Academic literature on the topic 'Renal Microperfusion'

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Journal articles on the topic "Renal Microperfusion"

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Burg, Maurice B. "Origins of Isolated Tubule Microperfusion Methodology." Physiology 3, no. 4 (1988): 176–80. http://dx.doi.org/10.1152/physiologyonline.1988.3.4.176.

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Understanding of renal function has been facilitated by the technique of perfusion of isolated renal tubule segments in vitro. The basic technology originated in the Laboratory of Kidney and Electrolyte Metabolism of the National Institutes of Health in the early 1960s and then was expanded to apply a variety of analytical methods to single tubules.
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CHEN, Chun-guang, and Yi-ping WANG. "Magnesium lithospermate B ameliorates renal cortical microperfusion in rats1." Acta Pharmacologica Sinica 27, no. 2 (2006): 217–22. http://dx.doi.org/10.1111/j.1745-7254.2006.00225.x.

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Ott, Christian, Joanna M. Harazny, Axel Schmid, et al. "Retinal microperfusion after renal denervation in treatment-resistant hypertensive patients." Clinical Research in Cardiology 104, no. 9 (2015): 782–89. http://dx.doi.org/10.1007/s00392-015-0845-0.

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Capasso, Giovambattista, Caterina Saviano, Francesca Ciani, Florian Lang, Ferdinando Russo, and Natale G. De Santo. "A decrease in renal medullary tonicity stimulates anion transport in Henle’s loop of rat kidneys." American Journal of Physiology-Renal Physiology 274, no. 4 (1998): F693—F699. http://dx.doi.org/10.1152/ajprenal.1998.274.4.f693.

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To investigate the effect of reduction in renal medulla osmolality on loop of Henle (LOH) net bicarbonate reabsorption, clearance and microperfusion experiments were performed on Sprague-Dawley rats. The decrease of renal medulla osmolality was induced by intravenous infusion of either a large dose of mannitol (mannitol protocol) or a hypotonic solution (hypotonic protocol) delivered at a rate to match the sodium and bicarbonate load of the control period. During the mannitol protocol, clearance data demonstrated a rise in glomerular filtration rate (GFR), renal plasma flow, urine pH, and frac
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Regus, Susanne, Felix Klingler, Werner Lang, et al. "Pilot study using intraoperative fluorescence angiography during arteriovenous hemodialysis access surgery." Journal of Vascular Access 20, no. 2 (2018): 175–83. http://dx.doi.org/10.1177/1129729818791989.

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Introduction: In this pilot study, we used indocyanine green fluorescence angiography during hemodialysis access surgery. The aim was to evaluate its relevance as a diagnostic tool to visualize changes in hand microperfusion. Patients and methods: In this prospective single-center study, 47 adult patients (33 male, 14 female) with renal disease (24 preemptive, 23 endstage) were enrolled. Surgical creation of an arteriovenous fistula was performed (22 forearm, 25 upper arm). Microperfusion of the ipsilateral hand and fingers was evaluated intraoperatively using indocyanine green fluorescence an
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Deng, Aihua, and Scott C. Thomson. "Renal NMDA receptors independently stimulate proximal reabsorption and glomerular filtration." American Journal of Physiology-Renal Physiology 296, no. 5 (2009): F976—F982. http://dx.doi.org/10.1152/ajprenal.90391.2008.

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N-methyl-d-aspartate receptors (NMDA) are expressed in the kidney, where little is known of their functional role. Several series of micropuncture experiments were performed in hydropenic rats using the NMDA channel blocker, MK801, and the NMDA coagonist, l-glycine, to probe NMDA for effects on single-nephron glomerular filtration rate (SNGFR) and proximal reabsorption ( Jprox). During intravenous infusion of MK801 or l-glycine, Henle's loop was perfused to manipulate SNGFR via tubuloglomerular feedback (TGF), thereby facilitating analysis of glomerulotubular balance. To confirm local actions
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Walter, S. J., D. G. Shirley, and R. J. Unwin. "Effect of vasopressin on renal lithium reabsorption: a micropuncture and microperfusion study." American Journal of Physiology-Renal Physiology 271, no. 1 (1996): F223—F229. http://dx.doi.org/10.1152/ajprenal.1996.271.1.f223.

