Relevant bibliographies by topics / Resistance to viral infection

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Resistance to viral infection'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Resistance to viral infection"

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Ceccherini-Silberstein, Francesca, Valeria Cento, Velia Chiara Di Maio, Carlo Federico Perno, and Antonio Craxì. "Viral resistance in HCV infection." Current Opinion in Virology 32 (October 2018): 115–27. http://dx.doi.org/10.1016/j.coviro.2018.10.005.

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Valkonen, J. P. T. "Mechanisms of resistance to viruses." Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (2002): S132—S135. http://dx.doi.org/10.17221/10337-pps.

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Resistance associated with a hypersensitive response (HR) and subsequent development of necrotic lesions (cell death) at the sites of virus infection can restrict virus movement in plants. Genes for HR are dominant and act on a gene-for-gene basis. Many viral proteins triggering HR have been identified. Also, several genes for HR-based virus resistance, or virus-induced cell death without resistance, have been isolated and characterized in plants, which provides novel insights to the mechanisms of virus resistance. Another international, major research frontier has formed more recently around
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Beisekova, M. K., A. Samat, А. B. Kurmanbayeva, N. N. Iksat, S. B. Zhangazin, and Zh K. Masalimov. "Role of anthocyanins in plant resistance to virus." BULLETIN OF THE L.N. GUMILYOV EURASIAN NATIONAL UNIVERSITY. BIOSCIENCE SERIES 150, no. 1 (2025): 117–33. https://doi.org/10.32523/2616-7034-2025-150-1-117-133.

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Viral infections pose a serious threat to crop production in Kazakhstan and worldwide, negatively affecting the growth, development, and productivity of agricultural crops. Under conditions of multiple stresses, such as drought, extreme temperatures, soil salinity, and pathogen damage, viruses aggravate physiological changes in plants, disrupting their metabolic pathways and reducing resistance to adverse factors. Particular attention is paid to the effect of viral infections on the biosynthesis of anthocyanins, important compounds involved in plant defense mechanisms. In this work, it was sho
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Joshi, Sachin C., Amit V. Kakad, Vardhaman A. Murkunde, and Gayatri N. Kanade. "Viral Infection-A Looming Catastrophe." World Journal of Pharmaceutical Sciences 10, no. 04 (2022): 38–43. http://dx.doi.org/10.54037/wjps.2022.100405.

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The common cold, influenza, chickenpox, human immunodeficiency virus (HIV), SARS-CoV-2, and other disease-causing (pathogenic) viruses crop up when most people hear the word "virus." Infectious diseases are predicted to account for over one-third (i.e., 20 million) of annual deaths worldwide, as per the World Health Organization (WHO). The rising globalized health sector has made major contributions to the improvement of human health. However, long-standing, developing, and re-emerging infectious disease risks continue to haunt the globe. With a pandemic on the scale of COVID-19, Antibiotic re
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Yergaliev, T. "Molybdenum and plant resistance to viral infection." BULLETIN of the L.N. Gumilyov Eurasian National University. BIOSCIENCE Series 135, no. 2 (2021): 63–70. http://dx.doi.org/10.32523/2616-7034-2021-135-2-63-70.

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Molybdenum takes an active part in several physiological processes necessary for the growth and development of plants and other domains of life. Molybdenum participates in numerous biochemical reactions and lack of this metal may affect the total amount of proteins in plants. More than fifty Mo-containing enzymes are currently known, although most of them were found in bacteria. Plants contain Mo-containing enzymes such as nitrate reductase, sulfite oxidase, aldehyde oxidase, xanthine dehydrogenase, and mitochondrial amidoxime reductase. Tungsten is another heavy metal, which due to highly sim
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Beck, Melinda A., and Colette C. Matthews. "Micronutrients and host resistance to viral infection." Proceedings of the Nutrition Society 59, no. 4 (2000): 581–85. http://dx.doi.org/10.1017/s0029665100000823.

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Previous work in our laboratory demonstrated that a virus could undergo rapid mutation in a host deficient in Se, leading to a normally avirulent virus acquiring virulence due to genome changes. Once these mutations occur, even a host with adequate Se-nutriture is susceptible to the newly virulent virus. What influence does the deficiency in Se have on the immune response of the host? Infection with myocarditic strains of coxsackievirus induces an inflammatory response in the cardiac tissue. It is this immune response that induces the heart damage, rather than direct viral effects on the heart
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Beutler, Bruce, Celine Eidenschenk, Karine Crozat, et al. "Genetic analysis of resistance to viral infection." Nature Reviews Immunology 7, no. 10 (2007): 753–66. http://dx.doi.org/10.1038/nri2174.

