Relevant bibliographies by topics / Pyuridae

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Pyuridae'

Author: Grafiati
Published: 2 June 2025
Last updated: 19 July 2025

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Journal articles on the topic "Pyuridae"

1

Monniot, Françoise. "Microcosmus anchylodeirus (Ascidiacea, Pyuridae) introduced in the Mediterranean Sea." Zootaxa 4175, no. 3 (2016): 222–30. https://doi.org/10.11646/zootaxa.4175.3.2.

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Rocha, Rosana M., and Bailey Keegan Counts. "Pyura (Tunicata: Ascidiacea: Pyuridae) on the coasts of Panama." Zootaxa 4564, no. 2 (2019): 491–513. https://doi.org/10.11646/zootaxa.4564.2.9.

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Monniot, Françoise. "Ascidians collected during the Madibenthos expedition in Martinique 3. Stolidobranchia, Pyuridae and Molgulidae." Zootaxa 4459, no. 3 (2018): 401–30. https://doi.org/10.11646/zootaxa.4459.3.1.

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Skinner, Luís Felipe, Rosana M. Rocha, and Bailey K. Counts. "Pyura gangelion and Pyura beta sp. nov. (Ascidiacea: Pyuridae): an exotic and a new tunicate from the West Atlantic." Zootaxa 4545, no. 2 (2019): 264–76. https://doi.org/10.11646/zootaxa.4545.2.6.

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Skinner, Luís Felipe, Rocha, Rosana M., Counts, Bailey K. (2019): Pyura gangelion and Pyura beta sp. nov. (Ascidiacea: Pyuridae): an exotic and a new tunicate from the West Atlantic. Zootaxa 4545 (2): 264-276, DOI: 10.11646/zootaxa.4545.2.6
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SEPÚLVEDA, R., J. M. CANCINO, and M. THIEL. "The peracarid epifauna associated with the ascidian Pyura chilensis (Molina, 1782) (Ascidiacea: Pyuridae)." Journal of Natural History 37, no. 13 (2010): 1555–69. https://doi.org/10.1080/00222930110099615.

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SEPÚLVEDA, R., CANCINO, J. M., THIEL, M. (2003): The peracarid epifauna associated with the ascidian Pyura chilensis (Molina, 1782) (Ascidiacea: Pyuridae). Journal of Natural History 37 (13): 1555-1569, DOI: 10.1080/00222930110099615, URL: https://www.tandfonline.com/doi/full/10.1080/00222930110099615
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Monniot, Claude. "Ascidies de Nouvelle-Calédonie VI. Pyuridae et Molgulidae." Bulletin du Muséum national d'histoire naturelle 11, no. 3 (1989): 475–507. http://dx.doi.org/10.5962/p.288279.

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Sanamyan, K. E., N. P. Sanamyan, and T. B. Morozov. "A new species of Microcosmus (Tunicata: Ascidiacea: Bolteniidae) from the Sea of Okhotsk." Zoosystematica Rossica 30, no. 2 (2021): 271–78. http://dx.doi.org/10.31610/zsr/2021.30.2.271.

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A large solitary ascidian Microcosmus armatus sp. nov., described from the Sea of Okhotsk, represents the first record of the genus Microcosmus Heller, 1877 in the cold waters of the Northwestern Pacific. The genus belongs to the family Bolteniidae formerly known as Pyuridae.
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Sanamyan, Karen E., Nadya P. Sanamyan, and Taras B. Morozov. "A new species of Microcosmus (Tunicata: Ascidiacea: Bolteniidae) from the Sea of Okhotsk." Zoosystematica Rossica 30, no. 2 (2021): 271–78. https://doi.org/10.31610/zsr/2021.30.2.271.

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A large solitary ascidian&nbsp;<em>Microcosmus armatus</em>&nbsp;<strong>sp. nov.</strong>, described from the Sea of Okhotsk, represents the first record of the genus&nbsp;<em>Microcosmus&nbsp;</em>Heller, 1877 in the cold waters of the Northwestern Pacific. The genus belongs to the family Bolteniidae formerly known as Pyuridae.
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ROCHA, ROSANA M., and BAILEY KEEGAN COUNTS. "Pyura (Tunicata: Ascidiacea: Pyuridae) on the coasts of Panama." Zootaxa 4564, no. 2 (2019): 491. http://dx.doi.org/10.11646/zootaxa.4564.2.9.

