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1

Knorpp, Carina. Plant mitochondrial biogenesis: Protein sorting, import, processing and degradation. Department of Biochemistry, Arrhenius Laboratories for Natural Sciences, Stockholm University, 1995.

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2

O'Regan, Seamus G. Cytosolic factors involved in the import of precursor proteins by plant mitochondria. University College Dublin, 1996.

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3

Sirk, Daniel. The effects of sub-lethal A[Beta](25-25) on mitochondrial protein import and degradation in differentiated PC12 cells. National Library of Canada, 2002.

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4

Cleary, Suzanne Patricia. Protein targeting to mitochondria and chloroplasts. typescript, 2000.

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5

service), ScienceDirect (Online, ed. Mitochondrial function: Mitochondrial protein kinases, protein phosphatases and mitochondrial diseases. Academic Press/Elsevier, 2009.

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6

Wadia, J. S. Changes in mitochondrial protein import during apoptosis. 2002.

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7

Phan, Nam. Stress induced modulation of mitochondrial protein import in differentiated PC12 cells: The role of Tom20. 2006.

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8

Protein Targeting to Mitochondria. Elsevier, 1996. http://dx.doi.org/10.1016/s1569-2558(09)x6001-7.

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9

Hartl, F. U. Protein Targeting to Mitochondria. Elsevier Science & Technology Books, 1997.

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10

Zimmermann, Richard, and Sven Lang, eds. Mechanisms of ER Protein Import. MDPI, 2022. http://dx.doi.org/10.3390/books978-3-0365-4093-1.

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11

Hartl, F. U. Protein Targeting to Mitochondria (Advances in Molecular and Cell Biology). Elsevier Science, 1996.

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12

The function of the Qcr7 protein of ubiquinol: Cytochrome c oxidoreductase in mitochondria of Saccharomyces cerevisiae. National Library of Canada, 2000.

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13

Kim, Chanhong. Substrate-dependent and organ-specific chloroplast protein import in planta. 2004.

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14

Caliebe, Alexander. Neue Komponenten des Protein-Import-Apparates aus Chloroplasten der Erbse (Pisum sativum L.). 1998.

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15

Clark, Steven Edward. Determinants for chloroplast import and processing of the light-harvesting chlorophyll a/b-binding protein precursor. 1991.

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16

Fernandez, Michael David. Import and maturation of spinach acyl carrier protein: Activation by a chloroplast holoACP synthase and coenzyme A. 1991.

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17

Lübeck, Jens. Topologie und Importverhalten von IEP110, einer neuen Komponente des Protein-Import-Apparates der inneren Chloroplasten-Hüllmembran aus Erbse (Pisum sativum L.). 1996.

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18

Streijger, Femke, Ward T. Plunet, and Wolfram Tetzlaff. Ketogenic Diet and Ketones for the Treatment of Traumatic Brain and Spinal Cord Injury. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0016.

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Ketogenic diets (KD)—high in fat, adequate in protein, and very low in carbohydrates—were developed almost a century ago and are still used clinically for drug-resistant epilepsy and some rare metabolic disorders. Possible new indications for cancers, diabetes, obesity, and neurodegenerative disorders are being trialed in humans based on a growing body of preclinical data showing efficacy. However the underlying mechanisms of KD remain incompletely understood. This chapter focuses on the neuroprotective effects of KD after spinal cord injury (SCI) and traumatic brain injury (TBI), and discusse
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19

van Geel, Björn M., Marc Engelen, and Stephan Kemp. X-linked Adrenoleukodystrophy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0061.

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X-linked adrenoleukodystrophy (X-ALD) is the most frequent peroxisomal disorder. Hallmarks are increased levels of plasma very long-chain fatty acids (VLCFA), mutations in the ABCD1 gene, impaired function of ALD-protein and, consequently, decreased import of VLCFA-CoA esters in peroxisomes and VLCFA beta-oxidation. Cerebral demyelination and axonal degeneration of the spinal cord are the main causes of neurological deficits. Endocrine dysfunction, particularly adrenocortical insufficiency, is very frequent. Based upon the age of onset of symptoms and the organs most severely affected, several
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