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Relevant bibliographies by topics / Reoviridae

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Academic literature on the topic 'Reoviridae'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Reoviridae"

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Urbano, Pasquale, and Francesco Giuseppe Urbano. "The Reoviridae family." Comparative Immunology, Microbiology and Infectious Diseases 17, no. 3-4 (August 1994): 151–61. http://dx.doi.org/10.1016/0147-9571(94)90040-x.

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Attoui, Houssam, Fauziah Mohd Jaafar, Mourad Belhouchet, Philippe de Micco, Xavier de Lamballerie, and Corina P. D. Brussaard. "Micromonas pusilla reovirus: a new member of the family Reoviridae assigned to a novel proposed genus (Mimoreovirus)." Journal of General Virology 87, no. 5 (May 1, 2006): 1375–83. http://dx.doi.org/10.1099/vir.0.81584-0.

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Micromonas pusilla reovirus (MpRV) is an 11-segmented, double-stranded RNA virus isolated from the marine protist Micromonas pusilla. Sequence analysis (including conserved termini and presence of core motifs of reovirus polymerase), morphology and physicochemical properties confirmed the status of MpRV as a member of the family Reoviridae. Electron microscopy showed that intact virus particles are unusually larger (90–95 nm) than the known size of particles of viruses belonging to the family Reoviridae. Particles that were purified on caesium chloride gradients had a mean size of 75 nm (a size similar to the size of intact particles of members of the family Reoviridae), indicating that they lost outer-coat components. The subcore particles had a mean size of 50 nm and a smooth surface, indicating that MpRV belongs to the non-turreted Reoviridae. The maximum amino acid identity with other reovirus proteins was 21 %, which is compatible with values existing between distinct genera. Based on morphological and sequence findings, this virus should be classified as the representative of a novel genus within the family Reoviridae, designated Mimoreovirus (from Micromonas pusilla reovirus). The topology of the phylogenetic tree built with putative polymerase sequences of the family Reoviridae suggested that the branch of MpRV could be ancestral. Further analysis showed that segment 1 of MpRV was much longer (5792 bp) than any other reovirus segment and encoded a protein of 200 kDa (VP1). This protein exhibited significant similarities to O-glycosylated proteins, including viral envelope proteins, and is likely to represent the additional outer coat of MpRV.

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Trask, Shane D., Karl W. Boehme, Terence S. Dermody, and John T. Patton. "Comparative analysis of Reoviridae reverse genetics methods." Methods 59, no. 2 (February 2013): 199–206. http://dx.doi.org/10.1016/j.ymeth.2012.05.012.

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Lozano, Luis-Fernando, Arthur A. Bickford, Anthony E. Castro, Joyce Swartzman-Andert, Richard Chin, Carol Meteyer, George Cooper, Bruce Reynolds, and Rosa Lynn Manalac. "Association of Reoviridae Particles in an Enteric Syndrome of Poults Observed in Turkey Flocks during 1988." Journal of Veterinary Diagnostic Investigation 1, no. 3 (July 1989): 254–59. http://dx.doi.org/10.1177/104063878900100311.

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An enteric syndrome of turkey poults, characterized by enteritis, crop mycosis, intestinal changes (pale, thin-walled ballooning with watery contents), and rickets, occurred during 1988 in 74 turkey flocks from different farms belonging to 9 California turkey growers. The flocks ranged in size from 9,000 to 120,000 birds. Pools of intestine sections from 618 birds, representing 78 field cases, were examined. Histopathological examination of the intestines showed a mild to severe atrophy with a reduced depth of crypts, which was more prominent in the distal part of the small intestine. Viral isolation attempts with primary cell cultures of chicken embryo kidney cells were negative. Examination by electron microscopy of negatively stained intestinal specimens revealed the presence of Reoviridae particles of 58.8 to 80 nm in diameter. Enzyme-linked immunosorbent assay results on the intestinal pools for mammalian and group A avian rotaviruses were negative. A statistically significant relationship was found for the presence of Reoviridae particles in the intestines of 10-21-day-old birds. Of the 7 most common pathological conditions analyzed, 2, rickets and intestinal changes (thin-walled ballooning intestine with watery contents), showed a statistically significant association with the presence of Reoviridae particles.

