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Relevant bibliographies by topics / Ras Genes

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Academic literature on the topic 'Ras Genes'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 January 2023

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Journal articles on the topic "Ras Genes"

1

Barbacid, M. "ras Genes." Annual Review of Biochemistry 56, no. 1 (1987): 779–827. http://dx.doi.org/10.1146/annurev.bi.56.070187.004023.

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Tamanoi, Fuyuhiko. "Yeast RAS genes." Biochimica et Biophysica Acta (BBA) - Reviews on Cancer 948, no. 1 (1988): 1–15. http://dx.doi.org/10.1016/0304-419x(88)90002-9.

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Turner, Helen, та Doreen A. Cantrell. "Distinct Ras Effector Pathways Are Involved in FcεR1 Regulation of the Transcriptional Activity of Elk-1 and NFAT in Mast Cells". Journal of Experimental Medicine 185, № 1 (1997): 43–54. http://dx.doi.org/10.1084/jem.185.1.43.

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Activation of Ras GTPases is a conserved feature of antigen receptor signaling, including FcεR1 activation of mast cells. Antigenic cross-linking of the FcεR1 on mast cells results in secretion of allergic mediators and induction of immediate early and cytokine genes. Here we examine the role of Ras in coupling the FcεR1 to transcriptional regulation. The transcription factors Elk-1, an immediate early gene regulator and the nuclear factor of activated T cells (NFAT), in the context of the IL-4 gene, are identified as Ras targets in mast cells. Ras mediates diverse effects via its diverse effe

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Carney, Ellen F. "Targeting multiple RAS genes." Nature Reviews Nephrology 10, no. 9 (2014): 482. http://dx.doi.org/10.1038/nrneph.2014.135.

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Takács, Tamás, Gyöngyi Kudlik, Anita Kurilla, Bálint Szeder, László Buday, and Virag Vas. "The effects of mutant Ras proteins on the cell signalome." Cancer and Metastasis Reviews 39, no. 4 (2020): 1051–65. http://dx.doi.org/10.1007/s10555-020-09912-8.

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AbstractThe genetic alterations in cancer cells are tightly linked to signaling pathway dysregulation. Ras is a key molecule that controls several tumorigenesis-related processes, and mutations in RAS genes often lead to unbiased intensification of signaling networks that fuel cancer progression. In this article, we review recent studies that describe mutant Ras-regulated signaling routes and their cross-talk. In addition to the two main Ras-driven signaling pathways, i.e., the RAF/MEK/ERK and PI3K/AKT/mTOR pathways, we have also collected emerging data showing the importance of Ras in other s

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Rajasekharan, Satish, and Thiagarajan Raman. "Ras and Ras mutations in cancer." Open Life Sciences 8, no. 7 (2013): 609–24. http://dx.doi.org/10.2478/s11535-013-0158-5.

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AbstractRas genes are pre-eminent genes that are frequently linked with cancer biology. The functional loss of ras protein caused by various point mutations within the gene, is established as a prognostic factor for the genesis of a constitutively active Ras-MAPK pathway leading to cancer. Ras signaling circuit follows a complex pathway, which connects many signaling molecules and cells. Several strategies have come up for targeting mutant ras proteins for cancer therapy, however, the clinical benefits remain insignificant. Targeting the Ras-MAPK pathway is extremely complicated due its intric

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Missero, Caterina, Maria Teresa Pirro, and Roberto Di Lauro. "Multiple Ras Downstream Pathways Mediate Functional Repression of the Homeobox Gene Product TTF-1." Molecular and Cellular Biology 20, no. 8 (2000): 2783–93. http://dx.doi.org/10.1128/mcb.20.8.2783-2793.2000.

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ABSTRACT Expression of oncogenic Ras in thyroid cells results in loss of expression of several thyroid-specific genes and inactivation of TTF-1, a homeodomain-containing transcription factor required for normal development of the thyroid gland. In an effort to understand how signal transduction pathways downstream of Ras may be involved in suppression of the differentiated phenotype, we have tested mutants of the Ras effector region for their ability to affect TTF-1 transcriptional activity in a transient-transfection assay. We find that V12S35 Ras, a mutant known to interact specifically with

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Tansey, M. G., G. C. Chu, and J. P. Merlie. "ARIA/HRG regulates AChR epsilon subunit gene expression at the neuromuscular synapse via activation of phosphatidylinositol 3-kinase and Ras/MAPK pathway." Journal of Cell Biology 134, no. 2 (1996): 465–76. http://dx.doi.org/10.1083/jcb.134.2.465.

