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Relevant bibliographies by topics / Qi wen yi xiang

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Journal articles

Academic literature on the topic 'Qi wen yi xiang'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 January 2023

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Journal articles on the topic "Qi wen yi xiang"

1

Wu, Fatima, Zheng Shu Shen, Huang Jichi, and Lu Weilian. "Zao qi Xiang-Gang xin wen xue zuo pin xuan, 1927-1941." World Literature Today 73, no. 4 (1999): 818. http://dx.doi.org/10.2307/40155275.

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2

DENG, YUNFEI. "Aralia wangshanensis (Araliaceae)—the legitimate name for Aralia franchetii." Phytotaxa 402, no. 1 (2019): 73. http://dx.doi.org/10.11646/phytotaxa.402.1.11.

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Aralia Linnaeus (1753: 273) consists of approximately 65 species distributed in eastern to southeastern Asia and the Americas (Wen, 1993). In the past there has been much disagreement over whether Aralia should be recognized in a broad or narrow sense (Li 1942, Hoo & Tseng 1965, 1978, Shang 1985a, 1985b, Shang & Li 1990, Wen 1993, 2002, 2011, Wen et al. 2002, Xiang & Lowry 2007). The broad sense of Aralia is supported by recent studies on the basis of morphological characters and molecular evidence (Wen 1993, Wen et al. 2001, 2002), and therefore some segregated genera, Coudenbergi

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3

Kim, Yon-Jae. "Yi-wei’s Taiyi-Yuanqi View and Its Pre-Experiential World-View of Qi-Xiang and Yun-Shu." Journal of the New Korean Philosophical Association 109 (July 31, 2022): 157–79. http://dx.doi.org/10.20433/jnkpa.2022.07.157.

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4

Houxuan, Hu. "16. An Interpretation of the Oracle-Bone Inscription Phrase: “The Sun and Moon Eclipsed”." Early China 9, S1 (1986): 35–38. http://dx.doi.org/10.1017/s0362502800003035.

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ABSTRACTThis paper assembles three oracle-bone inscriptions divined at the same time from the Wu Yi-Wen Ding period. Two are identical, being a paired divination inquiring into whether the event ri yue you shi was or was not auspicious. The third asks whether, in light of this ri yue you shi, it would be auspicious to sacrifice to Shang Jia.Since 1925, when Wang Xiang , first proposed th at the character yue should be read as xi, his position has been accepted by Liu Chaoyang De Xiaoqian , Chen Mengijia , Zhang Peiyü , Xu Zhentao , the Zhongguo Tianwenxue jianshi bianxiezu , and the Zhongguo T

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5

Wang, Bu Hai, Cai Yue Chen, Xian Zhang та ін. "Abstract 5091: The early change of serum interleukin 14α levels predicts the response to anti-PD-1 therapy in cancer". Cancer Research 82, № 12_Supplement (2022): 5091. http://dx.doi.org/10.1158/1538-7445.am2022-5091.

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Abstract Background: Targeting programmed cell death-1 (PD-1) or programmed cell death ligand-1 (PD-L1) has been shown improved clinical efficacy in a wide range of tumor types. We evaluated serum interleukin 14α (IL14α) as a biomarker to predict the response of anti- PD-1 therapy. Patients and methods: Thirty advanced cancer patients treated with PD-1 inhibitor were enrolled in this study. Serum levels of IL-14α were tested at baseline and after 2 cycles of treatment. Result: Among these 30 patients, the mean expression level of IL14α before treatment was 2.1±1.21, whereas the mean level of I

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6

Weng, Yu-Cheng, Yu-Lin Li, Xiang-Ling Hou, et al. "Abstract 1626: The orthotopic mouse study of cetuximab and dasatinib treatment on BRAF mutated colon cancer cells." Cancer Research 82, no. 12_Supplement (2022): 1626. http://dx.doi.org/10.1158/1538-7445.am2022-1626.

