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Relevant bibliographies by topics / Renal disease; Kidney failure

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Academic literature on the topic 'Renal disease; Kidney failure'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 February 2022

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Journal articles on the topic "Renal disease; Kidney failure"

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Stanisic, Marijana, Rajko Hrvacevic, Zoran Paunic, and Stanko Petrovic. "Nephronophthisis and medullary cystic kidney disease complex." Vojnosanitetski pregled 62, no. 9 (2005): 683–88. http://dx.doi.org/10.2298/vsp0509683s.

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Background. Nephronophthisis and medullary cystic kidney disease complex refers to the genetic heterogeneous group of inherited tubulointerstital nephritis. Nephronophthisis comprises at last 3 clinical manifestations, has the autosomal recessive pattern of inheritance, appears early in life and is the most frequent inherited kidney disease that causes terminal renal failure in childhood, while medullary cystic kidney disease has the autosomal dominant pattern of inheritance, is less frequent, and terminal renal failure appears later in life. These two forms have similar clinical and morphological findings but extrarenal manifestations, the median ages of occurrence of terminal renal failure, and siblings presence help us distinguish these diseases. Case report. In this article we illustrated the case of a 20- years old patient with the suspicion of having complex nephornophthisis and medullary cystic kidney disease based upon mild renal failure, seen in routinely taken laboratory findings and bilateral cysts in corticomedullary region of the kidneys verified on abdominal ultrasound examination. Conclusion. This disease should rise suspicion in children or adolescents with progressive renal failure, a typical clinical manifestation, blood and urine samples results, bilateral cysts in the corticomedullary region of the kidneys seen during ultrasound examination of the kidneys and family inheritance.

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Bereczki, Dániel. "Stroke in chronic renal failure." Orvosi Hetilap 149, no. 15 (April 2008): 691–96. http://dx.doi.org/10.1556/oh.2008.28292.

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Chronic kidney diseases and cardiovascular diseases have several common risk factors like hypertension and diabetes. In chronic renal disease stroke risk is several times higher than in the average population. The combination of classical risk factors and those characteristic of chronic kidney disease might explain this increased risk. Among acute cerebrovascular diseases intracerebral hemorrhages are more frequent than in those with normal kidney function. The outcome of stroke is worse in chronic kidney disease. The treatment of stroke (thrombolysis, antiplatelet and anticoagulant treatment, statins, etc.) is an area of clinical research in this patient group. There are no reliable data on the application of thrombolysis in acute stroke in patients with chronic renal disease. Aspirin might be administered. Carefulness, individual considerations and lower doses might be appropriate when using other treatments. The condition of the kidney as well as other associated diseases should be considered during administration of antihypertensive and lipid lowering medications.

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Leroy, Xavier, Laurent Lemaitre, Alexandre De La Taille, Marc Hazzan, Bruno Delepaul, Jerome Couturier, and Bernard Gosselin. "Bilateral Renal Oncocytosis With Renal Failure." Archives of Pathology & Laboratory Medicine 125, no. 5 (May 1, 2001): 683–85. http://dx.doi.org/10.5858/2001-125-0683-browrf.

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Abstract Oncocytosis is a term recently used to describe diffuse renal involvement by numerous oncocytic nodules. We report herein a case of a 53-year-old man with end-stage renal disease requiring hemodialysis. His kidneys were involved by numerous tumors. Histologic examination revealed more than 100 oncocytomas and an associated papillary renal cell carcinoma in the right kidney.

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Oh, Soo, Rabeet Khan, and Ahmed Ziada. "Polycystic kidney disease." InnovAiT: Education and inspiration for general practice 13, no. 6 (April 1, 2020): 326–35. http://dx.doi.org/10.1177/1755738020910762.

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Polycystic kidney disease (PKD) is a monogenic, hereditary disorder of the kidneys that leads to fluid-filled cysts within the renal tubes. It is one of the most common causes of end-stage renal failure. There are two types, the more common autosomal dominant (ADPKD) and the rarer autosomal recessive (ARPKD). ADPKD mostly presents in adulthood, whereas ARPKD is usually detected during antenatal screening or as a neonate. This article will focus on key points to understand and consider for the holistic management of PKD.

