Relevant bibliographies by topics / Régulations endocrine et paracrine

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Régulations endocrine et paracrine'

Author: Grafiati
Published: 7 December 2024
Last updated: 31 July 2025

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Journal articles on the topic "Régulations endocrine et paracrine"

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Gallo-Payet, N. "Nouveaux concepts sur la régulation de la sécrétion d'aldostérone ; interactions endocrines, paracrines, autocrines et neurocrines." médecine/sciences 9, no. 8-9 (1993): 943. http://dx.doi.org/10.4267/10608/3015.

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Kjekshus, Harald, Otto A. Smiseth, Randi Klinge, Erik Øie, Marit E. Hystad, and Håvard Attramadal. "Regulation of ET: pulmonary release of ET contributes to increased plasma ET levels and vasoconstriction in CHF." American Journal of Physiology-Heart and Circulatory Physiology 278, no. 4 (2000): H1299—H1310. http://dx.doi.org/10.1152/ajpheart.2000.278.4.h1299.

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Endothelin (ET) contributes to the increased systemic vascular resistance and elevated cardiac filling pressures seen in congestive heart failure (CHF). We investigated to what extent ET-mediated vasoconstriction in CHF occurs through an endocrine action of elevated plasma ET or by an autocrine/paracrine mechanism related to induction of vascular ET gene expression. Three weeks of pacing (225 beats/min) induced a marked release of ET-1 from the pulmonary circulation with a sixfold elevation of arterial plasma ET in CHF pigs compared with sham-operated pigs. Arterial plasma ET was the strongest
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Towler, Dwight A. "Bone morphogenetic proteins." Blood 114, no. 10 (2009): 2012–13. http://dx.doi.org/10.1182/blood-2009-06-228544.

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Abstract BMP2 and BMP4 play crucial inductive roles during development. In this issue of Blood, Shao et al demonstrate that an intricate network of paracrine BMP2/4 signals also regulates angiogenesis—and will very likely interact with endocrine BMP cues during wound repair.1
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Tonon, MC, C. Patte, J. Leprince, et al. "Les endozépines : peptides ubiquistes à activités intracrine, autocrine, paracrine, endocrine et exocrine." médecine/sciences 13, no. 5 (1997): 702. http://dx.doi.org/10.4267/10608/441.

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Sallon, C., I. Boulay, D. Logeart-Avramoglou, et al. "Nouvelles perspectives dans l’action paracrine et/ou endocrine des BMPs au niveau hypophysaire." Annales d'Endocrinologie 74, no. 4 (2013): 434. http://dx.doi.org/10.1016/j.ando.2013.07.698.

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Brandt, R. R., D. M. Heublein, M. T. Mattingly, M. R. Pittelkow, and J. C. Burnett. "Presence and secretion of atrial natriuretic peptide from cultured human aortic endothelial cells." American Journal of Physiology-Heart and Circulatory Physiology 268, no. 2 (1995): H921—H925. http://dx.doi.org/10.1152/ajpheart.1995.268.2.h921.

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The endothelium is the production site of several potent vasoactive substances that modulate vascular tone and growth. The present study was undertaken to investigate the presence and secretion of atrial natriuretic peptide (ANP) immunoreactivity from vascular endothelial cells. ANP immunoreactivity was present in cultured human aortic endothelial cells by both immunohistochemical staining and radioimmunoassay. ANP immunoreactivity was also detectable in culture medium from human aortic endothelial cells in low picogram concentrations. These findings suggest that vascular endothelium is a site
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Manshaei, Saba, Thea Willis, Dominic Withers, Jesus Gil, Cynthia Lilian Andoniadou, and Juan Pedro Martinez-Barbera. "Paracrine Signalling From SOX2-Expressing Pituitary Embryonic Cells Is Required for Terminal Differentiation of Hormone-Producing Cells." Journal of the Endocrine Society 5, Supplement_1 (2021): A547—A548. http://dx.doi.org/10.1210/jendso/bvab048.1115.

