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1

Hansson, Anna. "Cellular responses to respiratory chain dysfunction /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-493-7/.

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2

Silva, José Pablo. "The pathophysiology of respiratory chain dysfunction /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-234-9/.

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3

Li, Xi. "The respiratory chain in Neisseria species." Thesis, University of York, 2013. http://etheses.whiterose.ac.uk/3989/.

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This work presents the organization of respiratory chain in Neisseria species. The localization of redox proteins was determined. Lipid-modified azurin (Laz) and nitrite reductase (AniA) are mainly associated with outer membrane. All c-type cytochrome proteins are mainly associated with inner membrane. Cytochrome c5 is the major electron donor to AniA. Reduced form cytochrome c5 is able to donate electrons to AniA at a physiologically relevant rate. In addition, the second haem domain of cytochrome c5 is the direct donor to AniA. It presents a potential problem for inter-electron transfer betw
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4

Chen, Walter W. "Pathological features of mitochondrial respiratory chain dysfunction." Thesis, Massachusetts Institute of Technology, 2016. http://hdl.handle.net/1721.1/104099.

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Thesis: Ph. D., Massachusetts Institute of Technology, Department of Biology, 2016.<br>This electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.<br>Cataloged from student-submitted PDF version of thesis. "June 2016."<br>Includes bibliographical references.<br>Mitochondria are essential organelles that carry out a multitude of important metabolic processes in mammalian organisms. These processes include ATP generation by the respiratory chain, aspartate synthesis by matrix aminotransferases, and long-chain
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5

Capristo, Mariantonietta <1981&gt. "Respiratory chain complex I dysfunction in tumorigenesis." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2012. http://amsdottorato.unibo.it/4798/1/Capristo_Mariantonietta_Tesi.pdf.

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Diseases due to mutations in mitochondrial DNA probably represent the most common form of metabolic disorders, including cancer, as highlighted in the last years. Approximately 300 mtDNA alterations have been identified as the genetic cause of mitochondrial diseases and one-third of these alterations are located in the coding genes for OXPHOS proteins. Despite progress in identification of their molecular mechanisms, little has been done with regard to the therapy. Recently, a particular gene therapy approach, namely allotopic expression, has been proposed and optimized, although the results o
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6

Capristo, Mariantonietta <1981&gt. "Respiratory chain complex I dysfunction in tumorigenesis." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2012. http://amsdottorato.unibo.it/4798/.

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Diseases due to mutations in mitochondrial DNA probably represent the most common form of metabolic disorders, including cancer, as highlighted in the last years. Approximately 300 mtDNA alterations have been identified as the genetic cause of mitochondrial diseases and one-third of these alterations are located in the coding genes for OXPHOS proteins. Despite progress in identification of their molecular mechanisms, little has been done with regard to the therapy. Recently, a particular gene therapy approach, namely allotopic expression, has been proposed and optimized, although the results o
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7

Jackson, Margaret J. "Clinical and biochemical studies of respiratory chain disease." Thesis, University of Newcastle Upon Tyne, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294642.

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8

Heiske, Margit. "Modeling the respiratory chain and the oxidative phosphorylation." Thesis, Bordeaux 2, 2012. http://www.theses.fr/2012BOR21965/document.

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Mitochondria are cell organelles which play an essential role in the cell energy supply providing the universal high energetic molecule ATP which is used in numerous energy consuming processes. The core of the ATP production, oxidative phosphorylation (OXPHOS) consists of four enzyme complexes (respiratory chain) which establish, driven by redox reactions, a proton gradient over the inner mitochondrial membrane. The ATP-synthase uses this electrochemical gradient to phosphorylate ADP to ATP. Dysfunctioning of an OXPHOS complex can have severe consequences for the energy metabolism and cause ra
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9

Heiske, Margit. "Modeling the respiratory chain and the oxidative phosphorylation." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2013. http://dx.doi.org/10.18452/16720.

