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Journal articles on the topic 'Protein fibrillation'

Author: Grafiati

Published: 1 April 2023

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1

Gorensek-Benitez, Annelise H., Bryan Kirk, and Jeffrey K. Myers. "Protein Fibrillation under Crowded Conditions." Biomolecules 12, no. 7 (2022): 950. http://dx.doi.org/10.3390/biom12070950.

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Protein amyloid fibrils have widespread implications for human health. Over the last twenty years, fibrillation has been studied using a variety of crowding agents to mimic the packed interior of cells or to probe the mechanisms and pathways of the process. We tabulate and review these results by considering three classes of crowding agent: synthetic polymers, osmolytes and other small molecules, and globular proteins. While some patterns are observable for certain crowding agents, the results are highly variable and often depend on the specific pairing of crowder and fibrillating protein.

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2

St. Rammos, Kyriakos, George J. Koullias, Moustafa O. Hassan, et al. "Low Preoperative HSP70 Atrial Myocardial Levels Correlate Significantly with High Incidence of Postoperative Atrial Fibrillation after Cardiac Surgery." Cardiovascular Surgery 10, no. 3 (2002): 228–32. http://dx.doi.org/10.1177/096721090201000309.

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Purpose of the study. Atrial fibrillation after cardiac surgery is still a frequent encountered complication and has been associated with increased hospital length of stay and numerous postoperative complications. The pathogenesis of atrial fibrillation involves an overall sequence of perioperative events, collectively termed as ischemia-reperfusion injury. Heat-shock proteins have been found to provide increased protection during ischemia-reperfusion as well as increased postischemic cardiac functional recovery. We sought to determine whether preoperative atrial heat shock levels were correla

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3

Ohkubo, Kimie, Ichiro Watanabe, Yasuo Okumura, et al. "High-Sensitivity C-Reactive Protein: A Novel Predictor of Recurrence of Atrial Fibrillation After Initial Catheter Ablation of Paroxysmal Atrial Fibrillation." Journal of Nihon University Medical Association 75, no. 3 (2016): 118–22. http://dx.doi.org/10.4264/numa.75.3_118.

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4

Tachibana, Hideki, Shunta Kubomura, Kaoru Shinohara, and Ryohei Kono. "3P081 Seeded-Fibrillation of Lysozyme Disulfide-Variant Proteins(Protein: Property,The 48th Annual Meeting of the Biophysical Society of Japan)." Seibutsu Butsuri 50, supplement2 (2010): S159. http://dx.doi.org/10.2142/biophys.50.s159_1.

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5

Dev, Shubhda. "MOLECULAR DOCKING ANALYSIS OF NATRIURETIC PEPTIDE RECEPTOR-C TOWARDS THE DESIGN OF POTENTIAL ATRIAL FIBRILLATION INHIBITORS." Journal of Medical Pharmaceutical And Allied Sciences 9, no. 5 (2020): 2595–600. http://dx.doi.org/10.22270/jmpas.v9i5.974.

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Atrial fibrillation (AF) stands the most widely recognized kind of clinical arrhythmia. Right now accessible anti-Atrial Fibrillation drugs are restricted by just moderate adequacy and an unfavorable safety profile. There is a perceived requirement for enhanced antiarrhythmic agents including activities that are specific for the fibrillating atrium. Therefore, it is of interest to design an appropriate medication for the disease Atrial Fibrillation using Molecular Docking techniques through protein-ligand interaction analysis. Hence, we document the Molecular docking analysis of natriuretic pe

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6

Sata, Naoyuki, Naokazu Hamada, Takashi Horinouchi, et al. "C-reactive Protein and Atrial Fibrillation." Japanese Heart Journal 45, no. 3 (2004): 441–45. http://dx.doi.org/10.1536/jhj.45.441.

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7

Huang, Kun, Jian Dong, Nelson B. Phillips, Paul R. Carey, and Michael A. Weiss. "Proinsulin Is Refractory to Protein Fibrillation." Journal of Biological Chemistry 280, no. 51 (2005): 42345–55. http://dx.doi.org/10.1074/jbc.m507110200.

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8

Linse, S., C. Cabaleiro-Lago, W. F. Xue, et al. "Nucleation of protein fibrillation by nanoparticles." Proceedings of the National Academy of Sciences 104, no. 21 (2007): 8691–96. http://dx.doi.org/10.1073/pnas.0701250104.

