Relevant bibliographies by topics / Recepto 5-HT1A

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Recepto 5-HT1A'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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Journal articles on the topic "Recepto 5-HT1A"

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García-Pedraza, José Ángel, Oswaldo Hernández-Abreu, Mónica García, Asunción Morán, and Carlos M. Villalón. "Chronic 5-HT2 receptor blockade unmasks the role of 5-HT1F receptors in the inhibition of rat cardioaccelerator sympathetic outflow." Canadian Journal of Physiology and Pharmacology 96, no. 4 (2018): 328–36. http://dx.doi.org/10.1139/cjpp-2017-0191.

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Serotonin (5-hydroxytryptamine; 5-HT) inhibits the rat cardioaccelerator sympathetic outflow by 5-HT1B/1D/5 receptors. Because chronic blockade of sympatho-excitatory 5-HT2 receptors is beneficial in several cardiovascular pathologies, this study investigated whether sarpogrelate (a 5-HT2 receptor antagonist) alters the pharmacological profile of the above sympatho-inhibition. Rats were pretreated for 2 weeks with sarpogrelate in drinking water (30 mg/kg per day; sarpogrelate-treated group) or equivalent volumes of drinking water (control group). Animals were pithed and prepared for spinal sti
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Lee, T. J., M. Ueno, N. Sunagane, and M. H. Sun. "Serotonin relaxes porcine pial veins." American Journal of Physiology-Heart and Circulatory Physiology 266, no. 3 (1994): H1000—H1006. http://dx.doi.org/10.1152/ajpheart.1994.266.3.h1000.

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The effect of serotonin [5-hydroxytryptamine (5-HT)] on pial venous tone of the pig was examined using in vitro tissue bath techniques. Isolated pial venous rings exhibited spontaneous rhythmic contractions (SRC) on mechanical stretching and/or applications of several vasoactive substances, including norepinephrine. On the other hand, KCl induced sustained active muscle tone (SAT) without SRC. The SRC induced by mechanical stretching were not affected by tetrodotoxin, nitro-L-arginine, alpha- and beta-adrenergic, histaminergic, and muscarinic receptor antagonists, indicating that the SRC in po
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Montgomery, Stuart A., and Naomi Fineberg. "Is there a Relationship between Serotonin Receptor Subtypes and Selectivity of Response in Specific Psychiatric Illnesses?" British Journal of Psychiatry 155, S8 (1989): 63–69. http://dx.doi.org/10.1192/s0007125000291770.

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Identification of 5-HT receptor subtypes — 5-HT1A, 5-HT1B, 5-HT1c, 5-HT1D, 5-HT2 (possibly A and B), 5-HT3 subtypes, and possibly 5-HT4 — has encouraged the manufacture of 5-HT receptor inhibitors with greater subtype specificity. However, it appears that the receptors interact, and drugs initially thought to be specific may have multiple actions. For some conditions such as anxiety/depression, almost all receptors are implicated. Clinical studies provide clear evidence that manipulation of the 5-HT system has a role in treating depression, anxiety, obsessional illness, migraine, and eating di
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Reuter, U., S. Salomone, GW Ickenstein, and C. Waeber. "Effects of Chronic Sumatriptan and Zolmitriptan Treatment on 5-HT1 Receptor Expression and Function in Rats." Cephalalgia 24, no. 5 (2004): 398–407. http://dx.doi.org/10.1111/j.1468-2982.2004.00683.x.

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Triptans are commonly used anti-migraine drugs and show agonist action mainly at serotonin 5-HT1B/1D/1F receptors. It is not known whether frequent or long-term treatment with these drugs would alter 5-HT receptor function. We investigated the effects of protracted (14-18 days) sumatriptan and zolmitriptan treatment in rats on 5-HT1 receptor mRNA expression and function in tissues related to migraine pathophysiology. RT-PCR analysis revealed that 5-HT1B/1D/1F receptor mRNA was reduced in the trigeminal ganglion after treatment with either triptan (reduction by: sumatriptan 39% and zolmitriptan
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Nelson, David L., Lee A. Phebus, Kirk W. Johnson, et al. "Preclinical pharmacological profile of the selective 5-HT1F receptor agonist lasmiditan." Cephalalgia 30, no. 10 (2010): 1159–69. http://dx.doi.org/10.1177/0333102410370873.

