Relevant bibliographies by topics / Renal tissue regeneration

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Renal tissue regeneration'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2022

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Journal articles on the topic "Renal tissue regeneration"

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Tsuji, Kenji, and Shinji Kitamura. "Trophic Factors from Tissue Stem Cells for Renal Regeneration." Stem Cells International 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/537204.

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Stem cell therapies against renal injury have been advancing. The many trials for renal regeneration are reported to be effective in many kinds of renal injury models. Regarding the therapeutic mechanism, it is believed that stem cells contribute to make regeneration via not only direct stem cell differentiation in the injured space but also indirect effect via secreted factors from stem cells. Direct differentiation from stem cells to renal composed cells has been reported. They differentiate to renal composed cells and make functions. However, regarding renal regeneration, stem cells are discussed to secrete many kinds of growth factors, cytokines, and chemokines in paracrine or autocrine manner, which protect against renal injury, too. In addition, it is reported that stem cells have the ability to communicate with nearby cells via microvesicle-related RNA and proteins. Taken together from many reports, many secreted factors from stem cells were needed for renal regeneration orchestrally with harmony. In this review, we focused on the effects and insights of stem cells and regenerative factors from stem cells.
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Yokote, Shinya, Shuichiro Yamanaka, and Takashi Yokoo. "De NovoKidney Regeneration with Stem Cells." Journal of Biomedicine and Biotechnology 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/453519.

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Recent studies have reported on techniques to mobilize and activate endogenous stem-cells in injured kidneys or to introduce exogenous stem cells for tissue repair. Despite many recent advantages in renal regenerative therapy, chronic kidney disease (CKD) remains a major cause of morbidity and mortality and the number of CKD patients has been increasing. When the sophisticated structure of the kidneys is totally disrupted by end stage renal disease (ESRD), traditional stem cell-based therapy is unable to completely regenerate the damaged tissue. This suggests that whole organ regeneration may be a promising therapeutic approach to alleviate patients with uncured CKD. We summarize here the potential of stem-cell-based therapy for injured tissue repair andde novowhole kidney regeneration. In addition, we describe the hurdles that must be overcome and possible applications of this approach in kidney regeneration.
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Little, Melissa H., Alexander N. Combes, and Minoru Takasato. "Understanding kidney morphogenesis to guide renal tissue regeneration." Nature Reviews Nephrology 12, no. 10 (August 30, 2016): 624–35. http://dx.doi.org/10.1038/nrneph.2016.126.

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Bruno, Stefania, Stefano Porta, and Benedetta Bussolati. "Extracellular vesicles in renal tissue damage and regeneration." European Journal of Pharmacology 790 (November 2016): 83–91. http://dx.doi.org/10.1016/j.ejphar.2016.06.058.

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Salama, Samir A., Hany H. Arab, and Ibrahim A. Maghrabi. "Troxerutin down-regulates KIM-1, modulates p38 MAPK signaling, and enhances renal regenerative capacity in a rat model of gentamycin-induced acute kidney injury." Food & Function 9, no. 12 (2018): 6632–42. http://dx.doi.org/10.1039/c8fo01086b.

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Piron, Annie, Isabelle Leonard, Denis Nonclercq, Gerard Toubeau, Paul Falmagne, Jeanine-Anne Heuson-Stiennon, and Guy Laurent. "In vitro demonstration of a mitogenic activity in renal tissue extracts during regenerative hyperplasia." American Journal of Physiology-Renal Physiology 274, no. 2 (February 1, 1998): F348—F357. http://dx.doi.org/10.1152/ajprenal.1998.274.2.f348.

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Normal rat kidney (NRK-52E) cells, an established cell line of renal origin, were used as a bioassay system to reveal a possible mitogenic activity in tissue extracts prepared from kidneys undergoing tubular regeneration. Acute tubular injury was induced in female Wistar rats by a 4-day treatment with gentamicin at daily doses of 50 or 100 mg/kg twice daily. Animals were killed either 1 or 4 days after cessation of gentamicin administration. Proximal tubule regeneration in treated animals was confirmed by morphological examination after proliferating cell nuclear antigen staining. Tissue extracts from regenerating kidneys stimulated DNA synthesis in growth-arrested cells to a higher extent than extracts from intact kidneys. Sera from treated and control animals showed no difference with respect to mitogenic activity. The mitogenic effect of tissue extracts was sensitive to the tyrosine kinase inhibitor tyrphostin A46. The cell proliferative response to regenerating kidney extracts, but not that to intact kidney extracts, was partly suppressed by the addition of anti-insulin-like growth factor I (anti-IGF-I) antiserum. These data indicate that nephrogenic repair entails an elevation of biologically active IGF-I in kidney tissue.
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Bussolati, Benedetta, Akito Maeshima, Janos Peti-Peterdi, Takashi Yokoo, and Laura Lasagni. "Renal Stem Cells, Tissue Regeneration, and Stem Cell Therapies for Renal Diseases." Stem Cells International 2015 (2015): 1–2. http://dx.doi.org/10.1155/2015/302792.

