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Journal articles on the topic 'Quinpirole'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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1

de Haas, Ria, Annelies Nijdam, Tjalke A. Westra, Martien JH Kas, and Herman GM Westenberg. "Behavioral pattern analysis and dopamine release in quinpirole-induced repetitive behavior in rats." Journal of Psychopharmacology 25, no. 12 (2010): 1712–19. http://dx.doi.org/10.1177/0269881110389093.

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Obsessive–compulsive disorder (OCD) is a chronic and disabling psychiatric disease with a lifetime prevalence of 2–3%. People with OCD suffer from intrusive, unwanted and recurrent thoughts (obsessions) and/or repetitive ritualistic behaviors (compulsions). The aim of this study is to quantify the dimensions of ritualistic ‘compulsive-like’ behavior in quinpirole-induced behavior in rats by using T-pattern behavioral analysis. In addition, we investigated whether the behavioral effects elicited by quinpirole sensitization remained after 2 weeks of cessation of treatment. Finally, to study the

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Mannhold, R. "Quinpirole hydrochloride." Drugs of the Future 12, no. 6 (1987): 558. http://dx.doi.org/10.1358/dof.1987.012.06.51471.

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THORESON, WALLACE B., SALVATORE L. STELLA, ERIC J. BRYSON, JOHN CLEMENTS, and PAUL WITKOVSKY. "D2-like dopamine receptors promote interactions between calcium and chloride channels that diminish rod synaptic transfer in the salamander retina." Visual Neuroscience 19, no. 3 (2002): 235–47. http://dx.doi.org/10.1017/s0952523802192017.

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Activation of D2-like dopamine receptors in rods with quinpirole stimulates L-type calcium currents (ICa). This result appears inconsistent with studies showing that D2-like dopamine receptor activation diminishes rod signals in second-order retinal neurons. Since small reductions in [Cl−]i can inhibit photoreceptor ICa, we tested the hypothesis that enhancement of ICa with the D2/D4 receptor agonist, quinpirole, increases calcium-activated chloride currents (ICl(Ca)) causing an efflux of Cl− from rods that would provide a negative feedback inhibition of ICa. In agreement with studies from Xen

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Alam, Sayed Ibrar, Min Gi Jo, Tae Ju Park, et al. "Quinpirole-Mediated Regulation of Dopamine D2 Receptors Inhibits Glial Cell-Induced Neuroinflammation in Cortex and Striatum after Brain Injury." Biomedicines 9, no. 1 (2021): 47. http://dx.doi.org/10.3390/biomedicines9010047.

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Brain injury is a significant risk factor for chronic gliosis and neurodegenerative diseases. Currently, no treatment is available for neuroinflammation caused by the action of glial cells following brain injury. In this study, we investigated the quinpirole-mediated activation of dopamine D2 receptors (D2R) in a mouse model of traumatic brain injury (TBI). We also investigated the neuroprotective effects of quinpirole (a D2R agonist) against glial cell-induced neuroinflammation secondary to TBI in adult mice. After the brain injury, we injected quinpirole into the TBI mice at a dose of 1 mg/k

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FAN, SHIH-FANG, and STEPHEN YAZULLA. "Inhibitory interaction of cannabinoid CB1 receptor and dopamine D2 receptor agonists on voltage-gated currents of goldfish cones." Visual Neuroscience 21, no. 1 (2004): 69–77. http://dx.doi.org/10.1017/s0952523804041070.

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Dopamine is a light-adaptive signal that desensitizes the retina, while cannabinoids reportedly increase photosensitivity. The presynaptic membrane of goldfish retinal cones has dopamine D2 receptors and cannabinoid CB1 receptors. This work focused on whether dopamine D2 receptor agonist quinpirole and cannabinoid CB1 receptor agonist WIN 55212-2 (WIN) interacted to modulate voltage-dependent membrane currents of cones. A conventional patch-clamp method was used to record depolarization evoked whole-cell outward currents (Iout) and an inward calcium current (ICa) from the inner segment of cone

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Kohnomi, Shuntaro, Noriaki Koshikawa, and Masayuki Kobayashi. "D2-like dopamine receptors differentially regulate unitary IPSCs depending on presynaptic GABAergic neuron subtypes in rat nucleus accumbens shell." Journal of Neurophysiology 107, no. 2 (2012): 692–703. http://dx.doi.org/10.1152/jn.00281.2011.