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Li, Shu, Han Wang, and Guohui Zhou. "Synergism Between Southern rice black-streaked dwarf virus and Rice ragged stunt virus Enhances Their Insect Vector Acquisition." Phytopathology® 104, no. 7 (July 2014): 794–99. http://dx.doi.org/10.1094/phyto-11-13-0319-r.

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Southern rice black-streaked dwarf virus (SRBSDV), a tentative species in the genus Fijivirus, family Reoviridae, is a novel rice virus transmitted by the white-backed planthopper (Sogatella furcifera). Since its discovery in 2001, SRBSDV has spread rapidly throughout eastern and southeastern Asia and caused large rice losses in China and Vietnam. Rice ragged stunt virus (RRSV) (genus Oryzavirus, family Reoviridae) is a common rice virus vectored by the brown planthopper (Nilaparvata lugens). RRSV is also widely distributed in eastern and southeastern Asia but has not previously caused serious problems in China owing to its low incidence. With SRBSDV's spread, however, RRSV has become increasingly common in China, and is frequently found in co-infection with SRBSDV. In this study, we show that SRBSDV and RRSV interact synergistically, the first example of synergism between plant viruses in the family Reoviridae. Rice plants co-infected with both viruses displayed enhanced stunting, earlier symptoms, and higher virus titers compared with singly infected plants. Furthermore, white-backed and brown planthoppers acquired SRBSDV and RRSV, respectively, from co-infected plants at higher rates. We propose that increased RRSV incidence in Chinese fields is partly due to synergism between SRBSDV and RRSV.

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Donato, Celeste M., and Julie E. Bines. "Rotaviruses and Rotavirus Vaccines." Pathogens 10, no. 8 (July 29, 2021): 959. http://dx.doi.org/10.3390/pathogens10080959.

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Maramorosch, Karl. "Of Capsids and Capsomeres The Reoviridae Wolfgang K. Joklik." BioScience 35, no. 2 (February 1985): 116. http://dx.doi.org/10.2307/1309852.

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Li, Yanqiu, Jiamin Zhang, Yang Li, Li Tan, Wuguo Chen, Haishan Luo, and Yuanyang Hu. "Phylogenetic analysis of Heliothis armigera cytoplasmic polyhedrosis virus type 14 and a series of dwarf segments found in the genome." Journal of General Virology 88, no. 3 (March 1, 2007): 991–97. http://dx.doi.org/10.1099/vir.0.82673-0.

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Full-length nucleotide sequences for the genome segments (S1–S6) of Heliothis armigera cytoplasmic polyhedrosis virus type 14 (HaCPV-14) have been characterized. Each segment consists of a single open reading frame with conserved motifs AGAA and AGCU at the 5′ and 3′ ends, respectively. Comparison of the proteins of HaCPV-14 with those of other members of the family Reoviridae suggests that S1 encodes an RNA-dependent RNA polymerase (RdRp), whilst S2 encodes a major capsid protein of the virus. Phylogenetic analysis of RdRps from 16 viruses in the family Reoviridae reveals that the genera Cypovirus and Oryzavirus may have originated from a common insect virus ancestor. A series of viable dwarf segments originating from S5 of HaCPV-14 has been identified. Analysis of the predicted secondary structures for these dwarf segments suggests that the signals essential for replication and packaging are located within the terminal sequences of these segments.

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Ogden, Kristen. "Reoviridae transcription is more than an open-and-shut case." Nature Structural & Molecular Biology 26, no. 11 (November 2019): 991–93. http://dx.doi.org/10.1038/s41594-019-0328-5.

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López-Ferber, M., J. C. Veyrunes, and L. Croizier. "Drosophila S virus is a member of the Reoviridae family." Journal of Virology 63, no. 2 (1989): 1007–9. http://dx.doi.org/10.1128/jvi.63.2.1007-1009.1989.

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Dissertations / Theses on the topic "Reoviridae"

1

Grimes, Jonathan Mark. "Structural studies on Bluetongue virus." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260746.

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Walton, Alison. "Development of gene probes to P virus (Reoviridae) for disease diagnosis in crustaceans." Thesis, University of St Andrews, 1999. http://hdl.handle.net/10023/14861.