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Rahmawati, Wiwit, Heru Muryawan, and Farah Prabowo. "Renal imaging in children with chronic kidney disease." Paediatrica Indonesiana 53, no. 4 (August 31, 2013): 193. http://dx.doi.org/10.14238/pi53.4.2013.193-9.

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Background Chronic kidney failure is a cause of death inchildren. Diagnosing chronic kidney disease is often made byclinical manifestations, laboratory findings and ultrasonographyor other imaging tests. Early detection of chronic kidney diseaseis needed for education and management of the disease.Objective To describe renal imaging findings and mortality inchildren with chronic kidney disease .Methods This was a cross-sectional study on children with kidneydiseases who were inpatients at Dr. Kariadi Hospital from January2008 to June 2011. Data were taken from medical records. Chronickidney disease was confirmed by clinical manifestations, laboratoryfindings, and radiologic imaging. Renal ultrasound findings weredetermined by the radiologist responsible at that time. Resultswere presented as ft:equency distributions.Results Of 37 chronic kidney disease cases, 27 were males and 10were females. Subjects' most common complaints were dyspnea (7out of 3 7) and edema (30 out of 3 7) . Renal ultrasound imaging ofsubjects with chronic kidney disease yielded the following findings:reduced cortico-medullary differentiation (30 out of 3 7), bilateralechogenic kidneys (21 out of 3 7), reduced renal cortex thickness(4 out of 37) and small-sized kidneys (4 out of 37) . Eight of the37 children died. These 8 subjects had the following radiologicimaging findin gs: both kidneys appeared small in size (4 out ofS),reduced 'renal cortex' thickness (4 out of 8), echogenic kidneys(6 out of 8), and reduced cortico-medullary differentiation (8out of8).Conclusion Renal ultrasound imaging of pediatric subjects withchronic kidney disease revealed findings of reduced corticomedullarydifferentiation, bilateral echogenic kidneys, reducedrenal cortex thickness, and small kidneys bilaterally.

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Fatima, Tanveer, Aurangzeb Afzal, and Sania Ashraf. "CHRONIC KIDNEY DISEASE." Professional Medical Journal 25, no. 06 (June 10, 2018): 887–91. http://dx.doi.org/10.29309/tpmj/2018.25.06.276.

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Introduction: Hemodialysis is a process of removal of waste products andtoxic substances from the body using an extracorporeal system. During the procedure, lotsof hemodynamic and metabolic changes occur in the body as a result of which patientsundergoing hemodialysis may suffer from complications both acutely during or just after dialysisas well as in long term. Objective: To determine the frequencies of various acute intradialyticcomplications in our hemodialysis patients. Study Design: Cross sectional survey. Setting:Lahore General Hospital, Lahore. Period: 3 months from May 2017 to July 2017. Method:End stage renal disease patients on regular hemodialysis in the dialysis unit of a tertiary carehospital. A total of 81 patients were included in the study. Patients with acute renal failure andacute on chronic renal failure were excluded from the survey. Results: Common complicationsobserved in our studied population included muscle cramps (70.7%), post dialysis fatigue(57.3%), back ache (56.1%), intradialytic shivering (57.3%), hypoglycemia (21.4%), hypotension(37.8%), hypertension (8.5%), headache (13.4%), vomiting (13.4%) and anaphylaxis in 2.4%.Conclusion: Hemodialysis is a complex procedure and can cause many complications mostof which are not life threatening. With proper monitoring and immediate treatment thesecomplications can be overcome without causing interruption in hemodialysis.

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Den Hartogh, Danja J., and Evangelia Tsiani. "Health Benefits of Resveratrol in Kidney Disease: Evidence from In Vitro and In Vivo Studies." Nutrients 11, no. 7 (July 17, 2019): 1624. http://dx.doi.org/10.3390/nu11071624.

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Different diseases and disorders that affect the kidneys include, but are not limited to, glomerulonephritis, diabetic nephropathy, polycystic kidney disease, kidney stones, renal fibrosis, sepsis, and renal cell carcinoma. Kidney disease tends to develop over many years, making it difficult to identify until much later when kidney function is severely impaired and undergoing kidney failure. Although conservative care, symptom management, medication, dialysis, transplantation, and aggressive renal cancer therapy are some of the current strategies/approaches to kidney disease treatment, new preventative targeted therapies are needed. Epidemiological studies have suggested that a diet rich in fruits and vegetables is associated with health benefits including protection against kidney disease and renal cancer. Resveratrol, a polyphenol found in grapes and berries, has been reported to have antioxidant, anti-inflammatory, antidiabetic, hepatoprotective, neuroprotective, and anti-cancer properties. The current review summarizes the existing in vitro and in vivo animal and human studies examining the nephroprotective effects of resveratrol.