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Lee, Sang Jin, Hung-Jen Wang, Tae-Hyoung Kim, Jin San Choi, Gauri Kulkarni, John D. Jackson, Anthony Atala, and James J. Yoo. "In Situ Tissue Regeneration of Renal Tissue Induced by Collagen Hydrogel Injection." STEM CELLS Translational Medicine 7, no. 2 (January 29, 2018): 241–50. http://dx.doi.org/10.1002/sctm.16-0361.

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Cho, Y. S., H. C. Moon, C. Y. Choi, S. S. Kim, B. S. Kim, J. H. Han, K. J. Joo, C. H. Kwon, and H. J. Park. "32 Renal tissue regeneration using cellular transplantation in rats." European Urology Supplements 3, no. 2 (February 2004): 10. http://dx.doi.org/10.1016/s1569-9056(04)90034-6.

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Cho, Young Sam, Hong Chul Moon, Sang Su Kim, Cha Yang Choi, Byung Su Kim, Jeong Ho Han, Kwan lung Joo, Chil Hun Kwon, and Heung Jae Park. "1842: Renal Tissue Regeneration Using Cellular Transplantation in Rats." Journal of Urology 171, no. 4S (April 2004): 487. http://dx.doi.org/10.1016/s0022-5347(18)39034-7.

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Dissertations / Theses on the topic "Renal tissue regeneration"

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Poornejad, Nafiseh. "Decellularization and Recellularization Processes for Whole Porcine Kidneys." BYU ScholarsArchive, 2017. https://scholarsarchive.byu.edu/etd/6554.

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Concern over kidney disease has increased dramatically in recent years within the medical community. It is estimated that approximately one in fifteen Americans, nearly 20 million people, experience chronic kidney disease with most of those diagnosed progressing to kidney failure. The ultimate treatment available for end stage renal failure is whole kidney transplantation. However, there are very few kidneys available for patients to receive and those patients who are fortunate enough to receive an organ must remain on immunosuppressive medication for the remainder of their lives. The United States Department of Health & Human Services have reported that 18 people die every day while on the waiting list for organ donations. The treatment is fairly successful as 69% of patients who receive a kidney transplant are still alive 5 years after the transplant. Tissue engineered organs could be a promising alternative for whole organ transplantation. The overall objective is to repopulate appropriate decellularized scaffolds from pigs, which are not immunogenic, with a patient's own cells to achieve a functional organ. Therefore, there would be an inexhaustible source of organs ready for transplantation without the risk of immune rejection. The naturally obtained scaffolds devoid of immunogens are a potential matrix to create artificial kidneys. Repopulation of decellularized rat kidneys with renal progenitor cells has been reported in previous studies. This dissertation reports the scale-up of the previous technology and building of partially functional human-sized kidneys. In the first step, we investigated various cell lysing agents and developed an automated decellularization procedure for whole porcine kidney decellularization. We also developed a preservation method for native and decellularized kidneys to avoid spoilage before and after decellularization. We also developed a decontamination procedure for whole porcine kidneys. Finally, we recellularized whole porcine kidney scaffolds with renal epithelial cells and achieved partial repopulation of the renal structure.
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Books on the topic "Renal tissue regeneration"

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Tsai, Ching-Wei, Sanjeev Noel, and Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.

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Acute kidney injury (AKI), regardless of its aetiology, can elicit persistent or permanent kidney tissue changes that are associated with progression to end-stage renal disease and a greater risk of chronic kidney disease (CKD). In other cases, AKI may result in complete repair and restoration of normal kidney function. The pathophysiological mechanisms of renal injury and repair include vascular, tubular, and inflammatory factors. The initial injury phase is characterized by rarefaction of peritubular vessels and engagement of the immune response via Toll-like receptor binding, activation of macrophages, dendritic cells, natural killer cells, and T and B lymphocytes. During the recovery phase, cell adhesion molecules as well as cytokines and chemokines may be instrumental by directing the migration, differentiation, and proliferation of renal epithelial cells; recent data also suggest a critical role of M2 macrophage and regulatory T cell in the recovery period. Other processes contributing to renal regeneration include renal stem cells and the expression of growth hormones and trophic factors. Subtle deviations in the normal repair process can lead to maladaptive fibrotic kidney disease. Further elucidation of these mechanisms will help discover new therapeutic interventions aimed at limiting the extent of AKI and halting its progression to CKD or ESRD.
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Hastie, Nick, and Eve Miller-Hodges. WT1 and its disorders. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0329_update_001.

