Relevant bibliographies by topics / Receptores EP

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Receptores EP'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Receptores EP"

1

Cipollone, Francesco, and Donato Santovito. "EP Receptors and Coxibs." Circulation Research 113, no. 2 (2013): 91–93. http://dx.doi.org/10.1161/circresaha.113.301616.

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Sawyer, ST, SB Krantz, and K. Sawada. "Receptors for erythropoietin in mouse and human erythroid cells and placenta." Blood 74, no. 1 (1989): 103–9. http://dx.doi.org/10.1182/blood.v74.1.103.103.

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Abstract High and lower affinity receptors for erythropoietin (EP) were initially identified on a very pure population of EP-responsive erythroblasts obtained from the spleens of mice infected with anemia strain of Friend virus (FVA). The structure of the receptor for EP in these cells was determined to be proteins of 100 and 85 Kd by cross- linking 125I-EP. In this investigation, studies on the receptors for EP were extended to other mouse erythroid cells and human erythroid cells as well as to the placentas of mice and rats. Only lower affinity receptors for EP were detected on erythroblasts
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Sawyer, ST, SB Krantz, and K. Sawada. "Receptors for erythropoietin in mouse and human erythroid cells and placenta." Blood 74, no. 1 (1989): 103–9. http://dx.doi.org/10.1182/blood.v74.1.103.bloodjournal741103.

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High and lower affinity receptors for erythropoietin (EP) were initially identified on a very pure population of EP-responsive erythroblasts obtained from the spleens of mice infected with anemia strain of Friend virus (FVA). The structure of the receptor for EP in these cells was determined to be proteins of 100 and 85 Kd by cross- linking 125I-EP. In this investigation, studies on the receptors for EP were extended to other mouse erythroid cells and human erythroid cells as well as to the placentas of mice and rats. Only lower affinity receptors for EP were detected on erythroblasts purified
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Broudy, VC, N. Lin, M. Brice, B. Nakamoto, and T. Papayannopoulou. "Erythropoietin receptor characteristics on primary human erythroid cells." Blood 77, no. 12 (1991): 2583–90. http://dx.doi.org/10.1182/blood.v77.12.2583.2583.

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Abstract Erythropoietin (EP) exerts its effects on erythropoiesis by binding to a cell surface receptor. We examined EP receptor expression during normal human erythroid differentiation and maturation from the burst- forming unit-erythroid (BFU-E) to the reticulocyte level. In contrast to previous studies, we assessed EP receptor number and affinity in erythroid precursors immunologically purified from fresh bone marrow aspirates or fetal liver samples and in reticulocytes purified from peripheral blood. EP receptors were quantitated by equilibrium binding experiments with 125I EP. We found th
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Broudy, VC, N. Lin, M. Brice, B. Nakamoto, and T. Papayannopoulou. "Erythropoietin receptor characteristics on primary human erythroid cells." Blood 77, no. 12 (1991): 2583–90. http://dx.doi.org/10.1182/blood.v77.12.2583.bloodjournal77122583.

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Erythropoietin (EP) exerts its effects on erythropoiesis by binding to a cell surface receptor. We examined EP receptor expression during normal human erythroid differentiation and maturation from the burst- forming unit-erythroid (BFU-E) to the reticulocyte level. In contrast to previous studies, we assessed EP receptor number and affinity in erythroid precursors immunologically purified from fresh bone marrow aspirates or fetal liver samples and in reticulocytes purified from peripheral blood. EP receptors were quantitated by equilibrium binding experiments with 125I EP. We found that purifi
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Fang, K. M., W. H. Shu, H. C. Chang, J. J. Wang, and O. T. Mak. "Study of prostaglandin receptors in mitochondria on apoptosis of human lung carcinoma cell line A549." Biochemical Society Transactions 32, no. 6 (2004): 1078–80. http://dx.doi.org/10.1042/bst0321078.

