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1

Visentin, Lorenzo. "6 Endocrine System and Reproductive System Riddle J T E Endocrine System and Reproductive System 138PP 0-443-03939-9." Nursing Standard 4, no. 4 (October 24, 1989): 56. http://dx.doi.org/10.7748/ns.4.4.56.s82.

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2

Michal, Ješeta, Chmelíková Eva, Crha Igor, Sedmíková Markéta, Žáková Jana, and Ventruba Pavel. "Endocrine disruptive compounds and male reproduction." Medical Journal of Cell Biology 6, no. 4 (December 1, 2018): 131–34. http://dx.doi.org/10.2478/acb-2018-0021.

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AbstractEndocrine disruptors (EDs) are chemical substances that affect physiological processes in the organism via hormonal regulation. The EDs are present in the environment and objects of everyday use. They are often detected in food, particularly released from packaging of canned food, but also from plastic water bottles, and they are also found in cosmetics and fertilizers. They are commonly detected in children's toys, banknotes, receipts and many more objects. Permanent and long-term utilization of EDs has harmful effects on human reproductive health mainly by interference with sex hormo
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3

Коrytko, О. О. "Influence of Endocrine Diseases on Reproductive System Functioning." INTERNATIONAL JOURNAL OF ENDOCRINOLOGY, no. 7.71 (December 29, 2015): 94–98. http://dx.doi.org/10.22141/2224-0721.7.71.2015.72598.

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4

Kubokawa, K., Y. Tando, and S. Roy. "Evolution of the Reproductive Endocrine System in Chordates." Integrative and Comparative Biology 50, no. 1 (May 24, 2010): 53–62. http://dx.doi.org/10.1093/icb/icq047.

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5

Shepelska, Ninel, Mykola Prodanchuk, and Yana Kolianchuk. "Pesticides as endocrine distruptors of the reproductive system (literature review and own research)." JOURNAL OF THE NATIONAL ACADEMY OF MEDICAL SCIENCES OF UKRAINE, Issue 1; 2021 (May 26, 2021): 49–62. http://dx.doi.org/10.37621/jnamsu-2020-1-6.

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Currently, one of the main threats to human health is undoubtedly endocrine disruptors (ED), since they directly disrupt the processes of homeostasis maintenance, controlled by the endocrine system, the purpose of which is to maintain normal functions and development in a constantly changing environment. Pesticides can disrupt the physiological functioning of many endocrine axes, including the endocrine mechanisms that ensure reproductive health. It should be noted that research aimed at preventing chemically induced reproductive disorders in the human population is one of the central areas of
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6

Chedrese, P., Martina Piasek, and Michael Henson. "Cadmium as an Endocrine Disruptor in the Reproductive System." Immunology‚ Endocrine & Metabolic Agents in Medicinal Chemistry 6, no. 1 (February 1, 2006): 27–35. http://dx.doi.org/10.2174/187152206775528941.

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7

Choi, Donchan. "The Consequences of Mutations in the Reproductive Endocrine System." Development & Reproduciton 16, no. 4 (December 2012): 235–51. http://dx.doi.org/10.12717/dr.2012.16.4.235.

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8

Queiroz, Erika Kaltenecker Retto de, and William Waissmann. "Occupational exposure and effects on the male reproductive system." Cadernos de Saúde Pública 22, no. 3 (March 2006): 485–93. http://dx.doi.org/10.1590/s0102-311x2006000300003.

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A significant increase in the incidence of male infertility has been described in the international literature, raising questions about its causes. Part of this effect may result from synthetic toxic substances acting on the endocrine system (endocrine disruptors), many of which are routinely used in work processes. We provide a critical review of the specialized literature on work-related chemical substances capable of causing male infertility. Pesticides such as DDT, linuron, and others, heavy metals like mercury, lead, cadmium, and copper, and substances from various industrial uses and res
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9

Koifman, Sergio, Rosalina Jorge Koifman, and Armando Meyer. "Human reproductive system disturbances and pesticide exposure in Brazil." Cadernos de Saúde Pública 18, no. 2 (April 2002): 435–45. http://dx.doi.org/10.1590/s0102-311x2002000200008.