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Die oxidative Phosphorylierung (OXPHOS) spielt eine zentrale Rolle im Energiestoffwechsel der Zelle. Sie besteht aus der Atmungskette, deren vier Enzymkomplexe einen Protonengradienten über die innere mitochondriale Membran aufbauen, und der ATP-Synthase, die diesen Gradienten zur Phosphorylierung von ADP zu ATP, der zelluläre Energieeinheit, nutzt. In der vorliegenden Arbeit wurde ein thermodynamisch konformes OXPHOS Modell erstellt, welches auf Differentialgleichungen basiert. Dazu wurden Gleichungen entwickelt, welche die Kinetiken jedes OXPHOS-Komplexes über weite Bereiche von Substrat- u
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10

Taylor, Claire Louise. "Biochemical investigations of defects of the mitochondrial respiratory chain." Thesis, University of Newcastle Upon Tyne, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.281706.

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11

Kwong, Jennifer Quo-Yee. "The mitochondrial respiratory chain is a modulator of apoptosis /." Access full-text from WCMC, 2007. http://proquest.umi.com/pqdweb?did=1441191921&sid=24&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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12

Miley, Timothy Brian. "Studies of the respiratory chain of Methylococcus capsulatus (bath)." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1252.

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13

Brierley, Elizabeth Jane. "Defects of mitochondrial DNA and mitochondrial energy production in ageing." Thesis, University of Newcastle Upon Tyne, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323477.

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14

Eubel, Holger. "The supramolecular structure of the respiratory chain of higher plants." [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=975827413.

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15

Menezes, Minal Juliet. "Gene discovery and functional studies of mitochondrial respiratory chain disorders." Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/12688.

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Mitochondrial respiratory chain disorders (MRCD) are one of the most challenging inborn errors of metabolism to diagnose and treat, because of their phenotypic and genetic heterogeneity, and a paucity of validated therapies. The approach opted to diagnose our MRCD patient cohort was using next generation sequencing (NGS) technology, and in particular whole exome sequencing (WES). Using our research pipeline we were able to identify novel YARS2 variants in patients with a tissue specific MRCD. Novel disease gene discoveries (CYC1 and MRPS7) causing MRCD were identified using the WES approach, a
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16

Morris, Andrew Alan Myles. "Disorders of mitochondrial oxidation : clinical, biochemical & molecular studies." Thesis, University of Newcastle Upon Tyne, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294810.

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17

Taylor, Robert William. "Mitochondrial respiratory chain dysfunction in human pathology : investigation, pathogenicity and treatment." Thesis, University of Newcastle Upon Tyne, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.577189.

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The work presented in this thesis comprises 100 peer-reviewed publications, mostly original research papers but some key review articles are included, which highlight my ongoing research in understanding the role of mitochondrial respiratory chain dysfunction and mitochondrial DNA (mtDNA) mutation in human pathologies over a twenty year period, and in no small part have contributed to the development of my laboratory as a national referral centre in the UK for diagnostic biochemical and molecular genetic testing, funded by the NHS Specialist Commissioners. A significant proportion (at least 50
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18

Lowerson, Shelagh Anne. "Defects of the mitochondrial respiratory chain : biochemical studies and mathematical modelling." Thesis, University of Newcastle Upon Tyne, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297572.

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19

Owen, Mark R. "The mechanisms by which mild respiratory chain inhibitors regulate hepatic gluconeogenesis." Thesis, University of Bristol, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.358337.

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20

Nelson, Christy L. "Branched-chain amino acid nutrition and respiratory stability in premature infants." free to MU campus, others may purchase free online, 2002. http://wwwlib.umi.com/cr/mo/preview?3074432.

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21

Williams, Andrew. "Functional and molecular analysis of defects of the mitochondrial respiratory chain." Thesis, The University of Sydney, 1998. https://hdl.handle.net/2123/27688.

