Relevant bibliographies by topics / Prolonged exposure

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Prolonged exposure'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2022

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Journal articles on the topic "Prolonged exposure"

1

Johnson, Brett A., Cynthia C. Woo, Yu Zeng, Zhe Xu, Edna E. Hingco, Joan Ong, and Michael Leon. "Prolonged stimulus exposure reveals prolonged neurobehavioral response patterns." Journal of Comparative Neurology 518, no. 10 (May 15, 2010): 1617–29. http://dx.doi.org/10.1002/cne.22299.

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Underwood, Elizabeth L., JoLeigh Sutton, Ira Keith Ellis, Brian Qualls, Jon Zamber, and Brian N. Walker. "Prolonged coagulopathy after brodifacoum exposure." American Journal of Health-System Pharmacy 71, no. 8 (April 15, 2014): 639–42. http://dx.doi.org/10.2146/ajhp130537.

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Oprel, Danielle A. C., Chris M. Hoeboer, Maartje Schoorl, Rianne A. de Kleine, Marylene Cloitre, Ingrid G. Wigard, Agnes van Minnen, and Willem van der Does. "Effect of Prolonged Exposure, intensified Prolonged Exposure and STAIR+Prolonged Exposure in patients with PTSD related to childhood abuse: a randomized controlled trial." European Journal of Psychotraumatology 12, no. 1 (January 1, 2021): 1851511. http://dx.doi.org/10.1080/20008198.2020.1851511.

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WACHTER, KERRI. "Prolonged Exposure Therapy Lessens PTSD Symptoms." Clinical Psychiatry News 34, no. 2 (February 2006): 26. http://dx.doi.org/10.1016/s0270-6644(06)71163-8.

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Budeyri Gokgoz, Nilay, Fatma Gizem Avci, Kubra Karaosmanoglu Yoneten, Begum Alaybeyoglu, Elif Ozkirimli, Nihat Alpagu Sayar, Dilek Kazan, and Berna Sariyar Akbulut. "Response ofEscherichia colito Prolonged Berberine Exposure." Microbial Drug Resistance 23, no. 5 (July 2017): 531–44. http://dx.doi.org/10.1089/mdr.2016.0063.

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Kanariou, M., E. Petridou, E. Vrachnou, and D. Trichopoulos. "Lymphocyte alterations after prolonged sunlight exposure." Journal of Epidemiology and Biostatistics 6, no. 6 (November 1, 2001): 463–65. http://dx.doi.org/10.1080/135952201317225499.

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Salvadego, Desy, Michail E. Keramidas, Roger Kölegård, Irene Mavelli, Ola Eiken, Igor B. Mekjavic, and Bruno Grassi. "Prolonged Exposure to Hypoxia and Microgravity." Medicine & Science in Sports & Exercise 47 (May 2015): 220. http://dx.doi.org/10.1249/01.mss.0000477027.00901.ee.

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Steenkamp, Maria M., and Brett T. Litz. "Prolonged Exposure Therapy in Veterans Affairs." JAMA Psychiatry 71, no. 2 (February 1, 2014): 211. http://dx.doi.org/10.1001/jamapsychiatry.2013.3305.

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Feirer, Nicola, Christian Honemann, and Marcel Durieux. "PROLONGED BUPIVACAINE EXPOSURE INHIBITS BLOOD COAGULATION." Anesthesiology 89, Supplement (September 1998): 836A. http://dx.doi.org/10.1097/00000542-199809150-00011.

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Ushakov, I. B., and V. P. Fedorov. "The neuromorphological correlates of prolonged radiation exposure." Medicо-Biological and Socio-Psychological Problems of Safety in Emergency Situations, no. 3 (September 28, 2018): 86–97. http://dx.doi.org/10.25016/2541-7487-2018-0-3-86-97.