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AChR-inducing activity (ARIA)/heregulin, a ligand for erbB receptor tyrosine kinases (RTKs), is likely to be one nerve-supplied signal that induces expression of acetylcholine receptor (AChR) genes at the developing neuromuscular junction. Since some RTKs act through Ras and phosphatidylinositol 3-kinase (PI3K), we investigated the role of these pathways in ARIA signaling. Expression of activated Ras or Raf mimicked ARIA-induction of AChR epsilon subunit genes in muscle cells; whereas dominant negative Ras or Raf blocked the effect of ARIA. ARIA rapidly activated erk1 and erk2 and inhibition o

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Waldmann, V., and H. M. Rabes. "What's New in Ras Genes?" Pathology - Research and Practice 192, no. 9 (1996): 883–91. http://dx.doi.org/10.1016/s0344-0338(96)80067-7.

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Robbins, Stephen M., Meenal Khosla, Richard Thiery, Gerald Weeks, and George B. Spiegelman. "Ras-related genes inDictyostelium discoideum." Developmental Genetics 12, no. 1-2 (1991): 147–53. http://dx.doi.org/10.1002/dvg.1020120123.

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Dissertations / Theses on the topic "Ras Genes"

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Sumpter, Katherine Anne. "The role of RAS genes in lymphoma." Thesis, Institute of Cancer Research (University Of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.407119.

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Culine, Stéphane. "Genes de la superfamille ras et tumeurs humaines : etude de l'expression des genes rap, rab2 et rab3a." Paris 7, 1993. http://www.theses.fr/1993PA077037.

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Les proto-oncogenes h-ras, k-ras et n-ras appartiennent a une famille multigenique, dont les membres codent pour des proteines de 21 a 27 kilodaltons, caracterisees par leur capacite de se lier aux nucleotides guanyliques gdp et gtp. Sous l'influence d'un signal activateur d'amont, les proteines de la superfamille ras passent d'un etat inactif, lie au gd, a une forme activee, liee au gtp, interagissant avec un effecteur d'aval. Le passage, entre chaque etat de liaison au gdp ou au gtp, est sous la dependance de proteines dont le role regulateur est d'assurer un controle etroit de la balance gd

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Robbins, Stephen Mark. "Characterization of a ras and a ras-related gene and their developmental expression in the cellular slime mould Dictyostelium discoideum." Thesis, University of British Columbia, 1991. http://hdl.handle.net/2429/31318.

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Although it was previously reported that Dictyostelium discoideum possessed a single ras gene (Ddras) that was maximally expressed during the pseudoplasmodial stage of development, a second ras gene (DdrasG), has been isolated and characterized. It encodes a protein that is similar to the protein encoded by Ddras and the human ras proteins. However, in contrast to Ddras, the DdrasG gene was only expressed during growth and early development. The two ras proteins may fulfill different functions: the DdrasG protein having a role during cell growth and the Ddras protein having a role in signal tr

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CHARDIN, PIERRE. "Structure de la famille des genes ras chez l'homme." Paris 7, 1989. http://www.theses.fr/1989PA077029.

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Gray, Virginia Elaine. "The developmental expression of the Dictyostelium discoideum ras gene and preliminary detection of a second ras-homologous sequence in its genome." Thesis, University of British Columbia, 1987. http://hdl.handle.net/2429/26413.

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The expression of a mammalian ras gene analog was previously found by Reymond et al. to be developmentally regulated in Dictyostelium discoideum using Northern analysis of strain AX-3 RNA (1984, Cell 39;141) and by Pawson et al. using specific immunoprecipitation of in vivo synthesized proteins from strain V12M2 (1985, Mol. Cell Biol. 5;33). Due to differences in the results of the two studies, it was decided to further examine ras expression by applying both protein and RNA techniques to a single strain of D.discoideum, V12M2. RNA samples from strain V12M2 cells at different stages of develop

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Lehmann, Kerstin Elisabeth. "Regulation of epithelial cell transformation and survival by Raf activation." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248242.

