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Dissertations / Theses on the topic 'Receptors, adrenergic, beta'

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Published: 4 June 2021

Last updated: 31 January 2023

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1

Vela, Jose David. "Beta adrenergic modulation of peripheral nociceptors a dissertation /." San Antonio : UTHSC, 2008. http://proquest.umi.com.libproxy.uthscsa.edu/pqdweb?did=1588778261&sid=1&Fmt=2&clientId=70986&RQT=309&VName=PQD.

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2

Stern, Ligia R. "Regulation of beta-adrenergic receptors in cultured lung cells." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74250.

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The effects of glucocorticoids and agents affecting membrane phospholipid metabolism on $ beta$-adrenergic receptors ($ beta$AR) have been studied in A549 cultured human lung tumor cells and in IM-9 cultured human lymphocytes. Various glucocorticoids increased the density of $ beta$AR in A549 lung cells but not in IM-9 lymphocytes, although they did increase insulin receptor density in IM-9 lymphocytes. In contrast, bee venom phospholipase A$ sb2$ (0.05-0.2 units/ml), but not phospholipases C or D., downregulated $ beta$AR in IM-9 lymphocytes, but did not affect $ beta$AR in A549 lung cells. U

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3

Potter, Paul Charles. "In Vitro studies of factors which may influence ligand binding, function, immunogenicity and genetic regulation of the Beta-2 adrenergic receptor in asthma." Doctoral thesis, University of Cape Town, 1991. http://hdl.handle.net/11427/26483.

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This thesis records a series of experiments conducted to gain further insight into factors which influence the expression, ligand binding and functional activity of the beta-2 adrenergic receptor. These studies were prompted by previous reports that the postulated beta-2 adrenergic receptor abnormality in allergic asthma could be induced, induced by autoantibodies. I established and optimised beta-2 adrenergic receptor ligand binding and functional assays in guinea pig lung membranes and then conducted an original study of beta adrenergic receptor expression in the guinea pig foetal lung. I fo

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4

Zolty, Ronald. "Beta 1 and Alpha 2C adrenergic receptor polymorphisms and response to beta blockers in heart failure patients /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2007.

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Thesis (Ph.D. in Clinical Science) -- University of Colorado Denver, 2007.<br>Typescript. Includes bibliographical references (leaves 130-142). Free to UCD affiliates. Online version available via ProQuest Digital Dissertations;

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5

Crumbie, Hayley Elizabeth. "Beta-adrenergic receptor signalling and excitation-contraction coupling in the heart : impact of circadian rhythms and beta-3 receptors." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37500.

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A time-of-day variation in myocardial contraction and its response to sympathetic stimulation of β-adrenoceptors (b-ADR) exists, which is reflected by time-of-day variations in intracellular calcium [Ca2+]i regulation and electrical activity. There are three isoforms of β-ADR (β1/β2/β3) and the functional outcome of each receptor differs. β1/β2-ADRs result in positive inotropism, whereas β3-ADR induces a negative inotropic effect, associated with nitric oxide (NO) signalling. The aims of this thesis were to determine the role of the β3-ADR in the time-of-day variation in the response to sympat

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6

McLean, Alison Jane. "Ligand regulation of #beta#â†1- and #beta#â†2- adrenergic receptors and their GFP-tagged forms." Thesis, University of Glasgow, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323436.

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7

Salinas, Cristian Andres. "ROBUST EXPERIMENTAL DESIGN FOR ESTIMATING MYOCARDIAL BETA ADRENERGIC RECEPTOR CONCENTRATION USING POSITRON EMISSION TOMOGRAPHY." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1144088920.

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8

Ekpe, Enefiok David. "Beta-adrenergic receptors in tissues of sheep, effects of temperature and plane of nutrition." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ29032.pdf.

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9

Salamanca, Melissa, and Melissa Salamanca. "Beta₃-adrenergic receptors in the heart: Normal functions and potential roles in heart failure." Thesis, The University of Arizona, 2016. http://hdl.handle.net/10150/621455.

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Heart Failure is the state in which the heart is unable to pump enough blood to meet the needs of the body, resulting from events that progressively damage the myocardium and/or its ability to contract normally. Several compensatory mechanisms allow for short-term maintenance of adequate delivery of blood to the body. Progression from the initial events to heart failure is caused by the accumulation of pathologic changes and their effects, many of which result from chronic activation of these compensatory mechanisms. One of these mechanisms is the activation of the neurohormonal β-adrenergic

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10

Walker, Robert J. "Inflammatory cytokines modulated by beta adrenergic receptors on retinal Muller cells in diabetic retinopathy /." Available to subscribers only, 2007. http://proquest.umi.com/pqdweb?did=1467888651&sid=24&Fmt=2&clientId=1509&RQT=309&VName=PQD.