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This study reports the development of two important techniques, gene probes and haemocyte cultures that have not been previously available to investigate viral diseases in temperate water marine decapods. These techniques were used to investigate numerous aspects of a reovirus infection of the swimming crab Liocarcinus depurator, P virus, both in vivo and in vitro. The construction and subsequent use of a gene probe has revealed that, not only can virus be experimentally transmitted to L. depurator by injection, but that it is present in natural populations of crabs from the North Sea. Seasonal variation in both incidence of P infection and in incubation time was observed. The incidence of infection increased with increasing temperature whereas incubation time decreased with increasing temperature. In vivo, P virus was found to cause marked haemocytopenia in infected L. depurator and a cytopathic effect, vacuolisation of haemocytes was observed. This effect was not observed in the haemocytes of the shore crab, Carcinus maenas, providing evidence that P virus does not infect this species. To address the lack of techniques for in vitro studies, a cell culture system for crustacean haemocytes was developed. Primary culture of two haemocyte types, hyaline and semi-granular haemocytes was established for haemocytes of both L. depurator and C. maenas. High haemocyte viability was obtained for at least two weeks and, cells retained their functional capabilities in vitro. Having successfully established a haemocyte culture system and the gene probe E2b, it was then possible to begin investigations on P virus infections in vitro. P virus produced a number of effects on haemocytes of L. depurator in vitro. Haemocyte number and haemocyte viability decreased after addition of P virus and a number of cytopathic effects were observed such as necrosis, pycnosis and vacuolisation.

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BILLOIR, FREDERIQUE. "Strategies moleculaires pour la caracterisation genetique des arbovirus : application a l'identification et a la classification des virus appartenant aux familles reoviridae et flaviviridae (doctorat : maladies transmissibles et pathologie tropicale)." Aix-Marseille 2, 2000. http://www.theses.fr/2000AIX20652.

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Periz, Coloma Francisco Javier. "Single molecule fluorescence studies of viral transcription." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:d6e72aa8-060c-40fe-a07c-f695585dd43d.

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Rotaviruses are the single most common cause of fatal and severe childhood diarrhoeal illness worldwide (>125 million cases annually). Rotavirus shares structural and functional features with many viruses, such as the presence of segmented double-stranded RNA genomes selectively and tightly packed with a conserved number of transcription complexes in icosahedral capsids. Nascent transcripts exit the capsid through 12 channels, but it is unknown whether these channels specialise in specific transcripts or simply act as general exit conduits; a detailed description of this process is needed for understanding viral replication and genomic organisation. To test these opposing models, a novel single-molecule assay was developed for the capture and identification (CID) of newly synthesised specific RNA transcripts. CID combines the hybridisation of transcripts with biotinylated and FRET compatible labelled ssDNAs with the implementation of recent developments in single molecule fluorescence such as alternating laser excitation (ALEX) and total internal reflection fluorescence (TIRF) microscopy. CID identifies and quantifies specific transcripts of rotavirus based on a FRET/Stoichiometry (E*/S) value of the hybridised labelled probes. I used CID to pull down the capsid on the surface slide and identify partially extruded transcripts of three different segments 2, 6 and 11. The findings presented in this thesis support a model in which each channel specialises in extruding transcripts of a specific segment, that in turn is linked to a single transcription complex. The method can be extended to study other transcription systems including E.coli, and can be further developed as a potential diagnostic tool.

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Belhouchet, Mourad. "Analysis of an anti-silencing mechanism involved in immune evasion by vector-borne dsRNA animal viruses of family Reoviridae." Thesis, University of Oxford, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711672.

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Weyer, Camilla Theresa. "African horse sickness virus dynamics and host responses in naturally infected horses." Diss., University of Pretoria, 2010. http://hdl.handle.net/2263/25558.