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9

Hernández Madrid, Antonio, and Concepción Moro. "Atrial Fibrillation and C-Reactive Protein." Journal of the American College of Cardiology 49, no. 15 (2007): 1649–50. http://dx.doi.org/10.1016/j.jacc.2007.02.009.

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10

Wood, Mark A., Kenneth A. Ellenbogen, and Bruce S. Stambler. "Atrial fibrillation from liquid protein diet." American Heart Journal 127, no. 6 (1994): 1667–68. http://dx.doi.org/10.1016/0002-8703(94)90422-7.

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11

Colvin, V. L., and K. M. Kulinowski. "Nanoparticles as catalysts for protein fibrillation." Proceedings of the National Academy of Sciences 104, no. 21 (2007): 8679–80. http://dx.doi.org/10.1073/pnas.0703194104.

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12

Galea, Roberto, Maria Teresa Cardillo, Annalisa Caroli, et al. "Inflammation and C-Reactive Protein in Atrial Fibrillation: Cause or Effect?" Texas Heart Institute Journal 41, no. 5 (2014): 461–68. http://dx.doi.org/10.14503/thij-13-3466.

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Atrial fibrillation is associated with substantial morbidity and mortality rates. The incompletely understood pathogenesis of this cardiac dysrhythmia makes it difficult to improve approaches to primary and secondary prevention. Evidence has accumulated in regard to a relationship between inflammation and atrial fibrillation. Investigators have correlated the dysrhythmia with myocarditis, pericardiotomy, and C-reactive protein levels, suggesting that inflammation causes atrial fibrillation or participates in its onset and continuation. Conversely, other investigators suggest that atrial fibril

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13

Namsrai, Javkhlantugs, Ganchimeg Lkhamsuren, Kazuyoshi Ueda, and Akira Naito. "3P022 Structure and Interactions in Fibrillation of Human Calcitonin Hormone(01A. Protein: Structure,Poster)." Seibutsu Butsuri 53, supplement1-2 (2013): S215. http://dx.doi.org/10.2142/biophys.53.s215_4.

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14

Badran, Hala Mahfouz, and Magdi Elsayed Mahfouz. "Cytotoxin-associated gene-A bearing strains of Helicobacter pylori and atrial fibrillation due to ischemic origin: is there a link?" European Journal of Cardiovascular Prevention & Rehabilitation 14, no. 4 (2007): 518–20. http://dx.doi.org/10.1097/hjr.0b013e328011a2a0.

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Background Helicobacter pylori CagA strains could increase the risk for atrial fibrillation in patients with coronary artery disease Methods Serological status for H. pylori CagA using enzyme-linked immunosorbent assay, C-reactive protein, total leucocytic count and atrial size were determined in 185 coronary artery disease patients (with and without atrial fibrillation) and 80 healthy subjects (control). Results CagA strain showed a higher prevalence in the atrial fibrillation group. Atrial dimension and C-reactive protein (independent predictors of atrial fibrillation) were significantly inc

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15

Mari, Eleonora, Caterina Ricci, Silvia Pieraccini, Francesco Spinozzi, Paolo Mariani, and Maria Grazia Ortore. "Trehalose Effect on The Aggregation of Model Proteins into Amyloid Fibrils." Life 10, no. 5 (2020): 60. http://dx.doi.org/10.3390/life10050060.

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Protein aggregation into amyloid fibrils is a phenomenon that attracts attention from a wide and composite part of the scientific community. Indeed, the presence of mature fibrils is associated with several neurodegenerative diseases, and in addition these supramolecular aggregates are considered promising self-assembling nanomaterials. In this framework, investigation on the effect of cosolutes on protein propensity to aggregate into fibrils is receiving growing interest, and new insights on this aspect might represent valuable steps towards comprehension of highly complex biological processe

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16

Rho, Hoon Suk, Henk-Willem Veltkamp, Alexander Thomas Hanke, et al. "Systematic Investigation of Insulin Fibrillation on a Chip." Molecules 25, no. 6 (2020): 1380. http://dx.doi.org/10.3390/molecules25061380.

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A microfluidic protein aggregation device (microPAD) that allows the user to perform a series of protein incubations with various concentrations of two reagents is demonstrated. The microfluidic device consists of 64 incubation chambers to perform individual incubations of the protein at 64 specific conditions. Parallel processes of metering reagents, stepwise concentration gradient generation, and mixing are achieved simultaneously by pneumatic valves. Fibrillation of bovine insulin was selected to test the device. The effect of insulin and sodium chloride (NaCl) concentration on the formatio

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17

Macchi, Francesca, Maike Eisenkolb, Hans Kiefer, and Daniel E. Otzen. "The Effect of Osmolytes on Protein Fibrillation." International Journal of Molecular Sciences 13, no. 3 (2012): 3801–19. http://dx.doi.org/10.3390/ijms13033801.