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11

Fávaro-Moreira, Nádia Cristina 1981. "B-blockers effect in the temporomandibular joint pain in rats and humans and its modulation by gonadal hormones = Efeito dos B-bloqueadores na dor da articulação temporomandibular de ratos e humanos e sua modulação pelos hormônios." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/290581.

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Orientador: Cláudia Herrera Tambeli<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba<br>Made available in DSpace on 2018-08-24T16:27:32Z (GMT). No. of bitstreams: 1 Favaro-Moreira_NadiaCristina_D.pdf: 14440949 bytes, checksum: 19ac7e9f7196c13dfd756bd4150b1456 (MD5) Previous issue date: 2014<br>Resumo: Disfunção temporomandibular (DTM) é um termo coletivo que abrange uma série de problemas clínicos que envolvem os músculos mastigatórios e a articulação temporomandibular (ATM) e está comumente associada à inflamação. Apesar de medicamentos anti-in

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12

Tabrizchi, Reza. "Different modes of vasopressor actions of angiotensin and non-selective or selective beta-adrenoceptor antagonists." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/29439.

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Vasoconstriction can be initiated via the interaction of a number of chemicals with specific "receptive sites" known as the receptors. This thesis examines two distinctly different modes by which drugs initiate a contractile response, namely, (i) the interaction of angiotensin analogues with a heterogeneous population of angiotensin receptors in vascular smooth muscles, and (ii) the conditions whereby B-adrenoceptor antagonists interact with a-adrenoceptor antagonists thereby causing a pressor response. Conscious, unrestrained, instrumented-rats were used for the study. It has been suggested

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13

Rashdan, Nabil. "The effects of prenatal hypoxia on the levels of the α-subunits of G proteins in the heart of the Broiler chicken (Gallus gallus)". Thesis, Linköpings universitet, Zoologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-57191.

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Environmental stress during embryonic development could lead to growth restriction of the embryo, and act as a risk factor for the development of cardiovascular disease in adult life. A common environmental stressor that causes growth restriction is prenatal hypoxia, which has been shown to adversely affect adult health in mammalian models. Prenatal hypoxia causes an increase in catecholamines which results in over stimulation of the cardiac β-adrenergic receptors. Previous work on chickens has shown that prenatal hypoxia causes an increase in the sensitivity of β-adrenergic receptors to epine

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14

Maqueira, Iglesias Braudel. "Cloning and characterization of a novel family of β adrenergic-like octopamine receptors from Drosophila melanogaster". Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.615211.

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15

Ryberg, Mats. "Chronic treatment with [beta]-adrenoceptor agonists in asthmatics effects on salivary gland function and dental caries development /." Umeå : University of Umeå, 1991. http://catalog.hathitrust.org/api/volumes/oclc/23592154.html.

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16

Hunt, Desmond Gerard. "The role of [beta]-adrenergic receptors in regulating skeletal muscle glucose utilization at rest and during exercise /." Full text (PDF) from UMI/Dissertation Abstracts International, 2001. http://wwwlib.umi.com/cr/utexas/fullcit?p3008356.

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17

Alouane, Hamiroufou Loubna. "Préconditionnement myocardique et diabéte : effets aigus des AGEs sur le préconditionnement induit par la stimulation des récepteurs β1-adrénergiques et purinergiques. Rôle de la vitamine B6 (Pyridoxal-5- phosphate et pyridoxamine)". Thesis, Reims, 2012. http://www.theses.fr/2012REIMM202/document.

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Le diabète prédispose à des complications affectant divers organes comme le systémecardiovasculaire. La cardiopathie ischémique chez les patients diabétiques pourrait être liée àl'accumulation de produits avancés de glycation (AGEs). Dans les coeurs de rats ischémiques,l'expression des récepteurs aux AGEs et de ses ligands est considérablement augmentée etimpliquée dans les lésions de l'ischémie / reperfusion (I / R), même en absence de diabète. Ila été récemment rapporté que le myocarde humain diabétique ne peut pas être protégé par lepréconditionnement. Dans ce contexte, notre hypothèse étai

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18

Silva, Mariana de França Oliveira da. "Efeitos dos hormônios tireoidianos sobre a regulação da expressão de proteínas envolvidas com a lipólise no tecido adiposo branco subcutâneo e visceral." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-07122015-182041/.