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In the nucleus accumbens (NAc), a medium spiny (MS) neuron receives GABAergic inputs from two major sources: fast-spiking (FS) neurons and other, adjacent MS neurons. These two types of inhibitory synapses are considered to play different roles in output activities, i.e., FS→MS connections suppress output from the NAc whereas MS→MS connections contribute to lateral inhibition. In the present study, we focused on the electrophysiological properties of unitary inhibitory postsynaptic currents (uIPSCs) obtained from MS→MS connections and FS→MS connections and examined the effects of quinpirole, a

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Akopian, A., and P. Witkovsky. "D2 dopamine receptor-mediated inhibition of a hyperpolarization-activated current in rod photoreceptors." Journal of Neurophysiology 76, no. 3 (1996): 1828–35. http://dx.doi.org/10.1152/jn.1996.76.3.1828.

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1. Using the whole cell patch clamp method, we investigated the effect of dopamine on a hyperpolarization-activated current (Ih) in the inner segments of rod photoreceptors of the Xenopus retina. 2. Ih was elicited by hyperpolarizing voltage steps to -120 mV from a holding potential of -40 mV. Dopamine reversibly reduced Ih in a dose-dependent manner. Dopamine-mediated inhibition of Ih was blocked by the D2 dopamine antagonist sulpiride. 3. The D2 dopamine agonist quinpirole (0.1-20 microM) inhibited Ih whereas the D1 agonist SKF-38393 (100 microM) had no effect on Ih. Quinpirole-induced inhib

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Fedotova, Julia. "Effects of Stimulation and Blockade of Receptor on Depression-Like Behavior in Ovariectomized Female Rats." ISRN Pharmacology 2012 (March 1, 2012): 1–8. http://dx.doi.org/10.5402/2012/305645.

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The aim of the present study was to explore the hedonic effects of D2 receptor agonist, quinpirole and D2 receptor antagonist, and sulpiride alone or in combination with a low dose of 17β-E2-estradiol (17β-E2) in the adult ovariectomized female rats (OVX). OVX rats of Wistar strain were used in all experiments. Two weeks after surgery rats were chronically treated with vehicle, a low dose of 17β-E2 (5.0 μg/rat), quinpirole (0.1 mg/kg), sulpiride (10.0 mg/kg), quinpirole plus 17β-E2, or sulpiride plus 17β-E2 for 14 days before the forced swimming test. We found that sulpiride significantly decr

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9

Wang, Xiaoyan, Van Anthony Villar, Andrew Tiu, Kiran K. Upadhyay, and Santiago Cuevas. "Dopamine D2 receptor upregulates leptin and IL-6 in adipocytes." Journal of Lipid Research 59, no. 4 (2018): 607–14. http://dx.doi.org/10.1194/jlr.m081000.

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Leptin is a pro-inflammatory cytokine secreted by the adipose tissue. Dopamine D2 receptors (D2Rs) have anti-inflammatory effects in the brain and kidney tissues. Mouse and human adipocytes express D2R; D2R protein was 10-fold greater in adipocytes from human visceral tissue than subcutaneous tissue. However, the function of D2R in adipocytes is not well understood. 3T3-L1 cells were treated with D2-like receptor agonist quinpirole, and immunoblot and quantitative PCR were performed. Quinpirole increased the protein and mRNA expression of leptin and IL-6, but not adiponectin and visfatin (24 h

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Eilam, David, and Henry Szechtman. "Psychostimulant-Induced Behavior as an Animal Model of Obsessive-Compulsive Disorder: An Ethological Approach to the Form of Compulsive Rituals." CNS Spectrums 10, no. 3 (2005): 191–202. http://dx.doi.org/10.1017/s109285290001004x.