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The observation of reproductive disturbances in humans and in the wildlife has been reported in the last decade in different countries. Exposure to different chemicals possibly acting in the endocrine system or endocrine disruptors, including pesticides, has been a hypothesis raised to explain the observed changes. This paper aimed to present results of an epidemiological ecologic study carried out to explore population data on pesticides exposure in selected Brazilian states in the eighties and human reproductive outcomes in the nineties. Pearson correlation coefficients were ascertained betw
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10

Rattan, Saniya, and Jodi A. Flaws. "The epigenetic impacts of endocrine disruptors on female reproduction across generations†." Biology of Reproduction 101, no. 3 (May 11, 2019): 635–44. http://dx.doi.org/10.1093/biolre/ioz081.

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AbstractHumans and animals are repeatedly exposed to endocrine disruptors, many of which are ubiquitous in the environment. Endocrine disruptors interfere with hormone action; thus, causing non-monotonic dose responses that are atypical of standard toxicant exposures. The female reproductive system is particularly susceptible to the effects of endocrine disruptors. Likewise, exposures to endocrine disruptors during developmental periods are particularly concerning because programming during development can be adversely impacted by hormone level changes. Subsequently, developing reproductive ti
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11

Fujiwara, Hiroshi, Atsushi Ideta, Yoshihiko Araki, Yumi Takao, Yukiyasu Sato, Noboru Tsunoda, Yoshito Aoyagi, and Ikuo Konishi. "Immune System Cooperatively Supports Endocrine System-Primed Embryo Implantation." Journal of Mammalian Ova Research 26, no. 3 (October 2009): 122–28. http://dx.doi.org/10.1274/jmor.26.122.

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12

van Duursen, Majorie B. M., Julie Boberg, Sofie Christiansen, Lisa Connolly, Pauliina Damdimopoulou, Panagiotis Filis, Paul A. Fowler, et al. "Safeguarding Female Reproductive Health Against Endocrine Disrupting Chemicals—The FREIA Project." International Journal of Molecular Sciences 21, no. 9 (May 1, 2020): 3215. http://dx.doi.org/10.3390/ijms21093215.

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Currently available test methods are not well-suited for the identification of chemicals that disturb hormonal processes involved in female reproductive development and function. This renders women’s reproductive health at increasing risk globally, which, coupled with increasing incidence rates of reproductive disorders, is of great concern. A woman’s reproductive health is largely established during embryonic and fetal development and subsequently matures during puberty. The endocrine system influences development, maturation, and function of the female reproductive system, thereby making app
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13

Happ, G. M. "Maturation of the Male Reproductive System and its Endocrine Regulation." Annual Review of Entomology 37, no. 1 (January 1992): 303–20. http://dx.doi.org/10.1146/annurev.en.37.010192.001511.

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14

Sidorkiewicz, Iwona, Kamil Zaręba, Sławomir Wołczyński, and Jan Czerniecki. "Endocrine-disrupting chemicals—Mechanisms of action on male reproductive system." Toxicology and Industrial Health 33, no. 7 (May 3, 2017): 601–9. http://dx.doi.org/10.1177/0748233717695160.

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Endocrine-disrupting chemicals (EDCs) are exogenous compounds that can cause disturbances in the endocrine system and have multiple harmful effects on health by targeting different organs and systems in the human body. Mass industrial production and widespread use of EDCs have resulted in worldwide contamination. Accumulating evidence suggest that human exposure to EDCs is related to the impairment of male reproductive function and can interrupt other hormonally regulated metabolic processes, particularly if exposure occurs during early development. Investigation of studies absent in previous
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15

Nicolopoulou-Stamati, P. "The impact of endocrine disrupters on the female reproductive system." Human Reproduction Update 7, no. 3 (May 1, 2001): 323–30. http://dx.doi.org/10.1093/humupd/7.3.323.

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16

Leso, V., L. Fontana, A. Marinaccio, K. Leopold, C. Fanali, D. Lucchetti, A. Sgambato, and I. Iavicoli. "Palladium nanoparticle effects on endocrine reproductive system of female rats." Human & Experimental Toxicology 37, no. 10 (February 6, 2018): 1069–79. http://dx.doi.org/10.1177/0960327118756722.