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Sjögren, Anna-Karin. "The importance of isoprenylation and Nf1 deficiency in K-RAS-induced cancer /." Göteborg : The Wallenberg Laboratory, Department of Molecular and Clinical Medicine, Institute of Medicine at Sahlgrenska Academy, University of Gothenburg, 2009. http://hdl.handle.net/2077/21218.

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Lo, Chi-chuen Evans. "A study of BRAF and RAS genes in papillary thyroid carcinoma." Click to view the E-thesis via HKUTO, 2004. http://sunzi.lib.hku.hk/hkuto/record/B31972007.

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Lo, Chi-chuen Evans, and 盧致泉. "A study of BRAF and RAS genes in papillary thyroid carcinoma." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31972007.

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Mundt, Thomas. "Vergleichende Mutationsanalyse des k-ras-Genes in zytologischen Untersuchungsmaterialien bei nichtkleinzelligen Lungenkarzinomen." Doctoral thesis, Universitätsbibliothek Leipzig, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-91743.

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Die vorliegende Arbeit beschäftigte sich mit der Frage, inwieweit zytologische Untersuchungsmaterialien von Patienten mit nichtkleinzelligen Lungenkarzinomen geeignet sind, zur molekularbiologischen Analyse möglicher Mutationen des k-ras-Genes verwendet zu werden. Dafür wurden vier spezifische Polymerasekettenreaktionen mit anschließenden Restriktionsfragmentlängenpolymorphismus-Analysen kreiert und optimiert. In der Folge wurden die k-ras-Mutationen in den verschiedenen Codons nach einer Agarose-Gelelektrophorese unter der UV-Kamera detektiert. Es wurden vergleichende Untersuchungen mit Mater

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Books on the topic "Ras Genes"

1

NATO Advanced Research Workshop on Ras Oncogenes (1988 Athens, Greece). Ras oncogenes. Plenum Press, 1989.

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NATO Advanced Research Workshop on the Super-Family of Ras-Related Genes (1991 Hagia Pelagia, Greece). The superfamily of ras-related genes. Plenum Press, 1991.

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Spandidos, Demetrios A., ed. The Superfamily of ras-Related Genes. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-6018-6.

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Ahmed, Tariq. Search for RAS-like genes in the C.albicans genome. UMIST, 1995.

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Carlos, Lacal Juan, and McCormick Frank 1950-, eds. The Ras superfamily of GTPases. CRC Press, 1993.

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Media, Springer Science+Business, ed. Ras signaling: Methods and protocols. Humana Press, 2014.

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Verkoczy, Laurent Karl. Regulation studies of the human recombination activating genes (RAG-1 and RAG-2). National Library of Canada, 1995.

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Lazerine, Cameron. Rap-Up. Grand Central Publishing, 2008.

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Lang, Holly. The Notorious B.I.G.: A biography. Greenwood Press, 2007.

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Adamović, Tatjana. Identifiction and characterization of cancer genes in hormone-dependent tumors: Molecular genetic analysis in rat models of endometrial and mammary cancer. Dept. of Cell and Molecular Biology-Genetics, Lundberg Insitutute, Göteborgs University, 2006.

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Book chapters on the topic "Ras Genes"

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Wong, Yung H., Angel Pellicer, and Wen Liu. "RAS Genes." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_4951-2.

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Wong, Yung H., Angel Pellicer, and Wen Liu. "RAS Genes." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-46875-3_4951.

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Pellicer, Angel. "RAS Genes." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_4951.

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Hancock, J. F., K. Cadwallader, A. I. Magee, et al. "Prenylation of ras and ras-Related Proteins." In The Superfamily of ras-Related Genes. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-6018-6_2.

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Hayes, Tikvah K., Jeran K. Stratford, Andrea Wang-Gillam, and Channing J. Der. "RAS Genes and Cancer." In Ras Superfamily Small G Proteins: Biology and Mechanisms 1. Springer Vienna, 2014. http://dx.doi.org/10.1007/978-3-7091-1806-1_7.

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Bos, J. L. "Mutational Activation of Human ras Genes." In ras Oncogenes. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-1235-3_3.

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Cichutek, K., and P. H. Duesberg. "Conversion of ras Genes to Cancer Genes." In Haematology and Blood Transfusion / Hämatologie und Bluttransfusion. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72624-8_102.