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Relevance.Liquidators of the consequences of radiation accidents have an increase in neuropsychiatric diseases. However, the structural and functional rearrangement of brain neurons under prolonged low radiation exposure remains virtually unexplored, which makes it difficult to assess the pathogenesis of these diseases.Intention.To study morphological correlates of prolonged low radiation effects on brain neurons in the model of radiobiological experiment.Method.270 white outbred male rats aged 4 months were exposed to radiation-quanta of 60Co in equal portions in total doses of 10; 20; 50 and 100 cGy with a dose rate of 0.5 Gy/h over 5 days. Age control consisted of animals exposed to false radiation. Animal studies complied with GLP standards. Brain fragments were taken after 1 day, 6, 12, 18 and 24 months after irradiation. After a standard histological treatment, changes in the tinctorial properties of neurons, their morphometric parameters, total protein and nucleic acids were evaluated.Results and their analysis. At the end of the observation period, the number of normochromic neurons decreased and the number of hyper and hypochromic cells increased, as well as their destructive forms. The neuro-cellular index decreased over time suggesting the death of some neurons, but without a statistically significant decrease in their number per area. In the post-irradiation period there were observed phase changes in the size of the cytoplasm, nucleus and nucleolus of neurons, as well as the content of protein and nucleic acids (RNA in the cytoplasm and nucleoli, DNA in the nuclei), more pronounced in irradiated animals and sometimes different from those observed in age control animals. As a result, a number of neuromorphological parameters in some periods of observation were not consistent with those in control animals, with a certain instability in the structural and functional organization of neurons.Conclusion.Prolonged low-dose ionizing radiation does not cause significant organic changes in the brain. However, increased radiation exposure or other concomitant adverse factors can be associated with further instability of the structural and functional organization of neurons and serve as a material substrate for a number of deviations in the central nervous system.
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Dissertations / Theses on the topic "Prolonged exposure"

1

Duncan, Benedict. "Metabolic responses to acute and prolonged hypoxic exposure." Thesis, University of Brighton, 2017. https://research.brighton.ac.uk/en/studentTheses/b7ceef66-97ca-4c21-b388-34da9d84ca87.

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This thesis examined the metabolic effects of acute and intermediate hypoxic exposure in humans, specifically, physiological mechanisms associated with weight loss. Namely; increased metabolic rate, changes in substrate oxidation, altered lipid metabolism and changes in taste. Study one assessed the validity and reproducibility of an online gas analyser in normobaric hypoxia [Fraction of inspired oxygen: 0.12 (FiO2:0.12) equivalent to approximately 4,500m] (n=nine; two females, seven males). The MetaMax3x demonstrates good reproducibility between repeated trials. Differences exist between the system and the gold standard Douglas Bag method for measures of oxygen uptake (percent differences of V̇O2; 21%), carbon dioxide production (V̇CO2; 10%) and minute ventilation (V̇E; 5%). The second study investigated the free fatty acid (FFA) and triglyceride (TAG) response to an acute (45 minutes) hypoxic exposure (FiO2: 0.12) (n=10; five females, five males). A greater resting metabolic rate (RMR) (+28 ± 6 kcal.hr-1 ) was observed, through increased carbohydrate (CHO) and fat oxidation. Increased plasma FFA (+54%) and TAG (+26%) were observed, highlighting metabolic perturbations from acute exposure. Study three investigated the metabolic responses to an acute (60 minute) hypoxic exposure (FiO2: 0.12) at rest and a subsequent bout of moderate exercise in normoxia following a high fat meal (n=eight males). Experimental trials included a lipid ingestion prior to a rest period at hypoxia or normoxia followed by moderate intensity exercise (60% heart rate reserve). Control trials consisted of the same protocol without lipid ingestion. Acute, severe hypoxia increased energy expenditure (EE), (+22 ± 11 kcal.hr-1 ) CHO and fat oxidation following exposure. A prior acute bout of severe hypoxia did not alter EE and substrate use during subsequent moderate intensity exercise. An exercise bout, postlipid ingestion, resulted in lower triglyceride concentration. No changes in Meteorin-like were observed throughout trials. These findings suggest that an increase in RMR occurs following a single resting hypoxic exposure and independently to Meteorin-like protein. The fourth study observed reductions in body mass (-2.36 ± 1.41 kg) and increases in CHO oxidation during an altitude stay in Peru (18 days, 3400 m) (n=10; five females, five males). The reduction in body mass (-1.89 ± 1.31 kg) was sustained four weeks post-return to sea-level. Salt, sweet and bitter taste sensations were reduced at 3,400 m compared to sea-level. No changes in self-reported appetite were observed throughout the testing period. Furthermore no changes in circulating Meteorin-like protein were observed upon return to sea-level at one and four weeks post-altitude stay. Study five investigated the blood lipid response to a high lipid meal consumed one and four weeks post-return to sea-level following an altitude stay (18 days, 3400 m) (n=10; five females, five males). No lasting postprandial effects were observed. It is likely that a time dependent effect of hypoxia exists with regards to postprandial blood lipid responses. Taken together acute and intermediate exposure to hypoxic conditions alter substrate oxidation with the potential to induce losses in body mass, independently to changes in Meteorin-like protein and self-reported appetite. Specifically, prolonged stay at moderate altitude results in a greater dependency on CHO use. Increases in RMR were observed during an acute severe bout of hypoxia, although this was not a consistent effect throughout prolonged exposure and should be further investigated. Altered taste during an altitude stay may influence food preferences, energy intake and subsequent changes in body mass and should be considered an area of future investigation. Higher circulating levels of FFA and TAG, demonstrates a metabolic perturbation from a single, acute severe hypoxic exposure.
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Garzon-Villalba, Ximena Patricia. "Assessment of Prolonged Occupational Exposure to Heat Stress." Thesis, University of South Florida, 2016. http://pqdtopen.proquest.com/#viewpdf?dispub=10142405.

