Relevant bibliographies by topics / Prostaglandines – Synthèse

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Prostaglandines – Synthèse'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Prostaglandines – Synthèse"

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Yang, Ling, Xu Han, Leying Zhang, Ning Li, Zimo Zhao, and Jiachen Bai. "Changes in expression of prostaglandin synthase in ovine liver during early pregnancy." Canadian Journal of Animal Science 100, no. 3 (September 1, 2020): 432–39. http://dx.doi.org/10.1139/cjas-2019-0171.

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Liver can function as part of the innate and adaptive immune systems. We hypothesize that prostaglandins participate in the regulation of hepatic immune function during early pregnancy in sheep. The objective of this study was to elucidate expression of prostaglandin synthase in ovine liver during early pregnancy. Ovine livers were sampled on day 16 of the estrous cycle, and days 13, 16, and 25 of pregnancy, and the expression of prostaglandin synthases, including prostaglandin-endoperoxide synthase 1 (PTGS1), PTGS2, prostaglandin E synthase (PTGES), and aldo-keto reductase family 1, member B1, a prostaglandin F synthase (PGFS), were detected by quantitative real-time polymerase chain reaction, Western blot, and immunohistochemistry analysis. There were increases in the expression of mRNA and the proteins of PTGS2, PTGES, and PGFS in the livers during early pregnancy, but PTGS1 was decreased in the pregnant ewes. The PGFS protein was limited to the hepatocytes and the endothelial cells of the proper hepatic arteries and hepatic portal veins. In summary, the upregulation of PTGS2, PTGES, and PGFS and downregulation of PTGS1 may be involved in the maternal hepatic immune adjustment during early pregnancy in sheep.
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Berthou, F., F. Ceppo, M. Cormont, and J. F. Tanti. "P2096 Implication de la kinase Tpl2 dans l’expression de COX-2 et la synthèse des prostaglandines induites par les cytokines inflammatoires dans les adipocytes." Diabetes & Metabolism 39 (March 2013): A90—A91. http://dx.doi.org/10.1016/s1262-3636(13)72006-8.

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Tsuchida, Keiichiro, Takae Ibuki, and Kiyoshi Matsumura. "Bromoenol Lactone, an Inhibitor of Calcium-Independent Phospholipase A2, Suppresses Carrageenan-Induced Prostaglandin Production and Hyperalgesia in Rat Hind Paw." Mediators of Inflammation 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/605727.

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Prostaglandin (PG) E2and PGI2are essential to hyperalgesia in inflammatory tissues. These prostaglandins are produced from arachidonic acid, which is cleaved from membrane phospholipids by the action of phospholipase A2(PLA2). Which isozyme of PLA2is responsible for the cleavage of arachidonic acid and the production of prostaglandins essential to inflammation-induced hyperalgesia is not clear. In this study, we examined the effects of two PLA2isozyme-specific inhibitors on carrageenan-induced production of PGE2and PGI2in rat hind paw and behavioral nociceptive response to radiant heat. Local administration of bromoenol lactone (BEL), an inhibitor of calcium-independent PLA2(iPLA2), significantly reduced carrageenan-induced elevation of prostaglandins in the inflamed foot pad 3 h after injection. It also ameliorated the hyperalgesic response between 1 h and 3 h after carrageenan injection. On the other hand, AACOCF3, an inhibitor of cytosolic PLA2, suppressed neither prostaglandin production nor the hyperalgesic response. BEL did not suppress the mRNA levels of iPLA2β, iPLA2γ, cyclooxygenase-2, microsomal prostaglandin E synthase, prostaglandin I synthase, or proinflammatory cytokines in the inflamed foot pad, indicating that BEL did not suppress inflammation itself. These results suggest that iPLA2is involved in the production of prostaglandins and hyperalgesia at the inflammatory loci.
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Gómez-Foix, A. M., J. E. Rodriguez-Gil, J. J. Guinovart, and F. Bosch. "Prostaglandins E2 and F2α affect glycogen synthase and phosphorylase in isolated hepatocytes." Biochemical Journal 261, no. 1 (July 1, 1989): 93–97. http://dx.doi.org/10.1042/bj2610093.

