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Relevant bibliographies by topics / Pulmonary surfactant-associated protein A

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Academic literature on the topic 'Pulmonary surfactant-associated protein A'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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Journal articles on the topic "Pulmonary surfactant-associated protein A"

1

Weaver, Timothy E. "Pulmonary surfactant-associated proteins." General Pharmacology: The Vascular System 19, no. 3 (1988): 361–68. http://dx.doi.org/10.1016/0306-3623(88)90029-8.

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Weaver, T. E., J. A. Whitsett, W. M. Hull, and G. Ross. "Identification of canine pulmonary surfactant-associated glycoprotein A precursors." Journal of Applied Physiology 58, no. 6 (1985): 2091–95. http://dx.doi.org/10.1152/jappl.1985.58.6.2091.

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Surfactant-associated glycoproteins A were identified by two-dimensional sodium dodecyl sulfate-polyacrylamide gel electrophoresis of crude surfactant from canine alveolar lavage: an unglycosylated form (protein A1), 27,000–28,000 daltons; glycoprotein A2, 32,000–34,000 daltons; and glycoprotein A3, 37,000–38,000 daltons; pH at isoelectric point (pI) 4.5–5.0. Glycoproteins A2 and A3 were electroeluted and used to prepare a monospecific antiserum that identified proteins A1, A2, and A3 in immunoblots of crude surfactant obtained from dog lung lavage. This antiserum precipitated several proteins

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Qanbar, R., and F. Possmayer. "A Quantitative Method for Detecting Surfactant-Associated Protein C in Pulmonary Surfactant." Analytical Biochemistry 216, no. 2 (1994): 262–70. http://dx.doi.org/10.1006/abio.1994.1040.

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Weaver, Timothy E., Kathryn L. Kropp, and Jeffrey A. Whitsett. "In vitro sulfation of pulmonary surfactant-associated protein-35." Biochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology 914, no. 2 (1987): 205–11. http://dx.doi.org/10.1016/0167-4838(87)90065-3.

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Hawgood, S. "Pulmonary surfactant apoproteins: a review of protein and genomic structure." American Journal of Physiology-Lung Cellular and Molecular Physiology 257, no. 2 (1989): L13—L22. http://dx.doi.org/10.1152/ajplung.1989.257.2.l13.

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In recent years, as the complexity of the surfactant system has become more apparent, investigators with an increasingly diverse set of skills have been attracted to the study of this secretory product of the alveolar epithelium. In addition to advancing our knowledge of the mechanisms underlying the mechanical stability of the lung, recent studies of the surfactant system have also contributed information to less organ-specific biological phenomena such as exocytosis, endocytosis, cell differentiation and lipid-protein interactions in biomembranes. Pulmonary surfactant is not composed of a si

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PUTMAN, Esther, Lambert A. J. M. CREUWELS, Lambert M. G. van GOLDE, and Henk P. HAAGSMAN. "Surface properties, morphology and protein composition of pulmonary surfactant subtypes." Biochemical Journal 320, no. 2 (1996): 599–605. http://dx.doi.org/10.1042/bj3200599.

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Separation of surfactant subtypes is now commonly used as a parameter in assessing the amount of active compared with inactive material in various models of lung injury. The protein content, morphology and surface activity were determined of the heavy and light subtype isolated by differential centrifugation. Here we report the presence of surfactant proteins B and C in the heavy subtype but not in the light subtype. Adsorption studies revealed that separation of fast adsorbing bronchoalveolar lavage resulted in slowly adsorbing heavy and light subtypes. Surfactant, reconstituted from heavy an

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Weaver, T. E., V. K. Sarin, N. Sawtell, W. M. Hull, and J. A. Whitsett. "Identification of surfactant proteolipid SP-B in human surfactant and fetal lung." Journal of Applied Physiology 65, no. 2 (1988): 982–87. http://dx.doi.org/10.1152/jappl.1988.65.2.982.

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Surfactant proteolipid (SP-B) is one of several hydrophobic peptides detected in organic extracts of pulmonary surfactant and associated with the dramatic surface-active properties of surfactant phospholipids. In the present study human SP-B was identified as a protein with a relative molecular weight (Mr) of 7,500-8,000 under reducing conditions; protein of Mr 18,000 was detected under nonreducing conditions by immunoblot analysis of organic extracts of bovine and human surfactant utilizing an antiserum directed against a 60-amino acid synthetic SP-B peptide. This peptide antiserum was subseq

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Ikegami, Machiko, Shawn Grant, Thomas Korfhagen, Ronald K. Scheule, and Jeffrey A. Whitsett. "Surfactant protein-D regulates the postnatal maturation of pulmonary surfactant lipid pool sizes." Journal of Applied Physiology 106, no. 5 (2009): 1545–52. http://dx.doi.org/10.1152/japplphysiol.91567.2008.

