Relevant bibliographies by topics / Rat locomotion/brain receptors

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Rat locomotion/brain receptors'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Rat locomotion/brain receptors"

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Liu, Jun, and Larry M. Jordan. "Stimulation of the Parapyramidal Region of the Neonatal Rat Brain Stem Produces Locomotor-Like Activity Involving Spinal 5-HT7 and 5-HT2A Receptors." Journal of Neurophysiology 94, no. 2 (2005): 1392–404. http://dx.doi.org/10.1152/jn.00136.2005.

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Locomotion can be induced in rodents by direct application 5-hydroxytryptamine (5-HT) onto the spinal cord. Previous studies suggest important roles for 5-HT7 and 5-HT2A receptors in the locomotor effects of 5-HT. Here we show for the first time that activation of a discrete population of 5-HT neurons in the rodent brain stem produces locomotion and that the evoked locomotion requires 5-HT7 and 5-HT2A receptors. Cells localized in the parapyramidal region (PPR) of the mid-medulla produced locomotor-like activity as a result of either electrical or chemical stimulation, and PPR-evoked locomotor
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Kalyanasundar, B., Claudia I. Perez, Alvaro Luna, et al. "D1 and D2 antagonists reverse the effects of appetite suppressants on weight loss, food intake, locomotion, and rebalance spiking inhibition in the rat NAc shell." Journal of Neurophysiology 114, no. 1 (2015): 585–607. http://dx.doi.org/10.1152/jn.00012.2015.

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Obesity is a worldwide health problem that has reached epidemic proportions. To ameliorate this problem, one approach is the use of appetite suppressants. These compounds are frequently amphetamine congeners such as diethylpropion (DEP), phentermine (PHEN), and bupropion (BUP), whose effects are mediated through serotonin, norepinephrine, and dopaminergic pathways. The nucleus accumbens (NAc) shell receives dopaminergic inputs and is involved in feeding and motor activity. However, little is known about how appetite suppressants modulate its activity. Therefore, we characterized behavioral and
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BISOGNO, Tiziana, Dominique MELCK, Mikhail Yu BOBROV, et al. "N-acyl-dopamines: novel synthetic CB1 cannabinoid-receptor ligands and inhibitors of anandamide inactivation with cannabimimetic activity in vitro and in vivo." Biochemical Journal 351, no. 3 (2000): 817–24. http://dx.doi.org/10.1042/bj3510817.

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We reported previously that synthetic amides of polyunsaturated fatty acids with bioactive amines can result in substances that interact with proteins of the endogenous cannabinoid system (ECS). Here we synthesized a series of N-acyl-dopamines (NADAs) and studied their effects on the anandamide membrane transporter, the anandamide amidohydrolase (fatty acid amide hydrolase, FAAH) and the two cannabinoid receptor subtypes, CB1 and CB2. NADAs competitively inhibited FAAH from N18TG2 cells (IC50 = 19–100µM), as well as the binding of the selective CB1 receptor ligand, [3H]SR141716A, to rat brain
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Zaporozhets, Eugene, Kristine C. Cowley, and Brian J. Schmidt. "Neurochemical excitation of propriospinal neurons facilitates locomotor command signal transmission in the lesioned spinal cord." Journal of Neurophysiology 105, no. 6 (2011): 2818–29. http://dx.doi.org/10.1152/jn.00917.2010.

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Previous studies of the in vitro neonatal rat brain stem-spinal cord showed that propriospinal relays contribute to descending transmission of a supraspinal command signal that is capable of activating locomotion. Using the same preparation, the present series examines whether enhanced excitation of thoracic propriospinal neurons facilitates propagation of the locomotor command signal in the lesioned spinal cord. First, we identified neurotransmitters contributing to normal endogenous propriospinal transmission of the locomotor command signal by testing the effect of receptor antagonists appli
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Cowley, Kristine C., Eugene Zaporozhets, Jason N. MacLean, and Brian J. Schmidt. "Is NMDA Receptor Activation Essential for the Production of Locomotor-Like Activity in the Neonatal Rat Spinal Cord?" Journal of Neurophysiology 94, no. 6 (2005): 3805–14. http://dx.doi.org/10.1152/jn.00016.2005.

