Academic literature on the topic 'Receptors, adrenergic, beta'

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Journal articles on the topic "Receptors, adrenergic, beta"

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Brechet, S., P. Plaisancie, V. Dumoulin, JA Chayvialle, JC Cuber, and J. Claustre. "Involvement of beta1- and beta2- but not beta3-adrenoceptor activation in adrenergic PYY secretion from the isolated colon." Journal of Endocrinology 168, no. 1 (2001): 177–83. http://dx.doi.org/10.1677/joe.0.1680177.

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The secretion of PYY by endocrine L cells of the terminal gut is under the control of nutrients, the autonomic nervous system and hormones. Catecholamines, and the non-specific beta-adrenergic agonist isoproterenol induce PYY secretion from rat isolated colon or ileum. Because beta3-adrenergic receptors now appear to mediate many of the effects of catecholamines in the gastrointestinal tract, we investigated the involvement of beta1-, beta2-, and beta3-adrenoceptor stimulation in PYY secretion from the isolated, vascularly perfused rat colon. Infusion of 10(-6) M isoproterenol induced a transi
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Gesek, F. A., and K. E. White. "Molecular and functional identification of beta-adrenergic receptors in distal convoluted tubule cells." American Journal of Physiology-Renal Physiology 272, no. 6 (1997): F712—F720. http://dx.doi.org/10.1152/ajprenal.1997.272.6.f712.

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Renal nerve stimulation or circulating catecholamines activate the beta-adrenergic receptors that mediate direct effects on tubular transport. Three subtypes of beta-adrenergic receptors have been characterized: beta 1, beta 2, and beta 3. beta-Adrenergic-receptor effects on Na+ and Ca2+ transport in distal convoluted tubules (DCT) have not been established. The focus of this study was to 1) identify the subtypes of beta-adrenergic receptors in DCT cells and 2) examine functional responses to beta-receptor activation on adenosine 3',5'-cyclic monophosphate (cAMP) formation and Na+ and Ca2+ ent
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Scarpace, P. J., L. A. Baresi, and J. E. Morley. "Modulation of receptors and adenylate cyclase activity during sucrose feeding, food deprivation, and cold exposure." American Journal of Physiology-Endocrinology and Metabolism 253, no. 6 (1987): E629—E635. http://dx.doi.org/10.1152/ajpendo.1987.253.6.e629.

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Thermogenesis in brown adipose tissue (BAT) serves as a regulator of body temperature and weight maintenance. Thermogenesis can be stimulated by catecholamine activation of adenylate cyclase through the beta-adrenergic receptor. To investigate the effects of sucrose feeding, food deprivation, and cold exposure on the beta-adrenergic pathway, adenylate cyclase activity and beta-adrenergic receptors were assessed in rat BAT after 2 wk of sucrose feeding, 2 days of food deprivation, or 2 days of cold exposure. beta-Adrenergic receptors were identified in BAT using [125I]iodocyanopindolol. Binding
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Kiely, J., J. R. Hadcock, S. W. Bahouth та C. C. Malbon. "Glucocorticoids down-regulate β1-adrenergic-receptor expression by suppressing transcription of the receptor gene". Biochemical Journal 302, № 2 (1994): 397–403. http://dx.doi.org/10.1042/bj3020397.

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The expression of beta 2-adrenergic receptors is up-regulated by glucocorticoids. In contrast, beta 1-adrenergic receptors display glucocorticoid-induced down-regulation. In rat C6 glioma cells, which express both of these subtypes of beta-adrenergic receptors, the synthetic glucocorticoid dexamethasone stimulates no change in the total beta-adrenergic receptor content, but rather shifts the beta 1:beta 2 ratio from 80:20 to 50:50. Radioligand binding and immunoblotting demonstrate a sharp decline in beta 1-adrenergic receptor expression. Metabolic labelling of cells with [35S]-methionine in t
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Bahouth, S. W., та C. C. Malbon. "Human β-adrenergic receptors. Simultaneous purification of β1- and β2-adrenergic-receptor peptides". Biochemical Journal 248, № 2 (1987): 557–66. http://dx.doi.org/10.1042/bj2480557.