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Heat stress is a recognized occupational hazard present in many work environments. Its effects increase with increasing environmental heat loads. There is good evidence that exertional heat illness is associated with ambient thermal conditions in outdoor environments. Further, there is reason to believe that risk of acute injury may also increase with the ambient environment. For these reasons, the assessment of heat stress, which can be done through the characterization of the wet bulb globe temperature (WBGT), is designed to limit exposures to those that could be sustained for an 8-h day. The ACGIH Threshold Limit Value (TLV) for heat stress was based on limited data from Lind in the 1960s. Because there are practical limitations of using thermal indices, measurement of physiological parameters, such as body temperature and heart rate are used with environmental indices or as their alternative.

The illness and injury records from the Deepwater Horizon cleanup effort provided an opportunity to examine the effects of ambient thermal conditions on exertional heat illness and acute injury, and also the cumulative effect of the previous day’s environmental conditions. The ability of the current WBGT-based occupational exposure limits to discriminate unsustainable heat exposures, and the proposal of alternative occupational limits was performed on data from two progressive heat stress protocol trials performed at USF. The USF studies also provided the opportunity to explore physiological strain indicators (rectal temperature, heart rate, skin temperature and the Physiological Strain Index) to determine the threshold between unsustainable and sustainable heat exposures. Analysis were performed using Poisson models, conditional logistic regressions, logistic regressions, and receiver operator curves (ROC curves).

It was found that the odds to present an acute event, either exertional heat illness or acute injuries increased significantly with rising environmental conditions above 20 °C (RR 1.40 and RR 1.06, respectively). There was evidence of the cumulative effect from the prior day’s temperature and increased risk of exertional heat illness (RRs from 1.0–10.4). Regarding the accuracy of the current TLV, the results of the present investigation showed that this occupational exposure limit is extremely sensitive to predict cases associated with unsustainable heat exposures, its area under the curve (AUC) was 0.85; however its specificity was very low (specificity=0.05), with a huge percentage of false positives (95%). The suggested alternative models improved the specificity of the occupational exposure limits (specificities from 0.36 to 0.50), maintaining large AUCs (between 0.84 and 0.89). Nevertheless, any decision in trading sensitivity for specificity must be taken with extreme caution because of the steeped increment risk of heat related illness associated with small increments in environmental heat found also in the present study. Physiologic heat strain indices were found as accurate predictors for unsustainable heat stress exposures (AUCs from 0.74 to 0.89), especially when measurements of heart rate and skin temperature are combined (AUC=0.89 with a specificity of 0.56 at a sensitivity=0.95). Their implementation in industrial settings seems to be practical to prevent unsustainable heat stress conditions.

