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Journal articles on the topic 'Pylor'

Author: Grafiati

Published: 5 June 2025

Last updated: 15 July 2025

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1

Sachs, George, Karin Meyer-Rosberg, David R. Scott, Klaus Melchers, JaiMoo Shin, and Marie Besancon. "Acid Secretion and Helicobacter pylori." Digestion 58, no. 1 (1997): 8–13. http://dx.doi.org/10.1159/000201514.

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2

KHADEMI, BIJAN, Negar Azar Pira, Mohammad Javad Ashraf, and Abdul Hameed Chohedri. "HELICOBACTER PYLORI IN NASAL POLYPOSIS." Professional Medical Journal 19, no. 04 (2012): 455–61. http://dx.doi.org/10.29309/tpmj/2012.19.04.2256.

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Background: Nasal polyposis is an inflammatory condition of unknown etiology. Recently concern regarding GER orHelicobacter pylori as a possible pathologic cause of nasal polyps has been increasing. The present study was planned to investigate thepresence of Helicobacter Pylor in Nassal polyps by PCR , rapid Urease test and serology. Design: Case control study. Setting: ENT ward ofShiraz, Khalilli Hospital, Iran. Period: April 2006 to March 2008. Materials and Methods: 37 patients with nasal polyps who had undergonenasal endoscopic sinus surgery and 38 control subjects who had undergone septoplasty and turbinectomy. Biopsy specimens of nasal polypsand inferior turbinates were assessed by PCR and Rapid Urease test. Blood sample of both study and control subjects were evaluated for antiH.pylori Ig G by ELISA. HP status was regarded as positive, if 2 tests were positive. Results: Seropositivity was more common in the patientswith nasal polyps (72.97%) than in the control patients (31.57%) (P-value= 0.000) RUT was positive in 9 (24.3%) of 37 patients with nasalpolyps, but was not positive in control group (P-value= 0.001). only 3 of (8.1%) of 37 patients with nasal polyps were positive for both RUT andELISA (P-value =0.115). PCR was negative in all patients and controls. Conclusions: Polypoid tissue can be colonized by some other agentscontaining a urease enzyme other than Helicobacer Pylori. So, result of RUT can be false positive, and addition test may be performed. In theour study by using PCR , we were not able to confirm presence of Helicobacter pylori in the nasal polyps. However, further epidemiologic studiesusing different and specific diagnostic tests with control of documented GER is recommended.

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3

ST, Bozoglu. "A Rare Case: Pylor Stenosis with Anal Atresia and Vesicoureteral Reflux." Journal of Case Reports and Studies 9, no. 1 (2021): 1–7. http://dx.doi.org/10.15744/2348-9820.9.105.

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4

Eidt, Sebastian, and Manfred Stolte. "Antral Intestinal Metaplasia in Helicobacter pylori Gastritis." Digestion 55, no. 1 (1994): 13–18. http://dx.doi.org/10.1159/000201116.

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5

Graham, David Y. "We need to integrate new data concerningHelicobacter pylor into experimental ulcer research." Digestive Diseases and Sciences 40, no. 11 (1995): 2487–89. http://dx.doi.org/10.1007/bf02063261.

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6

Antoniou, Stavros, Athanasios Dimitriadis, Maria Kliridou, Katerina Pavlitou, Heleni Batzili, and Evanthia Malaka. "Prevalence of Helicobacter pylori Antibodies in CAPD Patients." Nephron 75, no. 3 (1997): 358–59. http://dx.doi.org/10.1159/000189563.

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7

萨, 础拉. "Application Progress of High-Dose Dual Therapy in the Eradication of Helicobacter Pylor." Journal of Clinical Personalized Medicine 03, no. 04 (2024): 1371–76. https://doi.org/10.12677/jcpm.2024.34194.

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8

Seyrek, Neslihan, Emine Kocabas, Salih Hazar, Saime Paydas, Necmi Aksaray, and Yahya Sagliker. "Helicobacter pylori Antibodies in Patients on Chronic Hemodialysis." Nephron 72, no. 4 (1996): 725–26. http://dx.doi.org/10.1159/000188978.

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9

Frieri, G., G. De Petris, A. Aggio, et al. "Gastric and Duodenal Juxtamucosal pH and Helicobacter pylori." Digestion 56, no. 2 (1995): 107–10. http://dx.doi.org/10.1159/000201229.

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10

Aydogdu, Bahattin, Serdar Sander, Oyhan Demirali, et al. "Infantile Hypertrophic Pylor Stenosis: The Most Common Cause of Bile-Free Vomiting in Children." Jinekoloji Obstetrik Pediatri ve Pediatrik Cerrahi Dergisi 4, no. 2 (2012): 69–73. http://dx.doi.org/10.5222/jopp.2012.069.

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11

Thomas, A. E., and J. S. Gillin. "Persistence of gastric lymphoma of mucosa-associated lymphoid tissue despite antibiotic eradication therapy for helicobacter pylor." Gastrointestinal Endoscopy 43, no. 4 (1996): 359. http://dx.doi.org/10.1016/s0016-5107(96)80277-x.

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12

Komilova, Baxmal Odilovna, and Musharraf Sadirovna Shodiyeva. "BOLALARDA OSHQOZON VA O'N IKKI BARMOQLI ICHAK YARASINI O'RGANISHNING IMMUNOGENETIK JIHATLARI." Innovation: The Journal of Social Scienses and Researches 1, no. 2 (2022): 86–90. https://doi.org/10.5281/zenodo.7238251.

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<em>HP bilan bog’liq patologiyasi bo’lgan 98 (53,8%) va gastroduodenal zonaning HP-salbiy patologiyasi bo’lgan 84 (46,2%) bolalar tekshirildi, ularda IL-4, TNF-a sitokinlarining tarkibi aniqlandi. Natijalar: o’rganilayotgan Th1-yordamchi sitokinlar ishlab chiqarishning ko’payishi H. pylori bilan bog’liq gastroduodenal patologiyada Th1 tipidagi immunitetning faollashishini ko’rsatadi. </em><em>Shu bilan birga, qon zardobida sitokinlar kontsentratsiyasining ortishi patologik jarayonning dinamikasini yanada aniqroq aks ettiradi. </em>

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13

&NA;. "ROLE OF HELICOBACTER PYLOR INFECTION AND NON-STEROIDAL ANTI-INFLAMMATORY DRUGS IN PEPTIC-ULCER DISEASE: A META-ANALYSIS." Infectious Diseases in Clinical Practice 11, no. 2 (2002): 91. http://dx.doi.org/10.1097/00019048-200202000-00019.

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14

Nowicki, Michael J., and Walter J. Coyle. "Severe Iron Deficiency Anemia and Asymptomatic Nodular Gastroduodenitis: An Uncommon Presentation of Helicobacter pylor Infection in a Child." Clinical Pediatrics 40, no. 2 (2001): 111–14. http://dx.doi.org/10.1177/000992280104000210.