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Beta-Adrenergic receptors from basal membranes of human placenta were purified from digitonin extracts by sequential rounds of affinity chromatography, hydrophobic chromatography, ion-exchange chromatography and steric-exclusion h.p.l.c. Basal membranes display both beta 1- and beta 2-adrenergic receptors, in the ratio 65:35. Affinity chromatography, hydrophobic chromatography on heptylamine-Sepharose and ion-exchange chromatography on DEAE-Sephacel removed most of the contaminating proteins, and final purification of the receptor to apparent homogeneity was achieved by steric-exclusion h.p.l.
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Feldman, R. D., G. W. Hunninghake, and W. L. McArdle. "Beta-adrenergic-receptor-mediated suppression of interleukin 2 receptors in human lymphocytes." Journal of Immunology 139, no. 10 (1987): 3355–59. http://dx.doi.org/10.4049/jimmunol.139.10.3355.

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Abstract Adrenergic receptor agonists are known to attenuate the proliferative response of human lymphocytes after activation; however, their mechanism of action is unknown. Since expression of interleukin 2 (IL-2) receptors is a prerequisite for proliferation, the effect of beta-adrenergic receptor agonists on lymphocyte IL-2 receptors was studied on both mitogen-stimulated lymphocytes and IL-2-dependent T lymphocyte cell lines. In both cell types the beta-adrenergic receptor agonist isoproterenol blocked the expression of IL-2 receptors, as determined with the IL-2 receptor anti-TAC antibody
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Nahorski, S. R. "The Beta-adrenergic receptors." Trends in Pharmacological Sciences 13 (January 1992): 296–97. http://dx.doi.org/10.1016/0165-6147(92)90089-o.

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Maisel, A. S., H. J. Motulsky, M. G. Ziegler, and P. A. Insel. "Ischemia- and agonist-induced changes in alpha- and beta-adrenergic receptor traffic in guinea pig hearts." American Journal of Physiology-Heart and Circulatory Physiology 253, no. 5 (1987): H1159—H1166. http://dx.doi.org/10.1152/ajpheart.1987.253.5.h1159.

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We have used radioligand binding techniques and subcellular fractionation to assess whether changes in expression of myocardial alpha 1- and beta-adrenergic receptors are mediated by a redistribution of receptors between various membrane fractions. Three fractions were prepared from the left ventricles of guinea pigs that underwent either 1 h of ischemia or injection of epinephrine (0.25 mg/kg ip): a crude membrane, a purified sarcolemma, and a light vesicle fraction. In control animals alpha 1-adrenergic receptors ([3H]prazosin binding) in light vesicles was only 25% of the total alpha 1-rece
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Wang, H. Y., M. Berrios та C. C. Malbon. "Localization of β-adrenergic receptors in A431 cells in situ. Effect of chronic exposure to agonist". Biochemical Journal 263, № 2 (1989): 533–38. http://dx.doi.org/10.1042/bj2630533.

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The status of beta-adrenergic receptors was investigated in A431 cells exposed to chronic stimulation by the beta-adrenergic agonist, (-)-isoproterenol. Specific binding of beta-adrenergic antagonist (-)-[125I]iodocyanopindolol declined to 60-80% below control values within 12 h of agonist treatment. This decline in ligand binding was also observed in high-speed membrane fractions prepared from agonist-treated cells. Immunoblots probed with anti-receptor antibodies revealed both that beta-adrenergic receptors from untreated and treated cells migrated as 65,000-Mr peptides and that the cellular
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Feldman, R. D., J. P. Christy, S. T. Paul, and D. G. Harrison. "Beta-adrenergic receptors on canine coronary collateral vessels: characterization and function." American Journal of Physiology-Heart and Circulatory Physiology 257, no. 5 (1989): H1634—H1639. http://dx.doi.org/10.1152/ajpheart.1989.257.5.h1634.

