Relevant bibliographies by topics / Proteïna AFP

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Proteïna AFP'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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Journal articles on the topic "Proteïna AFP"

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Lin, Min, and Michael L. Cleary. "MLL Translocation Partners AF4, AF5q31, and ENL Associate in a Higher Order Protein Complex with CDK9 and Cyclin T1." Blood 106, no. 11 (2005): 99. http://dx.doi.org/10.1182/blood.v106.11.99.99.

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Abstract The Mixed Lineage Leukemia (MLL) gene is frequently involved in chromosomal translocations that cause acute leukemia. More than 40 different genes have been identified as MLL translocation partners, with the expression of corresponding MLL fusion proteins. The MLL protein has histone methyltransferase activity and is required for embryonic development and hematopoiesis. Several proteins have been demonstrated to associate with MLL in a macromolecular complex, which is believed to have chromatin remodeling function. However, the C-terminal SET domain of MLL, which carries the histone m
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Yokoyama, Akihiko, and Hiroshi Okuda. "The Molecular Mechanism of Transcriptional Activation By MLL-AEP Fusion Proteins." Blood 126, no. 23 (2015): 2435. http://dx.doi.org/10.1182/blood.v126.23.2435.2435.

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Abstract Chromosomal translocations generate a variety of mixed lineage leukemia (MLL) fusion genes, which cause aggressive leukemia. Although >70 different fusion partners have been identified, the majority of the cases are caused by the chimeric genes of MLL and a component of the AEP co-activator complex (hereafter referred to as AEP), which comprises of AF4 family proteins (e.g. AF4, AF5Q31), ENL family proteins (e.g. ENL, AF9), and the P-TEFb elongation factor. MLL-AEP fusion proteins constitutively activate their target genes by recruiting AEP components to their target chromatin, whe
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Lin, Shan, Anetta Ptasinska, Salam Adli Assi, et al. "The Transcriptome Heterogeneity of MLL-Fusion ALL Is Driven By Fusion Partners Via Distinct Chromatin Binding." Blood 128, no. 22 (2016): 576. http://dx.doi.org/10.1182/blood.v128.22.576.576.

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Abstract Chromosome rearrangements involving the Mixed Lineage Leukemia (MLL) gene on chromosome 11q23 account for 15-20% of acute lymphoid leukemia (ALL) and confer poor prognosis. Such rearrangements generate the MLL-fusion proteins, in which the N-terminus of the MLL protein mediating chromatin interactions is fused with one of more than 70 different partner proteins. Proteins that are frequently involved in MLL translocations, including AF4, ENL, AF9 and AF10, were identified as components of the super elongation complex (SEC) or DOT1L complex (an H3K79 histone methyltransferase). Based on
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Bushweller, John H., Benjamin Leach, Ming-jin Chang, et al. "The Role of the Intrinsically Disordered and Multifunctional AF9 C-Terminal Domain in MLL-AF9 Leukemia,." Blood 118, no. 21 (2011): 3464. http://dx.doi.org/10.1182/blood.v118.21.3464.3464.

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Abstract Abstract 3464 Mixed Lineage Leukemia (MLL) fusion proteins disrupt HOX gene regulation through recruitment of transcriptional elongation factors, leading to acute leukemia. AF9 is one of the most common MLL fusion partners and has roles in normal regulation of HOX genes. AF9 has also been shown to interact with multiple transcriptional regulators, including AF4 family proteins which recruit and activate P-TEFb, suggesting a pivotal role in regulation of transcriptional elongation at HOX loci. The mechanism by which AF9 regulates normal transcription and contributes to dysregulated tra
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Lee, Young-Sun, Eunjung Ko, Eileen L. Yoon, et al. "Multiplexed Proteomic Approach for Identification of Serum Biomarkers in Hepatocellular Carcinoma Patients with Normal AFP." Journal of Clinical Medicine 9, no. 2 (2020): 323. http://dx.doi.org/10.3390/jcm9020323.