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Prostaglandin E2 (PGE2) and prostaglandin F2 alpha (PGF2 alpha) inactivated glycogen synthase and activated glycogen phosphorylase in rat hepatocytes in a dose- and time-dependent manner. These effects were dependent on the presence of Ca2+ in the incubation medium. When glycogen synthase was immunoprecipitated from cells incubated with [32P]Pi and then treated with PGE2 or PGF2 alpha, there was increased phosphorylation of the 88 kDa subunit of the enzyme. This phosphorylation affected two CNBr fragments of the glycogen synthase, CB-1 and CB-2, the same fragments that are phosphorylated by different glycogenolytic hormones. No phosphorylation of glycogen synthase by prostaglandins was observed in the absence of Ca2+. Thus the effect of PGE2 and PGF2 alpha on these glycogen-metabolizing enzymes supports a role for regulation by prostaglandins of glucose metabolism in parenchymal liver cells.
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Laurent, Fabrice, Martin F. Kagnoff, Tor C. Savidge, Muriel Naciri, and Lars Eckmann. "Human Intestinal Epithelial Cells Respond toCryptosporidium parvum Infection with Increased Prostaglandin H Synthase 2 Expression and Prostaglandin E2and F2α Production." Infection and Immunity 66, no. 4 (April 1, 1998): 1787–90. http://dx.doi.org/10.1128/iai.66.4.1787-1790.1998.

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ABSTRACT Cryptosporidium parvum is an important cause of diarrhea in humans and several animal species. Prostaglandins play a central role in regulating intestinal fluid secretion in animal models of cryptosporidiosis, but their cellular sources and mechanisms of induction are unclear. Here, we show that C. parvuminfection directly activates prostaglandin H synthase 2 expression and prostaglandin E2 and F2α production in human intestinal epithelial cells.
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Mitchell, MD, RJ Romero, SS Edwin, and MS Trautman. "Prostaglandins and parturition." Reproduction, Fertility and Development 7, no. 3 (1995): 623. http://dx.doi.org/10.1071/rd9950623.

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It seems likely that prostaglandins play a significant part in the mechanisms of parturition both at term and preterm. Concentrations of prostaglandins are increased in the blood, urine and amniotic fluid during labour. There are differences in the concentrations of prostaglandins in amniotic fluid from the forebag and hindbag. Nevertheless, if liquor is sampled only from the hindbag a highly significant increase in prostaglandin concentrations occurs throughout labour. Furthermore, we now have evidence that prostaglandin concentrations in amniotic fluid increase before the onset of labour. Prostaglandins are synthesized by uterine tissues and increased rates of production occur during labour. The amnion, chorion and decidua all contain mRNA for the newly-discovered inducible form of prostaglandin H synthase (PGHS-2) as well as mRNA for the constitutive form (PGHS-1). Using the reverse transcription polymerase chain reaction (RT-PCR) both mRNAs can be detected during late pregnancy whether women are in labour or not. PGHS-2 protein is detected by Western blot analysis in cells derived from all three tissues. There is regulation of PGHS-2 protein amounts by cytokines, phorbol esters and growth factors. For example, in amnion cells interleukin-1 beta induces a rapid increase in PGHS-2 mRNA levels followed by a decrease to undetectable levels within 4 h of treatment; PGHS-2 protein amounts are also elevated by this treatment. Administration of prostaglandins will induce labour and delivery, whereas inhibition of prostaglandin biosynthesis will delay labour and delivery. Hence, increased prostaglandin production is likely to be a key determinant of the onset and progression of the parturient process.
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Tang, Eva H. C., and Paul M. Vanhoutte. "Gene expression changes of prostanoid synthases in endothelial cells and prostanoid receptors in vascular smooth muscle cells caused by aging and hypertension." Physiological Genomics 32, no. 3 (February 2008): 409–18. http://dx.doi.org/10.1152/physiolgenomics.00136.2007.

