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Books on the topic 'Receptors, GABA-Benzodiazepine'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 January 2023

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1

1944-, Hindmarch I., ed. Benzodiazepines: Current concepts : biological, clinical, and social perspectives. Chichester [England]: Wiley, 1990.

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2

Giovanni, Biggio, Costa Erminio, and Capo Boi Conference on Neuroscience (6th : 1989 : Villasimius, Italy), eds. GABA and benzodiazepine receptor subtypes: Molecular biology, pharmacology, and clinical aspects. New York: Raven Press, 1990.

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3

W, Olsen Richard, and Venter J. Craig, eds. Benzodiazepine/GABA receptors and chloride channels: Structural and functional properties. New York: A.R. Liss, 1986.

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4

Martin, Sarter, Nutt David J. 1951-, and Lister Richard G, eds. Benzodiazepine receptor inverse agonists. New York: Wiley-Liss, 1995.

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5

1951-, Marescaux C., Vergnes M. 1935-, and Bernasconi R. 1929-, eds. Generalized non convulsive epilepsy: Focus on GABA-B receptors. Wien: Springer-Verlag, 1992.

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6

1944-, Martin Ian L., ed. The GABAA/benzodiazepine receptor as a target for psychoactive drugs. Austin: R.G. Landes, 1995.

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7

J, Rodger R., and Cooper S. J, eds. 5-HT1A agonists, 5-HT3 antagonists and benzodiazepines: Their comparative behavioural pharmacology. Chichester, West Sussex, Englad: Wiley, 1991.

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8

L, Erdő Sándor, and Bowery N. G, eds. GABAergic mechanisms in the mammalian periphery. New York: Raven Press, 1986.

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9

NATO Advanced Research Workshop on Molecular Biology of Neuroreceptors and Ion Channels (1988 Thera Island, Greece). Molecular biology of neuroreceptors and ion channels. Berlin: Springer-Verlag, 1989.

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10

Malison, Robert Thomas. Structural homology of the GABAa/benzodiazepine receptor as demonstrated by monoclonal antibodies and limited proteolysis. [New Haven: s.n.], 1987.

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11

Doble, Adam. GABA-A/benzodiazepine receptor as a target for psychoactive drugs. New York: Springer, 1996.

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12

The benzodiazepine receptor: Drug acceptor only or a physiologically relevant part of our central nervous system? Cambridge: Cambridge University Press, 1987.

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13

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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14

F, Squires Richard, ed. GABA and benzodiazepine receptors. Boca Raton, Fla: CRC Press, 1988.

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15

Hindmarch, Ian, G. Beaumont, and S. Brandon. Benzodiazepines: Current Concepts : Biological, Clinical and Social Perspectives (A Wiley Interscience Publication). John Wiley & Sons, 1990.

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16

E, Giesen-Crouse, ed. Peripheral benzodiazepine receptors. London: Academic Press, 1993.

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17

The Gaba Benzodiazepine Receptor As a Target for Psychoactive Drugs (Neuroscience Intelligence Unit). R G Landes Co, 1996.

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18

Ivan, Izquierdo, and Medina Jorge, eds. Naturally occurring benzodiazepines: Structure, distribution, and function. New York: Ellis Horwood, 1993.

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19

Olsen, Richard W. Benzodiazepine/Gaba Receptors and Chloride Channels: Structural and Functional Properties (Receptor Biochemistry and Methodology, Vol 5). Wiley-Liss, 1986.

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20

Marescaux, C., M. Vergnes, and R. Bernasconi. Generalized Non-Convulsive Epilepsy: Focus on GABA-B Receptors. Springer, 2012.

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21

Marescaux, C., M. Vergnes, and R. Bernasconi. Generalized Non-Convulsive Epilepsy: Focus on GABA-B Receptors. Springer London, Limited, 2013.

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22

Giovanni, Biggio, Costa Erminio, and Capo Boi Conference on Neuroscience (5th : 1987 : Villasimius, Italy), eds. Chloride channels and their modulation by neurotransmitters and drugs. New York: Raven Press, 1988.

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23

Ranney, Mize R., Marc R. E, and Sillito A. M, eds. GABA in the retina and central visual system. Amsterdam: Elsevier, 1992.

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24

(Editor), C. Marescaux, M. Vergnes (Editor), and R. Bernasconi (Editor), eds. Generalized Non Convulsive Epilepsy: Focus on Gaba-B Receptors ("Journal of Neural Transmission"). Springer-Verlag, Austria, 1992.

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25

E, Vartani͡a︡n M., ed. Neuronal receptors, endogenous ligands, and biotechnical approaches. Madison, Conn: International Universities Press, 1988.

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26

Marescaux, C., and M. Vergnes. Generalized Non Convulsive Epilepsy: Focus on Gaba-B Receptors (Journal of Neural Transmission Supplementum). Springer, 1992.

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27

(Editor), Erminio Costa, ed. Gaba Receptors and Anxiety: From Neurobiology to Treatment (Advances in Biochemical Psychopharmacology). Raven Pr, 1995.

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28

Giovanni, Biggio, Costa Erminio, and Capo Boi Conference on Neuroscience (4th : 1985 : Villasimius, Italy), eds. GABAergic transmission and anxiety. New York: Raven Press, 1986.

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29

Giovanni, Biggio, Concas Alessandra, and Costa Erminio, eds. GABAergic synaptic transmission: Molecular, pharmacological, and clinical aspects. New York: Raven Press, 1992.

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30

1944-, Hindmarch I., Ott H, and Collegium Internationale Neuro-psychopharmacologicum Congress, eds. Benzodiazepine receptor ligands, memory, and information processing: Psychometric, psychopharmacological, and clinical issues. Berlin: Springer-Verlag, 1988.

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31

Squires, R. F. Gaba and Benzodiazepine Receptor. CRC Press Inc, 1987.

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32

Peppin, John, Joseph V. Pergolizzi, Robert B. Raffa, and Steven L. Wright, eds. The Benzodiazepines Crisis. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780197517277.001.0001.

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Abstract:
When properly prescribed, benzodiazepines and related “Z” drugs, are usually safe and effective. However, some patients experience lack of efficacy, severe adverse effects, and/or protracted withdrawal symptoms. Unfortunately, there is no reliable way to predict outcome prior to treatment. Use has dramatically expanded, to the point where some experts suggest a disconnect with actual medical need. With increased and longer prescribing there has been a corresponding increase in the “down-side” of these drugs. Benzodiazepines, as all drugs, produce some degree of normal physiologic tolerance and physical dependence. But for some patients withdrawal can result in a bewildering array of symptoms, that can persist for protracted time periods, difficult to understand and live with. Although there is currently no clear mechanistic explanation, some potentials include alterations of receptor number, promoters of receptor protein synthesis or degradation, absorption, distribution, metabolism, and elimination, GABAA-receptor function or subtype-distribution, or involvement of peripheral benzodiazepine binding/receptor sites. This book attempts to bring benzodiazepine use under a more rational paradigm and reduce the incidence of side-effects and drug–drug interactions (DDI). It is the first devoted to take on this responsibility. Use, overuse/misuse, side-effects, DDI, physiology, and withdrawal are reviewed by expert clinicians and basic scientists in-depth. The book challenges the medical community to take seriously the use of this class of drug and to ameliorate prescribing behavior. The case is made for limiting initiation and duration (2–4 weeks) of use, and careful, supported discontinuation. We laud and suggest increased research into this class of drug and it’s “down-side.”
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33

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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