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Alpha fetoprotein (AFP) has been used as a serologic indicator of hepatocellular carcinoma (HCC). We aimed to identify an HCC-specific serum biomarker for diagnosis using a multiplexed proteomic technique in HCC patients with normal AFP levels. A total of 152 patients were included from Guro Hospital, Korea University. Among 267 identified proteins, 28 and 86 proteins showed at least a two-fold elevation or reduction in expression, respectively. Multiple reaction monitoring (MRM) analysis of 41 proteins revealed 10 proteins were differentially expressed in patients with liver cirrhosis and HCC
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Chen, Lili, Yuqing Sun, Jingya Wang, Hui Jiang, and Andrew G. Muntean. "Differential Regulation of c-Myc/Lin28 Discriminates Subclasses of Rearranged MLL Leukemia." Blood 126, no. 23 (2015): 163. http://dx.doi.org/10.1182/blood.v126.23.163.163.

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Abstract Rearrangements of the 11q23 locus account for ~70% of infant ALL and ~50% of infant AML1 and about 10% of leukemia overall. The prognosis for 11q23 patients is generally poor, however, outcomes vary depending on the fusion partner2. Rearrangements fuse the N-terminus of MLL with one of >70 different partner genes that includes both nuclear and cytoplasmic proteins. Despite the different intracellular localization of these partner proteins, to date, all studied MLL fusion proteins (MLL-FPs) localize to chromatin in the nucleus and drive aberrant transcriptional activation. Recent se
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Lin, Shan, Roger T. Luo, Mahesh Shrestha, Michael J. Thirman, and James C. Mulloy. "The full transforming capacity of MLL-Af4 is interlinked with lymphoid lineage commitment." Blood 130, no. 7 (2017): 903–7. http://dx.doi.org/10.1182/blood-2017-04-777185.

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Abstract Chromosome rearrangements involving the mixed-lineage leukemia gene (MLL) create MLL-fusion proteins, which could drive both acute lymphoblastic leukemia (ALL) and acute myeloid leukemia (AML). The lineage decision of MLL-fusion leukemia is influenced by the fusion partner and microenvironment. To investigate the interplay of fusion proteins and microenvironment in lineage choice, we transplanted human hematopoietic stem and progenitor cells (HSPCs) expressing MLL-AF9 or MLL-Af4 into immunodeficient NSGS mice, which strongly promote myeloid development. Cells expressing MLL-AF9 effici
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So, Chi Wai Eric, Piu Wong, Min Lin, and Michael L. Cleary. "Disease Models and Transformation Mechanisms Mediated by MLL-AF4 Family Oncoproteins in Human Leukemia." Blood 104, no. 11 (2004): 467. http://dx.doi.org/10.1182/blood.v104.11.467.467.

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Abstract The Mixed Lineage Leukemia (MLL) gene codes for a histone methyltransferase that is required for hematopoietic development. As a consequence of chromosomal translocations, MLL is fused with over 40 different genes to yield in-frame fusion proteins in acute leukemias. AF4, the most common fusion partner, accounts for 40% of MLL leukemias. The AF4-related proteins, LAF4 and AF5q31, are also fusion partners for MLL in rare cases of leukemia, whereas a fourth family member, FMR2, is a candidate protein for fragile X mental retardation syndrome. MLL fusions with AF4 family proteins manifes
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Choudhry, M. A., and I. J. McEwan. "In vitro regulation of reporter gene transcription by the androgen receptor AF1 domain." Biochemical Society Transactions 32, no. 6 (2004): 1103–6. http://dx.doi.org/10.1042/bst0321103.

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The androgen receptor (AR) is a ligand-activated transcription factor that regulates gene expression in response to the steroids testosterone and dihydrotestosterone. AR-dependent gene expression is likely to play an important role in a number of receptor-associated disorders, such as prostate cancer, spinal bulbar muscular atrophy, male type baldness and hirsutism. The AR contains two transactivation domains, termed AF1 (activation function 1) located in the N-terminus and AF2 (activation function 2) in the C-terminal ligand-binding domain. AF2 exhibits weak transcriptional activity, whereas
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Kundu, Arpita, Eric Kowarz, Jennifer Reis, and Rolf Marschalek. "Biology of t(6;11) Fusion Proteins and Their Role in MLL-Rearranged Acute Leukemia Lineage Determination." Blood 134, Supplement_1 (2019): 5033. http://dx.doi.org/10.1182/blood-2019-123070.