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The diagnosis of human mitochondrial respiratory chain defects is based on a staged procedure including: screening tests, enzymology of tissues and cultured cells and use of molecular techniques or cybrid technology to assign the site of the genetic defect This thesis examines aspects of each of these stages and presents improved methods for screening, enzymology and cybrid technology. Analyses of enzymes and proteins in the detection of respiratory chain defects are traditionally performed by manual assays. In chapters 2 and 3, I present automated methods for total protein, citrate s
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22

Marshall, Douglas Charles Alexander. "Structural and functional studies of respiratory chain proteins using infrared spectroscopy." Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1446092/.

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Attenuated total reflection Fourier transform infrared (FTIR) spectroscopy in the 4000-800 cm"1 range was applied to NADH:ubiquinone oxidoreductase (complex I) and cytochrome bc complex of the respiratory electron transfer chain. High signal:noise redox difference spectra were acquired using novel electrochemical and perfusion techniques to reversibly adjust protein redox state which, where appropriate, was monitored by simultaneous UV/visible spectroscopy. Interpretation of spectra was assisted by hydrogen-deuterium exchange, 15N labelling and redox titration. Model compound data, including p
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23

Parmar, Gaganvir. "Protein Factors Regulating Mitochondrial Respiratory Supercomplexes." Thesis, Université d'Ottawa / University of Ottawa, 2021. http://hdl.handle.net/10393/42350.

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24

Cogliati, Sara. "OPA1-dependent cristae remodeling disassembles respiratory chain supercomplexes, triggering apoptotic mitochondrial dysfunction." Doctoral thesis, Università degli studi di Padova, 2011. http://hdl.handle.net/11577/3421570.

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Mitochondria are key organelles in intermediatig cellular metabolism, energy production and calcium homeostasis (Rizzuto et al., 2000; Danial and Korsmeyer, 2004). They also integrate and amplify apoptosis induced by several intrinsic stimuli (Green and Reed, 1998). Their structure is extremely complex, being bound by two membranes. The inner membrane (IMM) can be further divided in two distinct compartments, the so called “boundary membrane” and the cristae, separated from the former by narrow tubular junctions. Such a functional versatility and complexity is controlled by a growing family of
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25

Neehaul, Yashvin. "Study of protein in the respiratory chain by IR spectroscopy and electrochemistry." Phd thesis, Université de Strasbourg, 2012. http://tel.archives-ouvertes.fr/tel-00827526.

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The field of molecular bioenergetics deals with the energy transduction in biological cells. In this project, respiration and more specifically proton and sodium pumping enzymes and their coupling to electron transfer have been in focus. First we have been interested in the Na+-pumping NADH:quinone reductase from Vibrio cholerae which is the entry site of electrons in the respiratory chain of several pathogens. The role of specific flavin cofactors and amino acids involved in Na+ transfer has been shown in a combined IR spectroscopic and electrochemical approach. The interaction between protei
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26

Pulkes, Teeratorn. "Studies on the genetics and molecular pathogenesis of mitochondrial respiratory chain disorders." Thesis, University College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411323.

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27

Pagnamenta, Alistair. "Identification of nuclear genes responsible for mitochondrial respiratory chain disorders in childhood." Thesis, University College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.437288.

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28

Tropeano, Concetta Valentina <1987&gt. "Respiratory Chain Complexes and Supercomplexes Organization in Cells with Defective Complex III." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2016. http://amsdottorato.unibo.it/7634/1/tropeano_concettavalentina_tesi.pdf.

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Cytochrome b is the only subunit of complex III (CIII) encoded by the mitochondrial DNA. Constituting the central core of the enzyme, the protein is essential for both assembly and catalytic activity of the complex. CIII can associate with complex I (CI) and complex IV to form supercomplexes (SCs). MTCYB mutations can affect CIII only or both CIII and CI, as a consequence of the importance of CIII on the stability of CI. Here, we have investigated the effects of two pathogenic mutations affecting MTCYB: the p.278Y>C missense mutation, causing the substitution of conserved Tyr278 close to the Q
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29

Tropeano, Concetta Valentina <1987&gt. "Respiratory Chain Complexes and Supercomplexes Organization in Cells with Defective Complex III." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2016. http://amsdottorato.unibo.it/7634/.

