Relevant bibliographies by topics / Prostaglandin E receptor EP2

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Academic literature on the topic 'Prostaglandin E receptor EP2'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Prostaglandin E receptor EP2"

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Breyer, Matthew D., and Richard M. Breyer. "Prostaglandin E receptors and the kidney." American Journal of Physiology-Renal Physiology 279, no. 1 (2000): F12—F23. http://dx.doi.org/10.1152/ajprenal.2000.279.1.f12.

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Prostaglandin E2 is a major renal cyclooxygenase metabolite of arachidonate and interacts with four G protein-coupled E-prostanoid receptors designated EP1, EP2, EP3, and EP4. Through these receptors, PGE2modulates renal hemodynamics and salt and water excretion. The intrarenal distribution and function of EP receptors have been partially characterized, and each receptor has a distinct role. EP1 expression predominates in the collecting duct where it inhibits Na+ absorption, contributing to natriuresis. The EP2 receptor regulates vascular reactivity, and EP2 receptor-knockout mice have salt-se
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Patwardhan, A. M., J. Vela, J. Farugia, K. Vela, and K. M. Hargreaves. "Trigeminal Nociceptors Express Prostaglandin Receptors." Journal of Dental Research 87, no. 3 (2008): 262–66. http://dx.doi.org/10.1177/154405910808700306.

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Orofacial inflammation is associated with prostaglandin release and the sensitization of nociceptive receptors such as the transient receptor potential subtype V1 (TRPV1). We hypothesized that certain PGE2 receptor subtypes (EP1–EP4) are co-expressed with TRPV1 in trigeminal nociceptors and sensitize responses to a TRPV1 agonist, capsaicin. Accordingly, combined in situ hybridization was performed with immunohistochemistry on rat trigeminal ganglia. We next evaluated the effects of specific EP2 and EP3 agonists (butaprost and sulprostone) in cultured trigeminal ganglia neurons. The results sho
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Zahner, Gunther, Melanie Schaper, Ulf Panzer, et al. "Prostaglandin EP2 and EP4 receptors modulate expression of the chemokine CCL2 (MCP-1) in response to LPS-induced renal glomerular inflammation." Biochemical Journal 422, no. 3 (2009): 563–70. http://dx.doi.org/10.1042/bj20090420.

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The pro-inflammatory chemokine CCL2 [chemokine (Cys-Cys motif) ligand 2; also known as MCP-1 (monocyte chemotactic protein-1)] is up-regulated in the glomerular compartment during the early phase of LPS (lipopolysaccharide)-induced nephritis. This up-regulation also occurs in cultured MCs (mesangial cells) and is more pronounced in MCs lacking the PGE2 (prostaglandin E2) receptor EP2 or in MCs treated with a prostaglandin EP4 receptor antagonist. To examine a possible feedback mechanism of EP receptor stimulation on CCL2 expression, we used an in vitro model of MCs with down-regulated EP recep
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BEK, MARTIN, ROLF NÜSING, PASCAL KOWARK, ANNA HENGER, PETER MUNDEL, and HERMANN PAVENSTÄDT. "Characterization of Prostanoid Receptors in Podocytes." Journal of the American Society of Nephrology 10, no. 10 (1999): 2084–93. http://dx.doi.org/10.1681/asn.v10102084.

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Abstract. Prostaglandins participate in the regulation of important glomerular functions and are involved in the pathogenesis of glomerular diseases. This study investigates the influence of prostaglandins on membrane voltage, ion conductances, cAMP accumulation, and cytosolic calcium activity ([Ca2+]i) in differentiated podocytes. Prostaglandin E2 (PGE2) caused a concentration-dependent depolarization and an increase of the whole cell conductance in podocytes (EC50 ≈ 50 nM). Compared with PGE2, the EP2/EP3/EP4 receptor agonist 11-deoxy-PGE1 caused an equipotent depolarization, whereas the DP
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Imig, John D., Matthew D. Breyer, and Richard M. Breyer. "Contribution of prostaglandin EP2 receptors to renal microvascular reactivity in mice." American Journal of Physiology-Renal Physiology 283, no. 3 (2002): F415—F422. http://dx.doi.org/10.1152/ajprenal.00351.2001.

