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Relevant bibliographies by topics / Rats Olfactometry Electronic dissertations / Dissertations / Theses

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Dissertations / Theses on the topic 'Rats Olfactometry Electronic dissertations'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Thomas, Rhiannon D. "Olfactory matching-to-sample in rats using a novel apparatus /." Electronic version (PDF), 2006. http://dl.uncw.edu/etd/2006/thomasr/rhiannonthomas.pdf.

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2

Poerstel, L. Brooke. "Evaluating equivalence relations in rats using an olfactory matching-to-sample procedure /." Electronic version (PDF), 2007. http://dl.uncw.edu/etd/2007-3/poerstell/laurelpoerstel.pdf.

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3

Rayburn-Reeves, Rebecca M. "A study of functional equivalence in rats using class-specific reinforcers and olfactory stimuli /." Electronic version (PDF), 2007. http://dl.uncw.edu/etd/2007-2/rayburn-reevesr/rebeccarayburn-reeves.pdf.

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4

MacQueen, David A. "Non-competitive NMDA receptor antagonist impairs olfactory memory span in rats." View electronic thesis (PDF), 2009. http://dl.uncw.edu/etd/2009-1/macqueend/davidmacqueen.pdf.

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5

Lefever, Timothy W. "Effects of olanzapine on olfactory delayed matching-to-sample in rats." View electronic thesis (PDF), 2009. http://dl.uncw.edu/etd/2009-3/lefevert/timothylefever.pdf.

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6

Stairs, Dustin J. "Effects of bupropion on nicotine self-administration and food-maintained responding in rats /." Electronic version (PDF), 2003. http://dl.uncw.edu/etd/2003/stairsd/dustinstairs.pdf.

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7

Bullard, Laura A. "The effects of the NMDA antagonist dizocilpine on an olfactory delayed match-to-sample task in rats." View electronic thesis (PDF), 2009. http://dl.uncw.edu/etd/2009-1/bullardl/laurabullard.pdf.

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8

Penã, Tracy M. "Generalized identity matching-to-sample in rats using olfactory stimuli /." Electronic version (PDF), 2003. http://dl.uncw.edu/etd/2003/penat/tracypena.html.

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9

Goldsbury, Robin Paulette, and University of Lethbridge Faculty of Arts and Science. "Early unilateral olfactory bulb lesion results in diffuse changes in behavior and overall cortical organization." Thesis, Lethbridge, Alta. : University of Lethbridge, Faculty of Arts and Science, 2007, 2007. http://hdl.handle.net/10133/683.

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The current work explores the behavioural and anatomical consequences of unilateral neonatal bulbectomy (OBX) in male and female rats at postnatal day 1 (PI) and P10. In adulthood the animals underwent a battery of motor and cognitive tests, and diffuse effects of early brain injury on the development of behavior were found. Disturbing olfactory sense input during development affected motor output. Rats normally display an equal distribution of right or left paw preference. In this study, both OBX sexes showed a shifted paw preference to the ipsilesional side, and forelimb deficits were found in a skilled reaching task. Lesion animals also showed enhanced performance on a visually driven spatial cognitive test. Cross-modal compensatory changes may be responsible. Morphological changes within the cerebral cortex are described, including bulbar changes, enlarged but fewer glomeruli, smaller accessory olfactory bulb, decreased downstream connectivity, and a rostral shift of the forebrain toward the olfactory bulb. Changes to the lateral cortex were found in both intact and lesion hemispheres, along with dendritic changes in the forelimb reaching area. Cellular regeneration within the lesion bulb was indicated. Changed shape and relative size increases compared to the intact bulb were found. BrdU labeling showed increased mitotic activity in P10 lesion animals. These findings demonstrate that the impact of olfactory injury during early development goes well beyond odor perception and discrimination, and that olfactory inputs during development significantly contribute to the development of the neocortex.
xix, 195 leaves : ill. (some col.) ; 29 cm.

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10

Yim, Tonia Tan-Ling, and University of Lethbridge Faculty of Arts and Science. "Multiple-object memory requires the hippocampus." Thesis, Lethbridge, Alta. : University of Lethbridge, Faculty of Arts and Science, 2007, 2007. http://hdl.handle.net/10133/678.

