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Books on the topic 'Protein-Energy Malnutrition'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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1

Waterlow, J. C. Protein-energy malnutrition. London: Edward Arnold, 1992.

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2

Bhat, B. Vishnu. Protein, energy, malnutrition. New Delhi: Peepee Publishers and Distributors, 2008.

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3

M, Grantham-McGregor Sally, and Tomkins A, eds. Protein-energy malnutrition. London: Edward Arnold, 1992.

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4

Peters, Caroline. Protein-energy malnutrition and the home environment: A study among children in Coast Province, Kenya. Nairobi, Kenya: Food and Nutrition Planning Unit, Ministry of Planning and National Development, 1987.

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5

Elnahas, S. A. Measurement of tonsil size in mild and moderate cases of protein-energy malnutrition. Roehampton: University of Surrey Roehampton, 2004.

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6

Celia, Holland, ed. The impact of helminth infections on human nutrition: Schistosomes and soil-transmitted helminths. London: Taylor & Francis, 1987.

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7

1909-, Bourne Geoffrey H., ed. Nutrition in the Gulf countries, malnutrition and minerals. Basel: Karger, 1987.

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8

Lutangu, Sikota. Report on field work one testing selected areas of Kasempa District: Study of factors leading to low recovery rate from malnutrition in the underfive children who receive high energy protein supplement in Kasempa District. Kabwe, Zambia: Pan African Institute for Development East and Southern Africa, 1998.

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9

Sserunjogi, Louise. Study on the prevalence of protein-energy malnutrition in children 5 years and under in Kiyeyi target area, Tororo District: January-April 1990. [Kampala]: Child Health and Development Centre, Makerere University, 1992.

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10

1928-, Isaacson Robert L., and Jensen Karl F, eds. The Vulnerable brain and environmental risks. New York: Plenum Press, 1994.

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11

Waterlow, J. C. Protein-energy Malnutrition. Hodder Arnold, 1993.

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12

Waterlow, J. C. Protein-Energy Malnutrition. Hodder Education Group, 1999.

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13

Waterlow, JC. Protein-Energy Malnutrition. Smith-Gordon, 2006.

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14

Omran, M. L. Assessment of protein energy malnutrition in older persons. 2000.

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15

Gallagher, Hubert John. The effects of protein-energy malnutrition on mononuclear phagocyte function. 1996.

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16

Hoorweg, Jan C. Protein-Energy Malnutrition and Intellectual Abilities: A Study of Teen-Age Ugandan Children. De Gruyter, Inc., 2019.

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17

Keshav, Satish, and Alexandra Kent. Starvation and malnutrition. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0332.

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Starvation is a state of severe malnutrition due to a reduction in macro- and micronutrient intake. The basis underlying starvation is an imbalance between energy intake and energy expenditure. The commonest cause of starvation is lack of available food, usually due to environmental, social, and economic reasons, although other causes include anorexia nervosa; depression and other psychiatric disorders; coma and disturbance of consciousness; intestinal failure; and mechanical failure of digestion, including poor dentition and intestinal obstruction. Protein energy malnutrition is usually seen in developing countries. This chapter discusses starvation and malnutrition, focusing on their etiology, symptoms, demographics, natural history, complications, diagnosis, treatment, and prognosis.

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18

Kumar, Navneet, Heather Henderson, Beverly D. Cameron, and Peter A. McCullough. Malnutrition, obesity, and undernutrition in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0106_update_001.

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Both overnutrition resulting in obesity and undernutrition leading to protein energy wasting contribute to chronic kidney disease-related morbidity and adverse outcomes. Early in the course of chronic kidney disease, goals should be set for a healthy body weight and lifelong efforts should be encouraged to attain and keep this goal. For patients with progressive chronic kidney disease, the development of weight loss and protein energy wasting is an ominous sign and is a clinical signal for a myriad of adverse catabolic processes that have been associated with poor outcomes including hospitalization and death, particularly for those with end-stage renal disease. Renal nutrition consultation at all stages of chronic kidney disease with frequent visits and education and counselling is needed to intercede early in both ends of the nutrition continuum in patients with chronic kidney disease.

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19

(Editor), R. L. Isaacson, and K. F. Jensen (Editor), eds. The Vulnerable Brain and Environmental Risks. Springer, 1994.

