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Journal articles on the topic 'Pusher syndrome'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 November 2024

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1

Matteo, Paci. "Pusher Syndrome." Physical Therapy 84, no. 6 (June 1, 2004): 580–83. http://dx.doi.org/10.1093/ptj/84.6.580.

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2

Roller, Margaret L. "The ‘Pusher Syndrome’." Journal of Neurologic Physical Therapy 28, no. 1 (March 2004): 29. http://dx.doi.org/10.1097/01.npt.0000284775.32802.c0.

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3

Dai, S., E. Clarac, A. Odin, A. Kistner, A. Chrispin, P. Davoine, M. Jaeger, C. Piscicelli, and D. Pérennou. "Lateropulsion syndrome or Pusher syndrome?" Annals of Physical and Rehabilitation Medicine 61 (July 2018): e64. http://dx.doi.org/10.1016/j.rehab.2018.05.141.

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4

Libois, Pierre-Yves, Denis Zanchetta, Adrien De Beer, Maud Ghislain, and Antoine Libois. "« Pusher syndrome » d’étiologie inhabituelle." Neurophysiologie Clinique 47, no. 5-6 (December 2017): 340–41. http://dx.doi.org/10.1016/j.neucli.2017.10.011.

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5

Dai, Shenhao, Céline Piscicelli, Emmanuelle Clarac, Anaïs Odin, Andrea Kistner, Anne Chrispin, Patrice Davoine, Marie Jaeger, and Dominic Pérennou. "Syndrome Pusher ou plutôt syndrome Lateropulsion ?" Neurophysiologie Clinique 48, no. 6 (December 2018): 326. http://dx.doi.org/10.1016/j.neucli.2018.10.039.

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6

Chen, Xiao-Wei, Cheng-He Lin, Hua Zheng, and Zhen-Lan Lin. "A Chinese Patient with Pusher Syndrome and Unilateral Spatial Neglect Syndrome." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 41, no. 4 (July 2014): 493–97. http://dx.doi.org/10.1017/s0317167100018540.

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Objective:To observe clinical manifestations, behavioral characteristics, and effects of rehabilitation on a patient with pusher syndrome and unilateral spatial neglect caused by right thalamic hemorrhage.Methods:Assessment of pusher syndrome was made by the Scale for Contraversive pushing (SCP), and unilateral spatial neglect syndrome was diagnosed using line cancellation, letter and star cancellation, line bisection tests and copy and continuation of graphic sequence test. Behavioral therapy, occupational therapy, reading training and traditional Chinese medicine methods were adopted for treatment of pusher syndrome and unilateral spatial neglect.Results:The patient showed typical pusher syndrome and unilateral spatial neglect symptoms. The pusher syndrome and unilateral spatial neglect symptoms were significantly improved following rehabilitation treatments.Conclusions:Pusher syndrome and unilateral spatial neglect syndrome occurred simultaneously after right thalamic hemorrhage. Early rehabilitation therapy can reduce the symptoms of pusher syndrome and unilateral spatial neglect syndrome and improve motor function.
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7

Mikołajewska, Emilia. "Posterior pusher syndrome — case report." Open Medicine 7, no. 3 (June 1, 2012): 354–57. http://dx.doi.org/10.2478/s11536-011-0145-7.

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AbstractPusher syndrome is classically described as disorder of body orientation in the coronal plane. It is characterized by a tilt towards the contralesional paretic side and a resistance to external attempts to rectify. It occurs mainly in stroke patients, however, non-stroke causes have been described too. In 2010 the concept of the posterior pusher syndrome had been proposed, defined as disturbance of body orientation in the sagittal plane with imbalance, posterior tilt and an active resistance to forward pulling or pushing. The author describes, on the basis of the literature and own research, symptoms and methods of the treatment of the little-known posterior pusher syndrome.
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8

Pérennou, D., B. Amblard, E. M. Laassel, and J. Pélissier. "Comprendre le « Pusher syndrome å." Annales de Réadaptation et de Médecine Physique 42, no. 7 (September 1999): 400. http://dx.doi.org/10.1016/s0168-6054(99)85089-2.

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9

Karnath, Hans-Otto, and Doris Broetz. "Understanding and Treating “Pusher Syndrome”." Physical Therapy 83, no. 12 (December 1, 2003): 1119–25. http://dx.doi.org/10.1093/ptj/83.12.1119.

