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1

Manic-depressive illness: Bipolar and recurrent depression. 2nd ed. Oxford University Press, 2007.

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2

Fieve, Ronald R. Bipolar breakthrough: The essential guide to going beyond moodswings to harness your highs, escape the cycles of recurrent depression, and thrive with bipolar II. Rodale, 2009.

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3

Richards, C. Steven. Relapse prevention for depression. American Psychological Association, 2010.

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4

Association, American Psychological, ed. Relapse prevention for depression. American Psychological Association, 2010.

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5

Gallagher-Thompson, Dolores. Depression and its recurrence in older persons with disabling illnesses. Rehabilitation Research and Training Center on Aging, Rancho Los Amigos Medical Center/University of Southern California, 1991.

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6

Maximizing psychotherapeutic gains and preventing relapse in emotionally distressed clients. Professional Resource Press, 1995.

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7

Ludgate, John W. Cognitive-behavioral therapy and relapse prevention for depression and anxiety. Professional Resource Press, 2009.

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8

1942-, Greden John F., ed. Treatment of recurrent depression. American Psychiatric Pub., 2001.

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9

Jamison, Kay Redfield, and Frederick Goodwin. Manic-Depressive Illness: Bipolar Disorders and Recurrent Depression. Oxford University Press, 2007.

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10

John F., M.D. Greden. Treatment of Recurrent Depression (Review of Psychiatry). American Psychiatric Publishing, Inc., 2001.

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11

John, Cobb, Goeting Nicola L. M, Duphar Medical Relations, and Royal Society of Medicine (Great Britain), eds. Prediction and treatment of recurrent depression. Duphar Medical Relations, 1990.

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12

Post, Robert M. Depression as a Recurrent, Progressive Illness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603342.003.0003.

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Clinical Highlights and summary of Chapter• Episodes of depression and bipolar illness progress in two ways:faster recurrences as a function of number of prior episodes, andgreater autonomy (decreased need for precipitation by stressors(Episode Sensitization)• Recurrent stressors result in increased reactivity to subsequent stressors(Stress sensitization) and bouts of stimulant abuse increase in severity with repetition(Stimulant-induced behavioral sensitization)• Each type of sensitization cross-sensitizes to the others and drives illness progression• Each type of sensitization involves speci
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13

Goodwin, Frederick K., and Kay Redfield Jamison. Manic-Depressive Illness: Bipolar Disorders and Recurrent Depression, 2nd Edition. 2nd ed. Oxford University Press, USA, 2007.

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14

Manic-Depressive Illness Bipolar Disorders and Recurrent Depression Volume I, (Volume I). 2nd ed. Oxford University Press, 2007.

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15

Levinson, Douglas F., and Walter E. Nichols. Genetics of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0024.

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Major depressive disorder (MDD) is a common and heterogeneous complex trait. Twin heritability is 35%–40%, perhaps higher in severe/recurrent cases. Adverse life events (particularly during childhood) increase risk. Current evidence suggests some overlap in genetic factors among MDD, bipolar disorder, and schizophrenia. Large genome-wide association studies (GWAS) are now proving successful. Polygenic effects of common SNPs are substantial. Findings implicate genes with effects on synaptic development and function, including two obesity-associated genes (NEGR1 and OLFM4), but not previous “can
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16

Guha, Maya B. The incidence of recent life events in women who experience recurrent episodes of unipolar depression. 1985.

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17

Dietz, Laura J., Laura Mufson, and Rebecca B. Weinberg. Family-based Interpersonal Psychotherapy for Depressed Preadolescents. Oxford University Press, 2018. http://dx.doi.org/10.1093/med-psych/9780190640033.001.0001.

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Family-based Interpersonal Psychotherapy (FB-IPT) for Depressed Preadolescents presents the rationale and basic principles for interpersonal psychotherapy (IPT) and for interpersonal psychotherapy for depressed adolescents (IPT-A), a developmental adaptation that is designed to treat adolescents, ages 12 to 18 years, with depression. The heart of this book introduces family-based interpersonal psychotherapy for depressed preadolescents (FB-IPT), a psychosocial treatment for preadolescent depression for children between 7 and 12 years. FB-IPT is conceptually rooted in an interpersonal model of
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18

Lam, Raymond W. Epidemiology and burden. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199692736.003.0002.