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AbstractRats treated chronically with the D2/D3, dopamine receptor agonist quinpirole show a pattern of behavior that meets a set of ethologically derived criteria of compulsive behavior in obsessive-compulsive disorder (OCD). Moreover, in both quinpirole-treated rats and OCD patients, the structure of compulsive rituals appear similar in being composed of relatively few motor acts that are organized in a flexible yet recurrent manner. In addition, the development of compulsive behavior in quinpirole-treated rats is attenuated by the OCD pharmacotherapeutic drug clomipramine. These similaritie

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Kostrzewa, Richard M., Ryszard Brus, Monika Rykaczewska, and Andrzej Plech. "Low-dose quinpirole ontogenically sensitizes to quinpirole-induced yawning in rats." Pharmacology Biochemistry and Behavior 44, no. 2 (1993): 487–89. http://dx.doi.org/10.1016/0091-3057(93)90496-g.

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Lagière, Mélanie, Marion Bosc, Sara Whitestone, et al. "A Subset of Purposeless Oral Movements Triggered by Dopaminergic Agonists Is Modulated by 5-HT2C Receptors in Rats: Implication of the Subthalamic Nucleus." International Journal of Molecular Sciences 21, no. 22 (2020): 8509. http://dx.doi.org/10.3390/ijms21228509.

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Dopaminergic medication for Parkinson’s disease is associated with troubling dystonia and dyskinesia and, in rodents, dopaminergic agonists likewise induce a variety of orofacial motor responses, certain of which are mimicked by serotonin2C (5-HT2C) receptor agonists. However, the neural substrates underlying these communalities and their interrelationship remain unclear. In Sprague-Dawley rats, the dopaminergic agonist, apomorphine (0.03–0.3 mg/kg) and the preferential D2/3 receptor agonist quinpirole (0.2–0.5 mg/kg), induced purposeless oral movements (chewing, jaw tremor, tongue darting). T

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Carroll, J. L., K. M. Boyle, M. J. Wasicko, and L. M. Sterni. "Dopamine D2 receptor modulation of carotid body type 1 cell intracellular calcium in developing rats." American Journal of Physiology-Lung Cellular and Molecular Physiology 288, no. 5 (2005): L910—L916. http://dx.doi.org/10.1152/ajplung.00414.2003.

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Carotid chemoreceptor type 1 cells release dopamine, which inhibits carotid chemoreceptor activity via dopamine D2 autoreceptors on type 1 cells. Postnatal changes in dopaminergic modulation may be involved in postnatal chemoreceptor development. The present study explores dopaminergic modulation of the intracellular calcium ([Ca2+]i) response to hypoxia in type 1 cells from 1, 3, and 11- to 16-day-old rats. Using fura-2, we studied the effects of quinpirole, a D2 receptor agonist, on type 1 cell [Ca2+]i response to 90-s hypoxia challenges (Po2 ∼1–2 mmHg). Cells were sequentially exposed to th

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Frederick, Michael J., and Sarah E. Cocuzzo. "Contrafreeloading in Rats Is Adaptive and Flexible: Support for an Animal Model of Compulsive Checking." Evolutionary Psychology 15, no. 4 (2017): 147470491773593. http://dx.doi.org/10.1177/1474704917735937.

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Contrafreeloading involves working unnecessarily to obtain a reward that is otherwise freely available. It has been observed in numerous species and can be adaptive when it provides an organism with updated information about available resources. Humans frequently update their knowledge of the environment through checking behaviors. Compulsive checking occurs when such actions are performed with excessive frequency. In a putative animal model of compulsive checking, rats treated chronically with the dopamine agonist quinpirole display exaggerated contrafreeloading for water. Although this effec

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Yc, Karyna, Luis Prado, and Hugo Merchant. "The scalar property during isochronous tapping is disrupted by a D2-like agonist in the nonhuman primate." Journal of Neurophysiology 121, no. 3 (2019): 940–49. http://dx.doi.org/10.1152/jn.00804.2018.