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Garzon-Villalba, Ximena Garzon-Villalba. "Assessment of Prolonged Occupational Exposure to Heat Stress." Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6240.

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Heat stress is a recognized occupational hazard present in many work environments. Its effects increase with increasing environmental heat loads. There is good evidence that exertional heat illness is associated with ambient thermal conditions in outdoor environments. Further, there is reason to believe that risk of acute injury may also increase with the ambient environment. For these reasons, the assessment of heat stress, which can be done through the characterization of the wet bulb globe temperature (WBGT), is designed to limit exposures to those that could be sustained for an 8-h day. The ACGIH Threshold Limit Value (TLV) for heat stress was based on limited data from Lind in the 1960s. Because there are practical limitations of using thermal indices, measurement of physiological parameters, such as body temperature and heart rate are used with environmental indices or as their alternative. The illness and injury records from the Deepwater Horizon cleanup effort provided an opportunity to examine the effects of ambient thermal conditions on exertional heat illness and acute injury, and also the cumulative effect of the previous day’s environmental conditions. The ability of the current WBGT-based occupational exposure limits to discriminate unsustainable heat exposures, and the proposal of alternative occupational limits was performed on data from two progressive heat stress protocol trials performed at USF. The USF studies also provided the opportunity to explore physiological strain indicators (rectal temperature, heart rate, skin temperature and the Physiological Strain Index) to determine the threshold between unsustainable and sustainable heat exposures. Analysis were performed using Poisson models, conditional logistic regressions, logistic regressions, and receiver operator curves (ROC curves). It was found that the odds to present an acute event, either exertional heat illness or acute injuries increased significantly with rising environmental conditions above 20 °C (RR 1.40 and RR 1.06, respectively). There was evidence of the cumulative effect from the prior day’s temperature and increased risk of exertional heat illness (RRs from 1.0–10.4). Regarding the accuracy of the current TLV, the results of the present investigation showed that this occupational exposure limit is extremely sensitive to predict cases associated with unsustainable heat exposures, its area under the curve (AUC) was 0.85; however its specificity was very low (specificity=0.05), with a huge percentage of false positives (95%). The suggested alternative models improved the specificity of the occupational exposure limits (specificities from 0.36 to 0.50), maintaining large AUCs (between 0.84 and 0.89). Nevertheless, any decision in trading sensitivity for specificity must be taken with extreme caution because of the steeped increment risk of heat related illness associated with small increments in environmental heat found also in the present study. Physiologic heat strain indices were found as accurate predictors for unsustainable heat stress exposures (AUCs from 0.74 to 0.89), especially when measurements of heart rate and skin temperature are combined (AUC=0.89 with a specificity of 0.56 at a sensitivity=0.95). Their implementation in industrial settings seems to be practical to prevent unsustainable heat stress conditions.
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Keller, Stephanie M. "HOMEWORK ADHERENCE IN PROLONGED EXPOSURE FOR CHRONICPOSTTRAUMATIC STRESS DISORDER." Case Western Reserve University School of Graduate Studies / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=case1413366984.

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Berry, Scott. "Chromatin-based memory of prolonged cold exposure in Arabidopsis thaliana." Thesis, University of East Anglia, 2015. https://ueaeprints.uea.ac.uk/58543/.

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All living organisms contain genes. Turning these genes on and off at the appropriate times controls much of an organism’s development and its responses to environmental conditions. In recent years, chromatin has emerged as an important player in orchestrating gene regulation. This thesis focuses on the role of chromatin in the maintenance of gene expression states and their inheritance through cell division. FLOWERING LOCUS C (FLC) in the plant Arabidopsis thaliana is repressed by the prolonged cold of winter, and repression is maintained in subsequent warm conditions. The molecular complexes involved in modulating FLC chromatin are vital for FLC regulation and are conserved among plants and animals, making FLC a paradigmatic system for understanding of the role of chromatin in gene regulation. After cold, FLC chromatin adopts a distinct configuration. In this study, experiments are used to show that this local chromatin ‘state’ instructs its own inheritance through cell division in growing plants. Thus, memory of winter cold is stored in the chromatin of the FLC gene. Mathematical models developed in this work focus on understanding how chromatin states are maintained and also re-established after DNA replication. Minimal models are used to investigate if a particular set of interactions between chromatin and chromatin-modifiers can give rise to the qualitative behaviours, and quantitative results that are observed experimentally. Models developed here make predictions for the FLC system, and more generally show how cis and trans determinants of gene expression can be integrated by chromatin. The role of transcription in determining chromatin states is also examined experimentally by studying the chromatin-associated protein LHP1. LHP1 is required for FLC repression and binds to modified histones associated with repressed FLC chromatin. In this work, it is shown that LHP1 also binds RNA and that this is important for its in vivo function.
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Mackos, Amy. "Infectious Colitis is Exacerbated by Prolonged Stressor Exposure: Implications for Probiotic Intervention." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1385463443.

