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Relevant bibliographies by topics / Prostate epithelial and cancer markers

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Academic literature on the topic 'Prostate epithelial and cancer markers'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Prostate epithelial and cancer markers"

1

Tribian, L. S., M. Lennartz, N. C. Blessin, et al. "Diagnostic role and prognostic impact of PSAP immunohistochemistry: A tissue microarray study on 31,358 cancer tissues from 127 different tumor types." American Journal of Clinical Pathology 160, Supplement_1 (2023): S47—S48. http://dx.doi.org/10.1093/ajcp/aqad150.107.

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Abstract Introduction/Objective Prostatic specific acid phosphatase (PSAP) protein is produced in prostate epithelial cells and it is used as an immunohistochemical marker for prostate cancer. However, studies have reported PSAP expression to occur in various other tumor entities as well. Methods/Case Report Prostatic specific acid phosphatase (PSAP) protein is produced in prostate epithelial cells and it is used as an immunohistochemical marker for prostate cancer. However, studies have reported PSAP expression to occur in various other tumor entities as well. Results (if a Case Study enter N

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Duffy, M. J. "New Cancer Markers." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 26, no. 5 (1989): 379–87. http://dx.doi.org/10.1177/000456328902600501.

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In recent years many new and improved cancer markers have become available. From a clinical point of view, the most useful of the new markers include CA 19–9 for pancreatic adenocarcinoma, CA 125 for epithelial ovarian cancer, CA 15–3 for breast cancer, prostate specific antigen for prostatic adenocarcinoma, placental alkaline phosphatase for testicular seminomas and neuron-specific enolase for small cell carcinoma of lung. None of these new markers are specific for cancer. Furthermore, none are organ specific, except prostate specific antigen for prostatic tissue. The main application of thes

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3

Hess, Megan, Hanbing Song, Jessica Hicks, et al. "Abstract 5323: Inflammation drives lineage plasticity and a club cell-like phenotype in prostate epithelial cells." Cancer Research 84, no. 6_Supplement (2024): 5323. http://dx.doi.org/10.1158/1538-7445.am2024-5323.

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Abstract Background: Club cells are a type of bronchiolar epithelial cell that serve a protective and antimicrobial function in the lung. An epithelial cell population in adult human prostates was recently identified as sharing a similar transcriptomic profile and morphology to lung club cells. Club-like epithelial cells were identified in the prostatic urethra, collecting ducts, and rarely in the peripheral zone of normal organ donor prostates. However, we recently reported that cells expressing club cell markers (CP, LTF, MMP7, PIGR, SCGB1A1) are present in the peripheral zone of radical pro

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Zhong, Jian, and Jian-Quan Hou. "A novel retinoic acid chalcone reverses epithelial‑mesenchymal transition in prostate cancer cells." Bangladesh Journal of Pharmacology 10, no. 2 (2015): 288. http://dx.doi.org/10.3329/bjp.v10i2.22602.

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<p>The present study was performed to investigate the effect of retinoic acid fluoro chalcone (RAFC) on lipopolysaccharide (LPS) induced epithelial-mesenchymal transition (EMT) in PC3 and CWR22rv1 prostate cell lines. Lipo-polysaccharide (LPS) was used to induce epithelial-mesenchymal transition in prostate carcinoma cell lines. The results revealed that treatment of PC3 and CWR22rv1 cells with LPS resulted in significant changes in the morphological features of the EMT. The mesenchymal marker, vimentin expression was significantly increased whereas the expression level of E‑cadherin was

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Ryabov, Vladimir M., Mikhail M. Baryshev, Mikhail A. Voskresenskiy, and Boris V. Popov. "Early Cell Cultures from Prostate Cancer Tissue Express Tissue Specific Epithelial and Cancer Markers." International Journal of Molecular Sciences 24, no. 3 (2023): 2830. http://dx.doi.org/10.3390/ijms24032830.

