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Relevant bibliographies by topics / Ramiprilat

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Academic literature on the topic 'Ramiprilat'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Ramiprilat"

1

Vree, Tom B., Erik Dammers, Ivan Ulc, Stefan Horkovics-Kovats, Miroslav Ryska, and IJsbrand Merkx. "Lack of Male-Female Differences in Disposition and Esterase Hydrolysis of Ramipril to Ramiprilat in Healthy Volunteers after a Single Oral Dose." Scientific World JOURNAL 3 (2003): 1344–58. http://dx.doi.org/10.1100/tsw.2003.122.

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The objective of this study was to identify differences in disposition and esterase hydrolysis of ramipril between male and female volunteers. Plasma concentration and area under the concentration-time curve until the last measured concentration (AUCt) data of ramipril and its active metabolite ramiprilat (-diacid) were obtained from a randomised, cross-over bioequivalence study in 36 subjects (18 females and 18 males). Participants received a single 5-mg oral dose of two different formulations of ramipril (Formulation I and II). Plasma ramipril and ramiprilat concentrations were determined ac

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Trobec, Katja Čvan, Iztok Grabnar, Jurij Trontelj, Mitja Lainščak, and Mojca Kerec Kos. "Population pharmacokinetics of ramipril in patients with chronic heart failure: A real-world longitudinal study." Acta Pharmaceutica 74, no. 2 (2024): 315–28. http://dx.doi.org/10.2478/acph-2024-0018.

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Abstract In patients with chronic heart failure (CHF), the use of angiotensin-converting enzyme inhibitors, including ramipril, is recommended to reduce the risk of heart failure worsening, hospitalisation, and death. Our aim was to investigate the influence of body composition on the pharmacokinetics of ramipril and its active metabolite ramiprilat and to evaluate the changes in pharmacokinetics after prolonged therapy. Twenty-three patients with CHF who were on regular therapy with ramipril participated at the first study visit ( median age 77 years, 65 % male, and 70 % New York Heart Associ

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M.Hareesh, Reddy* and Dr.A. Sambasiva Rao. "A COMPRAHENSIVE REVIEW OF PHARMACOLOGICAL AND THERAPEUTICAL ACTIVITIES OF ANTIHYPERTENSIVE DRUG RAMIPRIL." Indo American Journal of Pharmaceutical Sciences 04, no. 11 (2017): 3951–53. https://doi.org/10.5281/zenodo.1042737.

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Ramipril is an anti-hypertensive drug, a category of ACE inhibitor that inhibit the actions of angiotensin converting enzyme (ACE), thereby decreasing the production of angiotensin II and decreasing the breakdown of bradykinin. The decrease in an enzyme angiotensin II results in relaxation of arteriole smooth muscle leading to a lowering the total peripheral resistance, reducing blood pressure(BP) as the blood is pumped through widened. Ramipril, a precursor or prodrug, is converted to the active metabolite ramiprilat by carboxylesterase. It is mostly excreted by the kidneys. Its half-life is

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Khazaei, M., AM Sharifi, S. Golbidi, and I. Laher. "Reduction in Risk of Myocardial Infarction, Stroke, and Death from Cardiovascular Causes. Focus on Rampiril." Clinical Medicine. Therapeutics 1 (January 2009): CMT.S2095. http://dx.doi.org/10.4137/cmt.s2095.

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Several clinical studies demonstrate a beneficial role of angiotensin-converting enzyme (ACE) inhibitors in patients with myocardial infarction, hypertension and diabetes mellitus. This review focuses on the effects of ramipril, a weak inhibitor of ACE that is rapidly hydrolyzed to ramiprilat, an active metabolite. The Heart Outcome Prevention Evaluation (HOPE) study evaluated the effects of ramipril in patients with a high risk for cardiovascular events without pre-existing left ventricular dysfunction or heart failure. In this review, we summarized the effects of ramipril on myocardial infar

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Komers, R., and M. E. Cooper. "Acute renal hemodynamic effects of ACE inhibition in diabetic hyperfiltration: role of kinins." American Journal of Physiology-Renal Physiology 268, no. 4 (1995): F588—F594. http://dx.doi.org/10.1152/ajprenal.1995.268.4.f588.

