Relevant bibliographies by topics / Prostaglandin

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Prostaglandin'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Prostaglandin"

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Sirchak, Yelyzaveta S., Oleksandr O. Boldizhar, Yaroslav F. Filak, et al. "CHANGES IN PROSTAGLANDIN LEVELS IN BLOOD SERUM OF PATIENTS WITH GASTROESOPHAGEAL REFLUX DISEASE ON THE BACKGROUND OF THE OSTEOCHONDROSIS OF THE SPINE AND OBESITY." Wiadomości Lekarskie 75, no. 10 (2022): 2497–500. http://dx.doi.org/10.36740/wlek202210134.

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The aim: To study the features of changes in the level of prostaglandins (I2 and F2α) in blood serum of patients GERD on the background of OH of the cervical and thoracic spine and obesity. Materials and methods: The examined patients included 56 patients with GERD and OH of the cervical and thoracic spine. All patients had their blood serum prostaglandin (Pg) F2α and 6-keto prostaglandin F1α (blood prostacyclin – Pg I2) levels examined using the method of immunoassay analysis. Results: In all patients with GERD and OH an excessive body weight or obesity of varying degrees was found while anal
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Kirchner, K. A. "Role of prostaglandins in renin suppression during acute potassium loading." American Journal of Physiology-Renal Physiology 248, no. 3 (1985): F360—F365. http://dx.doi.org/10.1152/ajprenal.1985.248.3.f360.

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Acute K loading suppresses plasma renin activity (PRA) and may alter prostaglandin biosynthesis. To determine whether the effect of K on PRA was modulated through changes in the prostaglandin system, dietary NaCl-restricted rats were infused with prostaglandin I2. PRA was then determined before and 90 min after a KNO3 infusion. Control rats received only the prostaglandin infusion. PRA increased in rats receiving prostaglandins alone. K loading prevented the increase in PRA. To further exclude prostaglandins in PRA suppression during K loading, the effects of K on PRA were determined during pr
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Jara-Gutiérrez, Álvaro, and Victoriano Baladrón. "The Role of Prostaglandins in Different Types of Cancer." Cells 10, no. 6 (2021): 1487. http://dx.doi.org/10.3390/cells10061487.

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The prostaglandins constitute a family of lipids of 20 carbon atoms that derive from polyunsaturated fatty acids such as arachidonic acid. Traditionally, prostaglandins have been linked to inflammation, female reproductive cycle, vasodilation, or bronchodilator/bronchoconstriction. Recent studies have highlighted the involvement of these lipids in cancer. In this review, existing information on the prostaglandins associated with different types of cancer and the advances related to the potential use of them in neoplasm therapies have been analyzed. We can conclude that the effect of prostaglan
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Martens, Matthew D., Amy S. Fernando, and Joseph W. Gordon. "A new trick for an old dog? Myocardial-specific roles for prostaglandins as mediators of ischemic injury and repair." American Journal of Physiology-Heart and Circulatory Physiology 320, no. 6 (2021): H2169—H2184. http://dx.doi.org/10.1152/ajpheart.00872.2020.

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The small lipid-derived paracrine signaling molecules known as prostaglandins have been recognized for their ability to modulate many facets of cardiovascular physiology since their initial discovery more than 85 years ago. Although the role of prostaglandins in the vasculature has gained significant attention across time, a handful of historical studies have also directly implicated the cardiomyocyte in both prostaglandin synthesis and release. Recently, our understanding of how prostaglandin receptor modulation impacts and contributes to myocardial structure and function has gained attention
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Codde, James P., and Lawrence J. Beilin. "Dietary fish oil prevents dexamethasone induced hypertension in the rat." Clinical Science 69, no. 6 (1985): 691–99. http://dx.doi.org/10.1042/cs0690691.