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Dopamine, and specifically the D2 system, has been implicated in timing tasks where the absolute duration of individual time intervals is encoded discretely, yet the role of D2 during beat perception and entrainment remains largely unknown. In this type of timing, a beat is perceived as the pulse that marks equally spaced points in time and, once extracted, produces the tendency in humans to entrain or synchronize their movements to it. Hence, beat-based timing is crucial for musical execution. In this study we investigated the effects of systemic injections of quinpirole (0.005–0.05 mg/kg), a

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Maegawa, Hiroharu, Nayuka Usami, Chiho Kudo, Hiroshi Hanamoto, and Hitoshi Niwa. "Dopaminergic Modulation of Orofacial Mechanical Hypersensitivity Induced by Infraorbital Nerve Injury." International Journal of Molecular Sciences 21, no. 6 (2020): 1945. http://dx.doi.org/10.3390/ijms21061945.

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While the descending dopaminergic control system is not fully understood, it is reported that the hypothalamic A11 nucleus is its principle source. To better understand the impact of this system, particularly the A11 nucleus, on neuropathic pain, we created a chronic constriction injury model of the infraorbital nerve (ION-CCI) in rats. ION-CCI rats received intraperitoneal administrations of quinpirole (a dopamine D2 receptor agonist). ION-CCI rats received microinjections of quinpirole, muscimol [a gamma-aminobutyric acid type A (GABAA) receptor agonist], or neurotoxin 6-hydroxydopamine (6-O

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Luque-Rojas, María Jesús, Pablo Galeano, Juan Suárez, et al. "Hyperactivity induced by the dopamine D2/D3 receptor agonist quinpirole is attenuated by inhibitors of endocannabinoid degradation in mice." International Journal of Neuropsychopharmacology 16, no. 3 (2013): 661–76. http://dx.doi.org/10.1017/s1461145712000569.

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Abstract The present study was designed to investigate the effect of pharmacological inhibition of endocannabinoid degradation on behavioural actions of the dopamine D2/D3 receptor agonist quinpirole in male C57Bl/6J mice. In addition, we studied the effects of endocannabinoid degradation inhibition on both cocaine-induced psychomotor activation and behavioural sensitization. We analysed the effects of inhibition of the two main endocannabinoid degradation enzymes: fatty acid amide hydrolase (FAAH), using inhibitor URB597 (1 mg/kg); monoacylglycerol lipase (MAGL), using inhibitor URB602 (10 mg

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18

Zhu, Wylie H., Laura Conforti, and David E. Millhorn. "Expression of dopamine D2 receptor in PC-12 cells and regulation of membrane conductances by dopamine." American Journal of Physiology-Cell Physiology 273, no. 4 (1997): C1143—C1150. http://dx.doi.org/10.1152/ajpcell.1997.273.4.c1143.

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PC-12 cells depolarize during hypoxia and release dopamine. The hypoxia-induced depolarization is due to inhibition of an O2-sensitive K+ current. The role of dopamine released during hypoxia is uncertain, but it could act as an autocrine to modulate membrane conductance during hypoxia. The current study was undertaken to investigate this possibility. Reverse transcription-polymerase chain reaction and sequence analysis revealed that the D2 isoform of the dopamine receptor is expressed in rat PC-12 cells. Exogenously applied dopamine and the D2agonist quinpirole elicited inhibition of a voltag

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Liang, Jing, Vincent N. Marty, Yatendra Mulpuri, Richard W. Olsen, and Igor Spigelman. "Selective modulation of GABAergic tonic current by dopamine in the nucleus accumbens of alcohol-dependent rats." Journal of Neurophysiology 112, no. 1 (2014): 51–60. http://dx.doi.org/10.1152/jn.00564.2013.

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The nucleus accumbens (NAcc) is a key structure of the mesolimbic dopaminergic reward system and plays an important role in mediating alcohol-seeking behaviors. Alterations in glutamatergic and GABAergic signaling were recently demonstrated in the NAcc of rats after chronic intermittent ethanol (CIE) treatment, a model of alcohol dependence. Here we studied dopamine (DA) modulation of GABAergic signaling and how this modulation might be altered by CIE treatment. We show that the tonic current ( Itonic) mediated by extrasynaptic γ-aminobutyric acid type A receptors (GABAARs) of medium spiny neu

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Eagle, D. M., C. Schepisi, S. Chugh, et al. "Dissociable dopaminergic and pavlovian influences in goal-trackers and sign-trackers on a model of compulsive checking in OCD." Psychopharmacology 237, no. 12 (2020): 3569–81. http://dx.doi.org/10.1007/s00213-020-05636-3.