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Angiotensin converting enzyme (ACE) inhibitors not only reduce angiotensin II (ANG II) levels but also inhibit kinin degradation. The relative roles of ANG II and bradykinin in the acute action of ACE inhibitors on renal hemodynamic parameters in rats after 3 wk of diabetes were explored using antagonists of the ANG II type 1 (AT1) and the bradykinin B2 receptors. Conscious control and streptozotocin diabetic male Sprague-Dawley rats were randomized to receive vehicle, the ACE inhibitor, ramiprilat, the B2-receptor blocker, HOE-140, the AT1-receptor blocker, valsartan, or the combination of ra

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DESMOULINS, P. O., S. BURGAUD, and L. J. I. HORSPOOL. "Pharmacokinetics and pharmacodynamics of ramipril and ramiprilat in healthy cats." Journal of Veterinary Pharmacology and Therapeutics 31, no. 4 (2008): 349–58. http://dx.doi.org/10.1111/j.1365-2885.2008.00959.x.

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A.B., Gangurde Mohammed Awais* V. A. Bairagi Abdurrahman Sanaurrehman Karishma Rajashri. "FORMULATION DEVELOPMENT AND IN VITRO EVALUATION OF FAST DISSOLVING TABLETS OF RAMIPRIL SOLID DISPERSION." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 08 (2018): 8409–16. https://doi.org/10.5281/zenodo.1411687.

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<em>Ramipril is a prodrug belonging to the angiotensin</em>-<em>converting enzyme </em>(<em>ACE</em>) <em>inhibitor which is metabolized to Ramiprilat in the liver and, to a lesser extent, kidneys</em>.<em>The poor solubility and wett</em> <em>ability of Ramipril leads to poor dissolution and variations in bioavailability</em>.<em>Solid dispersion of Ramipril was prepared using Hydroxy propyl β</em>- <em>cyclodextrin to improve water solubility</em>. <em>Prepared solid dispersion was shown improved solubility in water of 97</em>.<em>5 µg</em>/<em>ml</em>. <em>Fast dissolving tablets

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Skoglof, A., P. O. Göthe, and J. Deinum. "Effect of temperature and chloride on steady-state inhibition of angiotensin I-converting enzyme by enalaprilat and ramiprilat." Biochemical Journal 272, no. 2 (1990): 415–19. http://dx.doi.org/10.1042/bj2720415.

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The kinetics of the steady-state inhibition of angiotension I-converting enzyme (EC 3.4.15.1) at 25 degrees C and 37 degrees C with enalaprilat and ramiprilat can be simulated, assuming only one inhibitor-binding site, consistent with a 1:1 stoichiometry if the protein concentration was determined by amino acid analysis. In this temperature range the apparent inhibition constants for ramiprilat and enalaprilat were roughly doubled by a decrease in the chloride concentration from 0.300 M to 0.120 M.

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Chernonosov, Alexander. "The Use of Dried Blood Spots for the Quantification of Antihypertensive Drugs." International Journal of Analytical Chemistry 2018 (August 1, 2018): 1–12. http://dx.doi.org/10.1155/2018/3235072.

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Hypertension or high blood pressure is a harbinger of cardiovascular diseases. There are several classes of drugs used to treat hypertension. This review discusses the use of dried blood spots (DBSs) for the quantification by mass spectrometry (MS), tandem mass spectrometry (MS/MS), or, in some cases, by fluorescence detection methods the following antihypertensive medications: angiotensin-converting enzyme inhibitors (ramipril, ramiprilat, captopril, and lisinopril); angiotensin II receptor antagonists (valsartan, irbesartan, losartan, and losartan carboxylic acid); calcium channel blockers (

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Safari, Fatemeh, Sohrab Hajizadeh, Shahnaz Shekarforoush, Gholamreza Bayat, Mohsen Foadoddini, and Ali Khoshbaten. "Influence of ramiprilat and losartan on ischemia reperfusion injury in rat hearts." Journal of the Renin-Angiotensin-Aldosterone System 13, no. 1 (2011): 29–35. http://dx.doi.org/10.1177/1470320311426025.

