Relevant bibliographies by topics / Proximal renal tubules

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Proximal renal tubules'

Author: Grafiati
Published: 10 September 2021
Last updated: 29 July 2025

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Journal articles on the topic "Proximal renal tubules"

1

Dominguez, J. H., M. Juhaszova, S. B. Kleiboeker, C. C. Hale, and H. A. Feister. "Na(+)-Ca2+ exchanger of rat proximal tubule: gene expression and subcellular localization." American Journal of Physiology-Renal Physiology 263, no. 5 (1992): F945—F950. http://dx.doi.org/10.1152/ajprenal.1992.263.5.f945.

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The activity of the Na(+)-Ca2+ exchanger, a membrane transporter that mediates Ca2+ efflux, has been described in amphibian and mammalian renal proximal tubules. However, demonstration of cell-specific expression of the Na(+)-Ca2+ exchanger in proximal renal tubules has been restricted to functional assays. In this work, Na(+)-Ca2+ exchanger gene expression in rat proximal tubules was characterized by three additional criteria: functional assay of transport activity in membrane vesicles derived from proximal tubules, expression of specific Na(+)-Ca2+ exchanger protein detected on Western blots
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Beyenbach, Klaus W. "Kidneys sans glomeruli." American Journal of Physiology-Renal Physiology 286, no. 5 (2004): F811—F827. http://dx.doi.org/10.1152/ajprenal.00351.2003.

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The evolution of the vertebrate kidney records three occasions, each separated by about 50 million years, when fish have abandoned glomeruli to produce urine by tubular mechanisms. The recurring dismissal of glomeruli suggests a mechanism of aglomerular urine formation intrinsic to renal tubules. Indeed, the transepithelial secretion of organic solutes and of inorganic solutes such as sulfate, phosphate, and magnesium can all drive secretory water flow in renal proximal tubules of fish. However, the secretion of NaCl via secondary active transport of Cl is the primary mover of secretory water
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Ramachandran, C., and M. G. Brunette. "The renal Na+/Ca2+ exchange system is located exclusively in the distal tubule." Biochemical Journal 257, no. 1 (1989): 259–64. http://dx.doi.org/10.1042/bj2570259.

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The movement of Ca2+ across the basolateral plasma membrane was determined in purified preparations of this membrane isolated from rabbit proximal and distal convoluted tubules. The ATP-dependent Ca2+ uptake was present in basolateral membranes from both these tubular segments, but the activity was higher in the distal tubules. A very active Na+/Ca2+ exchange system was also demonstrated in the distal-tubular membranes, but in proximal-tubular membranes this exchange system was not demonstrable. The presence of Na+ outside the vesicles gradually inhibited the ATP-dependent Ca2+ uptake in the d
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Maurel, Agnès, Odile Spreux-Varoquaux, Francesco Amenta, et al. "Vesicular monoamine transporter 1 mediates dopamine secretion in rat proximal tubular cells." American Journal of Physiology-Renal Physiology 292, no. 5 (2007): F1592—F1598. http://dx.doi.org/10.1152/ajprenal.00514.2006.

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Renal dopamine, synthesized by proximal tubules, plays an important role in the regulation of renal sodium excretion. Although the renal dopaminergic system has been extensively investigated in both physiological and pathological situations, the mechanisms whereby dopamine is stored and secreted by proximal tubule cells remain obscure. In the present study we investigated whether vesicular monoamine transporters (VMAT)-1 and -2, which participate in amine storing and secretion, are expressed in rat renal proximal tubules, and we defined their involvement in dopamine secretion. By combining RT-
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Takeuchi, Kazuhiro, Sayumi Kawamura, Yukihiro Wada, et al. "Renal Impairment of Proximal Tubular Injury Caused by Red Yeast Rice Supplement: Report of 2 Cases." Case Reports in Nephrology and Dialysis 14, no. 1 (2024): 128–37. http://dx.doi.org/10.1159/000540258.