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Surfactant protein (SP)-D plays an important role in host defense and pulmonary surfactant homeostasis. In SP-D-deficient ( Sftpd−/−) mice, the abnormal large surfactant forms seen at the ultrastructural level are taken up inefficiently by type II cells, resulting in an over threefold increase in the surfactant pool size. The mechanisms by which SP-D influences surfactant ultrastructure are unknown. We hypothesized that SP-D binds to surfactant immediately after being secreted and influences surfactant ultrastructure conversion. In newborn and adult sheep lungs, immunogold-labeled SP-D was ass

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Yu, S. H., and F. Possmayer. "Reconstitution of surfactant activity by using the 6 kDa apoprotein associated with pulmonary surfactant." Biochemical Journal 236, no. 1 (1986): 85–89. http://dx.doi.org/10.1042/bj2360085.

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Lipid extracts of bovine pulmonary surfactant containing the 6 kDa apoprotein, but lacking the 35 kDa apoprotein, can mimic the essential characteristics of pulmonary surfactant on a pulsating-bubble surfactometer. Reconstituted surfactant can be produced by combining silicic acid fractions containing 6 kDa apoprotein and phosphatidylglycerol with phosphatidylcholine. Treatment of the protein-containing fraction with proteolytic enzymes abolishes its efficacy. These results indicate that the presence of the 6 kDa apoprotein can account for some of the essential physical and biological characte

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Golubinskaya, E. P., T. G. Filonenko, Y. A. Ermola, et al. "Immunohistochemical evaluation of surfactant-associated protein in fibrosis-cavernous pulmonary tuberculosis." Innovative medicine of Kuban, no. 4 (December 28, 2019): 32–39. http://dx.doi.org/10.35401/2500-0268-2019-16-4-32-39.

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Background. Despite the progress and development of scientific directions in various fields of medicine, the problem of tuberculosis and its morphological manifestations remains relevant and is not fully disclosed due to complex pathogenesis, the presence of various clinical forms, therapeutic pathomorphosis, torpid to therapy, the presence of relapses. It is known that a surfactant system occupies a special place in the system of local lung protection.Aim. To study the condition of surfactant-assotiated protein A in the foci of specific destruction and in the surrounding intact lung tissue to

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Dissertations / Theses on the topic "Pulmonary surfactant-associated protein A"

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Li, Jing. "Processing, stability and interactions of lung surfactant protein C /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-582-8/.

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Worthman, Lynn-Ann D. "Surfactant protein A (SP-A) affects pulmonary surfactant morphology and biophysical properties." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0014/MQ34241.pdf.

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Ocampo, Minette C. "Protein-Lipid Interactions with Pulmonary Surfactant Using Atomic Force Microscopy." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1395050693.

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Cai, Jingfei. "Probing the Membrane Association Mechanisms for Pulmonary Collectins and Mammalian Phospholipase C." Thesis, Boston College, 2013. http://hdl.handle.net/2345/3872.

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Thesis advisor: Mary F. Roberts<br>Thesis advisor: Eranthie Weerapana<br>Peripheral proteins from mammals often exhibit multi-domain structures and require metal ions such as calcium as co-factors. This dissertation investigates two types of such proteins -- pulmonary collectins (surfactant proteins A and D) and phosphatidylinositol-specific phospholipase C (PLC) delta1 -- and their interactions with model membranes. One approach to work around the complexity brought upon by such multi-domain protein structure is to use a truncated construct or an isolated single domain. For pulmonary collecti

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Rova, M. (Meri). "The significance of surfactant protein gene polymorphisms in multifactorial infantile pulmonary diseases." Doctoral thesis, University of Oulu, 2005. http://urn.fi/urn:isbn:9514277481.

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Abstract Pulmonary surfactant is a lipid-protein mixture that lines the inner surface of the lung. The main function of surfactant is to reduce surface tension at the air-liquid interface, thus preventing alveolar collapse at the end of expiration. Lack of surfactant is the main cause of respiratory distress syndrome (RDS) in preterm infants. Very preterm babies are at risk of developing a lung disease called bronchopulmonary dysplasia (BPD). The surfactant proteins SP-A, -B, -C and -D have important functions in surfactant structure, homeostasis and innate immunity of the lung. The genes of t

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Henning, Lisa Novik. "Pulmonary surfactant protein a regulation of macrophage toll-like receptor expression, activity, and trafficking /." Columbus, Ohio : Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1211479279.