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This thesis investigates the role of the hippocampus in object memory. Currently, the role of the hippocampus in object recognition is unclear, with some studies demonstrating a delay-dependent impairment after hippocampal damage, others showing no impairment. The present thesis used the novel object recognition task and its variants to investigate various types of object memory in hippocampal lesion rats. In the first study, impairments were observed in discriminating object order and associating objects with contexts, while no impairment was observed in novel object recognition. In the second study, it was found that encountering another object shortly prior to or after encountering a target object impairs the recognition of the target object. In a control procedure, encountering a novel context either shortly before or after encountering the target object did not impair object recognition. In sum, in the absence of the hippocampus, object memory becomes vulnerable to interference, rendering rats unable to discern memories of multiple objects. The present thesis concludes that the hippocampus discriminates multiple objects via pattern separation. A stimulus-response model relating the role of the hippocampus to object memory is proposed.
vii, 150 leaves : ill. ; 29 cm. --

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11

Melvin, Neal, and University of Lethbridge Faculty of Arts and Science. "Differential neurogenesis in the adult rat dentate gyrus." Thesis, Lethbridge, Alta. : University of Lethbridge, Faculty of Arts and Science, 2008, 2008. http://hdl.handle.net/10133/727.

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Adult neurogenesis is a fundamental feature of mammalian nervous systems. Curiously, neurogenesis in the dentate gyrus is typically regarded as homogenous. This thesis challenges that view, and reports the discovery and characterization of a novel region of the dentate gyrus that consistently lacks basal neurogenesis. We demonstrate that this area, referred to as the neurogenically quiescent zone, represents approximately 1.5% of the total volume of the dentate gyrus, and that its location is invariant among animals. This region contains several critical cell types and molecular factors that are known to be critical to the neurogenic niche, including stem cells. We also present data that attempt to conceptualize the existence of this region in the context of early agerelated declines in neurogenesis. Finally, we demonstrate that, under some behavioural conditions, neurogenesis can be induced in this region, suggesting that, under basal conditions, it may simply lack the presence of pro-neurogenic factors.
xvi, 125 leaves : ill. ; 29 cm. --

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12

Nemati, Farshad, and University of Lethbridge Faculty of Arts and Science. "Contribution of brain with or without visual cortex lesion to exploratory locomotion in the rat." Thesis, Arts and Science, 2008. http://hdl.handle.net/10133/665.

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Over the past five decades spatial behavior has been a subject of research interest in psychology and neuroscience, in part based on philosophical theories of mental spatial representations. In order to continue uncovering the facts regarding spatial behavior, the focus of this thesis was on the contribution of entry point and visual inputs to the organization of exploratory locomotion and spatial representation in the rat. Despite the contribution of the hippocampus to spatial abilities, the exploratory locomotion is still visually organized in rats with damage to the hippocampus. On the other hand, recent studies have demonstrated a contribution of visual areas to the spatial ability of the rat. Nevertheless, the contribution of visual cortex to the organization of exploratory locomotion has not been studied in an open field. The experiments in this thesis were designed to characterize the organization of exploratory locomotion to the point of entry and/or visual cues. Rats were started from the edge or center of an open table near or on which a salient object could be placed. The main findings were that rats organized their exploratory locomotion to their point of entry and modified their behavior as they encountered objects. Also, rats with damage to visual cortex displayed an extra-attachment to the visual objects and in contrast to controls did not expand their exploratory locomotion with time. The results are discussed with respect to the centrality of the entry point in the organization of exploratory locomotion and the neural network that control visual exploration in the rat.
xiii, 220 leaves : ill. ; 29 cm. --

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13

Miller, Laurence L. "A competitive NMDA receptor antagonist potentiates the effects of morphine on spatial and discrimination learning /." Electronic version (PDF), 2005. http://dl.uncw.edu/etd/2005/millerl/laurencemiller.pdf.

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14

Salling, Michael C. "The effects of fluoxetine and environmental enrichment on recovery of function following focal dentate gyrus lesions." View electronic thesis, 2008. http://dl.uncw.edu/etd/2008-2/sallingm/michaelsalling.pdf.

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15

van, Waes Linda T. A., and University of Lethbridge Faculty of Arts and Science. "Does one plus one make two? Investigation of pharmacological effects and cortical injury on the developing brain." Thesis, Lethbridge, Alta. : University of Lethbridge, Deptartment of Neuroscience, 2008, 2009. http://hdl.handle.net/10133/782.