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Davis, Cindy. "Prolonged Exposure to non-school related media use and violence among urban youth." ScholarWorks, 2016. https://scholarworks.waldenu.edu/dissertations/2478.

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Youth violence is a pervasive and ongoing public health concern. Based on the paradigm of resilience, the purpose of this quantitative study was to test the relationship between prolonged (3 or more hours) of non-school related media use and youth violence. Secondary data from the Centers for Disease Control and Prevention (CDC) were utilized for this study. Logistic and multiple regression models were used to test whether exposure to prolonged non-school related media (video games and TV) use was associated with violent behavior, and whether there was a relationship between prolonged exposure to non-school related media use and electronic bullying among urban youth (N = 1228). Prolonged exposure to both types of non-school related media use was associated with violent behavior (carrying a weapon, physical fighting, perpetrating physical bullying), and playing video and computer games 3 or more hours per day was positively associated with electronic bullying. However, 3 or more hours of TV viewing per day was not associated with electronic bullying. Policy makers, constituents, and parents may benefit from a greater understanding of media exposure and urban youth violence. The knowledge gained from this study may promote positive social change within family systems by increasing parental awareness of what youth do in their unstructured free time and how this impacts subsequent behaviors. Public health professionals, community organizations, and social service agencies in urban communities could incorporate the results to create a culture that supports youth leadership programs that focus on limited use of non-school related media and on violence prevention.
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Mpoloka, Sununguko Wata. "Genetic effects of prolonged UV-B exposure in a Namaqualand daisy - Dimorphotheca sinuata." Doctoral thesis, University of Cape Town, 2001. http://hdl.handle.net/11427/4301.

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Bibliography: leaves 122-139.
This thesis describes investigations into the genetic effects of long term UV-B exposure in Namaqualand daisies (Dimorphotheea sinuata) grown for several generations under ambient and enhanced UV-B levels. Enhanced UV-B radiation was found to have a major effect on the biochemical composition of the chloroplast accompanied by impairment of photosynthetic function, involving a down-regulation of photosynthetic genes and an up-regulation of flavonoid biosynthesis.
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Baier, Allison L. "Investigating Mediators of Change in Prolonged Exposure Therapy and Sertraline for Chronic PTSD." Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1528480809790197.

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Galko, Elizabeth. "Pupil Constriction During Prolonged Exposure to Flickering Stimuli: Evidence for Cholinergic ipRGC Stimulation." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu155558064618568.

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Books on the topic "Prolonged exposure"

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Prolonged exposure. New York: St. Martin's Press, 1998.

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Ann, Hembree Elizabeth, and Rothbaum Barbara Olasov, eds. Prolonged exposure therapy for PTSD: Emotional processing of traumatic experiences : therapist guide. Oxford: Oxford University Press, 2007.

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Foa, Edna B. Prolonged exposure therapy for adolescents with PTSD: Emotional processing of traumatic experiences : therapist guide. Oxford: Oxford University Press, 2008.

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McLeay, D. J. Responses of arctic graling (Thymallus arcticus) to acute and prolonged exposure to Yukon placer miner sediment. S.l: s.n, 1987.

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Beaumont, Matthew William. Exposure of seasonally acclimated brown trout, Salmo trutta, to copper in soft, acidic water: Sub-lethal effects upon oxygen consumption and prolonged swimming performance. Birmingham: University of Birmingham, 1995.