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Introduction: Drug-induced tubulointerstitial injury is a common cause of renal impairment. Since the mechanisms of drug-induced tubular injury are diverse, various treatment approaches are needed according to the pathogenesis. Renal biopsy is indispensable to determine not only the pathological diagnosis, but also the underlying mechanism, and to guide appropriate treatment. Most recently, one of the red yeast supplements has been widely highlighted as a novel cause of tubular damage, mainly in Japan and Asia. However, neither detailed pathological findings nor the mechanism of renal impairme
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Miller, D. S. "Daunomycin secretion by killfish renal proximal tubules." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 269, no. 2 (1995): R370—R379. http://dx.doi.org/10.1152/ajpregu.1995.269.2.r370.

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Epifluorescence microscopy and video-image analysis were used to measure the uptake of the fluorescent anthracycline daunomycin by intact killifish renal proximal tubules. When tubules were incubated in medium containing 2-5 microM daunomycin, the drug accumulated in the cells and the tubular lumen. At steady state, luminal fluorescence was two to three times greater than cellular fluorescence. Luminal accumulation of daunomycin was reduced when tubules were exposed to the multidrug-resistance (MDR) transporter modifiers verapamil and cyclosporin A (CSA), but not tetraethylammonium (TEA), a mo
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Gewin, Leslie S. "Sugar or Fat? Renal Tubular Metabolism Reviewed in Health and Disease." Nutrients 13, no. 5 (2021): 1580. http://dx.doi.org/10.3390/nu13051580.

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The kidney is a highly metabolically active organ that relies on specialized epithelial cells comprising the renal tubules to reabsorb most of the filtered water and solutes. Most of this reabsorption is mediated by the proximal tubules, and high amounts of energy are needed to facilitate solute movement. Thus, proximal tubules use fatty acid oxidation, which generates more adenosine triphosphate (ATP) than glucose metabolism, as its preferred metabolic pathway. After kidney injury, metabolism is altered, leading to decreased fatty acid oxidation and increased lactic acid generation. This revi
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Syal, Ashu, Susan Schiavi, Sumana Chakravarty, Vangipuram Dwarakanath, Raymond Quigley, and Michel Baum. "Fibroblast growth factor-23 increases mouse PGE2 production in vivo and in vitro." American Journal of Physiology-Renal Physiology 290, no. 2 (2006): F450—F455. http://dx.doi.org/10.1152/ajprenal.00234.2005.

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Fibroblast growth factor-23 (FGF-23) has been implicated in the renal phosphate wasting in X-linked hypophosphatemia, tumor-induced osteomalacia, and autosomal dominant hypophosphatemic rickets. Recently, we demonstrated that Hyp mice have greater urinary PGE2 levels compared with C57/B6 mice and that indomethacin administration in vivo and in vitro ameliorates the phosphate transport defect in Hyp mice. To determine further whether altered prostaglandin metabolism plays a role in the renal phosphate transport defect in Hyp mice, we incubated renal proximal tubules with arachidonic acid. We fi
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Deebajah, M., Z. Qu, and P. Zhang. "GATA3 Is a Useful Immunohistochemical Marker to Differentiate Variants of Renal Tubular Lesions from Different Segments of Renal Tubules." American Journal of Clinical Pathology 156, Supplement_1 (2021): S152—S153. http://dx.doi.org/10.1093/ajcp/aqab191.325.

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Abstract Introduction/Objective GATA3 is found in glomerular mesangial cells, and the distal tubules & collecting ducts in metanephros and eventual kidneys, but not associated with the proximal tubules and loops of Henle. We hypothesize that GATA3 can be used as a marker to identify the origin of tubular differentiation in most renal tumors. Methods/Case Report Ten negative controls and 43 renal mass lesions (RCC, papillary, clear cell papillary, and chromophobe carcinomas, oncocytoma, and polycystic kidney disease). GATA3 nuclear stain was graded as negative (absent stain), equivocal and
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Li, Huiping, Xiyou Zhou, Deborah R. Davis, Di Xu, and Curt D. Sigmund. "An androgen-inducible proximal tubule-specific Cre recombinase transgenic model." American Journal of Physiology-Renal Physiology 294, no. 6 (2008): F1481—F1486. http://dx.doi.org/10.1152/ajprenal.00064.2008.