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Li, Xue, Xueping Zhou, and Fangfang Li. "Pelota: A double-edged sword in virus infection." PLOS Pathogens 21, no. 7 (2025): e1013328. https://doi.org/10.1371/journal.ppat.1013328.

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Pelota, a conserved ribosome rescue factor involved in mRNA surveillance, has emerged as a pivotal player in host–virus arms race. Beyond its canonical role in maintaining translational fidelity via No-Go Decay and Non-Stop Decay pathways, Pelota exhibits a dual function during viral infection-serving either as a restriction factor or as a susceptibility element depending on the virus species and their hosts. In DNA virus infections, notably with geminiviruses, a natural mutation in Pelota confers recessive resistance in tomato and pepper probably by impairing viral protein translation, offeri
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Mohamed, Atef, Zhenhui Jin, Toba Osman, et al. "Hotspot siRNA Confers Plant Resistance against Viral Infection." Biology 11, no. 5 (2022): 714. http://dx.doi.org/10.3390/biology11050714.

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A hallmark of antiviral RNA interference (RNAi) is the production of viral small interfering RNA (vsiRNA). Profiling of vsiRNAs indicates that certain regions of viral RNA genome or transcribed viral RNA, dubbed vsiRNA hotspots, are more prone to RNAi-mediated cleavage for vsiRNA biogenesis. However, the biological relevance of hotspot vsiRNAs to the host innate defence against pathogens remains to be elucidated. Here, we show that direct targeting a hotspot by a synthetic vsiRNA confers host resistance to virus infection. Using Northern blotting and RNAseq, we obtained a profile of vsiRNAs of
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Shen, Xiaoyun, Bo Feng, Weiyi Shi, Wenming Cheng, and Tiefeng Zhang. "Concomitant viral and bacterial pneumonia among patients in ICU with mechanical respiratory support." Journal of Infection in Developing Countries 16, no. 09 (2022): 1482–89. http://dx.doi.org/10.3855/jidc.12999.

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Introduction: The use of mechanical ventilators in the intensive care unit (ICU) is often associated with higher risk of respiratory tract infections, including ventilator-associated pneumonia (VAP). Concomitant bacterial-viral infection was reported to worsen patient's clinical condition. This study evaluated the rate of concomitant bacterial-viral infections in patients with VAP and analyzed their clinical outcomes.
 Methodology: In this retrospective observational study 107 patients diagnosed with VAP and admitted in ICU with mechanical ventilator support between April 2018 and May 201
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Dissertations / Theses on the topic "Resistance to viral infection"

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Heath, Sarah E. "Evolutionary consequences of viral resistance in the marine picoeukaryote Ostreococcus tauri." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31082.

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In marine environments, eukaryotic marine microalgae coexist with the viruses that infect them. Marine microalgae are the main primary producers in the oceans and are at the base of the marine food web. Viruses play important roles in top-down control of algae populations, cycling of organic matter, and as evolutionary drivers of their hosts. Algae must adapt in response to the strong selection pressure that viruses impose for resistance to infection. In addition to biotic selection pressures such as viral infections, algae must also adapt to their abiotic environment. Global climate change is
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Moody, Adrian John. "Mapping genetic resistance to infectious bursal disease." Thesis, University of Reading, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326754.

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Lowe, Alexander Paul. "The role of viral and bacterial infections in asthma exacerbations and corticosteroid resistance." Thesis, Cardiff University, 2013. http://orca.cf.ac.uk/47342/.

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Asthma is a chronic inflammatory disease of the airways characterised by early and late asthmatic responses (EAR & LAR) to allergen, airways hyperresponsiveness (AHR) to inhaled spasmogens, airway inflammation and airway oedema. Viral infections and lipopolysaccharide (LPS) from bacteria and environmental sources contribute to exacerbations of asthma and the development of insensitivity to corticosteroids. Complete insensitivity to oral corticosteroids is rare and most patients lie on a continuum of steroid responsiveness. This thesis aimed to examine the effect of viral infection and LPS in a
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Burgan, Sarah Catherine. "From Tolerance to Transmission: Linking Within-Individual to Community-Level Disease Processes." Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6193.

