Academic literature on the topic 'Renal circulation/physiology'

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Journal articles on the topic "Renal circulation/physiology"

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Rabadi, May M., Tammer Ghaly, Michael S. Goligorksy, and Brian B. Ratliff. "HMGB1 in renal ischemic injury." American Journal of Physiology-Renal Physiology 303, no. 6 (2012): F873—F885. http://dx.doi.org/10.1152/ajprenal.00092.2012.

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Factors that initiate cellular damage and trigger the inflammatory response cascade and renal injury are not completely understood after renal ischemia-reperfusion injury (IRI). High-mobility group box-1 protein (HMGB1) is a damage-associated molecular pattern molecule that binds to chromatin, but upon signaling undergoes nuclear-cytoplasmic translocation and release from cells. Immunohistochemical and Western blot analysis identified HMGB1 nuclear-cytoplasmic translocation and release from renal cells (particularly vascular and tubular cells) into the venous circulation after IRI. Time course
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Franchini, K. G., and A. W. Cowley. "Sensitivity of the renal medullary circulation to plasma vasopressin." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 271, no. 3 (1996): R647—R653. http://dx.doi.org/10.1152/ajpregu.1996.271.3.r647.

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Studies were carried out to determine the effects of physiological changes of plasma arginine vasopressin (AVP) on blood flow distribution in the renal cortex and medulla. Acute decerebration was performed so that studies could be carried out within the low physiological range of circulating AVP. Changes of renal cortical and medullary microcirculatory blood flow were measured with implanted optical fibers and laser-Doppler flowmetry, and total renal blood flow was measured with transit-time ultrasonography. During intravenous infusion of increasing doses of AVP, when plasma AVP was increased
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Cowley, A. W. "Role of the renal medulla in volume and arterial pressure regulation." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 273, no. 1 (1997): R1—R15. http://dx.doi.org/10.1152/ajpregu.1997.273.1.r1.

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The original fascination with the medullary circulation of the kidney was driven by the unique structure of vasa recta capillary circulation, which Berliner and colleagues (Berliner, R. W., N. G. Levinsky, D. G. Davidson, and M. Eden. Am. J. Med. 24: 730-744, 1958) demonstrated could provide the economy of countercurrent exchange to concentrate large volumes of blood filtrate and produce small volumes of concentrated urine. We now believe we have found another equally important function of the renal medullary circulation. The data show that it is indeed the forces defined by Starling 100 years
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Hsueh, Willa A., Yung S. Do, and Pou-Hsiung Wang. "Observations on the renal processing and sorting of prorenin." Canadian Journal of Physiology and Pharmacology 69, no. 9 (1991): 1327–30. http://dx.doi.org/10.1139/y91-196.

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Human prorenin is the biosynthetic precursor of renin. In general, prorenin is enzymatically inactive until it is converted to renin. The kidney is the major source of renin in the circulation, and is also an important source of circulating prorenin. The mechanisms of prorenin sorting and processing to renin in the juxtaglomerular cell may be a determinant of renal renin production. Therefore, our studies have focused on renal enzymes involved in "limited proteolysis" of prorenin to renin and on the morphology of prorenin sorting in the human juxtaglomerular cell.Key words: renin, processing,
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Roman, R. J., and A. P. Zou. "Influence of the renal medullary circulation on the control of sodium excretion." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 265, no. 5 (1993): R963—R973. http://dx.doi.org/10.1152/ajpregu.1993.265.5.r963.

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Although the role of the renal medullary circulation in the control of urinary concentrating ability is well established, its potential influence on tubular sodium reabsorption is not generally recognized. Nearly 30 years ago, changes in the intrarenal distribution of blood flow were first proposed to contribute to the natriuretic response to volume expansion. However, the lack of reliable methods for studying medullary blood flow limited progress in this area. The recent development of laser-Doppler flowmetry and videomicroscopic techniques for the study of the vasa recta circulation has rene
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Alonso-Galicia, Magdalena, John R. Falck, Komandla Malla Reddy, and Richard J. Roman. "20-HETE agonists and antagonists in the renal circulation." American Journal of Physiology-Renal Physiology 277, no. 5 (1999): F790—F796. http://dx.doi.org/10.1152/ajprenal.1999.277.5.f790.

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The present study examined the effects of a series of 20-hydroxyeicosatetraenoic acid (20-HETE) derivatives on the diameter of renal arterioles to determine the structural requirements of the vasoconstrictor response to 20-HETE. The vascular responses to 5-, 8-, 12-, 15-, 19-, 20-, 21-HETEs, arachidonic acid (AA), and saturated, partially saturated, dimethyl, carboxyl, and 19-carbon derivatives of 20-HETE (10−8 to 10−6 M) were assessed in rat renal interlobular arteries (65–125 μm). 20-HETE, 21-HETE, dimethyl-20-HETE, and a partially saturated derivative of 20-HETE, 20-hydroxyeicosa-5( Z),14(
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Reho, John J., Xiaoxu Zheng, James E. Benjamin, and Steven A. Fisher. "Neural programming of mesenteric and renal arteries." American Journal of Physiology-Heart and Circulatory Physiology 307, no. 4 (2014): H563—H573. http://dx.doi.org/10.1152/ajpheart.00250.2014.