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The present studies were performed to determine the contribution of EP2 receptors to renal hemodynamics by examining afferent arteriolar responses to PGE2, butaprost, sulprostone, and endothelin-1 in EP2 receptor-deficient male mice (EP2−/−). Afferent arteriolar diameters averaged 17.8 ± 0.8 μm in wild-type (EP2+/+) mice and 16.7 ± 0.7 μm in EP2−/− mice at a renal perfusion pressure of 100 mmHg. Vessels from both groups of mice responded to norepinephrine (0.5 μM) with similar 17–19% decreases in diameter. Diameters of norepinephrine-preconstricted afferent arterioles increased by 7 ± 2 and 20
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Hayashi, Yoshinori, Saori Morinaga, Xia Liu, et al. "An EP2 Agonist Facilitates NMDA-Induced Outward Currents and Inhibits Dendritic Beading through Activation of BK Channels in Mouse Cortical Neurons." Mediators of Inflammation 2016 (2016): 1–9. http://dx.doi.org/10.1155/2016/5079597.

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Prostaglandin E2(PGE2), a major metabolite of arachidonic acid produced by cyclooxygenase pathways, exerts its bioactive responses by activating four E-prostanoid receptor subtypes, EP1, EP2, EP3, and EP4. PGE2enables modulatingN-methyl-D-aspartate (NMDA) receptor-mediated responses. However, the effect of E-prostanoid receptor agonists on large-conductance Ca2+-activated K+(BK) channels, which are functionally coupled with NMDA receptors, remains unclear. Here, we showed that EP2 receptor-mediated signaling pathways increased NMDA-induced outward currents (INMDA-OUT), which are associated wit
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Tilley, Stephen L., John M. Hartney, Christopher J. Erikson, et al. "Receptors and pathways mediating the effects of prostaglandin E2 on airway tone." American Journal of Physiology-Lung Cellular and Molecular Physiology 284, no. 4 (2003): L599—L606. http://dx.doi.org/10.1152/ajplung.00324.2002.

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Prostaglandin E2(PGE2) has complex effects on airway tone, and the existence of four PGE2 [E-prostanoid (EP)] receptors, each with distinct signaling characteristics, has provided a possible explanation for the seemingly contradictory actions of this lipid mediator. To identify the receptors mediating the actions of PGE2 on bronchomotor tone, we examined its effects on the airways of wild-type and EP receptor-deficient mice. In conscious mice the administration of PGE2 increased airway responsiveness primarily through the EP1 receptor, although on certain genetic backgrounds a contribution of
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Breyer, M. D., L. Davis, H. R. Jacobson, and R. M. Breyer. "Differential localization of prostaglandin E receptor subtypes in human kidney." American Journal of Physiology-Renal Physiology 270, no. 5 (1996): F912—F918. http://dx.doi.org/10.1152/ajprenal.1996.270.5.f912.

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Four prostaglandin E2 (PGE2) receptors designated EP1, EP2, EP3, and EP4 have been pharmacologically identified, cloned, and sequenced. The present studies determined the intrarenal distribution of these EP-receptor subtypes in human kidney using in situ hybridization with riboprobes for the human EP receptors. mRNA for the phosphatidylinositol hydrolysis-coupled EP receptor was highly expressed in cortical, outer medullary, and inner medullary collecting duct. RNA for the Gi-coupled EP3 receptor was primarily expressed in the cortical and outer medullary collecting duct, as well as in the med
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Schweda, Frank, Jürgen Klar, Shuh Narumiya, Rolf M. Nüsing, and Armin Kurtz. "Stimulation of renin release by prostaglandin E2 is mediated by EP2 and EP4 receptors in mouse kidneys." American Journal of Physiology-Renal Physiology 287, no. 3 (2004): F427—F433. http://dx.doi.org/10.1152/ajprenal.00072.2004.

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PGE2 is a potent stimulator of renin release. So far, the contribution of each of the four PGE2 receptor subtypes (EP1–EP4) in the regulation of renin release has not been characterized. Therefore, we investigated the effects PGE2 on renin secretion rates (RSR) from isolated, perfused kidneys of EP1−/−, EP2−/−, EP3−/−, EP4−/−, and wild-type mice. PGE2 concentration dependently stimulated RSR from kidneys of all four knockout strains with a threshold concentration of 1 nM in EP1−/−, EP2−/−, EP3−/−, and wild-type mice, whereas the threshold concentration was shifted to 10 nM in EP4−/− mice. More
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Jadhav, Vikram, Anthony Jabre, Shinn-Zong Lin, and Tony Jer-Fu Lee. "EP1- and EP3-Receptors Mediate Prostaglandin E2–Induced Constriction of Porcine Large Cerebral Arteries." Journal of Cerebral Blood Flow & Metabolism 24, no. 12 (2004): 1305–16. http://dx.doi.org/10.1097/01.wcb.0000139446.61789.14.

