Relevant bibliographies by topics / PS1 mice

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'PS1 mice'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "PS1 mice"

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Chen, Min, Jiashuo Zheng, Guohao Liu та ін. "High Dietary Iron Disrupts Iron Homeostasis and Induces Amyloid-β and Phospho-τ Expression in the Hippocampus of Adult Wild-Type and APP/PS1 Transgenic Mice". Journal of Nutrition 149, № 12 (2019): 2247–54. http://dx.doi.org/10.1093/jn/nxz168.

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ABSTRACT Background Brain iron deposition is a feature of Alzheimer disease and may contribute to its development. However, the relative contribution of dietary iron remains unclear. Objectives We investigated the impact of high dietary iron on brain pathological changes and cognitive function in adult wild-type (WT) mice and amyloid precursor protein/presenilin 1 (APP/PS1) double transgenic mice. Methods Male WT mice and APP/PS1 mice aged 10 wk were fed either a control diet (66 mg Fe/kg) (WT-Ctrl and APP/PS1-Ctrl) or a high iron diet (14 g Fe/kg) (WT-High Fe and APP/PS1-High Fe) for 20 wk. I
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Han, Bing, Jin-Hua Wang, Yuan Geng, et al. "Chronic Stress Contributes to Cognitive Dysfunction and Hippocampal Metabolic Abnormalities in APP/PS1 Mice." Cellular Physiology and Biochemistry 41, no. 5 (2017): 1766–76. http://dx.doi.org/10.1159/000471869.

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Background/Aims: Stress response is determined by the brain, and the brain is a sensitive target for stress. Our previous experiments have confirmed that once the stress response is beyond the tolerable limit of the brain, particularly that of the hippocampus, it will have deleterious effects on hippocampal structure and function; however, the metabolic mechanisms for this are not well understood. Methods: Here, we used morris water maze, elisa and gas chromatography-time of flight/mass spectrometry to observe the changes in cognition, neuropathology and metabolomics in the hippocampus of APP/
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Zha, Lihuang, Zaixin Yu, Jian Fang, Li Zhou, Wen Guo, and Jun Zhou. "NLRC3 Delays the Progression of AD in APP/PS1 Mice via Inhibiting PI3K Activation." Oxidative Medicine and Cellular Longevity 2020 (December 24, 2020): 1–14. http://dx.doi.org/10.1155/2020/5328031.

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NLRC3 inhibits inflammatory responses. Neuroinflammation induces and accelerates the onset of Alzheimer’s disease (AD). This study is aimed at investigating whether NLRC3 plays a role in neuroinflammation, Aβ accumulation, and neuroprotection in AD mice. 12-month-old APP/PS1 transgenic and C57 mice were used for studies in vivo. In vitro, organotypic hippocampal slices were cultured. We found that the expression of NLRC3 was downregulated in the brain tissues of APP/PS1 mice. Mice in the APP/PS1 group had a significant attenuation of learning and memory ability compared to the control group, a
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Zhang, Q. L., L. Jia, X. Jiao, et al. "APP/PS1 Transgenic Mice Treated with Aluminum: An Update of Alzheimer's Disease Model." International Journal of Immunopathology and Pharmacology 25, no. 1 (2012): 49–58. http://dx.doi.org/10.1177/039463201202500107.

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There is still no animal model available that can mimic all the cognitive, behavioral, biochemical, and histopathological abnormalities observed in patients with Alzheimer's disease (AD). We undertook to consider the interaction between genetic factors, including amyloid precursor protein (APP) and presenilin-1 (PS1), and environmental factors, such as Aluminum (Al) in determining susceptibility outcomes when studying the pathogenesis of AD. In this article, we provide an AD model in APP/PS1 transgenic mice triggered by Al. The animal model was established via intracerebral ventricular microin
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Kim, Jin Woo, Tong-Shin Chang, Ji Eun Lee, et al. "Negative Regulation of the Sapk/Jnk Signaling Pathway by Presenilin 1." Journal of Cell Biology 153, no. 3 (2001): 457–64. http://dx.doi.org/10.1083/jcb.153.3.457.