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15

Brown, HannahSofia T., Priya Alagesan, Joseph Wawrzynski, Frances Wang, Meira Epplein, and Katherine S. Garman. "Mo1012 HEALTH DISPARITIES IN PATIENTS UNDERGOING UPPER ENDOSCOPY RELATED TO GASTRIC INTESTINAL METAPLASIA WITH ACTIVE H. PYLOR INFECTION." Gastroenterology 164, no. 6 (2023): S—730. http://dx.doi.org/10.1016/s0016-5085(23)02694-x.

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16

Yanaka, Akinori, Hideo Suzuki, Akira Nakahara, Naomi Tanaka, and Hiroshi Muto. "Prolonged acid inhibition increases apoptosis of fundic gland and accelerates gastric body atrophy in helicobacter pylor-infected C57/BL6 mice." Gastroenterology 118, no. 4 (2000): A888—A889. http://dx.doi.org/10.1016/s0016-5085(00)85692-9.

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17

Liston, R., M. A. Pitt, and A. K. Banerjee. "Lack of Association between Helicobacter pylori and Gastric Atrophy or Intestinal Metaplasia in Elderly Patients." Gerontology 42, no. 2 (1996): 97–103. http://dx.doi.org/10.1159/000213778.

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18

Ching, C. K., S. K. Lam, C. Y. Wong, et al. "Mass endoscopic screening for gastric cancer and initiation of Helicobacter pylor (Hp) eradication therapy in changle of China - a preliminary report." Gastroenterology 108, no. 4 (1995): A456. http://dx.doi.org/10.1016/0016-5085(95)26138-9.

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19

Smoot, Duane T., Linda E. Rosenthal, Harry L. T. Mobley, Oscar Iseri, Simin Zhu, and James H. Resau. "Development of a Human Stomach Explant Organ Culture System to Study the Pathogenesis of Helicobacter pylori." Digestion 46, no. 1 (1990): 46–54. http://dx.doi.org/10.1159/000200277.

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20

Novis, B. H., G. Gabay, G. Leichtmann, M. Peri, J. Bernheim, and I. S. Pomeranz. "Two Point Analysis 15-Minute ¹⁴C-Urea Breath Test for Diagnosing Helicobacter pylori Infection." Digestion 50, no. 1 (1991): 16–21. http://dx.doi.org/10.1159/000200735.

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21

Blecker, Uwe, and Yvan Vandenplas. "Usefulness of Specific IgM in the Diagnosis of Helicobacter pylori Infection." Pediatrics 93, no. 2 (1994): 342–43. http://dx.doi.org/10.1542/peds.93.2.342a.

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Serologic enzyme-linked immunosorbent assay (ELISA) tests for the detection of Helicobacter pylon-specific IgG have been demonstrated to be comparable to invasive techniques.1 Therefore they have been proposed as a inexpensive and reliable screening method for the diagnosis of H pylori infection.2 To study further the value of last-generation serologic tests as a screening technique for H pylori infection, we evaluated a last-generation ELISA kit for the detection of H pylori-specific IgM. During presurgical blood analysis venous blood was taken for last-generation ELISA tests for the detection of anti-H pylori IgG and IgM (Malakit Helicobacter pylori series, Biolab, Belgium) in 452 asymptomatic children (age 1 to 12 years, mean age 6.65 years) admitted for minor elective surgery.

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22

Hassan, Md Ali, MA Ahad, MH Rahman, MSH Bhuiyan, and MAH Khan. "Association between cytotoxin producing H. pylori and gastric carcinoma." Bangladesh Medical Journal Khulna 49, no. 1-2 (2017): 13–17. http://dx.doi.org/10.3329/bmjk.v49i1-2.31819.

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Background: Enormous studies have been conducted worldwide regarding CagA+ status of H. pylori in gastric carcinoma Objective: No study relating CagA+ status and gastric carcinoma has been carried out in our country yet. This study has been designed to see the association between CagA+ H. pylorl strain and gastric carcinomaMethods: For this purpose, a total number of 80 (eighty) patients were selected. Of the 80 (eighty) patients, 40 (forty) were selected as cases (malignant) and the remainder 40 (forty) were selected as controls (non malignant). H. pylori was detected by applying non invasive (H. pylori IgG serology and CagA-IgG serology) and invasive (Histology and rapid urease test) technique. Of them Histology was done by Modified giemsa stain which was regarded as gold standard, CagA IgG was detected by ELISA method.Results: In this study, among the 40 cases, 35 (thirty five) possess the CagA+ H. pylori strain and among the 40 controls, 33 (thirty three) bear the CagA+ H. pylori strain. In this study, no significant difference between case and control on the point of CagA-IgG status was found.Conclusion: H.pylori may be a simple initiator and not the actual cause of gastric carcinoma.Bang Med J (Khulna) 2016; 49 : 13-17

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23

Majaliwa, Nashon D., Muhammad Ismail, Edília Botão, et al. "Diagnostic performance of biopsy-based methods for determination of Helicobacter pylori infection in dyspeptic Mozambican patients." Journal of Infection in Developing Countries 18, no. 11 (2024): 1702–8. https://doi.org/10.3855/jidc.19518.

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Background: Helicobacter pylori (H. pylori) is a Gram-negative bacterium capable of colonizing the human stomach, which can lead to various gastrointestinal conditions. Several invasive and non-invasive methods exist for diagnosing H. pylori; however, none can be considered the gold standard. This study aimed to evaluate the performance of three biopsy-based methods (rapid urease test - RUT, histopathology - HIST, and polymerase chain reaction - PCR) in diagnosing H. pylori, and to assess their combined effect in confirming the infection. Methodology: Eighty dyspeptic patients were recruited for this study, and gastric biopsies were collected from each of them using upper digestive endoscopy. H. pylori was diagnosed using three biopsy-based methods: RUT, HIST, and PCR. RUT was performed using the commercially available PYLO DRYTM Kit, HIST was conducted with Hematoxylin & Eosin and Giemsa staining, and PCR was performed by amplifying the 16S rRNA and 23S rRNA genes. The patient had to test positive in at least two combined diagnostic methods to be confirmed as a case. Results: The three biopsy-based methods (RUT, HIST, and PCR) showed positivity rates of 100% (80/80), 35% (28/80), and 65% (52/80), respectively. When all methods were combined to confirm H. pylori infection, 75% (60/80) of cases were confirmed, while the remaining 25% (20/80) were classified as undetermined, as they were positive only for RUT. Conclusions: Despite slight differences, RUT and PCR performed well in diagnosing H. pylori compared to HIST. However, when all three methods were combined, they improved the accuracy of H. pylori diagnosis and infection confirmation.