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Havill, Steven F. Prolonged Exposure. Worldwide, 2016.

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Havill, Steven F. Prolonged Exposure (Missing Mystery 36). Poisoned Pen Press, 2001.

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Foa, Edna, Elizabeth A. Hembree, Barbara Olasov Rothbaum, and Sheila Rauch. Prolonged Exposure Therapy for PTSD. Oxford University Press, 2019. http://dx.doi.org/10.1093/med-psych/9780190926939.001.0001.

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This therapist guide of prolonged exposure (PE) treatment is accompanied by the patient workbook, Reclaiming Your Life from a Traumatic Experience. The treatment and manuals are designed for use by a therapist who is familiar with cognitive behavioral therapy (CBT) and who has undergone an intensive training workshop for prolonged exposure by experts in this therapy. The therapist guide instructs therapists to implement this brief CBT program that targets individuals who are diagnosed with posttraumatic stress disorder (PTSD) or who manifest PTSD symptoms that cause distress and/or dysfunction following various types of trauma. The overall aim of the treatment is to help trauma survivors emotionally process their traumatic experiences to diminish or eliminate PTSD and other trauma-related symptoms. The term prolonged exposure (PE) reflects the fact that the treatment program emerged from the long tradition of exposure therapy for anxiety disorders in which patients are helped to confront safe but anxiety-evoking situations to overcome their unrealistic, excessive fear and anxiety. At the same time, PE has emerged from the adaption and extension of Emotional Processing Theory (EPT) to PTSD, which emphasizes the central role of successfully processing the traumatic memory in the amelioration of PTSD symptoms. Throughout this guide, the authors highlight that emotional processing is the mechanism underlying successful reduction of PTSD symptoms.
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Chrestman, Kelly R., Eva Gilboa-Schechtman, and Edna B. Foa. Prolonged Exposure Therapy for PTSD: Teen Workbook. Oxford University Press, 2008. http://dx.doi.org/10.1093/med:psych/9780195331738.001.0001.

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This online treatment program adapts the principles of Dr. Foa's proven effective Prolonged Exposure Therapy for adolescents suffering from Post-traumatic Stress Disorder (PTSD), and is based on the principles of prolonged exposure and emotional processing for use with those individuals who suffer from PTSD. The treatment is presented in modules that can be individually tailored to fit the needs of each patient. Because many adolescent PTSD sufferers do not initiate therapy on their own, but are referred to therapy by social workers, parents, or other authority figures, their willingness to participate in their treatment can vary widely. The first element of this treatment, serves to assess the client's attitude, and increase motivation to change. Other modules introduce psychoeducation, real-life exposure, emotional processing, and relapse prevention. This online workbook provides additional information, monitoring forms, and worksheets to help clients take control of their treatment.
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Brown, Lily A., David Yusko, Hallie Tannahill, and Edna B. Foa. Prolonged Exposure Therapy for Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0030.

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This chapter presents an overview of prolonged exposure therapy (PE), a highly efficacious and effective treatment for post-traumatic stress disorder (PTSD). First, emotional processing theory is reviewed, which provides the theoretical basis for PE and the key mechanisms underlying PTSD symptom reduction. Next, a synthesis of the robust evidence for the efficacy and effectiveness of PE is provided. The chapter reviews evidence that in addition to ameliorating PTSD symptoms, PE reduces secondary symptoms such as depression, suicidal ideation, anger, and substance use disorders. The chapter describes evidence supporting the extension of PE with unique samples, including individuals with psychosis, persons with self-injurious behavior, and war veterans. The chapter concludes with a review of the status of PE dissemination and implementation efforts.
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Book chapters on the topic "Prolonged exposure"

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McLean, Carmen P., Anu Asnaani, and Edna B. Foa. "Prolonged Exposure Therapy." In Evidence Based Treatments for Trauma-Related Psychological Disorders, 143–59. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-07109-1_8.

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Nacasch, Nitsa, Lilach Rachamim, and Edna B. Foa. "Prolonged Exposure Treatment." In Future Directions in Post-Traumatic Stress Disorder, 245–51. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7522-5_11.

