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Relevant bibliographies by topics / Rat carotid body cells

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Academic literature on the topic 'Rat carotid body cells'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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Journal articles on the topic "Rat carotid body cells"

1

Tse, Amy, Lei Yan, Andy K. Lee, and Frederick W. Tse. "Autocrine and paracrine actions of ATP in rat carotid body." Canadian Journal of Physiology and Pharmacology 90, no. 6 (2012): 705–11. http://dx.doi.org/10.1139/y2012-054.

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Carotid bodies are peripheral chemoreceptors that detect lowering of arterial blood O2 level. The carotid body comprises clusters of glomus (type I) cells surrounded by glial-like sustentacular (type II) cells. Hypoxia triggers depolarization and cytosolic [Ca2+] ([Ca2+]i) elevation in glomus cells, resulting in the release of multiple transmitters, including ATP. While ATP has been shown to be an important excitatory transmitter in the stimulation of carotid sinus nerve, there is considerable evidence that ATP exerts autocrine and paracrine actions in carotid body. ATP acting via P2Y1 recepto

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Yamamoto, Y., and K. Taniguchi. "Expression of Tandem P Domain K+ Channel, TREK-1, in the Rat Carotid Body." Journal of Histochemistry & Cytochemistry 54, no. 4 (2006): 467–72. http://dx.doi.org/10.1369/jhc.5a6755.2005.

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TREK-1 is one of the important potassium channels for regulating membrane excitability. To examine the distribution of TREK-1 in the rat carotid body, we performed RT-PCR for mRNA expression and in situ hybridization and immunohistochemistry for tissue distribution of TREK-1. RT-PCR detected mRNA expression of TREK-1 in the carotid body. Furthermore, in situ hybridization revealed the localization of TREK-1 mRNA in the glomus cells. TREK-1 immunoreactivity was mainly distributed in the glomus cells and nerve fibers in the carotid body. TREK-1 may modulate potassium current of glomus cells and/

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Pallot, D. J., K. W. Al Neamy, and N. Blakeman. "Quantitative Studies of Rat Carotid Body Type I Cells." Cells Tissues Organs 126, no. 3 (1986): 187–92. http://dx.doi.org/10.1159/000146213.

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Martinez, A., L. Saldise, MJ Ramirez, et al. "Adrenomedullin expression and function in the rat carotid body." Journal of Endocrinology 176, no. 1 (2003): 95–102. http://dx.doi.org/10.1677/joe.0.1760095.

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Adrenomedullin (AM) immunoreactivity has been found in granules of the glomus (type I) cells of the carotid bodies in rats. The identity of these cells was ascertained by colocalization of immunoreactivities for AM and tyrosine hydroxylase in their cytoplasm. Exposure of freshly isolated carotid bodies to synthetic AM resulted in a concentration- and time-dependent degranulation of glomus cells as measured by dopamine (DA) release. DA release reached a zenith 30 min after exposure to AM (94.2% over untreated controls). At this time-point, the response to AM was similar to the one elicited by 5

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Makarenko, Vladislav V., Ying-Jie Peng, Guoxiang Yuan, et al. "CaV3.2 T-type Ca2+ channels in H2S-mediated hypoxic response of the carotid body." American Journal of Physiology-Cell Physiology 308, no. 2 (2015): C146—C154. http://dx.doi.org/10.1152/ajpcell.00141.2014.

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Arterial blood O2 levels are detected by specialized sensory organs called carotid bodies. Voltage-gated Ca2+ channels (VGCCs) are important for carotid body O2 sensing. Given that T-type VGCCs contribute to nociceptive sensation, we hypothesized that they participate in carotid body O2 sensing. The rat carotid body expresses high levels of mRNA encoding the α1H-subunit, and α1H protein is localized to glomus cells, the primary O2-sensing cells in the chemoreceptor tissue, suggesting that CaV3.2 is the major T-type VGCC isoform expressed in the carotid body. Mibefradil and TTA-A2, selective bl

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Di Giulio, C., P. G. Data, and S. Lahiri. "Chronic cobalt causes hypertrophy of glomus cells in the rat carotid body." American Journal of Physiology-Cell Physiology 261, no. 1 (1991): C102—C105. http://dx.doi.org/10.1152/ajpcell.1991.261.1.c102.