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24

Zohaib Hassan, Riaz Ahmed, Sajid Hussain, Muhammad Khalil Surani, Asif Karim, and Mubina Ifat. "Helicobacter pylori infection in children with recurrent abdominal pain." Professional Medical Journal 29, no. 05 (2022): 681–85. http://dx.doi.org/10.29309/tpmj/2022.29.05.6822.

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Objective: To find out the frequency of Helicobacter Pylori infection in children with recurrent abdominal pain. Study Design: Cross Sectional study. Setting: Pediatric Medicine, Children Hospital and Institute of Child Health, Multan. Period: November 2020 to May 2021. Material & Methods: A total of 148 children of both genders aged 4-12 years presenting with recurrent abdominal pain were included. Detailed history and physical examination was conducted. Frequency of helicobacter pylori was noted and it association with characteristics of the children studied was noted employing chi square test considering p-value<0.05 as significant. Results: In a total of 148 children with RAP, 83 (56.1%) were male. Overall, mean age was noted to be 7.6±1.9 years while 103 (69.6%) children were aged below 8 years. There were 77 (52%) children who belonged to rural areas whereas 103 (69.6%) were having poor socioeconomic status. Frequency of H. pylori was noted to be positive in 78 (52.7%) children with RAP. Male gender (p=0.001), age between 4-8 years (p=0.012) and poor socioeconomic status (p=0.001) were noted to have significant association with the frequency of H. pylori. Conclusion: Among children with RAP, frequency of H. pylori was very high. The H. pyloi infection was linked with male gender, younger age and low socioeconomic status.

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25

Verdú, E. F., R. Fraser, D. Tiberio, et al. "Prevalence of Helicobacterpylori Infection and Chronic Dyspeptic Symptoms among Immigrants from Developing Countries and People Born in Industrialized Countries." Digestion 57, no. 3 (1996): 180–85. http://dx.doi.org/10.1159/000201334.

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26

Shahbazi, Shaahin, Zahra Vahdat Shariatpanahi, and Nourkhoda Sadeghifard. "Effect of Proton Pump Inhibitor Based Triple Therapy with Apple Cider Vinegar on Helicobacter Pylori Eradication." Galen Medical Journal 3, no. 2 (2014): 90–94. http://dx.doi.org/10.31661/gmj.v3i2.97.

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Background: The purpose of this study was to evaluate the effect of apple cider vinegar plus PPI-based triple therapy on eradication of Helicobacter Pylori (H.pylori) infection.Materials and Methods: 116 patients with H. pylori infection were included in this randomized clinical trial. Patients in control group (n=59) received amoxicillin, clarithromycin, and pantaprazole twice daily. Patients in study group (57 patients) received the same regimen plus 30 mL of apple cider vinegar twice daily. The duration of therapy for study and control were 10 and 14 days respectively. Four weeks after the completion of therapy, Urea breath test was performed to assess the success of H.pylori infection eradication.Results: 86% of patients in control group in comparison to 88% in apple cider vinegar group responded to eradication therapy (P=0.41).Conclusion: Addition of apple cider vinegar to proton pomp inhibitor (PPI)-based triple therapy may decrease the duration of H. pylori treatment but it was not statistically significant. Thus, our results showed no efficacy of apple cider vinegar in H. pylory eradication treatment.

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27

Maylyan, Eduard A., Elena S. Zhadan, Andrey E. Bagriy, Olga A. Trunova та Evgeny V. Prokhorov. "Serum levels of 25-hydroxyvitamin D and interleukin-1β in Helicobacter pylori-infected women with postmenopausal osteoporosis". Человек и его здоровье 26, № 2 (2023): 59–65. http://dx.doi.org/10.21626/vestnik/2023-2/07.

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Dysfunction of the gastrointestinal system in Helicobacter pylori lesions significantly reduces the absorption of not only calcium (Ca), but also vitamin D and other nutrients, the intake of which is critical for bone formation. H. pylory can contribute to the formation of osteoporosis and due to the development of the inflammatory process. The inflammatory process is an important component of the pathogenesis of the above bone disease. Objective: to study of the levels of 25(OH)D and interleukin-1β in blood serum in postmenopausal OP in women with a positive test for antibodies to the Helicobacter pylori CagA antigen. Materials and methods. The study included 250 postmenopausal women, whose indicators (Me [Q1; Q3]) were 62 [56; 68] years, and the duration of the postmenopausal period is 13 [7; 20] years. In all patients, the content of 25(OH)D and interleukin-1β (IL-1β) concentrations were determined in blood serum samples. And total antibodies (IgA, IgM, IgG) to the CagA antigen of the pathogen Helicobacter pylori. Results. In the female group with postmenopausal osteoporosis, the presence of helicobacteriosis is associated with a decrease in the level of 25-hydroxyvitamin D in the blood serum by 14.8% (p=0.045). Serum IL-1β values in postmenopausal women do not depend on H. pylori infection (p>0.05). A negative correlation was found between the values of 25(OH)D and IL-1β in women with osteoporosis and a positive test for total antibodies to H. pylori (rs=-0.25; p<0.05). Conclusion. The results of the study demonstrate that the decrease in vitamin D depends on the infection with Helicobacter pylori in women with postmenopausal osteoporosis (p=0.045). The results of the studies obtained can be used in the diagnostic and treatment-and-prophylactic measures for women in the postmenopausal period.

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28

Philips, Cyriac, and Chhagan Bihari. "THE OCCURRENCE OF SIEVE-LIKE DUODENUM DUE TO HELICOBACTER PYLORI INFECTION IN A CIRRHOTIC PATIENT: CURRENT INSIGHTS, MANAGEMENT, AND THE IMPORTANCE OF H. PYLORI INFECTION AND ITS ASSOCIATIONS WITH CIRRHOSIS." International Journal of Surgery and Medicine 2, no. 4 (2016): 218. http://dx.doi.org/10.5455/ijsm.helicobacter-pylory-infection.

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29

Sirchak, Ye S., та S. V. Patskun. "ОСОБЛИВОСТІ КИСЛОТОУТВОРЮВАЛЬНОЇ ФУНКЦІЇ ШЛУНКА У ХВОРИХ НА ХРОНІЧНИЙ ГАСТРИТ ТА ЦУКРОВИЙ ДІАБЕТ 2 ТИПУ". Здобутки клінічної і експериментальної медицини, № 1 (25 квітня 2019): 125–28. http://dx.doi.org/10.11603/1811-2471.2019.v0.i1.10062.