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There is evidence for developmental origins of vascular dysfunction yet little understanding of maturation of vascular smooth muscle (VSM) of regional circulations. We measured maturational changes in expression of myosin phosphatase (MP) and the broader VSM gene program in relation to mesenteric small resistance artery (SRA) function. We then tested the role of the sympathetic nervous system (SNS) in programming of SRAs and used genetically engineered mice to define the role of MP isoforms in the functional maturation of the mesenteric circulation. Maturation of rat mesenteric SRAs as measure
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Conrad, Kirk P., and John M. Davison. "The renal circulation in normal pregnancy and preeclampsia: is there a place for relaxin?" American Journal of Physiology-Renal Physiology 306, no. 10 (2014): F1121—F1135. http://dx.doi.org/10.1152/ajprenal.00042.2014.

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During the first trimester of human pregnancy, the maternal systemic circulation undergoes remarkable vasodilation. The kidneys participate in this vasodilatory response resulting in marked increases in renal plasma flow (RPF) and glomerular filtration rate (GFR). Comparable circulatory adaptations are observed in conscious gravid rats. Administration of the corpus luteal hormone relaxin (RLN) to nonpregnant rats and humans elicits vasodilatory changes like those of pregnancy. Systemic and renal vasodilation are compromised in midterm pregnant rats by neutralization or elimination of circulati
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Evans, Roger G., Geoffrey A. Head, Gabriela A. Eppel, Sandra L. Burke, and Niwanthi W. Rajapakse. "Angiotensin II and neurohumoral control of the renal medullary circulation." Clinical and Experimental Pharmacology and Physiology 37, no. 2 (2010): e58-e69. http://dx.doi.org/10.1111/j.1440-1681.2009.05233.x.

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Tarrab, A., C. Mainardi, G. Chatellier, X. Girerd, M.-C. Plainfosse, and P.-F. Plouin. "40 Doppler measurement reproducibility in renal circulation." Journal of Hypertension 9, no. 9 (1991): 884. http://dx.doi.org/10.1097/00004872-199109000-00054.

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Dissertations / Theses on the topic "Renal circulation/physiology"

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Tam, Tin-lap Leonard, and 譚天立. "The influence of acute, chronic or chronic intermittent hypoxia on NO release from the renal circulation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B30401756.

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Lawand, Miguel José. "Comportamento da pressão arterial nos ratos SHR e Wistar-Kyoto expostos ao pneumoperitônio prolongado: estudo experimental com uso do dióxido de carbono para insuflação." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5132/tde-17122008-090833/.

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Para avaliar as repercussões da insuflação prolongada da cavidade peritoneal com gás carbônico sobre a hipertensão arterial essencial, utilizou-se ratos machos espontaneamente hipertensos (SHR) e como normotensos ratos machos Wistar-Kioto (WKY). No total foram utilizados 34 animais, sendo 22 SHRs e 12 WKYs, onde os ratos SHR foram distribuídos aleatoriamente aos grupos G1 e G3. O primeiro grupo (G1) com 12 animais SHRs e o segundo (G2) com 12 animais WKYs foram expostos a pneumoperitônio com dióxido de carbono por 120 minutos, enquanto que o terceiro grupo (G3) com 10 animais SHRs, passou por
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Lario, Fábio de Cerqueira. "Estudo da reserva de perfusão miocárdica pelo ecocardiograma com contraste em tempo real, em indivíduos com hipercolesterolemia grave, antes e após tratamento com inibidores da HMG-CoA redutase." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-31082009-160424/.

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INTRODUÇÃO: A hipercolesterolemia provoca alterações inflamatórias no sistema cardiovascular, induzindo disfunção endotelial e mudanças estruturais na microcirculação, com alterações significativas da homeostase vascular, processo este reversível com o tratamento hipolipemiante. Clinicamente, tais fenômenos podem ser demonstrados pela avaliação da reserva de fluxo coronário e da reatividade vascular periférica. A ecocardiografia de perfusão miocárdica em tempo real (EPMTR) possui características que a tornam ideal para a avaliação da microcirculação coronária, como a utilização de contrastes i
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Santos, João Manoel Theotonio dos. "Avaliação da reserva de fluxo miocárdico pela ecocardiografia com perfusão miocárdica em tempo real em pacientes com disfunção ventricular esquerda, antes e após reabilitação cardiovascular por exercício físico supervisionado." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-23052009-150130/.