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The widespread industrial application of nanomaterials (NMs) has dramatically increased the likelihood of environmental and occupational exposure of humans to such xenobiotics. This issue, together with the increasing public health interest in understanding the effects of chemicals on endocrine system, encouraged to investigate the disruptive potential of NMs on the endocrine function. Therefore, the aim of this study was to evaluate the effects of palladium nanoparticles (Pd-NPs) on the female reproductive system of Wistar rats, intravenously exposed to different doses (0.12, 1.2, and 12 µg/k
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17

Toppari, Jorma, Wiwat Rodprasert, and Jaakko J. Koskenniemi. "Exposure Variation and Endocrine Disruption of the Male Reproductive System." Hormone Research in Paediatrics 86, no. 4 (2016): 247–52. http://dx.doi.org/10.1159/000446436.

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18

Sweeney, M. F., N. Hasan, A. M. Soto, and C. Sonnenschein. "Environmental endocrine disruptors: Effects on the human male reproductive system." Reviews in Endocrine and Metabolic Disorders 16, no. 4 (December 2015): 341–57. http://dx.doi.org/10.1007/s11154-016-9337-4.

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19

Gonsioroski, Andressa, Vasiliki E. Mourikes, and Jodi A. Flaws. "Endocrine Disruptors in Water and Their Effects on the Reproductive System." International Journal of Molecular Sciences 21, no. 6 (March 12, 2020): 1929. http://dx.doi.org/10.3390/ijms21061929.

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Anthropogenic contaminants in water can impose risks to reproductive health. Most of these compounds are known to be endocrine disrupting chemicals (EDCs). EDCs can impact the endocrine system and subsequently impair the development and fertility of non-human animals and humans. The source of chemical contamination in water is diverse, originating from byproducts formed during water disinfection processes, release from industry and livestock activity, or therapeutic drugs released into sewage. This review discusses the occurrence of EDCs in water such as disinfection byproducts, fluorinated co
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20

Esina, M. M. "SYSTEM OF REPRODUCTION IN HYPOTHYREOSIS." V.F.Snegirev Archives of Obstetrics and Gynecology 4, no. 2 (June 15, 2017): 77–83. http://dx.doi.org/10.18821/2313-8726-2017-4-2-77-83.

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Hypothyreosis is one of the most common endocrine pathologies closely related with the reproductive system. In female patients with hypofunction of the thyroid gland, various disorders occur in the reproductive system: menstrual cycle deteriorations, miscarriage, infertility. Therefore, the study of thyroid function should be carried out as a screening in women planning pregnancy, as well as in infertile couples in the treatment of the infertility. When hypothyreosis is detected, L-thyroxine replacement therapy is necessary.
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21

Narayan, Edward, and Simone Parisella. "Influences of the stress endocrine system on the reproductive endocrine axis in sheep (Ovis aries)." Italian Journal of Animal Science 16, no. 4 (May 9, 2017): 640–51. http://dx.doi.org/10.1080/1828051x.2017.1321972.

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22

Guillette, L. J., and Taisen Iguchi. "Contaminant-induced endocrine and reproductive alterations in reptiles." Pure and Applied Chemistry 75, no. 11-12 (January 1, 2003): 2275–86. http://dx.doi.org/10.1351/pac200375112275.

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Many chemicals introduced into the environment by humans adversely affect embryonic development and the functioning of the vertebrate reproductive system. It has been hypothesized that many developmental alterations are due to the endocrine-disruptive effects of various environmental contaminants. The endocrine system exhibits an organizational effect on the developing embryo, altering gene expression and dosing. Thus, a disruption of the normal hormonal signals can permanently modify the organization and future functioning of the reproductive and endocrine system. We have worked extensively w
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23

Wen, Lixin, Rongfang Li, Ji Wang, and Jine Yi. "The reproductive stress hypothesis." Reproduction 158, no. 6 (December 2019): R209—R218. http://dx.doi.org/10.1530/rep-18-0592.