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African horse sickness (AHS) is a life threatening disease of equids caused by African horse sickness virus (AHSV), a member of the genus Orbivirus in the family Reoviridae. The virus is transmitted to horses by midges (Culicoides spp.) and the disease is most prevalent during the time of year, and in areas where the Culicoides spp. are most abundant, namely in late summer in the summer rainfall areas of the country. Whilst the clinical signs and presentation of the disease were well documented by Sir Arnold Theiler (1921), very little is known or documented about AHSV dynamics or the clinical pathological and serological responses of horses to natural infection with AHSV. This dissertation describes the history and current knowledge on AHS, and the methods and results of a prospective study on natural AHSV infection of horses, undertaken between 2009 and 2010 by the Equine Research Centre (ERC) at the University of Pretoria, Faculty of Veterinary Science, Onderstepoort. This study is the first documented study of its nature and included animals of various ages and therefore variable vaccination status. The objectives of the study were to describe the viral dynamics of AHSV infection in horses, to gain a better understanding of the clinical pathological and serological responses to natural AHS infection and to demonstrate early detection of AHS infection in horses under field conditions.
Dissertation (MSc)--University of Pretoria, 2010.
Veterinary Tropical Diseases
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Fulton, Jonathan Reid. "Intestinal and systemic cytotoxic T lymphocyte and humoral immune responses to oral and parenteral reovirus infection." Morgantown, W. Va. : [West Virginia University Libraries], 2006. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=4474.

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Thesis (Ph. D.)--West Virginia University, 2006.
Title from document title page. Document formatted into pages; contains xi, 288 p. : ill. Vita. Includes abstract. Includes bibliographical references.

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Burger, Liesel. "Silencing African horsesickness virus VP7 protein expression in vitro by RNA interference." Diss., Pretoria : [s.n.], 2006. http://upetd.up.ac.za/thesis/available/etd-06262008-125200.

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Mathers, Alicia R. "The effects of the route of viral infection on the balance of T helper immune responses." Morgantown, W. Va. : [West Virginia University Libraries], 2005. https://etd.wvu.edu/etd/controller.jsp?moduleName=documentdata&jsp%5FetdId=3825.

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Thesis (Ph. D.)--West Virginia University, 2005
Title from document title page. Document formatted into pages; contains ix, 155 p. : ill. Vita. Includes abstract. Includes bibliographical references.

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Pal, Kasturi. "Regulation of polymeric immunoglobulin receptor by reovirus in intestinal epithelial cells." Morgantown, W. Va. : [West Virginia University Libraries], 2006. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=4535.

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Thesis (Ph. D.)--West Virginia University, 2006.
Title from document title page. Document formatted into pages; contains x, 202 p. : ill. (some col.). Includes abstract. Includes bibliographical references.

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Books on the topic "Reoviridae"

1

(Editor), K. L. Tyler, and M. B. A. Oldstone (Editor), eds. Reoviruses I: Structure, Proteins, and Genetics (Current Topics in Microbiology and Immunology). Springer-Verlag Telos, 1998.

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2

TYLER, KENNETH L. Reoviruses Ii: CYTOPATHOGENICITY AND PATHOGENESIS (Current Topics in Microbiology & Immunology). SPRINGER-VERLAG, 1998.

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Reoviruses 2: Cytopathogenicity and pathogenesis. Berlin: Springer, 1998.

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1953-, Tyler Kenneth L., and Oldstone Michael B. A, eds. Reoviruses. Berlin: Springer, 1998.

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Roy, Polly. Reoviruses: Entry, Assembly and Morphogenesis. Springer, 2014.

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Reoviruses: Entry, Assembly and Morphogenesis (Current Topics in Microbiology and Immunology). Springer, 2006.

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Virally infected cells. New York: Plenum, 1989.

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Virally Infected Cells (Subcellular Biochemistry). Springer, 1989.

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Book chapters on the topic "Reoviridae"

1

Aspöck, Horst, and Gerhard Dobler. "Reoviridae." In Encyclopedia of Parasitology, 2320–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_2684.

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Mehlhorn, Heinz. "Reoviridae." In Encyclopedia of Parasitology, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-642-27769-6_2684-2.

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Gries, Oliver, and Thomas Ly. "Reoviridae [dsRNA]." In Infektologie - Kompendium humanpathogener Infektionskrankheiten und Erreger, 191–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-58219-0_24.

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Tyler, Kenneth L., and Bernard N. Fields. "Reoviridae: The Reoviruses." In Laboratory Diagnosis of Infectious Diseases Principles and Practice, 353–74. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3900-0_19.

