Relevant bibliographies by topics / R gene-mediated resistance

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Academic literature on the topic 'R gene-mediated resistance'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "R gene-mediated resistance"

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MIYASHITA, Shuhei, and Hideki TAKAHASHI. "R-gene-mediated resistance to plant viruses." Uirusu 65, no. 2 (2015): 199–208. http://dx.doi.org/10.2222/jsv.65.199.

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Parker, Jane E., Bart J. Feys, Erik A. Van Der Biezen, et al. "Unravelling R gene-mediated disease resistance pathways in Arabidopsis." Molecular Plant Pathology 1, no. 1 (2000): 17–24. http://dx.doi.org/10.1046/j.1364-3703.2000.00003.x.

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Tian, D., M. B. Traw, J. Q. Chen, M. Kreitman, and J. Bergelson. "Fitness costs of R-gene-mediated resistance in Arabidopsis thaliana." Nature 423, no. 6935 (2003): 74–77. http://dx.doi.org/10.1038/nature01588.

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Eschen-Lippold, Lennart, Simone Altmann, Christiane Gebhardt, Cornelia Göbel, Ivo Feussner, and Sabine Rosahl. "Oxylipins are not required for R gene-mediated resistance in potato." European Journal of Plant Pathology 127, no. 4 (2010): 437–42. http://dx.doi.org/10.1007/s10658-010-9621-1.

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Cantila, Aldrin Y., Nur Shuhadah Mohd Saad, Junrey C. Amas, David Edwards, and Jacqueline Batley. "Recent Findings Unravel Genes and Genetic Factors Underlying Leptosphaeria maculans Resistance in Brassica napus and Its Relatives." International Journal of Molecular Sciences 22, no. 1 (2020): 313. http://dx.doi.org/10.3390/ijms22010313.

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Among the Brassica oilseeds, canola (Brassica napus) is the most economically significant globally. However, its production can be limited by blackleg disease, caused by the fungal pathogen Lepstosphaeria maculans. The deployment of resistance genes has been implemented as one of the key strategies to manage the disease. Genetic resistance against blackleg comes in two forms: qualitative resistance, controlled by a single, major resistance gene (R gene), and quantitative resistance (QR), controlled by numerous, small effect loci. R-gene-mediated blackleg resistance has been extensively studied, wherein several genomic regions harbouring R genes against L. maculans have been identified and three of these genes were cloned. These studies advance our understanding of the mechanism of R gene and pathogen avirulence (Avr) gene interaction. Notably, these studies revealed a more complex interaction than originally thought. Advances in genomics help unravel these complexities, providing insights into the genes and genetic factors towards improving blackleg resistance. Here, we aim to discuss the existing R-gene-mediated resistance, make a summary of candidate R genes against the disease, and emphasise the role of players involved in the pathogenicity and resistance. The comprehensive result will allow breeders to improve resistance to L. maculans, thereby increasing yield.
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COOPER, W. C., L. JIA, and F. L. GOGGIN. "ACQUIRED AND R-GENE-MEDIATED RESISTANCE AGAINST THE POTATO APHID IN TOMATO." Journal of Chemical Ecology 30, no. 12 (2004): 2527–42. http://dx.doi.org/10.1007/s10886-004-7948-9.

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Warren, Randall F., Peter M. Merritt, Eric Holub, and Roger W. Innes. "Identification of Three Putative Signal Transduction Genes Involved in R Gene-Specified Disease Resistance in Arabidopsis." Genetics 152, no. 1 (1999): 401–12. http://dx.doi.org/10.1093/genetics/152.1.401.

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Abstract The RPS5 disease resistance gene of Arabidopsis mediates recognition of Pseudomonas syringae strains that possess the avirulence gene avrPphB. By screening for loss of RPS5-specified resistance, we identified five pbs (avrPphB susceptible) mutants that represent three different genes. Mutations in PBS1 completely blocked RPS5-mediated resistance, but had little to no effect on resistance specified by other disease resistance genes, suggesting that PBS1 facilitates recognition of the avrPphB protein. The pbs2 mutation dramatically reduced resistance mediated by the RPS5 and RPM1 resistance genes, but had no detectable effect on resistance mediated by RPS4 and had an intermediate effect on RPS2-mediated resistance. The pbs2 mutation also had varying effects on resistance mediated by seven different RPP (recognition of Peronospora parasitica) genes. These data indicate that the PBS2 protein functions in a pathway that is important only to a subset of disease-resistance genes. The pbs3 mutation partially suppressed all four P. syringae-resistance genes (RPS5, RPM1, RPS2, and RPS4), and it had weak-to-intermediate effects on the RPP genes. In addition, the pbs3 mutant allowed higher bacterial growth in response to a virulent strain of P. syringae, indicating that the PBS3 gene product functions in a pathway involved in restricting the spread of both virulent and avirulent pathogens. The pbs mutations are recessive and have been mapped to chromosomes I (pbs2) and V (pbs1 and pbs3).
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Makino, Seiko, Akiko Sugio, Frank White, and Adam J. Bogdanove. "Inhibition of Resistance Gene-Mediated Defense in Rice by Xanthomonas oryzae pv. oryzicola." Molecular Plant-Microbe Interactions® 19, no. 3 (2006): 240–49. http://dx.doi.org/10.1094/mpmi-19-0240.