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Mutations in the Wilms tumour suppressor gene, WT1, are associated with Wilms tumour in childhood. However, in addition WT1 has a key role in renal development, emerging roles in podocyte function, and a potential role in tissue regeneration. An understanding of WT1 is of increasing importance to clinical practice. WT1 is a complex gene with multiple isoforms. It is crucial for normal embryonic development, especially kidney development, where it is necessary for mesenchymal-to-epithelial transition to form the nephron. WT1 mutations lead to abnormalities in renal and genitourinary development, causing diseases such as Denys–Drash syndrome and Frasier syndrome as well as Wilms tumour. Recently, WT1 mutations have been recognized as a significant cause of isolated steroid-resistant nephrotic syndrome in children and young adults, without other associated syndromic features. WT1 continues to be expressed in adult podocytes, where it acts as a transcriptional activator of many podocyte genes. However, the specific role of WT1 in adult podocyte function remains poorly understood.
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Book chapters on the topic "Renal tissue regeneration"

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Kimelman, Nadav Bleich, Ilan Kallai, Dmitriy Sheyn, Wafa Tawackoli, Zulma Gazit, Gadi Pelled, and Dan Gazit. "Real-Time Bioluminescence Functional Imaging for Monitoring Tissue Formation and Regeneration." In Methods in Molecular Biology, 181–93. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-556-9_14.

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Wang, H. J., S. J. Lee, A. Atala, and J. J. Yoo. "In Situ Renal Regeneration." In In Situ Tissue Regeneration, 369–82. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-802225-2.00019-2.

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Li, Peng, Xiongbing Lu, Junhong Deng, Andrea Peloso, and Yuanyuan Zhang. "Urine Progenitor Cells for Potential Application in Renal Tissue Repair." In Kidney Transplantation, Bioengineering and Regeneration, 1067–73. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-801734-0.00077-1.

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Yoshikawa, Takahisa, Yuki Sato, and Motoko Yanagita. "Heterogeneity of Fibroblasts in Healthy and Diseased Kidneys." In Fibroblasts - Advances in Cancer, Autoimmunity and Inflammation [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99492.

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Chronic kidney disease (CKD) is a worldwide health problem affecting 9.1% of the world’s population. The treatments to prevent the progression of CKD remain limited, however. Resident fibroblasts in the kidneys play crucial roles in the pathological conditions commonly recognized in CKD, such as renal fibrosis, renal anemia, and peritubular capillary loss. Fibroblasts in the kidney provide structural backbone by producing extracellular matrix proteins and produce erythropoietin for normal hematopoiesis under physiological conditions. In the diseased condition, however, fibroblasts differentiate into myofibroblasts that produce excessive extracellular matrix proteins at the cost of the inherent erythropoietin-producing abilities, resulting in renal fibrosis and renal anemia. Pericytes, which are mesenchymal cells that enwrap peritubular capillaries and highly overlap with resident fibroblasts, detach from peritubular capillary walls in response to kidney injury, resulting in peritubular capillary loss and tissue hypoxia. Several reports have demonstrated the beneficial roles of fibroblasts in the regeneration of renal tubules Renal fibroblasts also have the potential to differentiate into a proinflammatory state, producing various cytokines and chemokines and prolonging inflammation by forming tertiary lymphoid tissues, functional lymphoid aggregates, in some pathological conditions. In this article, we describe the heterogenous functions of renal fibroblasts under healthy and diseased conditions.
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"Analyzing Gene Expression through Real Time PCR while Neo-tissue Regeneration using Developed Tissue Constructs." In Protocols used in Molecular Biology, edited by Divakar Singh, Tarun Minocha, Satyavrat Tripathi, Rupika Sinha, Shubhankar Anand, Hareram Birla, Vivek Kumar Pandey, et al., 15–34. BENTHAM SCIENCE PUBLISHERS, 2020. http://dx.doi.org/10.2174/9789811439315120010006.