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Pagel, J. F. "Prolonged Exposure Therapy." In Post-Traumatic Stress Disorder, 91–98. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-55909-0_11.

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Hoyer, Jürgen, and Katja Beesdo-Baum. "Prolonged Imaginal Exposure Based on Worry Scenarios." In Exposure Therapy, 245–60. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3342-2_14.

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Gallagher, Thea, Julie Petersen, and Edna B. Foa. "Prolonged exposure for PTSD." In Casebook to the APA Clinical Practice Guideline for the treatment of PTSD., 123–37. Washington: American Psychological Association, 2020. http://dx.doi.org/10.1037/0000196-006.

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Rauch, Sheila A. M., and Carmen P. McLean. "Theory and prolonged exposure." In Retraining the brain: Applied neuroscience in exposure therapy for PTSD., 3–18. Washington: American Psychological Association, 2021. http://dx.doi.org/10.1037/0000242-001.

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Rauch, Sheila A. M., and Carmen P. McLean. "Augmentation of prolonged exposure." In Retraining the brain: Applied neuroscience in exposure therapy for PTSD., 129–43. Washington: American Psychological Association, 2021. http://dx.doi.org/10.1037/0000242-012.

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Rachamim, Lilach, Liat Helpman, Eva Gilboa-Schechtman, and Elna Yadin. "Prolonged Exposure Treatment for Children." In Comprehensive Guide to Post-Traumatic Stress Disorders, 1331–47. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-08359-9_86.

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Rachamim, Lilach, Liat Helpman, Eva Gilboa-Schechtman, and Elna Yadin. "Prolonged Exposure Treatment for Children." In Comprehensive Guide to Post-Traumatic Stress Disorder, 1–13. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-08613-2_86-1.

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Rauch, Sheila A. M., and Carmen P. McLean. "What is prolonged exposure therapy?" In Retraining the brain: Applied neuroscience in exposure therapy for PTSD., 19–38. Washington: American Psychological Association, 2021. http://dx.doi.org/10.1037/0000242-002.

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Conference papers on the topic "Prolonged exposure"

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Celis Valdiviezo, Eduardo A., Angel O. Coz, and Zachary Q. Morris. "Progressive Desquamative Interstitial Pneumonitis Secondary To Prolonged Aluminum Exposure." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3214.

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Zwick, Harry, David O. Robbins, and Maqsood Nawim. "Transient Visual Effects Of Prolonged Small Spot Foveal Laser Exposure." In OE/LASE '89, edited by Kazuhiko Atsumi, Norman R. Goldblatt, and Stephen N. Joffe. SPIE, 1989. http://dx.doi.org/10.1117/12.952069.

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Czarnocki, K., E. Czarnocka, and T. Baum. "253. Occupational Prolonged Exposure Evaluation to Multiple Chemicals in Work Environment." In AIHce 2004. AIHA, 2004. http://dx.doi.org/10.3320/1.2758184.

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Pshchelko, Nikolai, Ekaterina Vodkailo, and Olga Tsareva. "Trouble-free Operation of Ionic Dielectrics During Prolonged Exposure to Voltage." In 2019 IEEE International Conference on Electrical Engineering and Photonics (EExPolytech). IEEE, 2019. http://dx.doi.org/10.1109/eexpolytech.2019.8906886.

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Czarnocki, K. "157. Biological Indicators in the Incidental and Prolonged Exposure Evaluation to Hydrocarbons." In AIHce 2001. AIHA, 2001. http://dx.doi.org/10.3320/1.2765672.

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Koltunowicz, T., G. Bajracharya, D. Djairam, Muhannad Al-Suhaily, and J. Smit. "The effects of prolonged exposure of paper insulation to fast repeating transients." In 2010 International Conference on High Voltage Engineering and Application (ICHVE). IEEE, 2010. http://dx.doi.org/10.1109/ichve.2010.5640836.

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Lu, Qing, Julie Newton, Michael Blackburn, and Sharon Rounds. "Prolonged Adenosine Exposure Increases Lung Endothelial Permeability Via RAC1-Mediated Oxidative Stress." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1978.