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The present study was designed to assess whether or not changes in genomic expression of cyclooxygenases (COX-1, COX-2), endothelial nitric oxide synthase (eNOS), and prostanoid synthases in the endothelium and of prostanoid receptors in vascular smooth muscle contribute to the occurrence of endothelium-dependent contractions during aging and hypertension. Gene expression was quantified by real-time PCR using isolated endothelial cells and smooth muscle cells (SMC) from the aorta of Wistar-Kyoto and spontaneously hypertensive rats. Genes for all known prostanoid synthases and receptors were present in endothelial cells and SMC, respectively. Aging caused overexpression of eNOS, COX-1, COX-2, thromboxane synthase, hematopoietic-type prostaglandin D synthase, membrane prostaglandin E synthase-2, and prostaglandin F synthase in endothelial cells and COX-1 and prostaglandin E2 (EP)4 receptors in SMC. Hypertension augmented the expression of COX-1, prostacyclin synthase, thromboxane synthase, and hematopoietic-type prostaglandin D synthase in endothelial cells and prostaglandin D2 (DP), EP3, and EP4 receptors in SMC. The increase in genomic expression of endothelial COX-1 explains why in aging and hypertension the endothelium has greater propensity to release cyclooxygenase-derived vasoconstrictive prostanoids. The expression of prostacyclin synthase was by far the most abundant, explaining why the majority of the COX-1-derived endoperoxides are transformed into prostacyclin, substantiating the role of prostacyclin as an endothelium-derived contracting factor. The expression of thromboxane synthase was increased in the cells of aging or hypertensive rats, explaining why the prostanoid can contribute to endothelium-dependent contractions. It is uncertain whether the gene modifications caused by aging and hypertension directly contribute to endothelium-dependent contractions or rather to vascular aging and the vascular complications of the hypertensive process.
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Chopra, Sahil, Paolo Giovanelli, Perla Abigail Alvarado-Vazquez, Sara Alonso, Minkyung Song, Tito A. Sandoval, Chang-Suk Chae, et al. "IRE1α–XBP1 signaling in leukocytes controls prostaglandin biosynthesis and pain." Science 365, no. 6450 (July 18, 2019): eaau6499. http://dx.doi.org/10.1126/science.aau6499.

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Inositol-requiring enzyme 1[α] (IRE1[α])–X-box binding protein spliced (XBP1) signaling maintains endoplasmic reticulum (ER) homeostasis while controlling immunometabolic processes. Yet, the physiological consequences of IRE1α–XBP1 activation in leukocytes remain unexplored. We found that induction of prostaglandin-endoperoxide synthase 2 (Ptgs2/Cox-2) and prostaglandin E synthase (Ptges/mPGES-1) was compromised in IRE1α-deficient myeloid cells undergoing ER stress or stimulated through pattern recognition receptors. Inducible biosynthesis of prostaglandins, including the pro-algesic mediator prostaglandin E2 (PGE2), was decreased in myeloid cells that lack IRE1α or XBP1 but not other ER stress sensors. Functional XBP1 transactivated the human PTGS2 and PTGES genes to enable optimal PGE2 production. Mice that lack IRE1α–XBP1 in leukocytes, or that were treated with IRE1α inhibitors, demonstrated reduced pain behaviors in PGE2-dependent models of pain. Thus, IRE1α–XBP1 is a mediator of prostaglandin biosynthesis and a potential target to control pain.
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Rice, G. E., M. H. Wong, and G. D. Thorburn. "Gestational changes in prostaglandin synthase activity of ovine cotyledonary microsomes." Journal of Endocrinology 118, no. 2 (August 1988): 265–70. http://dx.doi.org/10.1677/joe.0.1180265.

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ABSTRACT The capacity of cotyledonary microsomes, prepared from pregnant ewes (20–145 days of gestation), to metabolize exogenous arachidonic acid was quantified using a radiolabel technique. During gestation, the capacity of microsomes to metabolize arachidonic acid increased 25-fold, from 0·36±0·06μmol arachidonic acid/incubation (n = 8) at <100 days of gestation to 9·06±1 ·02μmol arachidonic acid/incubation at 130–145 days of gestation (n = 5; P<0·05). Arachidonic acid was metabolized to prostaglandin E2 and F2α, as determined by thin-layer chromatography and reverse-phase high performance liquid chromatography. The profile of prostaglandins synthesized by cotyledonary microsomes did not change throughout gestation. These data suggest that the increase in cotyledonary prostaglandin synthesis that occurs during late gestation and at term may reflect an increase in the tissue content of prostaglandin H2 synthase. J. Endocr. (1988) 118, 265–270
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Li, Ke, Jing Zhao, Mingxuan Wang, Lingzhi Niu, Yuanping Wang, Yanxia Li, and Yajuan Zheng. "The Roles of Various Prostaglandins in Fibrosis: A Review." Biomolecules 11, no. 6 (May 24, 2021): 789. http://dx.doi.org/10.3390/biom11060789.