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Os hormônios tireoidianos (HT) executam um papel lipolítico importante no Tecido Adiposo Branco (TAB), sendo este efeito mediado por meio do aumento da expressão de receptores beta adrenérgicos na membrana do adipócito, o que aumenta a sensibilidade deste tecido as catecolaminas. Sabe-se que os principais efetores da ação lipolítica nesse tecido são a lipase hormônio sensível (LHS) e a lipase dos triglicerídeos dos adipócitos (ATGL), as quais hidrolisam os triglicerídeos em ácidos graxos e glicerol. Além disso, outros componentes estão envolvidos na atividade lipolítica, como as perilipinas, p

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19

Johansson, Catarina. "Physiological changes in mice deficient in different subtypes of thyroid hormone receptors : a focus on studies of heart and muscle /." Stockholm, 1999. http://diss.kib.ki.se/1999/19990429joha/.

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20

Qing, Feng. "Studies of beta-adrenergic receptors in vivo in humans using the CGP-12177 ligand and positron emission tomography." Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325236.

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21

Inderwies, Tyra. "Alpha- and Beta-adrenergic receptors in the mammary gland and their influence on milking characteristics in dairy cows /." [S.l.] : [s.n.], 2002. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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22

Baragli, Alessandra. "Assembly and function of multimeric adenylyl cyclase signalling complexes." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111888.

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G protein coupled receptors, G proteins and their downstream effectors adenylyl cyclase (ACs) were thought to transiently interact at the plasma membrane by random collisions following agonist stimulation. However a growing number of studies have suggested that a major revision of this paradigm was necessary to account for signal transduction specificity and efficiency. The revised model suggests that signalling proteins are pre-assembled as stable macromolecular complexes together with modulators of their activity prior to receptor activation. How and where these signalling complexes form and

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23

Carie, Adam E. "Tumor suppressive effects of the Beta-2 adrenergic receptor and the small GTPase RhoB." [Tampa, Fla.] : University of South Florida, 2008. http://purl.fcla.edu/usf/dc/et/SFE0002330.

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24

Gowdak, Marcia Maria Godoy. "Efeito da ingestão aguda de gordura na resposta vasodilatadora muscular em portadores de polimorfismo nos receptores B2-adrenérgicos." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-20082007-160349/.

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Indivíduos portadores do glutamato na posição 27 do gene que codifica para o receptor beta2-adrenérgico têm resposta vasodilatadora muscular aumentada durante manobras fisiológicas. No entanto, o impacto do consumo agudo de gordura nessa resposta não é conhecido. Neste estudo, testou-se a hipótese de que o consumo gordura afetaria a resposta vasodilatadora aumentada destes indivíduos durante manobras fisiológicas. Vinte e cinco indivíduos saudáveis foram subdivididos em dois grupos: 11 homozigotos para o glutamato (Glu27Glu, 40+-3 anos; 65+-3kg) e 14 homozigotos para a glutamina (Gln27Gln, 40+

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25

Haley, James M. "Ex Vivo Evaluation of Myocardial Beta-Adrenergic Receptors in High-Fat Fed STZ and ZDF Models of Diabetes Using [3H]-CGP12177." Thesis, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/30363.

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Diabetes mellitus (DM) and hyperglycemia contribute to sympathetic nervous system (SNS) activation and cardiovascular dysfunction. SNS activation and increased norepinephrine levels downregulate cardiac β-adrenergic receptors (β-AR). The ADMIRE-HF trial identified reduced cardiac SNS innervation as an independent prognostic marker in heart failure. The β-AR antagonist [3H]-CGP12177 was used to quantify cardiac β-AR in ex vivo biodistribution studies in streptozotocin (STZ)-treated rats after 8 weeks of sustained hyperglycemia, and in the Zucker Diabetic Fatty (ZDF) rat model of type-2 diabetes

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26

Ferraz, Karina Coutinho 1986. "Efeitos da infusão intravenosa de metaloproteinase-2 da matriz recombinante humana (rhMMP-2) em respostas ß-adrenérgicas = Effects of intravenous infusion of recombinant human matrix metalloproteinase-2 (rhMMP-2) in ß-adrenergic responses." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/310017.