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1. This study was designed to examine the effect of dexamethasone treatment on tissue and urinary prostanoids, and to determine whether inhibition of prostaglandin biosynthesis by manipulation of dietary fatty acids accelerates the development of glucocorticoid hypertension. 2. Forty-eight rats were placed on either a 2-series prostaglandin ‘inhibitory’ diet (cod liver oil/linseed oil) or a control diet of saturated fat for an initial period of 4 weeks. The groups were then divided into two so that half of each received dexamethasone in their drinking water (2.5 mg/1) for 1 week whilst continu
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Leighton, B., L. Budohoski, F. J. Lozeman, R. A. J. Challiss та E. A. Newsholme. "The effect of prostaglandins E1, E2 and F2α and indomethacin on the sensitivity of glycolysis and glycogen synthesis to insulin in stripped soleus muscles of the rat". Biochemical Journal 227, № 1 (1985): 337–40. http://dx.doi.org/10.1042/bj2270337.

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Prostaglandins E1 and E2 increased the sensitivity of glycolysis to insulin in the isolated stripped soleus muscle of the rat, but prostaglandin F2 alpha had no effect. Indomethacin, which inhibits prostaglandin formation, markedly decreased the sensitivity of glycolysis to insulin. These findings suggest that prostaglandins of the E series increase the sensitivity of muscle glycolysis to insulin in vivo.
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Tiwary, Ekta, Muhan Hu, and Jeevan K. Prasain. "Sperm-Guiding Unconventional Prostaglandins in C. elegans: Synthesis and Signaling." Metabolites 11, no. 12 (2021): 853. http://dx.doi.org/10.3390/metabo11120853.

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Prostaglandins comprise a family of lipid signaling molecules derived from polyunsaturated fatty acids and are involved in a wide array of biological processes, including fertilization. Prostaglandin-endoperoxide synthase (a.k.a. cyclooxygenase or Cox) initiates prostaglandin synthesis from 20-carbon polyunsaturated fatty acids, such as arachidonic acid. Oocytes of Caenorhabditis elegans (C. elegans) have been shown to secrete sperm-guidance cues prostaglandins, independent of Cox enzymes. Both prostaglandin synthesis and signal transduction in C. elegans are environmentally modulated pathways
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Nassar, Bassam A., Yung-Sheng Huang, Alan T. J. McDonald, Kenneth D. Jenkins, and David F. Horrobin. "The influence of phenelzine and tranylcypromine on the release of prostaglandins from the rat mesenteric vascular bed." Canadian Journal of Physiology and Pharmacology 66, no. 9 (1988): 1206–9. http://dx.doi.org/10.1139/y88-198.

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We investigated the effects of phenelzine and tranylcypromine on the release of prostacyclin, thromboxane A2, prostaglandin E2, and prostaglandin E1 from the isolated perfused rat mesenteric vascular bed. Perfusion of the preparation with phenelzine in concentrations of 15, 45, and 135 μM for 150 min led to attenuated release of all four prostaglandins measured. Inhibition generally occurred with the lowest dose used and was most prominent with the highest concentration. Tranylcypromine also decreased prostaglandin formation. However, low doses were not effective in the suppression of prostacy
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Tsuchida, Keiichiro, Takae Ibuki, and Kiyoshi Matsumura. "Bromoenol Lactone, an Inhibitor of Calcium-Independent Phospholipase A2, Suppresses Carrageenan-Induced Prostaglandin Production and Hyperalgesia in Rat Hind Paw." Mediators of Inflammation 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/605727.

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Prostaglandin (PG) E2and PGI2are essential to hyperalgesia in inflammatory tissues. These prostaglandins are produced from arachidonic acid, which is cleaved from membrane phospholipids by the action of phospholipase A2(PLA2). Which isozyme of PLA2is responsible for the cleavage of arachidonic acid and the production of prostaglandins essential to inflammation-induced hyperalgesia is not clear. In this study, we examined the effects of two PLA2isozyme-specific inhibitors on carrageenan-induced production of PGE2and PGI2in rat hind paw and behavioral nociceptive response to radiant heat. Local
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Surbek, Hösli, and Holzgreve. "Current aspects of labor induction." Therapeutische Umschau 59, no. 12 (2002): 650–54. http://dx.doi.org/10.1024/0040-5930.59.12.650.