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Abstract Colorectal cancer is one of the most common cancer in the world. The mortality rate is the third among the top ten cancers. Among colorectal cancer patients, about 65-75% of patients have overexpression of epithelial growth factor receptors (EGFR). The antibody drug cetuximab that inhibits EGFR has also been developed. Moreover, overexpression of src protein is found in about 80% of patients with colorectal cancer. Higher levels of Src expression is related to the metastasis of colon cancer. Dasatinib is an oral taken tyrosine kinase inhibitor to inhibit the src phosphorylation. There

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7

Koo, Hui Min, Xiang-Ling Hou, Yu-Lin Li, et al. "Abstract 1001: Investigates the role of vesicle trafficking in E-cadherin dynamics in collective migrated colon cancer cells." Cancer Research 82, no. 12_Supplement (2022): 1001. http://dx.doi.org/10.1158/1538-7445.am2022-1001.

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Abstract Cell adherens junction plays an important role in cell communication and regulation. E-cadherin is an important protein as adherens junctions. Loss of E-cadherin leads to epithelial cells transit to mesenchymal cells. Studies has demonstrated that E-cadherin dominates cell contact inhibition, which plays an important role in cell growth, however, recent studies demonstrated collective cell migration required E-cadherin expression escaping cell growth inhibition is important in metastasis. E-cadherin dynamics has been shown to be regulated by vesicle trafficking, however, the detail me

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8

Winarso, Viriya Adhiguna, Yi-Wen Lu, Yu-Cheng Weng, et al. "Abstract 1578: Interaction of E-cadherin and EGFR in the collective cell migration of colon cancer cells." Cancer Research 82, no. 12_Supplement (2022): 1578. http://dx.doi.org/10.1158/1538-7445.am2022-1578.

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Abstract Collective cell migration is one of the most important cellular events where a cluster of cells moves together in a coordinated way. Many studies have found this phenomenon as the hallmark of cancer progression and metastasis. In epithelial single cell migration, downregulation of adherens junction (AJ) molecule E-cadherin occurs at the initial step known as the Epithelial to Mesenchymal Transition (EMT). However, in collective cell migration, E-cadherin junction is maintained to provide adhesion between cells. Moreover, it also mediates the cell-cell intercommunication that is critic

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9

Li, Yu-Lin, Yu-Cheng Weng, Viriya Adhiguna Winarso, et al. "Abstract 2263: The role of E-Cadherin mediated cell division in promoting collective colon cancer cell migration." Cancer Research 82, no. 12_Supplement (2022): 2263. http://dx.doi.org/10.1158/1538-7445.am2022-2263.

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Abstract Collective cell migration is the cell cohort migrates toward the direction. Other than single cell migration, collective cell migration requires cell-cell connection with E-cadherin, however, E-cadherin is the epithelial marker in epithelial mesenchymal transition and acts as tumor suppressor function. Therefore, the role of E-cadherin in collective cell migration is still under investigated. In the collected cells, cell migration and cell division are coordinated for the cohort motility, the mechanism is still not clear. This study is to investigate the role of E-cadherin orientation

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10

Hou, Xiang-Ling, Hui Min Koo, Yu-Cheng Weng, et al. "Abstract 1582: Chloroquine inhibites Rab11 mediated E-cadherin transport in collective colon cancer cell migration." Cancer Research 82, no. 12_Supplement (2022): 1582. http://dx.doi.org/10.1158/1538-7445.am2022-1582.

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Abstract Cell migration is regulated by multiple molecular pathways to change the morphology from epithelial to mesenchymal type and is important in cancer metastasis. In has been demonstrated epithelial mesenchymal transition (EMT) is related to E-cadherin expression level. The loss of E-cadherin expression results in tumor proliferation and metastasis. Recently, studies have been demonstrated the collective cancer cell migration which expresses high E-cadherin is a powerful mechanism of metastasis. Therefore, the role of E-cadherin in cancer metastasis is still not clear. Clathrin-mediated e

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