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Xanthomonas oryzae pv. oryzae and the closely related X. oryzae pv. oryzicola cause bacterial blight and bacterial leaf streak of rice, respectively. Although many rice resistance (R) genes and some corresponding avirulence (avr) genes have been characterized for bacterial blight, no endogenous avr/R gene interactions have been identified for leaf streak. Genes avrXa7 and avrXa10 from X. oryzae pv. oryzae failed to elicit the plant defense-associated hypersensitive reaction (HR) and failed to prevent development of leaf streak in rice cultivars with the corresponding R genes after introduction into X. oryzae pv. oryzicola despite the ability of this pathovar to deliver an AvrXa10:Cya fusion protein into rice cells. Furthermore, coinoculation of X. oryzae pv. oryzicola inhibited the HR of rice cultivar IRBB10 to X. oryzae pv. oryzae carrying avrXa10. Inhibition was quantitative and dependent on the type III secretion system of X. oryzae pv. oryzicola. The results suggest that one or more X. oryzae pv. oryzicola type III effectors interfere with avr/R gene-mediated recognition or signaling and subsequent defense response in the host. Inhibition of R gene-mediated defense by X. oryzae pv. oryzicola may explain, in part, the apparent lack of major gene resistance to leaf streak.
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Sugawara, Kyoko, Takuya Shiraishi, Tetsuya Yoshida, et al. "A Replicase of Potato virus X Acts as the Resistance-Breaking Determinant for JAX1-Mediated Resistance." Molecular Plant-Microbe Interactions® 26, no. 9 (2013): 1106–12. http://dx.doi.org/10.1094/mpmi-04-13-0094-r.

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Lectin-mediated resistance (LMR) has been suggested to comprise an uncharacterized branch of antiviral plant innate immunity. To unveil the feature of resistance conferred by jacalin-type lectin required for potexvirus resistance 1 (JAX1), a recently isolated LMR gene against potexviruses, we analyzed the resistance-breaking variants to find the viral component involved in resistance. We employed grafting-mediated inoculation, a high-pressure virus inoculation method, to obtain Potato virus X (PVX) variants that can overcome JAX1-mediated resistance. Whole-genome sequencing of the variants suggested that a single amino acid in the methyl transferase domain of the replicase encoded by PVX is responsible for this resistance-breaking property. Reintroduction of the amino-acid substitution to avirulent wild-type PVX was sufficient to overcome the JAX1-mediated resistance. These results suggest that viral replicase is involved in JAX1-mediated resistance. The residue that determines the resistance-breaking properties was highly conserved among potexviruses, suggesting a general role of the residue in potexvirus–JAX1 interactions.
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Römer, Patrick, Simone Hahn, Tina Jordan, Tina Strauß, Ulla Bonas, and Thomas Lahaye. "Plant Pathogen Recognition Mediated by Promoter Activation of the Pepper Bs3 Resistance Gene." Science 318, no. 5850 (2007): 645–48. http://dx.doi.org/10.1126/science.1144958.

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Plant disease resistance (R) proteins recognize matching pathogen avirulence proteins. Alleles of the pepper R gene Bs3 mediate recognition of the Xanthomonas campestris pv. vesicatoria (Xcv) type III effector protein AvrBs3 and its deletion derivative AvrBs3Δrep16. Pepper Bs3 and its allelic variant Bs3-E encode flavin monooxygenases with a previously unknown structure and are transcriptionally activated by the Xcv effector proteins AvrBs3 and AvrBs3Δrep16, respectively. We found that recognition specificity resides in the Bs3 and Bs3-E promoters and is determined by binding of AvrBs3 or AvrBs3Δrep16 to a defined promoter region. Our data suggest a recognition mechanism in which the Avr protein binds and activates the promoter of the cognate R gene.
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Dissertations / Theses on the topic "R gene-mediated resistance"

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Yildirim, Figen. "Elucidation Of R Gene Mediated Yellow Rust Disease Resistance Mechanism In Wheat By Dual Bait Yeast Two-hybrid Analysis." Phd thesis, METU, 2005. http://etd.lib.metu.edu.tr/upload/12606473/index.pdf.