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To facilitate the study of renal proximal tubules, we generated a transgenic mouse strain expressing an improved Cre recombinase (iCre) under the control of the kidney androgen-regulated protein (KAP) promoter. The transgene was expressed in the kidney of male mice but not in female mice. Treatment of female transgenic mice with androgen induced robust expression of the transgene in the kidney. We confirmed the presence of Cre recombinase activity and the cell specificity by breeding the KAP2-iCRE mice with ROSA26 reporter mice. X-Gal staining of kidney sections from male double transgenic mic
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Dissertations / Theses on the topic "Proximal renal tubules"

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SILBER, PAUL MICHAEL. "EARLY INDICATION AND PATHOGENESIS OF RENAL PROXIMAL TUBULE INJURY (ENZYMURIA)." Diss., The University of Arizona, 1987. http://hdl.handle.net/10150/184097.

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It is well known that a variety of toxicants can cause damage to the renal proximal tubule. However, the early pathogenesis of these deleterious interactions between a toxicant and this region of the nephron remain poorly understood. Thus, the purpose of this research was to attempt to answer three interrelated questions. First, what are the earliest changes in kidney function and structure after administration of tubule toxicants in vivo? Secondly, how do these structural/functional alterations change over time? Finally, are certain indicators of renal "dysfunction" more sensitive then other
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Shuprisha, Apichai. "Real-time assessment of organic anion secretion in isolated, perfused rabbit renal proximal tubules." Diss., The University of Arizona, 1999. http://hdl.handle.net/10150/289029.

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A newly developed epifluorescense microscopy system has been employed to measure net transepithelial secretion of fluorescein (FL) in real time in isolated perfused S2 segments of rabbit renal proximal tubules. Net FL secretion (K(t), ∼4 μM, and J(max), ∼280 fmol·min⁻¹·mm⁻¹) shares the same transport system with that of para -aminohippurate (PAH). The basolateral Na-DC cotransporter supports ∼25% of the "basal" FL secretion in the absence of exogenous αKG via recycling of αKG that has been exchanged for FL. Physiological αKG concentrations in the bath (∼10 μM) or in the perfusate (∼50 μM) stim
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RUEGG, CHARLES EDWARD. "MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS." Diss., The University of Arizona, 1987. http://hdl.handle.net/10150/184162.

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The convoluted (CPT) and straight (SPT) portions of the renal proximal tubule are susceptible to injury by a wide variety of chemical agents. These agents often affect the CPT or SPT selectively by proposed mechanisms usually attributed to tubular concentration, blood flow delivery patterns and tubuloglomerular feedback responses within the intact kidney. The innate cellular responses to chemical exposures remain virtually unexplored. Hence, the basic goal of this research was to develop an in vitro system that was conducive to examining the innate cellular differences in susceptibility betwee
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Tang, Chi-wai Sydney. "The many facets of the renal proximal tubular epithelial cell in human." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31992468.

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Tang, Chi-wai Sydney, and 鄧智偉. "The many facets of the renal proximal tubular epithelial cell inhuman." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31992468.

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Reece, Mark T. "Functional characterization of OCTRL2 : an organic cation transporter expressed in the renal proximal tubules." Thesis, McGill University, 1998. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=20973.

