Academic literature on the topic 'Prostate Testosterone'

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Journal articles on the topic "Prostate Testosterone"

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Ishola, Ismail O., Kayode O. Yemitan, Olasunmbo O. Afolayan, Charles C. Anunobi, and Tobi E. Durojaiye. "Potential of Moringa oleifera in the Treatment of Benign Prostate Hyperplasia: Role of Antioxidant Defence Systems." Medical Principles and Practice 27, no. 1 (2017): 15–22. http://dx.doi.org/10.1159/000486349.

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Objective: This study sought to evaluate the protective effect of ethanolic leaf extract of Moringa oleifera on testosterone-induced benign prostatic hyperplasia (BPH) in male Sprague-Dawley rats. Materials and Methods: BPH was induced in rats by the administration of testosterone propionate (3 mg/kg, s.c., in olive oil) for 4 weeks. M. oleifera (50, 100, or 200 mg/kg), celecoxib (20 mg/kg), or M. oleifera (50 mg/kg) + celecoxib (20 mg/kg) were orally administered daily 15 min before testosterone. On day 29, blood was collected to measure the levels of serum testosterone and prostate-specific
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Biancardi, Manoel F., Ana P. S. Perez, Cássia Regina Suzuki Caires, et al. "Prenatal exposure to testosterone masculinises the female gerbil and promotes the development of lesions in the prostate (Skene’s gland)." Reproduction, Fertility and Development 27, no. 7 (2015): 1000. http://dx.doi.org/10.1071/rd13387.

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Androgenic imbalance may disrupt prostate development, leading to morphological alterations in adulthood and predisposing this gland to develop diseases during ageing. However, little is known about the endocrine disruption of the prostate that is caused by androgenic compounds, especially in female experimental models. Therefore, this study aimed to evaluate the prostates of aged female gerbils exposed to testosterone at certain periods in intrauterine and postnatal life, to determine whether exposure at a particular age increases susceptibility to prostatic lesions in these animals. To this
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Xin, Qi, Mi-Jin Kwon, Ju-Woon Lee, et al. "Gamma Irradiated Rhodiola sachalinensis Extract Ameliorates Testosterone-Induced Benign Prostatic Hyperplasia by Downregulating 5-Alpha Reductase and Restoring Testosterone in Rats." Molecules 24, no. 21 (2019): 3981. http://dx.doi.org/10.3390/molecules24213981.

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The effect of Rhodiola sachalinensis Boriss extract irradiated with 50 kGy gamma rays (HKC) on benign prostatic hyperplasia (BPH) was investigated. Seven-week-old male SD rats received a subcutaneous injection of 20 mg/kg of testosterone propionate (TP) to induce BPH. Then, the testosterone only group received testosterone, the testosterone + finasteride group received testosterone and finasteride (5 mg/kg), the testosterone + HKC group received testosterone and HKC extract (500 mg/kg). Prostate weight and the dihydrotestosterone (DHT) levels in serum or prostate tissue were determined. The mR
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Arini, Luh Ari. "Studi Literatur Perbedaan Ekspresi Messenger Ribonucleid Acid (mRNA) Reseptor Androgen Setelah Pemberian Testosteron antara Penis dan Kelenjar Prostat Tikus Wistar Jantan (Rattus Norvegicus) Pascakastrasi." Jurnal Kesehatan Reproduksi 6, no. 1 (2019): 29. http://dx.doi.org/10.22146/jkr.37823.

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Background: Aging process (andropause) in men will cause a decrease in testosterone hormones, in other that decrese of androgen receptor (testosteron and dhyhidrtestosterone) in target organ, can be seen from AR mRNA expression. Andropause finaly impact of male reproductive organ, so given testosterone hormone therapy is important to restore the condition. Giving testosterone hormone causes increase the expression of AR mRNA in the prostate gland and penis, but in these two organs there is a different increase.Objective: to know the difference of expression AR mRNA after administration of test
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Vignozzi, Linda, Annamaria Morelli, Erica Sarchielli, et al. "Testosterone protects from metabolic syndrome-associated prostate inflammation: an experimental study in rabbit." Journal of Endocrinology 212, no. 1 (2011): 71–84. http://dx.doi.org/10.1530/joe-11-0289.

