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Dissertations / Theses on the topic 'Pylor'

Author: Grafiati

Published: 5 June 2025

Last updated: 1 August 2025

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1

CARUSO, ROBERTA. "Espressione e ruolo funzionale dei linfociti Th-17 nella gastrite correlata all'infezione da helicobacter pylor." Doctoral thesis, Università degli Studi di Roma "Tor Vergata", 2008. http://hdl.handle.net/2108/457.

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L'infezione da Helicobacter pylori (Hp) è caratterizzata da una marcata infiltrazione di cellule infiammatorie nella mucosa gastrica. I meccanismi molecolari che controllano la risposta infiammatoria associata all'infezione sono complessi, ma diverse evidenze suggeriscono che citochine, localmente prodotte, contribuiscano a sostenere tale infiammazione. Già precedentemente abbiamo dimostrato che la produzione dell' interleuchina (IL)-17 è aumentata nella mucosa gastrica di paziente con infezione da Hp, e che tale citochina stimoli la sintesi di IL-8, il principale fattore coinvolto nella che

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2

Minozzo, Bruno Rodrigo. "AÇÃO GASTROPROTETORA DA FRAÇÃO METANÓLICA DAS CASCAS DE Euphorbia umbellata (PAX) BRUYNS: ENVOLVIMENTO DE CICLOOXIGENASES, ÓXIDO NÍTRICO E SEU PAPEL ANTIOXIDANTE." UNIVERSIDADE ESTADUAL DE PONTA GROSSA, 2015. http://tede2.uepg.br/jspui/handle/prefix/96.

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Made available in DSpace on 2017-07-21T14:13:06Z (GMT). No. of bitstreams: 1 Bruno Rodrigo Minozzo.pdf: 2630206 bytes, checksum: 3bd9ced03193a1954969ce2be98510b3 (MD5) Previous issue date: 2015-12-02<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>A mucosa gástrica é continuamente exposta a agentes lesivos endógenos (ácido clorídrico, pepsina, bile, espécies reativas de oxigênio) e exógenos (álcool, anti-inflamatórios, fumo, Helicobacter pylori) que estão envolvidos na patogênese das úlceras. De uma forma geral é aceito que a doença ocorre quando há um desbalanço entre os f

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3

Ferreira, Pedro Manuel Negreiro de Moura. "Helicobacter pylori." Master's thesis, Universidade da Beira Interior, 2008. http://hdl.handle.net/10400.6/801.

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O Helicobacter pylori (HP) constitui uma entidade fundamental quando nos reportamos à patologia gastro-duodenal. As indicações para a erradicação desta bactéria constituem, actualmente, uma das grandes controvérsias na área da Gastroenterologia. Com o objectivo principal de estabelecer o actual ponto de situação no que diz respeito às indicações para a erradicação do HP, realizámos uma revisão sistemática aprofundada do tema com base em artigos científicos de boa qualidade metodológica. Existem duas indicações inequívocas para efectuar o tratamento de erradicação do HP: a doença péptic

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4

Loos, Chantal. ""Campylobacter pylori" : apport du laboratoire de bactériologie dans une étude multidisciplinaire." Paris 5, 1989. http://www.theses.fr/1989PA05P015.

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5

Garrett, June Kazumi. "Inhibition of Helicobacter pylori by Wild Blueberry Phenolics." Fogler Library, University of Maine, 2009. http://www.library.umaine.edu/theses/pdf/GarrettJK2009.pdf.

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6

Coelho, Filipa Maria Meireles da Cunha. "Helicobacter pylori : eficácia da terapêutica." Master's thesis, Universidade da Beira Interior, 2013. http://hdl.handle.net/10400.6/1412.