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Introduction: Lasmiditan (also known as COL-144 and LY573144; 2,4,6-trifluoro- N-[6-[(1-methylpiperidin-4-yl)carbonyl]pyridin-2yl]benzamide) is a high-affinity, highly selective serotonin (5-HT) 5-HT1F receptor agonist. Results: In vitro binding studies show a Ki value of 2.21 nM at the 5-HT1F receptor, compared with Ki values of 1043 nM and 1357 nM at the 5-HT1B and 5-HT1D receptors, respectively, a selectivity ratio greater than 470-fold. Lasmiditan showed higher selectivity for the 5-HT1F receptor relative to other 5-HT1 receptor subtypes than the first generation 5-HT1F receptor agonist LY
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Lindhe, Örjan, Per Almqvist, Matts Kågedal, et al. "Autoradiographic Mapping of 5-HT1B/1D Binding Sites in the Rhesus Monkey Brain Using [carbonyl-11C]zolmitriptan." International Journal of Molecular Imaging 2011 (October 12, 2011): 1–6. http://dx.doi.org/10.1155/2011/694179.

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Zolmitriptan is a serotonin 5-HT1B/1D receptor agonist that is an effective and well-tolerated drug for migraine treatment. In a human positron emission tomography study, [11C]zolmitriptan crossed the blood-brain barrier but no clear pattern of regional uptake was discernable. The objective of this study was to map the binding of [11C]zolmitriptan in Rhesus monkey brain using whole hemisphere in vitro autoradiography with [11C]zolmitriptan as a radioligand. In saturation studies, [11C]zolmitriptan showed specific (90%) binding to a population of high-affinity binding sites (Kd 0.95–5.06 nM). T
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Hamel, Edith. "The Biology of Serotonin Receptors: Focus on Migraine Pathophysiology and Treatment." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 26, no. 3 (1999): 2–6. http://dx.doi.org/10.1017/s0317167100000123.

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Serotonin receptors are highly heterogeneous and they have been regrouped within seven different families (5-HT1 - 5-HT7). With the exception of the 5-HT3 which is a ligand-gated ion channel, all others are G-protein coupled receptors with each family sharing structural, pharmacological and transductional characteristics. 5-HT receptors have been implicated in the regulation of several psychiatric and neurological disorders related to serotonergic neurotransmission, and specific receptor subtypes have recently been associated with either the pathogenesis or the treatment of migraine headache.
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Olivier, B., J. Schipper, J. A. M. van der Heyden, A. van Hest, J. Mos, and M. Th M. Tulp. "Preclinical evidence for the role of serotonin receptor-subtypes in depression." Acta Neuropsychiatrica 4, no. 2 (1992): 40–45. http://dx.doi.org/10.1017/s0924270800034888.

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SummarySerotonin (5-HT) plays an important role in depression and specific 5-HT reuptake blockers appear to be clinically important antidepressants. It is unclear however, which serotonergic mechanism is involved in the antidepressant effect, certainly when regarding the existence of at least seven 5-HT receptor subtypes. By testing different 5-HT ligands in two animal models of depression (forced swimming and DRL72-S test) and comparison with data from literature, evidence is provided for potential antidepressant qualities of 5-HT1A receptor-agonists and 5-HT1C receptor-antagonists. Compounds
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Cowen, P. J. "Serotonin Receptor Subtypes: Implications for Psychopharmacology." British Journal of Psychiatry 159, S12 (1991): 7–14. http://dx.doi.org/10.1192/s0007125000296190.

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Serotonin (5-HT) interacts with multiple brain 5-HT receptor subtypes to influence a wide range of behaviours. Three main families of 5-HT receptors (5-HT1, 5-HT2 and 5-HT3) have been described which differ in their binding affinity for selective ligands, their receptor-effector coupling mechanisms, and the behavioural processes they regulate. Nevertheless, manipulation of several different 5-HT receptor subtypes (5-HT1A, 5-HT1c, 5-HT2 and 5-HT3) may produce anxiolytic effects; 5-HT1A and 5-HT2 receptors may be involved in the aetiology of major depression and the therapeutic effects of antide
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Ghavami, A., K. L. Stark, M. Jareb, S. Ramboz, L. Segu, and R. Hen. "Differential addressing of 5-HT1A and 5-HT1B receptors in epithelial cells and neurons." Journal of Cell Science 112, no. 6 (1999): 967–76. http://dx.doi.org/10.1242/jcs.112.6.967.