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Real-time PCR offers a wide area of application to analyze the role of gene activity in various biological aspects at the molecular level with higher specificity, sensitivity and the potential to troubleshoot with post-PCR processing and difficulties. With the recent advancement in the development of functional tissue graft for the regeneration of damaged/diseased tissue, it is effective to analyze the cell behaviour and differentiation over tissue construct toward specific lineage through analyzing the expression of an array of specific genes. With the ability to collect data in the exponential phase, the application of Real-Time PCR has been expanded into various fields such as tissue engineering ranging from absolute quantification of gene expression to determine neo-tissue regeneration and its maturation. In addition to its usage as a research tool, numerous advancements in molecular diagnostics have been achieved, including microbial quantification, determination of gene dose and cancer research. Also, in order to consistently quantify mRNA levels, Northern blotting and in situ hybridization (ISH) methods are less preferred due to low sensitivity, poor precision in detecting gene expression at a low level. An amplification step is thus frequently required to quantify mRNA amounts from engineered tissues of limited size. When analyzing tissue-engineered constructs or studying biomaterials–cells interactions, it is pertinent to quantify the performance of such constructs in terms of extracellular matrix formation while in vitro and in vivo examination, provide clues regarding the performance of various tissue constructs at the molecular level. In this chapter, our focus is on Basics of qPCR, an overview of technical aspects of Real-time PCR; recent Protocol used in the lab, primer designing, detection methods and troubleshooting of the experimental problems.
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Kumar, Vineet, Shruti D. Vora, Foram A. Asodiya, Naveen Kumar, and Anil K. Gangwar. "Fourier Transform Infrared Spectroscopy of the Animal Tissues." In Real Perspective of Fourier Transforms and Current Developments in Superconductivity. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.94582.

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Animal tissues are extensively used as scaffolds for tissue engineering and regenerative therapies. They are typically subjected to decellularization process to obtain a cell-free extracellular matrix (ECM) scaffolds. It is important to identify chemical structure of the ECM scaffolds and Fourier transform infrared (FTIR) appears to be a technique of choice. In this chapter, FTIR spectra of native and decellularized buffalo aortae, buffalo diaphragms, goat skin, and native bovine cortical bone are presented. The transmittance peaks are that of organic collagen amide A, amide B, amide I, amide II and amide III chemical functional groups in both native and decellularized aortae, diaphragms and skin. In bone, the transmittance peaks are that of inorganic ν1, ν3 PO43−, OH− in addition to organic collagen amide A, amide B, amide I, amide II and amide III chemical functional groups. These important transmittance peaks of the tissue samples will help researchers in defining the chemical structure of these animal tissues.
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"6 Real-Time Demand: Information, Regeneration, and Organ Markets." In Tissue Economies, 160–80. Duke University Press, 2020. http://dx.doi.org/10.1515/9780822388043-008.

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Rama, R. R., and S. Skatulla. "Towards the Real-Time Modeling of the Heart." In Advances in Biomechanics and Tissue Regeneration, 139–80. Elsevier, 2019. http://dx.doi.org/10.1016/b978-0-12-816390-0.00008-x.

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Bastain, Theresa M., Lu Gao, and Frank D. Gilliland. "Stem Cells in the Real World: Environmental Impacts." In Stem Cells, Tissue Engineering and Regenerative Medicine, 485–96. WORLD SCIENTIFIC, 2015. http://dx.doi.org/10.1142/9789814612784_0023.

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Ahmed, Zaki, Kanwal Bilal, and Asad Ullah Khan. "Emerging Technologies as a Tool for Development of Human Values and Global Peace." In Advances in Educational Marketing, Administration, and Leadership, 267–304. IGI Global, 2016. http://dx.doi.org/10.4018/978-1-5225-0078-0.ch015.

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Nanotechnology is one of the unique technologies, which have the potential to narrow down the rich, and poor divide in nations. Nanotechnology has the potential to bridge the gap between developed and developing countries by developing a closer relationship to reduce involuntary sufferings. This can be testified by the proven role of nanotechnology in remediation of environment, providing health, clean water, harvesting water from air, eco-friendly housing from green nanomaterials, eradicating malaria, water borne diseases, human tissue regeneration, increasing agricultural yields, as generate innovations with embedded human values. The morally neutral threatening technologies like nanotechnology would lead to circumvent socio-political opposition, the rich and poor divide and address the involuntary sufferings by providing human value based solutions. Nanotechnology is the tool given by nature to transform the silos mentality to a collaborative mentality for real world problem solving and respond to the challenges of human sufferings.
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Conference papers on the topic "Renal tissue regeneration"

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Whited, Bryce M., Matthias C. Hofmann, Chris G. Rylander, Aaron S. Goldstein, Joseph W. Freeman, Mark Furth, Shay Soker, Ge Wang, Yong Xu, and Marissa N. Rylander. "Non-Destructive Real-Time Imaging of Cell Seeded Tissue Engineered Scaffolds." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53721.