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Yellow rust, caused by Puccinia striiformis Westend. f. sp. tritici Eriksson is one of the most severe leaf diseases of wheat. Aim of this study is to illuminate the downstream signaling pathways upon incompetible infection of rust pathogen in wheat, thus to understand the genes involved in resistance mechanism. The strategy used is the dual bait yeast two-hybrid analysis which is the most powerful method for in vivo detection of protein-protein interactions. The bait proteins used are<br>the domains of Yr10 yellow rust resistance gene, Rad6 gene which is considered to have a critical role in R gene mediated signaling pathway, and WR5 gene fragment which is an unknown protein having homology to the WD40 repeat containing protein with apoptosis related activity. Screening of a yeast prey library with these baits revealed proteins having mostly apoptosis related functions (SRP72, POR1, CSE1), translation initiation control in response to stress conditions (Gcn2p, Eap1p), phosphorylation (SKY1) and dephosphorylation activities (GAC1), cell cycle control (FAR1), oxidative stress control (OXR1), protein degradation control (TOM1), protein folding control (CPR7) and ion homeostasis in the cell (POR1, GAC1). The significance of the study can be summarized as i) being the first yeast two hybrid analysis of a wheat R gene, ii) being able to detect interacting partners with anticipated functions, iii) most importantly, initiating further detailed analysis of the key interactors.
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Mitrousia, Georgia. "Effectiveness of resistance against Leptosphaeria species (phoma stem canker) in oilseed rape." Thesis, University of Hertfordshire, 2016. http://hdl.handle.net/2299/17228.

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To improve understanding of the effectiveness of host resistance against Leptosphaeria spp., three aspects of effectiveness of resistance were investigated. With focus on the major Rlm-mediated resistance against L. maculans, changes in effectiveness of Rlm7-mediated resistance to prevent initiation of disease epidemics at the leaf spot stage were investigated in winter oilseed rape field experiments at five sites in the UK over the period with the cropping seasons 2009/2010 - 2013/2014. L. maculans isolates virulent against Rlm7 were identified in the UK. This may be associated with observed changes in lesion phenotypes on the Rlm7 cultivars in field conditions. However, despite increased severity of phoma leaf spotting on Rlm7 cultivars, there was no associated increase in phoma stem canker severity at the end of the cropping seasons. The effectiveness of winter oilseed rape cultivars for control of phoma stem canker (caused by L. maculans or L. biglobosa) was affected by the coexistence of the two Leptosphaeria species in oilseed rape crops. Weather conditions influenced ascospore release of both species and favoured L. biglobosa ascospore release in 2011, resulting in subsequent increased L. biglobosa phoma leaf spotting and stem canker severity. However, coexistence of Leptosphaeria spp. on oilseed rape crops was affected by the cultivar resistance against L. maculans. CE experiments showed that there were interactions between the two Leptosphaeria spp. in planta. Their coexistence on B. napus was influenced by the different host responses that they trigger during host colonisation. Effects of increased temperature on effectiveness of resistance against L. maculans and on severity of symptoms by Leptosphaeria spp. on B. napus were investigated. Increased temperature affected both Rlm4- and Rlm7-mediated resistance, when assessed by phenotypic and molecular techniques. Increased temperature was associated with increased symptom severity, for both L. maculans and L. biglobosa lesions on plants. Cultivar quantitative resistance background increased effectiveness of resistance against phoma stem canker pathogens at increased temperature and should be deployed in in strategies for adaptation to climate change to avoid increased phoma stem canker epidemics in the future.
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Books on the topic "R gene-mediated resistance"

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O'Brien, Donalee. DNA-mediated transfer of a neomycin resistance gene into the Y r mouse adrenocortical tumour cell line. 1987.

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Book chapters on the topic "R gene-mediated resistance"

1

Ishihara, Takeaki, Yukiyo Sato, and Hideki Takahashi. "Microarray Analysis of R-Gene-Mediated Resistance to Viruses." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1743-3_15.

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2

Moffett, P. "Mechanisms of Recognition in Dominant R Gene Mediated Resistance." In Advances in Virus Research. Elsevier, 2009. http://dx.doi.org/10.1016/s0065-3527(09)07501-0.

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