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The 5-HT1A and 5-HT1B serotonin receptors are expressed in a variety of neurons in the central nervous system. While the 5-HT1A receptor is found on somas and dendrites, the 5-HT1B receptor has been suggested to be localized predominantly on axon terminals. To study the intracellular addressing of these receptors, we have used in vitro systems including Madin-Darby canine kidney (MDCK II) epithelial cells and primary neuronal cultures. Furthermore, we have extended these studies to examine addressing in vivo in transgenic mice. In epithelial cells, 5-HT1A receptors are found on both apical and
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Dissertations / Theses on the topic "Recepto 5-HT1A"

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Weber, Karen Cacilda. "Modelagem molecular de compostos arilpiperazínicos e suas interações com o receptor 5-HT1A." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/75/75131/tde-05122008-165529/.

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Os inibidores seletivos da recaptação de serotonina (ISRSs) representam a classe mais importante de antidepressivos em uso clínico atualmente. Entretanto, esses medicamentos costumam levar de duas a seis semanas para apresentar os efeitos de sua ação terapêutica. Estudos clínicos mostram que quando um antagonista do receptor 5-HT1A é administrado juntamente com um ISRS, um aumento da concentração extracelular de serotonina é observado nas áreas terminais dos neurônios. Assim, a combinação de um antagonista do receptor 5-HT1A com um ISRS pode acelerar o início da ação antidepressiva, aumentando
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Lüttgen, Maria. "Serotonergic receptor subtypes in learning and memory : focus on 5-HT1A, 5-HT1B and 5-HT2A receptors /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-628-6148-4/.

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Hu, Xiao Jing. "Modulation of acetylcholine release by serotonergic 5-HT1A and 5-HT1B receptors : a microdialysis study in the awake rat /." Stockholm, 2007.

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Barf, Tjeerd Andries. "The medicinal chemistry of aryl triflates as applied to 5-HT1A and 5-HT1D receptor ligands /." [S.l. : [Groningen] : s.n.] ; [University Library Groningen] [Host], 1996. http://irs.ub.rug.nl/ppn/152303871.

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Centenaro, Lígia Aline. "Provocação social e comportamento agressivo : envolvimento dos receptores 5-HT1A e 5-HT1B no córtex pré-frontal." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/13070.

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A provocação social é um método utilizado em animais de laboratório para a indução de elevados níveis de agressividade, produzindo padrões comportamentais semelhantes ao de indivíduos violentos. Estudos prévios utilizando drogas que atuam seletivamente sobre os receptores 5-HT1A e 5-HT1B demonstraram uma redução pronunciada no comportamento agressivo. Um dos mais importantes sítios de ação para esses agentes serotonérgicos é o córtex pré-frontal, uma região cerebral particularmente relevante no controle inibitório da agressividade e da impulsividade. O objetivo do presente estudo foi verificar
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Zhou, Yi Yuan. "Structural Determinants of 5-Ht1a Receptor Interaction With Gαi Subunits". Thesis, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/19761.

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The 5-hydroxytryptamine (5-HT) system modulates numerous physiological and behavioural processes, and dysfunction within this system underlies many behavioural disorders, such as major depression. The 5-HT1A receptor is the primary somatodendritic autoreceptor that controls the firing rate of 5-HT neurons, but is also coupled to numerous signalling pathways. An understanding of 5-HT1A receptor signalling may lead to the development of antidepressant drugs that selectively target therapeutic pathways in treating depression. The 5-HT1A receptor is coupled to inhibitory G-proteins via its intrace
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Wong, Kit Yee. "Design and synthesis of 5-HT1A receptor ligands." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266349.

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Oller, Canet Sílvia. "Receptor 5-HT1A i ISRS: Escurçament de la resposta antidepressiva." Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/461153.

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Els fàrmacs antidepressius necessiten unes quantes setmanes per aconseguir una resposta clínica (Trivedi et al., 2006). Una possible explicació és que l’augment de la concentració de serotonina (5-HT) extracel·lular produïda pels inhibidors de la recaptació de serotonina (ISRS) estigui limitada per l’activació dels autoreceptors 5-HT1A. La utilització de molècules que antagonitzin el receptor 5-HT1A i el dessensibilitzin podria escurçar la resposta antidepressiva i/o augmentar-ne l’efecte. Es realitzen dos assajos clínics utilitzant dues molècules amb acció sobre el 5-HT1A: el Pindolol, un fàr
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Cockburn, Amanda M. "Estrogen regulation of the human 5-HT1A receptor gene." Thesis, University of Ottawa (Canada), 2004. http://hdl.handle.net/10393/26611.