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Prostate cancer (PCa) is a widespread oncological disease that proceeds in the indolent form in most patients. However, in some cases, the indolent form can transform into aggressive metastatic incurable cancer. The most important task of PCa diagnostics is to search for early markers that can be used for predicting the transition of indolent cancer into its aggressive form. Currently, there are two effective preclinical models to study PCa pathogenesis: patients derived xenografts (PDXs) and patients derived organoids (PDOs). Both models have limitations that restrict their use in research. I

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Olea-Flores, Monserrat, Juan C. Juárez-Cruz, Miriam D. Zuñiga-Eulogio, et al. "New Actors Driving the Epithelial–Mesenchymal Transition in Cancer: The Role of Leptin." Biomolecules 10, no. 12 (2020): 1676. http://dx.doi.org/10.3390/biom10121676.

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Leptin is a hormone secreted mainly by adipocytes; physiologically, it participates in the control of appetite and energy expenditure. However, it has also been linked to tumor progression in different epithelial cancers. In this review, we describe the effect of leptin on epithelial–mesenchymal transition (EMT) markers in different study models, including in vitro, in vivo, and patient studies and in various types of cancer, including breast, prostate, lung, and ovarian cancer. The different studies report that leptin promotes the expression of mesenchymal markers and a decrease in epithelial

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Khawaja, Hunain, and Cynthia Miranti. "Abstract B006: Identifying the molecular signature of bi-potent prostate epithelial cells through scRNA-seq." Cancer Research 83, no. 11_Supplement (2023): B006. http://dx.doi.org/10.1158/1538-7445.prca2023-b006.

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Abstract Introduction: Recent studies have shown that cells undergoing oncogenic processes usurp normal developmental pathways to escape regulators of unsolicited growth. Basal to luminal differentiation in the prostate serves as another cellular process that can go awry, the dysregulation of which can enable oncogenesis in the prostate. Studies have found bi-potent cells in prostate epithelium and speculate those to be cells of origin in prostate cancer. Methods and Results: Through single-cell RNA sequencing we investigated basal to luminal differentiation of normal human prostate epithelial

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Zhang, Yanting, Fei Cheng, and Lixia Li. "Exploring markers in nursing care of prostate cancer." Medicine 104, no. 4 (2025): e41357. https://doi.org/10.1097/md.0000000000041357.

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Prostate cancer is epithelial malignant prostate hyperplasia caused by a tumor. We found prostate cancer GSE141551 and GSE200879 profiles from gene expression omnibus database, followed by differentially expressed genes (DEGs) analysis, weighted gene co-expression network analysis, protein–protein interaction analysis, gene function enrichment analysis, and comparative toxicology database analysis. Finally, the gene expression heat map was drawn, and miRNA information regulating core DEGs was retrieved. A total of 1151 DEGs were found, most of them focusing on systematic development, cell deve

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Jadaan, Dima Y., Mutaz M. Jadaan, and John P. McCabe. "Cellular Plasticity in Prostate Cancer Bone Metastasis." Prostate Cancer 2015 (2015): 1–12. http://dx.doi.org/10.1155/2015/651580.

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Purpose.Experimental data suggest that tumour cells can reversibly transition between epithelial and mesenchymal states (EMT and MET), a phenomenon known as cellular plasticity. The aim of this review was to appraise the clinical evidence for the role of cellular plasticity in prostate cancer (PC) bone metastasis.Methods.An electronic search was performed using PubMed for studies that have examined the differential expression of epithelial, mesenchymal, and stem cell markers in human PC bone metastasis tissues.Results.The review included nineteen studies. More than 60% of the studies used ≤20

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Tribian, Laura Sophie, Maximilian Lennartz, Doris Höflmayer, et al. "Diagnostic Role and Prognostic Impact of PSAP Immunohistochemistry: A Tissue Microarray Study on 31,358 Cancer Tissues." Diagnostics 13, no. 20 (2023): 3242. http://dx.doi.org/10.3390/diagnostics13203242.