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Через високу захворюваність і смертність від хвороб, викликаних Helicobacter pylorі (HР), значні зусилля були спрямовані на визначення точних механізмів, за допомогою яких цей патоген викликає запалення шлунка, оскільки дані механізми не є достатньо дослідженими. Мета – оцінити особливості кислотоутворювальної функції шлунка у хворих на хронічний гастрит (ХГ) та цукровий діабет (ЦД) 2 типу залежно від наявності чи відсутності Helicobacter pylori. Матеріал і методи. Обстежено 80 пацієнтів, середній вік яких склав (57,6±2,3) року. Дане дослідження проводили за участі 45 (56,3 %) жінок та 35 (43,7 %) чоловіків. Всі хворі були поділені на дві групи залежно від інфікування Helicobacter pylori. До І групи увійшли 60 (75 %) осіб, які були НР-позитивними, а другу групу сформовано з 20 (25 %) осіб – НР-негативних хворих на ЦД 2 типу та хронічний гастрит. Результати. У пацієнтів із ЦД 2 типу та ХГ залежно від наявності чи відсутності НР виявлено достовірну різницю між кислотоутворювальною функцією шлунка. Тож пацієнти, які є НР-позитивними, частіше мали порушення кислотоутворювальної функції у вигляді вираженої гіперацидності (68,3 %), а пацієнти без НР мали помірну гіперацидність (75 %). Висновок. У більшості хворих на ЦД 2 типу та ХГ при експрес рН-метрії кислотоутворювальної функції шлунка виявлено гіперацидний стан.

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30

Laserna Estrada, Andres Felipe, Julián Esteban Barahona Correa, and Luz Helena Alba Talero. "Manejo de la infección por Helicobacter pylori: apreciación crítica de la literatura." Universitas Médica 59, no. 3 (2018): 1–13. http://dx.doi.org/10.11144/javeriana.umed59-3.pylo.

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Introducción: la infección por Helicobacter pylori es un problema de salud pública, dada su relación con cáncer gástrico. El incremento de la resistencia bacteriana limita la erradicación efectiva, a pesar del empleo de diferentes esquemas de tratamiento. Métodos: revisión de la literatura en la base de datos Pubmed/Medline entre el 1 de enero de 2015 y el 31 de diciembre de 2016 sobre el manejo del Helicobacter pylori. Resultados: se incluyeron 26 artículos. La terapia secuencial sobresale como opción de tratamiento de primera línea para escenarios como Colombia. La implementación de coadyuvantes puede influir en las tasas de erradicación. Los estudios de epidemiología local y costoefectividad son escasos. Conclusiones: el uso y conocimiento adecuado de los esquemas de manejo puede disminuir los costos para el sistema, la resistencia antimicrobiana y favorecer la erradicación de patógenos. Se requieren estudios para generar recomendaciones locales.

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31

SHAMS, F., G. N. TAYYAB, M. S. KHOKHAR, S. A. CHOUDHRY, A. HAFEEZ, and A. RAZA. "Persistence of Symptoms After Successful Eradication of Helicobacter Pylon in the Duodenal Ulcer Disease." Annals of King Edward Medical University 4, no. 4 (2020): 52–54. https://doi.org/10.21649/akemu.v4i4.3992.

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An Initial ulcer healing has never been a problem rather it is the relapse of the ulcer which is a clinical challenge, and if (Helicobacter Pylon) Hp eradication can be achieved, ulcer relapse is not expected. However it has been a common clinical experience that despite successful therapy these patients continue to suffer from upper GI symptoms. An audit was made to scrutinize the symptoms up to six months after the helicobacter pylori was successfully eradicated. 100 symptomatic endoscopically proven duodenal ulcer patients were included in the study and were given Lanzoprazole 30mg bid, Amoxicillin 1gm bid, Clarithromycin 250mg bid with and without Tinidazole 500mg bid for seven days. Ulcer healing and H pylon eradication was confirmed after three months by endoscopic assessment, mucosal rapid urease test and gastric antrum mucosal biopsy. 86 patients completed the seven day treatment and at the end of three months 72 patients were confirmed to be ulcer free and H. pylon negative. Only 51 patients could be followed upto six months after the eradication treatment and 20 patients were found to have upper GI type symptoms. The complaints were as follows: continuing abdominal pain (10 patients), water brash (9 patients), heart burn (5 patients), gas and flatulence (15 patients), and incomplete evacuation of bowels (4 patients). Majority of the patients had more than one symptom ( 16 patients ) and most common combination was of flatulence, gas, abdominal pain and water brash (8 patients).It appears that Hp eradication helps us in avoiding ulcer and its complications but a good percentage of people continue to complain of upper GI type symptoms afterwards as well.

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32

Moeller, Roger W. "A Post-Apocalyptic View of Archaeology." North American Archaeologist 21, no. 4 (2000): 351–66. http://dx.doi.org/10.2190/g9uj-px9m-pylr-d6c3.

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33

Dore, M. P., A. R. Sepulvedat, G. B. Cherchi, et al. "Helicobacter pylon DNA is not detected in atherosclerotic plaques obtained from patients with H. pylori infection." Digestive and Liver Disease 33 (November 2001): A81. http://dx.doi.org/10.1016/s1590-8658(01)80478-3.

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34

Iwai, Komei, Tetsuji Azuma, Takatoshi Yonenaga, et al. "Association between dental caries and Helicobacter pylori infection in Japanese adults: A cross-sectional study." PLOS ONE 17, no. 7 (2022): e0271459. http://dx.doi.org/10.1371/journal.pone.0271459.

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Helicobacter pylori (H. pylori) is widely known as a cause of gastric disorders. Presence of H. pylori in dental pulp has been reported. Dental caries may influence the presence or absence of systemic H. pylori infection by serving as a source of H. pylori. In this cross-sectional study, we examined whether H. pylori infection in blood were associated with dental caries in Japanese adults. The participants were 752 individuals (513 males and 239 females, mean age 53.8 years) who underwent both H. pylori testing (H. pylori antibody test and pepsinogen test) and dental checkups at the Asahi University Hospital Human Health Center between April 2018 and March 2019. Those diagnosed as positive for H. pylori antibody test or positive for serum pepsinogen test as H. pylori test in the human health checkup were judged as those with H. pylori infection in the blood. In our study, 83 participants (11%) were determined to be infected with H. pylori in the blood. The proportion of those with decayed teeth was higher in participants with H. pylori infection in blood than in those without H. pylori infection in blood (p< 0.001). The logistic analysis showed that presence of H. pylori infection in blood was positively associated with those with decayed teeth (OR, 5.656; 95% CI, 3.374 to 9.479) after adjusting for age, gender, gastric disease, regular dental checkups, antibiotic medication history, and decayed teeth. Furthermore, the proportion of H. pylori infection in blood increased according to number of decayed teeth (p< 0.001). The results indicate that H. pylori infection in blood were associated with decayed teeth. Untreated dental caries may have an impact on systemic H. pylori infection.

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Shafrir, Asher, Michal Shauly-Aharonov, Lior H. Katz, Ora Paltiel, Yishai Pickman, and Zvi Ackerman. "The Association between Serum Vitamin D Levels and Helicobacter pylori Presence and Eradication." Nutrients 13, no. 1 (2021): 278. http://dx.doi.org/10.3390/nu13010278.