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Presenilin 1 (PS1) plays a pivotal role in Notch signaling and the intracellular metabolism of the amyloid β-protein. To understand intracellular signaling events downstream of PS1, we investigated in this study the action of PS1 on mitogen-activated protein kinase pathways. Overexpressed PS1 suppressed the stress-induced stimulation of stress-activated protein kinase (SAPK)/c-Jun NH2-terminal kinase (JNK) in human embryonic kidney 293 cells. Interestingly, two functionally inactive PS1 mutants, PS1(D257A) and PS1(D385A), failed to inhibit UV-stimulated SAPK/JNK. Furthermore, H2O2- or UV-stimu
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Li, Dan, Chen Wang, Song Tan, and Yu Li. "Plant Compound Curcumin Increased the Expression of MAP2 in Hippocampal Neurons of APPswe/PS1dE9 Double Transgenic Mice." Advanced Materials Research 781-784 (September 2013): 717–20. http://dx.doi.org/10.4028/www.scientific.net/amr.781-784.717.

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In this study, we aim to investigate the effect of curcumin on the expression of MAP2 in hippocampus in APP/PS1 double transgenic mice. AD model was established with APP/PS1 double transgenic mice, which were fed for 6 months with different concentration of curcumin diet. Immunohistochemistry were applied to evaluate the expressive of MAP2 in hippocampus of transgenic mice. The expression of MAP2 in hippocampus were decreased in APP/PS1 double transgenic mice, but increased in concentration-dependent manner in curcumin treatment group,especially in CA1 and CA3 region. MAP2 may play an importan
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Brown, Desmond, Gobinda Sarkar, Teresa Decklever та ін. "SCIDOT-39. K16ApoE ENHANCES Aβ-ASSOCIATED 11C-PiB DEPOSITION AND PET SIGNAL IN APP/PS1 TRANSGENIC MICE". Neuro-Oncology 21, Supplement_6 (2019): vi279—vi280. http://dx.doi.org/10.1093/neuonc/noz175.1175.

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Abstract OBJECTIVE The K16ApoE peptide enhances delivery of multiple agents across the blood-brain barrier (BBB). Transgenic mouse models are central to elucidating the underlying pathophysiology of Alzheimer’s Disease (AD) and provide a system for evaluating novel therapeutic strategies. PET imaging plays a central clinical role in diagnosing human cases of AD but has had variable performance in mouse models. We investigated the role of K16ApoE to enhance delivery of a radiolabeled PET imaging tracer, 11C-PiB and assess whether this corresponds to improved PET sensitivity in APP/PS1 transgeni
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Leitner, Nicole R., Thomas Kolbe, Caroline Lassnig, Konrad Hochedlinger, Thomas Rülicke, and Mathias Müller. "PS1-69 Inducible STAT1 protein in mice." Cytokine 52, no. 1-2 (2010): 31–32. http://dx.doi.org/10.1016/j.cyto.2010.07.141.

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Lee, Yi-Heng, Hao-Chieh Hsu, Pei-Chen Kao, et al. "Augmented Insulin and Leptin Resistance of High Fat Diet-Fed APPswe/PS1dE9 Transgenic Mice Exacerbate Obesity and Glycemic Dysregulation." International Journal of Molecular Sciences 19, no. 8 (2018): 2333. http://dx.doi.org/10.3390/ijms19082333.

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Alzheimer’s disease (AD), a progressive neurodegenerative disease is highly associated with metabolic syndromes. We previously demonstrated that glycemic dysregulation and obesity are augmented in high fat diet (HFD)-treated APPswe/PS1dE9 (APP/PS1) transgenic mice. In the current study, the underlying mechanism mediating exacerbated metabolic stresses in HFD APP/PS1 transgenic mice was further examined. APP/PS1 mice developed insulin resistance and, consequently, impaired glucose homeostasis after 10 weeks on HFD. [18F]-2-fluoro-2-deoxy-d-glucose ([18F]-FDG) positron emission tomography showed
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Yuan, Li, Jun Zhang, Jun-Hong Guo, et al. "DAla2-GIP-GLU-PAL Protects Against Cognitive Deficits and Pathology in APP/PS1 Mice by Inhibiting Neuroinflammation and Upregulating cAMP/PKA/CREB Signaling Pathways." Journal of Alzheimer's Disease 80, no. 2 (2021): 695–713. http://dx.doi.org/10.3233/jad-201262.