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Abstract Rationale Checking is a functional behaviour that provides information to guide behaviour. However, in obsessive-compulsive disorder (OCD), checking may escalate to dysfunctional levels. The processes underpinning the transition from functional to dysfunctional checking are unclear but may be associated with individual differences that support the development of maladaptive behaviour. We examined one such predisposition, sign-tracking to a pavlovian conditioned stimulus, which we previously found associated with dysfunctional checking. How sign-tracking interacts with another treatmen

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Schaus, John M., Diane L. Huser, and Robert D. Titus. "Synthesis of the Dopamine Agonist (-)-Quinpirole." Synthetic Communications 20, no. 22 (1990): 3553–62. http://dx.doi.org/10.1080/00397919008051599.

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Kurylo, Daniel D., and Sarah Tanguay. "Effects of quinpirole on behavioral extinction." Physiology & Behavior 80, no. 1 (2003): 1–7. http://dx.doi.org/10.1016/s0031-9384(03)00218-x.

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Roane, David S., and Dennis Paul. "Evidence of hyperglycemic hyperalgesia by quinpirole." Pharmacology Biochemistry and Behavior 41, no. 1 (1992): 65–67. http://dx.doi.org/10.1016/0091-3057(92)90060-s.

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Ahlenius, Sven, Viveka Hillegaart, and Agneta Wijkström. "Quinpirole — A 5-HT receptor antagonist?" Neuroscience Letters 126, no. 1 (1991): 57–59. http://dx.doi.org/10.1016/0304-3940(91)90370-9.

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Seeman, Philip, and John M. Schaus. "Dopamine receptors labelled by [3H] quinpirole." European Journal of Pharmacology 203, no. 1 (1991): 105–9. http://dx.doi.org/10.1016/0014-2999(91)90796-s.

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Schmidt, Timo T., Ellis Rea, Julia Shababi-Klein, George Panagis, and Christine Winter. "Enhanced reward-facilitating effects of d-amphetamine in rats in the quinpirole model of obsessive–compulsive disorder." International Journal of Neuropsychopharmacology 16, no. 5 (2013): 1083–91. http://dx.doi.org/10.1017/s1461145712000983.

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Abstract The underlying neurobiology of addictive or repetitive behaviours, such as obsessive–compulsive disorder (OCD), involves dopaminergic dysregulation. While addictive behaviour depends strongly on mesolimbocortical dopaminergic responses, repetitive behaviours have been associated with dopaminergic dysregulation in the basal ganglia–thalamo–cortical circuitry. The present study investigates differences in brain stimulation reward in rats with quinpirole-induced compulsive checking behaviour, in order to examine if deficits in reward processing are also relevant for OCD. Rats were tested

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Richards, Toni L., Thomas L. Pazdernik, and Beth Levant. "Altered quinpirole-induced local cerebral glucose utilization in anterior cortical regions in rats after sensitization to quinpirole." Brain Research 1042, no. 1 (2005): 53–61. http://dx.doi.org/10.1016/j.brainres.2005.02.013.

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Nimitvilai, Sudarat, Maureen A. McElvain, Devinder S. Arora, and Mark S. Brodie. "Reversal of quinpirole inhibition of ventral tegmental area neurons is linked to the phosphatidylinositol system and is induced by agonists linked to Gq." Journal of Neurophysiology 108, no. 1 (2012): 263–74. http://dx.doi.org/10.1152/jn.01137.2011.

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Putative dopaminergic (pDAergic) ventral tegmental area neurons play an important role in brain pathways related to addiction. Extended exposure of pDAergic neurons to moderate concentrations of dopamine (DA) results in a time-dependent decrease in sensitivity of pDAergic neurons to DA inhibition, a process called dopamine inhibition reversal (DIR). We have shown that DIR is mediated by phospholipase C and conventional protein kinase C through concurrent stimulation of D2 and D1-like receptors. In the present study, we further characterized this phenomenon by using extracellular recordings in

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Watanabe, Katsushige, Takako Kita, and Hitoshi Kita. "Presynaptic Actions of D2-Like Receptors in the Rat Cortico-Striato-Globus Pallidus Disynaptic Connection In Vitro." Journal of Neurophysiology 101, no. 2 (2009): 665–71. http://dx.doi.org/10.1152/jn.90806.2008.