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• Depression is a highly prevalent condition – about 1 in 7 people will experience a depressive episode during their lifetime.• Many people with depression will have a recurrent or chronic course, leading to substantial impairment in psychosocial function.• Depression is now the leading cause of disability in developed countries and the fourth leading cause world wide....
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19

James, Anthony. Depressive Disorders in Childhood and Adolescence. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198801900.003.0008.

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This chapter focuses on depressive disorders in childhood and adolescence. Depression in children and adolescents is a complex and debilitating disease, and typically has a lifelong, chronic, and recurrent course. The peak age of onset of depression is between 13 and 15 years. After providing a clinical picture of depression, this chapter discusses early childhood depression and differential diagnosis, including paediatric bipolar disorder, psychotic depression and seasonal affective disorder, oppositional and conduct disorder, and substance misuse and medical conditions. It then examines como
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20

Naess, Halvor. Long-term prognosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198722366.003.0016.

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Knowledge of prognosis is important for patients in the prime of life in order to make informed decisions about treatment, choice of education, and profession. Median first-year mortality after first-ever cerebral infarction among young adults is about 4% while median annual average mortality after the first year is about 1.7%. Likewise, median first-year recurrence rate of cerebral infarction is 2% and thereafter 1.5% per year. Risk factors for recurrent cerebral infarction include hypertension, diabetes mellitus, symptomatic atherosclerosis, and smoking. Recurrent cerebral infarction and mor
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21

Bipolar II: Enhance Your Highs, Boost Your Creativity, and Escape the Cycles of Recurrent Depression--The Essential Guide to Recognize and Treat the Mood Swings of This Increasingly Common Disorder. Rodale Books, 2006.

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22

Henter, Ioline D., and Rodrigo Machado-Vieira. Novel therapeutic targets for bipolar disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0030.

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The long-term course of bipolar disorder (BD) comprises recurrent depressive episodes and persistent residual symptoms for which standard therapeutic options are scarce and often ineffective. Glutamate is the major excitatory neurotransmitter in the central nervous system, and glutamate and its cognate receptors have consistently been implicated in the pathophysiology of mood disorders and in the development of novel therapeutics for these disorders. Since the rapid and robust antidepressant effects of the N-methyl-D-aspartate (NMDA) antagonist ketamine were first observed in 2000, other NMDA
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23

Williams, J. Mark G. Depression. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780192627254.003.0011.

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Chapter 11 summarizes some of the ways in which psychological theory has contributed to the understanding of depression and how best to treat it. The nature of depression is outlined, along with the learning theory and social skills approach, self-control theory, cognitive theory and therapy of depression, evaluating the efficacy of cognitive therapy, the NIMH Treatment of Depression Collaborative Research Program, the prevention of relapse and recurrence, and mechanisms of change.
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24

Steven, Richards C., Perri Michael G, and American Psychological Association, eds. Relapse prevention for depression. American Psychological Association, 2010.

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25

Brommelhoff, Jessica A. Depression in Dementia Syndromes. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.007.

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Depressive symptoms are common in many dementia syndromes, and depressive disorders are much more common in older adults with dementia than in cognitively intact older adults. Depression may be a risk factor for, or a prodromal feature of, subsequent dementia. Several neuropathological mechanisms have been suggested to explain these relationships, including the role of underlying cerebrovascular risk factors for depression and cognitive impairment. Depression also may be present in dementia as an emotional reaction to cognitive decline, or as a recurrence of early and midlife depression. Diffe
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26

A Map for the Journey: Living Meaningfully With Recurring Depression. PublishAmerica, 2001.

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27

author, Sylvia Louisa Grandin, and Reilly-Harrington Noreen A. author, eds. The bipolar II disorder workbook: Managing recurring depression, hypomania & anxiety. New Harbinger Publications, 2013.

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28

Dietz, Laura J., Jennifer Silk, and Marlissa Amole. Depressive Disorders. Edited by Thomas H. Ollendick, Susan W. White, and Bradley A. White. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190634841.013.19.

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Depressive disorders onset early in development. Depression during childhood and adolescence is associated with serious disruptions in emotional, social, and occupational functioning into adulthood and a high likelihood of recurrence. This chapter discusses clinical manifestations, prevalence, and course of depression presenting in early childhood (ages 3–6), middle childhood/preadolescence (ages 7–12), and adolescence (13–18). An overview is presented of standardized interviews and questionnaires for clinical assessment of depression in children and adolescents; the chapter summarizes researc
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29

Erlinge, David, and Göran Olivecrona. Diagnosis and management of non-STEMI coronary syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0146.