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Holmes, Ian H. "Reoviridae: The Rotaviruses." In Laboratory Diagnosis of Infectious Diseases Principles and Practice, 384–413. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3900-0_21.

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Drews, Gerhart, Günter Adam, and Cornelia Heinze. "Viren mit Doppelstrang-RNA-Genom: Reoviridae." In Springer-Lehrbuch, 185–90. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18740-7_13.

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Emmons, Richard W. "Reoviridae: The Orbiviruses (Colorado Tick Fever)." In Laboratory Diagnosis of Infectious Diseases Principles and Practice, 375–83. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3900-0_20.

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Yukl, Steven, and Joseph K. Wong. "Colorado Tick Fever and Other Arthropod Borne Reoviridae." In Clinical Virology, 841–52. Washington, DC, USA: ASM Press, 2016. http://dx.doi.org/10.1128/9781555819439.ch35.

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Murphy, Frederick A., E. Paul J. Gibbs, Marian C. Horzinek, and Michael J. Studdert. "Reoviridae." In Veterinary Virology, 391–404. Elsevier, 1999. http://dx.doi.org/10.1016/b978-012511340-3/50024-6.

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"Reoviridae." In Fenner's Veterinary Virology, 275–91. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-375158-4.00015-8.

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Conference papers on the topic "Reoviridae"

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Santos, Edson Moura dos, and Marcia Helena Braga Catroxo. "ROTAVÍRUS BOVINO. DETECÇÃO POR TÉCNICAS DE MICROSCOPIA ELETRÔNICA DE TRANSMISSÃO." In I Congresso Nacional de Microbiologia Clínica On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1629.

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Introdução. O rotavírus, classificado na família Reoviridae, gênero Rotavirus, é a principal causa de diarreia em bovinos, acomete também diversos outros animais inclusive os humanos. Importante síndrome que afeta rebanhos bovinos de corte e leite em todo o mundo, causando graves prejuízos econômicos à bovinocultura. O vírus causa uma infecção altamente contagiosa, caracterizada por vômitos, anorexia, prostração, diarreia de consistência pastosa à líquida de cor variável e desidratação. Apresenta altos índices de morbidade e mortalidade, além de reduzir o ganho de peso e aumentar os custos da produção. Objetivo. Diagnóstico rápido do rotavírus bovino por microscopia eletrônica de transmissão. Materiais e Métodos. No período de 2011 a 2021, aproximadamente 430 amostras de fezes bovinas ou fragmentos de intestino delgado de casos clínicos foram enviadas ao Laboratório de Microscopia Eletrônica do Instituto Biológico de São Paulo, SP, Brasil, para diagnóstico viral. As amostras foram processadas para microscopia eletrônica de transmissão utilizando as técnicas de contrastação negativa (preparo rápido), imunomicroscopia e de imunocitoquímica. Na contrastação negativa, as amostras foram suspensas em tampão fosfato 0,1 M e pH 7,0, colocadas em contato com grades metálicas e contrastadas negativamente com molibdato de amônio a 2%. Na técnica de imunomicroscopia, as telas foram incubadas com anticorpo específico para o vírus e com gotas do antígeno. Após, as telas foram contrastadas com molibdato de amônio 2%. Na imunocitoquímica, as telas foram incubadas com a suspensão viral e com gotas do anticorpo primário. Em seguida, as grades foram incubadas em gotas de proteína A em associação com partículas de ouro coloidal (anticorpo secundário) e contrastadas com molibdato de amônio a 2%. Resultados. Ao microscópio eletrônico de transmissão, pela técnica de contrastação negativa foi visualizado um grande número de partículas de rotavírus, arredondadas, não envelopadas, icosaédricas, caracterizadas como partículas “completas” e “vazias”, medindo em média 70 nm de diâmetro em 51 amostras (11,8%). A presença de agregados formados pela interação antígeno-anticorpo caracterizou o resultado positivo obtido, na imunomicroscopia. Na técnica de imunocitoquímica, a reação antígeno-anticorpo foi evidenciada pelas partículas de ouro coloidal sobre os rotavírus. Conclusão. As técnicas utilizadas foram eficientes para o diagnóstico rápido dos rotavírus bovinos.

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