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We tested the hypothesis that chronic cobalt administration would induce carotid body cellular response along with polycythemia as found in chronic hypoxia if common oxygen-sensitive mechanisms were involved in the two instances. Morphometric studies were performed on carotid bodies in male rats that were chronically treated with cobalt chloride (0.17 mumol/kg, ip, daily for 6 wk) and in control rats that received blank saline injections. The rats were anesthetized, blood samples were collected for hematocrit, and the carotid bodies were surgically exposed and were perfused and superfused with

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7

Monti-Bloch, L., Vero´nica Abudara, and C. Eyzaguirre. "Electrical communication between glomus cells of the rat carotid body." Brain Research 622, no. 1-2 (1993): 119–31. http://dx.doi.org/10.1016/0006-8993(93)90810-a.

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Fung, Man-Lung, Siu-Yin Lam, Tung-Po Wong, Yung-Wui Tjong, and Po-Sing Leung. "Carotid Body AT4 Receptor Expression and its Upregulation in Chronic Hypoxia." Open Cardiovascular Medicine Journal 1, no. 1 (2007): 1–7. http://dx.doi.org/10.2174/1874192400701010001.

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Hypoxia regulates the local expression of angiotensin-generating system in the rat carotid body and the me-tabolite angiotensin IV (Ang IV) may be involved in the modulation of carotid body function. We tested the hypothesis that Ang IV-binding angiotensin AT4 receptors play a role in the adaptive change of the carotid body in hypoxia. The expression and localization of Ang IV-binding sites and AT4 receptors in the rat carotid bodies were studied with histochemistry. Specific fluorescein-labeled Ang IV binding sites and positive staining of AT4 immunoreactivity were mainly found in lobules in

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9

Otlyga, D. A., O. A. Junemann, E. G. Tsvetkova, K. R. Gorokhov, and S. V. Saveliev. "Immunohistochemical features of the human carotid body." CLINICAL AND EXPERIMENTAL MORPHOLOGY 9, no. 3 (2020): 61–67. http://dx.doi.org/10.31088/cem2020.9.3.61-67.

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Introduction. The carotid body is a chemoreceptor organ and the initial link of the reflex regulation of car-diovascular and respiratory systems. However, molecular genetic and immunohistochemical characteristics of the human carotid body remains underinvestigated. Although there are numerous studies of the second half of the 20th century devoted to the classical light-optical histology of the human organ, the immunohis-tochemical investigations are very few. The aim of our study was to clarify immunohistochemical features of the human carotid body in comparison with those of the most commonly

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Stea, A., and C. A. Nurse. "Chloride channels in cultured glomus cells of the rat carotid body." American Journal of Physiology-Cell Physiology 257, no. 2 (1989): C174—C181. http://dx.doi.org/10.1152/ajpcell.1989.257.2.c174.

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As part of our investigations on the chemosensory mechanisms in the rat carotid body, we are studying the physiology of the parenchymal glomus cells by the patch-clamp technique. Here we characterize a large-conductance chloride channel (approximately 296 pS) with random open and closed kinetics in inside-out patches of cultured glomus cells. The open-state probability (Po; mean = 0.61) was hardly affected by membrane potential (-50 to +50 mV) and cytoplasmic calcium (0-1 mM). Similarly, the channel did not appear to be regulated by cytoplasmic nucleotides (1 mM) or pH (6.5-8). Ion-substitutio

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Dissertations / Theses on the topic "Rat carotid body cells"

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O'Donnell, Jean. "Mechanism of excitation of carotid body chemoreceptor cells." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236119.

