Academic literature on the topic 'Psittaci'

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Journal articles on the topic "Psittaci"

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Santos, F., D. C. Leal, T. F. Raso, B. M. P. S. Souza, R. M. Cunha, V. H. R. Martinez, S. M. Barrouin-Melo, and C. R. Franke. "Risk factors associated with Chlamydia psittaci infection in psittacine birds." Journal of Medical Microbiology 63, no. 3 (March 1, 2014): 458–63. http://dx.doi.org/10.1099/jmm.0.060632-0.

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Chlamydia psittaci is the aetiological agent of chlamydiosis in birds, especially Psittaciformes. The objective of the present study was to detect C. psittaci by means of semi-nested PCR among psittacine birds sold at pet markets and kept as pet birds in Salvador, Bahia, Brazil. Questionnaires were used to identify risk factors involved in the epidemiology of the disease. In addition, the management of birds and cages was observed at each location studied. The frequency of C. psittaci infection was 10.6 % (33/311) in the psittacine birds studied. Birds kept in households were less frequently positive (3.4 %; 5/148) than those at pet markets (17.2 %; 28/163). Among the several factors analysed in the epidemiology of the disease, only population density (P = 0.001) and cage hygiene (P = 0.041) in birds at pet markets were significantly associated with C. psittaci infection. These results demonstrate the presence of C. psittaci infection in Psittaciformes kept as pets and held at pet markets in Salvador, Bahia, showing that this micro-organism is a public health concern. Control measures should be encouraged to prevent the spread of the agent among birds, as well as among employees and customers.
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Vaz, Frederico Fontanelli, Elenise Angelotti Bastos Sipinski, Gláucia Helena Fernandes Seixas, Nêmora Pauletti Prestes, Jaime Martinez, and Tânia Freitas Raso. "Molecular Survey of Pathogens in Wild Amazon Parrot Nestlings: Implications for Conservation." Diversity 13, no. 6 (June 16, 2021): 272. http://dx.doi.org/10.3390/d13060272.

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South America presents the greatest Psittacidae diversity in the world, but also has the highest numbers of threatened parrot species. Recently, exotic viruses have been detected in captive native psittacine birds in Brazil, however, their impacts on the health of wild parrots are still unknown. We evaluated the presence of Chlamydia psittaci, Psittacid alphaherpesvirus 1 (PsHV-1), avipoxvirus and beak and feather disease virus (BFDV) in wild Amazona aestiva, A. brasiliensis and A. pretrei nestlings and in wild caught A. aestiva nestlings seized from illegal trade. Samples were collected from 205 wild nestlings and 90 nestlings from illegal trade and pathogen-specific PCR was performed for each sample. Chlamydia DNA prevalence was 4.7% in A. aestiva and 2.5% in A. brasiliensis sampled from the wild. Sequencing revealed that the C. psittaci sample belonged to the genotype A. PsHV-1, avipoxvirus and BFDV DNA was not detected. These results have conservation implications since they suggest that wild parrot populations have a low prevalence of the selected pathogens and, apparently, they were not reached by the exotic BFDV. Stricter health protocols should be established as condition to reintroduction of birds to the wild to guarantee the protection of Neotropical parrots.
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Merchán, Brayan Marcel, Anny Jaramillo, Teresa Tortola, Pau Abrisqueta, Josep Castellvi, Ramón Medel, Katia Flores, et al. "Ocular Adnexal Marginal Zone Lymphomas (OAMZL): Lack of Evidence for Chlamydia Psittaci (C. Psittaci) Infection in a Clinicopathologic Study from a Single Institution." Blood 124, no. 21 (December 6, 2014): 4467. http://dx.doi.org/10.1182/blood.v124.21.4467.4467.