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The cerebral cortex, the neostriatum (Str), and the external segment of the globus pallidus (GPe) form a cortico-Str-GPe disynaptic connection, which is one of the major connections in the basal ganglia circuitries and a target of dopamine modulation. The aim of this study was to examine the actions of D2-like dopamine receptors (D2LRs) in this connection using rat brain slice preparations. Electrical stimulation of the frontal cortex evoked disynaptic inhibitory postsynaptic currents (IPSCs) in cesium-filled GPe neurons voltage-clamped at 0 mV. The IPSCs evoked by threshold stimulation were s

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Wu, Michael, Stefan M. Brudzynski, and Gordon J. Mogenson. "Functional interaction of dopamine and glutamate in the nucleus accumbens in the regulation of locomotion." Canadian Journal of Physiology and Pharmacology 71, no. 5-6 (1993): 407–13. http://dx.doi.org/10.1139/y93-061.

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The interaction of dopamine and glutamate in the nucleus accumbens in the regulation of locomotion was investigated. Microinjection of N-methyl-D-aspartic acid (NMDA, a glutamatergic NMDA receptor agonist) or α-amino-3-hydroxy-5-methylisoxazole-4-propionate (AMPA, a quisqualic receptor agonist which is a glutamatergic non-NMDA receptor agonist) into the nucleus accumbens caused a substantial increase in locomotor activity. This increase in locomotor activity was significantly reduced by prior administration of the dopamine D2 agonist quinpirole, but not the D1 agonist, SKF 38393, into the same

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Van Hartesveldt, Carol, Merle E. Meyer, and Thomas J. Potter. "Ontogeny of biphasic locomotor effects of quinpirole." Pharmacology Biochemistry and Behavior 48, no. 3 (1994): 781–86. http://dx.doi.org/10.1016/0091-3057(94)90346-8.

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Koller, W., G. Herbster, D. Anderson, R. Wack, and J. Gordon. "Quinpirole hydrochloride, a potential anti-parkinsonism drug." Neuropharmacology 26, no. 8 (1987): 1031–36. http://dx.doi.org/10.1016/0028-3908(87)90245-0.

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Kostrzewa, Richard M., and Florence P. Kostrzewa. "Neonatal 6-Hydroxydopamine Lesioning Enhances Quinpirole-Induced Vertical Jumping in Rats that were Quinpirole Primed During Postnatal Ontogeny." Neurotoxicity Research 21, no. 2 (2011): 231–35. http://dx.doi.org/10.1007/s12640-011-9268-5.

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Mizuta, Kentaro, Yi Zhang, Dingbang Xu, Eiji Masaki, Reynold A. Panettieri, and Charles W. Emala. "The dopamine D2 receptor is expressed and sensitizes adenylyl cyclase activity in airway smooth muscle." American Journal of Physiology-Lung Cellular and Molecular Physiology 302, no. 3 (2012): L316—L324. http://dx.doi.org/10.1152/ajplung.00130.2011.

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Dopamine receptors are G protein-coupled receptors that are divided into two subgroups, “D1-like” receptors (D1 and D5) that couple to the Gs protein and “D2-like” receptors (D2, D3, and D4) that couple to Gi. Although inhaled dopamine has been reported to induce bronchodilation in patients with asthma, functional expression of dopamine receptor subtypes has never been described on airway smooth muscle (ASM) cells. Acute activation of Gi-coupled receptors inhibits adenylyl cyclase activity and cAMP synthesis, which classically impairs ASM relaxation. In contrast, chronic activation of Gi-coupl

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Vargas, Gricelly, and Mary T. Lucero. "Dopamine Modulates Inwardly Rectifying Hyperpolarization-Activated Current (I h) in Cultured Rat Olfactory Receptor Neurons." Journal of Neurophysiology 81, no. 1 (1999): 149–58. http://dx.doi.org/10.1152/jn.1999.81.1.149.