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Metabolic syndrome (MetS) and benign prostatic hyperplasia (BPH)/lower urinary tract symptoms (LUTS) are often associated. One of their common denominators is hypogonadism. However, testosterone supplementation is limited by concerns for potential prostatic side effects. The objective was to determine whether MetS-associated prostate alterations are prevented by testosterone supplementation. We used a previously described animal model of MetS, obtained by feeding male rabbits a high-fat diet (HFD) for 12 weeks. Subsets of HFD rabbits were treated with testosterone or with the farnesoid X recep
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Herrera-Covarrubias, D., G. A. Coria-Avila, M. E. Hernandez, and N. Ismail. "STRESS DURING PUBERTY FACILITATES PRECANCEROUS PROSTATE LESIONS IN ADULT RATS." Experimental Oncology 39, no. 4 (2017): 269–75. http://dx.doi.org/10.31768/2312-8852.2017.39(4):269-275.

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Puberty can be a critical period for the long-term development of diseases, especially for stress-related disorders that depend on neuroendocrine and immune responses. Some organs like the prostate are prone to diseases that result from neuroendocrine or immune challenges, such as cancer. Aim: In the present study, we assessed the long-term effects of an acute pubertal stressor (immune-challenge) on the development of precancerous lesions in adult rats, and compared them with testosterone-induced prostatic lesions. Materials and Methods: Pubertal male rats received a single injection of lipopo
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Campos, Mônica S., Naiara C. S. Ribeiro, Rodrigo F. de Lima, et al. "Anabolic effects of chrysin on the ventral male prostate and female prostate of adult gerbils (Meriones unguiculatus)." Reproduction, Fertility and Development 30, no. 9 (2018): 1180. http://dx.doi.org/10.1071/rd17456.

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Chrysin is a bioflavonoid found in fruits, flowers, tea, honey and wine, which has antioxidant, anti-inflammatory, antiallergic and anticarcinogenic properties. This flavone has also been considered as beneficial for reproduction due its testosterone-boosting potential. Thus, the aim of this study was to evaluate the effects of chrysin on the prostate and gonads of male and female adult gerbils. In addition, a comparative analysis of the effects of testosterone on these same organs was conducted. Ninety-day-old male and female gerbils were treated with chrysin (50 mg kg−1 day−1) or testosteron
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Omoto, Yoko. "Estrogen Receptor-α Signaling in Growth of the Ventral Prostate: Comparison of Neonatal Growth and Postcastration Regrowth". Endocrinology 149, № 9 (2008): 4421–27. http://dx.doi.org/10.1210/en.2007-1413.

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A role for estrogen receptor (ER)-α in branching morphogenesis in the ventral prostate (VP) has previously been demonstrated; in the VP of ERα−/− mice, there are fewer side branches than in wild-type littermates. In the present study, we show that in the postnatal VP, fibroblast growth factor 10 (FGF10) is expressed in wild-type mice but not in ERα−/− mice, and because branching involves proliferation pathways also used in malignant growth, we investigated whether branching during regrowth of the VP after castration involves ERα and FGF10. ERα was not detectable in the prostates of sham-operat
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Yassin, A., K. AlRumaihi, R. Alzubaidi, S. Alkadhi, and A. Al Ansari. "Testosterone, testosterone therapy and prostate cancer." Aging Male 22, no. 4 (2019): 219–27. http://dx.doi.org/10.1080/13685538.2018.1524456.

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Kusama, Kazuya, Mayuko Miyagawa, Koichiro Ota, et al. "Cordyceps militaris Fruit Body Extract Decreases Testosterone Catabolism and Testosterone-Stimulated Prostate Hypertrophy." Nutrients 13, no. 1 (2020): 50. http://dx.doi.org/10.3390/nu13010050.

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The androgens testosterone and dihydrotestosterone (DHT) are essential for a variety of systemic functions in mature males. Alteration of these hormones results in late-onset hypogonadism (LOH) and benign prostate hyperplasia (BPH). The fruit bodies of fungi of the genus Cordyceps have been regarded as folk medicine or health food with tonic and antifatigue effects. The extract from the fruit body of Cordyceps militaris parasitizing Samia cynthia ricini (CM) was evaluated as a novel-candidate natural product for ameliorating male andropause symptoms. To explore the effects of CM on LOH and BPH
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Dissertations / Theses on the topic "Prostate Testosterone"

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Silva, Elcio Dias 1951. "Goserelina versus leuprolide na castração química de pacientes com câncer prostático = Goserelin versus leuprolide in the chemical castration of patients with prostate cancer." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/312273.