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Chromosomal translocations are genetic rearrangements where a chromosomal segment is transferred to a non-homologous chromosome which give rise to novel chimeras. Chromosomal rearrangements play a significant role in the development of acute leukemias (acute lymphoblastic leukemia (ALL) or acute myeloid leukemia (AML)). Chromosomal translocation events occurring at 11q23 involving the KMT2A or Mixed-Lineage Leukemia (MLL) gene (n=102) can be diagnosed in about 5-10% of all acute leukemia patients (Marschalek Ann Lab Med 2016), especially prevalent in infant acute leukemias (up to 70% of cases)
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Dissertations / Theses on the topic "Proteïna AFP"

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Moreno, Gonçalves Ana Beatriz. "Aplicaciones biotecnológicas del gen "afp" (Antifungical Protein) de "Aspergillus giganteus" para la protección de plantas frente a infección por patógenos." Doctoral thesis, Universitat de Barcelona, 2006. http://hdl.handle.net/10803/1000.

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Las plantas están constantemente sometidas a estreses ambientales y los hongos son sus principales patógenos. Actualmente, el control de las enfermedades que causan se realiza utilizando compuestos químicos, generando impacto en el medio ambiente. <br/><br/>Una alternativa es la obtención de plantas transgénicas resistentes. En un principio, la mayoría de los transgenes provenían de las propias plantas (genes involucrados en las respuestas de densa). Actualmente, y dada su reducida efectividad, se están identificando genes de defensa de otros organismos, como bacterias, insectos, animales y ho
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Baisden, Joseph M. "AFAP-110 is a cSrc activator." Morgantown, W. Va. : [West Virginia University Libraries], 2003. http://etd.wvu.edu/templates/showETD.cfm?recnum=2766.

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Thesis (Ph. D.)--West Virginia University, 2003.<br>Title from document title page. Document formatted into pages; contains v, 149 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references.
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Cherezova, Lidia Nikolayevna. "Determining the effects of phosphorylation on AFAP-110 function." Morgantown, W. Va. : [West Virginia University Libraries], 2002. http://etd.wvu.edu/templates/showETD.cfm?recnum=2492.

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Thesis (M.S.)--West Virginia University, 2002.<br>Title from document title page. Document formatted into pages; contains v, 105 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references.
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Dressel, Frank. "Sequenz, Energie, Struktur - Untersuchungen zur Beziehung zwischen Primär- und Tertiärstruktur in globulären und Membran-Proteinen." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2008. http://nbn-resolving.de/urn:nbn:de:bsz:14-ds-1222781322751-68621.

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Proteine spielen auf der zellulären Ebene eines Organismus eine fundamentale Rolle. Sie sind quasi die „Maschinen“ der Zelle. Ihre Bedeutung wird nicht zuletzt in ihrem Namen deutlich, welcher 1838 erstmals von J. Berzelius verwendet wurde und „das Erste“, „das Wichtigste“ bedeutet. Proteine sind aus Aminosäuren aufgebaute Moleküle. Unter physiologischen Bedingungen besitzen sie eine definierte dreidimensionale Gestalt, welche für ihre biologische Funktion bestimmend ist. Es wird heutzutage davon ausgegangen, dass diese dreidimensionale, stabile Struktur von Proteinen eindeutig durch die Abfol
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Rossell, Jacqueline. "Protein immobilisation for AFM." Thesis, University of Nottingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.404144.

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Johansson, Monika. "The role of nucleoside diphosphate kinase in plant mitochondria /." Uppsala : Dept. of Plant Biology and Forest Genetics, Swedish University of Agricultural Sciences, 2006. http://epsilon.slu.se/200674.pdf.

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Yarawsky, Alexander E. "Reversible assembly and amyloidogenesis of the staphylococcal biofilm protein, Aap." University of Cincinnati / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1560865959517373.

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Cervantes-Laurean, Daniel. "Preparation and Characterization of Model Conjugates for the Study of Proteins Modified by ADP-ribose." Thesis, University of North Texas, 1992. https://digital.library.unt.edu/ark:/67531/metadc935701/.