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Chromosome 11p15.5 harbors a gene or genes involved in Beckwith Wiedemann Syndrome (BWS) and that confer(s) susceptibility to Wilms' tumor, rhabdomyosarcoma, and hepatoblastoma. PowerBLAST of P1 artificial chromosome clones from this region identified two novel transcripts with open reading frames encoding putative proteins of 253 and 424 amino acids. The larger of the transcripts was shown by Northern blot to be predominantly expressed in the fetal and adult liver and kidney. This transcript shares homology with integral membrane organic cation transporters, such as the tetracycline resistanc
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Branco, Regiane Cardoso Castelo. "Efeito da angiotensina-(1-7) no fluxo reabsortivo de bicarbonato (JHCO3-) e na concentração citosólica de cálcio ([Ca2+]i): estudo por microperfusão tubular proximal, in vivo." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-25072012-135726/.

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O estudo avaliou os efeitos agudos da Ang-(1-7) na reabsorção de bicarbonato (JHCO3-) no túbulo proximal cortical de rato, in vivo, medindo o pH intratubular pelo microeletródio sensível a H+. O JHCO3- controle é 2,84 ± 0,08 nmol. cm-2. s-1 (49), a Ang-(1-7; 10-12 ou 10-9 M) o reduz (35 ou 61 %) e a Ang-(1-7; 10-6 M) o eleva (56 %). A inibição do receptor Mas (por A779) eleva o JHCO3- (30 %), abole o efeito inibidor da Ang-(1-7), mas não afeta seu efeito estimulador. A inibição do NHE3 (por S3226) diminui o JHCO3- (45 %), não altera o efeito inibidor da Ang-(1-7), mas transforma seu efeito est
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Pergher, Patrícia e. Silva. "Efeitos não-genômicos dos hormônios esteróides - aldosterona e corticosterona - sobre a acidificação do túbulo proximal (S2) de ratos: estudos de microperfusão tubular e capilar, in vivo." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-10122010-144456/.

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O objetivo foi determinar se aldosterona e corticosterona agem sobre a acidificação do túbulo proximal e se esses efeitos são genômicos e/ou não-genômicos. A reabsorção de HCO3- foi avaliada por microperfusão estacionária. Aldosterona e corticosterona perfundidas na luz tubular causaram aumento significante do JHCO3-. Na presença de etanol, actinomicina D, cicloheximida ou espironolactona, o JHCO3- foi estatisticamente igual ao valor controle (2,84 ± 0,079 nmol.cm-2.s-1). RU486 sozinho inibiu o efeito estimulador da aldosterona e corticosterona. Losartan não alterou o JHCO3-. Concanomicina ou
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Farah, Livia Xavier Soares. "Efeito do peptídeo-1 semelhante ao glucagon endógeno sobre a atividade do NHE3 em túbulo proximal renal." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/5/5166/tde-05102015-114814/.

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O peptídeo-1 semelhante ao glucagon (GLP-1) é um hormônio incretina secretado pelas células L do trato gastrointestinal e liberado imediatamente após a ingestão de alimento. O GLP-1 estimula a secreção de insulina pós-prandial moderando a elevação precoce da glicose no sangue. Embora primariamente envolvido na homeostase da glicose, o GLP-1 é capaz de induzir a diurese e natriurese, quando administrado em doses farmacológicas em humanos e em roedores. Estudos prévios do nosso laboratório demonstraram que o mecanismo de ação renal do GLP-1, bem como de agonistas sintéticos do receptor GLP-1R, e
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Lee, Sarah Angeline. "Curcumin Protects against Renal Ischemia by Activating the Unfolded Protein Response and Inducing HSP70." Yale University, 2009. http://ymtdl.med.yale.edu/theses/available/etd-04062009-215154/.

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The purpose of this study was to establish whether curcumin protects renal proximal tubule cells against ischemic injury, determine whether this postulated cytoprotective effect is mediated through the upregulation of HSP70, and investigate whether the mechanism by which curcumin induces HSP70 expression and confers its protective effect is through activation of the Unfolded Protein Response. LLC-PK1 cells were cultured on collagen-coated filters to mimic conditions of in vivo renal proximal tubule cells and induce cell polarization. Injury with and without curcumin treatment was studied by us
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Books on the topic "Proximal renal tubules"

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Najjar, Samer. Effects of ischemia and reperfusion on mitochondrial phosphate uptake in rat renal proximal tubules. s.n.], 1993.

