuse, Takeo, and Margareta B. Møller. "Delayed and Progressive Hearing Loss after Microvascular Decompression of Cranial Nerves." Annals of Otology, Rhinology & Laryngology 105, no. 2 (1996): 158–61. http://dx.doi.org/10.1177/000348949610500212.

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An unusual case of unilateral delayed and progressive hearing loss following a microvascular decompression operation on cranial nerves V, VII, and VIII on the left side is reported. Preoperative and postoperative audiologic evaluation revealed a mild high-frequency hearing loss for both ears, normal thresholds for the acoustic middle ear reflex response, and normal brain stem auditory evoked potentials. Three years after this microvascular decompression procedure, the patient noticed slowly decreasing hearing in her left ear, and subsequent serial audiograms revealed a progressive sensorineura

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35

Nakashima, Takahiro, Akira Ganaha, Shougo Tsumagari, et al. "Is the Conductive Hearing Loss in NOG-Related Symphalangism Spectrum Disorder Congenital?" ORL 83, no. 3 (2021): 196–202. http://dx.doi.org/10.1159/000512668.

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We describe a dominant Japanese patient with progressive conductive hearing loss who was diagnosed with <i>NOG</i>-related symphalangism spectrum disorder (<i>NOG</i>-SSD), a spectrum of congenital stapes fixation syndromes caused by <i>NOG</i> mutations. Based on the clinical features, including proximal symphalangism, conductive hearing loss, hyperopia, and short, broad middle, and distal phalanges of the thumbs, his family was diagnosed with stapes ankylosis with broad thumbs and toes syndrome (SABTT). Genetic analysis revealed a heterozygous substitutio

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36

NIELD, TOM, ANGELA D. RAMOS, and DAVID WARBURTON. "Late-Onset Hearing Loss." Pediatrics 83, no. 5 (1989): 807–8. http://dx.doi.org/10.1542/peds.83.5.807a.

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The findings of Hendricks-Munoz and Walton concerning the incidence of sensorineural hearing loss in infants with persistent pulmonary hypertension (persistent fetal circulation) substantiate our findings that, in some cases, the loss appears to be of delayed onset and progressive. We would, however, like to point out that delayed-onset sensorineural loss is not limited to infants with persistent pulmonary hypertension, because four of the 11 infants we reported did not have persistent pulmonary hypertension. Therefore, serial audiologic evaluations should also be done for any infant with sign

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37

Kang, Minjin, Jung Ah Kim, Mee Hyun Song, et al. "Novel Variant in CEP250 Causes Protein Mislocalization and Leads to Nonsyndromic Autosomal Recessive Type of Progressive Hearing Loss." Cells 12, no. 18 (2023): 2328. http://dx.doi.org/10.3390/cells12182328.

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Genetic hearing loss is the most common hereditary sensorial disorder. Though more than 120 genes associated with deafness have been identified, unveiled causative genes and variants of diverse types of hearing loss remain. Herein, we identified a novel nonsense homozygous variant in CEP250 (c.3511C>T; p.Gln1171Ter) among the family members with progressive moderate sensorineural hearing loss in nonsyndromic autosomal recessive type but without retinal degeneration. CEP250 encodes C-Nap1 protein belonging to the CEP protein family, comprising 30 proteins that play roles in centrosome aggreg

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38

Levy, Charles E., Albert T. Lash, Hal B. Janoff, and Frederick S. Kaplan. "Conductive Hearing Loss in Individuals with Fibrodysplasia Ossificans Progressiva." American Journal of Audiology 8, no. 1 (1999): 29–33. http://dx.doi.org/10.1044/1059-0889(1999/011).

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Fibrodysplasia ossificans progressiva (FOP) is a very rare genetic disorder that is characterized by progressive heterotopic ossification of soft tissues and congenital malformation of the great toes. Although previous case studies have reported hearing loss in individuals with FOP, there have been no large-scale studies regarding the nature or cause of the hearing loss. Here, we report the findings of a two-part study. In Part I, we report the findings of a postal survey regarding hearing loss that was sent to 102 individuals with FOP. In Part II, we report the findings of on-site hearing eva

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39

Dahle, Arthur J., Karen B. Fowler, John D. Wright, Suresh B. Boppana, William J. Britt, and Robert F. Pass. "Longitudinal Investigation of Hearing Disorders in Children with Congenital Cytomegalovirus." Journal of the American Academy of Audiology 11, no. 05 (2000): 283–90. http://dx.doi.org/10.1055/s-0042-1748054.

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AbstractThis investigation consisted of a longitudinal study of the effects of congenital cytomegalovirus (CMV) infection on hearing sensitivity in 860 children with documented asymptomatic or symptomatic congenital CMV infection. Of the 651 children with asymptomatic CMV infection, 48 (7.4%) developed sensorineural hearing loss (SNHL), compared to 85 (40.7%) of the children with symptomatic CMV infection. Children in both groups experienced latent effects consisting of delayed onset of loss, threshold fluctuations, and/or progressive loss of hearing. It can be concluded that congenital CMV in

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40

Hiramatsu, Ken, Shin-ya Nishio, Shin-ichiro Kitajiri, Tomohiro Kitano, Hideaki Moteki, and Shin-ichi Usami. "Prevalence and Clinical Characteristics of Hearing Loss Caused by MYH14 Variants." Genes 12, no. 10 (2021): 1623. http://dx.doi.org/10.3390/genes12101623.