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Micropuncture techniques were used to investigate the nephron site(s) responsible for the vasopressin-induced reductions in lithium clearance and fractional lithium excretion (FELi) in anesthetized Brattleboro rats lacking endogenous vasopressin. In rats treated intravenously with the vasopressin analogue 1-desamino-8-D-arginine vasopressin (DDAVP; 40 pg/min), FELi was significantly lower than in untreated animals (0.23 +/- 0.01 vs. 0.28 +/- 0.02, P < 0.05). Free-flow micropuncture showed that fractional lithium delivery (FDLi) to late proximal convolutions was identical in the two groups,
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Kirchner, K. A. "Increased loop chloride uptake precedes hypertension in Dahl salt-sensitive rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 262, no. 2 (1992): R263—R268. http://dx.doi.org/10.1152/ajpregu.1992.262.2.r263.

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When studied at equivalent renal perfusion pressures, loop segment chloride reabsorption is greater in hypertensive Dahl salt-sensitive (S) than Dahl salt-resistant (R) rats. To determine whether this difference in loop reabsorption is present before the onset of hypertension, volume expanded and euvolemic Dahl rats maintained on low NaCl diets were examined using micropuncture and in vivo microperfusion techniques. Neuroendocrine differences between groups were eliminated by renal denervation and fixing plasma aldosterone, norepinephrine, and vasopressin levels. After volume expansion, urinar
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Fischer, Krisztina, F. Can Meral, Yongzhi Zhang, et al. "High-resolution renal perfusion mapping using contrast-enhanced ultrasonography in ischemia-reperfusion injury monitors changes in renal microperfusion." Kidney International 89, no. 6 (2016): 1388–98. http://dx.doi.org/10.1016/j.kint.2016.02.004.

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Bank, N., H. S. Aynedjian, and B. F. Mutz. "Microperfusion study of proximal tubule bicarbonate transport in maleic acid-induced renal tubular acidosis." American Journal of Physiology-Renal Physiology 250, no. 3 (1986): F476—F482. http://dx.doi.org/10.1152/ajprenal.1986.250.3.f476.

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Microperfusion studies were carried out in rats to examine the abnormality in proximal tubule HCO3- transport caused by maleic acid administration. Permeability of the proximal tubule to HCO-3 was measured by perfusing proximal tubules with a HCO3- -free low-buffer isotonic equilibrium solution containing acetazolamide after plasma [HCO3-] had been raised by intravenous NaHCO3 infusion. Insulin recovery in the collected perfusate was approximately 100% in control and maleic acid-treated rats. CO2 influx measured by microcalorimetry was not significantly different in control vs. maleic acid-tre
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Dissertations / Theses on the topic "Renal Microperfusion"

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Bailey, Matthew Alexander. "Studies in renal cation transport in potassium replete and potassium-depleted rats." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.264703.

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Lima, Lucilia Maria Abreu Lessa Leite. "Efeito da uroguanilina sobre o transporte de hidrogênio em túbulos renais de rins de rato e em linhagens de células proximais e distrais." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-09022010-094843/.

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Uroguanilina (UGN) é um peptídeo normalmente sintetizado no intestino que modula o balanço de sódio através de ações renais. Investigamos os efeitos e os mecanismos de sinalização envolvidos na ação da UGN sobre a secreção de H+, em túbulos renais de ratos e em células LLC-PK1 (proximais) e MDCK-C11 (distais). Nos estudos in vivo foi utilizada a técnica de microperfusão estacionária, na qual medimos a secreção de H+ em túbulos proximais e distais de rins de rato, utilizando um microeletrodo sensível a H+. Nos estudos in vitro, para medir a atividade de NHE3 e H+-ATPase, utilizamos microscopia
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Lai, Enyin. "Interaction between Adenosine and Angiotensin II in Renal Afferent Arterioles of Mice." Doctoral thesis, Uppsala University, Department of Medical Cell Biology, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7702.