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Organ fibrosis is a common pathological result of various chronic diseases with multiple causes. Fibrosis is characterized by the excessive deposition of extracellular matrix and eventually leads to the destruction of the tissue structure and impaired organ function. Prostaglandins are produced by arachidonic acid through cyclooxygenases and various prostaglandin-specific synthases. Prostaglandins bind to homologous receptors on adjacent tissue cells in an autocrine or paracrine manner and participate in the regulation of a series of physiological or pathological processes, including fibrosis. This review summarizes the properties, synthesis, and degradation of various prostaglandins, as well as the roles of these prostaglandins and their receptors in fibrosis in multiple models to reveal the clinical significance of prostaglandins and their receptors in the treatment of fibrosis.
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Dissertations / Theses on the topic "Prostaglandines – Synthèse"

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Alphand, Véronique. "Synthèse de lactones optiquement activés par réaction de Baeyer-Villiger microbiologique." Aix-Marseille 2, 1990. http://www.theses.fr/1990AIX22020.

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Nous avons etudie la possibilite de realiser, a partir de diverses cetones, des reactions de baeyer-villiger asymetriques par voie microbiologique. Une etude exploratoire sur plusieurs microorganismes nous a permis de selectionner deux bacteries, a. Calcoaceticus ncib 9871 et acinetobacter td 63. La premiere, dotee d'une lactone hydrolase tres active, est utilisee en presence d'un inhibiteur; la seconde est naturellement depourvue de cette enzyme. Un certain nombre de cetones, choisies soit pour leur interet dans le domaine des aromes (cetones cyclopentaniques et cyclohexaniques), soit comme precurseurs de synthons chiraux (cetones de structure bicyclo 2. 2. 1 heptane, bicyclo 3. 2. 0 heptane et bicyclo 4. 2. 0 octane), ont ete testees avec succes. La regio- et l'enantioselectivite de ces biotransformations dependent de la structure des cetones de depart. Dans la plupart des cas, cependant, ces reactions sont hautement regio- et enantioselectives. De nombreuses lactones sont obtenues avec de bons rendements et des exces enantiomeriques eleves
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Boucher-Kovalik, Sofia. "Caractérisation de l'expression des enzymes de biosynthèse des prostaglandines dans des lignées cellulaires endométriales immortalisées." Thesis, Université Laval, 2010. http://www.theses.ulaval.ca/2010/27822/27822.pdf.

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Das, Saibal. "Synthèse de prostaglandines et nouvelles utilisations de liquides ioniques comme solvants." Rennes 1, 2007. http://www.theses.fr/2007REN1S044.

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Cette thèse est divisée en 5 parties: - La première est une étude bibliographique exhaustive des synthèses de prostaglandines réalisées au cours des 13 dernières années. - Dans le chapitre II on montre l'intérêt des liquides ioniques pour effectuer une réaction multicomposants, ce qui a permis d'effectuer la première synthèse totale du 12-épi-Carboprost. - Dans le chapitre III, l'utilisation d'une réaction de cyclisation radicalaire intramoléculaire a permis la synthèse totale du Carboprost et de quelques nouvelles prostaglandines. - Dans le chapitre IV nous montrons comment l'utilisation d'une lipase dans le liquide ionique [C8mim][PF6] offre une solution particulièrement efficace au problème du dédoublement de l'intermédiaire clé des deux approches précédentes - Dans le chapitre V nous avons montré comment les liquides ioniques peuvent constituer des solvants alternatifs très attractifs pour les réactions de mono- et gem-difluoration utilisant le DAST comme réactif de fluoration
This thesis is divided in 5 parts: - The first chapter is an extensive bibliographical study of the synthesis of Prostaglandins, performed during the last 13 years. - In the chapter II is reported the use of ionic liquids to perform a multicomponent reaction leading to the first total synthesis of the 12-epiCarboprost. In the chapter III an intramolecular radical cyclisation is used for the preparation of Carboprost and several other prostaglandins. In Chapter IV we demonstrate how the use of a lipase in the [C8mim][PF6] ionic liquid offers a very efficient solution to the problem of the resolution for the key intermediate of all previous synthesis. In chapter V, we demonstrate how the ionic liquids are very attractive alternative solvents for the mono- and gem-difluorination reactions using DAST as the fluorinating agent
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Moulard-Ravoisier, Thérèse. "Influence de médicaments à fort pouvoir électrodonneur sur les systèmes oxydasiques et peroxydasiques : conséquences sur l'hormonogénèse thyroi͏̈dienne et sur la synthèse des prostaglandines." Limoges, 1990. http://www.theses.fr/1990LIMO301B.