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Cojocaru, Manole, Inimioara Cojocaru, Isabela Silosi, and Camelia Vrabie. "Kidney Damage in Autoimmune Diseases." Journal of Medical Biochemistry 29, no. 2 (April 1, 2010): 61–65. http://dx.doi.org/10.2478/v10011-010-0007-x.

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Kidney Damage in Autoimmune DiseasesRenal involvement in autoimmunity has many facets. Glomerular, tubular and vascular structures are targeted and damaged as a consequence of autoimmune processes. Immunologically mediated kidney diseases represent the third most common cause of end-stage renal failure (after diabetic and hypertensive nephropathies). Appropriate evalution of patients with immune-mediated kidney diseases requires a meticulous history and physical examination, with particular attention to the urinalysis, tests of renal function and often renal biopsy. The thorough clinician should personally review microscopic urinalysis in any case in which there is a reasonable index of suspicion of immune-mediated renal disease. In this article we propose to highlight recent developments, with particular reference to renal autoimmunity. Systemic lupus erythe-matosus affects many parts of the body: primarily the skin and joints, but also the kidneys. Goodpasture's syndrome involves an autoantibody that specifically targets the kidneys and the lungs. IgA nephropathy is a form of glomerular disease that results when immunoglobulin A (IgA) forms deposits in the glomeruli, where it creates inflammation. Future research could look for how the disease occurs, and how to easily test for its presence so that early treatment could be started.

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Bonavia, Anthony, and Kai Singbartl. "Kidney Injury and Electrolyte Abnormalities in Liver Failure." Seminars in Respiratory and Critical Care Medicine 39, no. 05 (October 2018): 556–65. http://dx.doi.org/10.1055/s-0038-1673616.

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AbstractThe liver and kidney are key organs of metabolic homeostasis in the body and display complex interactions. Liver diseases often have direct and immediate effects on renal physiology and function. Conversely, acute kidney injury (AKI) is a common problem in patients with both acute and chronic liver diseases. AKI in patients with acute liver failure is usually multifactorial and involves insults similar to those seen in the general AKI population. Liver cirrhosis affects and is directly affected by aberrations in systemic and renal hemodynamics, inflammatory response, renal handling of sodium and free water excretion, and additional nonvasomotor mechanisms. Subsequent problems, for example, worsening ascites, hyponatremia, and AKI, often complicate management of patients with chronic progressive liver disease and add to their morbidity and mortality. Thus, AKI must be carefully defined and diagnosed in patients with liver disease. The kidney also plays a pivotal role in balancing acid–base disturbances resulting from advanced liver disease, making AKI in the setting of end-stage liver disease very difficult to manage clinically. While renal dysfunction in these patients often resolves following orthotopic liver transplant, dialysis may be required as a bridge to transplantation to mitigate the metabolic disarray found in these critically ill patients.

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Kelly, Katherine J., Jizhong Zhang, Mingsheng Wang, Shaobo Zhang, and Jesus H. Dominguez. "Intravenous renal cell transplantation for rats with acute and chronic renal failure." American Journal of Physiology-Renal Physiology 303, no. 3 (August 1, 2012): F357—F365. http://dx.doi.org/10.1152/ajprenal.00680.2011.

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Acute kidney injury (AKI) and chronic renal failure (CKD) are the most challenging problems in nephrology. Multiple therapies have been attempted but these interventions have minimal effects on the eventual outcomes, and all too often the result is end-stage renal disease (ESRD). The only effective therapy for ESRD is renal transplantation but only a small fraction of patients receive transplants. In this work we introduce a novel approach to transplantation designed to regenerate kidneys afflicted by severe AKI or CKD: intravenous renal cell transplantation (IRCT) with adult rat primary renal cells reprogrammed to express the SAA gene localized and engrafted in kidneys of rat recipients that had severe AKI or CKD. IRCT significantly resolved renal dysfunction and limited kidney damage, inflammation, and fibrosis. Severe CKD was successfully improved by IRCT using kidney cells from donor rats or by renal cell self-donation in a form of autotransplantation. We propose that IRCT with adult primary renal cells reprogrammed to express the SAA gene can be used to effectively treat AKI and CKD.