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Orientador: José Eduardo Tanus dos Santos<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-21T15:51:02Z (GMT). No. of bitstreams: 1 Ferraz_KarinaCoutinho_D.pdf: 4092717 bytes, checksum: 304af959216055cd75432ac292abc300 (MD5) Previous issue date: 2012<br>Resumo: Evidências clínicas e experimentais indicam o envolvimento de metaloproteinases da matriz extracelular (MMPs) na patogênese de diversas doenças, incluindo as doenças cardiovasculares. Particularmente, alterações na atividade da MMP-2 parecem desempenhar um im

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Vilardi, Bruna Maria Rodrigues. "Modulação da expressão de VEGF-C por mediador relacionado ao estresse em culturas de carcinomas espinocelulares de boca." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/25/25150/tde-13072011-164007/.

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Os mediadores do estresse, epinefrina e norepinefrina, participam da modulação de diversos processos celulares como a proliferação, a migração celular e a apoptose durante a tumorigênese, influenciando assim, o crescimento e a progressão tumoral. A presença de receptores beta-adrenérgicos e sua expressiva resposta ao estímulo do neurohormônio norepinefrina foram identificadas em várias linhagens de células tumorais, incluindo o carcinoma espinocelular de boca. O objetivo deste estudo foi investigar a influência da norepinefrina na expressão do fator de crescimento endotelial vascular do tipo C

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Hutchinson, Dana Sabine 1976. "Pharmacological characterisation and signalling pathways of recombinant and endogenously expressed mouse β₃-adrenoceptors". Monash University, Dept. of Pharmacology, 2001. http://arrow.monash.edu.au/hdl/1959.1/8516.

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29

Papi, Bianca Neofiti. "Avaliação da interação do hormônio tireoidiano com o sistema nervoso simpático, via receptor Beta2-adrenérgico, na regulação da massa e metabolismo ósseos." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-28092018-090735/.

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O hormônio tireoidiano (HT) é essencial para o desenvolvimento, maturação e metabolismo ósseos, enquanto que o sistema nervoso simpático (SNS) é, também, um potente regulador do remodelamento ósseo. Demonstrou-se que SNS regula negativamente a massa óssea, agindo via receptores ?2-adrenérgicos (?2-AR), expressos em osteoblastos. O nosso grupo demonstrou que os receptores ?2 adrenérgicos (?2-AR) também medeiam ações do SNS no esqueleto e que são expressos em osteoblastos, osteócitos, condrócitos e osteoclastos. Considerando-se que o HT interage com o SNS para regular uma série de processos fisi

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30

Souza, Germano Emílio Conceição. "Avaliação do bloqueio simpático cervico-torácico esquerdo por clipagem em portadores de insuficiência cardíaca sistólica sintomática com terapia medicamentosa otimizada." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-01022011-164015/.

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FUNDAMENTO: A modulação adicional do sistema nervoso simpático em pacientes com insuficiência cardíaca sistólica (ICS) sob tratamento clínico otimizado pode ser benéfica. Objetivo: Avaliar a exequibilidade e segurança do BSCTE em pacientes com ICS sintomática, refratária ao tratamento farmacológico incluindo beta-bloqueadores (BB). Secundariamente, avaliamos os seus efeitos no sistema cardiovascular tanto no perioperatório quanto após seis meses de seguimento. MÉTODOS: Ensaio clínico com duplo sorteio com \"concealed allocation\". Pacientes com ICS e Fração de ejeção do VE (FEVE) = 40%, CF da

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Warrier, Sunita. "cAMP BIOSENSORS AND SPATIOTEMPORAL cAMP SIGNALING IN ADULT CARDIAC MYOCYTES." Case Western Reserve University School of Graduate Studies / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=case1175718415.

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32

Davis, Danisha Marie, Suman Dalal, Connor James, Cerrone R. Foster, and Krishna Singh. "The Role of Osteopontin in Extracellular Matrix Remodeling Following Chronic Sympathetic Stimulation in The Aging Heart." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/122.