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Hypothesis/introduction: Our aim was to investigate whether a non-hypotensive dose of ramiprilat and losartan has myocardial protective effects during myocardial ischemia/reperfusion in vivo. Materials and methods: Three groups of rats were given 10 mg/kg per day of losartan for one (L-1W), four (L-4W) or 10 (L-10W) weeks. Another three groups were given 50 µg/kg per day of ramiprilat for one (R-1W), four (R-4W) or 10 (R-10W) weeks. The animals underwent 30 min of left anterior descending artery occlusion and subsequent reperfusion for 120 min. Results: Myocardial infarct size (IS) was reduced

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Dissertations / Theses on the topic "Ramiprilat"

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Schuckenböhmer, Jan Patrick [Verfasser]. "Einfluss des ACE-Hemmers Ramiprilat auf ventrikuläre Kardiomyozyten adulter Ratten / Jan Patrick Schuckenböhmer." Gießen : Universitätsbibliothek, 2014. http://d-nb.info/1068541342/34.

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Chajara, Abdessalam. "Etude de la réaction scléreuse de l'aorte thoracique de rat consécutive à une lésion à l'aide d'un cathéter à ballonet. : Applications pharmacologiques." Paris 13, 1993. http://www.theses.fr/1993PA132024.

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La réaction sclérotique des cellules musculaires lisses (migration, prolifération et synthèse des molécules de la matrice extracellulaire dans l'intima artérielle) joue un rôle majeur dans l'atherogenese, la restenose après angioplastie transluminale percutanée et l'épaississement artériel aberrant chez les hypertendus. Le but de nos travaux a été: premièrement, d'analyser la réaction sclérotique de l'aorte thoracique de rat en réponse à une lésion mécanique a l'aide d'un ballonnet gonflable, deuxièmement, de tester différents types de molécules (héparine, ramipril un inhibiteur de l'enzyme de

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Akashi, Ana Paula. "Efeitos do ramipril sobre a doença periodontal induzida experimentalmente em ratos." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/25/25149/tde-17052016-162345/.

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A doença periodontal (DP) corresponde a um grupo de doenças inflamatórias que acomete as estruturas periodontais de proteção e de suporte e pode levar à perda dentária. A etiologia está relacionada à placa dentobacteriana que leva à produção de grande quantidade de citocinas pró-inflamatórias importantes na destruição tecidual. A angiotensina (Ang) II também pode contribuir para a inflamação e destruição tecidual no periodonto agindo como mediador chave. A utilização de drogas que atuem na cascata do sistema renina-angiotensina (SRA) poderia interferir no estado de saúde ou inflamação do tecid

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Schulz, Sabine-Susan. "Untersuchungen zum myokardialen Sauerstoffradikal-Stoffwechsel am Tiermodell 30 - 36 Stunden und 6 Wochen nach Myokardinfarkt unter medikamentöser Therapie mittels Ramipril, Metoprolol und Kombinationstherapie Metoprolol, Ramipril." [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=973496827.

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Schulz, Sabine-Susan. "Untersuchungen zum myokardialen Sauerstoffradikal-Stoffwechsel am Tiermodell 30 - 36 Stunden und 6 Wochen nach Myokardinfarkt unter medikamentöser Therapie mittels Ramipril, Metoprolol und Kombinationstherapie Metoprolol/Ramipril." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2005. http://dx.doi.org/10.18452/15164.

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An Herzinsuffizienz sind in Deutschland weit mehr als 1 Mio. Menschen erkrankt. Ihre Häufigkeit steigt stetig an. Die Herzinsuffizienz wird als dominierende Herz-Kreislauf-Erkrankung des 21. Jahrhunderts angesehen. Die Hauptursache der Herzinsuffizienz ist die koronare Herzerkrankung besonderes nach stattgehabtem Myokardinfarkt. Sowohl für den akuten Myokardinfarkt als auch für die sich auf dieser Basis entwickelnde Herzinsuffizienz werden Veränderungen im Stoffwechsel der Sauerstoffradikale als pathophysiologisch bedeutsam angesehen. Es wird angenommen, dass als Folge der akuten Myokardischäm

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Kawahara, Elisa Ito. "Caracterização do fenótipo ósseo do modelo mgΔloxPneo da síndrome de Marfan e análise dos mecanismos de patogênese." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/41/41131/tde-21032017-152240/.