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Hosts have two main strategies for coping with infections: resistance and tolerance. Resistance is aimed at preventing or eliminating parasites, whereas the goal of tolerance is to maintain performance regardless of parasite burden. The balance between resistance and tolerance within a host may mediate host competence, or the propensity of a host to infect other hosts or vectors. Hosts with high tolerance and low resistance to an infection, for instance, may be highly competent and possess the greatest potential to act as superspreaders. These superspreading hosts will contribute disproportion
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Okleberry, Kevin M. "Metabolism of Selected Antiviral Agents in Cells Infected with Drug-Resistant and Wild-Type Strains of Murine Cytomegalovirus." DigitalCommons@USU, 1995. https://digitalcommons.usu.edu/etd/4657.

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Resistance of human viral pathogens to various antiviral drugs is a serious medical problem. Two modes of drug resistance in cytomegalovirus infections have been observed, the first being altered (decreased) drug metabolism by the infected cells, and the second reduced sensitivity of the viral deoxyribonucleic acid polymerase enzyme to the active form of the drug. Mice infected with the murine cytomegalovirus have been used extensively as an animal model for the human cytomegalovirus, and drug-resistant strains in this model have been identified. To better understand the mode of drug resistanc
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Lin, Kuan-Hung. "Viral Proteases as Drug Targets and the Mechanisms of Drug Resistance: A Dissertation." eScholarship@UMMS, 2016. https://escholarship.umassmed.edu/gsbs_diss/841.

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Viral proteases have been shown to be effective targets of anti-viral therapies for human immunodeficiency virus (HIV) and hepatitis C virus (HCV). However, under the pressure of therapy including protease inhibitors, the virus evolves to select drug resistance mutations both in the protease and substrates. In my thesis study, I aimed to understand the mechanisms of how this protease−substrate co-evolution contributes to drug resistance. Currently, there are no approved drugs against dengue virus (DENV); I investigated substrate recognition by DENV protease and designed cyclic peptides as inhi
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Lin, Kuan-Hung. "Viral Proteases as Drug Targets and the Mechanisms of Drug Resistance: A Dissertation." eScholarship@UMMS, 2009. http://escholarship.umassmed.edu/gsbs_diss/841.

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Viral proteases have been shown to be effective targets of anti-viral therapies for human immunodeficiency virus (HIV) and hepatitis C virus (HCV). However, under the pressure of therapy including protease inhibitors, the virus evolves to select drug resistance mutations both in the protease and substrates. In my thesis study, I aimed to understand the mechanisms of how this protease−substrate co-evolution contributes to drug resistance. Currently, there are no approved drugs against dengue virus (DENV); I investigated substrate recognition by DENV protease and designed cyclic peptides as inhi
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Ozen, Aysegul. "Structure and Dynamics of Viral Substrate Recognition and Drug Resistance: A Dissertation." eScholarship@UMMS, 2013. https://escholarship.umassmed.edu/gsbs_diss/677.

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Drug resistance is a major problem in quickly evolving diseases, including the human immunodeficiency (HIV) and hepatitis C viral (HCV) infections. The viral proteases (HIV protease and HCV NS3/4A protease) are primary drug targets. At the molecular level, drug resistance reflects a subtle change in the balance of molecular recognition; the drug resistant protease variants are no longer effectively inhibited by the competitive drug molecules but can process the natural substrates with enough efficiency for viral survival. Therefore, the inhibitors that better mimic the natural substrate bindin
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Ozen, Aysegul. "Structure and Dynamics of Viral Substrate Recognition and Drug Resistance: A Dissertation." eScholarship@UMMS, 2005. http://escholarship.umassmed.edu/gsbs_diss/677.

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Drug resistance is a major problem in quickly evolving diseases, including the human immunodeficiency (HIV) and hepatitis C viral (HCV) infections. The viral proteases (HIV protease and HCV NS3/4A protease) are primary drug targets. At the molecular level, drug resistance reflects a subtle change in the balance of molecular recognition; the drug resistant protease variants are no longer effectively inhibited by the competitive drug molecules but can process the natural substrates with enough efficiency for viral survival. Therefore, the inhibitors that better mimic the natural substrate bindin
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Svedhem, Johansson Veronica. "Kinetics of HIV-1 drug resistance mutations in vivo /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-671-9/.