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In this paper, we propose the reproductive stress hypothesis that describes the pregnant females response to reproductive events based upon the activation of the hypothalamic–pituitary–adrenal axis and sympathetic adrenomedullary system. The main components of the reproductive stress hypothesis can be summarized as follows: (1) events unique to reproduction including empathema, pregnancy, parturition and lactation cause non-specific responses in females, called active reproductive stress; (2) the fetus is a special stressor for pregnant females where endocrine hormones, including corticotropin
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24

Hohmann, John G., Thomas H. Teal, Donald K. Clifton, James Davis, Victor J. Hruby, Guoxia Han, and Robert A. Steiner. "Differential role of melanocortins in mediating leptin's central effects on feeding and reproduction." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 278, no. 1 (January 1, 2000): R50—R59. http://dx.doi.org/10.1152/ajpregu.2000.278.1.r50.

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Leptin serves as a humoral link coupling the status of energy reserves to the functional activity of the reproductive system. Leptin is thought to act through melanocortinergic pathways in the brain to regulate ingestive behaviors; however, whether melanocortins mediate leptin's actions on the neuroendocrine-reproductive axis is unknown. We tested this hypothesis first by determining whether the effects of leptin on feeding behavior and reproduction in the ob/ob mouse could be blocked by the melanocortin receptor (MC-R) antagonist SHU9119 and second, by examining the effects of the MC-R agonis
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25

Kamalova, Dilyafruz. "REPRODUCTIVE AND PERINATAL RISKS IN WOMEN WITH AUTOIMMUNE THYREOIDITIS." UZBEK MEDICAL JOURNAL 5, no. 1 (May 30, 2020): 44–48. http://dx.doi.org/10.26739/2181-0664-2020-5-7.

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Preservation and promotion of health, prevention of morbidity in Uzbekistan is the most important strategic direction of the state's social policy, the age structure of which is steadily shifting towards aging, which invariably leads to a decrease in the number of children and people of fertile age.Among the most common endocrine pathologies is the pathology of the thyroid system.
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26

Takeda, Ken. "The Effect of Endocrine Disrupting Chemicals on the Male Reproductive System." Waste Management Research 10, no. 4 (1999): 271–77. http://dx.doi.org/10.3985/wmr.10.271.

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27

Heindel, Jerrold J., and Kimberley A. Treinen. "Physiology of the Male Reproductive System: Endocrine, Paracrine and Autocrine Regulation." Toxicologic Pathology 17, no. 2 (February 1989): 411–45. http://dx.doi.org/10.1177/019262338901700219.

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28

Kogevinas, M. "Human health effects of dioxins: cancer, reproductive and endocrine system effects." Human Reproduction Update 7, no. 3 (May 1, 2001): 331–39. http://dx.doi.org/10.1093/humupd/7.3.331.

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29

Kogevinas, M. "Human health effects of dioxins: cancer, reproductive and endocrine system effects." APMIS 109, S103 (July 2001): S223—S232. http://dx.doi.org/10.1111/j.1600-0463.2001.tb05771.x.

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30

Pflieger-Bruss, S., H. C. Schuppe, and W. B. Schill. "The male reproductive system and its susceptibility to endocrine disrupting chemicals." Andrologia 36, no. 6 (December 2004): 337–45. http://dx.doi.org/10.1111/j.1439-0272.2004.00641.x.

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31

Yoshimura, Yasunori. "The Ovarian Renin–Angiotensin System in Reproductive Physiology." Frontiers in Neuroendocrinology 18, no. 3 (July 1997): 247–91. http://dx.doi.org/10.1006/frne.1997.0152.

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32

Nwankudu, O. N. "Endocrine, Reproductive, Neurophysiologic and Extraneous Activities of Estrogen in Vertebrates." Nigerian Veterinary Journal 41, no. 2 (April 16, 2021): 85–107. http://dx.doi.org/10.4314/nvj.v41i2.2.