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“Pusher syndrome” is a clinical disorder following left or right brain damage in which patients actively push away from the nonhemiparetic side, leading to a loss of postural balance. The mechanism underlying this disorder and its related anatomy have only recently been identified. Investigation of patients with severe pushing behavior has shown that perception of body posture in relation to gravity is altered. The patients experience their body as oriented “upright” when the body actually is tilted to the side of the brain lesion (to the ipsilesional side). In contrast, patients with pusher syndrome show no disturbed processing of visual and vestibular inputs determining visual vertical. These new insights have allowed the authors to suggest a new physical therapy approach for patients with pusher syndrome where the visual control of vertical upright orientation, which is undisturbed in these patients, is the central element of intervention.
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10

Baier, Bernhard, Jelena Janzen, Wibke Müller-Forell, Marcel Fechir, Notger Müller, and Marianne Dieterich. "Pusher syndrome: its cortical correlate." Journal of Neurology 259, no. 2 (August 10, 2011): 277–83. http://dx.doi.org/10.1007/s00415-011-6173-z.

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11

Santos-Pontelli, T. E. G., O. M. Pontes-Neto, and J. P. Leite. "‘Posterior pusher syndrome’ or ‘psychomotor disadaptation syndrome’?" Clinical Neurology and Neurosurgery 113, no. 6 (July 2011): 520–21. http://dx.doi.org/10.1016/j.clineuro.2011.01.012.

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12

Karnath, H. O., J. Suchan, and L. Johannsen. "Pusher syndrome after ACA territory infarction." European Journal of Neurology 15, no. 8 (August 2008): e84-e85. http://dx.doi.org/10.1111/j.1468-1331.2008.02187.x.

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13

Karnath, H. O., L. Johannsen, D. Broetz, and W. Kuker. "Posterior thalamic hemorrhage induces "pusher syndrome"." Neurology 64, no. 6 (March 21, 2005): 1014–19. http://dx.doi.org/10.1212/01.wnl.0000154527.72841.4a.

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14

Masdeu, J. C., P. B. Gorelick, H. O. Karnath, L. Johannsen, D. Broetz, and W. Kuker. "Posterior thalamic hemorrhage induces "pusher syndrome"." Neurology 65, no. 10 (November 21, 2005): 1682. http://dx.doi.org/10.1212/wnl.65.10.1682.

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15

Sawa, Kota, Kazu Amimoto, Takuya Miyamoto, and Miko Tamura. "Influence of Subjective Postural Vertical with Closed and Open Eyes in Patients with Hemiplegic and Pusher Behavior with Unilateral Spatial Neglect After Stroke: A Cross-Sectional Study." Brain Sciences 14, no. 11 (October 31, 2024): 1108. http://dx.doi.org/10.3390/brainsci14111108.

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Background: When integrating visual and somatosensory processing into the subjective postural vertical using the Romberg test, patients with hemiplegic can be sorted into either post-stroke or pushers with unilateral spatial neglect (USN). This study aimed to clarify the characteristics of the integrated processing of the integrated subjective postural vertical (ISPV) with open or closed eyes in patients with hemiplegic and/or pusher with USN. Methods: This cross-sectional study included 91 patients with hemiplegic and 45 with pusher and USN. The outcomes included the ratio and sum of SPV with the eyes closed and open. Statistical analyses were performed using the parametric and/or non-parametric Wilcoxon rank-sum test, Mann–Whitney U test, or chi-square test after the Shapiro–Wilk test. Results: The outcomes in the 91 patients with hemiplegic were as follows: moderate-to-severe ISPV with ratio, 1.64°; ISPV sum (ISPVS), 9.41°. The outcomes in the 45 patients with pusher and USN were as follows: moderate-to-severe, ISPV: 1.35°, and ISPVS: 13.96°. No significant differences were observed between the two groups in terms of demographic data or ISPV. However, the number of patients with pusher syndrome was significantly higher in the ISPVS group than in stroke patients with hemiplegic. Conclusions: Adaptation occurs by integrating sensory modalities, and the pusher behavior in patients with USN is characterized by the specific pathophysiology of a two-modality disorder with visual and somatosensory deficits. This study provides key insights into the pathophysiological characteristics of patients with pusher syndrome and USN.
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16

Bailey, Maggie J., and Linda Leivseth. "The ‘pusher’ syndrome in elderly stroke patients." British Journal of Therapy and Rehabilitation 7, no. 1 (January 2000): 11–16. http://dx.doi.org/10.12968/bjtr.2000.7.1.13908.