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Abstract Ocular adnexal lymphomas (OAL) represent up to 55% of all orbital tumors, being extranodal marginal zone lymphoma (OAMZL) the most frequent histological subtype reported in approximately 50% of patients and its etiology and pathogenesis are still not well understood. There are conflicting reports regarding the association between C. Psittaci infection and ocular adnexal lymphoma (OAMZL), in part because this association may vary between different geographical regions. Herein, we analyzed the clinical features and the presence of C. Psittaci infection in a cohort of 28 patients with OAMZL diagnosed from a single Spanish institution. All tumor samples were centrally reviewed by two pathologists, and C. Psittaci infection was determined in tumor specimens by a Semi-nested PCR method. Between January 1984 and April 2009, 28 patients were diagnosed with primary OAMZL. The median age was 70 years (range: 31- 89) and 40% were male. The great majority of patients (79%) were diagnosed in stage I-extranodal (IE), and six patients (21%) had bilateral ocular involvement. IgH rearrangement was confirmed in 25 out of 28 cases with OAMZL. PCR for C. Psittaci infection was negative in all 28 tumor specimens analyzed. Most patients (82%) were treated with immunochemotherapy combinations, being chlorambucil based regimens the most frequently used. Overall, 18 out of 24 patients (75%) achieved a complete response after first-line treatment. Median PFS was of 79 months (IC 95% 36 - 121). Whereas C. Psittaci infection in OAMZLs has been proved in up to 90% of patients in Northern Italy, in this Spanish cohort OAMZLs were not associated with C. Psittaci infection. These findings confirm that the association between C. Psittacci infection and OAML is highly heterogeneous even between the same geographic regions. In these regions where C. Psittaci infection is not prevalent, the search for alternative antigenic stimuli driving the appearance of this lymphoma is warranted. Disclosures No relevant conflicts of interest to declare.
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He, Qing-zhi, Huai-cai Zeng, Yan Huang, Yan-qun Hu, and Yi-mou Wu. "The Type III Secretion System (T3SS) ofChlamydophila psittaciIs Involved in the Host Inflammatory Response by Activating the JNK/ERK Signaling Pathway." BioMed Research International 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/652416.

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Chlamydophila psittaci(C. psittaci) is a human zoonotic pathogen, which could result in severe respiratory disease. In the present study, we investigated the role and mechanism of the type III secretion system (T3SS) ofC. psittaciin regulating the inflammatory response in host cells.C. psittaci-infected THP-1 cells were incubated with the specific T3SS inhibitor INP0007, inhibitors of ERK, p38, or JNK, and the levels of inflammatory cytokines were analyzed using Q-PCR and ELISA. The levels of ERK, p38, and JNK phosphorylation were analyzed by Western blot. Our results verified that INP0007 inhibited chlamydial growthin vitro, but the coaddition of exogenous iron completely reversed the growth deficit. INP0007 inhibited the growth ofC. psittaciand decreased the levels of IL-8, IL-6, TNF-α, and IL-1β. Exogenous iron restored the chlamydial growth but not the production of inflammatory cytokines. These results demonstrated that the expression of inflammatory cytokines during infection was associated with the T3SS which reduced by incubation with ERK and JNK inhibitors, but not with p38 inhibitor. We concluded that the T3SS elicited inflammatory responses by activating the JNK or ERK signaling pathways in the infection ofC. psittaci.
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Araujo, Simone Aparecida Almeida, Washington Luiz Assunção Pereira, Sandro Patroca da Silva, Jedson Ferreira Cardoso, Ednaldo da Silva Filho, Marcella Katheryne Marques Bernal, Fernanda Figueiredo Mendes, and Márcio Roberto Teixeira Nunes. "Clinical and molecular diagnosis of Chlamydophila in captive parrots in Pará State, Brazil." Semina: Ciências Agrárias 40, no. 6 (August 29, 2019): 2603. http://dx.doi.org/10.5433/1679-0359.2019v40n6p2603.

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Parrots (Order Psittaciformes) are globally distributed birds that, together with members of the Columbiformes, represent the most susceptible animals, in regards to infection by Chlamydophila psittaci, which is an obligate, zoonotic, intracellular bacterium that causes chlamydiosis in domestic and wild birds and psittacosis in humans. The aim of the present study was to assess the occurrence of C. psittaci in Brazilian psittacids kept in conservation breeding sites in Pará State, Brazil. Cloacal and oropharynx swab samples were collected from 201 psittacids that were distributed among four breeding sites: Metropolitan Area of Belém (C1 and C2), Northeastern Pará (C3), and Low Amazon (C4). The samples were screened for C. psittaci using semi-nested PCR, and the resulting incidence data were analyzed using proportion and chi-square tests. Chlamydophila infection was confirmed for all the breeding sites, with an overall prevalence of 31.84%, and no species-specific predisposition was observed. Furthermore, 13.93% of the sampled birds eliminated the infectious agent by the cloaca, whereas 11.44% eliminated the agent by the oropharynx, and 6.47% eliminated the agent by both routes. Moreover, there was a significant difference between the incidence of Chlamydophila infection of breeding sites C2 and C3 (p=0.029), which yielded the smallest and largest number of diagnosed cases, respectively. In the present study, most of the birds (27.86%) were considered unapparent carriers of Chlamydophila infection, and only 3.98% of the birds yielded both a positive diagnosis and clinical signs of chlamydiosis.
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Chu, Jun, Yongxia Guo, Guanlong Xu, Qiang Zhang, Zonghui Zuo, Qiang Li, Yihui Wang, and Cheng He. "Chlamydia psittaci Triggers the Invasion of H9N2 Avian Influenza Virus by Impairing the Functions of Chicken Macrophages." Animals 10, no. 4 (April 21, 2020): 722. http://dx.doi.org/10.3390/ani10040722.

