Academic literature on the topic 'Resorption (Physiology)'

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Journal articles on the topic "Resorption (Physiology)"

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Liesegang, A., M. L. Sassi, J. Risteli, R. Eicher, M. Wanner, and J. L. Riond. "Physiology of bone resorption during hypocalcemia in dairy cows." Journal of Animal Physiology and Animal Nutrition 80, no. 1-5 (September 12, 1998): 82–85. http://dx.doi.org/10.1111/j.1439-0396.1998.tb00507.x.

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Chattopadhyay, Naibedya. "Adiponectin Signaling Regulates Skeletal Physiology." INDIAN JOURNAL OF PHYSIOLOGY AND ALLIED SCIENCES 74, no. 02 (June 15, 2022): 39–40. http://dx.doi.org/10.55184/ijpas.v74i02.57.

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Bone remodelling is important to maintain the skeletal physiology. Bone loss with aging and hormonal pathologies may be result ofaltered bone remodelling leading to osteoporosis. Even in presence of existing therapies, there is an unmet clinical need to look forideal alternatives that would stimulate bone formation and keep resorption in check. Adiponectin and its derivatives could be a possiblecandidate for such therapy. Orally active small molecule AdipoR agonists may be a proposed solution for this.
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Ancajima Ramírez, Charton Dick, Frederik Maximiliano Parra Peña, Grace Teresa Panta Juárez, Luis Jaramillo Liviapoma, Ruth Marianella Huertas Coronado, and Marisel Roxana Valenzuela Ramosa. "Pregnancy, orthodontics and bone resorption." World Health Journal 2, no. 1 (April 23, 2021): 12–15. http://dx.doi.org/10.47422/whj.v2i1.10.

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Pregnant women present different changes in the skeletal system, such as the increase in calcium throughout this period, there are also small reductions in bone density. Orthodontic tooth movement is based on the principles of tissue resorption and formation at the level of the surrounding bone and periodontal ligament. It should be noted that there are multiple factors that affect the speed of this type of movement. During pregnancy and lactation, certain alterations in orthodontic dental movement may be perceived, caused by changes in bone homeostasis, alterations in tooth resorption and obs
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Lees, Rita L., and Johan N. M. Heersche. "Differences in regulation of pHi in large (≥10 nuclei) and small (≤5 nuclei) osteoclasts." American Journal of Physiology-Cell Physiology 279, no. 3 (September 1, 2000): C751—C761. http://dx.doi.org/10.1152/ajpcell.2000.279.3.c751.

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Osteoclasts are multinucleated cells that resorb bone by extrusion of protons and proteolytic enzymes. They display marked heterogeneity in cell size, shape, and resorptive activity. Because high resorptive activity in vivo is associated with an increase in the average size of osteoclasts in areas of greater resorption and because of the importance of proton extrusion in resorption, we investigated whether the activity of the bafilomycin A1-sensitive vacuolar-type H+-ATPase (V-ATPase) and amiloride-sensitive Na+/H+ exchanger differed between large and small osteoclasts. Osteoclasts were obtain
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Turner, R. T., L. S. Kidder, M. Zhang, S. A. Harris, K. C. Westerlind, A. Maran, and T. J. Wronski. "Estrogen has rapid tissue-specific effects on rat bone." Journal of Applied Physiology 86, no. 6 (June 1, 1999): 1950–58. http://dx.doi.org/10.1152/jappl.1999.86.6.1950.

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The decrease in cancellous bone formation after estrogen treatment is generally thought to be coupled with a prior decrease in bone resorption. To test the possibility that estrogen has rapid tissue-specific actions on bone metabolism, we determined the time course (1–32 h) effects of diethylstilbestrol on steady-state mRNA levels for immediate-response genes, extracellular matrix proteins, and signaling peptides in the proximal tibial metaphysis and uterus by using Northern blot and RNase protection assays. The regulation of signaling peptides by estrogen, although tissue specific, followed a
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Geng, W., and G. L. Wright. "Skeletal sensitivity to dietary calcium deficiency is increased in the female compared with the male rat." Canadian Journal of Physiology and Pharmacology 79, no. 5 (May 1, 2001): 379–85. http://dx.doi.org/10.1139/y01-005.