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17

Saj, A., J. Honoré, Y. Coello, and M. Rousseaux. "The visual vertical in the pusher syndrome." Journal of Neurology 252, no. 8 (July 27, 2005): 885–91. http://dx.doi.org/10.1007/s00415-005-0716-0.

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18

Baier, Bernhard, and Marianne Dieterich. "Pusher syndrome in patients with cerebellar infarctions?" Journal of Neurology 259, no. 7 (December 22, 2011): 1468–69. http://dx.doi.org/10.1007/s00415-011-6348-7.

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19

Sermsiripoka, U. "Rehabilitation of stroke patients with Pusher syndrome." Chulalongkorn Medical Journal 49, no. 3 (March 2005): 123–28. http://dx.doi.org/10.58837/chula.cmj.49.3.1.

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20

Drozd, Agnieszka, Maja Nowacka-Kłos, and Rita Hansdorfer-Korzon. "Possibilities of physiotherapeutic treatment in the case of patients with pusher syndrome." Fizjoterapia Polska 23, no. 3 (August 31, 2023): 146–64. http://dx.doi.org/10.56984/8zg143k5q.

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The consequences of damage to brain structures often includes various types of perceptual and motor disorders. One of them is Pusher Syndrome (PS). It is defined as a disturbance in the perception of the body position in the frontal plane, which in turn makes it difficult to maintain a correct vertical posture. It occurs most often in patients after a stroke of the right hemisphere of the brain, in the posterolateral region of the thalamus, and its frequency is estimated at about 9–50%. However, it is still a little-known syndrome and too rarely diagnosed. Correct, early diagnosis of pusher syndrome and the introduction of appropriate physiotherapy allows us to shorten the patient’s rehabilitation time and speed up his/her recovery. The objective of the study is to present the principles and possibilities of therapy for patients with pusher syndrome.
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21

Miler, Adrian. "Pusher syndrome – the role of the Bobath Concept." Medycyna Ogólna i Nauki o Zdrowiu 21, no. 3 (August 20, 2015): 250–53. http://dx.doi.org/10.5604/20834543.1165348.

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22

Pardo, Vicky, and Sujay Galen. "Treatment interventions for pusher syndrome: A case series." NeuroRehabilitation 44, no. 1 (February 20, 2019): 131–40. http://dx.doi.org/10.3233/nre-182549.

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23

Lee, Jong Hwa, Sang Beom Kim, Kyeong Woo Lee, and Ji Yeong Lee. "Somatosensory Findings of Pusher Syndrome in Stroke Patients." Annals of Rehabilitation Medicine 37, no. 1 (2013): 88. http://dx.doi.org/10.5535/arm.2013.37.1.88.

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24

Cardoen, Stefanie, and Patrick Santens. "Posterior pusher syndrome: A report of two cases." Clinical Neurology and Neurosurgery 112, no. 4 (May 2010): 347–49. http://dx.doi.org/10.1016/j.clineuro.2009.12.007.

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25

Wang, Hung-Che, and Ling-Fu Meng. "Phenomena Correlated to Stroke Patients with Pusher Syndrome." Rehabilitation Practice and Science 33, no. 1 (December 31, 2005): 39–46. http://dx.doi.org/10.6315/2005.33(1)05.

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26

De Bortoli, Patrícia, Silvia Alves Souza, Carolina Zamariola Margossian, Paulo Roberto Garcia Lucareli, José Eduardo Pompeu, and Sandra Maria Alvarenga Anti Pompeu. "Aspectos fisioterapêuticos e prognósticos da Síndrome de Pusher." O Mundo da Saúde 32, no. 2 (June 4, 2008): 215–20. http://dx.doi.org/10.15343/0104-7809.200832.2.12.

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27

Delva, Iryna, Olga Oksak, and Mykhaylo Delva. "TIME COURSE AND PREDICTORS OF RECOVERY FROM LATEROPULSION AFTER HEMISPHERIC STROKE (PROSPECTIVE STUDY)." Eastern Ukrainian Medical Journal 12, no. 1 (2024): 174–82. http://dx.doi.org/10.21272/eumj.2024;12(1):174-182.