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Books on the topic "Resistance to viral infection"

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Zumla, Alimuddin, David S. C. Hui, and Wing-Wai Yew. Emerging respiratory infections in the 21st century. Saunders, 2010.

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Burton, Dennis R., ed. Antibodies in Viral Infection. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-662-05783-4.

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Lane, Thomas E., ed. Chemokines and Viral Infection. Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/978-3-540-33397-5.

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G, Donders, Stray-Pedersen B, and Anteby E, eds. Viral infection in pregnancy. Elsevier, 2002.

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R, Burton Dennis, ed. Antibodies in viral infection. Springer, 2001.

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Sherlock, Sheila. Hepatitis C viral infection: An update. Blackwell Scientific, 1989.

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Sheila, Sherlock. Hepatitis C viral infection--an update. Blackwell Scientific Publications, 1989.

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Kreier, Julius P. Infection, Resistance, and Immunity. Routledge, 2022. http://dx.doi.org/10.1201/9780203750964.

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F, Mortensen Richard, ed. Infection, resistance, and immunity. Harper & Row, 1990.

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Clemens, Sorg, ed. Natural resistance to infection. Fischer, 1990.

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Book chapters on the topic "Resistance to viral infection"

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Johnson, Eleanor, Shravya Reddy Pothula, and Julie H. Wu. "Emerging Viral Infections." In Overcoming Antimicrobial Resistance of the Skin. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68321-4_6.

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Shukla, D. D., K. H. Gough, XiaoWen Xiao, M. J. Frenkel, and C. W. Ward. "Genetically Engineered Resistance in Plants Against Viral Infection." In Horticulture — New Technologies and Applications. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3176-6_18.

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Miller, Jennifer L., Tessa M. Burch-Smith, and S. P. Dinesh-Kumar. "Resistance to Viral Infections in Plants." In Handbook of Plant Virology. CRC Press, 2024. http://dx.doi.org/10.1201/9781003578611-18.

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Han, Ying-Shan, and Mark A. Wainberg. "Genotypic Assays for Monitoring Drug Resistance in HIV-1 Infection and for Other Chronic Viral Diseases." In Antimicrobial Drug Resistance. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47266-9_38.

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Swali, Ritu, Claire Wiggins, Sahira Farooq, Radhika A. Shah, and Emily Limmer. "Reemerging Viral Infections: Implications of Lack of Vaccination." In Overcoming Antimicrobial Resistance of the Skin. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68321-4_7.

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Sarmiento, Marion. "Innate Resistance to HSV-1 Infection: Mouse Macrophage Inhibition of Viral Replication." In Herpesviruses, the Immune System, and AIDS. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4613-1507-0_9.

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Martinez-Cajas, Jorge L., Marco Petrella, and Mark A. Wainberg. "The Use of Genotypic Assays for Monitoring the Development of Resistance to Antiviral Therapy for HIV-1 Infection and Other Chronic Viral Diseases." In Antimicrobial Drug Resistance. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-595-8_40.

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Lloyd, Alun, and Dominik Wodarz. "Drug resistance in acute viral infections: Rhinovirus as a case study." In Disease Evolution. American Mathematical Society, 2006. http://dx.doi.org/10.1090/dimacs/071/10.

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Kawamura, Tatsuyoshi. "Viral Infection." In Immunology of the Skin. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55855-2_19.

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Davies, Eryl. "Viral Infection." In The Final FFICM Structured Oral Examination Study Guide. CRC Press, 2022. http://dx.doi.org/10.1201/9781003243694-126.

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Conference papers on the topic "Resistance to viral infection"

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"The role of suppressor protein in acquired resistance to viral infection." In Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/plantgen2019-193.

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Solovyev, A. G., E. A. Tolstyko, A. A. Lezzhov, A. V. Pankratenko, E. A. Lazareva, and S. Y. Morozov. "PHLOEM TRANSPORT OF RNA: ROLES IN VIRAL INFECTION AND PLANT DEFENSE RESPONSE." In The All-Russian Scientific Conference with International Participation and Schools of Young Scientists "Mechanisms of resistance of plants and microorganisms to unfavorable environmental". SIPPB SB RAS, 2018. http://dx.doi.org/10.31255/978-5-94797-319-8-26-28.