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We investigated potential sex differences in bone resorption and the conservation of whole body bone mass in 24-week-old Sprague-Dawley rats maintained on a 1.0% calcium diet and then fed diets containing 0.02, 0.5, 1.0, or 1.75% calcium for 31 days. Lowering dietary calcium from 1.00% to 0.02% doubled whole skeleton bone resorption (urinary 3H-tetracycline loss). Female rats were more sensitive to calcium stress, exhibiting the maximal resorptive response when fed the 0.5% calcium diet, whereas the 0.02% calcium diet was required to elicit this response in males. Despite the evidence of incre
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Xie, Jingbo, Jian Guo, Zaeema Kanwal, Mingzheng Wu, Xiangyang Lv, Nihal Abdalla Ibrahim, Ping Li, Manal Ali Buabeid, El-Shaimaa A. Arafa, and Qingshan Sun. "Calcitonin and Bone Physiology: In Vitro, In Vivo, and Clinical Investigations." International Journal of Endocrinology 2020 (September 10, 2020): 1–20. http://dx.doi.org/10.1155/2020/3236828.

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Calcitonin was discovered as a peptide hormone that was known to reduce the calcium levels in the systemic circulation. This hypocalcemic effect is produced due to multiple reasons such as inhibition of bone resorption or suppression of calcium release from the bone. Thus, calcitonin was said as a primary regulator of the bone resorption process. This is the reason why calcitonin has been used widely in clinics for the treatment of bone disorders such as osteoporosis, hypercalcemia, and Paget’s disease. However, presently calcitonin usage is declined due to the development of efficacious formu
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Sims, Natalie A., and T. John Martin. "Osteoclasts Provide Coupling Signals to Osteoblast Lineage Cells Through Multiple Mechanisms." Annual Review of Physiology 82, no. 1 (February 10, 2020): 507–29. http://dx.doi.org/10.1146/annurev-physiol-021119-034425.

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Bone remodeling is essential for the repair and replacement of damaged and old bone. The major principle underlying this process is that osteoclast-mediated resorption of a quantum of bone is followed by osteoblast precursor recruitment; these cells differentiate to matrix-producing osteoblasts, which form new bone to replace what was resorbed. Evidence from osteopetrotic syndromes indicate that osteoclasts not only resorb bone, but also provide signals to promote bone formation. Osteoclasts act upon osteoblast lineage cells throughout their differentiation by facilitating growth factor releas
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Kullama, L. K., C. L. Agnew, L. Day, M. G. Ervin, and M. G. Ross. "Ovine fetal swallowing and renal responses to oligohydramnios." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 266, no. 3 (March 1, 1994): R972—R978. http://dx.doi.org/10.1152/ajpregu.1994.266.3.r972.

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Amniotic fluid (AF) volume regulation is dependent on a balance between fluid production and fluid resorption. We examined the effects of reduced AF volume on AF production by fetal urine and resorption by fetal swallowing and the response of these parameters to AF volume replacement. Eight time-dated pregnant ewes (125 +/- 1 days gestation) were studied before (day 1) and after (day 3) AF and fetal urine drainage. Drainage resulted in a significant decrease in AF volume (415 +/- 89 to 157 +/- 36 ml). Fetal urine osmolality increased (139 +/- 10 to 286 +/- 33 mosmol/kgH2O), while urine flow di
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Carano, A., P. H. Schlesinger, N. A. Athanasou, S. L. Teitelbaum, and H. C. Blair. "Acid and base effects on avian osteoclast activity." American Journal of Physiology-Cell Physiology 264, no. 3 (March 1, 1993): C694—C701. http://dx.doi.org/10.1152/ajpcell.1993.264.3.c694.