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Prostate-specific acid phosphatase (PSAP) is a marker for prostate cancer. To assess the specificity and prognostic impact of PSAP, 14,137 samples from 127 different tumor (sub)types, 17,747 prostate cancers, and 76 different normal tissue types were analyzed via immunohistochemistry in a tissue microarray format. In normal tissues, PSAP staining was limited to the prostate epithelial cells. In prostate cancers, PSAP was seen in 100% of Gleason 3 + 3, 95.5% of Gleason 4 + 4, 93.8% of recurrent cancer under androgen deprivation therapy, 91.0% of Gleason 5 + 5, and 31.2% of small cell neuroendoc

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Dissertations / Theses on the topic "Prostate epithelial and cancer markers"

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Knight, Jennifer Francesca. "Identification of Novel Markers for Prostatic Basal Epithelial Cells and Investigation of their expression and Potential Role in Prostate Cancers." Thesis, Institute of Cancer Research (University Of London), 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.498377.

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Valdman, Alexander. "Molecular genetic markers of prostate cancer development /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-618-9/.

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Larkin, Samantha. "Molecular characterisation of prostate cancer progression markers." Thesis, University of Portsmouth, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.511205.

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Shun, Kitty. "Study of two putative prostate cancer tumor markers." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111937.

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The following thesis is based on my study of two candidate prostate cancer tumor markers, namely telomerase and CD9. Accordingly, the thesis will be divided in two main chapters describing the results obtained for each protein. Although these proteins are de-regulated in prostate cancer cells, little is known about their regulation and function in prostate cells. For example, telomerase is generally inactive in normal cells and reactivated in cancer cells and CD9 is a transmembrane protein that is lost as prostate cancer progresses. In chapter 2, I will show that introduction of normal human c

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Lexander, Helena. "Protein expression in prostate cancer /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-617-4/.

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Bryant, Jennifer. "Neuroendocrine and epithelial markers of small cell lung cancer." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/neuroendocrine-and-epithelial-markers-of-small-cell-lung-cancer(c7c51e2c-6443-4021-b2ff-469966cd10b6).html.

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Small cell lung cancer (SCLC) is an extremely aggressive disease characterized by early metastasis and acquired resistance to therapy. SCLC is distinguished by its neuroendocrine (NE) component; the role of which is not fully understood in metastasis and response to therapy. Patients respond exceptionally well to first round chemotherapy; however, relapse with therapy-resistant tumours is virtually inevitable. Hypoxic regions within tumours can contribute towards metastasis and therapy resistance, highlighting hypoxia-targeted therapy as a novel approach for improving treatment for SCLC patien

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Fisken, Jane. "An investigation of serological tumour markers in epithelial ovarian cancer." Thesis, University of Edinburgh, 1992. http://hdl.handle.net/1842/19746.

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Epithelial ovarian cancer (EOC) accounts for over 80% of ovarian carcinomas. More than two-thirds of patients present with metastatic disease resulting in a poor five year survival of 25&37. Surgery is the mainstay of treatment; subsequently the majority of patients receive platinum based chemotherapy regimes. Although chemotherapy may improve progression free survival, it has little impact on overall survival. CA125 has an established role in monitoring response to chemotherapy and providing a lead time to clinical relapse. Its value in prognosis, however, requires clarification. Not all pati

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Brinham, Claire Louise. "Proteomic profiling of prostate cancer epithelial cell plasma membrane proteins." Thesis, University of York, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.444695.

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Scullin, P. "Molecular markers of response to systemic therapy in prostate cancer." Thesis, Queen's University Belfast, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.492490.

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Prostate cancer is a significant health problem worldwide. Glucocorticoids, including' dexamethasone have formed the backbone of treatment of androgenindependent disease for the past 50 years and are given with docetaxel in the only therapy with a proven survival benefit in this setting. This study explored the mecha'nism of dexamethasone action in the treatment of androgen-independent prostate cancer (AIPC). In the in vitro study' dexamethasone administration induced a suppression of NFKB transcriptional activity in AIPC cells, resulting in a decreased transcription . arid/or secretion of the

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Boddy, Jane Letitia. "Investigating new therapeutic markers in the management of prostate cancer." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428699.

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Books on the topic "Prostate epithelial and cancer markers"

1

Chen, Allen. Prostate cancer. Demos Medical, 2011.

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Vijayakumar, Srinivasan. Prostate cancer. Demos Medical, 2011.