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Recent efforts have been taken to survey and describe the ascidian fauna of the Pacific and Atlantic coasts of Panama; however, the genus Pyura still remains poorly known. Sampling events have been occurring since 2003. In this research, we describe two new species from the Atlantic coast of Panama: P. longispina sp. nov., P. lopezlegentilae sp. nov., and two new species from the Pacific Coast of Panama: P. carmanae sp. nov. and P. imesa sp. nov. We also supply a tabular key for all the Pyura species found in Panama.
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MONNIOT, FRANÇOISE. "Microcosmus anchylodeirus (Ascidiacea, Pyuridae) introduced in the Mediterranean Sea." Zootaxa 4175, no. 3 (2016): 222. http://dx.doi.org/10.11646/zootaxa.4175.3.2.

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Dissertations / Theses on the topic "Pyuridae"

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Lunawat, R. "An experimental study to find out the significance of sterile pyuria and evidence of inflammation in Overactive Bladder." Thesis, University College London (University of London), 2014. http://discovery.ucl.ac.uk/1418476/.

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Purpose: It has been reported that over 33 % of patients with OAB present with pyuria (≥10 wbc μl-1) on urine microscopy, but under a third of these have bacteriuria. To clarify this situation, an accomplished comparative scrutiny of the inflammatory state of the urothelium in OAB, was carried out. Materials and Methods: This was a prospective, blinded, observational study of idiopathic OAB patients compared with controls. CSU samples were obtained and submitted to fresh urine microscopy, routine culture and microscopic examination of spun sediment. Another sample of OAB patients and asymptomatic controls provided cystoscopic bladder biopsies for histopathology and electron microscopy. Results: 178 OAB patients and 21 controls provided spun sediment samples. 75 (42 %) of these OAB patients had microscopic pyuria (.1 wbc ƒÊl-1) with 25 of which (33 %) were culture-positive. None of the controls had pyuria, nor bacteriuria. In a 20 mm2 spun deposit there was an average of 48 wbc (95 % CI 26 to 589) in OAB with pyuria; 12 wbc (95 % CI 10 to 15) in OAB without pyuria; and 4 wbc (95 % CI 3 to 7) in controls. Biopsies from 79 OAB patients, showed chronic inflammation and hyperplasia in 69 (87 %; 95 % CI=78 to 92: only 20 % had pyuria) and none in 5 controls. EM of 22 OAB patients showed increased basement membrane thickness compared to 2 controls (H=48, df=2, p<0.001). Conclusions: The study shows evidence of chronic cystitis and urothelial hyperplasia associated with OAB irrespective of pyuria or bacteriuria. The phenomenon has been confirmed by 3 different methods.
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Müller, Sina [Verfasser]. "Auswirkung von Pyurie und Hämaturie auf den Protein-Kreatinin Quotienten im Urin bei Katzen in Abhängigkeit der Urinentnahmeart / Sina Müller." Gießen : Universitätsbibliothek, 2019. http://d-nb.info/1174938870/34.

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Yang, Ming-Hsin, and 楊旻鑫. "Pyuria and Post-operation Urinary Tract Infection following Diode Laser Vaporesection of the Prostate for Benign Prostate Obstruction." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/bbbck5.

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碩士<br>中山醫學大學<br>醫學研究所<br>106<br>Objective：Monopolar transurethral resection of prostate (m-TURP) remains the golden standard for benign prostate obstruction (BPO). Recently evolved laser surgical technique provides less perioperative complications with equivalent outcomes. New developed diode laser vaporesection (DiLRP) offers better hemostasis, shortens catheterization duration, and hospital stay. However, deep thermal penetration might cause prolong prostatic urethra inflammation and subsequent complication. We conduct a retrospective study to compare the pyuria duration and post-operative urinary tract infection sequelae (POUTIs) between DiLRP and m-TURP. Methods and Materials：From July 2011 to September 2015, we retrieved medical records on patients with lower urinary tract symptoms due to prostate obstruction underwent m-TURP and DiLRP. Demographic characteristics were recorded from a single center computerized database, including history of the underlying disease, use of anticoagulant or antibiotic before operation, the duration of indwelling catheter, PSA level, history of transrectal ultrasound biopsy, perioperative and postoperative details, hospitalization period. The duration of pyuira after operation was compared by kapalan-meire analysis; and risk factors were evaluated by Cox regression analysis. The POUTIs which include urinary tract infection, epididymitis, prostatitis, and infection related hospitalization were compared as well. Results：One hundred and twelve patients underwent DiLRP and 81 received m-TURP with the same surgeon at the same period. The mean age of the patients was 72±7.3 years in DiLRP group and 70±7.6 years in m-TURP group (p=0.069). There was more percentage of anticoagulant used in DiLEP group than in m-TURP group (18.5% vs 7.4 %, p=0.028). Operation time was longer, but post-operation normal saline irrigation interval was shorter in DiLRP in comparison with m-TURP respectively (62.8±20.6 vs 47.4±22.1 mins, p<0.001; 2.1±0.3 vs 2.5±0.9 days, p<0.001). The difference of post operation infection between groups is statistically significant in epididymitis (10.2% vs 1.2%, p=0.013) and POUTIs related hospitalization (8.3% vs 1.2 %, p=0.031). The DiLRP resulted in longer pyuria period (16 vs 12 weeks, p=0.0014), with factors including operative method by DiLRP (HR: 1.828, p=0.003) and age (HR: 0.665, p=0.040). Conclusion and Suggestion：According to our study, DiLRP associated with more POUTIs possibly caused by longer pyuria period. Further larger prospective study is necessary for the evaluation the association between post-operative pyuria and POUTIs.
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Books on the topic "Pyuridae"