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18

DERNELLIS, John, and Maria PANARETOU. "C-reactive protein and paroxysmal atrial fibrillation." Acta Cardiologica 56, no. 6 (2001): 375–80. http://dx.doi.org/10.2143/ac.56.6.2005701.

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19

Jeppesen, Martin D., Peter Westh, and Daniel E. Otzen. "The role of protonation in protein fibrillation." FEBS Letters 584, no. 4 (2010): 780–84. http://dx.doi.org/10.1016/j.febslet.2010.01.002.

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20

Ellinor, Patrick T., Adrian Low, Kristen K. Patton, Marisa A. Shea, and Calum A. MacRae. "C-Reactive Protein in Lone Atrial Fibrillation." American Journal of Cardiology 97, no. 9 (2006): 1346–50. http://dx.doi.org/10.1016/j.amjcard.2005.11.052.

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21

Pagano, Rodrigo S., Máximo López Medus, Gabriela E. Gómez, et al. "Protein Fibrillation Lag Times During Kinetic Inhibition." Biophysical Journal 107, no. 3 (2014): 711–20. http://dx.doi.org/10.1016/j.bpj.2014.06.029.

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22

Heegaard, Peter M. H., Ulrik Boas, and Daniel E. Otzen. "Dendrimer Effects on Peptide and Protein Fibrillation." Macromolecular Bioscience 7, no. 8 (2007): 1047–59. http://dx.doi.org/10.1002/mabi.200700051.

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23

Landreh, Michael, Jan Johansson, Anna Rising, Jenny Presto, and Hans Jörnvall. "Control of amyloid assembly by autoregulation." Biochemical Journal 447, no. 2 (2012): 185–92. http://dx.doi.org/10.1042/bj20120919.

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The assembly of proteins into amyloid fibrils can be an element of both protein aggregation diseases and a functional unit in healthy biological pathways. In both cases, it must be kept under tight control to prevent undesired aggregation. In normophysiology, proteins can self-chaperone amyloidogenic segments by restricting their conformational flexibility in an overall stabilizing protein fold. However, some aggregation-prone segments cannot be controlled in this manner and require additional regulatory elements to limit fibrillation. The present review summarizes different molecular mechanis

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24

Barros, Heloise R., Maria Kokkinopoulou, Izabel C. Riegel-Vidotti, Katharina Landfester та Héloïse Thérien-Aubin. "Gold nanocolloid–protein interactions and their impact on β-sheet amyloid fibril formation". RSC Advances 8, № 2 (2018): 980–86. http://dx.doi.org/10.1039/c7ra11219j.

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Formation of amyloid protein fibrils is associated with degenerative diseases. Here, the interaction mechanism between globular and fibrillar proteins with AuNPs were investigated in order to potentially control and reverse the fibrillation process.

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25

Li, Qiaoqiao, Yingyu Lai, Xiaoyan Gao, et al. "Involvement of plasminogen activator inhibitor-1 and its related molecules in atrial fibrosis in patients with atrial fibrillation." PeerJ 9 (June 2, 2021): e11488. http://dx.doi.org/10.7717/peerj.11488.

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Atrial fibrillation is the most common form of cardiac arrhythmia. Atrial fibrosis is a significant feature of atrial fibrillation though its mechanism is not well understood. We searched the Gene Expression Omnibus database to compare mRNA expression patterns between atrial fibrillation and sinus rhythm samples; one hundred and forty eight differentially expressed genes were identified. Most of these genes were significantly enriched in the extracellular matrix organization process and collagen-activated tyrosine kinase receptor signaling pathway. To screen hub genes involved in atrial fibros

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26

Tykhomyrov, A. A., Yu S. Kushnir, V. S. Nedzvetsky, T. V. Grinenko, and O. V. Kuryata. "Citicoline affects serum angiostatin and neurospecific protein levels in patients with atrial fibrillation and ischemic stroke." Ukrainian Biochemical Journal 91, no. 5 (2019): 34–45. http://dx.doi.org/10.15407/ubj91.05.034.