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Estrogens are reproductive hormones synthesized in the gonads of both male and female vertebrates. This review is geared towards uncovering some endocrine, reproductive, neurophysiologic and extraneous activities of estrogen in vertebrates. The three most common naturally occurring estrogens are: Estrone (E1), estradiol (E2), and estriol (E3). In primates, estradiol is the most potent and predominant estrogen during reproductive years. Estrogens are synthesized primarily in the female ovaries and in small quantities in the male testes and the adrenal glands, brain, and fat of both sexes. Estro
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33

Thomas, John A. "Drugs and Chemicals that Affect the Endocrine System." International Journal of Toxicology 17, no. 2 (February 1998): 129–38. http://dx.doi.org/10.1080/109158198226666.

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The mammalian endocrine system is very dynamic, and undergoes frequent physiological fluctions due to diurnal variations and cyclical hormonal feedback systems. Both hormonal modulations and chemicall drug perturbations can affect the reproductive systems in males and females. An endocrine disrup-tor, a contemporary term that has been used to define an agent that disrupts the endocrine system, is a hormone or antihormone mimic that can modulate endocrine signaling pathways. Unfortunately, this terminology is confusing and ambiguous and fails to account for the ever-changing endogenous hormonal
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34

García, María C., Miguel López, Clara V. Alvarez, Felipe Casanueva, Manuel Tena-Sempere, and Carlos Diéguez. "Role of ghrelin in reproduction." Reproduction 133, no. 3 (March 2007): 531–40. http://dx.doi.org/10.1530/rep-06-0249.

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Ghrelin, the endogenous ligand of GH secretagogue receptor type 1a, has emerged as a pleiotropic modulator of diverse biological functions, including energy homeostasis and, lately reproduction. Here, we review recent reports evaluating the reproductive effects and sites of action of ghrelin, with particular emphasis regarding its role as a molecule integrating reproductive function and energy status. Data gleaned from rodent studies clearly show that besides having direct gonadal effects, ghrelin may participate in the regulation of gonadotropin secretion and it may influence the timing of pu
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35

Latini, Giuseppe, Egeria Scoditti, Alberto Verrotti, Claudio De Felice, and Marika Massaro. "Peroxisome Proliferator-Activated Receptors as Mediators of Phthalate-Induced Effects in the Male and Female Reproductive Tract: Epidemiological and Experimental Evidence." PPAR Research 2008 (2008): 1–13. http://dx.doi.org/10.1155/2008/359267.

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There is growing evidence that male as well as female reproductive function has been declining in human and wildlife populations over the last 40 years. Several factors such as lifestyle or environmental xenobiotics other than genetic factors may play a role in determining adverse effects on reproductive health. Among the environmental xenobiotics phthalates, a family of man-made pollutants are suspected to interfere with the function of the endocrine system and therefore to be endocrine disruptors. The definition of endocrine disruption is today extended to broader endocrine regulations, and
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36

Grindon, Christina, Robert Combes, Mark T. D. Cronin, David W. Roberts, and John F. Garrod. "Integrated Decision-tree Testing Strategies for Developmental and Reproductive Toxicity with Respect to the Requirements of the EU REACH Legislation." Alternatives to Laboratory Animals 36, no. 1 (February 2008): 65–80. http://dx.doi.org/10.1177/026119290803600108.

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Liverpool John Moores University and FRAME conducted a research project, sponsored by Defra, on the status of alternatives to animal testing with regard to the European Union REACH (Registration, Evaluation and Authorisation of Chemicals) system for the safety testing and risk assessment of chemicals. The project covered all the main toxicity endpoints associated with the REACH system. This paper focuses on the prospects for the use of alternative methods (both in vitro and in silico) in developmental and reproductive toxicity testing. It considers many tests based on primary cells and cell li
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37

Grindon, Christina, Robert Combes, Mark T. D. Cronin, David W. Roberts, and John F. Garrod. "Integrated Decision-tree Testing Strategies for Developmental and Reproductive Toxicity with Respect to the Requirements of the EU REACH Legislation." Alternatives to Laboratory Animals 36, no. 1_suppl (October 2008): 123–38. http://dx.doi.org/10.1177/026119290803601s10.