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Prostaglandin E2 (PGE2) has been shown to dilate and constrict the systemic vascular beds, including cerebral vessels. The exact mechanism of PGE2-induced cerebral vasoconstriction, however, is less clarified. The authors' preliminary studies showed that PGE2 exclusively constricted the adult porcine basilar arteries. The present study, therefore, was designed to examine the receptor mechanisms involved in PGE2-induced constriction of large cerebral arteries in the adult pig. Results from an in vitro tissue-bath study indicated that PGE2 and its agonists 17-phenyl trinor PGE2 (17-PGE2), sulpro
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Dissertations / Theses on the topic "Prostaglandin E receptor EP2"

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Lee, Jinju. "IL-23 generates pathogenic Th17 cells by triggering T cell-intrinsic prostaglandin E2-EP2/4 signaling." Kyoto University, 2018. http://hdl.handle.net/2433/235123.

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Sonoshita, Masahiro. "Acceleration of intestinal polyposis through prostaglandin receptor EP2 in Apc[Δ716] knockout mice". Kyoto University, 2004. http://hdl.handle.net/2433/147468.

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Seno, Hiroshi. "Cyclooxygenase 2- and prostaglandin E2 receptor EP2-dependent angiogenesis in ApcΔ716 mouse intestinal polyps". Kyoto University, 2002. http://hdl.handle.net/2433/149346.

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Inazumi, Tomoaki. "Roles of Prostaglandin EP4 Receptor in Adipocytes." 京都大学 (Kyoto University), 2014. http://hdl.handle.net/2433/188727.

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Soontrapa, Kitipong. "Prostaglandin E2-prostaglandin E receptor subtype 4 (EP4) signaling mediates UV irradiation-induced systemic immunosuppression." Kyoto University, 2013. http://hdl.handle.net/2433/170077.

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Chandramouli, Anupama. "The Role of Prostaglandin E2/EP4 Prostanoid Receptor Signaling in Colorectal Carcinogenesis." Diss., The University of Arizona, 2009. http://hdl.handle.net/10150/195443.

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Colorectal cancer, among other tumors, is characterized by elevated levels of prostaglandins due to the up-regulation of cyclooxygenase -2 (COX-2), a key enzyme in the eicosanoid biosynthesis pathway. Prostaglandin E2 (PGE2) is an important prostaglandin that exerts its biological function via four transmembrane G protein coupled receptors (EP1-4), among which the EP4 receptor is the most important. The relevance of EP4 receptor to the carcinogenic process and the consequences of its interaction with PGE2 were explored in this dissertation.Despite the importance of the EP4 receptor in colon ca
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Furuyashiki, Miyako. "Facilitation of Th1-mediated immune response by prostaglandin E receptor EP1." Kyoto University, 2009. http://hdl.handle.net/2433/126454.

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Kunikata, Tomonori. "Suppression of allergic inflammation by the prostaglandin E receptor subtype EP3." Kyoto University, 2005. http://hdl.handle.net/2433/144482.

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Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(医学)<br>甲第11827号<br>医博第2903号<br>新制||医||906(附属図書館)<br>23587<br>UT51-2005-K493<br>京都大学大学院医学研究科脳統御医科学系専攻<br>(主査)教授 坂口 志文, 教授 湊 長博, 教授 宮地 良樹<br>学位規則第4条第1項該当
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Peng, Lin [Verfasser], and Udo [Akademischer Betreuer] Jeschke. "The role of G-protein coupled prostaglandin E2 receptors (EP2 and EP3) in unexplained recurrent miscarriage and cervical cancer / Lin Peng ; Betreuer: Udo Jeschke." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2021. http://d-nb.info/1239049366/34.

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Hori, Ryusuke. "PROSTAGLANDIN E RECEPTOR SUBTYPE EP4 AGONIST PROTECTS COCHLEAE AGAINST NOISE-INDUCED TRAUMA." Kyoto University, 2010. http://hdl.handle.net/2433/120543.

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Books on the topic "Prostaglandin E receptor EP2"

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Houillier, Pascal. Magnesium homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0027.

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Magnesium is critically important in the process of energy release. Although most magnesium is stored outside the extracellular fluid compartment, the regulated concentration appears in blood. Urinary magnesium excretion can decrease rapidly to low values when magnesium entry rate into the extracellular fluid volume is low, which has several important implications: cell and bone magnesium do not play a major role in the defence of blood magnesium concentration; while a major role is played by the kidney and especially the renal tubule, which adapts to match the urinary magnesium excretion and
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Book chapters on the topic "Prostaglandin E receptor EP2"

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Luo, Xu, and Zongyi Xie. "Prostaglandin E2 Receptor EP2 Subtype." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101753.

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Luo, Xu, and Zongyi Xie. "Prostaglandin E2 Receptor EP2 Subtype." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101753-1.

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Breyer, Richard M., Christopher R. J. Kennedy, Yahua Zhang, Youfei Guan, and Matthew D. Breyer. "Targeted gene disruption of the prostaglandin e2 ep2 receptor." In Advances in Experimental Medicine and Biology. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0193-0_49.