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Background: Alzheimer’s disease (AD) is a neurodegenerative disease characterized by progressive decline in cognitive function. Type 2 diabetes mellitus (T2DM) is an important risk factor for AD. Glucose-dependent insulinotropic polypeptide (GIP) has been identified to be effective in T2DM treatment and neuroprotection. Objective: The present study investigated the neuroprotective effects and possible mechanisms of DAla2GIP-Glu-PAL, a novel long-lasting GIP analogue, in APP/PS1 AD mice. Methods: Multiple behavioral tests were performed to examine the cognitive function of mice. In vivo hippoca
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Dissertations / Theses on the topic "PS1 mice"

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Whitman, Kathrine. "Mouse Model Behavior in APP/PS1 Mice Treated with a BBB-penetrating Erythropoietin Fusion Protein, cTfRMAb-EPO." Scholarship @ Claremont, 2019. https://scholarship.claremont.edu/cmc_theses/2092.

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Alzheimer’s disease (AD) is a devastating neurodegenerative condition in which a patient’s cognitive functioning, memory, and physical health progressively deteriorate. In order to treat physiological deterioration in AD, a neuroprotective recombinant human- erythropoietin (EPO) fusion protein was used. In addition to its ability to target amyloid beta (Aβ) aggregation, EPO has been shown to reduce inflammation, oxidative stress and synaptic loss. Recombinant human-erythropoietin (EPO) was combined with a chimeric transferrin receptor (TfR) monoclonal antibody (cTfRMAb) to form a fusion protei
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Dickey, Chad Anthony. "The influence of amyloid-beta, a major pathological marker in Alzheimer's disease, on molecular cognitive processes of APP+PS1 transgenic mice." [Tampa, Fla.] : University of South Florida, 2004. http://purl.fcla.edu/fcla/etd/SFE0000360.

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Schleif, William. "Effects of Long-Term Administration of Caffeine in a Mouse Model for Alzheimer’s Disease." Scholar Commons, 2005. https://scholarcommons.usf.edu/etd/854.

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A recent epidemiological study suggested that higher caffeine intake reduces the risk of Alzheimer's disease (AD). Caffeine, a widely consumed stimulatory drug, is a non-selective adenosine receptor antagonist that has been shown to increase plasma adenosine levels in rodents. To determine any long-term protective effects of caffeine in a controlled longitudinal study, caffeine was added to the drinking water of APPsw transgenic (Tg) mice between 4 and 9 1/2 months of age, with behavioral testing done during the last 6 weeks of treatment. The average daily intake of caffeine per mouse (1.5 mg)
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Li, Chaoyun [Verfasser], and Hermann [Akademischer Betreuer] Schlüsener. "Comparison of protective effects of four polyphenols on neuropathology and behavior of APP/PS1-21 transgenic mice, a model of Alzheimer’s disease / Chaoyun Li ; Betreuer: Hermann Schlüsener." Tübingen : Universitätsbibliothek Tübingen, 2014. http://d-nb.info/1196878277/34.

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Rey, Nolwen. "Vieillissement olfactif chez la souris normale et chez la souris APP/PS1, modèle de la maladie d'Alzheimer : implications de la neurogenèse et du système noradrénergique." Phd thesis, Université Claude Bernard - Lyon I, 2010. http://tel.archives-ouvertes.fr/tel-00595030.

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Au cours du vieillissement normal et du vieillissement pathologique de type Alzheimer, des altérations olfactives surviennent. Très précoces dans la maladie d'Alzheimer, ces troubles pourraient être signe du développement de la maladie, bien avant l'apparition des signes de déclin cognitif. Il nous paraissait donc important de caractériser et de différencier de manière précise les troubles olfactifs associés au vieillissement normal de ceux associés au vieillissement pathologique et leurs corrélats cellulaires. Notre première étude a pour objectif de clarifier le vieillissement de la fonction
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Ogbeide-Latario, Oghomwen. "The Role of Slow-Wave-Sleep in Hippocampus-Dependent Memory in Aging and Alzheimer's Disease." eScholarship@UMMS, 2021. https://escholarship.umassmed.edu/gsbs_diss/1136.