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Die Geburtseinleitung ist eines der wichtigsten therapeutischen Interventionsmittel in der Geburtshilfe und Schwangerschaftsmedizin. Das Ziel der Einleitung ist es, das bessere «perinatale Ergebnis» für Mutter und Kind im Gegensatz zum abwartenden Vorgehen zu erreichen. Zu den verschiedenen Methoden der Geburtseinleitung gehören insbesondere die Verabreichung von Oxytocin oder Prostaglandinen, die Amniotomie, und mechanische Methoden der Zervixdilatation. Der Erfolg der Geburtseinleitung ist vorab abhängig von der Geburtsbereitschaft des Uterus und von der verwendeten Methode. Bei sehr reifer
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Dissertations / Theses on the topic "Prostaglandin"

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Venturin, Gabriela Lovizutto. "Efeito regulador da PGE2 na produção de TNF-α e IL-17 na atividade microbicida na leishmaniose canina. /". Araçatuba, 2019. http://hdl.handle.net/11449/183297.

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Orientador: Valéria Marçal Felix de Lima<br>Resumo: A leishmaniose canina (CanL) é causada pelo parasito intracelular Leishmania infantum. Devido ao alto parasitismo na pele o cão é considerado o principal reservatório urbano da L. Infantum. A prostaglandina-E2 (PGE2) possui propriedades reguladoras potentes do sistema imunológico e pode se ligar aos receptores EP1, EP2 e EP4 que geram ativação celular ou EP3 que gera inibição de resposta celular. Na CanL o papel regulador da PGE2 ainda não foi estudado, por isso, os parâmetros foram avaliados em cultura de leucócitos esplênicos de cães com Ca
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Javed, T. "Synthesis of prostaglandins and prostaglandin analogues from norbornadiene." Thesis, University of Salford, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356170.

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Nielsch, A. S. "Effects of prostaglandins and prostaglandin synthetase inhibitors on liver toxicity." Thesis, University of Aberdeen, 1987. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU499301.

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A total of 22 non-steroidal anti-inflammatory drugs and derivatives were added to microsomes to study the denaturation of cytochrome P-450 to cytochrome P-420 in the absence of an NADPH-generating system. There was a highly significant correlation among the different compounds between the extent of denaturation of cytochrome P-450 and their surfactant potency. Endotoxin administration to rats caused a maximum decrease in hepatic microsomal enzymes after 24 hours. Significant decreases in cytochrome P-450 (40%), cytochrome b5 levels (22%), aminopyrine N-demethylase (31%) and biphenyl 4-hydroxyl
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Ujjan, Safdar Ali. "Prostaglandin D2 (PGD2) signalling and male germ cell : differentiation in the mouse embryonic testis." Thesis, Montpellier 2, 2014. http://www.theses.fr/2014MON20099.

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La détermination du sexe et la différenciation des cellules germinales est un processus très organisé qui commence au stade embryonnaire et se termine à la vie adulte. Dans les gonades embryonnaires l'expression de Sry suivie par l'expression de Sox9 initie le développement testiculaire tandis que, en l'absence de l'expression de Sry, les gènes associés au sexe féminin initient le développement des ovaires. Les cellules germinales qui ont migré vers les gonades nouvellement formées continuent leur prolifération intense jusqu'à ce qu'ils s'engagent dans la voie le mâle ou femelle. La décision d
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Gagliolo, Jean-Marie. "Rôle de la sénescence des fibroblastes dans la physiopathologie de la bronchopneumopathie chronique obstructive." Thesis, Paris Est, 2013. http://www.theses.fr/2013PEST1065.

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La sénescence, perte irréversible des capacités réplicatives des cellules associée à la sécrétion de médiateurs inflammatoires, pourrait participer au développement de l'atteinte pulmonaire dans la bronchopneumopathie chronique obstructive (BPCO) en initiant, maintenant et propageant un état inflammatoire. L'objectif de ce travail était d'évaluer les mécanismes de la sénescence impliqués dans l'induction et le maintien de l'inflammation au cours de la BPCO. Ainsi, des fibroblastes pulmonaires de témoins et de patients atteints de BPCO ont été mis en culture à long terme. Un phénotype sénescent
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Pereira, Priscilla Aparecida Tartari. "Efeitos antagônicos da prostaglandina D2 e prostaglandina E2 na resposta imune durante infecção experimental por Histoplasma capsulatum." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-06012014-081806/.