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Fusco, A., M. T. Berlingieri, M. Grieco, M. Santoro, and G. Vecchio. "The Cooperation between Viral ras Genes and Different Immortalizing Genes Induces the Transformation of Rat Thyroid Epithelial Cells." In ras Oncogenes. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-1235-3_41.

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Thorn, J. T., A. V. Todd, D. Warrilow, F. Watt, P. L. Molloy, and H. J. Iland. "Characterization of the Human N-ras Promoter Region." In The Superfamily of ras-Related Genes. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-6018-6_11.

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Nur-E-Kamal, M. S. A., and H. Maruta. "ras and rap1 GTPases Mutated at Position 64." In The Superfamily of ras-Related Genes. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-6018-6_12.

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Conference papers on the topic "Ras Genes"

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Киреева, Виктория, Viktoriya Kireeva, Г. Лифшиц, et al. "Advantages of a personalized approach to the prevention and treatment of cardiovascular diseases in the staff of the INC Of the SBRAS." In Topical issues of translational medicine: a collection of articles dedicated to the 5th anniversary of the day The creation of a department for biomedical research and technology of the Irkutsk Scientific Center Siberian Branch of RAS. INFRA-M Academic Publishing LLC., 2017. http://dx.doi.org/10.12737/conferencearticle_58be81ec9ed47.

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Purpose of the study. To test the functional associations of polymorphic variants of genes in the regulation of blood pressure and vascular tone in employees of the ISC SB RAS. Materials and methods. The study involved patients, employees of the ISC SB RAS, being under care of the outpatient clinic of the Hospital of the ISC SB RAS. During routine laboratory testing the patients were taken 2 ml of blood for genetic analysis and further molecular genetic study on “Hypertension”, “Endothelial dysfunction”, “Pharmacogenetics”, “Inflammatory response” panels. Results. In the analysis of 12 genes c

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Bahnassy, Abeer A., Salem E. Salem, Nehal Hussein, et al. "Abstract 4174: Molecular changes in the EGFR pathway and RAS/RAF genes identify subtypes of metastatic and nonmetastatic colorectal cancer associated with different outcomes." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4174.

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Singh, Anurag, Michael F. Sweeney, Patricia Greninger, and Jeffrey E. Settleman. "Abstract 5754: Identification of pharmacologically tractable genes that are associated with oncogenic K-Ras dependency in colorectal cancers." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5754.

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Ikeda, Yuji, Katsutoshi Oda, Shunsuke Nakagawa, et al. "Abstract 2957: Mutations of Cyclin D1 / beta-catenin are coexistent with mutations in Ras-PI3K pathway genes in endometrial cancer." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2957.

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Ohba, Taro, Kenji Sugio, Gouji Toyokawa, et al. "Abstract A155: Mutations of the EGFR, K-ras, and EML-4ALK genes in resected stage I lung adenocarcinoma and their clinical significance." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 12-16, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1535-7163.targ-11-a155.

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Arias-Romero, Luis E., Olga Villamar-Cruz, and Jonathan Chernoff. "Abstract A02: Pak1 kinase inhibition sensitizes 11q13 amplified breast cancer cells to platinum based chemotherapy via downregulation of Fanconi anemia genes." In Abstracts: AACR Special Conference on RAS Oncogenes: From Biology to Therapy; February 24-27, 2014; Lake Buena Vista, FL. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1557-3125.rasonc14-a02.

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Arima, Yoshimi, Kenjiro Kosaki, Chikako Hirose, and Hideyuki Saya. "Abstract 2730: Additional mutations in genes relevant to the Ras signaling pathway promote the malignant characteristics of NF1-associated malignant peripheral nerve sheath tumor (NF1-MPNST) cells." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2730.

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Gay, LM, JA Elvin, J.-A. Vergilio, et al. "Abstract P1-05-07: Comprehensive genomic profiling of clinically malignant phyllodes tumors of the breast reveals frequent mutation ofNF1and other genes associated with PI3K and RAS pathway activation." In Abstracts: 2016 San Antonio Breast Cancer Symposium; December 6-10, 2016; San Antonio, Texas. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.sabcs16-p1-05-07.