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Cardiovascular disease (CVD) is the leading cause of death in the United States. A common feature in most cardiac pathologies is the dysregulation of beta-adrenergic receptors (β-AR) and changes in the extracellular matrix (ECM). The ECM maintains strength and normal organization of cardiac tissue, while fibrosis (connective tissue scarring) is necessary for repair of damaged cardiac tissue. However, the dysregulation of the ECM leads to a number of cardiac disease pathologies. Osteopontin (OPN) is a protein with diverse biological functions in regulating the ECM such as bone resorption and ca

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33

Sun, Ruoyu [Verfasser], Reinhard [Akademischer Betreuer] Nießner, Peter B. [Gutachter] Luppa, and Reinhard [Gutachter] Nießner. "Nanodiscs incorporating functional human beta-1 adrenergic receptors as novel diagnostic approach for autoimmune dilated cardiomyopathy / Ruoyu Sun ; Gutachter: Peter B. Luppa, Reinhard Nießner ; Betreuer: Reinhard Nießner." München : Universitätsbibliothek der TU München, 2018. http://d-nb.info/1185069569/34.

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Campreciós, Figueras Genís. "Els receptors ErbB en la resposta a l’estimulació adrenèrgica crònica en el ratolí." Doctoral thesis, Universitat de Barcelona, 2010. http://hdl.handle.net/10803/31976.

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En aquest estudi s’ha abordat el paper que poden jugar els receptors de la família ErbB en l’estrès crònic per catecolamines en el ratolí. Se sabia que el Factor de Creixement Epidèrmic (EGF) interfereix en el fetge, el teixit adipós i el cor en la resposta màxima d’aquests òrgans a l’adrenalina disminuint els nivells de cAMP. A més a més, l’EGF és alliberat de les glàndules submaxil•lars en resposta a un estímul α-adrenèrgic. Aquesta interferència en el cor té un efecte cardioprotector durant una estimulació adrenèrgica aguda. En models fisiològics d’estrès per confrontació entre mascles, es

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Boer, Denile Cominato 1980. "Arritmogenese por catecolaminas em miocardio atrial e ventricular de ratos : metodologia e tipos de adrenoceptores envolvidos." [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/261797.

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Orientadores: Jose Wilson Magalhães Bassani, Rosana Almada Bassani<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Engenharia Eletrica e de Computação<br>Made available in DSpace on 2018-08-06T07:47:18Z (GMT). No. of bitstreams: 1 Boer_DenileCominato_M.pdf: 557480 bytes, checksum: e378c01e865d04a595d3f0e7ad3c60dd (MD5) Previous issue date: 2006<br>Resumo: Embora haja demonstração de que a estimulação simpática tenha efeito facilitatório sobre a indução de atividade espontânea em miocárdio há controvérsia sobre a participação dos tipos de adrenoceptores na mediação

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Lafaille, Francine. "Characterization of [125I]7-amino-8-iodo-ketanserin binding and comparative effects of long-term treatment with anxiolytic and antidepressant drugs on serotonin type 2 and beta-adrenergic receptors in rat brain." Thesis, McGill University, 1991. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=60548.

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Changes to 5-HT$ sb2$ receptors in rat brain following antidepressant and anxiolytic treatment were investigated. The B$ sb{ rm max}$ of ($ sp{125}$I) AMIK was found to be decreased significantly in rat fronto-parietal cortex after 21 days administration of antidepressant drugs including desmethylimipramine (30%), buspirone (47%) and adinazolam (17%). The anxiolytic drug, diazepam, which is devoid of intrinsic antidepressant action, did not significantly change the binding parameters. In comparison to 5-HT$ sb2$ receptors, the beta-adrenoceptors labelled by ($ sp{125}$I) cyanopindolol in membr

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Kurland, Lisa. "Pharmacogenetic Studies of Antihypertensive Treatment : With Special Reference to the Renin-Angiotensin-Aldosterone System." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5202-7/.

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Bagley, John Sorrell. "The beta adrenergic receptor in septic shock." Thesis, University of Aberdeen, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305466.

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The myocardial depression found during septic shock has been a subject of intense study for many years. Many theories exist as to the precise cause of this condition but in this thesis the role of the β-adrenergic receptor is examined. Some investigators have found reduced response to β-adrenergic stimulation in models of septic shock in animals. This is of interest to the clinician since adrenergic stimulation is a key manipulation in the management of septic shock. An animal model of septic shock was established using a comparatively small dose of <i>Escherichia coli</i> endotoxin infused in

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Higgins, Thomas John Diatchenko Luda. "Molecular characterization of [beta]2 adrenergic receptor haplotypes." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,1602.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2008.<br>Title from electronic title page (viewed Sep. 16, 2008). "... in partial fulfillment of the requirements for the degree of Master's of Science in the Curriculum in Neurobiology." Discipline: Neurobiology; Department/School: Medicine. On title page, [beta] appers as Greek character and 2 appears in subscript.