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Sivasubramaniyan, Kavitha. A novel antibody directed against SSEA-4 defines spontaneous epithelial-to-mesenchymal cell transition in human prostate cancer. s.n.], 2014.

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4

J, Catalona William, Coffey Donald S, Karr James P, and Organ Systems Program (National Cancer Institute). Prostate Cancer Working Group., eds. Clinical aspects of prostate cancer: Assessment of new diagnostic and management procedures : proceedings of a workshop of the Prostate Cancer Working Group of the National Cancer Institute's Organ Systems Program, held October 16-19, 1988, at Prout's Neck, Maine, U.S.A. Elsevier, 1989.

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Chen, Allen M., and Srinivasan Vijayakumar. Prostate Cancer. Springer Publishing Company, Incorporated, 2011.

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Chen, Allen M., Srinivasan Vijayakumar, and Thomas Charles R. Jr. Prostate Cancer. Springer Publishing Company, Incorporated, 2011.

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Cuzick, Jack, and Mangesh A. Thorat. Prostate Cancer Prevention. Springer London, Limited, 2014.

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Cuzick, Jack, and Mangesh A. Thorat. Prostate Cancer Prevention. Springer, 2014.

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9

(Editor), Zbigniew Petrovich, Luc Baert (Editor), and Luther W. Brady (Editor), eds. Carcinoma of the Prostate: Innovations in Management. Springer-Verlag Telos, 1996.

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Skinner, D. G., L. W. Brady, Luther W. Brady, Luc Baert, and Zbigniew Petrovich. Carcinoma of the Prostate: Innovations in Management. Springer London, Limited, 2011.

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Book chapters on the topic "Prostate epithelial and cancer markers"

1

Chung, Leland W. K., and Haiyen E. Zhau. "Stromal-Epithelial Interaction." In Prostate Cancer. Humana Press, 2001. https://doi.org/10.1007/978-1-59259-009-4_20.

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Finlay, Judith A., Stephen D. Mikolajczyk, Thomas M. Pribyl, R. Bruce Wallace, and Harry G. Rittenhouse. "Prostate Cancer Markers." In Cancer Diagnostics. Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-791-8_7.

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Brothman, Arthur R., and Briana J. Williams. "Prostate Cancer." In Human Cytogenetic Cancer Markers. Humana Press, 1997. http://dx.doi.org/10.1007/978-1-4612-3952-9_10.

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Cheetham, Philippa J. "Markers in Prostate Cancer." In Prostate Cancer. John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118347379.ch4.

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Gadzinski, Adam J., and Matthew R. Cooperberg. "Prostate Cancer Markers." In Cancer Treatment and Research. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-93339-9_3.

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Chu, T. Ming. "Prostate-Specific Antigen." In Serological Cancer Markers. Humana Press, 1992. http://dx.doi.org/10.1007/978-1-4612-0401-5_5.

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Shore, Neal D., and Karen Ventii. "Genomic Markers." In The Prostate Cancer Dilemma. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-21485-6_9.

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Bumbaširević, Uroš, and Miloš Petrović. "Biological Markers of Therapeutic Response in Prostate Cancer." In Prostate Cancer. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-51712-9_11.

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Challacombe, Benjamin, John Fitzpatrick, and Roger Kirby. "PCA3 and Other Urinary Markers." In Prostate Cancer Diagnosis. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-188-2_6.

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Duijvesz, Diederick, and Guido Jenster. "Tumor Markers." In Prostate Cancer: A Comprehensive Perspective. Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-2864-9_35.

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Conference papers on the topic "Prostate epithelial and cancer markers"

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Chen, Chun-Liang, Pawel Osmulski, Devalingam Mahalingam, et al. "Abstract 5588: Epithelial-to-mesenchymal markers of circulating tumor cells for detection of castration-resistant prostate cancer." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-5588.

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Suman, Suman, Dominique Z. Jones-Reed, M. L. Schmidt, et al. "Abstract 1483: Alteration of miR-186 expression modifies inflammatory markers in normal epithelial and prostate cancer cell models." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1483.