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Guy, Alexandre. "Synthèse biomimétique de la 15-F2t-IsoP. Synthèse de l'ent 5,6-dihydro-2,3-dinor-15-F2t-IsoP." Montpellier 2, 1998. http://www.theses.fr/1998MON20183.

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Les isoprostanes (isops), isomeres des prostaglandines, sont une nouvelle classe de produits naturels decouvertes dans les annees 1990. Les isops sont naturellement formees, lors de l'attaque oxydante de l'acide arachidonique (aa), par reaction de cyclisation radicalaire intramoleculaire avec introduction de trois atomes d'oxygene. La 15-f#2#t-isop, la plus abondante, possede de puissantes activites biologiques et est actuellement consideree comme un bon marqueur de la peroxydation lipidique. A cause de l'interet porte a ce compose, nous en avons developpe la synthese totale. La strategie retenue est une strategie biomimetique. En effet, l'etape cle de cette synthese utilise le mecanisme suppose de la formation in vivo des isops au moyen d'une cyclisation radicalaire de type 1,5,7-octadienyle d'un motif a 20 atomes de carbone mimant l'aa. La synthese biomimetique de la 15-f#2#t-isop a permis de confirmer la biosynthese des isops et sa formation possible a partir de l'aa esterifie. Nous avons aussi aborde la synthese de l'enantiomere d'un des metabolites urinaires de la 15-f#2#t-isop, l'ent-15(rs)-5,6-dihydro-2,3-dinor-15-f#2#t-isop. L'approche retrosynthetique est basee sur la formation d'un cycle pentanique polysubstitue de configuration adequate sur lequel sont fixes les chaines laterales du metabolite. L'obtention de ces deux molecules, en particulier la 15-f#2#t-isop, pouvant etre marquee au deuterium ou au tritium, presente un interet majeur pour des etudes biologiques. D'autre part, ces deux isops peuvent etre un index de peroxydation lipidique representatif des attaques oxydantes in vivo.
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Kuhn, Cyrille. "Nouvelles voies de synthèse énantiosélective de prostanoi͏̈des à activité antitumorale." Paris 5, 1996. http://www.theses.fr/1996PA05P622.

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Roulland, Emmanuel. "Application de la réaction de métathèse à la synthèse d'analogues de prostaglandines antitumorales." Paris 5, 2000. http://www.theses.fr/2000PA05P623.

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Henry, Olivier. "Synthèses totales de métabolites de la 15-F2t-Isoprostane." Montpellier 2, 2002. http://www.theses.fr/2002MON20009.

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Rodriguez, Ana. "Synthèse totale des prostaglandines, leucotriènes, lipoxines, acides hydroxyeicosatétraènoïques et de leurs analogues deutérés." Paris 7, 1999. http://www.theses.fr/1999PA077220.

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Perrin, Véronique. "Synthèse et caractérisation pharmacologique de nouveaux antagonistes potentiels des récepteurs du thromboxane A2." Lyon 1, 1996. http://www.theses.fr/1996LYO10314.

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Le thromboxane a#2 est un puissant constricteur des muscles lisses vasculaires et respiratoires ainsi qu'un agent de l'agregation plaquettaire. Il est implique dans des pathologies respiratoires, cardiovasculaires et renales. Compte tenu de l'etude des caracteristiques communes a de nombreux antagonistes connus, notre but etait de preparer des antagonistes des recepteurs du thromboxane a#2 possedant une structure 2-azanorbornane et ayant une fonction sulfone sur l'atome d'azote, ainsi que la chaine superieure, plus ou moins modifiee, des prostaglandines. Les differentes molecules obtenues ont ensuite ete soumises a des tests pharmacologiques in vivo sur les recepteurs vasculaires chez le rat conscient et in vitro sur les recepteurs plaquettaires chez le cobaye ; alors que les tests in vitro se sont averes en majorite negatifs, les tests in vivo ont permis de mettre en evidence une activite antagoniste sur les recepteurs vasculaires
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Books on the topic "Prostaglandines – Synthèse"

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Ragolia, Louis. Prostaglandin D2 synthase: A multitude of biological functions. Kerala, India: Research Signpost, 2007.