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Orientadoesr: Wagner Eduardo Matheus, Ubirajara Ferreira<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-20T13:05:18Z (GMT). No. of bitstreams: 1 Silva_ElcioDias_D.pdf: 1974038 bytes, checksum: e1e7b084d346a50a85fec60666fbf526 (MD5) Previous issue date: 2012<br>Resumo: Objetivo: avaliar a eficácia relativa do leuprolide 3,75 mg, leuprolide 7,5 mg e goserelina 3,6 mg quanto à redução da testosterona sérica, em níveis de castração. Método: foram avaliados 60 pacientes com carcinoma avançado de próstata, com indicação
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Grouf, Jaime L. "Characterization of value nutritions 5-androstenediol product and its proliferative effects on the LNCaP cell line." Link to electronic thesis, 2007. http://www.wpi.edu/Pubs/ETD/Available/etd-102707-170420/.

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Fávaro, Wagner José 1980. "Reatividade hormonal e ultra-estrutura do lobo ventral da prostata de ratos (Ratus norvegicus) submetidos ao uso cronico simultaneo de etanol e nicotina." [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/318011.

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Orientador: Valeria Helena Alves Cagno Quitete<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-06T20:48:50Z (GMT). No. of bitstreams: 1 Favaro_WagnerJose_M.pdf: 3700284 bytes, checksum: 4b1aec37ebb6981bbfdb52290cf218ca (MD5) Previous issue date: 2006<br>Resumo: o álcool e a nicotina têm ação prejudicial sobre o funcionamento da glândula prostática. Contudo, os mecanismos moleculares e celulares envolvidos nas alterações prostáticas, bem como suas correlações com a patogênese glandular permanecem desconhecidos. Assim,
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Biancardi, Manoel Francisco 1981. "Estudo da organogênese prostática normal e os efeitos da exposição androgênica intrauterina e puberal sobre a morfofisiologia da próstata de gerbilos senis." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/316157.

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Orientador: Sebastião Roberto Taboga<br>Texto em português e inglês<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-25T04:58:37Z (GMT). No. of bitstreams: 1 Biancardi_ManoelFrancisco_D.pdf: 15374332 bytes, checksum: b70012fe11a604131b1622adb07d2654 (MD5) Previous issue date: 2014<br>Resumo: Pesquisas recentes tem mostrado que a predisposição às desordens prostáticas tem origem nos momentos iniciais da vida. Portanto, o entendimento detalhado da organogênese prostática e dos mecanismos envolvidos na predisposição à estas de
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Constantino, Flávia Bessi. "Relativa influência da prolactina, sua inibição e combinação com testosterona sobre a próstata de ratos castrados comparação entre recrescimento glandular, diferenciação celular e atividade secretora /." Botucatu, 2017. http://hdl.handle.net/11449/148973.

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Orientador: Luis Antonio Justulin Junior<br>Resumo: A próstata é uma glândula do sistema genital masculino que possui grande importância na fertilidade. Estudos clássicos evidenciaram que para que se inicie o desenvolvimento prostático é necessário a presença de andrógeno produzido pelos testículos fetais. Porém, além da estimulação androgênica, estudos apontam para outros hormônios que também atuam na próstata como a Prolactina (PRL). PRL é um hormônio que é principalmente secretado por células lactotróficas da hipofise anterior e está envolvido em muitos processos biológicos, incluindo lacta
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Augusto, Taize Machado. "Modulação da expressão da heparanase-1 na próstata ventral de ratos e sua relação com aspectos gerais da fisiologia do órgão na castração e no imprinting estrogênico." [s.n.], 2011. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317557.

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Orientador: Hernandes Faustino de Carvalho<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-17T20:52:36Z (GMT). No. of bitstreams: 1 Augusto_TaizeMachado_D.pdf: 4110210 bytes, checksum: d70ae2c92435700f28fff9ee3cae63ac (MD5) Previous issue date: 2011<br>Resumo: A próstata é uma glândula do aparelho reprodutor importante na reprodução, sendo foco de várias afecções, dentre elas o carcinoma prostático. Ela é altamente dependente de testosterona e modulada por estrógenos, que desempenham papel fundamental no seu crescimento. A
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Mendes, Leonardo de Oliveira 1985. "Análise de receptores hormonais e marcadores inflamatórios no lobo ventral da próstata de ratos consumidores voluntários de etanol (UChB) = influência da terapia hormonal com testosterona." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/316146.

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Orientadores: Francisco Eduardo Martinez, Wellerson Rodrigo Scarano<br>Texto em português e inglês<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-25T05:09:17Z (GMT). No. of bitstreams: 1 Mendes_LeonardodeOliveira_D.pdf: 2740474 bytes, checksum: 7720a25e258ca28197be2547f35bc162 (MD5) Previous issue date: 2014<br>Resumo: O etanol provoca danos reprodutivos, diretamente nos tecidos ou indiretamente via eixo hipotalâmico-hiposário-gonadal. A próstata, hormônio-dependente, é susceptível aos efeitos do etanol. Evidências atribu
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Skaudickas, Darius. "Rausvažiedės ežiuolės Echinacea purpurea (L.) Moench antiandrogeninių savybių eksperimentiniai tyrimai." Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2005. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2005~D_20051209_100749-23074.