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Liverpool John Moores University and FRAME conducted a research project, sponsored by Defra, on the status of alternatives to animal testing with regard to the European Union REACH (Registration, Evaluation and Authorisation of Chemicals) system for the safety testing and risk assessment of chemicals. The project covered all the main toxicity endpoints associated with the REACH system. This paper focuses on the prospects for the use of alternative methods (both in vitro and in silico) in developmental and reproductive toxicity testing. It considers many tests based on primary cells and cell li
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38

Schwabl, Hubert, Willow R. Lindsay, Douglas G. Barron, and Michael S. Webster. "Endocrine correlates of mate choice and promiscuity in females of a socially monogamous avian mating system with alternative male reproductive phenotypes." Current Zoology 60, no. 6 (December 1, 2014): 804–15. http://dx.doi.org/10.1093/czoolo/60.6.804.

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Abstract While our understanding of male reproductive strategies is informed by extensive investigations into endocrine mechanisms, the proximate mechanisms by which females compete for mates and adjust reproduction to social environment remains enigmatic. We set out to uncover endocrine correlates of mate choice, social environment, and reproductive investment in female red-backed fairy-wrens Malurus melanocephalus. In this socially monogamous, yet highly sexually promiscuous species, females experience discrete variation in the phenotype of their mates, which vary in both plumage signals and
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39

Foster, Warren G. "Endocrine Disruption and Human Reproductive Effects: An Overview." Water Quality Research Journal 36, no. 2 (May 1, 2001): 253–71. http://dx.doi.org/10.2166/wqrj.2001.015.

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Abstract Humans of all ages interact with their environment on a daily basis and are thus exposed to a variety of both man-made and naturally occurring chemicals through the air they breath, the water they drink and the food they eat. The potential for exposure to environmental contaminants to impact the function of the reproductive system and affect normal development of the reproductive tract has become an area of increasing concern at all levels of society. Environment Canada and Health Canada jointly organized a workshop to review the current state of knowledge on endocrine disrupting comp
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40

Layton, Scott, and Jason Belden. "Engaging Undergraduates in the Scientific Process: Exploring Invertebrate Endocrine Disruption." American Biology Teacher 78, no. 5 (May 1, 2016): 410–16. http://dx.doi.org/10.1525/abt.2016.78.5.410.

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Engaging students in the process of science to increase learning and critical thinking has become a key emphasis in undergraduate education. Introducing environmental topics, such as the effects of endocrine-disrupting chemicals, into undergraduate courses offers a new means to increase student engagement. Daphnia magna can serve as a model organism for endocrine disruption, and its ease of handling, rapid reproduction rate, and clearly defined endpoints make it useful in short-term, student research projects. The concept of endocrine disruption can be tested through a 21-day reproductive stud
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41

Choi, Kyung-Chul, Eui-Bae Jeung, and Peter Leung. "Impact of Environmental Endocrine Disruption on the Reproductive System for Human Health." Immunology‚ Endocrine & Metabolic Agents in Medicinal Chemistry 6, no. 1 (February 1, 2006): 3–13. http://dx.doi.org/10.2174/187152206775528905.

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42

Brooks, A. N. "Comparative physiology of the reproductive endocrine system in laboratory rodents and humans." Pure and Applied Chemistry 70, no. 9 (September 1, 1998): 1633–46. http://dx.doi.org/10.1351/pac199870091633.

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43

Goldman, Jerome M., Ralph L. Cooper, Susan C. Laws, Georgia L. Rehnberg, Tammy L. Edwards, W. Keith McElroy, and Joy F. Hein. "Chlordimeform-induced alterations in endocrine regulation within the male rat reproductive system." Toxicology and Applied Pharmacology 104, no. 1 (June 1990): 25–35. http://dx.doi.org/10.1016/0041-008x(90)90279-4.

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44

Gibson, Douglas A., and Philippa T. K. Saunders. "Endocrine disruption of oestrogen action and female reproductive tract cancers." Endocrine-Related Cancer 21, no. 2 (October 25, 2013): T13—T31. http://dx.doi.org/10.1530/erc-13-0342.