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Altered regulation of the 5-HT1A receptor is implicated in depression. Since depression is twice as prevalent in women versus men and major hormonal events in women trigger depression, I evaluated estrogen regulation of the 5-HT1A receptor in rat RN46A raphe cells. Estrogen (100 nM) induced no change in transcriptional activity of the 5-HT1A promoter (-1515bp/ATG). A putative estrogen response element (1AERE, -433bp) was identified in human and mouse but not rat 5-HT1A promoter. At least one nuclear protein complex from RN46A cells expressing estrogen receptor (ERbeta) bound specifically to th
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Santos, Alice Hartmann dos. "Efeitos do canabidiol no comportamento agressivo induzido por isolamento social em camundongos." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/17/17133/tde-20072016-151736/.

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O Canabidiol (CBD), principal composto não-psicotomimético da Cannabis sativa, possui diversas propriedades farmacológicas, incluindo a indução de efeitos tipoantidepressivos e ansiolíticos em roedores após administração sistêmica. O isolamento social aumenta comportamentos agressivos em camundongos, condição denominada agressão induzida pelo isolamento social ou agressão territorial. Drogas ansiolíticas e antidepressivas podem atenuar comportamentos agressivos. Desse modo, o objetivo do presente trabalho foi avaliar se o CBD atenuaria comportamentos agressivos induzidos pelo isolamento social
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Books on the topic "Recepto 5-HT1A"

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Jin, Hui. Cloning of the human 5-HT1B receptor. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Roldan, Alma. An investigation of the impact of 5-HT1B receptor activation on ethanol self-administration behaviour. National Library of Canada, 2003.

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J, Rodger R., and Cooper S. J, eds. 5-HT1A agonists, 5-HT3 antagonists and benzodiazepines: Their comparative behavioural pharmacology. Wiley, 1991.

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Walsh, Richard A. Always Worth a Second Look. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0031.

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The serotonin syndrome is a toxic syndrome resulting from excessive stimulation of central 5-HT1A and 5-HT2A receptors. This is most commonly an iatrogenic syndrome, which in its most severe form can be fatal. It is more common for milder forms to present, and there is increasing recognition of serotoninergic drugs that can give rise to serotonin syndrome when used in combination. It is essential for physicians to be familiar with the clinical features of serotonin toxicity and similar syndromes discussed in this chapter that are marked by altered awareness, autonomic instability, changes in m
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Book chapters on the topic "Recepto 5-HT1A"

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Bloch, Michael H., Michael H. Bloch, Mark A. Geyer, et al. "5-HT1A Receptor." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1356.

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De Vry, J., T. Glaser, T. Schuurman, R. Schreiber, and J. Traber. "5-HT1A Receptors in Anxiety." In New Concepts in Anxiety. Macmillan Education UK, 1991. http://dx.doi.org/10.1007/978-1-349-11847-2_7.

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Fozard, J. R., A. K. Mir, and A. G. Ramage. "5-HT1A Receptors and Cardiovascular Control." In Serotonin. Palgrave Macmillan UK, 1989. http://dx.doi.org/10.1007/978-1-349-10114-6_19.

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Levine, Louise R., and William Z. Potter. "The 5-HT1A Receptor: an unkept promise?" In Anxiolytics. Birkhäuser Basel, 2000. http://dx.doi.org/10.1007/978-3-0348-8470-9_7.

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Peruche, B., F. Ausmeier, C. Backhauss, J. Nuglisch, J. H. M. Prehn, and J. Krieglstein. "Neuroprotective Effects of 5-HT1A Receptor Agonists." In Cerebral Ischemia and Basic Mechanisms. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78151-3_22.

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De Vry, J., T. Glaser, and J. Traber. "5-HT1A Receptor Partial Agonists as Anxiolytics." In Serotonin. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1912-9_68.

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Fillion, Gilles, Pascal Barone, Catherine Fayolle, and Marie-Paule Fillion. "Existence of a 5-HT1 Binding Site Different of 5-HT1A, 5-HT1B and 5-HT1C Subtypes and Coupled to a High Affinity Adenylate Cyclase Activation: A Functional 5-HT Receptor Involved in Neuromodulation ?" In Neuroreceptors and Signal Transduction. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4757-5971-6_25.