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Previous work has established that in vitro bath application of N-methyl-d-aspartic acid (NMDA) promotes locomotor activity in a variety of vertebrate preparations including the neonatal rat spinal cord. In addition, NMDA receptor activation gives rise to active membrane properties that are postulated to contribute to the generation or stabilization of locomotor rhythm. However, earlier studies yielded conflicting evidence as to whether NMDA receptors are essential in this role. Therefore in this study, we examined the effect of NMDA receptor blockade, using d-2-amino-5-phosphono-valeric acid
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Guo, Chunmei, Yang Yang, Yun'ai Su, and Tianmei Si. "Postnatal BDNF Expression Profiles in Prefrontal Cortex and Hippocampus of a Rat Schizophrenia Model Induced by MK-801 Administration." Journal of Biomedicine and Biotechnology 2010 (2010): 1–5. http://dx.doi.org/10.1155/2010/783297.

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Neonatal blockade of N-methyl-D-aspartic acid (NMDA) receptors represents one of experimental animal models for schizophrenia. This study is to investigate the long-term brain-derived neurotrophic factor (BDNF) expression profiles in different regions and correlation with “schizophrenia-like” behaviors in the adolescence and adult of this rat model. The NMDA receptor antagonist MK801 was administered to female Sprague-Dawley rats on postnatal days (PND) 5 through 14. Open-field test was performed on PND 42, and PND 77 to examine the validity of the current model. BDNF protein levels in hippoca
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Homma, Yutaka, R. D. Skinner, and E. Garcia-Rill. "Effects of Pedunculopontine Nucleus (PPN) Stimulation on Caudal Pontine Reticular Formation (PnC) Neurons In Vitro." Journal of Neurophysiology 87, no. 6 (2002): 3033–47. http://dx.doi.org/10.1152/jn.2002.87.6.3033.

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Stimulation of the pedunculopontine nucleus (PPN) is known to induce changes in arousal and postural/locomotor states. Previously, PPN stimulation was reported to induce prolonged responses (PRs) in extracellularly recorded PnC neurons in the decerebrate cat. The present study used intracellular recordings in semihorizontal slices from rat brain stem ( postnatal days 12–21) to determine responses in PnC neurons following PPN stimulation. Two-thirds (65%) of PnC neurons showed PRs after PPN stimulation. PnC neurons with PRs had higher amplitude afterhyperpolarizations (AHP) than non-PR (NPR) ne
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Chen, Gang, Yimin Liang, Fanghu Chen, Haifeng Wang, and Guoming Zhu. "The effect of lithium chloride on the motor function of spinal cord injury–controlled rat and the relevant mechanism." European Journal of Inflammation 17 (January 2019): 205873921985285. http://dx.doi.org/10.1177/2058739219852855.

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The objective of this study is to discuss the effect and mechanism of lithium chloride on the rehabilitation of locomotion post spinal cord injury (SCI) by observing the effect of lithium chloride on the expression of the brain-derived neurotrophic factor (BDNF)/tropomyosin receptor kinase B (TrkB) pathway. In total, 36 Sprague-Dawley (SD) rats were randomly divided into the sham operation group (n = 12), model group (n = 12), and lithium chloride group (n = 12). The sham operation group underwent laminectomy, while for the model group and the lithium chloride group with the NYU spinal cord im
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Wang, Xingxing, Tianna Zhou, George D. Maynard, et al. "Nogo receptor decoy promotes recovery and corticospinal growth in non-human primate spinal cord injury." Brain 143, no. 6 (2020): 1697–713. http://dx.doi.org/10.1093/brain/awaa116.

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Abstract After CNS trauma such as spinal cord injury, the ability of surviving neural elements to sprout axons, reorganize neural networks and support recovery of function is severely restricted, contributing to chronic neurological deficits. Among limitations on neural recovery are myelin-associated inhibitors functioning as ligands for neuronal Nogo receptor 1 (NgR1). A soluble decoy (NgR1-Fc, AXER-204) blocks these ligands and provides a means to promote recovery of function in multiple preclinical rodent models of spinal cord injury. However, the safety and efficacy of this reagent in non-
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Zarrindast, M. R., M. Nasehi, and M. Pournaghshband. "Effects of dopaminergic drugs in the dorsal hippocampus of rats in the MK801-induced anxiolytic-like behavior." European Psychiatry 26, S2 (2011): 441. http://dx.doi.org/10.1016/s0924-9338(11)72148-4.