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Abstract The pituitary gland is the master regulator of the endocrine system, housing six major hormone producing cell types. This gland is derived from Rathke’s Pouch, an invagination of the oral ectoderm. Hormone-producing pituitary cell lineages are derived from a population of embryonic cells expressing SOX2. ZFP36L1/Butyrate Response Factor 1 (BRF1) is an RNA binding protein that binds and targets mRNAs of various cytokines and chemokines for degradation prior to translation, attenuating secretion of inflammatory factors (Herranz et al. 2015). Here, we show that BRF1 is a novel marker exp
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Ojog, Victor. "Morphofunctional traits and reactivity of the portal vein." Moldovan Medical Journal 66, no. 2 (2023): 83–90. http://dx.doi.org/10.52418/moldovan-med-j.66-2.23.13.

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Background: Portal vein is the most enigmatic vessel of our body because it regulates own contractile performances using a special pace-maker mechanism represented by cells of Cajal. The contribution of various metabolic mediators and natural vasotropic agents in the control of the portal blood circuit is much less studied compared to the arterial system in general and the hepatic system in particular. The studies designed on the structure, function, and reactivity of the portal vein in different preconditioning have brought some common but also distinct evidence of the arterial system. Nitric
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Smekalova, Araksiya, Elena K. Montvila, Olga Konovalova, Olga Mityashova, and Irina Lebedeva. "PSII-32 In vitro effect of growth hormone on progesterone production by large preovulatory follicles depends on the hen age and follicular layers interaction." Journal of Animal Science 98, Supplement_4 (2020): 372. http://dx.doi.org/10.1093/jas/skaa278.654.

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Abstract Growth hormone (GH) is an endocrine and paracrine/autocrine regulator of the hen ovarian function, with the GH receptor concentration in the granulosa layer (GL) being maximum in the largest preovulatory follicle (Lebedeva et al. 2004, Biol.Reprod. 71:1174–1181). In the present study, GH effects on in vitro production of progesterone by GL from the two largest yellow follicles (F1 and F2) were investigated due to the hen age and the presence of the theca layer (TL). Young hens with long clutch (YLC, 32–33 week-old, >10 eggs per clutch) and old hens with short clutch (OSC, 74–76
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Bashirov, Farid V., Ilnur I. Salafutdinov, Michail E. Sokolov, et al. "Umbilical Cord Blood Mononuclear Cells for Ex-Vivo Gene Therapy." Blood 132, Supplement 1 (2018): 5797. http://dx.doi.org/10.1182/blood-2018-99-113462.

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Abstract Cell-mediated (ex-vivo) gene therapy for the treatment of adenosine deaminase (ADA)-deficient severe combined immunodeficiency (SCID) had started in 1990 and nowadays it is the first marketing approval of an ex vivo gene therapy in Europe. The method based on ex-vivo transduction of peripheral blood lymphocytes with retroviral vector carrying the functional ADA gene in 2002 have been improved to use hematopoietic stem cell (HSC) for ex-vivo transduction with 100% survival and the evidence of safety and efficacy. Remarkably, umbilical cord blood mononuclear cells (UCB-MC) were successf
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Dissertations / Theses on the topic "Régulations endocrine et paracrine"

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Jeanne, Fabian. "Evοlutiοns des systèmes GΝRΗ et des hοrmοnes glycοprοtéiques dans les cοntrôles endοcrine et paracrine de la spermatοgénèse chez la rοussette, Scyliοrhinus canicula". Electronic Thesis or Diss., Normandie, 2024. http://www.theses.fr/2024NORMC225.

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La spermatogenèse est un processus hautement spécialisé de prolifération et de différenciation cellulaire conduisant à la production de spermatozoïdes haploïdes à partir de cellules souches spermatogoniales diploïdes. Chez les Gnathostomes, les fonctions testiculaires sont principalement contrôlées de façon endocrine par l’axe hypothalamo-hypophyso-gonadique (HHG) impliquant notamment les GnRHs hypothalamiques et les hormones gonadotropes, FSH et LH, dont l’émergence s’est produite à la racine des Vertébrés cartilagineux. De plus, des fonctions paracrines des GnRHs et de la thyrostimuline ont
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Lejeune, Hervé. "Régulations endocrines, paracrines et autocrines des cellules de Leydig : des modèles animaux au testicule humain, étude in vitro." Lyon 1, 1998. http://www.theses.fr/1998LYO1H114.

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Cadranel, Jacques. "1,25 (OH) 2d3 médiateur endocrine et paracrine au cours des granulomatoses pulmonaires." Paris 5, 1993. http://www.theses.fr/1993PA05CD03.