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Dissertations / Theses on the topic "Renal disease; Kidney failure"

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Chakrabarti, Shubro. "Mechanisms of fibrosis in feline chronic kidney disease." Thesis, Royal Veterinary College (University of London), 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572451.

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McLelland, Elizabeth Victoria. "Identification of factors associated with, and preventative strategies in, diabetic nephropathy." Thesis, University of Wolverhampton, 1999. http://hdl.handle.net/2436/93526.

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Bergsten, Alicia. "Molecular studies of complications in end stage renal disease : focus on expression and variations of candidate susceptibility genes /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-425-2/.

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Tong, Ka-hang Matthew, and 湯嘉恆. "Cost-effectiveness of screening for chronic kidney disease: a systematic review." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45174179.

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Goldstein, D. Jordi. "Effects of selective manipulation of fatty acids in experimental chronic renal disease." Thesis, Boston University, 1993. https://hdl.handle.net/2144/31818.

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Thesis (D.Sc.N.S.)--Boston University, Henry M. Goldman School of Graduate Dentistry, 1993 (Nutritional Sciences).
Includes bibliography (leaves 176-187)
This dissertation has been presented in two related studies: A. Fish Oil Reduces Proteinuria and Interstitial Injury but not GIomerulosclerosis in the Milan Nomotensive Rat Rats of the Milan Normotensive strain (MNS) spontaneously develop severe Proteinuria and excessive glomemlar thromboxane (Tx)A2 PrOduction at a young age. These are accompanied by podocyte alterations and progressive focal glomerulosclerosis (FGS) and interstitial fibrosis. Since previous studies showed that pharmacologic... [TRUNCATED]

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Yeung, Nga-man, and 楊雅雯. "A guideline of nurse-delivered pre-dialysis education programme for stage 4 chronic kidney disease patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44626988.

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Gregory, Deborah M. "Patients' perceptions of their experiences with end-stage renal disease (ESRD) and hemodialysis treatment." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0031/MQ47421.pdf.

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Xiao, Shen. "Alterations of vascular endothelial nitric oxide synthase activity and substrate availability in chronic renal disease." Morgantown, W. Va. : [West Virginia University Libraries], 1999. http://etd.wvu.edu/templates/showETD.cfm?recnum=794.

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Thesis (Ph. D.)--West Virginia University, 1999.
Title from document title page. Document formatted into pages; contains xvi, 184 p. : ill. Vita. Includes abstract. Includes bibliographical references.

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Yang, Bin. "Involvement of programmed cell death (apoptosis) and its regulators in experimental chronic renal scarring." Thesis, University of Sheffield, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.369894.

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Kennedy, David J. "Cardiovascular complications of ischemic renal disease : the effect of renal dysfunction on cardiac disease and the central role of cardiotonic steroids in the pathogenesis of uremic cardiomyopathy." Connect to full-text via OhioLINK ETD Center, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1145302915.

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Thesis (Ph.D.)--Medical University of Ohio, 2005.
"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Medical Sciences." Major advisor: Joseph I. Shapiro. Includes abstract. Document formatted into pages: v, 265 p. Title from title page of PDF document. Bibliography: pages 52-59,94-100,129-134,171-176,200-263.

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Books on the topic "Renal disease; Kidney failure"

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Lerma, Edgar V., and Matthew R. Weir. Chronic kidney disease and hypertension. New York: Humana Press, 2015.

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Handbook of chronic kidney disease management. Philadelphia: Wolters Kluwer/Lippincott Williams & Wilkins Health, 2011.

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Australian Institute of Health and Welfare. Chronic kidney disease in Australia, 2005. Canberra: Australian Institute of Health and Welfare, 2005.

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Australian Institute of Health and Welfare. An overview of chronic kidney disease in Australia, 2009. Canberra: Australian Institute of Health and Welfare, 2009.

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1941-, Guarnieri G., Panzetta G, and Toiga G, eds. Metabolic and nutritional abnormalities in kidney disease. Basel: Karger, 1992.

