Relevant bibliographies by topics / Protein Akt

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Protein Akt'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Protein Akt"

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Guda, B. B., V. V. Pushkarev, O. V. Zhuravel, et al. "Protein kinase Akt activity in human thyroid tumors." Ukrainian Biochemical Journal 88, no. 5 (2016): 90–95. http://dx.doi.org/10.15407/ubj88.05.090.

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Abraham, Edward. "Akt/protein kinase B." Critical Care Medicine 33, Suppl (2005): S420—S422. http://dx.doi.org/10.1097/01.ccm.0000191715.31970.d8.

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Nakamura, Akito, Mikihiko Naito, Takashi Tsuruo, and Naoya Fujita. "Freud-1/Aki1, a Novel PDK1-Interacting Protein, Functions as a Scaffold To Activate the PDK1/Akt Pathway in Epidermal Growth Factor Signaling." Molecular and Cellular Biology 28, no. 19 (2008): 5996–6009. http://dx.doi.org/10.1128/mcb.00114-08.

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ABSTRACTThe phosphoinositide 3-kinase (PI3K)/3-phosphoinositide-dependent protein kinase 1 (PDK1)/Akt pathway regulates various cellular functions, especially cell survival and cell cycle progression. In contrast to other survival pathways, there have been few reports of scaffold proteins that regulate signaling cascade specificity in this pathway. Here we identify a 5′ repressor element under dual-repression binding protein 1 (Freud-1)/Aktkinase-interacting protein1(Aki1) as a novel scaffold for the PDK1/Akt pathway. Freud-1/Aki1 (also known as CC2D1A) expression induced formation of a PDK1/A
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Hill, Michelle M., and Brian A. Hemmings. "Inhibition of protein kinase B/Akt." Pharmacology & Therapeutics 93, no. 2-3 (2002): 243–51. http://dx.doi.org/10.1016/s0163-7258(02)00193-6.

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Yang, Z. Z., O. Tschopp, A. Baudry, B. Dümmler, D. Hynx, and B. A. Hemmings. "Physiological functions of protein kinase B/Akt." Biochemical Society Transactions 32, no. 2 (2004): 350–54. http://dx.doi.org/10.1042/bst0320350.

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The genetic manipulation of mice has become an essential and elegant method for studying the function of proteins in physiology, and for testing the veracity of information obtained from cell culture experiments. During the past few years, a variety of transgenic and knockout mouse models of PKB (protein kinase B)/Akt have been generated and investigated. In this paper, we focus on the phenotypes of these PKB/Akt overexpression and mutant mice that may help to elucidate the functions exerted by PKB/Akt in mammals.
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CALLEJA, Véronique, Simon M. AMEER-BEG, Borivoj VOJNOVIC, Rudiger WOSCHOLSKI, Julian DOWNWARD, and Banafshé LARIJANI. "Monitoring conformational changes of proteins in cells by fluorescence lifetime imaging microscopy." Biochemical Journal 372, no. 1 (2003): 33–40. http://dx.doi.org/10.1042/bj20030358.

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To be able to detect in situ changes in protein conformation without perturbing the physiological environment would be a major step forward in understanding the precise mechanism occurring in protein interaction. We have developed a novel approach to monitoring conformational changes of proteins in intact cells. A double-labelled fluorescent green fluorescent protein–yellow fluorescent protein (GFP–YFP) fusion protein has been constructed, allowing the exploitation of enhanced-acceptor-fluorescence (EAF)-induced fluorescence resonance energy transfer (FRET). Additionally, a novel fusion partne
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Rotwein, Peter. "Variation in Akt protein kinases in human populations." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 313, no. 6 (2017): R687—R692. http://dx.doi.org/10.1152/ajpregu.00295.2017.

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The three Akt kinases are related proteins that are essential for normal growth and metabolic regulation and are implicated as key signaling mediators in many physiological and pathophysiological processes. Each Akt is activated by common biochemical signals that act downstream of growth factor and hormone receptors via phosphatidylinositol-3 kinase, and each controls several downstream pathways. The importance of Akt actions in human physiology is strengthened by the rarity of modifying mutations in their genes and by the devastating impact caused by these mutations on growth and development
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Faridi, Jesika, Janet Fawcett, Lihong Wang, and Richard A. Roth. "Akt promotes increased mammalian cell size by stimulating protein synthesis and inhibiting protein degradation." American Journal of Physiology-Endocrinology and Metabolism 285, no. 5 (2003): E964—E972. http://dx.doi.org/10.1152/ajpendo.00239.2003.