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Dans ce travail, nous montrons tout d'abord une production de 1,25 (OH)2D3 par les cellules pulmonaires fraîchement recueillies par lavage bronchoalvéolaire (LBA) chez des sujets tuberculeux hypercalcémiques (annexe I). Ensuite, après avoir constaté une corrélation entre les capacités de production de la 1,25 (OH)2D3 par ces cellules alvéolaires et les anomalies du bilan phosphocalciques (annexe II), nous avons identifié les cellules responsables de cette production chez des sujets tuberculeux non hypercalcémiques (annexe III). Ainsi, nous avons montré qu'une activité 25(OH)D3-1-a-hydroxylase
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Lafont, Anne-Gaëlle. "Caractérisation et rôle fonctionnel d'une famille de peptides calciotropes (CT et CGRP) : nouvelles données chez un téléostéen et deux mollusques céphalopodes." Paris 6, 2006. http://www.theses.fr/2006PA066190.

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Afin de mieux comprendre leur histoire évolutive, nous avons recherché la présence de la CT, du CGRP et de leurs organes cibles, chez un téléostéen, Anguilla anguilla, et deux céphalopodes, Nautilus macromphalus et Sepia officinalis. Contrairement à l’anguille, aucune molécule biologiquement apparentée à la CT n’a été détectée chez les céphalopodes. La co-localisation du CGRP et de son récepteur dans le système nerveux central de l’anguille et de la seiche, suggère que ce peptide pourrait jouer un rôle de neuromédiateur ou neurotransmetteur comme décrit chez les mammifères. L’implication du CG
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Darbei͏̈da, Halima. "Régulations auto-paracrine et-ou intracrine dans la surrénale ovine : augmentation par les glucocorticoïdes des productions d'AMPc et de corticostéroïdes par les cellules corticosurrénaliennes fœtales et adultes en culture primaire, en réponse à l'ACTH." Bordeaux 1, 1990. http://www.theses.fr/1990BOR10591.

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Les cellules corticosurrenaliennes (cc) adultes et ftales, cultivees en presence de dexamethasone (d) ou de cortisol (f), produisent 3 a 6 fois plus d'ampc et 8 a 20 fois plus de glucocorticoides (g), en reponse a l'acth, que les cellules temoins. Au contraire, la culture des cc, en presence d'aminoglutethimide ou de ru 38486, se traduit par une diminution dose-dependante de la secretion d'ampc induite par l'acth. Les g elevent la reponse maximale en ampc mais ne modifient pas l'ed50 de l'acth; ils n'augmentent pas la reponse ampc a la forskoline. La reponse steroidogene des cc cultivees en pr
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Book chapters on the topic "Régulations endocrine et paracrine"

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"8. Le système immunitaire – inter-relations entre les trois voies de communication nerveuse, immunitaire et endocrine : la neuro-immunoendocrinologie." In Cybernétique et régulations physiologiques. EDP Sciences, 2025. https://doi.org/10.1051/978-2-7598-3674-1.c009.

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Bang, Peter, and Kerstin Hall. "Insulin-like growth factors as endocrine and paracrine hormones." In The Insulin-like Growth Factors. Oxford University PressOxford, 1992. http://dx.doi.org/10.1093/oso/9780198542704.003.0007.

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Abstract Three decades ago, Salmon and Daughaday (1957) proposed that the growth-promoting action of growth hormone was mediated through secondary substances. The search for such factors led to the discovery of the insulin-like growth factors (IGFs). These were discovered as three different biological activities in plasma; the growth hormone-regulated, sulphation factor activity, the non-suppressible insulin-like activity, and multiplication-stimulating activity (Daughaday et al. 1972, 1987).
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Smith, G. P., and J. Gibbs. "The development and proof of the CCK hypothesis of satiety." In Multiple Cholecystokinin Receptors in the CNS. Oxford University PressOxford, 1992. http://dx.doi.org/10.1093/oso/9780198577560.003.0017.

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Abstract In 1973, we reported that the intraperitoneal (i.p.) administration of CCK to rats shortly before a test meal decreased the size of that meal (Gibbs et al. 1973a). The effect was obtained with an impure extract of CCK as well as with the synthetic C-terminal octapeptide CCK-8 (Gibbs et al. 1973b). The effect was dose-related and did not appear to be non-specific or toxic. On the basis of these results, we proposed that endogenous CCK released from the mucosal cells of the upper small intestine by food ingested during a meal acted to terminate that meal by a hormonal mode of action. We
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