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Bomback, Andrew. Chronic kidney disease and hypertension essentials 2011. Sudbury, Mass: Jones and Bartlett Publishers, 2011.

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Bakris, George L. Chronic kidney disease and hypertension essentials 2011. Sudbury, Mass: Jones and Bartlett Publishers, 2011.

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Centers for Medicare & Medicaid Services (U.S.). Medicare for children with chronic kidney disease. Baltimore, MD: Centers for Medicare & Medicaid Services, 2004.

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Halper, Thomas. The misfortunes of others: End-stage renal disease in the United Kingdom. Cambridge: Cambridge University Press, 1989.

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A, Rettig Richard, and Levinsky Norman G. 1929-, eds. Kidney failure and the federal government. Washington, D.C: National Academy Press, 1991.

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Book chapters on the topic "Renal disease; Kidney failure"

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De Broe, Marc. "Vitamin D and Progression of Renal Failure." In Vitamin D in Chronic Kidney Disease, 249–65. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32507-1_14.

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Means, Robert T. "Anemia of Renal Failure/Chronic Kidney Disease." In Anemia in the Young and Old, 147–56. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-96487-4_8.

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Martin, Kevin J., and Esther A. González. "How to Minimize Bone Disease in Renal Failure." In Improving Prognosis for Kidney Disorders, 125–31. Dordrecht: Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-017-1848-6_15.

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Müller, Andreas, and Martin Meier. "Assessment of Renal Volume with MRI: Experimental Protocol." In Methods in Molecular Biology, 369–82. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-0978-1_21.

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AbstractRenal length and volume are important parameters in the clinical assessment of patients with diabetes mellitus, kidney transplants, or renal artery stenosis. Kidney size is used in primary diagnostics to differentiate between acute (rather swollen kidneys) and chronic (rather small kidney) pathophysiology. Total kidney volume is also an established biomarker in studies for the treatment of autosomal dominant polycystic kidney disease (ADPKD). There are several factors influencing kidney size, and there is still a debate on the value of the measured kidney size in terms of renal function or cardiovascular risk. The renal volume is most often calculated by measuring the three axes of the kidney, on the assumption that the organ resembles an ellipsoid. By default, the longitudinal and transverse diameters of the kidney are measured. In animal models renal length and volume1 are also important parameters in the assessment of organ rejection after transplantation and in determination of kidney failure due to renal artery stenosis, recurrent urinary tract infections, or diabetes mellitus. In general total kidney volume (TKV) is a valuable parameter for predicting prognosis and monitoring disease progression in animal models of human diseases like polycystic kidney disease (PKD) or acute kidney injury (AKI) and chronic kidney disease (CKD).This chapter is based upon work from the COST Action PARENCHIMA, a community-driven network funded by the European Cooperation in Science and Technology (COST) program of the European Union, which aims to improve the reproducibility and standardization of renal MRI biomarkers. This analysis protocol is complemented by two separate chapters describing the basic concept and experimental procedure.

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Ahmadmehrabi, Shadi, Hernan Rincon-Choles, and W. H. Wilson Tang. "Heart Failure in a Patient with End-Stage Kidney Disease on Renal Replacement Therapy." In Cardiorenal Syndrome in Heart Failure, 107–20. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-21033-5_8.

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Quaia, Emilio. "Renal Failure." In Radiological Imaging of the Kidney, 745–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-87597-0_29.

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Quaia, Emilio. "Renal Failure." In Radiological Imaging of the Kidney, 739–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54047-9_29.

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Harvey, Elizabeth, and Walid A. Farhat. "Renal Calculi." In Pediatric Kidney Disease, 1135–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-52972-0_44.

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Alexander, R. Todd, and Detlef Bockenhauer. "Renal Tubular Acidosis." In Pediatric Kidney Disease, 973–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-52972-0_36.

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Takahashi, Shori, Michio Nagata, and Hiroshi Saito. "Renal Vasculitis in Children." In Pediatric Kidney Disease, 733–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-52972-0_27.

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Conference papers on the topic "Renal disease; Kidney failure"

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Huang, Zhongping, Jie Ren, and Anilchandra Attaluri. "Experimental Study of a Hybrid Renal Replacement System." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14326.