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Endocrine disrupting chemicals (EDC) are ubiquitous and persistent compounds that have the capacity to interfere with normal endocrine homoeostasis. The female reproductive tract is exquisitely sensitive to the action of sex steroids, and oestrogens play a key role in normal reproductive function. Malignancies of the female reproductive tract are the fourth most common cancer in women, with endometrial cancer accounting for most cases. Established risk factors for development of endometrial cancer include high BMI and exposure to oestrogens or synthetic compounds such as tamoxifen. Studies on
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45

Cottone, Erika, Valentina Pomatto, and Patrizia Bovolin. "Role of the Endocannabinoid System in the Central Regulation of Nonmammalian Vertebrate Reproduction." International Journal of Endocrinology 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/941237.

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The endocannabinoid system (ECS) has a well-documented pivotal role in the control of mammalian reproductive functions, by acting at multiple levels, that is, central (CNS) and local (gonads) levels. Since studies performed in animal models other than mammals might provide further insight into the biology of these signalling molecules, in the present paper we review the comparative data pointing toward the endocannabinoid involvement in the reproductive control of non-mammalian vertebrates, focussing in particular on the central regulation of teleost and amphibian reproduction. The morphofunct
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46

Fusani, Leonida, Daniele Della Seta, Francesco Dessì-Fulgheri, and Francesca Farabollini. "Altered reproductive success in rat pairs after environmental-like exposure to xenoestrogen." Proceedings of the Royal Society B: Biological Sciences 274, no. 1618 (April 24, 2007): 1631–36. http://dx.doi.org/10.1098/rspb.2007.0064.

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Endocrine-disrupting compounds (EDCs) have the capacity of altering the normal function of the endocrine system. EDCs have shown dramatic effects on the reproductive biology of aquatic wildlife and may affect human reproduction as well. Studies on EDCs in mammalian species have often investigated the effects of short-term, high doses on male and female reproductive physiology. However, it is difficult to predict from such studies the effects of EDC on populations that are exposed to very low doses throughout their life via contaminated food and water. We studied the effects of EDC on mammalian
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47

Wright, I. A., and A. R. Peters. "Nutrition: reproduction interactions in cows." Proceedings of the British Society of Animal Production (1972) 1991 (March 1991): 35. http://dx.doi.org/10.1017/s0308229600019863.

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In any cattle production system, reproductive performance is of central importance. The output from beef cow systems is calves, while lactation in the dairy cow is only achieved following parturition. On a world basis, the major cause of poor reproductive performance is inadequate nutrition, and in most cases this is represented by an inadequate energy supply, although in specific circumstances particular vitamin, mineral or disease problems can occur. This review considers how nutrition, and in particular energy nutrition may act via the endocrine system to influence reproductive performance
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48

Caro, Samuel P., Charlotte A. Cornil, Kees van Oers, and Marcel E. Visser. "Personality and gonadal development as sources of individual variation in response to GnRH challenge in female great tits." Proceedings of the Royal Society B: Biological Sciences 286, no. 1902 (May 2019): 20190142. http://dx.doi.org/10.1098/rspb.2019.0142.

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Seasonal timing of reproduction is a key life-history trait, but we know little about the mechanisms underlying individual variation in female endocrine profiles associated with reproduction. In birds, 17β-oestradiol is a key reproductive hormone that links brain neuroendocrine mechanisms, involved in information processing and decision-making, to downstream mechanisms in the liver, where egg-yolk is produced. Here, we test, using a simulated induction of the reproductive system through a Gonadotropin-Releasing Hormone (GnRH) challenge, whether the ovary of pre-breeding female great tits respo
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Kumar, Vikas, Chandrajeetbalo Majumdar, and Partha Roy. "Effects of endocrine disrupting chemicals from leather industry effluents on male reproductive system." Journal of Steroid Biochemistry and Molecular Biology 111, no. 3-5 (September 2008): 208–16. http://dx.doi.org/10.1016/j.jsbmb.2008.06.005.

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50

Joffe, M. "Myths about endocrine disruption and the male reproductive system should not be propagated." Human Reproduction 17, no. 2 (February 1, 2002): 520–21. http://dx.doi.org/10.1093/humrep/17.2.520.

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