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27

Ohkubo, Kimie, Ichiro Watanabe, Yasuo Okumura, et al. "Usefulness of High Sensitivity C-Reactive Protein in Predicting Recurrence of Atrial Fibrillation after Electrical Cardioversion." Journal of Nihon University Medical Association 74, no. 5 (2015): 233–37. http://dx.doi.org/10.4264/numa.74.5_233.

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28

Borges, Jordana I., Malka S. Suster, and Anastasios Lymperopoulos. "Cardiac RGS Proteins in Human Heart Failure and Atrial Fibrillation: Focus on RGS4." International Journal of Molecular Sciences 24, no. 7 (2023): 6136. http://dx.doi.org/10.3390/ijms24076136.

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The regulator of G protein signaling (RGS) proteins are crucial for the termination of G protein signals elicited by G protein-coupled receptors (GPCRs). This superfamily of cell membrane receptors, by far the largest and most versatile in mammals, including humans, play pivotal roles in the regulation of cardiac function and homeostasis. Perturbations in both the activation and termination of their G protein-mediated signaling underlie numerous heart pathologies, including heart failure (HF) and atrial fibrillation (AFib). Therefore, RGS proteins play important roles in the pathophysiology of

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29

Mahmoudi, Morteza, Hamid R. Kalhor, Sophie Laurent, and Iseult Lynch. "Protein fibrillation and nanoparticle interactions: opportunities and challenges." Nanoscale 5, no. 7 (2013): 2570. http://dx.doi.org/10.1039/c3nr33193h.

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30

Kashchiev, Dimo. "Kinetics of protein fibrillation controlled by fibril elongation." Proteins: Structure, Function, and Bioinformatics 82, no. 9 (2014): 2229–39. http://dx.doi.org/10.1002/prot.24586.

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31

Li, Jun, Junjie Xiao, Dandan Liang, et al. "Inhibition of mitochondrial translocator protein prevents atrial fibrillation." European Journal of Pharmacology 632, no. 1-3 (2010): 60–64. http://dx.doi.org/10.1016/j.ejphar.2010.01.014.

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32

Vasilets, L. M., A. V. Agafonov, O. V. Khlynova, et al. "Prediction of atrial fibrillation according to levels of serum markers of inflammation during arterial hypertension." Kazan medical journal 93, no. 4 (2012): 642–46. http://dx.doi.org/10.17816/kmj1561.

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Aim. To study the parameters of inflammation during atrial fibrillation in patients with arterial hypertension and to determine the possibility of their predictive significance in relation to development of the arrhythmia. Methods. Examined were 97 individuals with arterial hypertension, mean age 50.53±8.10 years. Formed were two groups: patients with arterial hypertension without the arrhythmia and patients with atrial fibrillation on the background of arterial hypertension. Among the examined patients with atrial fibrillation revealed were patients with persistent or recurrent persistent for

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33

van Wijk, Stan W., Wei Su, Leonoor F. J. M. Wijdeveld, Kennedy S. Ramos, and Bianca J. J. M. Brundel. "Cytoskeletal Protein Variants Driving Atrial Fibrillation: Potential Mechanisms of Action." Cells 11, no. 3 (2022): 416. http://dx.doi.org/10.3390/cells11030416.

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The most common clinical tachyarrhythmia, atrial fibrillation (AF), is present in 1–2% of the population. Although common risk factors, including hypertension, diabetes, and obesity, frequently underlie AF onset, it has been recognized that in 15% of the AF population, AF is familial. In these families, genome and exome sequencing techniques identified variants in the non-coding genome (i.e., variant regulatory elements), genes encoding ion channels, as well as genes encoding cytoskeletal (-associated) proteins. Cytoskeletal protein variants include variants in desmin, lamin A/C, titin, myosin

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34

Savelieva, Irina, and A. John Camm. "A New Biomarker in Atrial Fibrillation: Monocyte Chemoattractant Protein-1-Induced Protein." Cardiology 144, no. 3-4 (2019): 122–24. http://dx.doi.org/10.1159/000502253.

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35

Psychari, Stavroula N., Dionysios Tsoukalas, Dimitrios Varvarousis, et al. "Opposite relations of epicardial adipose tissue to left atrial size in paroxysmal and permanent atrial fibrillation." SAGE Open Medicine 6 (January 2018): 205031211879990. http://dx.doi.org/10.1177/2050312118799908.