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Cytochrome b is the only subunit of complex III (CIII) encoded by the mitochondrial DNA. Constituting the central core of the enzyme, the protein is essential for both assembly and catalytic activity of the complex. CIII can associate with complex I (CI) and complex IV to form supercomplexes (SCs). MTCYB mutations can affect CIII only or both CIII and CI, as a consequence of the importance of CIII on the stability of CI. Here, we have investigated the effects of two pathogenic mutations affecting MTCYB: the p.278Y>C missense mutation, causing the substitution of conserved Tyr278 close to the Q
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30

Tabrizi, Sarah Joanna. "Mitochondrial dysfunction in the pathogenesis of neurodegeneration." Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325866.

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31

Reed, J. S. "The function of ubiquinone in mitochondrial electron transport." Thesis, University of Southampton, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.376174.

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32

Bouverot, Romain. "Etudes structurales de la protéine ACAD9 et des facteurs d'assemblage du complexe 1 de la chaîne respiratoire mitochondriale pour établir leur implication dans les processus neurodégénératifs." Thesis, Université Grenoble Alpes (ComUE), 2019. http://www.theses.fr/2019GREAV005.

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Les mitochondries sont en charge de la bioénergétique cellulaire, tout particulièrement dans le cerveau humain, au sein duquel les neurones sont extrêmement demandeurs en énergie et hautement dépendant de la phosphorylation oxydative. En effet, celles-ci génèrent un potentiel énergétique grâce à une chaîne de transport d’électrons, ou chaîne respiratoire, composée de quatre complexes protéiques ancrés dans la membrane interne mitochondriale. La chaine respiratoire permet la production d’énergie via la phosphorylation oxydative d’ADP en ATP par l’ATP synthéase dans la matrice mitochondriale. Le
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33

Sabuncu, Sinan. "Investigation of enzymes from the respiratory chain by using electrochemical and spectroscopic techniques." Thesis, Strasbourg, 2017. http://www.theses.fr/2017STRAF017/document.

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Le présent travail porte sur l’étude de deux protéines de la famille des oxydases à hème-fer par des techniques de spectroscopie et d’électrochimie. Le premier chapitre décrit l’étude du cytochrome bo3 oxydase issue d’E. coli. Nous nous sommes intéressés à l’étude des interactions enzyme-quinone par l’utilisation de quinones avec des longueurs chaines isoprenyl différentes. Notre but est de mieux comprendre le rôle de la longueur de la chaine des quinones sur l’activité catalytique de l’enzyme et sur les propriétés redox des cofacteurs à hème. Dans l’étape suivante, on a étudié les résidus imp
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34

Alderson, Jesse. "Studies on the aerobic respiratory chain of the human gastric pathogen Helicobacter pylori." Thesis, University of Sheffield, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324459.

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35

Pham, Nhu-An. "Generation of oxidative stress by the respiratory chain following treatment with DNA damaging agents." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0003/MQ46064.pdf.

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36

Schweizer, Ina [Verfasser], and Thorsten [Akademischer Betreuer] Friedrich. "Studies on the Escherichia coli aerobic respiratory chain with an emphasis on complex I." Freiburg : Universität, 2016. http://d-nb.info/1122647670/34.

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37

Sasarman, Florin. "Adult mitochrondrial myopathy associated with generalized respiratory chain deficiency : molecular mechanism and genetic basis." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84321.

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Cellular ATP is synthesized by the mitochondrial oxidative phosphorylation (OXPHOS) system, composed of five enzyme complexes (Complexes I--V), which consist collectively of over 80 subunits. The majority of these subunits are encoded by nuclear genes, and 13 of them, by mitochondrial DNA (mtDNA). OXPHOS deficiencies resulting in mitochondrial disorders can be caused by either nuclear or mitochondrial mutations; however, most pathogenic mutations reported in adults occur in mtDNA. Such mutations often impair mitochondrial translation and are associated with a characteristic muscle patho
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38

Hwang, Ming. "The role of complex II of the mitochondrial respiratory chain in cell death induction." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/25068.