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Carlson, Tracy Karin. "The Effects of Pulmonary Surfactant Protein-D on Innate Immune Cells and Tuberculosis Pathogenesis." The Ohio State University, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=osu1299708141.

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Frangolias, Despina Daisy. "Candidate genes other than the CFTR gene as possible modifiers of pulmonary disease severity in cystic fibrosis." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/527.

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Cystic fibrosis (CF) is a single gene Mendelian disorder characterized by pulmonary disease and pancreatic insufficiency. Pulmonary disease is the major cause of death in CF patients. Although some cystic fibrosis transmembrane conductance regulator (CFTR) genotypes are associated with less severe disease, patients possessing the same genotype show great variation in pulmonary disease severity and progression. Genes involved in modulating the inflammatory response and genes increasing susceptibility to infection are proposed as modifiers of pulmonary disease severity. Polymorphisms selected f

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Scholz, Dietmar. "Analyse der Surfaktantprotein-A-Gene bei Patienten mit Verdacht auf einen Surfaktantproteindefekt." Doctoral thesis, [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=962717843.

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Krause, Lauren Kendall. "Gene Expression patterns in High-Altitude Pulmonary Edema: A Gene Microway Analysis." Yale University, 2008. http://ymtdl.med.yale.edu/theses/available/etd-08152007-111828/.

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Multiple modulating genes and environmental factors have been implicated in the pathogenesis of high-altitude pulmonary edema (HAPE). However, at the present time, there exists an incomplete understanding of the molecular mechanisms and pathways which underlie constitutional susceptibility. Genome-wide measurements of gene expression in peripheral blood mononuclear cells (PBMCs) were performed using microarray technology. Comparison of gene expression profiles of HAPE-susceptible and resistant individuals resulted in the identification of several previously undescribed candidate genes. RhoA an

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Books on the topic "Pulmonary surfactant-associated protein A"

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Kaushik, Nag, ed. Lung surfactant function and disorder. Taylor & Francis, 2005.

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Lung Surfactant Function and Disorder (Lung Biology in Health and Disease). Informa Healthcare, 2005.

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Mueller, Christian. Acute dyspnoea in the emergency department. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0009.

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Acute dyspnoea is a very common symptom in the acute cardiac care setting. In contrast to current beliefs, acute dyspnoea, as the leading symptom in the emergency department, is associated with about twice the mortality risk, compared to acute chest pain. Rapid and accurate identification of the cause of dyspnoea is critical to the initiation of specific and effective treatment. In most patients, a rapid and accurate diagnosis in the emergency department can be achieved by a combination of vital signs, including pulse oximetry, detailed patient history, physical examination, blood tests (inclu

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Mueller, Christian. Acute dyspnoea in the emergency department. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0009_update_001.

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Acute dyspnoea is a very common symptom in the acute cardiac care setting. In contrast to current beliefs, acute dyspnoea, as the leading symptom in the emergency department, is associated with about twice the mortality risk, compared to acute chest pain. Rapid and accurate identification of the cause of dyspnoea is critical to the initiation of specific and effective treatment. In most patients, a rapid and accurate diagnosis in the emergency department can be achieved by a combination of vital signs, including pulse oximetry, detailed patient history, physical examination, blood tests (inclu

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Lheureux, Philippe, and Marc Van Nuffelen. Management of benzodiazepine poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0320.

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The wide use of benzodiazepines is associated with some inconveniences and are most frequently implicated in acute self-poisoning and accidental poisoning in children. Some of them are recognized as submission drugs, used to commit date rape or robbery. Prolonged use of a benzodiazepine leads to dependence, with a risk of developing a life-threatening withdrawal syndrome. Overdose has usually a good prognosis—most patients recover well with careful observation and prevention of complications, although care should be taken with elderly people, and patients with chronic obstructive pulmonary dis

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Book chapters on the topic "Pulmonary surfactant-associated protein A"

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Pérez-Gil, J., A. Cruz, M. L. F. Ruano, E. Miguel, I. Plasencia, and C. Casals. "Interaction of Pulmonary Surfactant-Associated Proteins with Phospholipid Vesicles." In Molecular Dynamics of Biomembranes. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-61126-1_31.

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Possmayer, Fred, Amanda Cockshutt, and Shou-Hwa Yu. "Pulmonary surfactant-associated proteins: their role in surface tension reduction." In The Surfactant System of the Lung. Macmillan Education UK, 1991. http://dx.doi.org/10.1007/978-1-349-12553-1_2.

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Panos, Ralph J., and James P. Bridges. "Mutations in Surfactant Protein C and Interstitial Lung Disease." In Molecular Basis of Pulmonary Disease. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-59745-384-4_6.