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"Computational analysis of viral drug resistance and treatment efficacy: case study for HIV-infection." In Bioinformatics of Genome Regulation and Structure/Systems Biology (BGRS/SB-2022) :. Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, 2022. http://dx.doi.org/10.18699/sbb-2022-205.

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Wali, S., D. Goldblatt, J. R. Flores Gonzalez, M. J. Tuvim, B. F. Dickey, and S. E. Evans. "Inducible Epithelial Resistance Prevents CD8+T Cell-Dependent Lethal Immunopathology Following Respiratory Viral Infections." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1202.

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Баранов, Д. Ю., С. В. Долгов, and В. Р. Тимербаев. "KNOCKOUT OF TOMATO TRANSLATION ELONGATION FACTOR GENES USING CRISPR-CAS9 TECHNOLOGY." In Биотехнология в растениеводстве, животноводстве и сельскохозяйственной микробиологии. Crossref, 2022. http://dx.doi.org/10.48397/arriab.2022.22.xxii.094.

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Фитопатогенные вирусы наносят значительный вред сельскохозяйственному производству. Несмотря на развитие технологий по сдерживанию вирусных болезней томата, включающих как методы классической селекции, так и биотехнологические методы, данные заболевания вызывают серьезные потери урожая по всему миру. Самовоспроизведение фитовирусов включает взаимодействие между вирусными компонентами и факторами растения-хозяина, поэтому мутации в соответствующих генах последних могут обеспечить резистентность к вирусной инфекции. В настоящее время, имеются данные, что белки комплекса эукариотического фактора
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Marii, Liliana, Larisa Andronic, Svetlana Smerea, and Natalia Balasova. "Evaluarea rolului genotipului în răspunsul antioxidativ la tomatele infectate cu virusuri." In VIIth International Scientific Conference “Genetics, Physiology and Plant Breeding”. Institute of Genetics, Physiology and Plant Protection, Republic of Moldova, 2021. http://dx.doi.org/10.53040/gppb7.2021.41.

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Studying the particularities of manifestation of defensive indicators – POX and PPO in case of in-fection with 2 types of viruses of different virus-host combinations (sensitive, tolerant, resistant) was per-formed in basis of analysis of variance. The obtained results denote a significant contribution of all ana-lyzed factors in the variability of PPO and POX indices, the major contribution returning to the genotype, followed by viral infection, the type of viral infection with a variable dose of contribution depending on the applied matrix. The PPO index expressed a higher specificity of the
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Sharma, Richa, Vijay Sharma, and Gaurav Gandhi. "IDDF2019-ABS-0207 First report of primary tenofovir resistance in A hepatitis B viral hepatitis patient from india without human immunodeficiency virus co-infection." In International Digestive Disease Forum (IDDF) 2019, Hong Kong, 8–9 June 2019. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2019. http://dx.doi.org/10.1136/gutjnl-2019-iddfabstracts.282.

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Pereira, Maria Luiza Scardua, Sarah Santos Gonçalves, Creuza Rachel Vicente, Carolina Salume Xavier, Bárbara Ellen Santos Carvalhais, and Kenia Valeria Santos. "Can the therapeutic protocol recommended for gonococcal infection be affected by the COVID-19 pandemic?" In XIII Congresso da Sociedade Brasileira de DST - IX Congresso Brasileiro de AIDS - IV Congresso Latino Americano de IST/HIV/AIDS. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/dst-2177-8264-202133p173.

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Introduction: The World Health Organization has warned of antibiotics misuse in the COVID-19 pandemic. Currently, the clinical management protocol for patients with suspected or confirmed SARS-CoV-2 infection includes the use of empirical antibiotic therapy only for the treatment of bacterial pneumonia in patients admitted with severe acute respiratory syndrome. This protocol includes ceftriaxone, azithromycin, piperacillin/tazobactam, vancomycin, and meropenem. In Brazil, ceftriaxone 500 mg plus azithromycin 1 g is the recommended treatment regimen for mild and moderate anogenital gonococcal
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Khaled, Salma, Catalina Petcu, Lina Bader, et al. "Prevalence and Potential Determinants of COVID-19 Vaccine Hesitancy and Resistance in Qatar." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0074.