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Acute coronary syndromes are classified as ST segment elevation myocardial infarction (STEMI), non-STEMI (NSTEMI) or unstable angina. Most patients with NSTEMI present with a history of chest pain that has subsided spontaneously before or soon after arrival at the emergency room, but with positive cardiac markers (usually troponin T or I) indicative of myocardial infarction. NSTEMI has a risk of recurrent myocardial infarction of 15–20% and a 15% chance of 1-year mortality. Patients with non-STE-acute coronary syndromes are at similar risk as a STEMI patient at 1 year. The strongest objective
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30

Strain, James J., and Michael Blumenfield, eds. Depression as a Systemic Illness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603342.001.0001.

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Depression has been declared by the World Health Organization in March of 2017 to be the illness with the greatest burden of disease in the world. This volume attempts to examine the current state of our understanding of depressive disorders, from the animal models, allostatie load, patterns of recurrence, effects on other illnesses, for example, cancer, neurological, cardiovascular, wound healing, etc. It is from this perspective that the editors declare that depression is a systemic illness, not just a mental disorder. Therefore, primary care physicians need to know how to diagnose, treat, a
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31

Calarco, Margaret Marie. PSYCHOLOGICAL AND BIOLOGICAL DIFFERENCES AMONG FIRST-EPISODE AND RECURRENTLY DEPRESSED WOMEN DURING DEPRESSION AND RECOVERY. 1992.

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32

Lam, Raymond W. Clinical management. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199692736.003.0005.

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• Clinical management of depression includes screening, assessment, developing a therapeutic alliance, selecting treatment(s), monitoring, and follow up.• The treatment of depression has two phases: the acute phase to achieve full remission of symptoms, and the maintenance phase to prevent relapse and recurrence.•...
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33

Bielecky, Amber. The higher prevalence of major depressive disorder observed in lower socioeconomic status groups: A result of incidence, duration and/or recurrence? 2006.

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34

McCarty, Richard. Stress and Mental Disorders: Insights from Animal Models. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780190697266.001.0001.

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Stress has now been recognized as an important factor in the development or recurrence of various mental disorders, from major depressive disorder to bipolar disorder to anxiety disorders. Stressful stimuli appear to exert their effects by acting upon individuals with susceptible genotypes. Over the past 50 years, animal models have been developed to study these dynamic interactions between stressful stimuli and genetically susceptible individuals during prenatal and postnatal development and into adulthood. This book begins with a discussion of the history of psychiatric diagnosis and the rec
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35

Muralidharan, Anjana, David J. Miklowitz, and W. Edward Craighead. Psychosocial Treatments for Bipolar Disorder. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0010.

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Pharmacological interventions remain the primary treatment for bipolar disorder. However, adjunctive psychosocial interventions have the potential to increase adherence to medication regimens, decrease hospitalizations and relapses, decrease severity of symptoms, improve quality of life, and enhance mechanisms for coping with stress. Group psychoeducation, designed to provide information to bipolar patients about the disorder and its treatment, leads to lower rates of recurrence and greater adherence to medication among remitted bipolar patients at both short- and long-term follow-up. Cognitiv
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36

Markon, Kristian E. From Comorbidity to Constructs. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.014.

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Comorbidity models have become central to psychopathology theory and research, not only because they have clarified our understanding of how and why disorders co-occur but also because they have clarified our understanding of what the disorders are. This chapter reviews basic types of comorbidity models, recurring issues in comorbidity modeling, and discuss emerging issues in the area. Using recent epidemiological, repeated-measures data on depression and anxiety as an example, two different models of comorbidity are compared, one in which comorbidity arises due a shared liability dimension (i
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37

Elleray, Michelle. Robin Hyde. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199609932.003.0024.

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This chapter explores the novels of Iris Guiver Wilkinson, who wrote as Robin Hyde. Three of her novels— Check to Your King (1936), Passport to Hell (1936), and Nor the Years Condemn (1938)—counter claims of historical absence or irrelevance by fictionalizing historical people involved in key moments in New Zealand's history, specifically the mid-nineteenth century efforts to establish New Zealand as a colony, the First World War, and the Great Depression. Meanwhile, with Wednesday's Children (1937), Hyde turns to history's antithesis, fantasy, as an alternative route to investigating New Zeal
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