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Hamon, M., S. El Mestikawy, M. B. Emerit, and H. Gozlan. "Molecular Structure of the Central 5-HT1A Receptor." In Serotonin. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1912-9_3.

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Hamon, M. "The Main Features of Central 5-HT1A Receptors." In Serotoninergic Neurons and 5-HT Receptors in the CNS. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-60921-3_9.

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Lesch, K. P., and M. Osterheider. "5-Hydroxytryptamine1A (5-HT1A) Receptor Responsivity in Anxiety Disorders and Depression." In New Concepts in Anxiety. Macmillan Education UK, 1991. http://dx.doi.org/10.1007/978-1-349-11847-2_8.

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Conference papers on the topic "Recepto 5-HT1A"

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Cocchi, M., M. C. Menziani, F. Fanelli та P. G. de Benedetti. "Theoretical QSAR and QSSR analyses of 5-HT1A serotonin and α1-adrenergic receptors ligands". У The first European conference on computational chemistry (E.C.C.C.1). AIP, 1995. http://dx.doi.org/10.1063/1.47824.

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Lin, Ling, Guoliang Liu, and Yinpo Zhang. "Research on the Effects of 5-HT1A Receptor in Hippocampal CA1 Region in the Cognition of PTSD Rats." In 2016 2nd International Conference on Materials Engineering and Information Technology Applications (MEITA 2016). Atlantis Press, 2017. http://dx.doi.org/10.2991/meita-16.2017.81.

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PAUWELS, Petrus J. "HUMAN 5-HT1D RECEPTORS: CLONING DISCOVERIES AND THEIR IMPACT ON HUMAN DRUG DESIGN." In IX World Congress of Psychiatry. WORLD SCIENTIFIC, 1994. http://dx.doi.org/10.1142/9789814440912_0026.

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Zou, Ying, Wei Wang, Wenyang Li, and Hongyu Jin. "The effect of chronic-intermittent-hypoxia on 5-HT1A receptors and GIRK-2 in dorsal raphe nucleus of rats." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2540.

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"Influence of 5-HT1A receptors overexpression in the hippocampus on behavior and the brain serotonin system of BTBR mice – the model of autism." In SYSTEMS BIOLOGY AND BIOINFORMATICS (SBB-2020). Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences., 2020. http://dx.doi.org/10.18699/sbb-2020-04.

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Demolle, D., E. J. Cragoe, and J. M. Boeynaems. "MECHANISMS INVOLVED IN 5-HT STIMULATION OF PROSTACYCLIN PRODUCTION BY BOVINE AORTIC SMOOTH MUSCLE CELLS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642841.

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Serotonin (5-HT) stimulates prostacyclin (PGI2) production by bovine aortic smooth muscle cells in culture via 5-HT2 receptors (1). These cells express a synthetic phenotype (2), whereas the majority of the smooth muscle cells in the media from adult arteries are in a contractile state. We have now shown that 5-HT (1-10 μM) also stimulates PGI2 production by a preparation of contractile smooth muscle cells : explants from bovine aortic media cultured for short periods. This effect is independent from 5-HT2 receptors : it is only partially inhibited (±30%) by ketan-serin (a selective and potent
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Reports on the topic "Recepto 5-HT1A"

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Yan, Qingshan. Ethanol and Mesolimbic Serotonin/Dopamine Interactions Via 5-HT1B Receptors. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada455523.

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Halker Singh, Rashmi B., Juliana H. VanderPluym, Allison S. Morrow, et al. Acute Treatments for Episodic Migraine. Agency for Healthcare Research and Quality (AHRQ), 2020. http://dx.doi.org/10.23970/ahrqepccer239.

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Objectives. To evaluate the effectiveness and comparative effectiveness of pharmacologic and nonpharmacologic therapies for the acute treatment of episodic migraine in adults. Data sources. MEDLINE®, Embase®, Cochrane Central Registrar of Controlled Trials, Cochrane Database of Systematic Reviews, PsycINFO®, Scopus, and various grey literature sources from database inception to July 24, 2020. Comparative effectiveness evidence about triptans and nonsteroidal anti-inflammatory drugs (NSAIDs) was extracted from existing systematic reviews. Review methods. We included randomized controlled trials
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