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Wong, P. S. K. The use of NMR spectroscopy to follow intracellular sodium content in rat rental proximal tubules. University of Birmingham, 1994.

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Jones, Caroline Elizabeth Mary. The development, evaluation and use of freshly isolated renal proxinal tubule systems in the fischer rat. Aston University. Department of Pharmaceutical Sciences, 1990.

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Speeckaert, Marijn, and Joris Delanghe. Tubular function. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0008.

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Assessment of tubular function is more complicated than the measurement of glomerular filtration rate. Different functions may be affecting according to the different segments of tubule involved. Key tests include concentrating and diluting capacity, and fractional excretion of sodium. Tubular proteinuria occurs when glomerular function is normal, but when the proximal tubules have a diminished capacity to reabsorb and to catabolize proteins, causing an increased urinary excretion of the low-molecular-mass proteins that normally pass through the glomerulus. Proximal tubular dysfunction is char
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Bockenhauer, Detlef, and Robert Kleta. Approach to the patient with renal Fanconi syndrome, glycosuria, or aminoaciduria. Edited by Robert Unwin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0041_update_001.

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Up to 80% of filtered salt and water is returned back into the circulation in the proximal tubule. Several solutes, such as phosphate, glucose, low-molecular weight proteins, and amino acids are exclusively reabsorbed in this segment, so their appearance in urine is a sign of proximal tubular dysfunction. An entire orchestra of specialized apical and basolateral transporters, as well as paracellular molecules, mediate this reabsorption. Defects in proximal tubular function can be isolated (e.g. isolated renal glycosuria, aminoacidurias, or hypophosphataemic rickets) or generalized. In the latt
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Murer, Heini, Jürg Biber, and Carsten A. Wagner. Phosphate homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0025.

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Inorganic phosphate ions (H2PO4−/ HPO42−) (abbreviated as Pi) are involved in formation of bone and generation of high-energy bonds (e.g. ATP), metabolic pathways, and regulation of cellular functions. In addition, Pi is a component of biological membranes and nucleic acids. Only about 1% of total body Pi content is present in extracellular fluids, at a plasma concentration in adults within the range 0.8–1.4 mMol/L (at pH 7.4 mostly as HPO42−), with diurnal variations of approximately 0.2 mM. A small amount of plasma Pi is bound to proteins or forms complexes with calcium. Under normal, balanc
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Schreuder, Michiel F. Renal tubular dysgenesis. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0350.

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Renal tubular dysgenesis involves the absence or incomplete differentiation of proximal tubular nephron segments. Due to the lack of a patent nephron, it is characterized by (fetal) anuria and subsequent oligohydramnios, pulmonary hypoplasia, premature birth with severe and refractory arterial hypotension, and fetal or neonatal death. The main cause for renal tubular dysgenesis is a genetic mutation in the renin–angiotensin system, which has shown an autosomal recessive trait. Maternal use of angiotensin-converting enzyme inhibitors or angiotensin II receptor blockers during pregnancy can have
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Horst-Liu, Zeya. PDZ proteins based heteromultimeric complexes in the renal proximal tubule. 2006.

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Walsh, Stephen B. Approach to the patient with renal tubular acidosis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0036.

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The renal tubular acidoses are a collection of syndromes characterized by defective urinary acidification. These syndromes have classically caused some confusion, and many opine that the widely used numerical system (type 1, 2) should be abandoned. We consider distal renal tubular acidosis and proximal renal tubular acidosis separately, and briefly cover hypoaldosteronism. Distal (Type 1) renal tubular acidosis is a syndrome of hypokalaemia, metabolic acidosis, kidney stones, nephrocalcinosis, and osteomalacia or rickets. It is caused by failure of the acid secreting α‎‎‎-intercalated cells in
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Houillier, Pascal. Magnesium homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0027.