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Vargas, Gricelly and Mary T. Lucero. Dopamine modulates inwardly rectifying hyperpolarization-activated current ( I h) in cultured rat olfactory receptor neurons. J. Neurophysiol. 81: 149–158, 1999. The presence of dopamine receptors in olfactory receptor neurons (ORNs) suggests that odor sensitivity may be modulated by neurotransmitters at the level of primary sensory neurons. Using standard patch-clamp techniques on rat ORNs, we found that 1 μM dopamine, 500 μM SQ 22536 (SQ, an adenylyl cyclase inhibitor), 20 and 50 μM quinpirole (a selective dopamine D2 receptor agonist), and 1 mM adenosine

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Congar, Patrice, Annie Bergevin, and Louis-Eric Trudeau. "D2 Receptors Inhibit the Secretory Process Downstream From Calcium Influx in Dopaminergic Neurons: Implication of K+ Channels." Journal of Neurophysiology 87, no. 2 (2002): 1046–56. http://dx.doi.org/10.1152/jn.00459.2001.

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Dopaminergic (DAergic) neurons possess D2-like somatodendritic and terminal autoreceptors that modulate cellular excitability and dopamine (DA) release. The cellular and molecular processes underlying the rapid presynaptic inhibition of DA release by D2 receptors remain unclear. Using a culture system in which isolated DAergic neurons establish self-innervating synapses (“autapses”) that release both DA and glutamate, we studied the mechanism by which presynaptic D2 receptors inhibit glutamate-mediated excitatory postsynaptic currents (EPSCs). Action-potential evoked EPSCs were reversibly inhi

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Bertrand, F., J. Thiery, S. Picard, and B. Malpaux. "Implication of D2-like dopaminergic receptors in the median eminence during the establishment of long-day inhibition of LH secretion in the ewe." Journal of Endocrinology 163, no. 2 (1999): 243–54. http://dx.doi.org/10.1677/joe.0.1630243.

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In ewes, photoperiod modulates LH release and dopaminergic terminals in the median eminence (ME) have a critical role in the establishment of long-day inhibition of LH secretion. This study was undertaken to determine the type of dopaminergic receptors, D1-like or D2-like, that mediate the action of dopamine on LH secretion at the ME level in this situation. This was assessed, in ovariectomized and estradiol-treated ewes, with the use of reverse microdialysis in the ME in three experiments: first, when LH secretion was stimulated by short days, by determining the response to three doses (0.01,

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Kontis, D., V. Boulougouris, S. Papadopoulos, et al. "Directional Persistence in the Rewarded Alternation Model of Obsessive-compulsive Disorder is Responsive to both Dopaminergic and Serotonergic Manipulations." European Psychiatry 24, S1 (2009): 1. http://dx.doi.org/10.1016/s0924-9338(09)71264-7.

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Rationale:In the rewarded alternation model of obsessive compulsive disorder (OCD), the serotonin agonist m-chlorophenylpiperazine (mCPP) increases persistent behaviour, while chronic pretreatment with selective serotonin reuptake inhibitor (SSRI-fluoxetine) but not benzodiazepine or desipramine abolishes mCPP effects. However, we noted that acute SSRI administration also causes transient persistence increases, counteracted by mCPP pretreatment.Objectives:This studya.further explores the apparent cross-tolerance between fluoxetine and mCPP andb.extends the model by investigating its sensitivit

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Paíno, Carlos L. "Evaluation of Activity Patterns in Quinpirole-Treated Rats." Journal of Behavioral and Brain Science 04, no. 07 (2014): 291–96. http://dx.doi.org/10.4236/jbbs.2014.47030.

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GORRITI, M., B. FERRER, I. DELARCO та ін. "Acute Δ-tetrahydrocannabinol exposure facilitates quinpirole-induced hyperlocomotion". Pharmacology Biochemistry and Behavior 81, № 1 (2005): 71–77. http://dx.doi.org/10.1016/j.pbb.2005.02.004.

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Nowak, PrzemysŁaw, Łukasz Labus, Richard M. Kostrzewa, and Ryszard Brus. "DSP-4 prevents dopamine receptor priming by quinpirole." Pharmacology Biochemistry and Behavior 84, no. 1 (2006): 3–7. http://dx.doi.org/10.1016/j.pbb.2006.03.024.