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The present studies were performed to examine the role of beta-adrenergic receptors in modulating smooth muscle tone in mature coronary collaterals. To examine the beta-adrenergic receptor population present, radioligand binding-cover slip autoradiographic studies were performed on sections of native canine coronary vessels and sections of coronary collaterals developed after placement of Ameroid constrictors. Specific binding of the nonselective beta-adrenergic antagonist [125I]iodopindolol to vascular smooth muscle in segments of both collaterals and native coronary arteries was saturable an
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Dissertations / Theses on the topic "Receptors, adrenergic, beta"

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Vela, Jose David. "Beta adrenergic modulation of peripheral nociceptors a dissertation /." San Antonio : UTHSC, 2008. http://proquest.umi.com.libproxy.uthscsa.edu/pqdweb?did=1588778261&sid=1&Fmt=2&clientId=70986&RQT=309&VName=PQD.

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Stern, Ligia R. "Regulation of beta-adrenergic receptors in cultured lung cells." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74250.

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The effects of glucocorticoids and agents affecting membrane phospholipid metabolism on $ beta$-adrenergic receptors ($ beta$AR) have been studied in A549 cultured human lung tumor cells and in IM-9 cultured human lymphocytes. Various glucocorticoids increased the density of $ beta$AR in A549 lung cells but not in IM-9 lymphocytes, although they did increase insulin receptor density in IM-9 lymphocytes. In contrast, bee venom phospholipase A$ sb2$ (0.05-0.2 units/ml), but not phospholipases C or D., downregulated $ beta$AR in IM-9 lymphocytes, but did not affect $ beta$AR in A549 lung cells. U
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Potter, Paul Charles. "In Vitro studies of factors which may influence ligand binding, function, immunogenicity and genetic regulation of the Beta-2 adrenergic receptor in asthma." Doctoral thesis, University of Cape Town, 1991. http://hdl.handle.net/11427/26483.

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This thesis records a series of experiments conducted to gain further insight into factors which influence the expression, ligand binding and functional activity of the beta-2 adrenergic receptor. These studies were prompted by previous reports that the postulated beta-2 adrenergic receptor abnormality in allergic asthma could be induced, induced by autoantibodies. I established and optimised beta-2 adrenergic receptor ligand binding and functional assays in guinea pig lung membranes and then conducted an original study of beta adrenergic receptor expression in the guinea pig foetal lung. I fo
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Zolty, Ronald. "Beta 1 and Alpha 2C adrenergic receptor polymorphisms and response to beta blockers in heart failure patients /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2007.

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Thesis (Ph.D. in Clinical Science) -- University of Colorado Denver, 2007.<br>Typescript. Includes bibliographical references (leaves 130-142). Free to UCD affiliates. Online version available via ProQuest Digital Dissertations;
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Crumbie, Hayley Elizabeth. "Beta-adrenergic receptor signalling and excitation-contraction coupling in the heart : impact of circadian rhythms and beta-3 receptors." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37500.

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A time-of-day variation in myocardial contraction and its response to sympathetic stimulation of β-adrenoceptors (b-ADR) exists, which is reflected by time-of-day variations in intracellular calcium [Ca2+]i regulation and electrical activity. There are three isoforms of β-ADR (β1/β2/β3) and the functional outcome of each receptor differs. β1/β2-ADRs result in positive inotropism, whereas β3-ADR induces a negative inotropic effect, associated with nitric oxide (NO) signalling. The aims of this thesis were to determine the role of the β3-ADR in the time-of-day variation in the response to sympat
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McLean, Alison Jane. "Ligand regulation of #beta#←1- and #beta#←2- adrenergic receptors and their GFP-tagged forms." Thesis, University of Glasgow, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323436.