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In a pilot study, simultaneous infection with Chlamydia psittaci (C. psittaci) and H9N2 virus induced 20% mortality and severe avian airsacculitis, shedding light on animal models of poultry respiratory diseases. However, the pathogenesis is still unclear. In the current study, we hypothesized that C. psittaci infection execrates macrophage function and facilitates H9N2 infection. To explore the potential mechanism, we studied the effect of C. psittaci and H9N2 on the functions of HD11 cells in vitro by simultaneous infection of C. psittaci and H9N2. At the same time, we used infection with C. psittaci or H9N2 alone as the control groups. The results showed that coinfection with C. psittaci and H9N2 could significantly aggravate the mortality of HD11 cells compared to C. psittaci or H9N2 infection alone. In addition, coinfection with C. psittaci and H9N2 did not induce high C. psittaci loads compared to C. psittaci infection alone at 12- and 24-hours post-inoculation (hpi), but coinfection with C. psittaci and H9N2 could increase the loads of H9N2 compared to H9N2 alone in HD11 cells at 12 hpi. More importantly, inducible nitric oxide synthase (iNOS) expression levels, enzyme activity, nitric oxide (NO) production, and phagocytosis were reduced significantly in the group with C. psittaci and H9N2 coinfection compared to those of H9N2 or C. psittaci alone at 24 hpi. Finally, C. psittaci infection induced robust expressions of type Th2 cytokines interleukin (IL)-4 and IL-10, while interferon gamma (IFN-γ) and tumor necrosis factor-α (TNF-α) displayed a significant decrease compared to H9N2 infection alone at 24 hpi. All the above data indicate that C. psittaci infection can facilitate H9N2 invasion and to aggravate severe avian airsacculitis by impairing macrophage functions.
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Anstey, Susan I., Vasilli Kasimov, Cheryl Jenkins, Alistair Legione, Joanne Devlin, Jemima Amery-Gale, James Gilkerson, et al. "Chlamydia Psittaci ST24: Clonal Strains of One Health Importance Dominate in Australian Horse, Bird and Human Infections." Pathogens 10, no. 8 (August 11, 2021): 1015. http://dx.doi.org/10.3390/pathogens10081015.

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Chlamydia psittaci is traditionally regarded as a globally distributed avian pathogen that can cause zoonotic spill-over. Molecular research has identified an extended global host range and significant genetic diversity. However, Australia has reported a reduced host range (avian, horse, and human) with a dominance of clonal strains, denoted ST24. To better understand the widespread of this strain type in Australia, multilocus sequence typing (MLST) and ompA genotyping were applied on samples from a range of hosts (avian, equine, marsupial, and bovine) from Australia. MLST confirms that clonal ST24 strains dominate infections of Australian psittacine and equine hosts (82/88; 93.18%). However, this study also found novel hosts (Australian white ibis, King parrots, racing pigeon, bovine, and a wallaby) and demonstrated that strain diversity does exist in Australia. The discovery of a C. psittaci novel strain (ST306) in a novel host, the Western brush wallaby, is the first detection in a marsupial. Analysis of the results of this study applied a multidisciplinary approach regarding Chlamydia infections, equine infectious disease, ecology, and One Health. Recommendations include an update for the descriptive framework of C. psittaci disease and cell biology work to inform pathogenicity and complement molecular epidemiology.
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Andersen, Arthur A. "Two New Serovars of Chlamydia Psittaci from North American Birds." Journal of Veterinary Diagnostic Investigation 9, no. 2 (April 1997): 159–64. http://dx.doi.org/10.1177/104063879700900209.