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Introdução: O Helicobacter pylori adquiriu grande importância durante as últimas décadas, ao ser reconhecido como um importante patogénio que infeta uma grande parte da população humana. Esta bactéria, localizada na mucosa gástrica, apresenta uma relação causal com algumas patologias, tais como: carcinoma gástrico, úlcera péptica, entre outras. A sua erradicação representa um grande desafio para os clínicos. O aumento gradual da falha do tratamento obriga a utilizar novos esquemas terapêuticos e a procurar novas soluções. As taxas de eficácia de erradicação do Helicobacter pylori têm vindo a d

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7

Wanken, Amy Elizabeth. "Helicobacter pylori colonization of the mouse gastric mucosa the Entner-Doudoroff pathway and development of a promoter-trapping system /." Columbus, Ohio : Ohio State University, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1059079727.

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Thesis (Ph. D.)--Ohio State University, 2003.<br>Title from first page of PDF file. Document formatted into pages; contains xiii, 145 p.; also includes graphics (some col.). Includes abstract and vita. Advisor: Kathryn Eaton, Dept. of Veterinary Biosciences. Includes bibliographical references (p. 130-145).

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8

Illingworth, David Simon. "Studies on Helicobacter pylori." Thesis, University of Reading, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333335.

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9

Salamina, M. "Helicobacter pylori Pathogenic Factors." Doctoral thesis, Università degli studi di Padova, 2014. http://hdl.handle.net/11577/3423803.

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From 1994, Helicobacter pylori was classified by WHO (World Health Organization) as a class I carcinogen and its infection has been associated to gastroduodenal disease. It colonizes more than half of worldwide population, with a prevalent infection rate in developed countries. In spite of the majority of infected people are asymptomatic, around 20% develop severe pathologies like peptic ulcers and the 1% lymphoma of the mucosa-associated lymphoid tissue (MALT) and stomach cancer. This significant epidemiological study both of the unique characteristics of H. pylori inspired many scientists, a

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10

Rader, Bethany Anne. "Autoinducer-2 regulation of motility in Helicobacter pylori /." view abstract or download file of text, 2006. http://proquest.umi.com/pqdweb?did=1251819321&sid=6&Fmt=2&clientId=11238&RQT=309&VName=PQD.

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Thesis (Ph. D.)--University of Oregon, 2006.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 80 - 90). Also available for download via the World Wide Web; free to University of Oregon users.

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11

Massiere, Jessica. "La transition épithélio-mésenchymateuse dans les cellules épithéliales gastriques : rôle des microARN régulés par Helicobacter pylori." Thesis, Bordeaux 2, 2011. http://www.theses.fr/2011BOR21835/document.

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Les microARN sont de petits ARN non codant régulant post-transcriptionnellement l’expression de certains gènes. Du fait de leur fort potentiel régulateur, une modification de leur expression peut conduire à l’apparition de pathologies telles que le cancer ou l’inhibition des mécanismes de défense contre des pathogènes. Notre objectif est de caractériser le rôle de certains miARN dans la formation de cancer gastrique dû à Helicobacter pylori. En effet, cette bactérie peut conduire à l’apparition d’adénocarcinome gastrique et de lymphome du MALT. Sa virulence est essentiellement due à la protéin

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12

Boublil, Laurence Yae͏̈l. "Maladie ulcéreuse : physiopathologie, traitements ; rôle d'hélicobacter pylori." Paris 5, 1998. http://www.theses.fr/1998PA05P006.

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13

Carles, Bertrand. "La gastrite chronique associée à Hélicobacter pylori : résultats et suivi à long terme aprés éradication de la bactérie chez des patients ulcéreux duodénaux." Bordeaux 2, 1995. http://www.theses.fr/1995BOR23010.

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14

Yasar, Zemine. "Die Rolle der Helicobacter pylori Infektion bei dyspeptischen Beschwerden und Hyperemesis gravidarum während der Schwangerschaft." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-65787.

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15

Ngoyi, Esther Nina. "Résistance de Helicobacter pylori aux antibiotiques et d’autres substances antimicrobiennes. : Aspects moléculaires des mécanismes de détection." Thesis, Bordeaux, 2016. http://www.theses.fr/2016BORD0442/document.