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Modification of proteins by ADP-ribose has been shown to be a versatile modification with respect to the amino acid side chain. The results described here will allow the study of the biological importance of ADP-ribose glycation and also allow differentiation on crude extracts between enzymatic modifications from protein ADP-ribose glycation that can occur due to the presence of NAD glycohydrolases.
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Xu, Ping. "Sensing and analyzing unfolded protein response during heterologous protein production :." Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 205 p, 2008. http://proquest.umi.com/pqdweb?did=1555621341&sid=2&Fmt=2&clientId=8331&RQT=309&VName=PQD.

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Ndabambi, Nonkululeko. "Recombinant expression of the pRb- and p53-interacting domains from the human RBBP6 protein for in vitro binding studies." Thesis, University of the Western Cape, 2004. http://etd.uwc.ac.za/index.php?module=etd&amp.

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The aim of this thesis was to produce DNA expression constructs and use them to investigate the feasibility of recombinantly expression proteins for future interaction studies between human RBBP6 and p53 and pRb proteins.
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Books on the topic "Proteïna AFP"

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Althaus, Felix R., and Christoph Richter. ADP-Ribosylation of Proteins. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-83077-8.

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Althaus, Felix R., Helmuth Hilz, and Sydney Shall, eds. ADP-Ribosylation of Proteins. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70589-2.

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1945-, Richter Ch, ed. ADP-ribosylation of proteins: Enzymology and biological significance. Springer-Verlag, 1987.

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cAMP signaling: Methods and protocols. Humana Press, 2015.

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1959-, Gabai Vladimir L., ed. Heat shock proteins and cytoprotection: ATP-deprived mammalian cells. R.G.Landes, 1996.

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Kabakov, Alexander E. Heat shock proteins and cytoprotection: ATP-deprived mammalian cells. Springer, 1997.

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Kim, Sang Geon. AMPK-S6K1 signaling pathway as a target for treating hepatic insulin resistance. Nova Science, 2009.

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Kim, Sang Geon. AMPK-S6K1 signaling pathway as a target for treating hepatic insulin resistance. Nova Science Publishers, 2010.

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Whitfield, James F. Calcium in cell cycles and cancer. 2nd ed. CRC Press, 1995.

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1949-, Althaus F. R., Hilz H. 1924-, Shall S. 1932-, and International Symposium on ADP-Ribosylation Reactions (7th : 1984 : Vitznau, Switzerland), eds. ADP-ribosylation of proteins. Springer-Verlag, 1985.

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Book chapters on the topic "Proteïna AFP"

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Toftager-Larsen, K. "CARBOHYDRATE MICROHETEROGENEITY OF MATERNAL SERUM AFP IN PREGNANCY." In Pregnancy Proteins in Animals, edited by Jann Hau. De Gruyter, 1986. http://dx.doi.org/10.1515/9783110858167-007.

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Westphal, Ulrich. "Androgen-Binding Protein (ABP)." In Steroid-Protein Interactions II. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-82486-9_9.

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Masignani, V., M. Pizza, and R. Rappuoli. "Common Features of ADP—Ribosyltransferases." In Bacterial Protein Toxins. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-05971-5_2.

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Popoff, M. R. "Molecular Biology of Actin-ADP-Ribosylating Toxins." In Bacterial Protein Toxins. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-05971-5_13.

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Lyubchenko, Yuri L. "AFM Visualization of Protein–DNA Interactions." In Single-molecule Studies of Proteins. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4921-8_4.

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Barbieri, Joseph T., and Drusilla L. Burns. "Bacterial Toxins that Covalently Modify Eukaryotic Proteins by ADP-Ribosylation." In Bacterial Protein Toxins. ASM Press, 2014. http://dx.doi.org/10.1128/9781555817893.ch15.

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Jacobson, Elaine L., Daniel Cervantes-Laurean, and Myron K. Jacobson. "Glycation of proteins by ADP-ribose." In ADP-Ribosylation: Metabolic Effects and Regulatory Functions. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2614-8_27.

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Unsay, Joseph D., and Ana J. García-Sáez. "AFM to Study Pore-Forming Proteins." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8894-5_10.

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Patton, W. A., N. Vitale, J. Moss, and M. Vaughan. "Mechanism of Cholera Toxin Action: ADP-Ribosylation Factors as Stimulators of Cholera Toxin-Catalyzed ADP-Ribosylation and Effectors in Intracellular Vesicular Trafficking Events." In Bacterial Protein Toxins. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-05971-5_7.