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Burlon, Drew C. "THE PRESYNAPTIC REGULATION OF ISOLATED NEONATAL RAT CAROTID BODY TYPE I CELLS BY HISTAMINE." Wright State University / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=wright1252943387.

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Ricker, Ellen M. "The inhibitory effects of opioids on voltage-gated calcium influx in neonatal rat carotid body type I cells." Wright State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=wright1424262410.

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Dunn, Eric J. "Effect of Somatostatin on Voltage-Gated CalciumInflux in Isolated Neonatal Rat Carotid Body Type I Cells." Wright State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=wright1432132454.

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Jackson, Adele. "Oxygen sensing, plasticity and catecholaminergic functions in cultured chromaffin cells of rat carotid body and adrenal medulla : modulation by chronic hypoxia and acetylcholine receptors /." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0018/NQ30096.pdf.

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Richmond, Patrick Henry. "The regulation of intracellular pH (pHi) and its role in chemoreception in the carotid body type-1 cell of the neonatal rat." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359506.

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Rakoczy, Ryan J. "Measuring the Effects of High-Fat Diet on Breathing and Oxygen-Sensitivity of the Carotid Body Type I Cell." Wright State University / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=wright1505728876488752.

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Arnold, Alexandre José Tavolari. "O papel do corpúsculo carotídeo na insuficiência cardíaca induzida pela doxorrubicina." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-05062018-121228/.

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A insuficiência cardíaca (IC) é o estágio final de diversas patologias cardíacas e apresenta alta morbimortalidade. Dentre as causas, estão os efeitos cardiotóxicos em pacientes tratados com doxorrubicina (Dox). A fisiopatologia da IC apresenta aumento da atividade barorreflexa e marcada hiperatividade simpática (HS), estado compensatório à redução do débito cardíaco. Porém, a HS prolongada culmina em alterações deletérias para o sistema cardiovascular (SC) com piora do quadro de sintomas. Atualmente muito se discute sobre o papel dos corpúsculos carotídeos (CC) na fisiopatologia da IC devido

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Shaw, Karen. "Stimulus-secretion coupling in the rat carotid body." Thesis, University of Leicester, 1989. http://hdl.handle.net/2381/35148.

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The effects of a variety of agents known to stimulate chemoafferent activity were employed to define in greater detail the stimulus-secretion coupling mechanism in the rat carotid body superfused in vitro. Hypoxia, carbachol and sodium cyanide were independently able to elicit amine release. Hypoxia-evoked release was calcium dependent and was reduced by nitrendipine suggesting the involvement of voltage-dependent calcium channels in the secretory response. The effect of carbachol on catecholamine release was abolished by atropine indicating the presence of muscarinic cholinergic receptors on

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Paulet, Julia. "MATURATION OF THE CAROTID BODY OXYGEN-SENSOR DURING RAT DEVELOPMENT." Wright State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=wright1337703804.

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Books on the topic "Rat carotid body cells"

1

D, Lambris John, Peers Chris, Cohen Irun R, et al., eds. Arterial Chemoreceptors: Arterial Chemoreceptors. Springer Netherlands, 2009.

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2

Constancio, Gonzalez, ed. The carotid body chemoreceptors. Landes Bioscience, 1997.

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3

Arterial Chemoreception Advances in Experimental Medicine and Biology. Springer, 2012.

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4

Phenylephrine-induced electrophysiological changes in cultured neonatal rat ventricular myocytes. 1995.

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Phenylephrine-induced electrophysiological changes in cultured neonatal rat ventricular myocytes. 1995.

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Phenylephrine-induced electrophysiological changes in cultured neonatal rat ventricular myocytes. 1995.

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7

Phenylephrine-induced electrophysiological changes in cultured neonatal rat ventricular myocytes. 1995.