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A Síndrome de Marfan (SMF) é uma doença de caráter autossômico dominante que acomete o tecido conjuntivo. As principais manifestações clínicas afetam o sistema cardiovascular, ótico e ósseo. A SMF é causada por mutações no gene FBN1, que codifica a proteína extracelular fibrilina-1, componente principal das microfibrilas, que formam as fibras elásticas. Estudos mostraram que mutações no gene da fibrilina-1 levam a um aumento indiscriminado do TGF-Β ativo na matriz, que resulta nos principais fenótipos da doença. O Sistema Renina Angiotensina (RAS) tem como produto principal a Angiotensina

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Miranda, Roberto Dischinger [UNIFESP]. "Estudo duplo-cego e randomizado para avaliar a eficácia e a tolerabilidade da combinação fixa Anlodipina/Ramipril comparada ao Anlodipino isolado em pacientes hipertensos." Universidade Federal de São Paulo (UNIFESP), 2009. http://repositorio.unifesp.br/handle/11600/9577.

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Made available in DSpace on 2015-07-22T20:50:09Z (GMT). No. of bitstreams: 0 Previous issue date: 2009-06-24. Added 1 bitstream(s) on 2015-08-11T03:26:18Z : No. of bitstreams: 1 Publico-135.pdf: 902121 bytes, checksum: dc8fe74d80e38756535c78264933c2a2 (MD5)<br>Introdução: A utilização de anti-hipertensivos com diferentes mecanismos de ação em combinação fixa pode oferecer vantagens na adesão ao tratamento, na eficácia e na tolerabilidade. A combinação de um bloqueador dos canais de cálcio com um inibidor da enzima conversora da angiotensina pode reduzir a incidência de edema periférico, al

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Vinçon, Markus. "Hämodynamische Untersuchungen an Typ-2-diabetischen Ratten Einfluss des ACE-Inhibitors Ramipril /." [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=971238464.

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Agosto-Plazanet, Elizabeth. "Effets persistants d'une exposition au froid ou d'une inhibition de l'enzyme de conversion sur le métabolisme glucidique insulino-dépendant." Lyon 1, 1998. http://www.theses.fr/1998LYO1T001.

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Demolis, Pierre. "Application du doppler transcranien aux études de pharmacologie clinique." Paris 11, 1994. http://www.theses.fr/1994PA11T015.

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Books on the topic "Ramiprilat"

1

Leeker, Markus. Einfluss des ACE-Hemmers Ramipril auf die Resorption von humanem Serumalbumin aus dem Dünndarm der Ratte. 2003.

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Klein-Galczinsky, Christoph. Doppelblinde randomisierte prospektive Pilotstudie zur Überprüfung der Wirkung des ACE-Hemmers Ramipril auf die Infarktgrösse bei akutem Myokardinfarkt. 1998.

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Rossius, Birthe. Die Beeinflussung von Morphologie und Hämodynamik des Herzens nach Myokardinfarkt durch Therapie mit dem ACE-Hemmer Ramipril im Tierversuch. 1999.

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Rademacher, Markus. Wirkung des Calciumantagonisten Felodipin, des Konversionsenzymhemmers Ramipril sowie von Vasopressin auf Endothelin, hormonelle Regulationsmechanismen und Nierenfunktion bei gesunden Freiwilligen. 1995.

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Adler, Klaudia Ruth Krimhilde. Einfluss von Atorvastatin und Ramipril auf die Anzahl und Funktion von endothelialen Progenitorzellen bei Patienten mit stabiler kornarer Herzerkrankung. 2006.

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Main, Universität Frankfurt am, ed. Langzeitbehandlung herzinsuffizienter Patienten (NYHA III/IV) mit dem Konversionshemmer "Ramipril" (Delix®) oder Placebo unter Beibehaltung der konventionellen Therapie. 1995.

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Diehl, Stefanie. Effekt einer langzeitbehandlung mit dem ace-hemmer ramipril und dem AT₁-rezeptorantagonisten losartan auf die cyclin-expression in myokardialen kapillaren. 2000.