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Palazzo, Luca, Dominic I. James, Ian D. Waddell, and Ivan Ahel. "Studying Catabolism of Protein ADP-Ribosylation." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6993-7_26.

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Conference papers on the topic "Proteïna AFP"

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Timmons, Sheila, and Jack Hawiger. "REGULATION OF PLATELET RECEPTORS FOR FIBRINOGEN AND VON WILLEBRAND FACTOR BY PROTEIN KINASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644674.

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Positive and negative regulation of platelet receptors for adhesive proteins, fibrinogen (F) and von Willebrand Factor (vWF) determines whether binding of these ligands will or will not take place. We have shown previously that ADP stimulates and cyclic AMP inhibits binding of F and vWF to human platelets. Now we show that positive regulation of F and vWF binding to platelets via the glycoprotein 11b/1111a complex is dependent on platelet Protein Kinase C, a calcium- and phospholipid-dependent enzyme. A potent activator of Protein Kinase C, phorbol-12-myristoyl-13-acetate (PMA) induced saturab
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Gear, LR A., D. Freas, and J. D. Carty. "EARLY (< 5 SEC) PHOSPHORYLATIONS OF PLATELET PROTEINS FOLLOWING ACTIVATION BY ADP AND ADRENALIN, SEPARATELY AND IN COMBINATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643640.

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Understanding the earliest events (&lt; 1 sec) in signal transduction of platelets is important, since there is evicenee that “shape change,” aggregation and secretion can all begin within this period. We have employed a guenched-flow approach to study these early events and found that thrombin can induce rapid phosphorylation of myosin light-chain kinase (20K) and a 47K protein (Blood, 67, 1738, 1986). To investigate the role of rapid phosphorylations in platelet activation, we have studied the influence of adrenalin and ADP during early (0.3 to 5 sec) stimulation. Aggregation in washed human
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Fujimura, K., T. Fujimoto, M. Takemoto, K. Oda, S. Maehama, and A. Kuramoto. "INTERACTION OF MEMBRANE GLYCOPROTEIN GPIIb AND Ilia WITH CYTOSKELETAL PROTEINS DURING PLATELET ACTIVATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643515.

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Experiments were designed and performed to analyse the cytoskeleton assembly and the interaction of glycoprotein (GP)IIb, IIIa and cytoskeletal proteins during platelet activation. A23187 stimulated 125I labeled platelets were solubilised with Triton X-100 solution and centrifuged. The insoluble fraction were analysed by two dimensional electrophoresis and the soluble fraction were fractionated with 5-25% sucrose gradient centrifugation and analysed by SDS PAGE. In Triton X-100 insoluble fraction, high molecular weight protein fraction(MW &gt; 106) was present after stimulation which were cons
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Yuping, Sun, Ma Xiaoli, Wang Yunshan, and Hu Anla. "Downregulation of AFP Protein Expression by RNAi Inhibits the Growth of AFP-Producing Gastric Adenocarcinoma Cell Line." In 2007 1st International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2007. http://dx.doi.org/10.1109/icbbe.2007.15.

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Jakobs, K. H., P. Gierschik, and R. Grandt. "THE ROLE OF GTP-BINDING PROTEINS EXHIBITING GTPase ACTIVITY IN PLATELET ACTIVATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644773.

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Activation of platelets by agonists acting via cell surface-located receptors apparently involves as an early event in transmembrane signalling an interaction of the agonist-occupied receptor with a guanine nucleotide-binding regulatory protein (G-protein). The activated G-protein, then, transduces the information to the effector molecule, being responsible for the changes in intracellular second messengers. At least two changes in intracellular signal molecules are often found to be associated with platelet activation by agonists, i.e., increases in inositol trisphosphate and diacylglycerol l
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Ma, Liang, Meixiang Xu, and Andres F. Oberhauser. "Nanoscale Analysis of the Effect of Pathogenic Mutations on Polycystin-1." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13093.