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1. INTRODUCTION 1.1. Work actuality In recent years the number of males with urination disorders mostly induced by prostate pathology has significantly increased [Gas et al., 1998; Khan, Khan et al., 2005; Amaral, Coeli et al., 2004]. Benign prostate hyperplasia (BPH) affects males at a much younger age making them complain of urination disorders of different types and intensity. It is quite a common pathology among males (which is) conditioned by life style and nutrition [Saga & Sugimura, 2004; Shabbir & Mumtaz, 2004; Cambell, 2005]. Clinical symptoms of BPH are not restricted only to u
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Biancardi, Manoel Francisco 1981. "Ação androgênica intra-uterina e pós-natal sobre a próstata de roedores." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317915.

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Orientador: Sebastião Roberto Taboga<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-16T00:12:46Z (GMT). No. of bitstreams: 1 Biancardi_ManoelFrancisco_M.pdf: 6869231 bytes, checksum: 32275d5e0c79d81cecdc898bc5e42bb9 (MD5) Previous issue date: 2010<br>Resumo: A próstata é uma glândula encontrada não somente em machos de mamíferos, podendo estar presente em fêmeas de alguns roedores e também em mulheres. Os andrógenos são essenciais durante o desenvolvimento e também para a manutenção da homeostasia prostática ao long
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Greenhough, Katherine Jane. "Studies on the regulation of DNA synthesis in the rat ventral prostate by androgens." Thesis, University of Leeds, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252627.

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Books on the topic "Prostate Testosterone"

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Alves, Ines Teles, Jan Trapman, and Guido Jenster. Molecular biology of prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0059.

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Prostate cancer is a heterogeneous disease that arises through the acquisition of key malignant hallmarks. At the molecular level, prostate tumours are dependent upon the androgen receptor pathway, which affects cell function, growth, and behaviour through downstream androgen-regulated genes. Prostate cancer requires this activity and manipulates the AR pathway to maintain signalling. For example, mutation of the AR (to bind ligands other than androgens) or amplification/duplication of the AR allows signalling to continue in the absence of testosterone. Around 50% of prostate cancers have a ge
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Key, Timothy J., and Alison J. Price. Epidemiology of prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0058.

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Prostate cancer is the second most common malignancy and the sixth most common cause of cancer death for men worldwide. The highest incidence and mortality rates are in populations that originated in Africa, such as African Americans. Rates are also high in Western countries and generally low in East and South Asia. Incidence rates are increasing in some countries which until recently had low rates, but are not changing much in countries which already have high rates. The only well-established risk factors are increasing age, African ancestry, family history of the disease, and certain genetic
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New Testosterone Treatment: How You and Your Doctor Can Fight Breast Cancer, Prostate Cancer, and Alzheimer's. Prometheus Books, Publishers, 2013.

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Mukherji, Deborah, Aurelius Omlin, Carmel Pezaro, and Johann De Bono. Novel therapies and emerging strategies for the treatment of patients with castration-resistant prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0069.

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Castration-resistant prostate cancer (CRPC) represents a final stage of this malignancy for many men and is defined as the progression of prostate cancer despite castrate levels of testosterone. CRPC may present as a rising PSA, the development of new metastases, or worsening of known metastases. Recent advances have resulted in five new treatments for CRPC: the immunotherapy sipuleucel-T; the cytotoxic cabazitaxel; the androgen biosynthesis inhibitor abiraterone acetate; the radioisotope radium-223; and the antiandrogen enzalutamide. These have all improved overall survival in randomized phas
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Muñoz, George, and Mikhail Kogan. Men’s Health. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190466268.003.0010.

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The aim of this chapter is to review nutrition, hormones, natural substances, and integrative aspects to men’s health specific to the geriatric population. This segment of the population is growing, and they are more active and want to remain so for as long as possible. Older men therefore are entitled to and demand healthy options and lifestyle education and interventions to meet their future goals. This chapter covers exercise, nutrition, and food recommendation for the healthy aging male. Topics include prostate health, sexuality, erectile dysfunction, and hormone deficiencies, specifically
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Book chapters on the topic "Prostate Testosterone"

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Frick, Julian, Andreas Jungwirth, and Erwin Rovan. "Androgens and the prostate." In Testosterone. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-72185-4_9.