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Renken, Felix. Humorale Faktoren und Baroreflexfunktion bei Patienten mit diabetischer Neuropathie unter Therapie mit dem ACE-Hemmer Ramipril und dem [alpha]₁-Blocker Doxazosin. 2001.

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Krämer, Dietmar Gebhard. Reduktion der Proteinurie bei chronischer Glomerulonephritis durch den vasodilatierenden Betablocker Celiprolol im Vergleich mit dem ACE-Hemmer Ramipril und dem Betablocker Atenolol. 1998.

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Kröhn, Thorsten. Wirkung des Angiotensin II-AT1-Rezeptorantagonisten Losartan auf die Nierenfunktion von mit PEEP beatmeten, wachen Hunden: Vergleich mit dem ACE-Hemmer Ramipril. 1996.

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Book chapters on the topic "Ramiprilat"

1

de Groot, Anton C. "Ramipril." In Monographs In Contact Allergy. CRC Press, 2022. http://dx.doi.org/10.1201/9781003158004-432.

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Becker, B. F., M. Heier, and E. Gerlach. "Experimental Evidence for Cardioprotection Afforded by Ramipril, an Inhibitor of Angiotensin Converting Enzyme." In New Concepts in Viral Heart Disease. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73610-0_44.

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Saunders, John. "Inhibitors of angiotensin converting enzyme as effective antihypertensive agents." In Top Drugs. Oxford University Press, 2000. http://dx.doi.org/10.1093/hesc/9780198501008.003.0001.

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This chapter discusses the role of angiotensin converting enzyme (ACE) in reducing the blood pressure of hypertensive patients. The ACE is vital in the cascade of proteolytic reactions which ultimately control the levels of angiotensin II, a potent vasoconstrictor. The chapter provides a brief background of the treatment of hypertension in the 1970s, highlighting the impact of the discovery of captopril, a landmark in rational drug design. It explains the synthesis of captopril, as well as the syntheses of enalaprilat and lisinopril. The chapter also discusses the synthesis of quinapril, which

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"Ramipril." In Meyler's Side Effects of Drugs: The International Encyclopedia of Adverse Drug Reactions and Interactions. Elsevier, 2006. http://dx.doi.org/10.1016/b0-44-451005-2/00425-3.

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Papich, Mark G. "Ramipril." In Saunders Handbook of Veterinary Drugs. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-24485-5.00499-x.

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"Ramipril." In Meyler's Side Effects of Drugs. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-444-53717-1.01390-1.

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"Ramipril." In Drugs Handbook 2012–2013. Bloomsbury Academic, 2011. http://dx.doi.org/10.5040/9781350363595.art-1486.

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Davis, Stephen. "Ramipril." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.62512-7.

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"Ramipril." In Checkliste Arzneimittel A–Z, edited by Detlev Schneider and Frank Richling. Georg Thieme Verlag, 2013. http://dx.doi.org/10.1055/b-0034-82719.

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Papich, Mark G. "Ramipril." In Papich Handbook of Veterinary Drugs. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-323-70957-6.00470-2.

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Conference papers on the topic "Ramiprilat"

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Her Oktaviono, Yudi, Ferry Sandra, Achmad Lefi, and Christian Pramudita Budianto. "Secretome And Ramiprilat Effects On Endothelial Progenitor Cells Proliferation In Chronic Coronary Syndrome Patient." In ICBBE '20: 2020 7th International Conference on Biomedical and Bioinformatics Engineering. ACM, 2020. http://dx.doi.org/10.1145/3444884.3444910.

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Her Oktaviono, Yudi, Ilma Alfia Isaridha, Ferry Sandra, Achmad Lefi, and Agus Subagjo. "Ramiprilat Effects on Endothelial Progenitor Cells Migration is Increased by Human Umbilical Cord Blood-Mesenchymal Stem Cells derived Secretome." In ICBBE '20: 2020 7th International Conference on Biomedical and Bioinformatics Engineering. ACM, 2020. http://dx.doi.org/10.1145/3444884.3444914.