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Gyomorey, Sandor. Temporal expression of prostaglandin endoperoxide H synthase type 2 and prostaglandin receptors at birth. Ottawa: National Library of Canada, 1998.

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Yu, Denise. Regulation of prostaglandin H2 synthase type-2 enzyme by cortisol and progesterone. Ottawa: National Library of Canada, 1999.

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Cyclooxygenases: Methods and protocols. New York, N.Y: Humana Press, 2010.

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Harlan, John E. Solution structure and function of ligand interaction of prostaglandin H₂ synthase. 1993.

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A, Willoughby D., and Tomlinson Annette 1942-, eds. Inducible enzymes in the inflammatory response. Basel: Birkhaüser, 1999.

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Parman, Toufan. Role of embryonic prostaglandin H synthase-catalyzed free radical formation and reactive oxygen species-mediated macromolecular damage in chemical teratogenesis. 2001.

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Jeng, Winnie. Free radical determinants of endogenous and amphetamine-enhanced neurodegenerative disease: Prostaglandin H synthase-catalyzed free radical formation, reactive oxygen species-mediated oxidative DNA damage and glucose-6-phosphate dehydrogenase-catalyzed neuroprotection. 2004.

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Book chapters on the topic "Prostaglandines – Synthèse"

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Degen, Gisela H., Christoph Vogel, and Josef Abel. "Prostaglandin Synthases." In Enzyme Systems that Metabolise Drugs and Other Xenobiotics, 189–229. Chichester, UK: John Wiley & Sons, Ltd, 2002. http://dx.doi.org/10.1002/0470846305.ch6.

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Schomburg, Dietmar, and Dörte Stephan. "Prostaglandin-F synthase." In Enzyme Handbook 10, 123–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-57756-7_36.

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Schomburg, Dietmar, and Dörte Stephan. "Prostaglandin-endoperoxide synthase." In Enzyme Handbook, 739–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-57942-4_153.

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Watanabe, Kikuko, Toshiko Suzuki-Yamamoto, Mikio Nishizawa, Motonari Fukui, Kazunori Ishimura, and Seiji Ito. "Roles of Prostaglandin F Synthase." In Advances in Prostaglandin and Leukotriene Research, 73–76. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9721-0_13.

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Hickok, Noreen J., Mary Alosio, and Richard S. Bookman. "Prostaglandin Endoperoxide Synthase from Human Cell Line Lu-65." In Prostaglandins, Leukotrienes, and Lipoxins, 47–55. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4946-4_5.

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Shimokawa, Teruhiko, and William L. Smith. "Amino Acids Essential to Catalysis by Prostaglandin Endoperoxide Synthase." In Prostaglandins, Leukotrienes, Lipoxins, and PAF, 79–87. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-0727-1_9.

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Bailey, J. Martyn. "Translational Control of Prostaglandin Synthase by Growth Factors and Glucocorticoids." In Prostaglandins, Leukotrienes, Lipoxins, and PAF, 1–14. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-0727-1_1.

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Nakayama, Tomohiro, Masayoshi Soma, Duolikun Rehemudula, and Katsuo Kanmatsuse. "Mutation of the Human Prostacyclin Synthase Gene." In Advances in Prostaglandin and Leukotriene Research, 65–68. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9721-0_11.

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Herschman, Harvey R., Weilin Xie, and Srinivasta Reddy. "Function and Regulation of Prostaglandin Synthase 2." In Advances in Experimental Medicine and Biology, 3–8. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4793-8_1.

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Hayaishi, Osamu. "Prostaglandin D Synthase, β-Trace and Sleep." In Advances in Experimental Medicine and Biology, 347–50. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1810-9_74.

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Conference papers on the topic "Prostaglandines – Synthèse"

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Sheehan, S. J., A. B. Latif, S. R. Bibby, R. C. Kester, and S. M. Rajah. "THE RECOVERY OF ENDOTHELIAL CELL AND PLATELET PROSTAGLANDIN SYNTHESES AFTER ADMINISTRATION OF ASPIRIN AND INDOBUFEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643389.