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Expression of constitutively active Akt3 was found to increase the size of MCF-7 cells approximately twofold both in vitro and in vivo. A regulatable version of Akt1 (MER-Akt) was also found capable of inducing a twofold increase in the size of H4IIE rat hepatoma cells. Rapamycin, a specific inhibitor of mTOR function, was found to inhibit the Akt-induced increase in cell size by 70%, presumably via inhibition of the Akt-induced increase in protein synthesis. To determine whether Akt could be inhibiting protein degradation, thereby contributing to its ability to induce an increase in cell size
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Bang, Sookhee, Catherine Steenstra, and Sangwon F. Kim. "Striatum specific protein, Rhes regulates AKT pathway." Neuroscience Letters 521, no. 2 (2012): 142–47. http://dx.doi.org/10.1016/j.neulet.2012.05.073.

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Fayard, E. "Protein kinase B/Akt at a glance." Journal of Cell Science 118, no. 24 (2005): 5675–78. http://dx.doi.org/10.1242/jcs.02724.

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Dissertations / Theses on the topic "Protein Akt"

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Yan, Weisi. "Functional proteomic study of Akt reveals novel substrates in translation /." Access full-text from WCMC, 2008. http://proquest.umi.com/pqdweb?did=1619246521&sid=9&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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Zhang, Yun. "Activation of Erk1/2 and Akt in astrocytes under ischemia /." View Abstract or Full-Text, 2002. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202002%20ZHANGY.

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Thesis (M. Phil.)--Hong Kong University of Science and Technology, 2002.<br>Includes bibliographical references (leaves 98-115). Also available in electronic version. Access restricted to campus users.
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Park, Sungman. "AKT function and human oncogenesis." [Tampa, Fla.] : University of South Florida, 2007. http://purl.fcla.edu/usf/dc/et/SFE0001885.

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Wu, Hoi Ti. "Regulation of the pro-survival AKT signaling cascade : roles of receptor tyrosine kinases, G proteins, and their crosstalks /." View abstract or full-text, 2006. http://library.ust.hk/cgi/db/thesis.pl?BICH%202006%20WU.

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Lu, Bin. "Roles of cellular FLICE-inhibitory protein (c-FLIP) and Pl3K/Akt in Fas (CD95)-induced NF-[kappa]B activation and apoptosis through death effector domains." Morgantown, W. Va. : [West Virginia University Libraries], 2005. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=4344.

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Thesis (Ph. D.)--West Virginia University, 2005.<br>Title from document title page. Document formatted into pages; contains viii, 95 p. : ill. (some col.). Includes abstract. Includes bibliographical references (p. 82-95).
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Tucka, Joanna Barbara. "Regulation of vascular smooth muscle cell survival by the Akt pathway." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610282.

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Xia, Chang. "The role of reactive oxygen species and PI3K/AKT signaling in tumor angiogenesis." Morgantown, W. Va. : [West Virginia University Libraries], 2006. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=4714.

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Thesis (Ph. D.)--West Virginia University, 2006.<br>Title from document title page. Document formatted into pages; contains xi, 261 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references.
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Rato, Leila Sofia Coelho. "Vimentin interacts with the Akt/mTOR pathway mediating cell growth." Master's thesis, Universidade de Aveiro, 2017. http://hdl.handle.net/10773/22372.

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Mestrado em Bioquímica - Bioquímica Clínica<br>A vimentina é uma proteína da classe III dos filamentos intermédios que promove processos tais como proliferação, migração e invasão celular através da interação com diferentes vias de sinalização. No entanto, o papel da vimentina no crescimento celular é ainda pouco conhecido. Neste estudo, observamos que fibroblastos isolados de embriões de ratinhos sem vimentina (Vim -/- MEFs) eram mais pequenos que o tipo normal (WT). Assim, o objetivo deste estudo era entender de que forma a vimentina regula o crescimento celular. Com recurso a modelos in vit
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Matthews, Jason Aaron. "Investigation of the effects of increased levels of O-GlcNAc protein modification on protein kinase C and Akt." [Tampa, Fla] : University of South Florida, 2006. http://purl.fcla.edu/usf/dc/et/SFE0001723.