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Gyorffy, Balazs, Iwona Stelniec-Klotz, Christian Sigler, Attila Szijarto, Yu Qian, and Reinhold Schäfer. "Abstract PR06: Impact of RAL signal transduction on genetic program and growth control in KRAS- and BRAF-mutated colorectal cells and prognostic potential of pathway-responsive genes in cancer patients." In Abstracts: AACR Special Conference on RAS Oncogenes: From Biology to Therapy; February 24-27, 2014; Lake Buena Vista, FL. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1557-3125.rasonc14-pr06.

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Rodrigues, Antonio Rony da S. P., and Edinalda Maria Cavalcante. "ESTUDO GENÉTICO DO CÂNCER DE TIREOIDE – UMA REVISÃO." In I SIMPÓSIO MARANHENSE DE GENÉTICA E GENÔMICA EM SAÚDE. Doity - Plataforma de Eventos, 2022. http://dx.doi.org/10.55664/simaggens2022.005.

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INTRODUÇÃO: O câncer de tireoide é um dos agravos mais comuns ao sistema endócrino, com o maior aumento anual de incidência em diferentes países, principalmente devido à melhoria das tecnologias de diagnóstico, sendo mais proeminente em regiões com acesso aos cuidados de saúde amplamente disponíveis. Alguns eventos moleculares são descritos tanto na carciogênese da tireoide quanto na evolução do tumor glandular. Dados do Atlas do câncer no Genoma Humano dividiu os carcinomas papilíferos de tireoide nas categorias BRAF e RAS, com base nos resultados do exoma do sequenciamento de DNA, RNA e perf

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Reports on the topic "Ras Genes"

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Friedman, Haya, Chris Watkins, Susan Lurie, and Susheng Gan. Dark-induced Reactive Oxygen Species Accumulation and Inhibition by Gibberellins: Towards Inhibition of Postharvest Senescence. United States Department of Agriculture, 2009. http://dx.doi.org/10.32747/2009.7613883.bard.

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Dark-induced senescence could pose a major problem in export of various crops including cuttings. The assumption of this work was that ROS which is increased at a specific organelle can serve as a signal for activation of cell senescence program. Hormones which reduce senescence in several crops like gibberellic acid (GA) and possibly cytokinin (CK) may reduce senescence by inhibiting this signal. In this study we worked on Pelargonium cuttings as well as Arabidopsis rosette. In Pelargonium the increase in ROS occurred concomitantly with increase in two SAGs, and the increase persisted in isol

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Horwitz, Benjamin, and Barbara Gillian Turgeon. Secondary Metabolites, Stress, and Signaling: Roles and Regulation of Peptides Produced by Non-ribosomal Peptide Synthetases. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7696522.bard.

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Fungal pathogens of plants produce a diverse array of small molecules. Often referred to as secondary metabolites because they were thought to be dispensable for basic functions, they may indeed have central roles as signals for the fungal cell, and in interactions with the host. We have identified more than a dozen genes encoding nonribosomal peptide synthetases (NPS) in Cochliobolusheterostrophus, the agent of southern corn leaf blight. The aim of this project was to identify roles of these genes in stress responses and signaling. The first objective was to test a complete collection of C. h

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Arazi, Tzahi, Vivian Irish, and Asaph Aharoni. Micro RNA Targeted Transcription Factors for Fruit Quality Improvement. United States Department of Agriculture, 2008. http://dx.doi.org/10.32747/2008.7592651.bard.

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Fruits are unique to flowering plants and represent an important component of human and animal diets. Development and maturation of tomato fruit is a well-programmed process, and yet, only a limited number of factors involved in its regulation have been characterized. Micro-RNAs (miRNAs) are small, endogenous RNAs that regulate gene expression in animals and plants. Plant miRNAs have a vital role in the generation of plant forms through post-transcriptional regulation of the accumulation of developmental regulators, especially transcription factors. Recently, we and others have demonstrated th

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Horwitz, Benjamin A., and Barbara Gillian Turgeon. Fungal Iron Acquisition, Oxidative Stress and Virulence in the Cochliobolus-maize Interaction. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7709885.bard.

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Our project focused on genes for high affinity iron acquisition in Cochliobolus heterostrophus, a necrotrophic pathogen of maize, and their intertwined relationship to oxidative stress status and virulence of the fungus on the host. An intriguing question was why mutants lacking the nonribosomal peptide synthetase (NRPS) gene (NPS6) responsible for synthesis of the extracellular siderophore, coprogen, are sensitive to oxidative stress. Our overall objective was to understand the mechanistic connection between iron stress and oxidative stress as related to virulence of a plant pathogen to its h

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Delmer, Deborah P., Douglas Johnson, and Alex Levine. The Role of Small Signal Transducing Gtpases in the Regulation of Cell Wall Deposition Patterns in Plants. United States Department of Agriculture, 1995. http://dx.doi.org/10.32747/1995.7570571.bard.