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Global COVID-19 pandemic containment necessitates understanding the risk of hesitance or resistance to vaccine uptake in different populations. The Middle East and North Africa currently lack vital representative vaccine hesitancy data. We conducted the first representative national phone survey among the adult population of Qatar, between December 2020 and January 2021, to estimate the prevalence and identify potential determinants of vaccine willingness: acceptance (strongly agree), resistance (strongly disagree), and hesitance (somewhat agree, neutral, somewhat disagree). Bivariate and mult
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Rabbani, Naila, Paul John Thornalley, Maryam Al-Motawa, and Mingzhan Xue. "Vulnerabilities of the SARS-Cov-2 Virus to Proteotoxicity – Opportunity for Repurposed Chemotherapy of COVID-19 Infection." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0291.

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The global pandemic of COVID-19 disease caused by infection with the SARS-CoV-2 Coronavirus, has produced an urgent requirement and search for improved treatments whilst effective vaccines are developed. A strategy for improved drug therapy is to increase levels of endogenous reactive metabolites for selective toxicity to SARS-CoV-2 by preferential damage to the viral proteome. Key reactive metabolites producing major quantitative damage to the proteome in physiological systems are: Reactive oxygen species (ROS) and the reactive glycating agent methylglyoxal (MG); cysteine residues and arginin
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Reports on the topic "Resistance to viral infection"

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Lapidot, Moshe, and Vitaly Citovsky. molecular mechanism for the Tomato yellow leaf curl virus resistance at the ty-5 locus. United States Department of Agriculture, 2016. http://dx.doi.org/10.32747/2016.7604274.bard.

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Tomato yellow leaf curl virus (TYLCV) is a major pathogen of tomato that causes extensive crop loss worldwide, including the US and Israel. Genetic resistance in the host plant is considered highly effective in the defense against viral infection in the field. Thus, the best way to reduce yield losses due to TYLCV is by breeding tomatoes resistant or tolerant to the virus. To date, only six major TYLCV-resistance loci, termed Ty-1 to Ty-6, have been characterized and mapped to the tomato genome. Among tomato TYLCV-resistant lines containing these loci, we have identified a major recessive quan
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Lapidot, Moshe, Linda Hanley-Bowdoin, Jane E. Polston, and Moshe Reuveni. Geminivirus-resistant Tomato Plants: Combining Transgenic and Conventional Strategies for Multi-viral Resistance. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7592639.bard.

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Begomoviruses, which constitute one genus of the Geminiviridae family, are single-stranded DNA viruses that infect many dicotyledonous crops important to large agricultural industries as well as to subsistence growers. Although all begomoviruses are transmitted by whiteflies (Bemisia tabaci), they have proven difficult to manage even with heavy insecticide applications. The begomovirus, Tomato yellow leaf curl virus (TYLCV), has been a problem in tomato production in Israel since the 1950s and in the United States since 1997. Approximately 89 begomoviruses have now been reported to infect toma
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Gal-On, Amit, Shou-Wei Ding, Victor P. Gaba, and Harry S. Paris. role of RNA-dependent RNA polymerase 1 in plant virus defense. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7597919.bard.

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Objectives: Our BARD proposal on the impact of RNA-dependent RNA polymerase 1 (RDR1) in plant defense against viruses was divided into four original objectives. 1. To examine whether a high level of dsRNA expression can stimulate RDR1 transcription independent of salicylic acid (SA) concentration. 2. To determine whether the high or low level of RDR1 transcript accumulation observed in virus resistant and susceptible cultivars is associated with viral resistance and susceptibility. 3. To define the biogenesis and function of RDR1-dependent endogenous siRNAs. 4. To understand why Cucumber mosai
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David, Lior, Yaniv Palti, Moshe Kotler, Gideon Hulata, and Eric M. Hallerman. Genetic Basis of Cyprinid Herpes Virus-3 Resistance in Common Carp. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7592645.bard.

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The goal of this project was to provide scientific and technical basis for initiating the development of breeding protocols using marker assisted selection for viral disease resistance in common carp. The specific objectives were: 1) Establishing families and characterizing the phenotypic and genetic variation of viral resistance; 2) Measuring the dynamics of immune response and developing a method to measure the long term immune memory; 3) Developing markers and generating a new genetic linkage map, which will enable initial QTL mapping; and, 4) Identifying genetic linkage of markers and cand
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Dolja, Valerian V., Amit Gal-On, and Victor Gaba. Suppression of Potyvirus Infection by a Closterovirus Protein. United States Department of Agriculture, 2002. http://dx.doi.org/10.32747/2002.7580682.bard.