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Soares, Daniel de Freitas G., Ernani Luis Rhoden, and Abraham Morgentaler. "Testosterone Therapy and Prostate Cancer." In Testosterone. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-46086-4_15.

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Schröder, Fritz H. "Androgens and carcinoma of the prostate." In Testosterone. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-662-00814-0_13.

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Soares, Daniel de Freitas G., Ernani Luis Rhoden, and Abraham Morgentaler. "Correction to: Testosterone Therapy and Prostate Cancer." In Testosterone. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-46086-4_20.

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Salter, Carolyn A., and John P. Mulhall. "Testosterone and Prostate Effects." In Controversies in Testosterone Deficiency. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-77111-9_7.

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Schulman, Claude, and Abraham Morgentaler. "Testosterone and Prostate Safety." In Clinical Uro-Andrology. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45018-5_11.

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Morgentaler, Abraham, and Claude Schulman. "Testosterone and Prostate Safety." In Frontiers of Hormone Research. KARGER, 2008. http://dx.doi.org/10.1159/000176054.

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Alba, Frances, Claudio A. Romero, and Run Wang. "The Role of Androgens in Prostate Cancer." In Androgen Deficiency and Testosterone Replacement. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-179-0_6.

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Kit, Ng Kok. "Testosterone Therapy, Prostate Safety, and Other Safety Issues." In Men's Health. Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-4766-4_7.

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Balk, Steven P. "Increased Expression of Genes Converting Adrenal Androgens to Testosterone in Castration-Recurrent Prostate Cancer." In Androgen Action in Prostate Cancer. Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-69179-4_5.

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Conference papers on the topic "Prostate Testosterone"

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Thomas-Ahner, Jennifer M., Lei Wan, Hsueh-Li Tan, et al. "Abstract 3701: Tomato carotenoids and testosterone modulate mRNA and miRNA profiles during prostate carcinogenesis." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3701.

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Sissung, Tristan M., C. Tyler Kirkland, Kelie Reece, Julia Tyler Arnold та William D. Figg. "Abstract 4535: Thalidomide inhibits the TGFβ-mediated synthesis of testosterone from DHEA in prostate cancer". У Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-4535.

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Ide, Hisamitsu, Yu Jingsong, Lu Yan, Satoru Muto, and Shigeo Horie. "Abstract 4385: Testosterone induces DNA damage signaling in response to oxidative stress in prostate cancer cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4385.

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Mout, Lisanne, Corrina de Ridder, Debra Stuurman, et al. "Abstract 1099: Testosterone abolishes cabazitaxel efficacy in anin vivomodel of prostate cancer and affects intratumoral concentrations." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1099.

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Cook, Michael B., Daniel C. Beachler, Lauren E. Parlett, et al. "Abstract 5050: Testosterone supplementation in relation to prostate cancer in a US commercial insurance claims database." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-5050.

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Cook, Michael B., Daniel C. Beachler, Lauren E. Parlett, et al. "Abstract 5050: Testosterone supplementation in relation to prostate cancer in a US commercial insurance claims database." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-5050.

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Ozten, Nur, Michael J. Schlicht, and Maarten C. Bosland. "Abstract 5579: Selenomethionine does not protect against testosterone plus 17 beta-estradiol-induced prostate carcinogenesis and prostatic oxidative stress in NBL rats." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-5579.

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Adaramoye, Oluwatosin A., Solomon E. Owumi, Olubukola Akanni, Oluwabunmi E. Olapade-Olaopa, Olusoji J. Abiola, and Oluyemi Akinloye. "Abstract A007: Methyl jasmonate and finasteride synergistically reduce testosterone-induced benign prostatic hyperplasia through regulation of inflammatory and apoptotic processes in rats." In Abstracts: AACR Special Conference: Prostate Cancer: Advances in Basic, Translational, and Clinical Research; December 2-5, 2017; Orlando, Florida. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.prca2017-a007.

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Peskoe, Sarah B., William G. Nelson, Corinne E. Joshu, Sabine Rohrmann, Katherine A. McGlynn, and Elizabeth A. Platz. "Abstract 3614: Are circulating testosterone and PSA levels associated in a nationally representative sample of men without a diagnosis of prostate cancer." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3614.

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Agurs-Collins, Tanya D., Kepher Makambi, Hyunuk Seung, Bruce J. Trock, and Kevin J. Cullen. "Abstract A78: Associations between IGF-axis, testosterone and nutrients in healthy African American and Caucasian men at increased risk for prostate cancer." In Abstracts: AACR International Conference on the Science of Cancer Health Disparities‐‐ Sep 30-Oct 3, 2010; Miami, FL. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1055-9965.disp-10-a78.

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