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Background: The success of Helicobacter pylori (H. pylori) eradication depends on several host and treatment factors. Serum vitamin D levels may be associated with H. pylori infection and eradication rates. We investigated the association between vitamin D and H. pylori infection and eradication, using a large electronic database based on medical records from a population-based health maintenance organization. Methods: Data regarding adults who underwent H. pylori testing and had vitamin D measurements within one month of H. pylori testing were collected. H. pylori infection was ascertained using urea breath or stool antigen tests. A negative H. pylori test following a positive result implied eradication. Multivariate regression models were constructed to assess associations between H. pylori infection, eradication, and vitamin D. Results: Among 150,483 members who underwent H. pylori testing from 2009 to 2018, 27,077 (18%) had vitamin D measurements. Vitamin D levels were inversely associated with H. pylori infection, p < 0.001. The odds of a positive H. pylori test were 31% higher among patients with vitamin D levels <20 ng/mL, compared with those with levels ≥20 ng/mL (OR 1.31, 99% CI 1.22–1.4, p < 0.001). Purchase of vitamin D supplements was associated with a negative subsequent H. pylori test (p < 0.001). Mean vitamin D levels were moderately higher in those with successful vs. failed H. pylori eradication (19.34 ± 9.55 vs. 18.64 ± 9.61, p < 0.001). Conclusions: Vitamin D levels are associated with H. pylori infection. Increased vitamin D levels are associated with successful H. pylori eradication. Vitamin D may have a role in H. pylori eradication.

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Jurnalis, Yusri Dianne, Yorva Sayoet, and Sari Dewi. "HELICOBACTER PYLORI INFECTION IN CHILDREN." Majalah Kedokteran Andalas 35, no. 1 (2011): 43. http://dx.doi.org/10.22338/mka.v35.i1.p43-49.2011.

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AbstrakFaktor risiko infeksi Helicobacter pylori adalah tinggal di negara berkembang, kondisi sosial ekonomi yang rendah, jumlah anggota keluarga yang banyak, etnik dan genetik. Tatalaksana dan diagnosis Helicobacter pylori belum memuaskan karena adanya resistensi antibiotik pada pasien Helicobacter pylori. Kami melaporkan seorang pasien perempuan usia 8 tahun 6 bulan yang terinfeksi Helicobacter pylori. Diagnosis ditegakkan berdasarkan anamnesis, pemeriksaan fisik, dan hasil laboratorium. Pasien diduga terinfeksi Helicobacter pylori karena mengalami nyeri perut berulang. Dari laboratorium didapatkan serologi IgG Helicobacter pylori positif. Pada hasil endoskopi biopsi ditemukan kuman Helicobacter pylori. Pasien mendapat therapi eradikasi lini pertama untuk infeksi Helicobacter pylori yaitu amoksisilin, klaritromisin dan omeprazol selama dua minggu. Setelah dua minggu pengobatan keluhan pasien tidak ada.Kata kunci: Helicobacter pylori, anak, nyeri perut berulangAbstractRisk factors for acquiring Helicobacter pylori infection include residency of developing country, poor socioeconomic conditions, crowded family, and possibly an ethnic or genetic as predispositions. The diagnosis and management Helicobacter pylori has not been satisfied yet, however, there is problem of increasing resistancy antibiotic due to Helicobacter pylori. Objective: We report a 8 year and 6 month old girl who suffered from Helicobacter pylori. The diagnosis was based on history, clinical finding, and laboratory work-up. Suspicion on the presence of Helicobacter pylori was started when the girl had recurrent abdominal pain. Serology IgG Helicobacter pylori was positive and we had done endoscopic examination and biopsy. Therapy this patient was first line eradication Helicobacter pylori which give amoxicillin, clarithromycin and omeprazole for two weeks. There are no sympthoms after two weeks therapyKey word: Helicobacter pylori, children, recurrent abdominal painLAPORAN KASUS

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Tongtawee, Taweesak, Wareeporn Wattanawongdon, and Theeraya Simawaranon. "Effects of periodontal therapy on eradication and recurrence of Helicobacter pylori infection after successful treatment." Journal of International Medical Research 47, no. 2 (2019): 875–83. http://dx.doi.org/10.1177/0300060518816158.

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Objectives This study aimed to evaluate the effects of periodontal therapy on the efficacy of Helicobacter pylori eradication and on the recurrence of infection after eradication. Methods We conducted a prospective randomized trial on 698 gastric H. pylori-infected patients, of whom 347 received gastric H. pylori treatment alone and 342 received gastric H. pylori treatment plus periodontal therapy. The presence of H. pylori and associated virulence genes were detected by real-time polymerase chain reaction. Results After eradication of gastric H. pylori infection, the recurrence of gastric H. pylori was significantly lower in the gastric H. pylori treatment plus periodontal therapy group than in the group receiving gastric H. pylori treatment alone (OR 0.67; 95% CI 0.45 to 0.99), whereas the eradication rate was not significantly different (OR 0.87; 95% CI 0.68 to 0.98). There was a close relationship between the presence of H. pylori in saliva and its presence in the stomach. Conclusions The oral cavity is an important reservoir for gastric H. pylori infection. Adjunctive periodontal therapy could enhance the efficiency of H. pylori treatment and reduce the recurrence of gastric H. pylori infection.

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Syam, Ari F. "Current situation of Helicobacter pylori infection in Indonesia." Medical Journal of Indonesia 25, no. 4 (2017): 263–6. http://dx.doi.org/10.13181/mji.v25i4.1408.

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Data epidemiologi infeksi Helicobacter pylori (H. pylori) terus berubah dalam beberapa dekade terakhir. Indonesia dilaporkan memiliki prevalensi infeksi H. pylori yang rendah dibandingkan dengan negara lain di Asia. Beberapa penelitian di Indonesia melaporkan bahwa sanitasi yang buruk, usia, agama, etnis merupakan faktor risiko untuk infeksi H. pylori. Dibandingkan dengan tes diagnostik lainnya, tes urine merupakan tes yang dapat diandalkan untuk mendeteksi H. pylori di Indonesia karena tes tersebut bersifat non-invasif dengan harga yang cukup terjangkau dan memiliki akurasi yang tinggi. Meskipun banyak penelitian telah dilakukan mengenai prevalensi infeksi H. pylori pada beberapa etnis di Indonesia, peneliti masih memiliki beberapa pertanyaan yang belum terjawab mengenai infeksi H. pylori di Indonesia. Oleh karena itu, diperlukan untuk membangun pusat penelitian H. pylori yang menyediakan fasilitas untuk kultur, evaluasi resistensi antibiotik, dan memperoleh informasi genotipe yang dapat menjelaskan perbedaan dalam infeksi H. pylori di antara berbagai etnis di Indonesia The epidemiology of Helicobacter pylori (H. pylori) has been changing over the past decades. Indonesia was reported have a low prevalence of H. pylori infection compared to other countries in Asia. Some studies in Indonesia have evaluated that poor sanitation, age, religion, ethnicity are the risk factors for H. pylori infection. Compared to other diagnostic tests, the urine test will be reliable for the detection of H. pylori in Indonesia because it is non-invasive and low cost with high accuracy. Although we have already performed studies on the prevalence of H. pylori infection in several ethnics, we still have some questions that remain unclear regarding H. pylori infection in Indonesia. Therefore, we have a need to build a H. pylori center that provide facilities for culturing, evaluating antibiotic resistance, and obtaining the genotype information that may explain the differences in H. pylori infection among ethnic groups in Indonesia.