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Introdução: A insuficiência cardíaca é uma síndrome clínica, complexa e progressiva, que pode resultar de qualquer distúrbio funcional ou estrutural do coração que altere sua capacidade de enchimento e/ou ejeção, sendo que a maior parte dos pacientes evolui com disfunção ventricular esquerda (DVE). O exercício físico é aceito como um importante coadjuvante no tratamento desta condição clínica por promover significativa melhora da capacidade funcional dos pacientes, entretanto os mecanismos pelos quais isto ocorre ainda não estão totalmente elucidados. Neste contexto, a Ecocardiografia com Perf
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Books on the topic "Renal circulation/physiology"

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Principles of renal physiology. 3rd ed. Chapman & Hall, 1994.

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Lote, Christopher J. Principles of renal physiology. 2nd ed. Chapman and Hall, 1990.

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Principles of Renal Physiology. Springer, 2012.

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Principles of Renal Physiology. Croom Helm, Ltd., 1987.

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Principles of Renal Physiology. Springer, 2000.

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Lote, Christopher J. Principles of Renal Physiology. Springer, 2012.

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Lote, C. J. Principles of Renal Physiology. 4th ed. Springer, 2000.

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Principles of Renal Physiology: Fifth Edition. Springer, 2012.

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Book chapters on the topic "Renal circulation/physiology"

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Braam, Branko, Steven Yip, and William A. Cupples. "Anatomy, Physiology and Pathophysiology of Renal Circulation." In PanVascular Medicine. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-37078-6_146.

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Braam, Branko, Steven Yip, and William A. Cupples. "Anatomy, Physiology, and Pathophysiology of Renal Circulation." In PanVascular Medicine. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-37393-0_146-1.

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Sembulingam, K., and Prema Sembulingam. "Renal Circulation." In Essentials of Medical Physiology. Jaypee Brothers Medical Publishers (P) Ltd., 2012. http://dx.doi.org/10.5005/jp/books/11696_129.

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Sembulingam, K., and Prema Sembulingam. "Renal Circulation." In Essentials of Medical Physiology. Jaypee Brothers Medical Publishers (P) Ltd., 2010. http://dx.doi.org/10.5005/jp/books/11093_51.

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Sembulingam, K., and Prema Sembulingam. "Renal Circulation." In Essentials of Medical Physiology. Jaypee Brothers Medical Publishers (P) Ltd., 2006. http://dx.doi.org/10.5005/jp/books/10283_51.

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Sembulingam, K., and Prema Sembulingam. "Renal Circulation." In Essentials of Physiology for Dental Students. Jaypee Brothers Medical Publishers (P) Ltd., 2016. http://dx.doi.org/10.5005/jp/books/12902_42.

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Sembulingam, K., and Prema Sembulingam. "Renal Circulation." In Essentials of Physiology for Dental Students. Jaypee Brothers Medical Publishers (P) Ltd., 2011. http://dx.doi.org/10.5005/jp/books/11397_50.

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Wolf, Gunter. "Cell physiology of vascular smooth muscle cells and mesangial cells and the impact of this on the control of renal circulation." In Advances in Organ Biology Volume 9. Elsevier, 2000. http://dx.doi.org/10.1016/s1569-2590(00)09056-x.

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Zhu, Yi. "Atomic Force Microscopy Reveals the Role of Vascular Smooth Muscle Cell Elasticity in Hypertension." In Atomic Force Microscopy - Basic Principles to Advanced Applications [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95761.

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The vascular smooth muscle cell (VSMC) mechanical properties not only provide intrinsic cellular functions, but also influence many vascular and circulation functions in physiology. In this report, the VSMCs of thoracic aorta from 16-18 week age Wistar-Kyoto normotensive rats (WKY) and spontaneously hypertensive rats (SHR) were used as research subjects to reveal hypertension mechanism at a single cell level using atomic force microscopy (AFM). The apparent elastic modulus was significantly increased in VSMCs from SHRs compared to those from WKYs. Treatment with cytochalasin D (CD), ML7, Y27632 and lysophosphatidic acid (LPA) modulated VSMC stiffness of WKYs and SHRs. A spectral analysis approach was applied to further investigate the time- dependent change in VSMC elasticity of WKYs and SHRs. This report demonstrated the efficacy of real-time analysis of VSMC elasticity by AFM nano-indentation, and revealed real-time functional differences in biomechanical characteristics of VSMCs with drug treatments.
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Conference papers on the topic "Renal circulation/physiology"

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Fillioe, Seth, Paul Dent, Bin Deng, et al. "Direct noninvasive, real-time observation of thermoregulation physiology: periodic fluctuations in hematocrit and vascular volume in the peripheral circulation." In Advanced Biomedical and Clinical Diagnostic and Surgical Guidance Systems XVII, edited by Anita Mahadevan-Jansen. SPIE, 2019. http://dx.doi.org/10.1117/12.2509125.

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