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Nantel, Francois, Danielle Denis, Simon Wong, et al. "Altered Bronchodilation and Pulmonary Inflammation in Prostanoid EP2 Receptor Knockout Mice." In Advances in Prostaglandin and Leukotriene Research. Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9721-0_18.

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Ortiz-Vega, Sara, and Barrie Ashby. "Human Prostacyclin Receptor: Cloning and Co-Expression with EP3 Prostaglandin Receptor." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1810-9_50.

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Meyer, Jutta, Thomas Hohlfeld, and Karsten Schrör. "Characterization of the Prostaglandin EP3-Receptor from Porcine Heart." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1810-9_24.

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Breyer, Richard M., Ronald B. Emeson, Linda S. Davis, and Matthew D. Breyer. "Structure and Localization of the Rabbit Prostaglandin EP3 Receptor." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5325-0_38.

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Sakuma, Toshiki, Toru Akaike, and Susumu Minamisawa. "Prostaglandin E2 Receptor EP4 Inhibition Constricts the Rat Ductus Arteriosus." In Molecular Mechanism of Congenital Heart Disease and Pulmonary Hypertension. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1185-1_42.

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Fedyk, Eric R., Sarah G. Harris, Josue Padilla, and Richard P. Phipps. "Prostaglandin Receptors of the EP2 and EP4 Subtypes Regulate B Lymphocyte Activation and Differentiation to IGE-Secreting Cells." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1810-9_31.

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Khatibi, Nikan H., Vikram Jadhav, Brenden Matus, et al. "Prostaglandin E2 EP1 Receptor Inhibition Fails to Provide Neuroprotection in Surgically Induced Brain-Injured Mice." In Intracerebral Hemorrhage Research. Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0693-8_46.

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Conference papers on the topic "Prostaglandin E receptor EP2"

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Park, Joo Hun, Shunichiro Iwasawa, Jun Ikari, et al. "Prostaglandin E Stimulates Lung Fibroblasts Hepatocyte Growth Factor Release Through The EP2 Receptor." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1924.

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Togo, S., O. Holz, X. Wang, et al. "Endogenous Prostaglandin E 2 in COPD Lung Fibroblast Inhibits Platelet-Derived Growth Factor-Stimulated Cell Migration through a Prostaglandin E Receptor EP2 and 4." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2947.

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Reader, Jocelyn C., Xinrong Ma, Namita Kundu, Olga Goloubeva, and Amy Fulton. "Abstract 3250: Prostaglandin E2 receptor EP1 suppresses breast cancer metastasis." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3250.

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Peng, L., Y. Ye, H. Yang, et al. "Trophoblast derived prostaglandin E2 receptor 2 (EP2) is downregulated in recurrent miscarriage and regulates cell proliferation and inflammatory cytokines in vitro." In Kongressabstracts zur Tagung 2020 der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe (DGGG). © 2020. Thieme. All rights reserved., 2020. http://dx.doi.org/10.1055/s-0040-1717686.

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Shin, Vivian Y., Man T. Siu, John C. Ho, Isabella Cheuk, Jiawei Chen, and Ava Kwong. "Abstract P2-07-07: Prostaglandin E receptor 2 (EP2) regulates breast cancer stem-cell like property and promotes epithelial-mesenchymal transition." In Thirty-Seventh Annual CTRC-AACR San Antonio Breast Cancer Symposium; December 9-13, 2014; San Antonio, TX. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.sabcs14-p2-07-07.

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Aso, Hiromichi, Satoru Ito, Akemi Mori, et al. "Prostaglandin E2 Inhibits Platelet-Derived Growth Factor-Induced Cell Migration Via EP2 And EP4 Receptors In Human Airway Smooth Muscle Cells." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2420.

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Nonomura, Kazuhiko, Katsuyoshi Miyama, Kiyoshi Kanazawa, et al. "Abstract 893: Anticancer effect and mechanism of prostaglandin E2 receptor EP4 antagonist." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-893.

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Reader, Joceyln C., Paul Staats, Olga Goloubeva, et al. "Abstract 5169: Functional studies of the prostaglandin E2 receptor EP4 in ovarian cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5169.

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Reader, Jocelyn C., Xinrong Ma, Namita Kundu, Olga Goloubeva, Joseph Kao, and Amy Fulton. "Abstract 1456: Prostaglandin E2 receptor EP1 as a metastasis suppressor in breast cancer." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-1456.

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Ma, Xinrong, Namita Kundu, Peter Collin, and Amy M. Fulton. "Abstract 609: Frondoside antagonizes the prostaglandin E receptor EP4 and inhibits breast tumor metastasis." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-609.

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