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Five Chlamydia psittaci isolates (1 turkey, 1 psittacine, 1 human, and 2 pigeon isolates) failed to react with serovar-specific monoclonal antibodies to known avian and mammalian C. psittaci serovars and were presumed to represent 1 or more new serovars. The isolates were characterized using restriction endonuclease analysis of the whole genome, polymerase chain reaction-restriction fragment length polymorphism of the major outer membrane protein genome, monoclonal antibody comparisons, and growth in tissue culture. Monoclonal antibodies were produced to the human isolate (MP) and to the psittacine isolate (VS225). The monoclonal antibody results show that the isolates represent 2 new avian serovars (serovars E and F). The restriction fragment length polymorphism analysis of the major outer membrane protein genome demonstrated that the isolates are distinct. The whole genome restriction endonuclease analysis data and the growth patterns in tissue culture indicate that the new serovars are similar to avian serovars recognized previously. A subspecies monoclonal antibody that reacted with serovars A and B also reacted with serovar E, indicating that these serovars are closely related. The results show that these isolates represent 2 new avian serovars, making them the fifth and sixth avian serovars identified in North American birds.
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Lagae, S., and D. Vanrompay. "Innate immune response in avian macrophages elicited by Chlamydia psittaci." Vlaams Diergeneeskundig Tijdschrift 84, no. 3 (June 1, 2015): 133–41. http://dx.doi.org/10.21825/vdt.v84i3.16601.

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Chlamydia psittaci is a gram-negative, obligate, intracellular bacterium, which mainly infects birds and mammals. Not much is known about innate immunity initiated by C. psittaci. The focus of the present study is on chicken macrophage activation and expression of cytokine, chemokine, caspase-1, iNOS and TLR genes during the early phase and mid-cycle period of the developmental cycle of the highly virulent C. psittaci strain 92/1293. C. psittaci significantly augmented the transcript levels for all genes investigated, especially during the mid-cycle period. These results demonstrate a robust innate immune response of chicken macrophages initiated by a C. psittaci infection.
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Cong, W., S. Y. Huang, X. X. Zhang, D. H. Zhou, M. J. Xu, Q. Zhao, A. D. Qian, and X. Q. Zhu. "Chlamydia psittaci exposure in pet birds." Journal of Medical Microbiology 63, no. 4 (April 1, 2014): 578–81. http://dx.doi.org/10.1099/jmm.0.070003-0.

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Chlamydia psittaci is a zoonotic pathogen with a wide range of avian hosts and may be transmitted to humans and cause severe disease. To assess the risk of psittacosis posed by pet birds, the seroprevalence of Chlamydia psittaci antibodies in 360 Eurasian siskins (Carduelis spinus), 289 oriental skylarks (Alauda arvensis) and 36 black-tailed grosbeaks (Coccothraustes migratorius) in Gansu province, north-western China was detected by an indirect haemagglutination assay. Twenty-seven out of 289 (9.34 %) Alauda arvensis, 45 out of 360 (12.50 %) Carduelis spinus and 2 out of 36 (5.56 %) Coccothraustes migratorius were positive for Chlamydia psittaci infection at a cut-off dilution of 1 : 16. The prevalence of Chlamydia psittaci was higher in Carduelis spinus (12.5 %) than in Alauda arvensis (9.34 %) and Coccothraustes migratorius (5.56 %); however, the differences were not statistically significant (P>0.05). Statistical analysis indicated that Chlamydia psittaci seroprevalence in adult pet birds (12.4 %, 67/540) was significantly higher than that in juvenile pet birds (4.83 %, 7/145) (P<0.01). There was no statistical difference in Chlamydia psittaci seroprevalence between male (12.4 %) and female (8.27 %) birds. To our knowledge, this is the first report indicating the seroprevalence of Chlamydia psittaci exposure in pet birds in China. Our results indicate that close contact with pet birds poses the risk of zoonotic transmission of Chlamydia psittaci.
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Dissertations / Theses on the topic "Psittaci"

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McCafferty, Michael Campbell. "Ovine cell mediated immunity to Chlamydia psittaci." Thesis, University of Edinburgh, 1992. http://hdl.handle.net/1842/20002.