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Introduction. In recent years, considerable attention has been paid to the abnormality of body verticality perception in stroke patients. Most often, misperception of body verticality is manifested in the form of lateropulsion and repulsion syndrome. Objective: to study the timing of recovery from lateropulsion (pusher syndrome) and to determine the predictors of lateropulsion resolution in patients with hemispheric strokes. Material and methods. We included in the study patients with hemispheric strokes occurring within the last month. 61 patients were diagnosed with lateropulsion and 9 patients with pusher syndrome, according to the Scale for Contraversive Pushing. After initial examination, patients were subsequently invited for a weekly examination until the body's verticality was normalized. Results. Recovery time from pusher syndrome was significantly longer – 9.0 (95% confidence interval: 7.1–10.4) weeks compared to recovery time from lateropulsion – 5.9 (95% confidence interval: 5.5–6.3) weeks. Among all the studied factors, only spatial hemineglect was a significant independent predictor of a much longer resolution time of lateropulsion (hazard ratio 2.36; 95% confidence interval: 1.20–4.27). The mean duration of lateropulsion in patients with spatial hemineglect was 6.3 (95% confidence interval: 5.8–6.8) weeks, whereas in patients without spatial hemineglect, it was 4.8 (95% confidence interval: 4.3–5.4) weeks. In a subgroup of patients without spatial hemineglect, higher Fazekas scale values were a significant independent predictor of longer resolution time of lateropulsion (hazard ratio 2.38; confidence interval 95%: 1.25–4.48). Conclusions. After hemispheric strokes recovery time from pusher syndrome is much longer than recovery from lateropulsion. Recovery time from lateropulsion is determined by spatial hemineglect and leukoaraiosis severity.
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28

Johannsen, L., D. Broetz, T. Naegele, and H. O. Karnath. ""Pusher syndrome" following cortical lesions that spare the thalamus." Journal of Neurology 253, no. 4 (February 3, 2006): 455–63. http://dx.doi.org/10.1007/s00415-005-0025-7.

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29

Broetz, Doris, Leif Johannsen, and Hans-Otto Karnath. "Time course of ‘pusher syndrome’ under visual feedback treatment." Physiotherapy Research International 9, no. 3 (August 2004): 138–43. http://dx.doi.org/10.1002/pri.314.

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30

Paci, Matteo, and Luca Nannetti. "The pusher syndrome in a patient with cerebellar infarction." Physiotherapy Research International 10, no. 3 (September 2005): 176–77. http://dx.doi.org/10.1002/pri.8.

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31

Kwon, Yong Hyun, Jung Won Kwon, Sang Young Park, Mi Young Lee, Sung Ho Jang, and Chung Sun Kim. "Presence of Ideomotor Apraxia in Stroke Patients with Pusher Syndrome." Journal of Physical Therapy Science 23, no. 4 (2011): 635–38. http://dx.doi.org/10.1589/jpts.23.635.

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32

dos Santos Pontelli, Taiza Elaine Grespan, Octavio Marques Pontes-Neto, José Fernando Colafêmina, Draulio Barros de Araújo, Antonio Carlos Santos, and João Pereira Leite. "Posture control in Pusher syndrome: influence of lateral semicircular canals." Brazilian Journal of Otorhinolaryngology 71, no. 4 (July 2005): 448–52. http://dx.doi.org/10.1016/s1808-8694(15)31197-6.

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33

Yang, M. X., and Y. Jiang. "Study of the burke lateropulsion scale to evaluate Pusher syndrome rehabilitation." Physiotherapy 101 (May 2015): e683. http://dx.doi.org/10.1016/j.physio.2015.03.3527.

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34

Zhang, L., Q. Zhang, Y. Su, X. Zheng, L. Zhang, and S. Chen. "A new treatment of Pusher syndrome based on central integration concept." Annals of Physical and Rehabilitation Medicine 61 (July 2018): e37. http://dx.doi.org/10.1016/j.rehab.2018.05.082.

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35

Honoré, Jacques, Arnaud Saj, Thérèse Bernati, and Marc Rousseaux. "The pusher syndrome reverses the orienting bias caused by spatial neglect." Neuropsychologia 47, no. 3 (February 2009): 634–38. http://dx.doi.org/10.1016/j.neuropsychologia.2008.11.008.

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36

Popalzai, Adeel, Ricardo Cruz, and Lyn Weiss. "Poster 404: Rehabilitation of Pusher Syndrome Following Stroke: A Case Report." PM&R 1 (September 2009): S280. http://dx.doi.org/10.1016/j.pmrj.2009.08.432.

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37

Reding, M., A. David, and B. Volpe. "2-44-07 Neuroimaging study of the pusher syndrome post stroke." Journal of the Neurological Sciences 150 (September 1997): S129. http://dx.doi.org/10.1016/s0022-510x(97)85489-5.