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Elfira, Vanda, Dwi Prasetyo, Dzulfikar DLH, and Kusnandi Rusmil. "Perbedaan Status Gizi dan Perawakan Pendek pada Anak Sakit Perut Berulang dengan Helicobacter Pylori Positif dan Negatif." Sari Pediatri 20, no. 5 (2019): 303. http://dx.doi.org/10.14238/sp20.5.2019.303-8.

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Latar belakang. Sakit perut berulang (SPB) merupakan keluhan yang paling sering pada anak. Infeksi Helicobacter pylori (H. pylori) saat ini merupakan salah satu penyebab organik terbanyak pada anak SPB. Infeksi H. pylori dapat menyebabkan malnutrisi dan perawakan pendek, tetapi hal ini masih kontroversial.Tujuan. Mengetahui perbedaan status gizi dan perawakan pendek antara anak SPB dengan infeksi H. pylori positif dan negatif.Metode Penelitian potong lintang analitik dilakukan pada anak SMP dan SMA di Bandung yang mengalami SPB. Infeksi H. pylori berdasarkan pemeriksaan serologis menggunakan kit BioM pylori. Analisis perbedaan status gizi dan perawakan pendek antara anak SPB dengan infeksi H. pylori positif dan negatif menggunakan uji chi square.Hasil. Terdapat 224 subjek mengalami SPB dari 1658 subjek yang disurvey. Sebanyak 99 subjek memenuhi kriteria inklusi. H. pylori positif pada 45 subjek. Uji beda memperlihatkan perbedaan proporsi pada status gizi kurang dan infeksi H. pylori positif, namun belum bermakna secara statistik. Pada uji beda perawakan pendek dengan infeksi H. pylori positif tidak didapatkan perbedaan bermakna.Kesimpulan. Tidak terdapat perbedaan bermakna status gizi dan perawakan pendek pada anak SPB dengan infeksi H. pylori positif dan infeksi H. pylori negatif.

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Huang, Wei, San Jiang, and Wanshou Jiang. "A Model-Driven Method for Pylon Reconstruction from Oblique UAV Images." Sensors 20, no. 3 (2020): 824. http://dx.doi.org/10.3390/s20030824.

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Pylons play an important role in the safe operation of power transmission grids. Directly reconstructing pylons from UAV images is still a great challenge due to problems of weak texture, hollow-carved structure, and self-occlusion. This paper presents an automatic model-driven method for pylon reconstruction from oblique UAV images. The pylons are reconstructed with the aid of the 3D parametric model library, which is represented by connected key points based on symmetry and coplanarity. First, an efficient pylon detection method is applied to detect the pylons in the proposed region, which are obtained by clustering the line segment intersection points. Second, the pylon model library is designed to assist in pylon reconstruction. In the predefined pylon model library, a pylon is divided into two parts: pylon body and pylon head. Before pylon reconstruction, the pylon type is identified by the inner distance shape context (IDSC) algorithm, which matches the shape contours of pylon extracted from UAV images and the projected pylon model. With the a priori shape and coplanar constraint, the line segments on pylon body are matched and the pylon body is modeled by fitting four principle legs and four side planes. Then a Markov Chain Monte Carlo (MCMC) sampler is used to estimate the parameters of the pylon head by computing the maximum probability between the projected model and the extracted line segments in images. Experimental results on several UAV image datasets show that the proposed method is a feasible way of automatically reconstructing the pylon.

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Hiengrach, Pratsanee, Wimonrat Panpetch, Ariya Chindamporn, and Asada Leelahavanichkul. "Helicobacter pylori, Protected from Antibiotics and Stresses Inside Candida albicans Vacuoles, Cause Gastritis in Mice." International Journal of Molecular Sciences 23, no. 15 (2022): 8568. http://dx.doi.org/10.3390/ijms23158568.

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Due to (i) the simultaneous presence of Helicobacter pylori (ulcer-induced bacteria) and Candida albicans in the stomach and (ii) the possibility of prokaryotic–eukaryotic endosymbiosis (intravacuolar H. pylori in the yeast cells) under stresses, we tested this symbiosis in vitro and in vivo. To that end, intravacuolar H. pylori were induced by the co-incubation of C. albicans with H. pylori under several stresses (acidic pH, non-H. pylori-enrichment media, and aerobic environments); the results were detectable by direct microscopy (wet mount) and real-time polymerase chain reaction (PCR). Indeed, intravacuolar H. pylori were predominant under all stresses, especially the lower pH level (pH 2–3). Interestingly, the H. pylori (an amoxicillin-sensitive strain) inside C. albicans were protected from the antibiotic (amoxicillin), while extracellular H. pylori were neutralizable, as indicated by the culture. In parallel, the oral administration of intravacuolar H. pylori in mice caused H. pylori colonization in the stomach resulting in gastritis, as indicated by gastric histopathology and tissue cytokines, similar to the administration of free H. pylori (extra-Candida bacteria). In conclusion, Candida protected H. pylori from stresses and antibiotics, and the intravacuolar H. pylori were able to be released from the yeast cells, causing gastric inflammation with neutrophil accumulations.

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Ahmed, Suha Hussein, and Saad Abdul Kareem Mohammed. "Helicobacter pylori infection in pregnant women and it’s correlation with the alterations of some trace elements levels in the serum at Maternity Teaching Hospital in Erbil City." Al Mustansiriyah Journal of Pharmaceutical Sciences 18, no. 2 (2018): 41–50. http://dx.doi.org/10.32947/ajps.v18i2.474.

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Helicobacter pylori (H. pylori) is a spiral-shaped pathogenic bacterium found on the human gastric mucosa, Warren and Marshall isolated H pylori for the first time in 1982. It is one of the most common worldwide human infections [1].H. pylori play a vital role in the development of chronic gastritis, gastric ulcer, duodenal ulcer, gastric adenocarcinoma, and gastric mucosa-associated lymphoid tissue lymphoma [2]. The current study included (120) pregnant women, (50) positive anti -H. Pylori Ig and (70) negative anti- H. Pylori Ig with pregnancy for first, second and third semester at mean age/ years 28.36 for the positive anti-H. Pylori and 26.17 for the negative anti-H. Pylori appeared that an alteration of zinc level in serum of positive anti- H. Pylori Ig groups was (48.904 ± 18.3486) (μg/dl) comparing with the negative groups (90.757 ± 9.2727) with the highly significant difference (P < 0.01). While serum copper levels of positive anti-H. Pylori Ig group was (μg/dl), (100.412 ± 23.8234), documented as normal highly significant (P < 0.01) compared to the negative anti- H. Pylori Ig group (114.971 ± 20.4995).In this study, the GIT disorder with anti-H. Pylori Ig positive groups were (32, 64%) and anti-H. Pylori Ig negative groups were (32, 45.7%), with significant difference (P < 0.05).