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Salinas, Cristian Andres. "ROBUST EXPERIMENTAL DESIGN FOR ESTIMATING MYOCARDIAL BETA ADRENERGIC RECEPTOR CONCENTRATION USING POSITRON EMISSION TOMOGRAPHY." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1144088920.

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Ekpe, Enefiok David. "Beta-adrenergic receptors in tissues of sheep, effects of temperature and plane of nutrition." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ29032.pdf.

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Salamanca, Melissa, and Melissa Salamanca. "Beta₃-adrenergic receptors in the heart: Normal functions and potential roles in heart failure." Thesis, The University of Arizona, 2016. http://hdl.handle.net/10150/621455.

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Heart Failure is the state in which the heart is unable to pump enough blood to meet the needs of the body, resulting from events that progressively damage the myocardium and/or its ability to contract normally. Several compensatory mechanisms allow for short-term maintenance of adequate delivery of blood to the body. Progression from the initial events to heart failure is caused by the accumulation of pathologic changes and their effects, many of which result from chronic activation of these compensatory mechanisms. One of these mechanisms is the activation of the neurohormonal β-adrenergic
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Walker, Robert J. "Inflammatory cytokines modulated by beta adrenergic receptors on retinal Muller cells in diabetic retinopathy /." Available to subscribers only, 2007. http://proquest.umi.com/pqdweb?did=1467888651&sid=24&Fmt=2&clientId=1509&RQT=309&VName=PQD.

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Books on the topic "Receptors, adrenergic, beta"

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Perkins, John P., ed. The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3.

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Advances in adrenergic receptor biology. Elsevier, 2011.

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1946-, Frishman William H., ed. Beta₃-adrenergic agonism: A new concept in human pharmacotherapy. Futura, 1995.

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Cruickshank, J. M. Beta-blockers in clinical practice. Churchill Livingstone, 1988.

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Prichard, Brian N. C. 1932-, ed. Beta-blockers in clinical practice. Churchill Livingstone, 1987.

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Owens, Helen. The effect of beta-adrenergic receptor antagonists on the temporal accommodative response. Aston University. Department of Vision Sciences, 1991.

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1944-, Costello J. F., and Mann Ronald D. 1928-, eds. Beta agonists in the treatment of asthma: The proceedings. Parthenon Pub. Group, 1992.

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Richard, Beasley, and Pearce Neil, eds. The Role of beta receptor agonist therapy in asthma mortality. CRC Press, 1993.

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Drugs looking for diseases: Innovative drug research and the development of the beta blockers and the calcium antagonists. Kluwer Academic Publishers, 1991.

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Perkins, John P. Beta-Adrenergic Receptors. Humana Press, 2012.

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Book chapters on the topic "Receptors, adrenergic, beta"

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Stadel, Jeffrey M., and Robert J. Lefkowitz. "Beta-Adrenergic Receptors." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_1.

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Makaritsis, Konstantinos, and Filippos Triposkiadis. "Beta Adrenergic Receptors." In Introduction to Translational Cardiovascular Research. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-08798-6_5.

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Hill, Keith, Tom Baranowski, Walter Schmidt, et al. "Beta-Adrenergic Receptors." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2142.

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Caron, Marc G., and Robert J. Lefkowitz. "Structure-Function Relationships." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_2.

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Perkins, John P., William P. Hausdorff, and Robert J. Lefkowitz. "Mechanisms of Ligand-Induced Desensitization of Beta-Adrenergic Receptors." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_3.

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Ross, Elliott M. "Reconstitution of the Beta-Adrenergic Receptor and Its Biochemical Functions." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_4.

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Malbon, Craig C., Cary P. Moxham, and Harvey J. Brandwein. "Antibodies to Beta-Adrenergic Receptors." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_5.

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Wolfe, Barry B. "Autoradiographic Studies of Beta-Adrenergic Receptors." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_6.