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38

Karnath, Hans-Otto. "Pusher Syndrome – a frequent but little-known disturbance of body orientation perception." Journal of Neurology 254, no. 4 (March 25, 2007): 415–24. http://dx.doi.org/10.1007/s00415-006-0341-6.

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39

Lagerqvist, J., and E. Skargren. "Pusher syndrome: Reliability, validity and sensitivity to change of a classification instrument." Advances in Physiotherapy 8, no. 4 (January 2006): 154–60. http://dx.doi.org/10.1080/14038190600806596.

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40

Chitambira, Benjamin, and Susan Evans. "Repositioning stroke patients with pusher syndrome to reduce incidence of pressure ulcers." British Journal of Neuroscience Nursing 14, no. 1 (February 2, 2018): 16–21. http://dx.doi.org/10.12968/bjnn.2018.14.1.16.

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41

Lafosse, C., M. Pappens, M. Troch, L. Vereeck, and E. Kerckhofs. "P1.082 Evidence for a pathologically re-aligned graviceptive misperception in the pusher syndrome." Parkinsonism & Related Disorders 14 (February 2008): S31. http://dx.doi.org/10.1016/s1353-8020(08)70179-2.

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42

Clark, Ellen, Keith D. Hill, and T. David Punt. "Responsiveness of 2 Scales to Evaluate Lateropulsion or Pusher Syndrome Recovery After Stroke." Archives of Physical Medicine and Rehabilitation 93, no. 1 (January 2012): 149–55. http://dx.doi.org/10.1016/j.apmr.2011.06.017.

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43

Pape, A. E., G. Karabin, N. Schenke, T. Duning, and H. Hildebrandt. "Eine Pilotstudie zur Behandlung der kontraversiven Lateropulsion (»Pusher-Syndrom«) durch Prismenadaption." Neurologie & Rehabilitation 29, no. 02 (2023): 100–106. http://dx.doi.org/10.14624/nr2302003.

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Zusammenfassung Hintergrund: Patienten mit unilateralen Hirnschäden zeigen häufig eine kontraversive Lateropulsion (»pusher syndrome«, PS). Prismenadaption (PA) führt zu einer Neuausrichtung des propriozeptiven und visuellen Koordinatensystems und könnte daher eine wirksame Behandlungsoption für das PS sein. Methode: Das Studiendesign bestand aus einer Serie von Einzelfallinterventionen (n=14) mit vor- und nachheriger Baseline Messung. Patienten in einem subakuten Zustand nach einem zerebrovaskulären Ereignis der rechten Hemisphäre und mit einem mindestens moderatem PS (gemessen anhand der Klinischen Skala für Contraversive PusherSymptomatik (SCP) und einer Abweichung der Sitzbalance zwischen der linken und rechten Körperhälfte von mindestens 10 Prozent) wurden eingeschlossen. Die quantitativ gemessene Veränderung der Gleichgewichtsverteilung beim Sitzen war der primäre Outcome Parameter. Nach drei Baseline-Messungen im Abstand von drei Tagen (Tage 1, 4, 7) folgten die Interventionssitzungen mit PA im Abstand von drei Tagen (Tage 10, 13, 16). Nach 14 Tagen (Tag 30) wurde eine Folgemessung durchgeführt. Wir erhoben auch den Kraftgrad, den Frührehabilitation Barthel-Index (FRBI) und die Functional Independence Measure (FIM) am Tag 1 und zum Zeitpunkt der Nachfolgeuntersuchung. Die Lokalisation der Läsion wurde manuell in den MRIcron-Standard-Hirnatlas übertragen. Ergebnisse: Jede PA-Intervention verbesserte das Sitzgleichgewicht signifikant, was auf eine sofortige Wirkung von PA auf PS hindeutet. Die PA-Effekte nahmen jedoch über den Zeitraum von drei Tagen teilweise wieder ab, und, wie die Baseline-Phase zeigte, es trat eine signifikante Erholung auch unabhängig von PA auf. Die Patienten verbesserten sich auch in der SCP, dem FRBI, der FIM und der Muskelkraft. Aufgrund der generell hohen Remission kann nicht geschlossen werden, dass letztere Verbesserungen mit den PA-Interventionen zusammenhingen. Die Patienten zeigten überlappende Läsionen im rechten Putamen, im präzentralen Gyrus und in der Corona radiata. Schlussfolgerung: PA führte zu einer kurzfristigen Verbesserung der kontraversiven Lateropulsion, die aber womöglich als Nacheffekt der PA zu interpretieren ist. Es bleibt damit unklar, ob sie sich auch nachhaltig auf die Erholung vom PS auswirkt Schlüsselwörter: Schlaganfallrehabilitation, Prismenadaption, Pusher-Syndrom, Lateropulsion, Sitzbalance
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44