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Kadota, Tamami, Masakazu Hamada, Ryota Nomura, et al. "Distribution of Helicobacter pylori and Periodontopathic Bacterial Species in the Oral Cavity." Biomedicines 8, no. 6 (2020): 161. http://dx.doi.org/10.3390/biomedicines8060161.

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The oral cavity may serve as a reservoir of Helicobacter pylori. However, the factors required for H. pylori colonization are unknown. Here, we analyzed the relationship between the presence of H. pylori in the oral cavity and that of major periodontopathic bacterial species. Nested PCR was performed to detect H. pylori and these bacterial species in specimens of saliva, dental plaque, and dental pulp of 39 subjects. H. pylori was detected in seven dental plaque samples (17.9%), two saliva specimens (5.1%), and one dental pulp (2.6%) specimen. The periodontal pockets around the teeth, from which dental plaque specimens were collected, were significantly deeper in H. pylori-positive than H. pylori-negative subjects (p < 0.05). Furthermore, Porphyromonas gingivalis, a major periodontopathic pathogen, was detected at a significantly higher frequency in H. pylori-positive than in H. pylori-negative dental plaque specimens (p < 0.05). The distribution of genes encoding fimbriae (fimA), involved in the periodontal pathogenicity of P. gingivalis, differed between H. pylori-positive and H. pylori-negative subjects. We conclude that H. pylori can be present in the oral cavity along with specific periodontopathic bacterial species, although its interaction with these bacteria is not clear.

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U, Anagha, and Binitha A. "A Single Case Study of Treating H-Pyroli Infection with Coconut Oil." International Journal of Ayurvedic Medicine 11, no. 2 (2020): 322–25. http://dx.doi.org/10.47552/ijam.v11i2.1215.

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Helicobacter pylori (H-Pylori) are a Gram negative spiral organism in GI Tract. Final effect of H-pylori on the GI tract is variable and determined by microbial and host factors. The particular end result of H-Pylori varies from chronic gastritis to PUD, Gastric MALT Lymphoma. A case presented with chronic gastritis associated H-Pylori infection. Which showed resemblance with Clinical features of vatha –pitha Parinaamashoola (pain during digestion). It was intervened successfully with samanasnehapana (internal oleation therapy) of coconut oil. Coconut oil administered for 15 days. Helicobacter pylori immunoglobulin (IgG H-pylori) evaluated before and after the treatment. A positive H-pylori infection became negative after the treatment.

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LUPU, Vasile Valeriu, Gabriela PĂDURARU, Anca ADAM, Ana-Maria DĂBULEANU, Ancuţa IGNAT, and Marin BURLEA. "Helicobacter pylori colonization and oral health in children." Romanian Journal of Medical Practice 12, no. 2 (2017): 41–44. http://dx.doi.org/10.37897/rjmp.2017.1.8.

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Helicobacter pylori (H. pylori) is a microaerophilic gram-negative bacterium infecting approximately one half of the world’s population. The oral cavity and dental plaque may be a reservoir for H. pylori infection. Diagnosis of H. pylori infection in children differs from that of adults. Although H. pylori has long been known to be detected in the oral cavity, the significance of such findings are controversial. Oral H. pylori may play an important role in re-infection of the gastric mucosa. The gold standard for eradicating H. pylori infection is standard triple therapy. The studies have shown promising results in the management of both oral and gastric H. pylori.

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Luzza, Francesco, Antonio Contaldo, Maria Imeneo, et al. "Testing for Serum IgG Antibodies to Helicobacter pylori Cytotoxin‐Associated Protein Detects Children with Higher Grades of Gastric Inflammation." Journal of Pediatric Gastroenterology and Nutrition 29, no. 3 (1999): 302–7. http://dx.doi.org/10.1002/j.1536-4801.1999.tb02419.x.

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ABSTRACTBackground:Little information is available about the relationships between Helicobacter pylori cytotoxin‐associated protein (CagA) and clinicopathologic features in children. The purpose of this study was to test whether determining serum IgG antibodies to CagA is a useful tool for detecting more severe disease.Methods:One hundred twenty‐seven consecutive children (age, range, 0.75‐17.8 years; median, 9.4 years) referred for gastroscopy were included in the study. Antral and corpus biopsies were taken for gastric histology and H. pylori detection. Major symptoms and endoscopic findings were recorded. A serum sample was drawn from each child and assayed for IgG antibodies CagA by a commercial enzyme‐linked immunosorbent assay.Results:Sixty‐three (50%) children had no evidence of H. pylori infection, 28 (22%) were H. pylori positive/CagA positive, and 36 (28%) were H. pylori positive/CagA negative. There were no differences in clinical diagnosis and occurrence of any predominant symptom according to H. pylori and CagA status. Findings of antral nodularity were more frequent (p = 0.003) in H. pylori‐positive/CagA‐positive children than in H. pylori‐positive/CagA‐negative children. The gastritis score was significantly higher in H. pylori‐positive/CagA‐positive children than in H. pylori‐positive/CagA‐negative children (5.7 ± 1.9 vs. 3.8 ± 1.6, respectively; p = 0.0003), either in the antral (p = 0.0002) or in the corpus (p = 0.001) mucosa. Inflammation (p = 0.0001) and activity (p = 0.0001) scores were both higher in H. pylori‐positive/CagA‐positive children than in H. pylori‐positive/CagA‐negative children, but the H. pylori density score was not significantly different (p = NS). In no case was normal gastric mucosa found in H. pylori‐positive/CagA‐positive children. Lymphocytic gastritis (p = 0.0008) and lymphoid follicles (p = 0.000003) were a more frequent finding in H. pylori‐positive children than in H. pylori negative children, irrespective of CagA status.Conclusion:Testing for serum IgG to CagA detects higher grades of gastric inflammation among children with H. pylori infection. It may be useful in targeting H. pylori‐positive/CagA‐positive children for antimicrobial therapy while reducing the need for endoscopy and gastric biopsy.

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Della Bella, Chiara, Sofia D’Elios, Sara Coletta, et al. "Increased IL-17A Serum Levels and Gastric Th17 Cells in Helicobacter pylori-Infected Patients with Gastric Premalignant Lesions." Cancers 15, no. 6 (2023): 1662. http://dx.doi.org/10.3390/cancers15061662.