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Osteoclasts generate a massive acid flux to mobilize bone calcium. Local extracellular acidification by polarized vacuolar-type H(+)-ATPase, balanced by contralateral HCO3-(-)Cl- exchange to maintain physiological intracellular pH, is theorized to drive this process. It follows that extracellular pH, PCO2, or HCO3- concentration ([HCO3-]) should impact bone matrix dissolution. However, the effects on bone resorption of the concentrations of these ions or their transmembrane gradients are unknown. Furthermore, because bone management is a vital process, regulatory feedback may minimize such eff
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Dissertations / Theses on the topic "Resorption (Physiology)"

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Dreyer, Craig William. "Clast cell activity in a model of aseptic root resorption." Title page, table of contents and abstract only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phd778.pdf.

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Ngan, Daniel C. S. "The genetic contribution to orthodontic root resorption : a retrospective twin study." Thesis, The University of Sydney, 2003. http://hdl.handle.net/2123/4781.

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Brice, Garth Loyola. "The interrelationship of epithelial rests of Malassez with orthodontic root resorption and repair in man /." Title page, contents and summary only, 1988. http://web4.library.adelaide.edu.au/theses/09DM/09dmb849.pdf.

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Malek, Sameh. "Physical properties of human premolar cementum : a structural correlation." Thesis, The University of Sydney, 2000. http://hdl.handle.net/2123/4696.

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Kebsch, Mark Peter. "The effect of fluoride administration on rat serum osteocalcin expression during orthodontic tooth movement : the Osteocalcin Project." Thesis, The University of Sydney, 2004. http://hdl.handle.net/2123/4795.

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容冠宇 and Koon-yu Samuel Yung. "Effects of green tea on bone loss in mature ovariectomized rat." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31970333.

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Wu, Andy Tzong Jong. "The extent of root resorption following the application of light (25g) and heavy (225g) controlled rotational orthodontic forces for 4 weeks: a micro-CT study." Thesis, The University of Sydney, 2009. https://hdl.handle.net/2123/28194.

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In 1932 Beck and Marshall defined root resorption as the destruction of formed tooth structure. Root resorption results in the loss of substance from dentine or cementum and can present as either a physiological or pathological process. Physiological root resorption can occur in both deciduous and permanent dentition. Root resorption of the deciduous dentition is a normal and essential physiological process that facilitates natural exfoliation. Pathological resorption is an inflammatory process that is triggered by numerous factors. Root resorption following orthodontic treatment is i
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Wong, Mark. "Radiographic evaluation of root morphology in association with apical root resorption." 1997. http://catalog.hathitrust.org/api/volumes/oclc/48166828.html.

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Dreyer, Craig William. "Clast cell activity in a model of aseptic root resorption / Craig William Dreyer." Thesis, 2002. http://hdl.handle.net/2440/21834.

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Kavukcuoglu, Nadire Beril. "Correlating the nanoscale mechanical and chemical properties of knockout mice bones." 2007. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13470.

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Books on the topic "Resorption (Physiology)"

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Harvey, Wilson. Prostaglandins in bone resorption. Boca Raton, Fla: CRC Press, 1988.

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Duchesne, Louis. Cycle biochimique des éléments nutritifs majeurs dans le bassin versant du lac Clair, Duchesnay, Québec. [Charlesbourg]: Forêt Québec, Direction de la recherche forestière, 1999.

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Maria, Bijvoet Olav Leonardus, ed. Bisphosphonate on bones. Amsterdam: Elsevier, 1995.

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Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1989.

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Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1989.

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Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1986.

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Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1989.

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Skiba, Grzegorz. Fizjologiczne, żywieniowe i genetyczne uwarunkowania właściwości kości rosnących świń. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_gs_2020.