Kim, Min-Su. "Effect of Robot Assisted Rehabilitation Based on Visual Feedback in Post Stroke Pusher Syndrome." Journal of the Korea Academia-Industrial cooperation Society 17, no. 10 (October 31, 2016): 562–68. http://dx.doi.org/10.5762/kais.2016.17.10.562.

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45

Hwang, Ki-Kyeong, Su-Young Song, Yeong-Taek Doo, Se-Won Yoon, and Jeong-Woo Lee. "Physical Therapy Clinical Practice and Documentation for Pusher Syndrome in Stroke Patients: Case Report." Journal of the Korean Academy of Clinical Electrophysiology 9, no. 1 (June 30, 2011): 41–49. http://dx.doi.org/10.5627/kace.2011.9.1.041.

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46

Takase, M., M. Shirai, H. Matsushita, H. Umehara, S. Wakabayashi, A. Doi, and I. Inoue. "The severity of unilateral spatial neglect was positively correlated with that of Pusher syndrome." Annals of Physical and Rehabilitation Medicine 61 (July 2018): e35. http://dx.doi.org/10.1016/j.rehab.2018.05.078.

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47

Meng, Lijiao, Raymond C. C. Tsang, Yanlei Ge, Qifan Guo, and Qiang Gao. "rTMS for poststroke pusher syndrome: study protocol for a randomised, patient-blinded controlled clinical trial." BMJ Open 12, no. 8 (August 2022): e064905. http://dx.doi.org/10.1136/bmjopen-2022-064905.

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IntroductionPoststroke pusher syndrome (PS) prevalence is high. Patients with PS require longer rehabilitation with prolonged length of stay. Effective treatment of PS remains a challenge for rehabilitation professionals. Repetitive transcranial magnetic stimulation (rTMS) is a non-invasive neuromodulation technique that is effective and recommended in the clinical guidelines of stroke rehabilitation. However, the role of rTMS for PS has not been examined. The study is to assess the efficacy of a specific rTMS programme for patients with PS in reducing pushing behaviour, enhancing motor recovery and improving mobility, as well as testing the safety of rTMS for patients with PS.Methods and analysisA randomised, patient and assessor blinded sham-controlled trial with two parallel groups will be conducted. Thirty-four eligible patients with PS will be randomly allocated to receive either rTMS or sham rTMS for 3 weeks. The primary assessment outcome is the pushing behaviour measured by the Burke Lateropulsion Scale and Scale for Contraversive Pushing. The secondary outcomes are the motor functions and mobility measured by the Fugl-Meyer Assessment Scale (motor domain) and Modified Rivermead Mobility Index, and any adverse events. Assessment will be performed at baseline and 1 week, 2 weeks and 3 weeks after intervention. Repeated-measures analysis of variance will be used for data analysis with the level of significance level set at 0.05.Ethics and disseminationThe protocol has been approved by the Biomedical Ethics Committee of West China Hospital, Sichuan University on 23 March 2022 (2022-133). The trial findings will be published in peer-reviewed journals.Trial registration numberChinese Clinical Trial Registry (ChiCTR2200058015).
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48

Johannsen, Leif, Monika Fruhmann Berger, and Hans-Otto Karnath. "Subjective visual vertical (SVV) determined in a representative sample of 15 patients with pusher syndrome." Journal of Neurology 253, no. 10 (June 20, 2006): 1367–69. http://dx.doi.org/10.1007/s00415-006-0216-x.

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49

Ticini, Luca Francesco, Uwe Klose, Thomas Nägele, and Hans-Otto Karnath. "Perfusion Imaging in Pusher Syndrome to Investigate the Neural Substrates Involved in Controlling Upright Body Position." PLoS ONE 4, no. 5 (May 29, 2009): e5737. http://dx.doi.org/10.1371/journal.pone.0005737.

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Jang, Sung Ho, and Han Do Lee. "Recovery of an injured medial lemniscus with concurrent recovery of pusher syndrome in a stroke patient." Medicine 97, no. 22 (June 2018): e10963. http://dx.doi.org/10.1097/md.0000000000010963.

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