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The plant virus family Polyviridae is the largest and most destructive of all plant viruses. Despite the continuous effort to develop resistant plant varieties, there is a desperate need for novel approaches conferring wide-range potyvirus resistance. Based on experiments with the tobacco etch potyvirus (TEV)-derived gene expression vector, we suggested approach for screening of the candidate resistance genes. This approach relies on insertion of the genes into a virus vector and evaluation of the phenotypes of the resulting recombinant viruses. The genes which suppress infection by the recomb
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Palukaitis, Peter, Amit Gal-On, Milton Zaitlin, and Victor Gaba. Virus Synergy in Transgenic Plants. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7573074.bard.

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Transgenic plants expressing viral genes offer novel means of engendering resistance to those viruses. However, some viruses interact synergistically with other viruses and it is now known that transgenic plants expressing particular genes of one virus may also mediate synergy with a second virus. Thus, our specific objectives were to (1) determine if transgenic plants resistant to one virus showed synergy with another virus; (2) determine what viral sequences were essential for synergy; and (3) determine whether one of more mechanisms were involved i synergy. This project would also enable an
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Wang, X. F., and M. Schuldiner. Systems biology approaches to dissect virus-host interactions to develop crops with broad-spectrum virus resistance. United States-Israel Binational Agricultural Research and Development Fund, 2020. http://dx.doi.org/10.32747/2020.8134163.bard.

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More than 60% of plant viruses are positive-strand RNA viruses that cause billion-dollar losses annually and pose a major threat to stable agricultural production, including cucumber mosaic virus (CMV) that infects numerous vegetables and ornamental trees. A highly conserved feature among these viruses is that they form viral replication complexes (VRCs) to multiply their genomes by hijacking host proteins and remodeling host intracellular membranes. As a conserved and indispensable process, VRC assembly also represents an excellent target for the development of antiviral strategies that can b
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Mawassi, Munir, Adib Rowhani, Deborah A. Golino, Avichai Perl, and Edna Tanne. Rugose Wood Disease of Grapevine, Etiology and Virus Resistance in Transgenic Vines. United States Department of Agriculture, 2003. http://dx.doi.org/10.32747/2003.7586477.bard.

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Rugose wood is a complex disease of grapevines, which occurs in all growing areas. The disease is spread in the field by vector transmission (mealybugs). At least five elongated-phloem- limited viruses are implicated in the various rugose wood disorders. The most fully characterized of these are Grapevine virus A (GV A) and GVB, members of a newly established genus, the vitivirus. GVC, a putative vitivirus, is much less well characterized than GV A or GVB. The information regarding the role of GVC in the etiology and epidemiology of rugose wood is fragmentary and no sequence data for GVC are a
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Gafni, Yedidya, and Vitaly Citovsky. Molecular interactions of TYLCV capsid protein during assembly of viral particles. United States Department of Agriculture, 2007. http://dx.doi.org/10.32747/2007.7587233.bard.

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Tomato yellow leaf curl geminivirus (TYLCV) is a major pathogen of cultivated tomato, causing up to 100% crop loss in many parts of the world. The present proposal, a continuation of a BARD-funded project, expanded our understanding of the molecular mechanisms by which CP molecules, as well as its pre-coat partner V2, interact with each other (CP), with the viral genome, and with cellular proteins during assembly and movement of the infectious virions. Specifically, two major objectives were proposed: I. To study in detail the molecular interactions between CP molecules and between CP and ssDN
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Chejanovsky, Nor, and Bruce A. Webb. Potentiation of Pest Control by Insect Immunosuppression. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7592113.bard.

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The restricted host range of many baculoviruses, highly pathogenic to Lepidoptera and non-pathogenic to mammals, limits their use to single or few closely related Lepidopteran species and is an obstacle to extending their implementation for pest control. The insect immune response is a major determinant of the ability of an insect pathogen to efficiently multiply and propagate. We have developed an original model system to study the Lepidopteran antiviral immune response based on Spodoptera littoralis resistance to AcMNPV (Autographa californica multiple nucleopolyhedrovirus) infection and the
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