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<p>Renal arterioles represent the most important effecter site in the control of renal perfusion and filtration. Adenosine (Ado), angiotensin II (Ang II) and nitric oxide (NO) interact in modulating arteriolar tone. The present work investigates the mechanism of this interaction. We tested the hypothesis that AT<sub>1</sub> receptor (AT<sub>1</sub>AR) mediated NO release in isolated perfused afferent arterioles. Further, special attention was given to mechanisms of Ado-Ang II -interactions.</p><p>We found (I) that Ang II specifically induces NO release via AT<sub>1</sub>AR in arterioles. The e
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Gao, Xiang. "Local Purinergic Control of Arteriolar Reactivity in Pancreatic Islets and Renal Glomeruli." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk cellbiologi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-230770.

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Local control of regional blood flow is exerted mainly through the arterioles. An adequate minute-to-minute regulation of blood perfusion of the kidney and the pancreas is obtained by the modulation of arteriolar reactivity, which will influence the organ function. The importance of purinergic signaling in this concept has been addressed, with special emphasis on the role of the adenosine A1 receptor. The effects of adenosine on two specialized vascular beds, namely the renal glomerulus and the pancreatic islets, have been examined. Characteristic for these regional circulations is their very
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Branco, Regiane Cardoso Castelo. "Efeito da angiotensina-(1-7) no fluxo reabsortivo de bicarbonato (JHCO3-) e na concentração citosólica de cálcio ([Ca2+]i): estudo por microperfusão tubular proximal, in vivo." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-25072012-135726/.

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O estudo avaliou os efeitos agudos da Ang-(1-7) na reabsorção de bicarbonato (JHCO3-) no túbulo proximal cortical de rato, in vivo, medindo o pH intratubular pelo microeletródio sensível a H+. O JHCO3- controle é 2,84 ± 0,08 nmol. cm-2. s-1 (49), a Ang-(1-7; 10-12 ou 10-9 M) o reduz (35 ou 61 %) e a Ang-(1-7; 10-6 M) o eleva (56 %). A inibição do receptor Mas (por A779) eleva o JHCO3- (30 %), abole o efeito inibidor da Ang-(1-7), mas não afeta seu efeito estimulador. A inibição do NHE3 (por S3226) diminui o JHCO3- (45 %), não altera o efeito inibidor da Ang-(1-7), mas transforma seu efeito est
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Pergher, Patrícia e. Silva. "Efeitos não-genômicos dos hormônios esteróides - aldosterona e corticosterona - sobre a acidificação do túbulo proximal (S2) de ratos: estudos de microperfusão tubular e capilar, in vivo." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-10122010-144456/.

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O objetivo foi determinar se aldosterona e corticosterona agem sobre a acidificação do túbulo proximal e se esses efeitos são genômicos e/ou não-genômicos. A reabsorção de HCO3- foi avaliada por microperfusão estacionária. Aldosterona e corticosterona perfundidas na luz tubular causaram aumento significante do JHCO3-. Na presença de etanol, actinomicina D, cicloheximida ou espironolactona, o JHCO3- foi estatisticamente igual ao valor controle (2,84 ± 0,079 nmol.cm-2.s-1). RU486 sozinho inibiu o efeito estimulador da aldosterona e corticosterona. Losartan não alterou o JHCO3-. Concanomicina ou
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Book chapters on the topic "Renal Microperfusion"

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Windhager, Erich E. "Micropuncture and Microperfusion." In Renal Physiology. Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4614-7545-3_4.

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Oza, Narendra B., Catherine M. Murphy, Debbie Beasley, Norman G. Levinsky, and James S. Kaufman. "A Micro-Kininogenase Assay for Studies of Kallikrein in Renal Micropuncture/Microperfusion." In Advances in Experimental Medicine and Biology. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4615-9543-4_85.

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Conference papers on the topic "Renal Microperfusion"

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Heeman, Wido, Hanno Maassen, Joost Calon, et al. "Real-time visualization of renal microperfusion using laser speckle contrast imaging." In Biomedical Applications of Light Scattering XI, edited by Adam Wax and Vadim Backman. SPIE, 2021. http://dx.doi.org/10.1117/12.2577538.

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