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20

Bender, David A. 6. Under-nutrition. Oxford University Press, 2014. http://dx.doi.org/10.1093/actrade/9780199681921.003.0006.

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Severe under-nutrition is generally associated with developing countries where food is in short supply, affecting some 162 million people world-wide, but malnutrition is also seen in about 2 per cent of the population of developed countries. ‘Under-nutrition’ highlights the three conditions classified as protein-energy malnutrition: marasmus, which affects adults and children; kwashiorkor, which affects young children; and cachexia, which is associated with advanced cancer and other chronic diseases, and involves increased metabolic rate as well as reduced food intake. Malnutrition leads to impaired immune responses, predisposing to infection, and muscle loss resulting in increased fatiguability, inability to work, and falls.

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21

Puntis, John. Nutritional assessment. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0003.

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Routine assessment of nutritional status should be part of normal practice when seeing any patient. The purpose is to document objective nutritional parameters (e.g. anthropometry), identify nutritional deficiencies, and establish nutritional needs. Protein–energy malnutrition has many adverse consequences including growth failure (identified by reference to standard growth charts). Worldwide, malnutrition contributes to a third of deaths in children under 5 years of age, and one in nine people don’t have enough food to lead an active and healthy life. In developed countries, malnutrition complicates both acute and chronic illness with negative effects on outcomes. In clinical practice, a useful approach to nutritional assessment is to consider three elements: ‘what you are’ (i.e. body habitus—underweight for height; short for age; etc.), ‘what you can do’ (functional activity), and ‘what you eat’ (current nutritional intake).

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22

Misulis, Karl E., and E. Lee Murray. Nutritional Deficiencies and Toxicities. Edited by Karl E. Misulis and E. Lee Murray. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190259419.003.0029.

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Nutritional disorders are often encountered in hospital neurology practice, especially deficiencies of vitamins B1 and B12. Medical conditions can predispose to nutritional disorders. This chapter discusses the presentation, diagnosis, and management of B12 deficiency, B1 deficiency, protein-energy malnutrition, folate deficiency especially in the context of pregnancy, B6 deficiency, B6 toxicity, copper deficiency, and vitamin D deficiency. Wernicke encephalopathy and Korsakoff syndrome are also discussed.

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23

Egreteau, Pierre-Yves, and Jean-Michel Boles. Assessing nutritional status in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0204.

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Decreased nutrient intake, increased body requirements, and/or altered nutrient utilization are frequently combined in critically-ill patients. The initial nutritional status and the extent of the disease-related catabolism are the main risk factors for nutrition- related complications. Many complications are related to protein energy malnutrition, which is frequent in the ICU setting. Assessing nutritional status pursues several different goals. Nutritional assessment is required for patients presenting with clinical evidence of malnutrition, with chronic diseases, with acute conditions accompanied by a high catabolic rate, and elderly patients. Recording the patient’s history, nutrient intake, and physical examination, and subjective global assessment allows classification of nutritional status. All the traditional markers of malnutrition, anthropometric measurements and plasma proteins, lose their specificity in the sick adult as each may be affected by a number of non-nutritional factors. Muscle function evaluated by hand-grip strength in cooperative patients and serum albumin provide an objective risk assessment. Several nutritional indices have been validated in specific groups of patients to identify patients at risk of nutritionally-mediated complications and, therefore, the need for nutritional support. A strong suspicion remains the best way of uncovering potentially harmful nutritional deficiencies.

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24

Wise, Matt, and Paul Frost. Nutritional support in the critically ill. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0334.

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Major injury evokes a constellation of reproducible hormonal, metabolic, and haemodynamic responses which are collectively termed ‘the adaptive stress response’. The purpose of the adaptive stress response is to facilitate tissue repair and restore normal homeostasis. If critical illness is prolonged, the adaptive stress response may become maladaptive, in essence exerting a parasitic effect leaching away structural proteins and impairing host immunity. Primarily therapy should be directed towards the underlying illness, as nutritional support per se will not reverse the stress response and its sequelae. Nonetheless, adequate nutritional support in the early stages of critical illness may attenuate protein catabolism and its adverse effects. This chapter covers nutritional assessment; detection of malnutrition; energy and protein requirements; monitoring the effectiveness of nutritional replacements; nutritional delivery; complications; and refeeding syndrome.

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25

Nutritional Problems and Education: Selected Topics (World Review of Nutrition and Dietetics). S. Karger AG (Switzerland), 1986.

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