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Magnesium is critically important in the process of energy release. Although most magnesium is stored outside the extracellular fluid compartment, the regulated concentration appears in blood. Urinary magnesium excretion can decrease rapidly to low values when magnesium entry rate into the extracellular fluid volume is low, which has several important implications: cell and bone magnesium do not play a major role in the defence of blood magnesium concentration; while a major role is played by the kidney and especially the renal tubule, which adapts to match the urinary magnesium excretion and
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Book chapters on the topic "Proximal renal tubules"

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Pavelka, Margit, and Jürgen Roth. "Parathyroid Hormone Response of Renal Proximal Tubules." In Functional Ultrastructure. Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_119.

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Green, Carol E., Jack E. Dabbs, Katherine L. Allen, Charles A. Tyson, and Elmer J. Rauckman. "Characterization of Isolated Renal Proximal Tubules for Nephrotoxicity Studies." In Nephrotoxicity. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-2040-2_111.

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Mandel, Lazaro J., William R. Jacobs, Rick Schnellmann, Maria Sgambati, Ann LeFurgey, and Peter Ingram. "Mechanisms of Anoxic Injury to Transport and Metabolism of Proximal Renal Tubules." In Cell Calcium Metabolism. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_49.

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Kaimori, Jun-ya, Masaru Takenaka, and Kousaku Okubo. "16 Quantification of Gene Expression in Mouse and Human Renal Proximal Tubules." In Laser Capture Microdissection. Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-853-6:209.

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Christensen, Erik Ilsø, and Søren Nielsen. "Protein Handling from Apical and Basolateral Surfaces in Rat and Rabbit Renal Proximal Tubules." In Endocytosis. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-84295-5_40.

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Chobanian, M. C., L. A. Fahien, and P. C. Brazy. "Metabolic Requirement for Inorganic Phosphate by Renal Proximal Tubules: Influence upon L-Glutamine Metabolism." In Contributions to Nephrology. KARGER, 1997. http://dx.doi.org/10.1159/000059849.

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Yanagawa, Norimoto, and Ok D. Jo. "Possible Role of Calcium in Parathyroid Hormone Action on Phosphate Transport in Rabbit Renal Proximal Tubules." In Phosphate and Mineral Homeostasis. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5206-8_13.

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Lote, Chris. "The proximal tubule." In Principles of Renal Physiology. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4086-7_5.

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Lote, Christopher J. "The proximal tubule." In Principles of Renal Physiology. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-6470-2_5.

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Lote, Christopher J. "The Proximal Tubule." In Principles of Renal Physiology. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3785-7_5.

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Conference papers on the topic "Proximal renal tubules"

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Westwood, Brian M., Hossam A. Shaltout, and Mark C. Chappell. "Modeling of Angiotensin Peptide Metabolism in Renal Proximal Tubules." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-190990.

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The recent discovery of angiotensin converting enzyme 2 (ACE2) as a functional peptidase within the renin-angiotensin system (RAS) has added a new layer of complexity to the enzymatic cascade of this hormonal system. ACE2 is highly expressed in the proximal tubules of the kidney, an important tissue site involved in blood pressure regulation. Therefore, we derived a model for the processing of Ang I which is the immediate precursor to the biologically active peptides Ang II and Ang-(1-7) based on metabolism data in isolated proximal tubules of the sheep kidney (1). Given the individual experim
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Hansson, Jennifer, Kjell Hultenby, Catharina Cramnert, David Lindgren, Håkan Axelson, and Martin E. Johansson. "Abstract 5010: Characterization of a novel cell type in human renal proximal tubules with connection to renal cell carcinoma development." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5010.

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Safithri, Fathiyah, Desy Andari, and Fifa Yuniarmi. "Improvement of Renal Proximal Tubules after Black Cumin (Nigella Sativa) Extract Administration in Rat with CCl4-induced Chronic Renal Damage." In The Health Science International Conference. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009120000210026.