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The use of tissue engineered scaffolds in combination with progenitor cells has emerged as a promising strategy to restore or replace tissues damaged by disease or trauma. In addition to being biocompatible and exhibiting appropriate mechanical properties, scaffolds must be designed to sustain cell attachment, proliferation, and differentiation to ultimately produce the desired tissue once implanted in the patient [1]. Conventional techniques used to assess successful scaffold design include cell viability stains, DNA assays, and histological sectioning/staining. While significant information can be gained from using these methodologies, they are destructive to the sample and only provide snapshots of scaffold and cell development at a limited number of time points. Consequently, key temporal and spatial information relating to tissue regeneration in the scaffold is lost utilizing these techniques. Thus, the ability to non-destructively monitor cell viability, proliferation, and differentiation in real-time is of great importance for scaffold design and tissue engineering [2].
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Debbaut, Charlotte, David De Wilde, Christophe Casteleyn, Pieter Cornillie, Manuel Dierick, Luc Van Hoorebeke, Diethard Monbaliu, Ye-Dong Fan, and Patrick Segers. "Electrical Analog Models to Simulate the Impact of Partial Hepatectomy on Hepatic Hemodynamics." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14266.

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Due to the growing shortage of donor livers, more patients are waiting for liver transplantation. Efforts to expand the donor pool include the use of living donor liver transplantation (LDLT) and split liver transplantation. LDLT involves a healthy person undergoing a partial hepatectomy to donate a part of his liver to a patient with severe liver failure. Afterwards, the regenerative capacity of the organ allows the livers of both donor and recipient to regrow to normal liver masses. The procedure is not without risk as serious complications may occur (such as cholestasis, ascites, gastrointestinal bleeding and renal impairment). An inadequate liver mass compared to the body mass may result in the small-for-size syndrome (SFSS). In both donor and recipient, LDLT may lead to portal hypertension associated with the elevated intrahepatic resistance of a smaller liver, and an increased portal venous (PV) inflow per gram of liver tissue compared to the total liver before resection. Excessive hyperperfusion and shear stress may damage the sinusoidal endothelial cells and lead to graft dysfunction.
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Wang, Chengyang, Qudus Hamid, Jessica Snyder, Halim Ayan, and Wei Sun. "A Novel Automation System for Microplasma Surface Patterning and Biologics Printing." In ASME/ISCIE 2012 International Symposium on Flexible Automation. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/isfa2012-7106.

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In the field of tissue engineering, regenerative medicine, and life sciences, the topological biochemical cues regulate cell attachment and alignment within the construct. In a native biological system, these cues are inherent. However, most of the biological materials utilized in the fabrication of tissue construct do not possess the appropriate cues required to develop an architecture to support the cell attachment and growth of a functional tissue Therefore the ability to manipulate structural and biochemical cues plays an important role in biofabrication process, and it is a key element to evaluate a engineered cellular model. Plasma surface functionalization and biologics printing have been investigated and validated as two effective techniques to guide cell functions by creating microenvironments. The objective of this work is to develop a novel dual functional platform for freeform microplasma surface patterning and biologics printing process as well as to study the underlying process science and the process induced cellular functions. The microplasma jet system was assembled by two parts. The upper part is a plastic NPT connector surrounding an extending high voltage copper electrode. The lower part is a dielectric Pasteur pipette connected with a capillary micro-scale nozzle tip. The lower part is interchangeable and the diameter of the tip ranges from 50 μm to 1 mm. With up to 20 kV output capability, a high-voltage power supply was connected to the copper electrode through the NPT connector which also served as gas inlet. A high-voltage probe linked to an oscilloscope is used to monitor the real time voltage. The whole microplasma jet system was set up on automation platform, which allows X-Y-Z motion control and switch control. This integrated system operates at atmospheric pressured environment. All tissue constructs could be fabricated at room temperature without the use of a mask. Clear polystyrene microplates were used as plasma treatment substrates. After O2-He mixed microplasma treatment, 7F2 mouse osteoblastic cells were cultured in the microplates for cell biology studies. We demonstrated the capability of our dual functional platform by applying microplasma in the polystyrene wells and control group (without any treatment) in other wells of the same microplate substrate. The results show that the microplasma treatment changed the surface properties and improved cell attachment. This dual functional freeform system allows for surface patterning and printing of cells, proteins, growth factors, etc. to fabricate three-dimensional tissue constructs.
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