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Aging and Alzheimer’s disease (AD), are associated with disabling sleep and cognitive deficits. Specifically, aging and Alzheimer’s disease is associated with reduced quantity and quality of the deepest stage of sleep, called slow-wave-sleep (SWS). Interestingly, SWS has been implicated in hippocampus-dependent memory in mice. More importantly, sleep deprivation, aging, and AD are all associated with deficits in memory. Therefore, I hypothesize that, in aging and AD, the sleep deficits are, at least in part, responsible for memory impairments and increasing the quantity and quality of SWS will
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Soler, David C. "The PP1 gamma isoforms restore spermatogenesis but not fertility in PP1 gamma null mice." [Kent, Ohio] : Kent State University, 2009. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=kent1259087463.

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Thesis (Ph.D.)--Kent State University, 2009.<br>Title from PDF t.p. (viewed May 17, 2010). Advisor: Srinivasan Vijayaraghavan. Keywords: sperm; spermatogenesis; PP1gamma2; PP1gamma1; mice; transgene. Includes bibliographical references (p. 102-123).
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Cheng, Lina. "Regulation of protein phosphatase 1, PP1 [gamma] 2, in testis/spermatozoa by PPP1R11, PPP1R7 and PPP1R2." [Kent, Ohio] : Kent State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=kent1208813693.

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Laird, Fiona Mary. "Behavioural and neurochemical specification of cognitive deficits displayed in mice with targeted manipulations of PSD-95." Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.620202.

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Pontén, Emeli. "Astrocyte response after irradiation of the juvenile brain : -­‐ a study on C57BL/6 strain mice (p21)." Thesis, Örebro universitet, Institutionen för medicinska vetenskaper, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-55163.

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Books on the topic "PS1 mice"

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Singh, Nalini. Mine to Possess. Penguin Group USA, Inc., 2008.

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Zipf, R. Karl. Compendium of structural testing data for 20-psi coal mine seals. Dept. of Health and Human Services, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, Pittsburgh Research Laboratory, 2009.

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Kogan, Ilana. An in vivo model for PSA production by breast cancer cell-lines growing as xenografts in scid mice. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Singh, Nalini. Mine to Possess (Psy-Changelings, Book 4). Berkley, 2008.

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Book chapters on the topic "PS1 mice"

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Sahu, Bijayani, Amy R. Mackos, Angela M. Floden, Loren E. Wold, and Colin K. Combs. "Particulate Matter Exposure Exacerbates Amyloid-β Plaque Deposition and Gliosis in APP/PS1 Mice." In Advances in Alzheimer’s Disease. IOS Press, 2021. http://dx.doi.org/10.3233/aiad210013.

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Background: Alzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by the accumulation of amyloid-β (Aβ) plaques, neuroinflammation, and neuronal death. There are several well-established genetic and environmental factors hypothesized to contribute to AD progression including air pollution. However, the molecular mechanisms by which air pollution exacerbates AD are unclear. Objective: This study explored the effects of particulate matter exposure on AD-related brain changes using the APP/PS1 transgenic model of disease. Methods: Male C57BL/6;C3H wild type and APP/PS1 mice were exposed to either filtered air (FA) or particulate matter sized under 2.5 μm (PM2.5) for 6 h/day, 5 days/week for 3 months and brains were collected. Immunohistochemistry for Aβ, GFAP, Iba1, and CD68 and western blot analysis for PS1, BACE, APP, GFAP, and Iba1 were performed. Aβ ELISAs and cytokine arrays were performed on frozen hippocampal and cortical lysates, respectively. Results: The Aβ plaque load was significantly increased in the hippocampus of PM2.5-exposed APP/PS1 mice compared to their respective FA controls. Additionally, in the PM2.5-exposed APP/PS1 group, increased astrocytosis and microgliosis were observed as indicated by elevated GFAP, Iba1, and CD68 immunoreactivities. PM2.5 exposure also led to an elevation in the levels of PS1 and BACE in APP/PS1 mice. The cytokines TNF-α, IL-6, IL-1β, IFN-γ, and MIP-3α were also elevated in the cortices of PM2.5-exposed APP/PS1 mice compared to FA controls. Conclusion: Our data suggest that chronic particulate matter exposure exacerbates AD by increasing Aβ plaque load, gliosis, and the brain inflammatory status.
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Zhang, Zhihua, Wenping Gao, Xiaojian Wang, et al. "mTOR-Autophagy: A Key for Geniposide Protection in APP/PS1 Transgenic Mice." In Prime Archives in Aging. Vide Leaf, Hyderabad, 2020. http://dx.doi.org/10.37247/paaging.1.2020.1.