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Kidney failure is a major issue in the United States. The numbers of kidney failure patients are rapidly increasing with the simultaneous rise in diabetes, obesity and hypertension1. Kidney transplantation has shown excellent results, but insufficiency of donors has been a limiting factor. Most end-stage renal disease (ESRD) patients depend on hemodialysis (HD) for survival which is highly expensive. On an average ESRD patients receive 3 dialysis treatment a week and 4 hours per treatment, i.e., approximately 12 hours a week. Technology has not yet reached to a state where all the kidney functions can be mimicked. The only major kidney function being performed in HD is toxin removal. Even the toxins are not being continuously removed from the patients. To compensate the toxin and fluid removal of a whole week within 12 hours, high volumes of fluid are removed in HD treatments. Patients suffer due to the high fluid removal in a short period of time. Also the patients are restricted from taking fluids between the HD treatments. More frequent HD can improve both survival rate and life quality of patients with chronic kidney disease since normal people has his kidneys functioning continuously. It is a well known fact that daily dialysis offers many benefits over regular intermittent HD1. But providing daily dialysis is not affordable currently. Therefore, new modes of delivering continuous renal support are required.

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Rebouças Filho, Pedro Pedrosa, Suane Pires Pinheiro Da Silva, Jefferson Silva Almeida, Elene Firmeza Ohata, Shara Shami Araujo Alves, and Francisco Dos Santos Hercules Silva. "An Approach to Classify Chronic Kidney Diseases using Scintigraphy Images." In XXXII Conference on Graphics, Patterns and Images. Sociedade Brasileira de Computação - SBC, 2019. http://dx.doi.org/10.5753/sibgrapi.est.2019.8318.

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Chronic kidney diseases cause over a million deaths worldwide every year. One of the techniques used to diagnose the diseases is renal scintigraphy. However, the way that is processed can vary depending on hospitals and doctors, compromising the reproducibility of the method. In this context, we propose an approach to process the exam using computer vision and machine learning to classify the stage of chronic kidney disease. An analysis of different features extraction methods, such as Gray-Level Co-occurrence Matrix, Structural Co-occurrence Matrix, Local Binary Patters (LBP), Hu's Moments and Zernike's Moments in combination with machine learning methods, such as Bayes, Multi-layer Perceptron, k-Nearest Neighbors, Random Forest and Support Vector Machines (SVM), was performed. The best result was obtained by combining LBP feature extractor with SVM classifier. This combination achieved accuracy of 92.00% and F1-score of 91.00%, indicating that the proposed method is adequate to classify chronic kidney disease in two stages, being a high risk of developing end-stage renal failure and other outcomes, and otherwise.

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McNally, Andrew, A. George Akingba, and Philippe Sucosky. "Computational Hemodynamic Assessment of a Novel Modular Anastomotic Valve Device for Improving Hemodialysis Vascular Access Patency." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14560.

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End-stage renal disease (ESRD) occurs as a result of any renal injury that chronically decreases renal excretory and regulatory function. ESRD patients are most commonly treated with hemodialysis (HD) to manage their renal failure while awaiting kidney transplant. Current practices for maintenance of HD vascular access consist of arteriovenous fistulas (AVFs) or grafts (AVGs), which are both fraught with problems that compromise the patency and use of these surgically created shunts. The major cause of shunt failure is thrombosis caused by occlusion of the outflow venous anastomosis and draining vein. Intimal hyperplasia (IH), which consists of the thickening of the innermost layer of the vessel wall, is the initial pathological event leading to shunt stenosis/thrombosis and has been associated with the presence of flow disturbances and abnormal wall shear stress (WSS) at the graft-vein anastomosis. Therefore, the improvement of HD via the enhancement of vascular access patency requires the development of a novel vascular access technology preserving the normal hemodynamics of the native vein.

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Smith, Robert E., Nicole C. Docherty, P. Alex Smith, Duncan J. Maitland, and Alan C. Glowczwski. "A Vascular Access Port for Dialysis With a Polyurethane Foam Seal: A Pilot Study." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3318.