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PGs (prostaglandins) are synthesized through the cyclo-oxygenase (COX-1 and -2) pathway in a variety of cells in response to various physiological stimuli. All cells require at least one pathway for apoptosis, and mitochondrial play a central role in regulation of apoptosis. In a previous study, incubation of A549 cells with NS-398 (a COX-2-specific inhibitor) induced apoptosis and inhibited cell proliferation, and the concentrations of different PGs between various cellular compartments were found to be changed. To determine whether PG receptors are involved in this regulation, Western-blot a
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Wognum, AW, PM Lansdorp, RK Humphries, and G. Krystal. "Detection and isolation of the erythropoietin receptor using biotinylated erythropoietin." Blood 76, no. 4 (1990): 697–705. http://dx.doi.org/10.1182/blood.v76.4.697.697.

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Abstract Procedures have been developed to label human erythropoietin (Ep) with biotin to detect and isolate the Ep-receptor. The labeling method used the abundant carbohydrate groups on Ep and resulted in biologically active biotin-Ep (b-Ep) containing 8 to 10 biotins per Ep molecule. Specific binding of b-Ep to cells from spleens of mice made anemic by phenylhydrazine injections was demonstrated using 125I-labeled streptavidin. B-Ep, together with fluorescently tagged streptavidin, was found to specifically detect Ep-receptor-bearing cells by flow cytometry. This was demonstrated in several
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Wognum, AW, PM Lansdorp, RK Humphries, and G. Krystal. "Detection and isolation of the erythropoietin receptor using biotinylated erythropoietin." Blood 76, no. 4 (1990): 697–705. http://dx.doi.org/10.1182/blood.v76.4.697.bloodjournal764697.

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Procedures have been developed to label human erythropoietin (Ep) with biotin to detect and isolate the Ep-receptor. The labeling method used the abundant carbohydrate groups on Ep and resulted in biologically active biotin-Ep (b-Ep) containing 8 to 10 biotins per Ep molecule. Specific binding of b-Ep to cells from spleens of mice made anemic by phenylhydrazine injections was demonstrated using 125I-labeled streptavidin. B-Ep, together with fluorescently tagged streptavidin, was found to specifically detect Ep-receptor-bearing cells by flow cytometry. This was demonstrated in several ways. Fir
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Audoly, Laurent P., Stephen L. Tilley, Jennifer Goulet, et al. "Identification of specific EP receptors responsible for the hemodynamic effects of PGE2." American Journal of Physiology-Heart and Circulatory Physiology 277, no. 3 (1999): H924—H930. http://dx.doi.org/10.1152/ajpheart.1999.277.3.h924.

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To identify the E-prostanoid (EP) receptors that mediate the hemodynamic actions of PGE2, we studied acute vascular responses to infusions of PGE2using lines of mice in which each of four EP receptors (EP1 through EP4) have been disrupted by gene targeting. In mixed groups of males and females, vasodepressor responses after infusions of PGE2were significantly diminished in the EP2 −/− and EP4 −/− lines but not in the EP1 −/− or EP3 −/− lines. Because the actions of other hormonal systems that regulate blood pressure differ between sexes, we compared the roles of individual EP receptors in male
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Kim, Soon Ok, Siabhon M. Harris, and Diane M. Duffy. "Prostaglandin E2 (EP) Receptors Mediate PGE2-Specific Events in Ovulation and Luteinization Within Primate Ovarian Follicles." Endocrinology 155, no. 4 (2014): 1466–75. http://dx.doi.org/10.1210/en.2013-2096.

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Prostaglandin E2 (PGE2) is a key mediator of ovulation. All 4 PGE2 receptors (EP receptors) are expressed in the primate follicle, but the specific role of each EP receptor in ovulatory events is poorly understood. To examine the ovulatory events mediated via these EP receptors, preovulatory monkey follicles were injected with vehicle, the PG synthesis inhibitor indomethacin, or indomethacin plus PGE2. An ovulatory dose of human chorionic gonadotropin was administered; the injected ovary was collected 48 hours later and serially sectioned. Vehicle-injected follicles showed normal ovulatory eve
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Dissertations / Theses on the topic "Receptores EP"

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Gomes, Renata Nascimento. "Análise do perfil dos prostanoides e do seu papel no controle da migração celular em glioblastoma." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-23012017-094918/.