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The combined research of the groups of Delmer, Levine and Johnson has led to a number of interesting findings with respect to the function of the small GTPase Rac in plants and also opened up new leads for future research. The results have shown: 1) The Rac13 protein undergoes geranylgeranlyation and is also translocated to the plasma membrane as found for Rac in mammals; 2) When cotton Rac13 is highly- expressed in yeast, it leads to an aberrant phenotype reminiscent of mutants impaired in actin function, supporting a role for Rac13 in cytoskeletal organization; 3) From our searches, there is

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Gould, Michael, and V. A. Ford. Mapping Mammary Carcinoma Suppressor Genes in the Laboratory Rat. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada298730.

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Horwitz, Benjamin, and Nicole M. Donofrio. Identifying unique and overlapping roles of reactive oxygen species in rice blast and Southern corn leaf blight. United States Department of Agriculture, 2017. http://dx.doi.org/10.32747/2017.7604290.bard.

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Plants and their fungal pathogens both produce reactive oxygen species (ROS). CytotoxicROS act both as stressors and signals in the plant-fungal interaction. In biotrophs, a compatible interaction generates little ROS, but is followed by disease. An incompatible interaction results in a strong oxidative burst by the host, limiting infection. Necrotrophs, in contrast, thrive on dead and dying cells in an oxidant-rich local environment. Rice blast, Magnaportheoryzae, a hemibiotroph, occurs worldwide on rice and related hosts and can decimate enough rice each year to feed sixty million people. Co

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Mawassi, Munir, Adib Rowhani, Deborah A. Golino, Avichai Perl, and Edna Tanne. Rugose Wood Disease of Grapevine, Etiology and Virus Resistance in Transgenic Vines. United States Department of Agriculture, 2003. http://dx.doi.org/10.32747/2003.7586477.bard.

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Rugose wood is a complex disease of grapevines, which occurs in all growing areas. The disease is spread in the field by vector transmission (mealybugs). At least five elongated-phloem- limited viruses are implicated in the various rugose wood disorders. The most fully characterized of these are Grapevine virus A (GV A) and GVB, members of a newly established genus, the vitivirus. GVC, a putative vitivirus, is much less well characterized than GV A or GVB. The information regarding the role of GVC in the etiology and epidemiology of rugose wood is fragmentary and no sequence data for GVC are a

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Droby, Samir, Michael Wisniewski, Ron Porat, and Dumitru Macarisin. Role of Reactive Oxygen Species (ROS) in Tritrophic Interactions in Postharvest Biocontrol Systems. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7594390.bard.

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To elucidate the role of ROS in the tri-trophic interactions in postharvest biocontrol systems a detailed molecular and biochemical investigation was undertaken. The application of the yeast biocontrol agent Metschnikowia fructicola, microarray analysis was performed on grapefruit surface wounds using an Affymetrix Citrus GeneChip. the data indicated that 1007 putative unigenes showed significant expression changes following wounding and yeast application relative to wounded controls. The expression of the genes encoding Respiratory burst oxidase (Rbo), mitogen-activated protein kinase (MAPK)

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McElwain, Terry, Eugene Pipano, Guy Palmer, Varda Shkap, Stephen Hines, and Douglas Jasmer. Protection of Cattle Against Babesiosis: Immunization with Recombinant DNA Derived Apical Complex Antigens of Babesia bovis. United States Department of Agriculture, 1995. http://dx.doi.org/10.32747/1995.7612835.bard.

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Bovine babesiosis caused by Babesia bovis continues to be a significant deterrent to global livestock production. Current control methods have both biological and technical drawbacks that have stimulated research on improved methods of vaccination. This BARD project has focused on characterization of candidate Babesia bovis vaccine antigens located in the apical complex, a unique group of subcellular organelles - including rhoptries, micronemes, and spherical bodies - involved in the invation of erythrocytes. Spherical bodies and rhoptries were partially purified and their contents characteriz

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