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Alsulami, Haneen Hamed. "INVESTIGATING THE EFFECT OF CIGARETTE SMOKING ON THE NKX3.1 AND TMPRSS2 GENES ASSOCIATED WITH MALE FERTILITY." In Dubai International Conference on Research in Life-Science & Healthcare, 22-23 February 2024. Global Research & Development Services, 2024. http://dx.doi.org/10.20319/icrlsh.2024.3041.

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Cigarette's smoking has a wide negative impact on human health, and it's have been related to many serious issues like cancer, heart disease, respiratory system, and number of health problems. Also, smoking can affect fertility in men by affecting the sperm on several levels. Our research will investigate the genetic risk factors in male by focusing on NKX3.1 and TMPRSS2 genes related to male fertility and investigate the correlation between the gene’s polymorphisms of three groups (smokers, non-smokers, and infertile men). The NKX3.1 or NK3 homeobox 1 located on chromosome 8 is the first know

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Kolijn, Kimberley, Esther I. Verhoef, and Geert J. L. H. van Leenders. "Abstract 1590: N-cadherin is a key marker for epithelial-to-mesenchymal transition in clinical prostate cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1590.

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Glavaris, Stephanie, Heather Chalfin, Changxue Lu, et al. "Abstract B070: Epithelial-marker absent and disease-marker specific circulating and disseminated tumor cells are rarely detected in men with localized prostate cancer." In Abstracts: AACR Special Conference: Prostate Cancer: Advances in Basic, Translational, and Clinical Research; December 2-5, 2017; Orlando, Florida. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.prca2017-b070.

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Faugeroux, Vincent, Céline Lefebvre, Emma Pailler, et al. "Abstract 4953: Whole-exome sequencing of single circulating tumor cells according to epithelial-mesenchymal marker expression in metastatic prostate cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4953.

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Kampan, Nirmala Chandralega. "Unravelling the role of immune markers in epithelial ovarian cancer." In JSGO 2023. Korean Society of Gynecologic Oncology, 2023. http://dx.doi.org/10.3802/jgo.2023.34.s2.ayd-2.

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Wu, Tianying, Bruce Bracken, and Susan Kasper. "Abstract LB-178: Oxidation markers in relation to prostate cancer and BPH and progression of prostate cancer." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-lb-178.

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O'Neil, P., M. Zhao, X. Wang, and D. D. Nolte. "Interferometric detection of early markers for epithelial ovarian cancer and prognostic markers for acute lymphocytic leukemia." In BiOS, edited by Tuan Vo-Dinh, Warren S. Grundfest, and Anita Mahadevan-Jansen. SPIE, 2010. http://dx.doi.org/10.1117/12.841859.

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Borziak, Kirill, and Joseph Finkelstein. "Gene Expression Markers of Prognostic Importance for Prostate Cancer Risk in Patients with Benign Prostate Hyperplasia." In 2022 44th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2022. http://dx.doi.org/10.1109/embc48229.2022.9871422.

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Reports on the topic "Prostate epithelial and cancer markers"

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Lapointe, Jacques. Identification of Prostate Cancer Prognostic Markers. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada595253.

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Huang, Shuang. Identifying Early Diagnosis Markers of Prostate Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada442717.

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Huang, Shuang. Identifying Early Diagnosis Markers of Prostate Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada433991.

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Ritter, Mark A. Molecular Markers and Prostate Cancer Radiation Response. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada415011.

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Ritter, Mark A. Molecular Markers and Prostate Cancer Radiation Response. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada423342.

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Huang, Shuang. Identifying Early Diagnosis Markers of Prostate Cancer. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada462063.

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Bancroft, F. C. Novel Technology for Cloning Prostate Cancer Cell Markers. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada405355.

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Wu, Fangting. Exosome Mediates Stemness Transfer from Prostate Epithelial Progenitors to Prostate Cancer Cells. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada590430.

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Tyner, Angela L. Regulation of the Epithelial-Mesenchymal Transition in Prostate Cancer. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada594294.

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Hoon, Dave S. Serum Genetic Markers as Surrogates of Prostate Cancer Progression. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada485699.

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