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Demontis, Francesca, and Gino Serra. "Failure of memantine to “reverse” quinpirole-induced hypomotility." World Journal of Psychiatry 6, no. 2 (2016): 215. http://dx.doi.org/10.5498/wjp.v6.i2.215.

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Baladi, Michelle G., Amy H. Newman, Yvonne M. Thomas, and Charles P. France. "Drinking sucrose enhances quinpirole-induced yawning in rats." Behavioural Pharmacology 22, no. 8 (2011): 773–78. http://dx.doi.org/10.1097/fbp.0b013e32834d0f3c.

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SCHAUS, J. M., D. L. HUSER, and R. D. TITUS. "ChemInform Abstract: Synthesis of the Dopamine Agonist (-)-Quinpirole." ChemInform 22, no. 30 (2010): no. http://dx.doi.org/10.1002/chin.199130277.

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Hartesveldt, Carol Van, Georgia A. Cottrell, Thomas Potter, and Merle E. Meyer. "Effects of intracerebral quinpirole on locomotion in rats." European Journal of Pharmacology 214, no. 1 (1992): 27–32. http://dx.doi.org/10.1016/0014-2999(92)90091-h.

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Bananej, Maryam, Ahmad Karimi-Sori, Mohammad Reza Zarrindast, and Shamseddin Ahmadi. "D1 and D2 dopaminergic systems in the rat basolateral amygdala are involved in anxiogenic-like effects induced by histamine." Journal of Psychopharmacology 26, no. 4 (2011): 564–74. http://dx.doi.org/10.1177/0269881111405556.

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Involvement of the dopamine receptors in the basolateral amygdala (BLA) in the effects of histamine on anxiety-like behaviors of the elevated plus maze in male Wistar rats was investigated. The results showed that bilateral intra-BLA injections of histamine (2.5, 5 and 7.5 µg/rat) induced an anxiogenic-like effect, revealed by decreases in percentage of open arm time (%OAT) and open arm entries (%OAE). Intra-BLA administration of dopamine D1 receptor agonist, SKF38393 (0.25 µg/rat), and dopamine D2 receptor agonist, quinpirole (0.03 and 0.05 µg/rat), decreased %OAT but not %OAE. Conversely, in

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Hadipour-Niktarash, Arash, Karen S. Rommelfanger, Gunasingh J. Masilamoni, Yoland Smith, and Thomas Wichmann. "Extrastriatal D2-like receptors modulate basal ganglia pathways in normal and parkinsonian monkeys." Journal of Neurophysiology 107, no. 5 (2012): 1500–1512. http://dx.doi.org/10.1152/jn.00348.2011.

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According to traditional models of the basal ganglia-thalamocortical network of connections, dopamine exerts D2-like receptor (D2LR)-mediated effects through actions on striatal neurons that give rise to the “indirect” pathway, secondarily affecting the activity in the internal and external pallidal segments (GPi and GPe, respectively) and the substantia nigra pars reticulata (SNr). However, accumulating evidence from the rodent literature suggests that D2LR activation also directly influences synaptic transmission in these nuclei. To further examine this issue in primates, we combined in vivo

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Damase-Michel, Christine, Jean-Louis Montastruc, Marie-Antoinette Tran, Claude Gharib, Ghislaine Geelen, and Paul Montastruc. "Involvement of vasopressin in the cardiovascular effects of quinpirole." European Journal of Pharmacology 184, no. 1 (1990): 179–83. http://dx.doi.org/10.1016/0014-2999(90)90680-5.

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RAHMINIWATI, Min, and Masakazu NISHIMURA. "Diazepam-Induced Hyperphagia in Mice is Sensitive to Quinpirole." Journal of Veterinary Medical Science 61, no. 7 (1999): 777–80. http://dx.doi.org/10.1292/jvms.61.777.

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Kostrzewa, Richard M., and Ryszard Brus. "Ontogenic homologous supersensitization of quinpirole-induced yawning in rats." Pharmacology Biochemistry and Behavior 39, no. 2 (1991): 517–19. http://dx.doi.org/10.1016/0091-3057(91)90219-r.

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