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Enzootic Abortion of Ewes (EAE) is an economically important disease of ewes, caused by the gram negative, intracellular bacterium, Chlamydia psittaci. The disease results in lamb loss from abortion and the perinatal death of weak lambs. Vaccines have controlled EAE for more than thirty years, however in the last decade vaccine efficacy has been poor. The primary aim of this project was to examine the cell mediated immune responses to C.psittaci in sheep and to identify potentially immunoprotective antigens for future vaccine studies, by their ability to stimulate both T-cell proliferation and cytokine production. Preliminary studies determined the parameters of an antigen driven, ovine lymphocyte transformation assay for C.psittaci, employing whole chlamydial elementary bodies (EB) as antigen. It was shown that peripheral blood mononuclear cells (PMBC) from post abortion animals would proliferate in response to chlamydial EB, although lymphocytes from naive ewes also proliferated to a lesser degree. Further studies in mice showed this latter response could be caused by a cross reaction with harmless, enteric bacteria. The development of proliferative responses of the PMBC to both EB and mitogens was also measured during gestation. Infection at this time led to the development of lasting T-cell responses and a transient suppression of the response to the T-cell mitogen, Con A. In addition, both mitogen and antigen specific responses were disrupted in the immediate pre-parturition period. These responses returned to control levels soon after lambing. The T-cell proliferative response was further characterised by probing chlamydial EB which had been separated by SDS page electrophoresis and blotted onto nitrocellulose. Individual antigens were then added to cultures of PBMC and T-cell lines generates from post abortion animals. Four antigens were identified with approximate weights of 70, 50, 38 and 30kDa which stimulated proliferation. The ability of individual chlamydial proteins to stimulate cytokine production in these cultures was also tested. The four antigens above also stimulated the production of γ-IFN in the PBMC and T-cell lines from all sheep tested. Finally, the importance of γ-IFN in a chlamydial infection was investigted in an in vivo mouse model, where neutralising γ-IFN with a monoclonal antibody resulted in an increase in the severity of infection in both thymic and athymic mice, when compared with control animals. Increased numbers of viable chlamydiae were isolated from tissues and increased pathological changes and serum interferon levels were demonstrated. The results presented in this thesis provide evidence for the involvement of cell mediated responses in ovine immunity to C.psittaci. Both T-cell proliferation and γ-IFN production can be measured, although how the responses interact with B-cells and antibody has yet to be elucidated.
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Brown, Jeremy Keith. "Immune regulation of Chlamydia psittaci persistence in sheep." Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/22786.

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Chlamydia psittaci is the causative agent of ovine enzootic abortion (OEA), the single most common cause of diagnosed infectious ovine abortion in the United Kingdom. Diagnosis and control of OEA is complicated by the ability of C. psittaci to establish a chronic low level or persistent infection in ewes infected out-with pregnancy. Persistent C. psittaci infection was reproduced in vitro by maintaining infected ovine fibroblastic ST.6 cells in the presence of recombinant ovine interferon gamma (ROvIFN-γ) at physiologically relevant concentrations for up to 63 days after infection. ROvIFN-γ restricted C. psittaci growth in a dose dependent manner, concentrations of 25-100 units of biological activity per millilitre (U/ml) induced and maintained persistence whereas concentrations of 250 U/ml or more eradicated C. psittaci from infected cultures. Attempts to identify and characterise the source of IFN-γ in immune sheep using T-cell lines raised against recombinant C. psittaci proteins were unsuccessful. The mechanisms of ROvIFN-γ mediated restriction of C. psittaci were investigated. Pretreatment of ST.6 cells with recombinant interferon alpha or double stranded RNA at concentrations matched with ROvIFN-γ for anti-viral activity did not restrict C. psittaci growth in ST.6 cells, indicating that the mechanism through which ROvIFN-γ restricts C. psittaci in ovine cells is independent from its anti-viral activities. Nitric oxide was not detected in the supernates of infected ST.6 cells pretrated with ROvIFN-γ and addition of L-NMMA, a potent inhibitor of inducible nitric oxide synthase, did not alter the anti- C. psittaci effects of ROvIFN-γ in this cell line. Furthermore, nitric oxide production could not be demonstrated in primary ovine alveolar macrophages treated with combinations of ROvIFN-γ, LPS, heat inactivated C. psittaci and/or live C. psittaci. Addition of exogenous L-tryptophan partially reversed the anti-chlamydial effects of ROvIFN-γ in ST.6 cells that had been infected with C. psittaci over a 48 hours period and also resulted in the recovery of infectious Chlamydiae from persistently infected ST.6 cells maintained in 50 U/ml of ROvIFN-γ. These findings support a role for up-regulation of tryptophan catabolism in ROvIFN-γ mediated restriction of both C. psittaci growth and persistence in ovine cells.
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Hughes, Susan Elizabeth. "Protective humoral immune responses against ovine abortifacient Chlamydia psittaci." Thesis, University of Edinburgh, 1998. http://hdl.handle.net/1842/29811.