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O glioblastoma (GBM) é o tumor mais frequente do sistema nervoso central com um alto grau de malignidade e um prognóstico desfavorável. Apesar dos avanços nas técnicas cirúrgicas e de radioterapia e/ou quimioterapia, não há tratamento eficiente disponível para o GBM. Os prostanoide são derivados do ácido araquidônico e estão envolvidas com vários processos do desenvolvimento e progressão do câncer. O objetivo deste estudo foi analisar in vitro o perfil de diferentes prostanoides nas linhagens de GBM. Além de analisar o papel dos prostanoides e dos receptores na migração celular de GBM. Os resu
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Reschke, Cristina Ruedell. "RECEPTORES EP1 E EP3 MODULAM AS CRISES EPILÉPTICAS INDUZIDAS POR PENTILENOTETRAZOL E ÁCIDO CAÍNICO EM CAMUNDONGOS." Universidade Federal de Santa Maria, 2013. http://repositorio.ufsm.br/handle/1/3852.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>Epilepsy is one of the most common neurologic disorders. It has been suggested that seizures may be facilitaded by inflammation. PGE2 is one of the most important inflammatory mediators, and facilitates pentylenetetrazol (PTZ)-induced seizures by stimulating EP1 and EP3 receptors. However, up to the present moment, no study has investigated whether EP1 and EP3 receptors blocking attenuate seizures induced by convulsants other than PTZ. It is also unknown whether Na+,K+-ATPase activity alterations are involved in such an effect. The
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Ratzlaff, Viviane. "Padronização e validação de um novo modelo de febre induzida pela injeção intratecal de prostaglandina e2 em ratos jovens." Universidade Federal de Santa Maria, 2006. http://repositorio.ufsm.br/handle/1/11148.

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The fever response, besides being part of host defense response to infection or inflammation, is associated with discomfort and anxiety and may constitute a risk for febrile seizures in children. Therefore, antipyretic therapy is routinely prescribed for febrile patients. The animal models of fever using the systemic injection of lipopolysaccharide (LPS) and Baker yeast, described in the literature, are suitable for screening of novel antipyretics, but they do not provide information regarding the mechanism of action of these compounds. Therefore, the present study aimed to describe and valida
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Milne, Stuart Angus. "Characterisation of the inhibitory EP-receptors present in human monocytes." Thesis, University of Edinburgh, 1998. http://hdl.handle.net/1842/22496.

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Rahal, Sherine S. "The role of prostaglandin EP receptors in a model of glomerulonephritis." Thesis, University of Ottawa (Canada), 2004. http://hdl.handle.net/10393/26750.

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Prostaglandin E2 (PGE2) interacts with four E-Prostanoid (EP) receptor subtypes---designated EP1--4 . In glomerulonephritis (GN), a renal inflammatory disease, inhibition of enhanced renal PGE2 synthesis by nonsteroidal-anti-inflammatory drugs (NSAIDs), results in both beneficial anti-proteinuric effects and deleterious side effects on renal blood flow (RBF) and Na+ homeostasis, implying that one or more EP subtypes may mediate these actions. We set out to investigate the role of the EP1 receptor in GN since it localizes to the collecting duct, where it may regulate Na+ homeostasis, in podocyt
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Slipetz, Deborah M. "The prostaglandin E₂ EP₄ receptor : the elucidation of agonist mediated receptor regulation and a novel therapeutic role for an EP₄ agonist in allergic lung inflammation." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111868.

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Prostaglandin (PG) E2 is involved in a number of physiological and path op hysiologicaI events in many tissues. PGE2 mediates its actions through interaction with specific plasma-membrane G-protein coupled receptors (GPCR's). There are four subtypes of PGE2 receptors: EP1, EP2, EP3 and EP4, classified by their different pharmacological profiles and signal transduction pathways. EP4 agonists have potential therapeutic benefit in diseases such as osteoporosis (EP4 mediates the bone anabolic actions of PGE 2), but possibly in other areas such as asthma. A characteristic feature of GPCR's is their
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Li, Sau Wei. "Pharmacological characterization of human neutrophil prostanoid EP receptors : modulation of neutrophil function." Thesis, University of Bath, 1995. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307121.