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Limmer, Sebastian. "Alpha1-Adrenozeptor-vermittelte Regulation der Akt/Protein-Kinase B in der humanen Prostata." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-177826.

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Books on the topic "Protein Akt"

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US GOVERNMENT. An Act to to Protect Seniors from Fraud. U.S. G.P.O., 2000.

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US GOVERNMENT. An Act to Protect Infants Who Are Born Alive. U.S. G.P.O., 2002.

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GOVERNMENT, US. An Act to Define and Protect the Institution of Marriage. U.S. G.P.O., 1996.

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US GOVERNMENT. An Act to Protect Religious Liberty, and for Other Purposes. U.S. G.P.O., 2000.

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Canada, Canada Parks. Preserve and protect: An overview of the National Parks Act. Parks Canada, 1986.

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Ezratty, Harry A. How to collect and protect works of art. Omni Arts, 1988.

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1928-, Wagner Bernard Meyer, Museum for African Art (New York, N.Y.), and National Museum of Health and Medicine (U.S.), eds. To cure and protect: Sickness and health in African art. Museum for African Art, 1999.

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Act up--Paris. Editions J. Di Sciullo, 2009.

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R, Ettema, Melville Bruce W, National Cooperative Highway Research Program., National Research Council (U.S.). Transportation Research Board., American Association of State Highway and Transportation Officials., and United States. Federal Highway Administration., eds. Countermeasures to protect bridge abutments from scour. Transportation Research Board, 2007.

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United States. Congress. Senate. Committee on the Judiciary. The PROTECT Act: Report together with additional views (to accompany S. 151). U.S. G.P.O., 2003.

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Book chapters on the topic "Protein Akt"

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Donato, Dominique M., Steven K. Hanks, Kenneth A. Jacobson, et al. "Protein Kinase B/Akt." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101097.

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Girardi, Cristina, and Maria Ruzzene. "CK2 Function in the Regulation of Akt Pathway." In Protein Kinase CK2 Cellular Function in Normal and Disease States. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-14544-0_8.

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Jain, Shruti, and D. S. Chauhan. "Linear and Nonlinear Modeling of Protein Kinase B/AkT." In Advances in Intelligent Systems and Computing. Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0135-2_7.

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Enomoto, Atsushi, Liang Weng, and Masahide Takahashi. "Critical Roles of the AKT Substrate Girdin in Disease Initiation and Progression." In Protein Modifications in Pathogenic Dysregulation of Signaling. Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55561-2_15.

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Todisco, Andrea, Nonthalee Pausawasdi, Saravanan Ramamoorthy, John Del Valle, Rebecca W. Van Dyke, and Frederick K. Askari. "Functional Role of Protein KinaseB/Akt in Gastric Acid Secretion." In Mechanisms and Consequences of Proton Transport. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0971-4_24.

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Fayard, Elisabeth, Gongda Xue, Arnaud Parcellier, Lana Bozulic, and Brian A. Hemmings. "Protein Kinase B (PKB/Akt), a Key Mediator of the PI3K Signaling Pathway." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/82_2010_58.

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Srivastava, Ashok K., Nihar R. Pandey, and Antoine Blanc. "Activation of Mitogen-Activated Protein Kinases and Protein Kinase B/Akt Signaling by Oxidative Stress in Vascular Smooth Muscle Cells: Involvement in Vascular Pathophysiology." In Pathophysiology of Cardiovascular Disease. Springer US, 2004. http://dx.doi.org/10.1007/978-1-4615-0453-5_30.

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Iturri, Pablo, Vincent Joseph, Gloria Rodrigo, Aida Bairam, and Jorge Soliz. "Inhibition of Protein Kinases AKT and ERK1/2 Reduce the Carotid Body Chemoreceptor Response to Hypoxia in Adult Rats." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-18440-1_31.

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Schnell, Santiago. "Knitting Proteins." In The Art of Theoretical Biology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-33471-0_27.

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Rieser, Jens, Jörg Ackermann, and Ina Koch. "Green Protein Interaction Wheel." In The Art of Theoretical Biology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-33471-0_56.