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Objectives: Atrial fibrillation has been associated with obesity in epidemiological studies. Epicardial adipose tissue is an ectopic fat depot in the proximity of atria, with endocrine and inflammatory properties that is implicated in the pathophysiology of atrial fibrillation. Inflammation also has a role in atrial arrhythmogenesis. The aim of this study was to investigate the potential relations of epicardial adipose tissue to left atrial size and to adiponectin and the pro-inflammatory mediators, high-sensitivity C-reactive protein, and interleukin-6 in paroxysmal and permanent atrial fibri

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36

Chen, Yongqing, Fangju Su, Juanping Han, Piqi Jiao, and Wenyun Guo. "Expression of Rho Kinase and Its Mechanism in the Left Atrial Appendage in Patients with Atrial Fibrillation." Heart Surgery Forum 21, no. 1 (2018): 044. http://dx.doi.org/10.1532/hsf.1851.

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Aim: To study the expression of Rho kinase (Rho associated coil forming protein kinase-1, ROCK-1) and its substrate myosin phosphatase target subunit 1 (myosin phosphatase target subunit-1, MYPT-1), connexin 40 (Cx40) and connexin 43 (Cx43) in the left atrial appendage of patients with atrial fibrillation, and explore the role of ROCK signaling pathway in patients with atrial fibrillation and its underlying mechanism. Methods: 40 patients undergoing open heart surgery were divided into two groups; atrial fibrillation group (AF group) and sinus rhythm group (SR group). About 100 mg of left atri

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37

Panigrahi, Gitanjali P., Ankita R. Rane, Sirisha L. Vavilala та Sinjan Choudhary. "Deciphering the anti-Parkinson’s activity of sulphated polysaccharides from Chlamydomonas reinhardtii on the α-Synuclein mutants A30P, A53T, E46K, E57K and E35K". Journal of Biochemistry 166, № 6 (2019): 463–74. http://dx.doi.org/10.1093/jb/mvz064.

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Abstract Parkinsonism-linked mutations in alanine and glutamic acid residues of the pre-synaptic protein α-Synuclein (α-Syn) affect specific tertiary interactions essential for stability of the native state and make it prone to more aggregation. Many of the currently available drugs used for the treatment of Parkinson’s disease (PD) are not very effective and are associated with multiple side effects. Recently, marine algae have been reported to have sulphated polysaccharides which offers multiple pharmaceutical properties. With this background, we have isolated sulphated polysaccharides from

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38

Pala, Arda Aybars. "C-reactive protein/albumin ratio in predicting atrial fibrillation after coronary artery bypass grafting." Cardiovascular Surgery and Interventions 9, no. 2 (2022): 66–72. http://dx.doi.org/10.5606/e-cvsi.2022.1233.

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Objectives: In the present study, the purpose was to investigate the usability of the preoperative C-reactive protein/albumin ratio as a predictor of the development of postoperative atrial fibrillation in patients who undergo coronary artery bypass grafting. Patients and methods: A total of 336 patients (228 males, 108 females; mean age: 58.1±8.5 years; range 35 to 88 years) who underwent isolated coronary artery bypass grafting with cardiopulmonary bypass between January 2019 and January 2021 were reviewed in the single-center, retrospective study. Those with postoperative sinus rhythm were

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39

Milošević, Jelica, Radivoje Prodanović, and Natalija Polović. "On the Protein Fibrillation Pathway: Oligomer Intermediates Detection Using ATR-FTIR Spectroscopy." Molecules 26, no. 4 (2021): 970. http://dx.doi.org/10.3390/molecules26040970.

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Oligomeric intermediates on the pathway of amyloid fibrillation are suspected as the main cytotoxins responsible for amyloid-related pathogenicity. As they appear to be a part of the lag phase of amyloid fibrillation when analyzed using standard methods such as Thioflavin T (ThT) fluorescence, a more sensitive method is needed for their detection. Here we apply Fourier transform infrared spectroscopy (FTIR) in attenuated total reflectance (ATR) mode for fast and cheap analysis of destabilized hen-egg-white lysozyme solution and detection of oligomer intermediates of amyloid fibrillation. Stand

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40

Bai, Juan, Chongyu Ren, Wei Hao, Rui Wang, and Ji-Min Cao. "Chemical sympathetic denervation, suppression of myocardial transient outward potassium current, and ventricular fibrillation in the rat." Canadian Journal of Physiology and Pharmacology 86, no. 10 (2008): 700–709. http://dx.doi.org/10.1139/y08-075.