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Oki, Hideo. Pyuritan: Kindaika no seishin kozo (Chuko shinsho). Chuo Koronsha, 1992.

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Heyns, Chris. Tuberculosis and parasitic infestations involving the urogenital system. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0006.

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Urogenital tuberculosis is caused by Mycobacterium tuberculosis, which evokes a granulomatous tissue reaction leading to caseous necrosis, fibrosis, and eventual calcification. It most commonly presents as cystitis with sterile pyuria but can show many other symptoms and signs requiring a high index of suspicion to make the diagnosis. Schistosomiasis (Bilharzia) affecting the urinary tract is caused by the flatworm Schistosoma haematobium. Humans are infested by contact with fresh water harbouring the intermediate snail host. Echinococcosis (hydatid disease), is caused by the tapeworm Echinococcus granulosis or multilocularis. Human infection results from close contact with the parasite host (usually dogs and sheep). Filariasis, caused by the roundworm Wuchereria bancrofti, is transmitted by mosquito bite
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Izzedine, Hassan, and Victor Gueutin. Drug-induced chronic tubulointerstitial nephritis. Edited by Adrian Covic. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0087.

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The chronic form of drug-induced tubulointerstitial nephritis (CTIN) is an insidious disease and most probably represents the common final response pattern of the kidney to a variety of agents (including analgesics, lithium, antineoplastic chemotherapeutic agents, like cisplatin and nitrosoureas, and immunosuppressive drugs, such as ciclosporin and tacrolimus). Drug-induced CTIN is usually asymptomatic, presenting with slowly progressive renal impairment. Because of its insidious nature, CTIN is often diagnosed incidentally on routine laboratory screening or evaluation of CKD. The diagnosis of drug-induced CTIN largely depends on the history of exposure to a nephrotoxic drug. Clinical investigations may show modest elevation in serum creatinine, evidence of tubular dysfunction (e.g. renal tubular acidosis), or Fanconi syndrome (i.e. aminoaciduria, glycosuria, hypophosphataemia, and hypouricaemia). Urinalysis may be normal or show low-grade proteinuria (&lt; 1.5 g/day) and/or pyuria. Diagnosis depends on renal biopsy, which reveals variable cellular infiltration of the interstitium, tubular atrophy, and fibrosis. Analgesic nephropathy is possibly still the most common category of CTIN worldwide. The amount of phenacetin-acetaminophen combination required to cause CTIN has been estimated to be at least 2–3 kg over many years. Lithium-induced CTIN occurs in a small subset of patients receiving long-term lithium therapy, who have had repeated episodes of lithium toxicity, with high serum drug levels. CTIN induced by ciclosporin or tacrolimus is common among patients receiving kidney, heart, liver, and pancreas transplants. The mechanism appears to be dependent largely on the potent vasoconstrictive effects of these drugs. The recognition of a potential association between a patient’s renal disease and exposure to a drug is crucial, because, unlike many other forms of renal disease, drug-induced CTIN can be prevented and even reversed, by avoiding additional drug exposure.
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Book chapters on the topic "Pyuridae"

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Mookerjee, Basab K., and Saleem Khan. "Bacteriuria and Pyuria." In Assessment of Urinary Sediment by Electron Microscopy. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1851-4_5.

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Kunin, Calvin M. "Recurrent Fever, Chills, Pyuria, and Bacteriuria in an Adult Male Following Urinary Catheterization." In Contemporary Internal Medicine. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4615-6713-4_12.