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van Groen, Thomas, Inga Kadish, Aileen Funke, Dirk Bartnik, and Dieter Willbold. "Treatment with Aβ42 Binding d-Amino Acid Peptides Reduce Amyloid Deposition and Inflammation in APP/PS1 Double Transgenic Mice." In Advances in Protein Chemistry and Structural Biology Volume 88. Elsevier, 2012. http://dx.doi.org/10.1016/b978-0-12-398314-5.00005-2.

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Brahma, K., B. Pal, C. Das, et al. "Mineral resource evaluation based on AHP." In Mine Planning and Equipment Selection 2004. Taylor & Francis, 2004. http://dx.doi.org/10.1201/9780203023419.pt1.

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Yang, Jasmine J., and Stephanie B. Seminara. "Reproductive Deficits in Mice LackingTacr3: Closing the Gap between Mice and Men." In BASIC/TRANSLATIONAL/CLINICAL - Late Breaking Research in Endocrinology. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p20.p1-773.

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Holm, Anders, Per-Olof Grande, Bjorn Olde, Fredrik Leeb-Lundberg, and Bengt-Olof Nilsson. "The GPER1 Agonist G-1 Attenuates Aortic Sprouting in Mice." In The Endocrine Society's 93rd Annual Meeting & Expo, June 4–7, 2011 - Boston. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part1.p1.p1-7.

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Lin, Yuezhen, Iyabo Osifeso, Xiaojun Ma, Owen McGuinness, Roy Smith, and Yuxiang Sun. "Ablation of Ghrelin Receptor in Leptin-Deficient Mice Has Paradoxical Effects on Glucose Homeostasis Compared to Ghrelin-Ablated Leptin-Deficient Mice." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part2.p11.p2-517.

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Kaur, V., J. Wood, JL Hall, and MK Thomas. "Metabolic Consequences of TCF7L2 Deficiency in Mice." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part1.p10.p1-465.

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Blouet, Clemence, and Gary Schwartz. "Mediobasal Hypothalamic Crtc1 Regulates Energy Balance in Mice." In BASIC/TRANSLATIONAL/CLINICAL - Late Breaking Research in Endocrinology. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p20.p1-774.

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Lee, Nicola, Daniela Sousa, Amanda Sainsbury, Paul Baldock, and Herbert Herzog. "NPY Signaling in Osteoblasts Controls Glucose Metabolism in Mice." In BASIC/TRANSLATIONAL/CLINICAL - Late Breaking Research in Endocrinology. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p20.p1-781.

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Conference papers on the topic "PS1 mice"

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Luo, Yin P., Li Zhang, Xiao Y. Wu, et al. "Cerebral blood microcirculation measurement in APP/PS1 double transgenic mice at the preclinical stage of Alzheimer’s disease: preliminary data on the early intervention of anodal transcranial direct current stimulation *." In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9175875.

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Schrenk, Bernhard, and Fotini Karinou. "TDM Operation of an Optically Synchronized Low-Cost Coherent Receiver Fed with Mice." In 2018 Photonics in Switching and Computing (PSC). IEEE, 2018. http://dx.doi.org/10.1109/ps.2018.8751222.