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Sympathetic denervation is frequently observed in heart disease. To investigate the linkage of sympathetic denervation and cardiac arrhythmia, we developed a rat model of chemical sympathectomy by subcutaneous injections of 6-hydroxydopamine (6-OHDA). Cardiac sympathetic innervation was visualized by means of a glyoxylic catecholaminergic histofluorescence method. Transient outward current (Ito) of ventricular myocytes was recorded with the whole-cell configuration of the patch clamp technique. We observed that sympathectomy (i) decreased cardiac sympathetic nerve density and norepinephrine le

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41

SO, Masatomo, Hisashi YAGI, and Yuji GOTO. "Ultrasonication-Induced Acceleration of Amyloid Fibrillation and Protein Crystallization." Seibutsu Butsuri 54, no. 6 (2014): 297–302. http://dx.doi.org/10.2142/biophys.54.297.

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42

Fändrich, M., and C. M. Dobson. "Amyloid-type fibrillation of the all alpha protein myoglobin." Biochemical Society Transactions 28, no. 5 (2000): A408. http://dx.doi.org/10.1042/bst028a408c.

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43

Cabaleiro-Lago, Celia, Fiona Quinlan-Pluck, Iseult Lynch та ін. "Inhibition of Amyloid β Protein Fibrillation by Polymeric Nanoparticles". Journal of the American Chemical Society 130, № 46 (2008): 15437–43. http://dx.doi.org/10.1021/ja8041806.

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44

Korantzopoulos, Panagiotis, Dimitrios Galaris, Dimitrios Papaioannides, and Stelianos Kokkoris. "C-reactive protein and oxidative stress in atrial fibrillation." International Journal of Cardiology 88, no. 1 (2003): 103–4. http://dx.doi.org/10.1016/s0167-5273(02)00386-8.

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45

Boos, Christopher J., and Gregory Y. H. Lip. "C-Reactive Protein and Lone Atrial Fibrillation: Potential Confounders." American Journal of Cardiology 98, no. 7 (2006): 991–92. http://dx.doi.org/10.1016/j.amjcard.2006.05.005.

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46

Marott, Sarah C. W., Børge G. Nordestgaard, Jeppe Zacho, et al. "Does Elevated C-Reactive Protein Increase Atrial Fibrillation Risk?" Journal of the American College of Cardiology 56, no. 10 (2010): 789–95. http://dx.doi.org/10.1016/j.jacc.2010.02.066.

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47

Volpatti, Lisa R., Ulyana Shimanovich, Francesco Simone Ruggeri, et al. "Micro- and nanoscale hierarchical structure of core–shell protein microgels." Journal of Materials Chemistry B 4, no. 48 (2016): 7989–99. http://dx.doi.org/10.1039/c6tb02683d.

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48

Akbarian, Mohsen, Ehsan Rezaie, Fatemeh Farjadian, et al. "Inhibitory effect of coumarin and its analogs on insulin fibrillation /cytotoxicity is depend on oligomerization states of the protein." RSC Advances 10, no. 63 (2020): 38260–74. http://dx.doi.org/10.1039/d0ra07710k.

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The effect of the applied compounds on insulin fibrillation at two pHs. By and large, the compounds through changing the oligomerization states and altering structure integrity of insulin can govern the fibrillation process.

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Meng, Xiaoyun, Larissa A. Munishkina, Anthony L. Fink та Vladimir N. Uversky. "Effects of Various Flavonoids on theα-Synuclein Fibrillation Process". Parkinson's Disease 2010 (2010): 1–16. http://dx.doi.org/10.4061/2010/650794.

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α-Synuclein aggregation and fibrillation are closely associated with the formation of Lewy bodies in neurons and are implicated in the causative pathogenesis of Parkinson's disease and other synucleinopathies. Currently, there is no approved therapeutic agent directed toward preventing the protein aggregation, which has been recently shown to have a key role in the cytotoxic nature of amyloidogenic proteins. Flavonoids, known as plant pigments, belong to a broad family of polyphenolic compounds. Over 4,000 flavonoids have been identified from various plants and foodstuffs derived from plants a

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50

Basu, Aalok, Sagar Bhayye, Sonia Kundu, Aatryee Das, and Arup Mukherjee. "Andrographolide inhibits human serum albumin fibril formations through site-specific molecular interactions." RSC Advances 8, no. 54 (2018): 30717–24. http://dx.doi.org/10.1039/c8ra04637a.

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