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8

Studies on calcitonin in the carotid body chief cells during development and in SIDS: Immunohistochemical localization, quantitative analysis and detection of calcitonin mRNA by in situ hybridization. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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9

G, O'Regan R., and International Symposium on Arterial Chemoreceptors (12th : 1993 : Dublin, Ireland), eds. Arterial chemoreceptors: Cell to system. Plenum Press, 1994.

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Book chapters on the topic "Rat carotid body cells"

1

Powell, Frank L., Tatsumi Kusakabe, and Mark H. Ellisman. "Effects of Chronic Hypoxia on Rat Carotid Body and Toad Carotid Labyrinth Glomus Cells." In Advances in Experimental Medicine and Biology. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2572-1_68.

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Zhong, Huijun, and Colin Nurse. "Co-Cultures of Rat Petrosal Neurons and Carotid Body Type 1 Cells." In Frontiers in Arterial Chemoreception. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5891-0_27.

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Gauda, Estelle B., Reed Cooper, and Shereé M. Johnson. "Autonomic Ganglion Cells : Likely Source of Acetylcholine in the Rat Carotid Body." In Advances in Experimental Medicine and Biology. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9280-2_64.

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4

Böck, P. "Localization of Lysosomal Enzymes in Chromaffin Cells of the Rat Carotid Body." In Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_22.

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Murali, Sindhubarathi, Min Zhang, and Colin A. Nurse. "Paracrine Signaling in Glial-Like Type II Cells of the Rat Carotid Body." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-18440-1_5.

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Buckler, K. J. "Role of Potassium Channels in Hypoxic Chemoreception in Rat Carotid Body Type-I Cells." In Frontiers in Arterial Chemoreception. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5891-0_11.

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Vaughan-Jones, R. D., K. J. Buckler, C. Peers, and P. C. G. Nye. "Regulation of Intracellular pH in Type I Cells of the Neonatal Rat Carotid Body." In Advances in Experimental Medicine and Biology. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2966-8_35.

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Mokashi, A., A. Roy, C. Rozanov, P. Daudu, and S. Lahiri. "Effect of Barium on Rat Carotid Body Glomus Cell [Ca2+]i and Carotid Chemosensory Response." In Oxygen Sensing. Springer US, 2002. http://dx.doi.org/10.1007/0-306-46825-5_71.

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Peers, Chris, Christopher N. Wyatt, and Keith J. Buckler. "Actions of Nicotinic Agonists on Isolated Type I Cells of the Neonatal Rat Carotid Body." In Advances in Experimental Medicine and Biology. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2572-1_17.

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Wyatt, Christopher N., and Chris Peers. "Ca2+-Activated K+-Channels from Isolated Type I Carotid Body Cells of the Neonatal Rat." In Advances in Experimental Medicine and Biology. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2572-1_18.

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Conference papers on the topic "Rat carotid body cells"

1

Agarwal, Amit, Dong Yang, Insook Kim, and John L. Carroll. "Purinergic Modulation Of Carotid Body Glomus Cell Hypoxia Response During Postnatal Development In Rats." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4187.

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Attaluri, Anilchandra, Liang Zhu, and Zhongping Huang. "Targeted Brain Hypothermia Induced by an Interstitial Cooling Device in Human Neck: An Experimental Study." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205558.

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In recent years, mild or moderate hypothermia during which brain temperature is reduced to 30–35°C, has been proposed for clinical use as an adjunct for achieving protection from cerebral ischemia during cardiac bypass injury [Nussmeier 2002], carotid endarterectomy [Jamieson et al., 2003] and resection of extra-cranial aneurysm [Wagner and Zuccarello 2005], as well as stroke and traumatic brain injury [Marion et al., 1996; Marion 1997]. It has been shown that a reduction in brain temperature as small as 2°C substantially reduced ischemic cell damage [Clark et al., 1996], or improved significa

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3

Dryjski, Maciej, Eileen Mikat, and Thorir D. Bjornsson. "IN VIVO POTENCY OF HEPARIN AND HEPARINOIDS TO INHIBIT RAT SMOOTH MUSCLE CELL PROLIFERATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644604.