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Contexte : Amélioration de la prise en charge de l’infection à H. pylori. Matériels et Méthodes : Détection de H. pylori, la résistance à la clarithromycine, la tétracycline, la lévofloxacine, et la détermination des gènes de pathogénicité ont été réalisées par PCR en temps réel, du gène de l’ARNr 23 S, de l’ARNr 16S, PCR classique et séquençage. L’évaluation de la stabilité du mutant résistant par rapport à l’isolat sensible était obtenue par compétition en culture sur cellules gastriques AGS sur une longue période, suivie du séquençage du génome entier. L’évaluation de l’effet de l’extrait d

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16

Rigot, Agnès. "Hélicobacter pylori et gastrites chez l'enfant : à propos de 14 observations." Bordeaux 2, 1991. http://www.theses.fr/1991BOR23113.

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17

Queralt, i. Díaz Núria. "Detecció d'Helicobacter pylori en aigua." Doctoral thesis, Universitat de Barcelona, 2013. http://hdl.handle.net/10803/104263.

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Aquesta Tesi Doctoral té com objectius principals l’estudi d’Helicobacter pylori en mostres aquàtiques de Catalunya, en aigües procedents de sistemes dentals de consultes de dentistes, en saliva i en femtes de pacients amb símptomes gastrointestinals mitjançant el mètode de la PCR. També es va estudiar la supervivència d’Helicobacter pylori en aigua dolça usant un model de laboratori aplicant diferents tècniques d’anàlisi i es va interpretar el canvi de morfologia, la viabilitat i la culturabilitat de la bactèria així com la detecció i quantificació del seu ADN durant un període de temps concr

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18

Goto, Hidemi. "Helicobacter pylori and gastric diseases." Nagoya University School of Medicine, 2003. http://hdl.handle.net/2237/5386.

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19

Phillips, Rosemary Helen. "Metal ions and Helicobacter pylori." Thesis, King's College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408125.

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20

Kivi, Mårten. "Aspects of Helicobacter pylori transmission /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-422-8/.

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21

Jones, Hilary Francis. "Helicobacter pylori survival in macrophages /." Title page and abstract only, 2004. http://web4.library.adelaide.edu.au/theses/09SBT/09sbtj762.pdf.

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22

Thomas, Julian Edward. "Helicobacter pylori infection in childhood." Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300216.

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23

Chatsuwan, Tanittha. "Antibiotic resistance in Helicobacter pylori." Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/24320.

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Helicobacter pylori is a gram negative, microaerophilic bacteria that plays an important role in chronic gastritis and peptic ulcer disease. Multiple antimicrobial therapies including combinations of clarithromycin, metronidazole or amoxycillin and proton pump inhibitor have been used to eradicate H. pylori. Antimicrobial resistance in H. pylori has been associated with treatment failure. In this study, antimicrobial susceptibility patterns of amoxycillin, ciprofloxacin, clarithromycin, erythromycin, tetracycline and metronidazole were evaluated in 110 H. pylori strains isolated from 454 antra

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24

Cunha, Ana Raquel Barreira. "Cancro Gástrico e Helicobacter Pylori." Master's thesis, Instituto de Ciências Biomédicas Abel Salazar, 2009. http://hdl.handle.net/10216/52733.

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25

Koskenpato, Jari. "Helicobacter pylori and functional dyspepsia." Helsinki : University of Helsinki, 2001. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/koskenpato/.

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26

Cunha, Ana Raquel Barreira. "Cancro Gástrico e Helicobacter Pylori." Dissertação, Instituto de Ciências Biomédicas Abel Salazar, 2009. http://hdl.handle.net/10216/52733.

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27

Ben, Cheikh M'Hamed Laurence De Korwin Jean-Dominique. "Diagnostic en médecine générale." [S.l] : [s.n], 2003. http://www.scd.uhp-nancy.fr/docnum/SCDMED_T_2003_GONDA_BEN_CHEIKH_M_HAMED_LAURENCE.pdf.

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28

Palau, de Miguel Montserrat. "Detection of Helicobacter pylori Microevolution and Multiple Infection, and Genomic Analysis of Helicobacter pylori Strains." Doctoral thesis, Universitat de Barcelona, 2019. http://hdl.handle.net/10803/668148.