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Homer, John, Ashley Whitson, Bruce Whisner, Jeff Yonkey, and Dave Yantek. "Explosion Testing of Relief Valves for Underground Refuge Alternatives." In ASME 2019 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/imece2019-10592.

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Abstract Federal regulations require refuge alternatives (RAs) to be installed in underground coal mines. RAs provide miners safe shelter from life-threatening environments during a mine emergency when escape is not possible. Built-in-place (BIP) RAs require ventilation systems that supply breathable air to occupants. Relief valves provide critical functions to the ventilation system by limiting pressure within the RA, allowing ventilation air to exit while preventing contamination ingress, and protecting occupants from external pressure due to mine explosions. As such, relief valves for BIP R
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Broome, Larry E., Hakim Bouamar, James F. Nelson, and Lu-Zhe Sun. "Abstract PS16-18: Inhibition of mTOR signaling by rapamycin abrogates mammary stem/progenitor cell activity in aged mice." In Abstracts: 2020 San Antonio Breast Cancer Virtual Symposium; December 8-11, 2020; San Antonio, Texas. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.sabcs20-ps16-18.

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Pestell, Richard G., Xuanmao Jiao, Andrew V. Kossenkov, et al. "Abstract PS17-58:Pparg1induces an EGF-EphA2 receptor tyrosine kinase module to promote ErbB2- mammary adenocarcinoma in mice." In Abstracts: 2020 San Antonio Breast Cancer Virtual Symposium; December 8-11, 2020; San Antonio, Texas. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.sabcs20-ps17-58.

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Sainsbury, Bre-Anne, David Sainsbury, and Diana Carroll. "Back-analysis of PC1 cave propagation and subsidence behaviour at the Cadia East mine." In Fourth International Symposium on Block and Sublevel Caving. Australian Centre for Geomechanics, Perth, 2018. http://dx.doi.org/10.36487/acg_rep/1815_10_sainsbury.

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Ju, Cunxiang, Mingkun Zhang, Rui Mao, et al. "Abstract 2228: PD1/CTLA4 humanized mice - A great model for pre-clinical toxicology and efficacy evaluation of macromolecular drugs." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-2228.

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Quixabeira, Dafne C. A., Víctor Cervera-Carrascón, Riikka Havunen, Mikko Siurala та Akseli Hemminki. "Abstract A08: Adenovirus coding for TNF-α and IL-2 proteins mediates abscopal effect in mice receiving anti-PD1 immunotherapy". У Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; November 27-30, 2018; Miami Beach, FL. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm18-a08.

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Marcovina, S., R. Coppola, M. P. Protti, C. Gelfi, and P. M. Mannucci. "EDTA-DEPENDENT MONOCLONAL ANTIBODIES TO HUMAN PROTEIN S." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644294.

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Splenocytes from a Balb/c mouse immunized with purified human protein S (PS) were fused with murine hybridoma cell line SP2/0-Agl4 and cultured in Iscove's medium without addition of fetal bovine serum. Hybrid supernatants were screened for the presence of specific antibodies by solid-phase ELISA and cloned by the limiting dilution technique. Pour clones, named S2, S3, S8, and S10, were selected, recloned several times, and injected intraperitoneally into Balb/c mice for the production of antibody-rich ascitic fluid. The monoclonal antibodies (Mabs), all of IgGl subclass with k light chain, we
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Rosato, RR, D. Davila-Gonzalez, DS Choi, B. Dave, and JC Chang. "Abstract P6-14-02: An anti-PD1 antibody-based therapy results in dramatic reduction of TNBC PDX tumors in humanized mice models." In Abstracts: 2016 San Antonio Breast Cancer Symposium; December 6-10, 2016; San Antonio, Texas. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.sabcs16-p6-14-02.

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Reports on the topic "PS1 mice"

1

Compendium of structural testing data for 20-psi coal mine seals. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 2009. http://dx.doi.org/10.26616/nioshpub2009151.

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Strengthening existing 20-psi mine ventilation seals with carbon fiber-reinforced polymer reinforcement. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 2008. http://dx.doi.org/10.26616/nioshpub2008106.

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