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O Histoplasma capsulatum é um fungo dimórfico, patogênico e responsável por graves lesões pulmonares. A infecção é adquirida pela inalação de conídios e posterior conversão para leveduras nos alvéolos e bronquíolos, onde são fagocitadas por macrófagos alveolares residentes e leucócitos que migram para o local da infecção. Recentemente, demonstramos que animais infectados com H. capsulatum e tratados com inibidor da síntese de prostaglandinas apresentaram diminuição de carga fúngica nos pulmões e baço, aumento da produção de nitrito e da fagocitose de leveduras por macrófagos alveolares, e maio
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Oliveira, Milena Lopes. "Ação do 17β-estradiol na síntese de PGF2α endometrial em vacas." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/10/10131/tde-25082017-095013/.

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O 17&#946;-E2 estimula a expressão de receptores endometriais, ER e OXTR. A ativação de OXTR induz a ativação da cadeia de síntese de PGF2&#945;. A hipótese do presente estudo é que as enzimas de síntese de PGF2&#945; são reguladas pelo 17&#946;-E2. Objetivou-se determinar os efeitos do 17&#946;-E2 na expressão gênica e proteica, assim como na localização de proteínas envolvidas na síntese de PGF2&#945;. Vacas Nelore multíparas, solteiras e cíclicas foram sincronizadas por aplicação de BE e inserção de dispositivo de P4. Após 8 dias realizou-se a remoção do dispositivo de P4 e aplicação de PGF
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Feltrin, Isabella Rio. "Efeitos do 17β-estradiol na abundância de transcritos para enzimas envolvidas na síntese de PGF2α endometrial em fêmeas bovinas no final do diestro". Botucatu, 2020. http://hdl.handle.net/11449/192193.

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Orientador: Claudia Maria Bertan Membrive<br>Resumo: Em bovinos, o 17β-estradiol (17β-E2) estimula a expressão de receptores de estradiol (ER) e ocitocina (OXTR) no endométrio. A ativação de OXTR induz a ativação de uma complexa cascata que resulta na síntese de PGF2α. A hipótese é que o tratamento com 17β-E2, 15 dias após o estro (D15), modula a expressão gênica das proteínas quinase (PKC) e fosfolipase A2 (PLA2), ambas envolvidas na síntese de PGF2α e dependentes de cálcio. Objetivou-se neste estudo determinar os efeitos do 17β-E2 na abundância de trancritos (PKCα, PKCβ, PLA2G4, AKR1B1, AKR1
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Gyomorey, Sandor. "Temporal expression of prostaglandin endoperoxide H synthase type 2 and prostaglandin receptors at birth." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0023/MQ40864.pdf.

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Matthews, Jane Simone. "Characterisation of prostaglandin E receptors." Thesis, University of Edinburgh, 1993. http://hdl.handle.net/1842/19997.

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Prostaglandin (PG) E<SUB>2</SUB> has been shown to act on at least three distinct receptor subtypes, designated EP<SUB>1</SUB>, EP<SUB>2</SUB> and EP<SUB>3</SUB>, with recent evidence suggesting the possibility of a fourth EP-receptor subtype. Whilst good antagonists are not available, a range of PGE analogues of differing selectivity for the three receptor subtypes may be used to determine which is effective in a given system. The activity of these analogues has not yet been assessed on the putative EP<SUB>4</SUB>-receptor containing preparation. This research has involved the use of such com
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Books on the topic "Prostaglandin"

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Javed, Tariq. Synthesis of prostaglandins and prostaglandin analogues from norbornadiene. University of Salford, 1985.

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Diehm, Curt, Helmut Sinzinger, and Waltraud Rogatti, eds. Prostaglandin E1. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76910-8.

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Heidrich, H., H. Böhme, and Waltraud Rogatti, eds. Prostaglandin E1. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73943-9.

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Sinzinger, Helmut, and Waltraud Rogatti, eds. Prostaglandin E1 in Atherosclerosis. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71679-9.

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Wasinger, Thomas. Prostaglandin receptors and prostaglandin metabolism in the terminal vascular bed of the brain. Facultas Universitätsverlag, 1989.

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Taipei Conference on Prostaglandin and Leukotriene Research (1988). Taipei Conference on Prostaglandin and Leukotriene Research. Edited by Samuelsson Bengt, Wong Patrick Y. -K, and Sun Frank F. Raven, 1988.

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Gyomorey, Sandor. Temporal expression of prostaglandin endoperoxide H synthase type 2 and prostaglandin receptors at birth. National Library of Canada, 1998.