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This thesis examined how pharmacological treatment and cortical injury during development affects brain plasticity. Rats were given either a low dose of perinatal fluoxetine or a mild postnatal day 7 Hypoxic‐Ischemic (HI) injury, both, or neither. The functional outcome was assessed using a series of behavioral tasks and anatomical measures. To assess how HI affects the development of motor maps, forelimb motor maps were evoked at P19. The findings indicate that fluoxetine treatment or HI injury mostly negatively affected functional outcome. The combined treatment with fluoxetine and HI injury only interacted on a limited number of measures. There was no delay in the emergence of evoked motor movements, or change in map location in the HI animals. These results suggest that the pharmacological treatment and cortical injury described in this thesis may have different mechanisms whereby plastic changes are induced and the interaction between these two mechanisms is limited.
xii, 169 leaves : ill. ; 29 cm.

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16

Faraji, Jamshid, and University of Lethbridge Faculty of Arts and Science. "Structural alterations in the hippocampus and spatial behavior by stress in male and female rats : protections, and recovery in water-based and dry-land tasks." Thesis, Lethbridge, Alta : University of Lethbridge, Faculty of Arts and Science, 2008, 2008. http://hdl.handle.net/10133/725.

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Stress-related cognitive changes are still a matter of debate. In some particular neuropathological conditions such as focal ischemia, cognitive functions have been shown to be significantly impaired. These conditions, however, may be improved by some factors such as steroid hormones. The purpose of the current thesis was to assess the structural and functional effects of corticosterone-related experiences on the hippocampus before and after endothelin-1 (ET-1)-induced stroke. We found corticosterone-related experiences enhance the hippocampal recovery, and improve its function in both wet and dryland tasks after ET-1-induced focal stroke. Structural and functional effects of such experiences prior to the focal ischemia in the hippocampus, however, showed that stress, not corticosterone is a strong inhibitor for hippocampal recovery.
xii, 252 leaves : ill. ; 29 cm. --

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17

Baker, Mike, and University of Lethbridge Faculty of Arts and Science. "Role of epigenetic changes in direct and indirect radiation effects." Thesis, Lethbridge, Alta. : University of Lethbridge, Faculty of Arts and Science, 2008, 2008. http://hdl.handle.net/10133/650.

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For over 100 years, cancer radiation therapy has provided patients with increased survival rates. Despite this success, radiation exposure poses a threat to the progeny of exposed parents. It causes transgenerational genome instability that is linked to transgenerational carcinogenesis. The exact mechanisms in which this instability occurs have yet to be discovered. Current evidence points to their epigenetic nature, specifically changes in DNA methylation. Using mouse and rat models, this thesis investigated the transgenerational effects of radiation in the offspring from parents who received whole body or localized exposure to ionizing radiation (IR). Both types of exposure resulted in significant global DNA hypomethylation in the somatic tissues of the progeny. These changes were paralleled by the significantly decreased levels of methyltransferases and methyl-CpG-binding protein. In summary, our results suggest that both localized and whole body parental exposures to IR result in transgenerational epigenetic instability within the unexposed offspring.
vii, 106 leaves : ill. ; 29 cm.

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18

Bell, Heather C., and University of Lethbridge Faculty of Arts and Science. "Playful feedback and the developing brain." Thesis, Lethbridge, Alta. : University of Lethbridge, Faculty of Arts and Science, 2008, 2008. http://hdl.handle.net/10133/747.

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The prefrontal cortex (PFC) has long been thought to be the seat of social behaviours in mammals. Lesions of the orbitofrontal cortex (OFC), a subregion of the PFC, are known to cause social deficits in humans. Interestingly, social deficits are also seen in rats with OFC lesions. Rats that are deprived of peer play during development exhibit behaviour similar to OFC-ablated animals. Another subregion of the PFC, the medial prefrontal cortex (mPFC) is interconnected with the OFC. The mPFC and OFC have been shown to be reciprocally responsive to a variety of inuences, in terms of dendritic morphology. It was hypothesized that social experiences are necessary for the proper development of the OFC, and that, because of the interconnectivity, the mPFC would also be sensitive to social experience. The social condition in which juvenile rats were raised was manipulated, and the OFC and mPFC were shown to be differentially responsive to specific aspects of social experience. It was already known that OFC lesions produce specific social deficits, but the contribution of the mPFC to the production of social behaviour was unknown. To investigate the contribution of the mPFC to the performance of social behaviour, animals were given mPFC lesions, and their social play behaviour was quantified. mPFC-ablated animals had altered play patterns that were distinct from those seen in the OFC-ablated animals. It was concluded that the OFC and mPFC are differentially responsive to social stimuli during development, and that the OFC and mPFC make discrete contributions to the production of social behaviour. The results were interpreted in an evolutionary context.
x, 93 leaves : ill. ; 29 cm.

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