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Abdel-Magid, Ahmed F., Mary Ellen Bos, Urs Eggmann, et al. "Solution Phase Synthesis of the Pulmonary Surfactant KL4: A 21 Amino Acid Synthetic Protein." In ACS Symposium Series. American Chemical Society, 2003. http://dx.doi.org/10.1021/bk-2004-0870.ch011.

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Keough, K. M. W., J. Pérez-Gil, G. Simatos, et al. "Hydrophobic Pulmonary Surfactant Proteins in Model Lipid Systems." In Progress in Membrane Biotechnology. Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-7454-0_17.

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Whitsett, Jeffrey A. "Gene structure of the pulmonary surfactant proteins SP-A, SP-B and SP-C." In The Surfactant System of the Lung. Macmillan Education UK, 1991. http://dx.doi.org/10.1007/978-1-349-12553-1_1.

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Al-Saiedy, Mustafa, Francis Green, and Matthias Amrein. "The Effects of Free Radicals on Pulmonary Surfactant Lipids and Proteins." In Oxidative Stress in Lung Diseases. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9366-3_1.

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Schäfer, A., P. F. Nielsen, T. Voss, et al. "Primary structure elucidation of human and bovine pulmonary surfactant SP-C proteins with covalent bis-(cysteinyl4,5)-thioester-palmitate residues." In Peptides 1990. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3034-9_148.

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Snyder, Jeanne M. "The Biology of the Surfactant-Associated Proteins." In Pulmonary Surfactant: Biochemical, Functional, Regulatory, and Clinical Concepts. CRC Press, 2019. http://dx.doi.org/10.1201/9780367812812-5.

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Morrell, Nicholas W. "Pulmonary hypertension." In Oxford Textbook of Medicine. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.161502_update_002.

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Symptoms of unexplained exertional breathlessness or symptoms out of proportion to coexistent heart or lung disease should alert the clinician to the possibility of pulmonary hypertension, and the condition should be actively sought in patients with known associated conditions, such as scleroderma, hypoxic lung disease, liver disease, or congenital heart disease. Heterozygous germ-line mutations in the gene encoding the bone morphogenetic protein type II receptor (...

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Conference papers on the topic "Pulmonary surfactant-associated protein A"

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W. Agudelo, C., S. Mendizuri, E. Area-Gomez, et al. "Alveolar type 2 cell LDL receptor associated protein 1 regulates surfactant homeostasis and pulmonary function." In ERS Lung Science Conference 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/23120541.lsc-2020.107.

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Arciniegas Flores, R. A., I. A. Vital, K. Medepalli, D. DeMarzo, M. K. Glassberg Csete, and R. A. Alvarez. "Pulmonary Fibrosis Due to a Novel Surfactant Protein Mutation." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5437.

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Furuhashi, K., S. Sakurai, H. Yasui, et al. "Surfactant Protein D in Patients with Chronic Pulmonary Aspergillosis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3694.

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Aono, Yoshinori, Julie G. Redford, Michael F. Beers, and Joe Rae Wright. "The Role Of Surfactant Protein D In Bleomycin Induced Pulmonary Fibrosis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2460.

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Korfei, M., K. Guenther, W. Seeger, and A. Guenther. "Cellular Consequences of Uncharacterized Surfactant Protein-A2 (SFTPA2)-Gene Mutations Associated with Familial Idiopathic Pulmonary Fibrosis (IPF) and Lung Cancer." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7407.

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Bækvad-Hansen, Marie, Morten Dahl, Anne Tybjærg-Hansen, and Børge G. Nordestgaard. "Surfactant Protein-B 121ins2 Heterozygosity, Reduced Pulmonary Function And COPD In Smokers." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2909.

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Noutsios, G. T., A. Grozić, J. G. Ledford, A. Kim, W. Yoo, and E. H. Chang. "Pulmonary Surfactant Protein A2 Polymorphism rs1965708 Associates with Upper Airway Disease Prevalence." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7409.

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Veith, Chrisine, Sigrid Schmitt, Werner Seeger, Norbert Weissmann, and Grazyna Kwapiszewska. "The Cytoskeletal Protein – Cofilin Is Associated With Pulmonary Arterial Hypertension." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1187.

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Lau, Gee W., Zhizhou Kuang, Yonghua Hao, Daniel J. Hassett, and Henry Akinbi. "Pseudomonas Aeruginosa Flagellum Regulates Multiple Mechanisms Of Resistance To Pulmonary Surfactant Protein-A." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6105.

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Mazur, Witold, Tuula Toljamo, Pentti Nieminen, et al. "Elevation Of Pulmonary Surfactant Protein A In Sputum Supernatants Of Current Cigarette Smokers." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3007.

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