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Brandão, Joana Bader Sadala, Isabela Abud De Andrade, Maria Eduarda Alencar Santos, Paula Cristina Rios Rodriguez, and Rafael Xavier Cunha. "INFECÇÃO POR SARS-COV-2 E RELAÇÃO FISIOPATOLÓGICA COM INJÚRIA RENAL." In I Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1000.

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INTRODUÇÃO: A Covid-19 é caracterizada por sua alta transmissibilidade e, em casos graves, pela sua acentuada resposta imune e sua tempestade de citocinas, que pode resultar em perda da tolerância periférica dos órgãos, ocasionando uma inflamação exacerbada e destruição tecidual. É sabido que o vírus Sars-CoV-2 pode gerar injúrias em órgãos e sistemas além do pulmonar e imune, como o sistema renal. Um importante ponto de análise foi a ação da enzima conversora de angiotensina 2 (ACE2), altamente presente nas células tubulares proximais e podócitos, levando a um quadro de hematúria e proteinúri
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Taub, Mary L., and Sunil Sudarshan. "Abstract 4364: Oncometabolite L-2-hydroxyglutarate blocks differentiation of renal proximal tubule cells in matrigel." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4364.

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Taub, Mary L., and Sunil Sudarshan. "Abstract 4364: Oncometabolite L-2-hydroxyglutarate blocks differentiation of renal proximal tubule cells in matrigel." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4364.

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Gronda, Edoardo, Massimo Iacoviello, and Arduino Arduini. "Sodium glucose co-transporter inhibitors put cardiovascular medicine at a crossroads – Cardiorenal interaction and clinical implications." In 7th International Congress of Cardionephrology KARNEF 2025. Punta Niš, 2025. https://doi.org/10.46793/karnef25.215g.

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Heart and kidney have essential homeostatic role in circulation balance to cope with variation of body biology needs, however the two organs physiology is designed to satisfy opposing demands. The heart has to provide with oxygen and nutrition of different organs and apparatus based on intercurrent demand without any predefined schedule. In contrast, the kidneys have to adapt the body’s fluid and electrolyte content to cope with changes in the internal and external environment. The heart and kidney function confront different physiological needs, namely the energy the body requires versus the bo
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Kumar, Balawant, Rizwan Ahmad, Pinelopi Kapitsino, et al. "Abstract 1767: Rho-GTPase inhibits claudin-2 expression to promote proximal tubular epithelial cell plasticity and renal cell carcinoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1767.

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Kumar, Balawant, Rizwan Ahmad, Pinelopi Kapitsino, et al. "Abstract 1767: Rho-GTPase inhibits claudin-2 expression to promote proximal tubular epithelial cell plasticity and renal cell carcinoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1767.

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Kaewpaiboon, Sunisa, Titpawan Nakpheng, and Teerapol Srichana. "Biocompatibility of Polymyxin B Sulfate Based on Sodium Deoxycholate Sulfate Formulations with Kidney Cell Lines, Macrophage Cells, and Red Blood Cells." In 5th International Conference and Exhibition on Pharmaceutical Sciences and Technology 2022. Trans Tech Publications Ltd, 2022. http://dx.doi.org/10.4028/p-7490x3.

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Antibiotic-resistant has emerged without new drug challenges. Polymyxin B (PMB) was the last resort therapy for multiple-drug resistant Gram-negative bacteria. However, the toxicity of PMB including nephrotoxicity (61%) and neurotoxicity (7%) was dose-limitation. PMB-based sodium deoxycholate sulfate (SDCS) formulations were prepared in the 2-different mole ratios of SDCS to PMB (5:1 and 10:1). Particle size, zeta-potential, and drug content were evaluated. The biocompatibility of PMB formulations was investigated with normal human primary renal proximal tubule epithelial cells (PCS-400-010),
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