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Cheng, Chi-Hui. "Case 33. An 8-Month-Old Female Infant with Fever and Pyuria: Acute Lobar Nephronia." In Paediatric Infectious Diseases. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-7276-8_33.

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Lowenstam, Heinz A., and Stephen Weiner. "Chordata." In On Biomineralization. Oxford University Press, 1989. http://dx.doi.org/10.1093/oso/9780195049770.003.0011.

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In the field of biomineralization the phylum Chordata is the most intensively studied, as one of its subphyla, the Craniata, includes our own species. The Craniata are often referred to as the vertebrates, a term that alludes to the importance of the endoskeleton in denning the essential character of these animals. The phylum Chordata also contains three other subphyla, only one of which has members that form mineralized hard parts. They belong to the Urochordata or tunicates. In fact, mineralization is confined to several families of a single class of urochordates, the Ascidiacea. Table 9.1 is a compilation of the known biogenic minerals formed by members of the Chordata, together with the sites at which they form and their presumed functions. The table includes no less than 17 different minerals, which should dispel any notion that mineralization in the chordates is synonymous with "calcium phosphate" deposition. It is, of course, true that the mineralized skeletal hard parts of most of the Craniata or vertebrates contain a calcium phosphate mineral, usually in the carbonated form called dahllite. However, the vertebrates also form four different carbonate minerals that are most commonly found in the vestibulary apparatus (see Chapter 10). They form three different iron minerals, which includes magnetite found in the navigation system of various vertebrate genera. The Ascidiacea also form a diverse array of minerals. Interestingly, however, their diversity is essentially confined to one class, the Pyuridae, which form no less than six different minerals, including two phosphate minerals. In this chapter we first describe biomineralization processes in the Ascidiacea followed by detailed discussions of mineralization processes in the Chordata or vertebrates. For convenience, the section on vertebrate mineralization is divided according to the major mineralized tissues: bone (dentin), cartilage, and tooth enamel. Mineralization in the vestibulary apparatus is discussed in Chapter 10.
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"Pyuria." In Clinical Veterinary Advisor. Elsevier, 2012. http://dx.doi.org/10.1016/b978-1-4160-9979-6.00434-7.

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"pyuria, n." In Oxford English Dictionary, 3rd ed. Oxford University Press, 2023. http://dx.doi.org/10.1093/oed/1076578343.

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"4.2 Leukozyturie/Pyurie." In Urologische Differenzialdiagnose, edited by Joachim W. Thüroff. Georg Thieme Verlag, 2007. http://dx.doi.org/10.1055/b-0034-41114.

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De Broe, Marc E., Patrick C. D’Haese, and Monique M. Elseviers. "Chronic tubulointerstitial nephritis." In Oxford Textbook of Medicine. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.210902_update_001.

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Chronic tubulointerstitial nephritis is usually asymptomatic, presenting with slowly progressive renal impairment. Urinalysis may be normal or show low-grade proteinuria (&lt;1.5 g/day) and/or pyuria. Diagnosis depends on renal biopsy, which reveals variable cellular infiltration of the interstitium, tubular atrophy, and fibrosis. There are many causes including sarcoidosis, drugs (prescribed and nonprescribed), irradiation, toxins, and metabolic disorders....
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Alexander, Mariam P., and Ajay K. Singh. "Parenchymal Renal Disease." In The Brigham Intensive Review of Internal Medicine. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199358274.003.0062.

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Parenchymal renal disease can be considered anatomically under the headings of glomerular, tubular, tubulointerstitial, and vascular disease. Most patients present with a clinical syndrome of nephron injury. The clinical features of parenchymal renal disease frequently depend on the component of the kidney that is affected—for example, glomerulonephritis presents with worsened kidney function, hypertension, hematuria, proteinuria, and red cell casts. Tubulointerstitial nephritis presents with azotemia, pyuria and/or white cell casts.
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Ikeda, Ichiro, Makoto Oda, Muneki Sakakura, and Kojiro Yasunaga. "Presence of Platelet-Activating Factor in Pyuria in Humans." In Platelet-Activating Factor and Structurally Related Alkyl Ehter Lipids. AOCS Publishing, 1992. http://dx.doi.org/10.1201/9781439832042.ch76.

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Conference papers on the topic "Pyuridae"

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Cox, Jennifer, M. Kenosi, Laura Whitla, and Montaseur Nadeem. "GP290 Can we rely on pyuria to diagnose urinary tract infection in children?" In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.349.

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