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IntroductionExcitatory transmission through glutamate receptors constitutes the main mode of synaptic signaling in the brain regions that are critical for cognition such as learning and anxiety.ObjectivesThe possible involvement of dorsal hippocampal (intra-CA1) dopaminergic receptor mechanism on the anxiolytic-like response induced by NMDA receptor antagonist, MK801 has been investigated in the present study.MethodsThe male wistar rats were used and the elevated plus maze apparatus has been used to test parameters (%OAT, %OAE, locomotor activity, grooming, rereading and defection) of anxiety-
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Dissertations / Theses on the topic "Rat locomotion/brain receptors"

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Coles, S. K. "Controls of the locomotor system in the rat." Thesis, University of Oxford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233493.

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Boulay, Denis. "Etude chez le rat des modifications comportementales et neurochimiques après administrations répétées d'agonistes dopaminergiques indirects : la cocaïne ou le GBR12783." Rouen, 1995. http://www.theses.fr/1995ROUE5042.

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Nous avons recherché si un blocage répété du système de capture de la dopamine par un inhibiteur spécifique de la capture neuronale de dopamine, le GBR12783, pouvait mimer les sensibilisations observées après administrations de cocaïne, inhibiteur non sélectif de la recapture des monoamines. Des administrations répétées de GBR12783 ont induit une sensibilisation à ses effets stimulants moteurs, se développant au cours du temps et pouvant persister plusieurs semaines après la dernière administration. L'expression de cette sensibilisation dépend en partie de la durée des traitements chroniques ;
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Cohen, Ricky Israel. "Rat brain oligodendrocytes express muscarinic and adrenergic receptors." Thesis, McGill University, 1996. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=42006.

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The aim of the studies underlying this thesis was to characterize the muscarinic and adrenergic receptors expressed in rat brain oligodendrocytes' (OLs); determine if ligand binding alters second messenger levels classically associated with these families of receptors, such as inositol phosphates (InsP), intracellular calcium ( (Ca$ rm sp{2+} rbrack sb{i}),$ and cyclic AMP (cAMP); and the role of neurotransmitters, acetylcholine (Ach) or norepinephrine (NE) on oligodendrocyte growth.<br>CCH (carbachol), a stable Ach analog, caused a concentration and time dependent increase in the accumulation
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Cohen, Ricky Israel. "Rat brain oligodendrocytes express muscarinic and adrenergic receptors." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0022/NQ29913.pdf.

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Mahalekam, Malani. "Natriuretic peptide receptors in developing and adult rat brain." Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368618.

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Howard, Helen Clare. "The pharmacological characterisation of oxytocin receptors in the rat brain." Thesis, University of Bristol, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299594.

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Pasquini, Filoteo. "High resolution radioautographic visualization of delta opioid receptors in rat brain." Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=64051.

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Smith, Steven Andrew. "The role of Peroxisome proliferator-activated receptors in the rat brain /." St. Lucia, Qld, 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17982.pdf.

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Gibbins, Jonathan Martin. "The purification of corticotropin-releasing factor (CRF) receptors from rat brain." Thesis, University of Reading, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260813.

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Schindler, Marcus. "The distribution of somatostatin receptors in the rat and human brain." Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627298.

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Books on the topic "Rat locomotion/brain receptors"

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Bowlby, Deborah Anne. Regulation of wstrogen receptors in the rat brain. National Library of Canada, 1994.

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Cao, Chenglong. Immunological screening of a rat brain cDNA library for genes encoding potential novel glutamate receptors. National Library of Canada, 1993.

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Billinton, Andrew. Brain GABA[Beta] receptors and GBR1 mRNA in rat seizure models and human temporal lobe epilepsy. University of Birmingham, 1999.

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Clarke, Wendy Elizabeth. Intracellular compartmentalisation of fibroblast growth factor-2 and associated high and low affinity receptors after cerebral injury to the adult rat brain. University of Birmingham, 1999.

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Seale, J. V. A quantitative immunohistochemical analysis of the distribution of insulin receptors in the rat brain for regions associated with memory compared to regions not associated with memory. University of Surrey Roehampton, 2001.