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Breakdown of a native chlamydial vaccine against ovine enzootic abortion (OEA), coupled with the identification of the protective capacity of the major outer membrane protein (MOMP) of C. psittaci, has encouraged research into the use of recombinant forms of MOMP in a third generation OEA vaccine. The prospect of testing all recombinant forms of MOMP in pregnant sheep trials is a daunting one, both in terms of time and money. To circumvent these problems, it was decided that a model system would be required to assess the efficacies of recombinant MOMP constructs. The initial model developed (active immunisation of mice) did not appear to reflect immunisation in sheep. Difficulties were encountered in eliciting seroconversion in mice vaccinated with recombinant antigens. It also demonstrated a possible H-2 link to the MOMP-specific antibody response. Problems associated with the model were overcome to some extent, by the development of a second model (passive transfer of sheep sera to mice). In this case, it was demonstrated that infection of mice susceptible to C. psittaci, could be reduced by passively transferring sera from sheep which had been vaccinated with recombinant antigens. The mechanism of neutralisation in vivo was also examined for this model. The model was further used to identify protective regions within the MOMP by passive transfer of affinity purified antibodies and monoclonal antibodies. Significant protection was afforded by the VS1 and VS2 regions of the MOMP.
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Hulin, Virginie. "Circulation des Chlamydiaceae en filières avicoles, exposition des professionnels et étude de la survie de Chlamydia psittaci." Thesis, Paris Est, 2016. http://www.theses.fr/2016PESC1016/document.

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La chlamydiose aviaire, causée par la bactérie Chlamydia psittaci, représente un risque zoonotique important. L’infection chez l’animal est principalement asymptomatique, mais chez l’Homme elle peut entrainer des pneumopathies atypiques sévères et causer la mort dans les cas les plus graves. Les personnes infectées sont principalement celles exposées régulièrement à des oiseaux, particulièrement dans le cadre professionnel. En France, de nombreux cas humains sont liés à une exposition à des canards Mulard, espèce utilisée pour la production du foie gras. Afin d’évaluer la prévalence des Chlamydiaceae chez les volailles et de caractériser les souches circulantes, des suivis ont été réalisés à différents stades de l’élevage (couvoir, pré-gavage et gavage pour la filière canard, et abattoirs impliqués dans l’abattage de différentes espèces aviaires). Des prélèvements d’air et de poussières ainsi que des suivis sérologiques et biologiques de personnels volontaires ont été effectués en parallèle afin d’évaluer l’exposition des professionnels travaillant au contact des volailles. Des prélèvements environnementaux ainsi que des essais in vitro visant à étudier la survie de C. psittaci ont été réalisés afin de tenter de mieux caractériser les voies de contamination des oiseaux, ce qui pourrait, à terme, permettre de maitriser le risque de contamination par C. psittaci chez l’animal et donc de réduire l’exposition des professionnels. Les résultats ont démontré une prévalence importance de C. psittaci chez le canard Mulard, au contraire des autres volailles qui hébergent très majoritairement C. gallinacea. L’exposition des professionnels aux Chlamydia est réelle, tout au long du processus d’élevage des volailles, mais plus particulièrement en élevages de canards Mulard et à l’abattoir, aussi il convient pour les professionnels de se protéger à chaque contact avec les animaux. L’hypothèse d’une contamination environnementale des animaux se fait de plus en plus claire, avec notamment la mise en évidence d’un lien existant entre les procédures de nettoyage et désinfection et l’excrétion des canards, la description de la survie de C. psittaci en dehors de tout hôte vivant, ou encore la mise en évidence d’une possible survie de C. psittaci au sein d’A. castellanii. La mise au point de moyens de lutte efficaces permettant de réduire voire de supprimer l’excrétion chez les volailles est également nécessaire, dans le but de diminuer l’exposition des professionnels
Avian chlamydiosis is a factor of economic loss to the poultry industry as well as a risk for zoonotic transmission to human. Chlamydia psittaci is the primary avian chlamydial pathogen with zoonotic potential. Although being mainly asymptomatic in birds, it can cause a disease called “psittacosis” in humans, with severe atypical pneumonia that leads to death in the most severe cases. Persons affected are mainly those whose occupations put them at risk of exposure, and a number of recent reports in France have confirmed that most of the human cases seemed to be linked to poultry, especially mule ducks. Currently there is evidence suggesting that avian chlamydiosis in poultry involves a new chlamydial agent, namely C. gallinacea. In order to evaluate the presence of Chlamydiaceae in poultry and the exposure of workers, we conducted four studies in the poultry industries, in duck hatchery, breeding farms and slauhgterhouse, as well as a studie in two poultry slaughterhouses including samples from voluntary workers. Results showed an important asymptomatic carriage of C. psittaci by mule ducks and a real, invisible and unpredictable exposure of workers. The species C. gallinacea was really prevalent in poultry othe than ducks and we still ignore its impact on human. Contamination of animals on farm seems to be mainly made via the environment. In vitro studies have been done to examine the survival of C. psittaci as a function of temperature in a non-nutritiv middle and showed that viable bacteria were still detectable after two months. Finally, the possible interactions between C. psittaci and an amoeba, Acanthamoeba castellanii, were studied and seem to show that the bacteria was able to enter the amoeba but we still ignore if it can survive or not
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Rayes, H. M. "Serological, cultural and experimental studies of Chlamydia psittaci from sheep." Thesis, University of Liverpool, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233854.