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Insel, Paul A. "Beta-Adrenergic Receptors in Pathophysiologic States and in Clinical Medicine." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_7.

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Stiles, Gary L. "Drug and Hormonal Regulation of the Beta-Adrenergic Receptor—Adenylate Cyclase System." In The Beta-Adrenergic Receptors. Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_8.

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Conference papers on the topic "Receptors, adrenergic, beta"

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Matuskova, Lenka, Barbora Czippelova, Zuzana Turianikova, et al. "Beta-adrenergic receptors gene polymorphisms effects on cardiovascular control." In 2022 12th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2022. http://dx.doi.org/10.1109/esgco55423.2022.9931348.

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Croce, Anna C., V. Mares, V. Lisa, D. Krajci, and Giovanni F. Bottiroli. "Modulation of Photofrin II accumulation in C6 glioma cells by stimulation of beta-adrenergic receptors." In BiOS Europe '97, edited by Kristian Berg, Benjamin Ehrenberg, Zvi Malik, Johan Moan, and Abraham Katzir. SPIE, 1997. http://dx.doi.org/10.1117/12.297827.

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Rousseau, Benedicte, Sengottuvelan Murugan, Ajay Palagani, Ananya Tirupathur, Nupur Kadam, and Dipak K. Sarkar. "Abstract 6067: Beta-2 adrenergic receptors role in tumor aggressiveness of triple negative breast cancer." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-6067.

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Alves, Mateus Feitosa, Marcus Scotti, Luciana Scotti, Sócrates dos Santos, and Margareth de Fátima Diniz. "Alkaloids Menispermaceae family: a new source of compounds selective for beta-adrenergic receptors." In MOL2NET 2016, International Conference on Multidisciplinary Sciences, 2nd edition. MDPI, 2016. http://dx.doi.org/10.3390/mol2net-02-14004.

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Rousseau, Benedicte, Ananya Tirupathur, Sengottuvelan Murugan, and Dipak K. Sarkar. "Abstract 3440: Beta-2 adrenergic receptors role in tumor aggressiveness of MDA-MB-231 breast cancer cells." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-3440.

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Rousseau, Benedicte, Ananya Tirupathur, Sengottuvelan Murugan, and Dipak K. Sarkar. "Abstract 3440: Beta-2 adrenergic receptors role in tumor aggressiveness of MDA-MB-231 breast cancer cells." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-3440.

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Romano, Veronica, Domenico Cozzolino, Giorgio Zinno, Stefano Palermi, and Domiziano Tarantino. "The effects of beta (2)-adrenergic receptors activation on the cardiovascular system and on the skeletal muscle: A narrative review." In Journal of Human Sport and Exercise - 2021 - Winter Conferences of Sports Science. Universidad de Alicante, 2021. http://dx.doi.org/10.14198/jhse.2021.16.proc3.53.

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Cano, E. J., J. A. Gonzalez, J. L. Perez-Lara, F. Casado, A. Zhang, and C. Zeana. "Beta-Adrenergic Receptor Blockers in E. coli Bacteremia: Severity of Presentation and Outcomes." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6499.

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Carroll, Christopher L., Craig Schramm, Indira Sadhu, and Aaron R. Zucker. "Beta-Adrenergic Receptor Haplotype Associated With Recurrent Near Fatal Asthma Exacerbations In Children." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2509.

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Khan, Y., SJ Fowler, and G. Tavernier. "S140 Beta-2-adrenergic receptor gene polymorphisms in severe asthma: a systematic review." In British Thoracic Society Winter Meeting, Wednesday 17 to Friday 19 February 2021, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2021. http://dx.doi.org/10.1136/thorax-2020-btsabstracts.145.

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Reports on the topic "Receptors, adrenergic, beta"

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Slotkin, Theodore. Beta-Adrenergic Receptors Regulating Growth and Replication of Breast Cancer cells: Basic and Therapeutic Implications. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada395273.

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