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In the past decades, Helicobacter pylori has received the attention of many researchers because of its known relation with gastric cancer. Although many studies have tried to decipher the exact relation between the bacteria and cancer state, and several virulence factors have been discovered, an exact answer has not been found yet. Further work should be made in order to study more accurately the genome of this bacterium and to understand its precise involvement. The bacterium is characterised for a highly genetic diversity, meaning it is continuously changing in order to adapt itself to its h

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29

Rapp, Nathalie. "Gastrites chroniques et formes folliculaires associées à hélicobacter pylori : aspects histologiques et endoscopiques : étude prospective de 445 observations." Bordeaux 2, 1993. http://www.theses.fr/1993BOR2M139.

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30

Warren, John Robin. "The discovery and pathology of H pylori /." Title page and contents only, 1999. http://web4.library.adelaide.edu.au/theses/09MD/09mdw289.pdf.

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31

Ye, Fang. "The role of DNA supercoiling in the coordinated regulation of gene expression in Helicobacter pylori." Doctoral thesis, [S.l. : s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=973410698.

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32

Lundin, Annelie. "Diversity and persistence of Helicobacter pylori /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-885-8/.

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33

Wong, Wai-man Raymond, and 王衛民. "The pathology, diagnosis and treatment of helicobacter pylori infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B23331707.

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34

Pantzar, Martina. "Interactions of vitronectin and plasminogen with Helicobacter pylori." Lund : Lund University, 1998. http://catalog.hathitrust.org/api/volumes/oclc/68945086.html.

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35

Schröder, Gunnar. "TraG-like transporter proteins of type IV secretion systems." [S.l. : s.n.], 2003. http://www.diss.fu-berlin.de/2003/179/index.html.

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36

Niehus, Eike. "Untersuchungen zur Regulation Motilitäts-assoziierter Gene in Helicobacter pylori." [S.l. : s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=974441813.

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37

Houben, Martinus Henricus Maria Gerardus. "Clinical aspects in Helicobacter pylori infections." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2000. http://dare.uva.nl/document/81155.

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38

Gideonsson, Pär. "Helicobacter pylori : molecular insights into regulation of adhesion properties." Doctoral thesis, Umeå universitet, Institutionen för medicinsk kemi och biofysik, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-120466.

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Helicobacter pylori infects the human stomach and triggers an inflammatory response that damages the gastric tissue. This host-pathogen interplay has dire consequences as up to 20 % of infected individuals develop peptic ulcer disease or gastric cancer. Given that half of the world’s population is infected, the number of afflicted humans is staggering and also tells that H. pylori is extremely efficient in spreading and maintaining infection. To enable persistent infection many factors play a role, but one important feature of H. pylori is its impressive ability to adhere to the slimy gastric

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39

Pyndiah, Slovénie. "Etude du protéome de Helicobacter Pylori : Approches moléculaire et biochimique." Bordeaux 2, 2005. http://www.theses.fr/2005BOR21203.

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Nous avons construit plusieurs plasmides intégratifs et réplicatifs pour appliquer la méthode TAP-tag à l'étude du protéome de la bactérie Helicobacter pylori. Le transfert des plasmides chez H. Pylori a échoué cependant les derniers résultats obtenus montrent que l'intégration d'ADN exogène par double recombinaison homologue dans le génome bactérien semble être une alternative. Une seconde approche utilisant la méthode BN-PAGE d'électrophorèse en conditions natives couplée à une analyse en LC-MS/MS a permis l'identification de 91 complexes membranaires et cytoplasmiques dont 36 complètement n

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40

Björkholm, Britta. "Helicobacter pylori : cellular interactions and pathogenesis /." Stockholm : [Karolinska institutets bibl.], 2001. http://diss.kib.ki.se/2001/91-7349-084-9/.

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41

Unge, Peter. "Pharmacological therapy of Helicobacter pylori infection /." Linköping : Univ, 2002. http://www.bibl.liu.se/liupubl/disp/disp2002/med734s.pdf.

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42

Gunn, Melanie Catherine. "Clinical relevance of Helicobacter pylori genotypes." Thesis, University of Leicester, 2000. http://hdl.handle.net/2381/29349.