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Samuelsson, Bengt, R. Paoletti, Giancarlo C. Folco, E. Granström, and S. Nicosia, eds. Advances in Prostaglandin and Leukotriene Research. Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9721-0.

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Walden, Thomas L., and Haywood N. Hughes, eds. Prostaglandin and Lipid Metabolism in Radiation Injury. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5457-4.

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1934-, Harris Jules E., Braun Donald P, and Anderson Kenning M, eds. Prostaglandin inhibitors in tumor immunology and immunotherapy. CRC Press, 1994.

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Book chapters on the topic "Prostaglandin"

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Donato, Dominique M., Steven K. Hanks, Kenneth A. Jacobson, et al. "Prostaglandin." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101094.

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Gooch, Jan W. "Prostaglandin." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14584.

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Giangiacomo, Annette. "Prostaglandin Analogues." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_121-2.

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Hubl, W. "Prostaglandin E2." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_2394-1.

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Degen, Gisela H., Christoph Vogel, and Josef Abel. "Prostaglandin Synthases." In Enzyme Systems that Metabolise Drugs and Other Xenobiotics. John Wiley & Sons, Ltd, 2002. http://dx.doi.org/10.1002/0470846305.ch6.

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Giangiacomo, Annette. "Prostaglandin Analogues." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_121.

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Hubl, W. "Prostaglandin E2." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_2394.

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Wilson, John Fawcett. "Prostaglandin D2." In The Immunoassay Kit Directory. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0679-5_85.

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Wilson, John Fawcett. "Prostaglandin E2." In The Immunoassay Kit Directory. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0679-5_86.

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Wilson, John Fawcett. "Prostaglandin F2α." In The Immunoassay Kit Directory. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0679-5_87.

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Conference papers on the topic "Prostaglandin"

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Maher, SA, MA Birrell, and MG Belvisi. "Prostaglandin E2Mediates Cough Via the EP3Receptor." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6056.

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Zhou, Kaile. "Prostaglandin activity in established cell lines." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.108381.

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Konya, Viktoria, Eva M. Sturm, Petra Luschnig-Schratl, and Akos Heinemann. "Endothelial Prostaglandin I2 Restrains Eosinophil Trafficking." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6401.

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Sheehan, S. J., A. B. Latif, S. R. Bibby, R. C. Kester, and S. M. Rajah. "THE RECOVERY OF ENDOTHELIAL CELL AND PLATELET PROSTAGLANDIN SYNTHESES AFTER ADMINISTRATION OF ASPIRIN AND INDOBUFEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643389.

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Suppression of endothelial prostacyclin (PGI2) by cyclooxygenase inhibition may contribute to the formation of neointimal hyperplasia. While aspirin (ASA) inhibits platelet function for several days, it returns to normal within 24 hours after indobufen (IDB), a reversible cyclooxygenase inhibitor, suggesting that prostacyclin may recover at an even faster rate. In a randomised, controlled study, recovery of platelet and endothelial cell prostaglandin synthesis was compared in 49 New Zealand white rabbits following indobufen (3.5 mg/kg) or aspirin (5 mg/kg). Prostacyclin in arterial incubates a
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Van Nueten, J. M., W. J. Janssens, and F. De Clerck. "VASOCONSTRICTION IN RESPONSE TO HUMAN PLATELET-VESSEL WALL INTERACTIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644598.

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Human blood platelets, stimulated with thrombin, induced contractions of isolated basilar artery segments of the dog. These platelet-mediated vascular contractions were inhibited in a concentration-dependent way by flunarizine, a Ca2+-entry blocker, selective for vascular tissues. This inhibition increased gradually as a function of time after contact with flunarizine to reach its maximum after 60-90 min. Biochemical and pharmacological analyses, using the 5-HT2-serotonergic antagonist ritanserin, the thromboxane A2/prostaglandin endo-peroxide antagonist BM 13.177 and the fatty acid cyclo-oxyg
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Spears, Mark, Rekha Chaudhuri, Charles McSharry, et al. "Elevated Sputum Prostaglandin D2 (PGD2) Concentrations In Asthma." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4448.