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Grove, Kevin Lyle. Angiotensin II receptors in the hypothalamus of the adult and developing female rat brain. 1994.

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7

Autoradiographic localization of insulin receptors in rat brain: Prominence in olfactory and limbic areas. National Institute of Arithitis, Diabetes, and Digestive and Kidney Diseases], 1986.

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Cao, Chenglong. Immunological screening of a rat brain cDNA library for genes encoding potential novel glutamate receptors. 1994.

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9

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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Bird, Mark F., and David G. Lambert. Deorphanization of ORL-1/LC132 by reverse pharmacology in two landmark studies. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0026.

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Deorphanization of ORL-1/LC132 in 1995 by reverse pharmacology in two simultaneously published landmark studies added a new member to the opioid family of G-protein coupled receptors. Meunier and Reinscheid used cells expressing recombinant ORL-1 (human) or LC132 (rat) and the presumed intracellular inhibition of cyclic AMP formation to ‘fish’ for endogenous peptide ligands in rat whole-brain and pig hypothalamic extracts. Both studies reported the isolation of a 17-amino-acid peptide, which was named nociceptin and orphanin FQ by the two authors, respectively. The behaviour of the isolated pe
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Book chapters on the topic "Rat locomotion/brain receptors"

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Wood, P. L., L. L. Martin, and C. A. Altar. "[3H]Tryptamine Receptors in Rat Brain." In Neuropsychopharmacology of the Trace Amines. Humana Press, 1985. http://dx.doi.org/10.1007/978-1-4612-5010-4_9.

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Clarke, P. B. S., and A. Pert. "Autoradiographical Evidence of Nicotinic Receptors in Rat Brain." In Tobacco Smoking and Nicotine. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1911-5_11.

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Mendelsohn, Laurane G. "Visualization of IGF-2 Receptors in Rat Brain." In Insulin, Insulin-like Growth Factors, and Their Receptors in the Central Nervous System. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5380-5_21.

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Chabot, Jean-Guy, Satyabrata Kar, and Rémi Quirion. "Ontogenic Profile of Epidermal Growth Factor Receptors in Rat Brain." In Receptors in the Developing Nervous System. Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1540-7_5.

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Ben-Shachar, Dorit, and Moussa B. H. Youdim. "Brain Iron and Dopamine D2 Receptors in the Rat." In Advances in Behavioral Biology. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2179-8_33.

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Taylor, J. E. "Binding of Neuromediators to Their Receptors in Rat Brain." In Rökan. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73686-5_11.

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Ikezaki, Kiyonobu, T. Nomura, M. Takahashi, B. F. Zieroth, and M. Fukui. "MRI Contrast Enhancement by Mn-TPPS in Experimental Rat Brain Tumor with Peripheral Benzodiazepine Receptors." In Brain Edema IX. Springer Vienna, 1994. http://dx.doi.org/10.1007/978-3-7091-9334-1_95.

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Scharfman, Helen E. "Presynaptic and Postsynaptic Actions of Somatostatin in Area CA1 and the Dentate Gyrus of Rat and Rabbit Hippocampal Slices." In Presynaptic Receptors in the Mammalian Brain. Birkhäuser Boston, 1993. http://dx.doi.org/10.1007/978-1-4684-6825-0_4.

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Winkler, T., H. S. Sharma, E. Stålberg, and J. Westman. "Benzodiazepine Receptors Influence Spinal Cord Evoked Potentials and Edema Following Trauma to the Rat Spinal Cord." In Brain Edema X. Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6837-0_66.

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Kitabgi, Patrick, and Jean-Pierre Vincent. "Effects of Cations and Nucleotides on Neurotensin Binding to Rat Brain Synaptic Membranes." In Neural and Endocrine Peptides and Receptors. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5152-8_21.

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Conference papers on the topic "Rat locomotion/brain receptors"

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Fukayama, Osamu, Noriyuki Taniguchi, Takafumi Suzuki, and Kunihiko Mabuchi. "RatCar system for estimating locomotion states using neural signals with parameter monitoring: Vehicle-formed brain-machine interfaces for rat." In 2008 30th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2008. http://dx.doi.org/10.1109/iembs.2008.4650416.

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