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Bhatti, Mehwish S., Waqas Rasheed, Tong Boon Tang, and Augustinus Laude. "Investigating eye-strain due to prolonged exposure to low resolution multimedia using LSFG." In 2016 6th International Conference on Intelligent and Advanced Systems (ICIAS). IEEE, 2016. http://dx.doi.org/10.1109/icias.2016.7824096.

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Ozaki, Yo, and Ralph H. Zee. "Lifetime of advanced material components in space nuclear systems due to prolonged exposure." In Proceedings of the 12th symposium on space nuclear power and propulsion: Conference on alternative power from space; Conference on accelerator-driven transmutation technologies and applications. AIP, 1995. http://dx.doi.org/10.1063/1.47191.

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Bandara, M., JT Dziodzio, and JA Yahwak. "Prolonged Exposure to Nasogastric Tube as a Risk Factor for Ventilator Associated Pneumonia." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1735.

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Reports on the topic "Prolonged exposure"

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Mader, Thomas, and Christopher L. Blanton. Refractive Changes during Prolonged Exposure to Altitude Following Refractive Surgery. Fort Belvoir, VA: Defense Technical Information Center, October 1994. http://dx.doi.org/10.21236/ada290263.

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Gahm, Gregory, Greg Reger, Albert Rizzo, Patricia Koenen-Woods, and Nancy Skopp. Comparing Virtual Reality Exposure Therapy to Prolonged Exposure in the Treatment of Soldiers with PTSD. Fort Belvoir, VA: Defense Technical Information Center, June 2014. http://dx.doi.org/10.21236/ada613612.

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Gahm, Gregory, Greg Reger, Kevin Holloway, Albert Rizzo, Patricia Koenen-Woods, Kimberlee Zetocha, and Nancy Skopp. Comparing Virtual Reality Exposure Therapy to Prolonged Exposure in the Treatment of Soldiers with PTSD. Fort Belvoir, VA: Defense Technical Information Center, June 2012. http://dx.doi.org/10.21236/ada567846.

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Gahm, Gregory, Greg Reger, Albert Rizzo, Patricia Koenen-Woods, and Nancy Skopp. Comparing Virtual Reality Exposure Therapy to Prolonged Exposure in the Treatment of Soldiers with PTSD. Fort Belvoir, VA: Defense Technical Information Center, June 2013. http://dx.doi.org/10.21236/ada588910.

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Gahm, Gregory. Comparing Virtual Reality Exposure Therapy to Prolonged Exposure in the Treatment of Soldiers with PTSD. Fort Belvoir, VA: Defense Technical Information Center, June 2011. http://dx.doi.org/10.21236/ada555161.

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Gahm, Gregory, and Greg Reger. Comparing Virtual Reality Exposure Therapy to Prolonged Exposure in the Treatment of Soldiers with PTSD. Fort Belvoir, VA: Defense Technical Information Center, June 2010. http://dx.doi.org/10.21236/ada543849.

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Foa, Edna B., Carmen McLean, and Laurie J. Zandberg. Implementation of Prolonged Exposure in the Army: Is Consultation Necessary for Effective Dissemination? Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada610843.

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Rauch, Sheila. Randomized Controlled Trial of Sertraline, Prolonged Exposure Therapy and Their Combination of OEF/OIF with PTSD. Fort Belvoir, VA: Defense Technical Information Center, December 2011. http://dx.doi.org/10.21236/ada580287.

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Gahm, Gregory, Greg Reger, Mary V. Ingram, Mark Reger, and Albert Rizzo. A Multisite, Randomized Clinical Trial of Virtual Reality and Prolonged Exposure Therapy for Active Duty Soldiers with PTSD. Fort Belvoir, VA: Defense Technical Information Center, February 2015. http://dx.doi.org/10.21236/ada614117.

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Gahm, Gregory, Greg Reger, Mary V. Ingram, Mark Reger, and Albert Rizzo. A Multisite, Randomized Clinical Trial of Virtual Reality and Prolonged Exposure Therapy for Active Duty Soldiers with PTSD. Fort Belvoir, VA: Defense Technical Information Center, December 2012. http://dx.doi.org/10.21236/ada579116.

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