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Background: Helicobacter pylori infection is characterized by an inflammatory infiltrate that might be an important antecedent of gastric cancer. The purpose of this study was to evaluate whether interleukin (IL)-17 inflammation is elicited by gastric T cells in Helicobacter pylori patients with gastric intestinal metaplasia and dysplasia (IM/DYS). We also investigated the serum IL-17A levels in Helicobacter pylori patients with gastric intestinal metaplasia and dysplasia, and patients with Helicobacter pylori non-atrophic gastritis (NAG). Methods: the IL-17 cytokine profile of gastric T cells was investigated in six patients with IM/DYS and Helicobacter pylori infection. Serum IL-17A levels were measured in 45 Helicobacter pylori-infected IM/DYS patients, 45 Helicobacter pylori-infected patients without IM/DYS and in 45 healthy controls (HC). Results: gastric T cells from all IM/DYS patients with Helicobacter pylori were able to proliferate in response to Helicobacter pylori and to produce IL-17A. The Luminex analysis revealed that IL-17A levels were significantly increased in Helicobacter pylori IM/DYS patients compared to healthy controls and to Helicobacter pylori gastritis patients without IM/DYS (452.34 ± 369.13 pg/mL, 246.82 ± 156.06 pg/mL, 169.26 ± 73.82 pg/mL, respectively; p < 0.01, p < 0.05). Conclusions: the results obtained indicate that Helicobacter pylori is able to drive gastric IL-17 inflammation in IM/DYS Helicobacter pylori-infected patients, and that IL-17A serum levels are significantly increased in Helicobacter pylori-infected patients with IM/DYS.

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Zeng, Judeng, Chuan Xie, Lin Zhang, et al. "Host Cell Antimicrobial Responses against Helicobacter pylori Infection: From Biological Aspects to Therapeutic Strategies." International Journal of Molecular Sciences 23, no. 18 (2022): 10941. http://dx.doi.org/10.3390/ijms231810941.

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The colonization of Helicobacter pylori (H. pylori) in human gastric mucosa is highly associated with the occurrence of gastritis, peptic ulcer, and gastric cancer. Antibiotics, including amoxicillin, clarithromycin, furazolidone, levofloxacin, metronidazole, and tetracycline, are commonly used and considered the major treatment regimens for H. pylori eradication, which is, however, becoming less effective by the increasing prevalence of H pylori resistance. Thus, it is urgent to understand the molecular mechanisms of H. pylori pathogenesis and develop alternative therapeutic strategies. In this review, we focus on the virulence factors for H. pylori colonization and survival within host gastric mucosa and the host antimicrobial responses against H. pylori infection. Moreover, we describe the current treatments for H. pylori eradication and provide some insights into new therapeutic strategies for H. pylori infection.

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Simán, J. Henrik, Lars Engstrand, Göran Berglund, Claes-Henrik Florén, and Arne Forsgren. "Evaluation of Western Blot CagA Seropositivity in Helicobacter pylori-Seropositive and -Seronegative Subjects." Clinical Diagnostic Laboratory Immunology 12, no. 2 (2005): 304–9. http://dx.doi.org/10.1128/cdli.12.2.304-309.2005.

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ABSTRACT CagA seropositivity is an important risk factor for gastric adenocarcinoma and duodenal ulcer. However, CagA seropositivity is also found in Helicobacter pylori-seronegative subjects. Is CagA seropositivity in these subjects a sign of a past H. pylori infection, or does it represent a false-positive reaction? This study investigates the intensity of the CagA immune reaction and the variation in CagA seroprevalence with year of birth for 650 subjects belonging to the Malmö Preventive Medicine cohort. CagA and H. pylori seroprevalences were determined by Western blot analysis (Helicoblot 2.1) and enzyme-linked immunosorbent assay. The peak intensity of the CagA band was significantly lower in H. pylori-seronegative subjects than in those with concomitant H. pylori seropositivity. In H. pylori-seropositive subjects, peak CagA intensity had a bimodal distribution. The prevalence of CagA-seropositive but H. pylori-seronegative subjects increased successively and significantly with year of birth, in contrast to the prevalence of CagA-seropositive and H. pylori-seropositive subjects, which decreased significantly. However, within H. pylori-seropositive and -seronegative subgroups, CagA seroprevalences were constant for different birth cohorts. If CagA seropositivity in H. pylori-seronegative subjects represents a past H. pylori infection, there must have been some mechanisms of eradication that were more common in younger subjects and that were of more importance than the presence of gastric atrophy and the longer duration and higher prevalence of H. pylori infection found in older subjects. Antibiotic treatment of H. pylori was not common practice at the time of enrollment. On the other hand, a false-positive reaction would be constant and independent of birth cohorts, as with the H. pylori-seronegative subgroup of our study. Peak CagA intensity in H. pylori-seronegative subjects corresponded to the lower part of the bimodal distribution of peak CagA intensity in H. pylori-seropositive subjects. We conclude that a major proportion of CagA seropositivity in H. pylori-seronegative subjects represents a false-positive reaction. Peak CagA intensity has a bimodal distribution in H. pylori-seropositive subjects. Low-intensity CagA seropositivity in H. pylori-seropositive subjects is indeterminate, representing both false-positive and true-positive reactions.

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Eissa, Adil, and Saad Mirza. "The Relevance of Helicobacter pylori Infection to Iron Deficiency Anemia in Duhok City." Iraqi Journal of Medical Sciences 19, no. 1 (2021): 33–38. http://dx.doi.org/10.22578/ijms.19.1.5.

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Background:Helicobacter pylori (H. pylori) infection had been criticized for many deleterious effects and had been amalgamated to iron deficiency by many authors, frequently based on correlative rather than direct relationship studies and often opposed by others. Objective: To evaluate the role of H. pylori infection in the etiology of iron deficiency anemia and to study the impact of the bacterial eradication on the response to iron therapy. Methods:The current study represents an interventional prospective study and involved 52 non-pregnant females with iron deficiency anemia. All patients were tested for the presence of active H. pylori infection by stool antigen test and they followed after one month of iron therapy. Patients with positive H. pylori infection followed for another month after eradication of H. pylori and iron therapy. Results: Fifteen patients (28.85%) were positive for H. pylori. Hematological and biochemical data were not different among both groups (H. pylori positive and negative) at presentation despite significant better response among H. pylori negative individuals. Continuation of iron therapy after eradication of H. pylori infection improve the response to therapy significantly. Conclusion: Eradication of H. pylori enhances the response to iron therapy significantly. Keywords: Iron deficiency anemia, H. pylori, iron therapy, eradication Citation: Eissa AA, Mirza SS. The relevance of Helicobacter pylori Infection to iron deficiency anemia in Duhok City. Iraqi JMS. 2021; 19(1): 33-38. doi: 10.22578/IJMS.19.1.5

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