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Arulkumaran, Shankari. "The roles of prostanoid EP receptors in the control of contractions of human myometrium." Thesis, Imperial College London, 2012. http://hdl.handle.net/10044/1/9305.

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The primary function of the uterus during pregnancy is to harbour the growing fetus in a quiescent environment. Upon maturation of the fetus, the uterus generates forceful contractions during labour. Prostaglandins are central in the parturition process. PGE2 in particular, is produced in large quantities by fetal membranes and possible roles include cervical ripening and the stimulation of uterine contractions. PGE2 exhibits a wide spectrum of physiological actions depending on the distribution and subtypes of EP receptors. EP1 and EP3 mediate contractions, but there is no consensus about the
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Akram, Asha. "Examining the potential of PGE₂ receptor EP₄ as a neuroprotective target following ischaemic injury." Thesis, University of Leicester, 2013. http://hdl.handle.net/2381/28245.

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Ischaemic stroke instigates a series of pathological mechanisms which contribute to injury. Despite significant research in this field successful clinical treatment is limited. This highlights the need to identify novel therapeutic targets in order to assess whether clinical investigation is warranted. The enzyme cyclooxygenase-2 (COX-2) is a key contributor to inflammatory injury following stroke. Upregulation of this enzyme results in increased prostanoid synthesis, which mediate many physiological and pathological functions. Prostaglandin E₂ (PGE₂) is a key mediator in inflammation and acti
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Katsuyama, Masato. "A Study on the Structure and Function of the Prostaglandin E Receptor Subtype EP[2]." 京都大学 (Kyoto University), 1998. http://hdl.handle.net/2433/73162.

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Book chapters on the topic "Receptores EP"

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Takeuchi, Koji, Shinichi Kato, Yusaku Komoike, Yoshihiro Ogawa, and Masanori Takeeda. "Gastric Cytoprotection by Prostaglandin E2 — Relation to EP Receptor Subtypes —." In Mechanisms and Consequences of Proton Transport. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0971-4_21.

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Zeng, Li, Songzhu An, and Edward J. Goetzl. "EP Receptor Subtype-Dependence of Regulation of Immune Cellular Functions by Prostaglandin E2." In Frontiers in Bioactive Lipids. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5875-0_23.

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Takeuchi, K., S. Kato, and A. Tanaka. "Gastrointestinal Protective Action of Prostaglandin E2 and EP Receptor Subtypes." In Frontiers of Gastrointestinal Research. KARGER, 2002. http://dx.doi.org/10.1159/000060970.

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Jones, Robert L. "EP Prostanoid Receptors." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.60088-1.

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Jones, Robert L. "EP-1 Prostanoid Receptor." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.60089-3.

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Jones, Robert L. "EP-2 Prostanoid Receptor." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.60090-x.

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Jones, Robert L. "EP-3 Prostanoid Receptor." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.60336-8.

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Jones, Robert L. "EP-4 Prostanoid Receptor." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.60337-x.

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Takeuchi, Koji. "Prostaglandin EP Receptors and Their Roles in Mucosal Protection and Ulcer Healing in the Gastrointestinal Tract." In Advances in Clinical Chemistry. Elsevier, 2010. http://dx.doi.org/10.1016/s0065-2423(10)51005-9.

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Conference papers on the topic "Receptores EP"

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Morelli, H., D. Villalba, S. D. Shah, A. P. Nayak, and R. B. Penn. "Airway Smooth Muscle Contraction: Distinct EP Receptors, Different Outcomes." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1253.

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Holt, Dawn M., Xinrong Ma, Namita Kundu, and Amy M. Fulton. "Abstract 5308: Targeting the Cox2 pathway prostaglandin EP receptors to regulate NK cell functions." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5308.

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Nayak, A. P., D. Villalba, J. V. Michael, S. W. Twardus, S. S. An, and R. B. Penn. "Differential Regulation of Airway Smooth Muscle Function by E-Prostanoid (EP) Receptor Subtypes." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4283.

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Van Geffen, Chiel, Sandra Beer-Hammera, Bernd Nürnberg, and Saeed Kolahian. "Generation and activation of myeloid derived suppressor cells using prostaglandin E2 and EP receptor agonists in vitro." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3871.

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