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Tönnies, Kirsten. "Darstellung der Haltungsbedingungen von Ziervögeln anhand der Praxis in 50 Zoofachgeschäften in den Jahren 1994 bis 1996 und Beurteilung der dort vorgefundenen Haltungsbedingungen unter Berücksichtigung bestehender rechtlicher und anderer Vorgaben." Giessen VVB Laufersweiler, 2009. http://d-nb.info/99599403X/04.

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Saad, M. Z. "In vitro and in vivo studies on Chlamydia psittaci (ovis) infection." Thesis, University of Liverpool, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.380157.

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Johnson, F. W. A. "Studies on Chlamydia psittaci associated ovine foetopathy in the United Kingdom." Thesis, University of Liverpool, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.372676.

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Schmit, Jean-Luc. "Modèle expérimental de pneumopathie murine à Chlamydia psittaci : application à l'évaluation des antibiotiques." Paris 11, 1991. http://www.theses.fr/1991PA114840.

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Castan, Bernard. "Etude des pneumopathies a chlamydia observees dans un centre hospitalier general de janvier 1989 a avril 1992." Toulouse 3, 1992. http://www.theses.fr/1992TOU31144.

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Books on the topic "Psittaci"

1

Bertolini, Renzo. Psittacosis (ornithosis): A summary of the occupational health concern. Hamilton, Ont: Canadian Centre for Occupational Health and Safety, 1989.

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Kidd, Catherine. Psittacine flute. Montréal: Conundrum Press, 2000.

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Beynon, Peter H. Manual of psittacine birds. Gloucestershire: British Small Veterinary Association, 1996.

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Johnson, Kurt A. 1991 psittacine captive breeding survey: A survey of private aviculture in the United States. Washington, DC (1250 24th St., NW, Washington 20037): Traffic USA, World Wildlife Fund-US, 1992.

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Allen, Catherine M. 1990 psittacine captive breeding survey: A survey of private aviculture in the United States. Washington, DC, U.S.A. (1250 24th St., NW, Washington 20037): Traffic USA, World Wildlife Fund-US, 1991.

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Homberger, Dominique G. The lingual apparatus of the African grey parrot, Psittacus erithacus Linné (Aves: Psittacidae): Description and theoretical mechanical analysis. Washington, DC: The American Ornithologists' Union, 1986.

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Currier, Angela R. Interferon-induced effector mechanisms in murine Chlamydia psittaci infection. 1998.

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Currier, Angela R. Interferon-induced effector mechanisms in murine Chlamydia psittaci infection. 1998.

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Paguirigan, Angelica M. Characterization of Chlamydia psittaci-induced indoleamine 2,3-dioxygenase in human monocyte-derived macrophage cultures. 1993.

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Cho, Kathy S. Effect of transforming growth factor-beta on indoleamine 2,3-dioxygenase-mediated inhibition of Chlamydia psittaci replication. 1995.

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Book chapters on the topic "Psittaci"

1

Stöcker, W. "Chlamydia psittaci." In Springer Reference Medizin, 567–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_721.

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Stöcker, W. "Chlamydia psittaci." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_721-1.