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Conference papers on the topic "Protein Akt"

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Montgrain, Philippe R., Rick Quintana, Leonard J. Deftos, and Randolph H. Hastings. "Akt Regulates Parathyroid Hormone-Related Protein Expression In Lung Cancer." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2066.

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Yang, Jilai, Qi Shen, David J. Newman, A. Douglas Kinghorn, and Steven M. Swanson. "Abstract 2815: Sylvestrol inhibits protein synthesis by downregulating the AKT/mTOR pathway." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2815.

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Zhang, Qiang, Meimei Wan, David A. Horita, et al. "Abstract A25: Yin Yang 1 promotes AKT activation through direct protein binding." In Abstracts: AACR Special Conference on Chromatin and Epigenetics in Cancer - June 19-22, 2013; Atlanta, GA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.cec13-a25.

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Hsu, Alice H., Kathryn J. Curry, Kang-Sup Shim, et al. "Abstract 189: Protein kinase C alpha suppresses AKT activation in endometrial cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-189.

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Wei, Tao, Suyong Choi, Darya Buehler, Richard A. Anderson, and Paul F. Lambert. "Abstract 1426: A PI3K/AKT scaffolding protein, IQ motif-containing GTPase associating protein 1 (IQGAP1), promotes head and neck carcinogenesis." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-1426.

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Hsu, Alice H., Kathryn J. Curry, Kang-Sup Shim та ін. "Abstract 4720: Protein kinase C alpha (PKCα) regulates PI3K/AKT signaling in endometrial cancer". У Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4720.

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Visuttijai, Kittichate, Katarina Ejeskär, and Afrouz Behboudi. "Abstract 2166: Analyses of protein expression of PI 3-kinase/AKT signaling in response to altered expression of motor protein MYO1C." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2166.

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Peng, Jei-Ming, Shao-Wen Hung, Ching-Feng Chiu, et al. "Abstract C40: Recombinant viral protein suppresses metastasis of ovarian adenocarcinoma cells through negative Akt signaling." In Abstracts: Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-c40.

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Cho, Eunho. "Abstract 1115: NANOG-AKT signaling increases metastatic capacity via stabilizes HDAC1 protein through Chfr inactivation." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1115.

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Cho, Eunho. "Abstract 1115: NANOG-AKT signaling increases metastatic capacity via stabilizes HDAC1 protein through Chfr inactivation." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1115.

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Reports on the topic "Protein Akt"

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Vojtek, Anne B. Celluar and Molecular Roles of the Akt Protein Kinase in Breast Carcinomas. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada395131.

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Vojtek, Anne B. Cellular and Molecular Roles of the Akt Protein Kinase in Breast Carcinomas. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada407622.

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Kraft, Andrew S. Overcoming Resistance to Inhibitors of the AKT Protein Kinase by Modulation of the Pim Kinase Pathway. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada597882.

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Kraft, Andrew S. Overcoming Resistance to Inhibitors of the Akt Protein Kinase by Modulation of the Pim Kinase Pathway. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada615425.

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Berman, Helen. Protein Data Bank Project at Rutgers University. Office of Scientific and Technical Information (OSTI), 2002. http://dx.doi.org/10.2172/805813.

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Tian, Yongchi, and Jia Chen. Protein-Assisted Redox Sensing at Biomimetic Electrode. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada412000.

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Li, Fenglei. Automated High Throughput Protein Crystallization Screening at Nanoliter Scale and Protein Structural Study on Lactate Dehydrogenase. Office of Scientific and Technical Information (OSTI), 2006. http://dx.doi.org/10.2172/892735.

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Sohn, Lydia, T. C. Messina, L. N. Dunkleberger, et al. Probing Interactions at the Nanoscale. Sensing Protein Molecules. Office of Scientific and Technical Information (OSTI), 2003. http://dx.doi.org/10.2172/940829.

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Dabney-Smith, Carole. RECOVERY ACT - Thylakoid Assembly and Folded Protein Transport by the Tat Pathway. Office of Scientific and Technical Information (OSTI), 2016. http://dx.doi.org/10.2172/1326395.

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Yuan, V., H. Frauenfelder, R. W. Harper, et al. Parity violation in proton-proton scattering at intermediate energies. Office of Scientific and Technical Information (OSTI), 1986. http://dx.doi.org/10.2172/5610738.

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