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Maria Martins Lima, Ylana, Paulo Roberto da Silva Ribeiro, Luis Henrique Silva Queiroz, and Raychimam Douglas Santana Bezerra. "DISPERSÃO SÓLIDA AMORFA DO ANTI-HIPERTENSIVO RAMIPRIL." In 2nd (ICAIC) International Conference for Academia and Industry Co-operation & 2nd (IMMSEM) International Meeting in Materials Science and Engineering of Maranhão. Even3, 2021. http://dx.doi.org/10.29327/2ndicaic2ndimmsem2020.421893.

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Plümacher, S., T. Paul, and M. Sigler. "Expectoration of Bronchial Casts Caused by Ramipril Treatment." In 51st Annual Meeting German Society for Pediatric Cardiology. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1679097.

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Maria Martins Lima, Ylana, Paulo Roberto da Silva Ribeiro, Raychimam Douglas S. Bezerra, and Luis Henrique Silva Queiroz. "CARACTERIZAÇÃO ESPECTROSCÓPICA DOS CO-AMORFOS DO ANTIHIPERTENSIVO RAMIPRIL." In 3rd (ICAIC) International Conference for Academia and Industry Co-operation & 3rd (IMMSEM) International Meeting in Materials Science and Engineering of Maranhão. Even3, 2022. http://dx.doi.org/10.29327/3rd-icaica-3rd-imnsem-267048.576224.

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De Sousa Silva, Wanderson, Ylana Maria Martins Lima, and Paulo Roberto da Silva Ribeiro. "OBTENÇÃO E CARACTERIZAÇÃO DE COAMORFOS DE RAMIPRIL COM ÁCIDO P-AMINOBENZOIC." In 3rd (ICAIC) International Conference for Academia and Industry Co-operation & 3rd (IMMSEM) International Meeting in Materials Science and Engineering of Maranhão. Even3, 2022. http://dx.doi.org/10.29327/3rd-icaica-3rd-imnsem-267048.576866.

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Dodia, N., A. L. Blackman, and M. S. Heavner. "Ramipril-Associated Liver Injury: A Case Report and Brief Review of the Literature." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3544.

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Logoyda, Liliya, and Kateryna Typlynska. "Development of the HPLC method for the determination of related substances in ramipril tablets." In MOL2NET'22, Conference on Molecular, Biomedical & Computational Sciences and Engineering, 8th ed. - MOL2NET: FROM MOLECULES TO NETWORKS. MDPI, 2022. http://dx.doi.org/10.3390/mol2net-08-13919.

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Ansbro, E., S. Masri, D. Prieto-Merino, et al. "Fixed dose combination drugs for cardiovascular disease in Lebanon." In MSF Scientific Days International 2022. MSF-USA, 2022. http://dx.doi.org/10.57740/8697-vn33.

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INTRODUCTION Cardiovascular disease (CVD) is the leading cause of death and disability globally, including in humanitarian contexts. Fixed-dose combination (FDC) drugs are cost-effective for primary and secondary prevention of CVD. From 2012 until the end of 2020, MSF provided care for CVD patients from Syrian refugee and host populations in primary care clinics in Tripoli, north Lebanon. In this implementation study, we assessed whether FDC use is linked with adherence to CVD medications and treatment simplification in a humanitarian setting. METHODS Our prospective, before-and-after cohort s

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Romanel Vinco, Lara, Julia Duarte de Souza Silva, Barbara Citelis Silva Vargas, Bianca Moreira Belido, Andressa Morais Mota Freitas, and Márcia Valéria Azevedo Gomes de Carvalho. "Síndrome nefrótica por lesão mínima em adulto diabético: um relato de caso." In Semana Científica da Faculdade de Medicina de Campos. Faculdade de Medicina de Campos, 2023. http://dx.doi.org/10.29184/anaisscfmc.v22023p47.

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Introdução: A síndrome nefrótica (SN) é uma glomerulopatia que acomete adultos e crianças, sendo causada por doenças primárias renais ou por patologias secundárias, como a diabetes mellitus tipo 2. As causas primárias são as mais prevalentes, tendo as alterações glomerulares mínimas (AGM) como as mais comuns de ocorrerem na infância, sendo a maioria dos casos antes dos cinco anos de idade. É uma patologia que cursa com proteinúria maciça, com excreção urinária maior que 3,5g/dia, e é caracterizada pela hipoproteinemia, edema e hiperlipidemia. Ademais, a SN quando ocorre em adultos diabéticos,

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