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43

Boughan, P. K. "Innate immune defence to Helicobacter pylori." Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1444551/.

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Helicobacter pylori exhibits tropism for the human stomach causing a spectrum of complications ranging from gastritis to gastric cancer in susceptible individuals. The mechanism(s) that allow the bacteria to persist and cause disease are unfolding. p-defensins are a family of endogenous, epithelial anti-microbial peptides that engage in host defense most prominently at mucosal surfaces. We and others have previously shown that human p-defensin (hBD)-2 and -3 are potent bactericidal agents against H. pylori. At present the identity of signalling pathways involved in host-bacterial cross talk le

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44

Talbi, Patrice. "Macrolides et éradication de Hélicobacter pylori." Bordeaux 2, 1994. http://www.theses.fr/1994BOR23066.

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45

Гребеник, Людмила Іванівна, Людмила Ивановна Гребеник, Liudmyla Ivanivna Hrebenyk та В. А. Сурков. "Уреазні дихальні тести на Helicobacter pylory". Thesis, Видавництво СумДУ, 2009. http://essuir.sumdu.edu.ua/handle/123456789/7095.

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46

Boonjakuakul, Jenni Kim. "Analysis of Helicobacter pylori gene expression /." For electronic version search Digital dissertations database. Restricted to UC campuses. Access is free to UC campus dissertations, 2003. http://uclibs.org/PID/11984.

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47

Ephrati, Véronique. "Hélicobacter pylori et les récidives d'ulcères." Paris 5, 1991. http://www.theses.fr/1991PA05P043.

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48

Castro, Dionicio Inés. "Caractérisation des métabolites secondaires responsables de l'activité anti-Helicobacter pylori d'une plante médicinale péruvienne Cordia lutea Lam. (Boraginaceae)." Thesis, Toulouse 3, 2020. http://www.theses.fr/2020TOU30059.

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Cordia lutea Lam. (Flor de Overo) est une plante utilisée en médecine traditionnelle péruvienne pour le traitement des troubles gastro-intestinaux, des hépatites et des douleurs rénales. Onze composés ont été isolés à partir de l'extrait éthanolique des fleurs de cette plante médicinale sur la base d'un fractionnement bioguidé. Dix de ces composés (cordiasecosides A-J), représentent les premiers exemples de 9,10-seco-29-norcycloartane glycosides. Leurs structures ont été déterminées par des analyses de données RMN et MS ainsi que des analyses cristallographiques aux rayons X. Ces composés ont

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49

Korkut, Dursun Nizam. "Identification et caractérisation structurale du système Toxine-Antitoxine aapA1/IsoA1 de Helicobacter pylori : De l’analyse globale des systémes par bio informatique à l’étude structurale par Résonnance Magnétique Nucluéaire." Thesis, Bordeaux, 2015. http://www.theses.fr/2015BORD0355/document.

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Les systèmes Toxine Antitoxine (TA) sont présents chez la plupart des génomes bactériens. Nous rapportons dans cette étude la présence de tels systèmes chez la bactérie pathogène Helicobacter pylori. Ce nouveau système TA de type I, de la même manière que les autres systèmes de type I décrits, est composé d’une toxine peptidique membranaire (AapA) dont la traduction est inhibée par un ARNnc (IsoA) suivant une interaction côté 5’ non traduit de l’ARNm. La structuration particuliere de l’ARNm a permit l’identification d’orthologue de ce système dans les chromosomes des genres Helicobacter et Cam

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50

Bourzac, Kevin. "A molecular and cellular investigation of Helicobacter pylori's cag pathogenicity island and interactions with host cells /." view abstract or download file of text, 2006. http://proquest.umi.com/pqdweb?index=0&did=1251819291&SrchMode=1&sid=1&Fmt=2&VInst=PROD&VType=PQD&RQT=309&VName=PQD&TS=1181341914&clientId=11238.

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Thesis (Ph. D.)--University of Oregon, 2006.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 80-91). Also available for download via the World Wide Web; free to University of Oregon users.

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