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Suppression of endothelial prostacyclin (PGI2) by cyclooxygenase inhibition may contribute to the formation of neointimal hyperplasia. While aspirin (ASA) inhibits platelet function for several days, it returns to normal within 24 hours after indobufen (IDB), a reversible cyclooxygenase inhibitor, suggesting that prostacyclin may recover at an even faster rate. In a randomised, controlled study, recovery of platelet and endothelial cell prostaglandin synthesis was compared in 49 New Zealand white rabbits following indobufen (3.5 mg/kg) or aspirin (5 mg/kg). Prostacyclin in arterial incubates and platelet thromboxane A2 (TXA2) production in serum were measured by radioimmunoassay of their meta-bolities, 6Keto PGF1 and thromboxane B2. Platelet prostaglandin synthesis was also monitored by changes in serum malondiealdehyde (MDA).At one hour, both ASA and IDB significantly inhibited PGI2 (p<0.005). TXA2 (p<0.005), and MDA (p<0.02). After IDB, TXA2 and MDA levels returned to normal by 24 hours while inhibition continued in the ASA group (p<0.01). PGI2 synthesis following IDB recovered at 8 hours against 24 hours in the ASA group. We conclude that while both drugs inhibit platelet and endothelial prostaglandins, the earlier recovery of prostacyclin synthesis following indobufen may allow the endothelium to resume its physiological function.
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Fajt, Merritt L., Silvana Balzar, John B. Trudeau, Haizhen Hu, and Sally E. Wenzel. "Epithelial Hematopoietic Prostaglandin (PG) D Synthase Is Increased In Asthma." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1440.

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Woolbright, Benjamin Leeland, Jordan Barney, Van Schloegel Schloegel, Erika Abbott, and John Arthur Taylor. "Abstract 3867: Microsomal prostaglandin E synthase-1: A therapeutic target in bladder cancer." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-3867.

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Kock, Anna, Karin Larsson, Raouf Joan, Marina Korotkova, John-Inge Johnsen, Per-Johan Jakobsson, and Per Kogner. "Abstract 1632: Microsomal prostaglandin E synthase 1: a novel therapeutic target of neuroblastoma." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1632.

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Woolbright, Benjamin Leeland, Jordan Barney, Van Schloegel Schloegel, Erika Abbott, and John Arthur Taylor. "Abstract 3867: Microsomal prostaglandin E synthase-1: A therapeutic target in bladder cancer." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-3867.

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Tsai, Fu-Ming, Chang-Chieh Wu, Rong-Yaun Shyu, and Shun-Yuan Jiang. "Abstract 5384: Hrev107 enhances prostaglandin D2 synthase-mediated cancer cells suppression in the testis." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5384.

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Türk, Murat, Oğuz Köktürk, Zeynep Işıkdoğan, and Canan Demirtaş. "Urinary biomarkers for obstructive sleep apnea syndrome: Lipocalin type prostaglandin-D synthase and F2-isoprostane." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2535.

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Kock, Anna, Agnes Rasmuson, Marina Korotkova, Helena Idborg, John Inge Johnsen, Per-Johan Jakobsson, and Per Kogner. "Abstract 2746: Microsomal prostaglandin E2 synthase-1 may provide a novel specific therapeutic target in neuroblastoma." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2746.

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Gunji, Yoko, Tadashi Sato, Nobuhiro Kanaji, Amy Nelson, Jun Ikari, Shunichiro Iwasawa, JooHun Park, et al. "Microsomal Prostaglandin E Synthase-1 Is Induced In Lung Fibroblasts By IL-1b And Turns Over." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1922.

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Xiao, Lei, Magdalena Ornatowska, Hongmei Cao, Rui Yu, Guiqing Zhao, Ruxana T. Sadikot, and John W. Christman. "Lipopolysaccharide (LPS)-induced Expression Of Prostaglandin E Synthase-1 (PGES-1) Mediates The Late Phase Prostaglandin (PG) E2 Production In Mouse Bone Marrow-derived Macrophages (BMDM)." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1292.

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Reports on the topic "Prostaglandines – Synthèse"

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Fisher, Robert W. The Role of Prostaglandin G/H Synthase 2 in the Apoptosis of Human Mammary Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, June 1999. http://dx.doi.org/10.21236/ada373969.

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Fisher, Robert. The Role of Prostaglandin G/H Synthase 2 in the Apoptosis of Human Mammary Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, June 1998. http://dx.doi.org/10.21236/ada357964.

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You might also be interested in the extended bibliographies on the topic 'Prostaglandines – Synthèse' for particular source types:
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