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Belacel, Ouari Milia. "Contrôle de la voie de l’AMPc vasculaire par les phosphodiestérases en situation physiopathologique." Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLS504/document.

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L’AMPc est un second messager exerçant un rôle vasculoprotecteur majeur, par ses effets relaxants et inhibiteurs de la prolifération et de la migration cellulaires. Les concentrations intracellulaires d’AMPc sont finement régulées par leur synthèse via les adénylates cyclases et leur dégradation par les phosphodiestérases (PDEs). Nous avons évalué l’impact de l’environnement cellulaire sur la voie de signalisation couplée au récepteur β-adrénergique (β-AR/AMPc/PDE) dans les cellules musculaires lisses vasculaires (CMLVs), ainsi que les altérations potentielles de celle-ci en situation patholog

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41

Snyder, Ellen Amanda Flood Patrick M. "[beta]2-adrenergic receptor modulation of macrophage inflammatory mediator production." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,1344.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2007.<br>Title from electronic title page (viewed Apr. 25, 2008). "... in partial fulfillment of the requirements for the degree of Master of Science in the Curriculum of Oral Biology." Discipline: Oral Biology; Department/School: Dentistry. On title page, [beta] appears as Greek character and 2 appears as subscript.

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Robson, Alice. "In vitro reconstitution and stability of a beta-adrenergic receptor." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424005.

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43

Vincent, Karla Kristine. "Transactivation of Beta 2 Adrenergic Receptor by Bradykinin type 2 Receptor via heterodimerization." Diss., Georgia Institute of Technology, 2009. http://hdl.handle.net/1853/37117.

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Although a long standing convention maintained that G Protein Coupled Receptors (GPCRs) exist in the plasma membrane solely as monomers, substantial work over the last two decades has demonstrated that these ubiquitous receptors can and in many cases, preferentially, exist as homodimers, heterodimers, or higher order oligomers. Often, two GPCRs of the same class heterodimerize; it is less common for two GPCRs of different signaling pathways to interact. The work presented here studied the physical and functional interaction of two GPCRs from discrete classes, the Beta 2 Adrenergic Receptor

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44

Allcock, Stephen M. J. "Interactions between calpain proteases #beta#-adrenergic receptor and skeletal muscle growth." Thesis, University of Nottingham, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.358286.

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45

Clausen, Lisa. "Effects of beta-2 adrenergic receptor agonists in DOK7 congenital myasthenic syndrome." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:9360c51b-8497-47ca-bd16-e917a3614a25.

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Congenital myasthenic syndromes (CMS) are a rare group of heterogeneous disorders, characterised by compromised neuromuscular transmission and symptoms of fatiguable muscle weakness. CMS is caused by mutations in genes that affect the structure and function of the neuromuscular junction (NMJ). In about 20% of CMS cases, patients have mutations in the gene DOK7; the protein product, DOK7, is crucial for maintaining the dense aggregation of acetylcholine receptor (AChR) clusters at the NMJ. DOK7-CMS patients do not respond to treatment with acetylcholinesterase inhibitors which are the first lin

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Plank, David Michael. "Calcium dynamics, β-adrenergic receptor blockade, and cardiac function in failing and non-failing hearts". University of Cincinnati / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1053431441.

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47

Owens, Helen. "The effect of beta-adrenergic receptor antagonists on the temporal accommodative response." Thesis, Aston University, 1991. http://publications.aston.ac.uk/14611/.

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It is well established that the steady-state accommodative response is characterised by temporal changes in lens power having 2 dominant frequency components: a low frequency component (LFC: < 0.6Hz) and a high frequency component (HFC: 1.0-2.2Hz). This thesis investigates various aspects of these microfluctuations of accommodation. The HFC of accommodative fluctuations was shown to be present in central and peripheral lens zones, although the magnitude of the rms of accommodative microfluctuations was found to be reduced in the lens periphery. These findings concur with the proposal that the

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48

Clausen, Lisa K. J. "Effects of beta-2 adrenergic receptor agonists in DOK7 congenital myasthenic syndromes." Thesis, University of Oxford, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.712079.

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Yu, Esther Jeong. "Cloning and characterization of a novel β-adrenergic like receptor from Drosophila melanogaster". Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.620357.

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Wardle, Robert L. "Functional antagonism between muscarinic receptor and beta-adrenergic receptor agonists in equine trachealis muscle in vitro /." The Ohio State University, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487858417981441.

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