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Poskitt, K. R., J. T. C. Irwin, C. M. Backhouse, and C. N. McCollum. "MICROEMBOLISATION DURING SURGICAL SHOCK: EFFECT OF PROSTAGLANDIN E1." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643450.

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Embolisation of microaggregates following major surgery may be a cause of pulmonary arterio-venous shunt and postoperative respiratory failure (1). Prostaglandin E1 may prevent intravascular aggregation and we studied this possibility in a pig model of surgical shock.Following autologous platelet labelling with Indium, 16 pigs (20-30kg) were randomised to receive a perioperative infusion of PGE1 (100ng/kg/min) or placebo. Arterial and Swann Ganz catheters were inserted under anaesthesia prior to surgery consisting of midline laparotomy, exteriorisation of small bowel 1.5 hours of aortic clampi
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Perelman, Juliy M., Eduard V. Nekrasov, Anna G. Prikhodko, and Galina A. Makarova. "A metabolite of prostaglandin D2, 15-deoxy-?12,14-prostaglandin J2 (15d-PGJ2), in exhaled breath condensate (EBC) of asthma patients." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4946.

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Kock, Anna, Karin Larsson, Linda Ljungblad, et al. "Abstract 3986: Characterization of prostaglandin signaling in primary neuroblastoma." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3986.

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Oga, Toru, Daiji Sakata, Kiminobu Tanizawa, et al. "Prostaglandin F2±-FP Signaling Facilitates Bleomycin-induced Pulmonary Fibrosis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1967.

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Reports on the topic "Prostaglandin"

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Moreno, Jacqueline. Vitamin D-Prostaglandin Interactions and Effects on Prostate Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada446263.

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Moreno, Jacqueline. Vitamin D-Prostaglandin Interactions and Effects in Prostate Cancer. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada463443.

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Naseem, Syed M., K. A. Mereish, Rikki Solow, and Harry Hines. Toxin-Induced Activation of Rat Hepatocyte Prostaglandin Synthesis and Phospholipid Metabolism. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada221157.

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McDonagh, Marian, Andrea C. Skelly, Amy Hermesch, et al. Cervical Ripening in the Outpatient Setting. Agency for Healthcare Research and Quality (AHRQ), 2021. http://dx.doi.org/10.23970/ahrqepccer238.

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Abstract:
Objectives. To assess the comparative effectiveness and potential harms of cervical ripening in the outpatient setting (vs. inpatient, vs. other outpatient intervention) and of fetal surveillance when a prostaglandin is used for cervical ripening. Data sources. Electronic databases (Ovid® MEDLINE®, Embase®, CINAHL®, Cochrane Central Register of Controlled Trials, and Cochrane Database of Systematic Reviews) to July 2020; reference lists; and a Federal Register notice. Review methods. Using predefined criteria and dual review, we selected randomized controlled trials (RCTs) and cohort studies o
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Liebert, Monica. Increase in Breast Cancer Stem Cells in Response to Prostaglandin E2. Revision. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada583511.

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Kim, Sangmi. Urinary Level of Prostaglandin E2 Metabolite and Risk of Incident Breast Cancer. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada559390.

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Fields, Michael, Mordechai Shemesh, Philip A. Fields, Anna-Riitta Fuchs, and Michael Diskin. Bovine Relaxin: A Placental Source and Effects on Prostaglandin and Steroid Metabolism. United States Department of Agriculture, 1988. http://dx.doi.org/10.32747/1988.7568097.bard.

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Fields, Michael, Mordechai Shemesh, and Phillip A. Fields. Bovine Relaxin: A Placental Source and Effects on Prostaglandin and Steroid Metabolism. United States Department of Agriculture, 1991. http://dx.doi.org/10.32747/1991.7594379.bard.

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Will, James A., Ricardo Saban, and Nian-Ji Li. Pharmacologic Studies on the In Vitro Bronchodilating Vasoactive Actions of Oligo-PGB (Prostaglandin B). Defense Technical Information Center, 1988. http://dx.doi.org/10.21236/ada193138.

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Eck, Stephen L. The Role of Breast Cancer Derived Prostaglandin E2 in the Elaboration of a Therapeutic Immune Response. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada409420.

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You might also be interested in the extended bibliographies on the topic 'Prostaglandin' for particular source types:
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