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Variants in MYH14 are reported to cause autosomal dominant nonsyndromic hereditary hearing loss (ADNSHL), with 34 variants reported to cause hearing loss in various ethnic groups. However, the available information on prevalence, as well as with regard to clinical features, remains fragmentary. In this study, genetic screening for MYH14 variants was carried out using a large series of Japanese hearing-loss patients to reveal more detailed information. Massively parallel DNA sequencing of 68 target candidate genes was applied in 8074 unrelated Japanese hearing-loss patients (including 1336 with

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41

van Aarem, Annelies, Alfred J. L. G. Pinckers, William J. Kimberling, Patrick L. M. Huygen, Elisabeth M. Bleeker-Wagemakers, and W. R. J. Cremers. "Stable and Progressive Hearing Loss in Type 2A Usher's Syndrome." Annals of Otology, Rhinology & Laryngology 105, no. 12 (1996): 962–67. http://dx.doi.org/10.1177/000348949610501206.

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Audiograms were traced or additionally performed on 23 Usher's syndrome patients in 10 Dutch multi-affected families, all linked to chromosome 1q (USH2A locus). Serial audiograms, available in 13 patients, were used for a regression analysis of binaural pure tone average on age (follow-up, 9 to 32 years) to test for “significant progression,” ie, a significant regression coefficient, here called the “annual threshold increase” (ATI, expressed in decibels per year). A significant ATI (>1 dB/y) was observed in 3 patients. Analysis of variance of ATI demonstrated significant heterogeneity; hea

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42

Gilroy, Patricia A., and Carl L. Reams. "Delayed Progressive Sensorineural Hearing Loss after Electrocution Injury." Otolaryngology–Head and Neck Surgery 129, no. 2 (2003): P262. http://dx.doi.org/10.1016/s0194-59980301191-4.

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43

Ichijo, Hiroaki, Shin-Ichi Saito, Shigetoshi Fujita, and Hideichi Shinkawa. "Vestibular Function in Bilateral Progressive Sensorineural Hearing Loss." Acta Oto-Laryngologica 115, sup519 (1995): 193–96. http://dx.doi.org/10.3109/00016489509121902.

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44

HICKSON, L. M. H., and D. ALCOCK. "Progressive hearing loss in children with congenital cytomegalovirus." Journal of Paediatrics and Child Health 27, no. 2 (1991): 105–7. http://dx.doi.org/10.1111/j.1440-1754.1991.tb00362.x.

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45

Kenna, Margaret A., and Nancy Sculerati. "Management of Progressive Sensorineural Hearing Loss in Children." Otolaryngology–Head and Neck Surgery 112, no. 5 (1995): P117. http://dx.doi.org/10.1016/s0194-5998(05)80294-3.

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Educational objectives: To cite the differential diagnosis of progressive sensorineural hearing loss in children and to provide a reasonable algorithm of both medical and surgical treatment modalities for this condition.

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Gilroy, P. "Delayed progressive sensorineural hearing loss after electrocution injury." Otolaryngology - Head and Neck Surgery 129, no. 2 (2003): P262. http://dx.doi.org/10.1016/s0194-5998(03)01191-4.

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47

Koitschev, Assen, Katharina Gramlich, Sandra Hansmann, et al. "Progressive familial hearing loss in Muckle-Wells syndrome." Acta Oto-Laryngologica 132, no. 7 (2012): 756–62. http://dx.doi.org/10.3109/00016489.2012.656321.

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48

Ravecca, Francesca, Stefano Berrettini, Francesca Forli, et al. "Cx26 Gene Mutations in Idiopathic Progressive Hearing Loss." Journal of Otolaryngology 34, no. 02 (2005): 126. http://dx.doi.org/10.2310/7070.2005.04017.

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Lyos, A. T., M. A. Marsh, H. A. Jenkins, and N. J. Coker. "Progressive Hearing Loss After Transverse Temporal Bone Fracture." Archives of Otolaryngology - Head and Neck Surgery 121, no. 7 (1995): 795–99. http://dx.doi.org/10.1001/archotol.1995.01890070081017.

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Sakuma, Naoko, Hideaki Moteki, Hela Azaiez, et al. "Novel PTPRQ Mutations Identified in Three Congenital Hearing Loss Patients With Various Types of Hearing Loss." Annals of Otology, Rhinology & Laryngology 124, no. 1_suppl (2015): 184S—192S. http://dx.doi.org/10.1177/0003489415575041.

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Abstract:

Objectives: We present 3 patients with congenital sensorineural hearing loss (SNHL) caused by novel PTPRQ mutations, including clinical manifestations and phenotypic features. Methods: Two hundred twenty (220) Japanese subjects with SNHL from unrelated and nonconsanguineous families were enrolled in the study. Targeted genomic enrichment with massively parallel DNA sequencing of all known nonsyndromic hearing loss genes was performed to identify the genetic cause of hearing loss. Results: Four novel causative PTPRQ mutations were identified in 3 cases. Case 1 had progressive profound SNHL with

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