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Bones are multifunctional passive organs of movement that supports soft tissue and directly attached muscles. They also protect internal organs and are a reserve of calcium, phosphorus and magnesium. Each bone is covered with periosteum, and the adjacent bone surfaces are covered by articular cartilage. Histologically, the bone is an organ composed of many different tissues. The main component is bone tissue (cortical and spongy) composed of a set of bone cells and intercellular substance (mineral and organic), it also contains fat, hematopoietic (bone marrow) and cartilaginous tissue. Bones a
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Book chapters on the topic "Resorption (Physiology)"

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Mundy, G. R. "Hormonal Factors Which Regulate Bone Resorption." In Physiology and Pharmacology of Bone, 215–47. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77991-6_6.

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MORRISON, B. "Physiology of Cerebrospinal Fluid Secretion, Recirculation, and Resorption." In Cerebrospinal Fluid in Clinical Practice, 11–17. Elsevier, 2009. http://dx.doi.org/10.1016/b978-141602908-3.50006-6.

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Turner, Helen E., Richard Eastell, and Ashley Grossman. "Bone." In Endocrinology (Oxford Desk Reference), 330–61. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199672837.003.0012.

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This chapter discusses the anatomy and physiology of the bone, including mineralization, and outlines techniques in bone remodelling. It describes formation and resorption hormonal markers that are part of the bone remodelling cycle, such as procollagens and serums. It describes how diagnostic measures in these formation markers are increased for focal bone disorders like Paget’s disease, fibrous dysplasia, osteomalacia, bone metastases, myeloma, primary hyperparathyroidism, thyrotoxicosis, and acromegaly. The chapter also discusses osteoporosis, including causes, symptoms, and treatment options. Clinical suggestions for bone diagnoses and diseases are provided, based on dual-energy X-ray absorptiometry (commonly abbreviated as DXA), plain radiography, and bone biopsy. The chapter also defines osteogenesis imperfecta and describes its epidemiology and management. In addition, it outlines sclerosing bone disorders such as osteopetrosis, pycnodysostosis, and hyperostosis type Worth, as well as fibrodysplasia ossificans progressive.
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"Aktive Na+-Resorption und die FolgenNa+-Resorption proximaler Tubulus." In Physiologie, edited by Hans-Christian Pape, Armin Kurtz, and Stefan Silbernagl. Stuttgart: Georg Thieme Verlag, 2014. http://dx.doi.org/10.1055/b-0034-98533.

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"Resorption organischer Substanzen." In Taschenatlas Physiologie, edited by Stefan Silbernagl and Agamemnon Despopoulos. Stuttgart: Georg Thieme Verlag, 2007. http://dx.doi.org/10.1055/b-0034-89407.

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"Resorption organischer Substanzen." In Taschenatlas Physiologie, edited by Stefan Silbernagl and Agamemnon Despopoulos. Stuttgart: Georg Thieme Verlag, 2012. http://dx.doi.org/10.1055/b-0034-49892.

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"Fall 31 Resorption von Nahrungsbestandteilen." In Physiologie, edited by Kerstin Walter. Stuttgart: Georg Thieme Verlag, 2006. http://dx.doi.org/10.1055/b-0034-28289.

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"Resorption von Na+- und Cl-." In Taschenatlas Physiologie, edited by Stefan Silbernagl and Agamemnon Despopoulos. Stuttgart: Georg Thieme Verlag, 2007. http://dx.doi.org/10.1055/b-0034-89409.

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"7.8 Die Resorption der Nahrungsbestandteile." In Kurzlehrbuch Physiologie, edited by Jens Huppelsberg and Kerstin Walter. Stuttgart: Georg Thieme Verlag, 2009. http://dx.doi.org/10.1055/b-0034-22345.

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"Resorption von Na+ und Cl−." In Taschenatlas Physiologie, edited by Stefan Silbernagl and Agamemnon Despopoulos. Stuttgart: Georg Thieme Verlag, 2012. http://dx.doi.org/10.1055/b-0034-49894.

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