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The final response of endothelial cell injury in the arterial wall is characterized by proliferation of smooth muscle cells (SMC) in the intima to form a fibro-musculo-elastic plaque. Recent in vivo and in vitro studies have shown that heparin can inhibit proliferation of SMC. These studies, however, have not elucidated the relationships between heparin dose or concentration and its in vivo antiproliferative response. In the present study, we investigated the potency of standard heparin (SH), low molecular weight heparin (LMWH) and a mixture of sulfated glycosaminoglycans (Organon 10172) with

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Liu, Honghai, Julie X. Yun, Russel K. Pirlo, et al. "The Dependence of Mechanical Properties of Adult Rat Myocytes on Cell Alignment." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193024.

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In response to damage, stress and cell death cardiac muscle undergoes remodeling in which cardiomyocytes de-differentiate and re-differentiate. An understanding of the mechanisms involved in this process may lead to therapies to promote and enhance the repair of damaged cardiac tissue. However, due to the complexity of native environments, it is hard to investigate this remodeling process directly on tissues isolated from the body. Therefore, it is important to construct a cell-culture model that will replicate the most relevant characteristics of that tissue in controlled environments with gr

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Lieber, Samuel C., Nadine Aubry, Jayashree Pain, Gissela Diaz, Song-Jung Kim, and Stephen S. Vatner. "Measurement of the Transverse Apparent Elastic Modulus in Mammalian Cardiac Myocytes." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-41469.

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Transverse mechanical properties of mammalian cardiac myocytes, was determined by using atomic force microscopy (AFM). The AFM can be used as a nano-indentation device allowing transverse stiffness measurements to be conducted on biological cells in a physiological environment. This enables real-time biomechanical and physiological processes to be monitored with nano-scale resolution. Cellular mechanical properties were determined by indenting the cell’s body, and analyzing the indentation data with classical infinitesimal strain theory (CIST). This calculation was accomplished by modeling the

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De Clerck, F., R. Van de Wiele, B. Xhonneux, et al. "PLATELET TXA2 SYNTHETASE INHIBITION AND TXA2/PROSTAGLANDIN ENDOPEROXIDE RECEPTOR BLOCKADE COMBINED IN ONE MOLECULE (R 68070)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643465.

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F 68070, an oxime-alkane carboxylic acid derivative (Janssen Pharmaceutica), is a potent inhibitor of thromboxane A2 (TXA2) synthetase activity (IC50 in vitro against thrombin-stimulated human platelets in plasma : R 68070 : 2.9 x 10-8 M; CGS 13080 : 6 x 10-8 M; OKY-1581 : 8.2 x 10-8 M; dazmegrel : 2.6 x 10-8 M; dazoxiben : 2.3 x 10-8 M).The compound specifically inhibits platelet TXA2 synthetase activity (14C-arachidonic acid metabolism by washed human platelets) without effect on the cyclo-oxygenase, lipoxygenase (platelets, RBL cells) or prostacyclin synthetase activities (rat aortic rings)

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Zhang, Qingwei, Wei Zhang, Donggang Yao, Peter I. Lelkes, and Jack G. Zhou. "The Co-Continuous Micro-Porous PLLA Scaffolds and Their Application for ACL Reconstruction." In ASME 2010 International Mechanical Engineering Congress and Exposition. ASMEDC, 2010. http://dx.doi.org/10.1115/imece2010-38291.

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Anterior cruciate ligament (ACL) reconstructive surgery is a major health concern world-wide because of a large aging population and increased occurrence of sport-related damage. Tissue engineering is a rapidly growing interdisciplinary field that offers a promising new approach for ACL repair. In order to overcome the shortages of current existing surgical fixation devices, we are combining gradient cellular structure (GCS) injection molding technique and biomedical engineering to develop novel surgical fixation devices (screw, anchor, plate, pin, staple, etc.) that not only incorporate bioac

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