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Essig, Andreas. "Chlamydia psittaci." In Lexikon der Infektionskrankheiten des Menschen, 161–63. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_183.

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Schulz, Eva-Cathrin. "Erläutern Sie Chlamydia psittaci!" In Mikrobiologie für die mündliche Prüfung, 59. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80349-9_60.

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Mårdh, Pers-Anders, Jorma Paavonen, and Mirja Puolakkainen. "Chlamydia psittaci Infections in Humans." In Chlamydia, 263–67. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0719-8_33.

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Mårdh, Pers-Anders, Jorma Paavonen, and Mirja Puolakkainen. "Chlamydia psittaci Infection in Birds." In Chlamydia, 269–70. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0719-8_34.

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Mårdh, Pers-Anders, Jorma Paavonen, and Mirja Puolakkainen. "Chlamydia psittaci Infection in Mammals." In Chlamydia, 271–75. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0719-8_35.

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Ferreri, Andrés J. M., Riccardo Dolcetti, Silvia Govi, and Maurilio Ponzoni. "Ocular Adnexal Lymphoma of MALT-Type and Its Association with Chlamydophila psittaci Infection." In Bacteria and Cancer, 139–63. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-2585-0_6.

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Byrne, Gerald I., Lynne E. Guagliardi, Robin E. Huebner, and Donna M. Paulnock. "Immunomodulation and Chlamydia: Immunosuppression and the Protective Immune Response to C. Psittaci in Mice." In Host Defenses and Immunomodulation to Intracellular Pathogens, 343–52. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4757-5421-6_33.

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Kaplan, Gisela. "Psittacine Cognition." In Encyclopedia of Animal Cognition and Behavior, 1–19. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-47829-6_1761-1.

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Conference papers on the topic "Psittaci"

1

Lyapina, Anna M., Maxim S. Lavrukhin, Larisa P. Padilo, Maria A. Khizhnyakova, Nadejda N. Filonova, Onega V. Ulianova, Galina N. Maslyakova, et al. "LASCA monitoring of dose-dependent dynamics of tumor growth under the influence of Chlamydia psittaci AMK-16 specific ornithosis antigen." In Saratov Fall Meeting 2020: Optical and Nano-Technologies for Biology and Medicine, edited by Valery V. Tuchin and Elina A. Genina. SPIE, 2021. http://dx.doi.org/10.1117/12.2588596.

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Padilo, Larisa P., Onega V. Ulianova, Galina Maslyakova, Alla Bucharskaya, Sergey Dobdin, Irina Subbotina, Anatoly Skripal, et al. "Can the infection, caused by Chlamydia psittaci, produce the stimulation of the growth of a malignant tumor: studying by using t-LASCA technique on animal model." In Saratov Fall Meeting 2019: Optical and Nano-Technologies for Biology and Medicine, edited by Valery V. Tuchin and Elina A. Genina. SPIE, 2020. http://dx.doi.org/10.1117/12.2563841.

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Grammar, Alex W., and Robert L. Williams. "Design of a Robotic Gripper Based on a Psittacus Erithacu Beak." In ASME 2012 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/detc2012-70244.

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Abstract:
A high versatility, low degrees-of-freedom (DOF) gripper was designed based on avian morphology. Grasping mechanisms for robotic manipulators are often developed for application-specific tasks, such as manipulating a single part or performing a repetitive action. In contrast, more dexterous grippers are complex, multiple-DOF mechanisms. A simple, minimal-DOF, versatile gripper has been developed based on the morphology of the Psittacus Erithacu (African Grey Parrot) beak shape. This species is highly intelligent and uses its beak for digging, gripping, climbing, and foraging. Giving a robot a similar capability would allow the platform to pick up targets such as single, small seeds, liquids, large irregular rocks and soft Robocup style balls. By using the beak as a model for a grasping mechanism the design maintains its versatility without the need for a complex system and allows a large range of targets to be gripped. This gripper is intended for use in the new open-source humanoid robot DARwIn-OP.
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"Mitogenomics and phylogenetics of vulnerable and endangered birds of genera Aratinga and Psittacus." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-148.

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Pepperberg, Irene M. "Referential use of American English speech by an African Grey parrot (Psittacus erithacus): phonological output reflects cognitive capacities." In Medical Imaging 1997, edited by Eric A. Hoffman. SPIE, 1997. http://dx.doi.org/10.1117/12.274029.

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