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End-Stage Renal Disease (ESRD) is a condition wherein the kidneys are incapable of removing toxins from the body. Over 660,000 Americans suffer from ESRD, with millions more in the early stages, known as chronic kidney disease [1]. The only cure for ESRD is a kidney transplant, but the majority of patients receive dialysis every 2–3 days to filter their blood while on the transplant waitlist. Efficient dialysis requires approximately 600 mL/min of flow, which is commonly achieved by directly connecting an artery to a vein in the arm. Such a shunt may be created with an intervening prosthetic graft or by suturing the vein to the artery directly (termed an arteriovenous fistula, or AVF). Though accepted as the gold standard, AVF’s may take >6 weeks to heal and become useable, and 35–50% will never become accessible [1]. Needle trauma to the AVF can weaken the vessel wall and produce aneurysms or hematomas, which leak blood, potentially causing infection or clotting off the AVF [2]. These complications are costly: hemodialysis patients on average cost Medicare over $84,000 per year, and Medicare is the primary payer for more than 80% of nearly 500,000 dialysis patients in the U.S. [1]. An improved dialysis access method is needed to address the clinical shortcomings and high costs associated with AVF’s. A device has been developed to improve clinical outcomes and to reduce the failure rates associated with AVF’s. This device is a type of vascular access port which integrates with the external wall of the venous portion of the AVF, providing structural support to the vessel and preventing the types of trauma which lead to aneurysmal dilation or hematoma formation. The top and bottom sections are implanted independently within the patient’s soft tissue, allowing them to separate gradually as the AVF dilates during maturation. The result is a palpable and easy-to-access port which should improve AVF longevity (Figure 1). Two unique design features were identified as key to the success of this vascular access port: 1. Type of membrane or seal 2. Proper tissue integration into the implant This technical brief examines the selection of the proper membrane or seal on the port.

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Evan, Andrew P., Sharon B. Bledsoe, James C. Williams, Andrew P. Evan, James E. Lingeman, and James A. McAteer. "Bone Genes in the Kidney of Stone Formers." In RENAL STONE DISEASE 2: 2nd International Urolithiasis Research Symposium. AIP, 2008. http://dx.doi.org/10.1063/1.2998053.

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Coe, Fredric L. "Introduction: Kidney Stone Research, Lessons From Human Studies." In RENAL STONE DISEASE: 1st Annual International Urolithiasis Research Symposium. AIP, 2007. http://dx.doi.org/10.1063/1.2723554.

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Gochoo, Munkhjargal, Jun-Wei Hsieh, Chien-Hung Lee, Yun-Chih Chen, and Yu-Chi Shih. "Chronic Kidney Disease Stage Classification Using Renal Artery Doppler-Derived Parameters." In 2019 IEEE International Conference on Systems, Man and Cybernetics (SMC). IEEE, 2019. http://dx.doi.org/10.1109/smc.2019.8913899.

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McAteer, James A., Andrew P. Evan, Lynn R. Willis, Bret A. Connors, James C. Williams, Yuri A. Pishchalnikov, and James E. Lingeman. "Shock Wave Injury to the Kidney in SWL: Review and Perspective." In RENAL STONE DISEASE: 1st Annual International Urolithiasis Research Symposium. AIP, 2007. http://dx.doi.org/10.1063/1.2723588.

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Johnson, Emma, Krishna Shah, Ananna Rahman, Kieran Mccafferty, Simon Tiberi, and Heinke Kunst. "Extrapulmonary tuberculosis in patients with chronic kidney disease receiving renal replacement therapy." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2976.

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De La Zerda, D. J., R. A. Alvarez, and J. Villamizar. "Pulmonary Hypertension in Chronic Kidney Disease After Renal Transplant: Risk Factor or Consequence." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5084.

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Reports on the topic "Renal disease; Kidney failure"

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Dominguez, Jesus, K. J. Kelly, and Jizhong Zhang. Intravenous Renal Cell Transplantation for Polycystic Kidney Disease. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada597871.

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Stewart, B. J. Mass Spectrometry Data Set for Renal Cell Carcinoma and Polycystic Kidney Disease Cell Models. Office of Scientific and Technical Information (OSTI), January 2017. http://dx.doi.org/10.2172/1342001.

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Zhang, Yong, Jingjing Li, Anjun Wang, Tian Li, and Yan-Hong Tuo. Effects of intensive blood pressure control on mortality and cardio-renal function in chronic kidney disease patients. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2021. http://dx.doi.org/10.37766/inplasy2021.3.0001.

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