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The activity of proteins and their complexes often involves the conversion of chemical energy (stored or supplied) into mechanical work through conformational changes. Mechanical forces are also crucial for the regulation of the structure and function of cells and tissues. Thus, the shape of eukaryotic cells is the result of cycles of mechano-sensing, mechano-transduction, and mechano-response. Recently developed single-molecule atomic force microscopy (AFM) techniques can be used to manipulate single molecules, both in real time and under physiological conditions, and are ideally suited to di
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Gralnick, H. R., L. M. Magurder, K. Hansmann, et al. "THE SURFACE EXPRESSION OF ALPHA GRANULE PROTEINS FOLLOWING THROMBIN STIMULATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643859.

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We have studied the platelet glycoproteins (GP) GPIb and the GPIIb/IIIa and the expression of alpha granule proteins (AGP) on the platelet (P) surface following thrombin (T) stimulation. The platelets were separated from plasma proteins on a arabinogalactan gradient. The P were stimulated with purified alpha T 0.1u/108p. Either monospecific polyclonal or murine monoclonal antibodies were used to detect the P glycoprotein and AGP. The platelets were analyzed on an EPICS V Flow Cytometer. Resting P had small amounts of AGP (2-8%) present on their surface. Within 1-3 min. after T stimulation sign
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Gray, R. J., Larry J. Kim, Lawrence J. DeLucas, Ali Arabshahi, and W. William Wilson. "Dynamic temperature-induced crystallization of proteins." In AIP Conference Proceedings Volume 387. ASCE, 1997. http://dx.doi.org/10.1063/1.52068.

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Ehrenberg, Anders. "Proteins, dynamics solutes with solvent properties." In AIP Conference Proceedings Volume 180. AIP, 1988. http://dx.doi.org/10.1063/1.37870.

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Discher, Dennis E., and Colin Johnson. "Alternative Splicing for Mechanical Resilience: The Softening Effect of Filamin’s Hinge." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176751.

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Alternative splicing within proteins is common but not well understood in its influence on protein structure and stability. Filamins are ubiquitous actin-crosslinking proteins with two dozen Immunolgobulin (Ig) repeats and one alternatively-spliced ‘hinge’ that has been hypothesized to add flexibility. The hinge is also predicted to perturb folding. The molecular mechanics of filamins are probed here by AFM-forced extension, with a particular focus on the ∼30 aa hinge between repeats R15 and R16. After re-examining full-length filamin to clarify the single molecule limit for AFM experiments on
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Reports on the topic "Proteïna AFP"

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Zhu, Xiaoyang, and Tim P. Lodge. Controlling Protein Conformation & Activities on Block-Copolymer Nanopatterns. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada520626.

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Weiss, Shimon. Biochemical and Physiological Characterization: Development & Apply Optical Methods for Charaterizing Biochemical Protein-Protein Interactions in MR-1. Office of Scientific and Technical Information (OSTI), 2006. http://dx.doi.org/10.2172/890585.

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Bednarek, Sebastian, Y. Role of AtCDC48 & the AtCDC48 Regulatory Protein Family, PUX, in Plant Cell Morphogenesis. Office of Scientific and Technical Information (OSTI), 2009. http://dx.doi.org/10.2172/977066.

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Imagawa, Walter T. Mechanisms of Altered Control of Proliferation by Cyclic Amp/Protein Kinase A During Mammary Tumor Progression. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada374059.

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LiWang, Andy. HBCU Equipment for AFOSR Project 13RSL012: The Mechanism by which ADP Regulates the Structure and Function of the Protein KaiC. Defense Technical Information Center, 2015. http://dx.doi.org/10.21236/ad1001100.

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Siuzdak, Gary. Ecosystems and Networks Integrated with Genes and Molecular Assemblies (ENIGMA): Component 5: Imaging Protein Conformations, Shapes & Assemblies in Solution & Administration project (Final Scientific/Technical Report, Subcontract No. 6974584). Office of Scientific and Technical Information (OSTI), 2021. http://dx.doi.org/10.2172/1797991.

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Stanley J. Opella. Structural Biology of The sequestration & Transport of Heavy Metal Toxins: NMR Structure Determination of Proteins Containing the CYS-X-Y-Metal Binding Motif. Office of Scientific and Technical Information (OSTI), 2004. http://dx.doi.org/10.2172/822065.

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Pawlowski, David R. Automated Sample Preparation (ASP): Development of a Rapid Method to Sequentially Isolate Nucleic Acids and Protein from Any Sample Type by a Cartridge-Based System. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada608052.

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