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Complex II of the respiratory chain (RC) recently emerged as a prominent regulator of cell death. In both cancer as well as neurodegenerative diseases, mutations in its subunits have been found along with other genetic alterations indirectly affecting this complex. Moreover, anticancer compounds were developed that target complex II and cause cell death in a tumour-specific way. I found evidence that this protein assembly is specifically activated for cell death via the dissociation of its SDHA and SDHB subunits from the membrane-anchoring proteins through mitochondrial Ca2+ influx. The relati
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39

Homberg, Bettina [Verfasser]. "Regulation of Respiratory Chain Supercomplex Formation and the Involvement of Rcf-proteins / Bettina Homberg." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2021. http://d-nb.info/1235222667/34.

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40

Nunoura, Takuro. "Study of Aerobic Respiratory Chain of Novel Facultative Aerobic and Hyperthermophilic Archaeon Pyrobaculum oguniense." Kyoto University, 2002. http://hdl.handle.net/2433/149916.

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Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(農学)<br>甲第9630号<br>農博第1258号<br>新制||農||844(附属図書館)<br>学位論文||H14||N3662(農学部図書室)<br>UT51-2002-G388<br>京都大学大学院農学研究科応用生物科学専攻<br>(主査)教授 内田 有恆, 教授 加藤 暢夫, 助教授 左子 芳彦<br>学位規則第4条第1項該当
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41

Spinazzi, Marco. "Optimized protocols for the analysis of mithocondrial respiratory chain enzymes in cells and tissues." Doctoral thesis, Università degli studi di Padova, 2012. http://hdl.handle.net/11577/3422449.

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Mitochondria are fundamental cellular organelles which decide about life and death of the cells. They are critically involved in a number of essential functions, including the production of energy through the respiratory chain enzymes, regulation of apoptosis, calcium metabolism and production of reactive oxygen species. Dysfunction of mitochondrial metabolism has been recognized a key player in a number of conditions, including mitochondrial disorders, diabetes, cancer, ageing, neurodegeneration and intoxications from drugs and poisons. Therefore, a reliable assessment of mitochondrial respir
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42

Desmard, Mathieu. "Effets antibactériens sur Pseudomonas aeruginosa des donneurs de monoxyde de carbone." Thesis, Paris Est, 2010. http://www.theses.fr/2010PEST0039.

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La recherche de nouvelles molécules pour combattre Pseudomonas.aeruginosa est d'une grande importance. L'utilisation des antibiotiques a spectre large a grandement accru la résistance de P.aeruginosa aux antibiotiques. Malgré cette situation, aucune nouvelle drogue active sur P.aeruginosa n'a été introduite en pratique clinique durant les 2 dernières décennies. Le monoxyde de carbone (CO) pourrait agir comme un inhibiteur efficace de la chaîne respiratoire de P.aeruginosa mais l'utilisation pratique de ce gaz comme molécule antibactérienne est gênée par sa toxicité et les difficultés de manipu
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Bowman, Amy. "Investigating the role of mitochondrial respiratory chain activity and mitochondrial DNA damage in skin ageing." Thesis, University of Newcastle upon Tyne, 2014. http://hdl.handle.net/10443/2502.

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Ageing describes the progressive functional decline of an organism over time, leading to an increase in susceptibility to age-related diseases and eventually to death, and it is a phenomenon observed across a wide range of organisms. Despite a vast repertoire of ageing studies performed over the past century, the exact causes of ageing remain unknown. For over 50 years it has been speculated that mitochondria play a key role in the ageing process, due mainly to correlative data showing an increase in mitochondrial dysfunction, mitochondrial DNA (mtDNA) damage, and reactive oxygen species (ROS)
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44

El, Khoury Youssef. "Mid and far infrared spectroelectrochemical studies on the metal−ligand interactions in respiratory chain enzymes." Strasbourg, 2010. https://publication-theses.unistra.fr/public/theses_doctorat/2010/EL_KHOURY_Youssef_2010.pdf.

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La thèse contient trois parties principales, la coordination du Cu, l'inhibition par le Zn, et le lointain infrarouge. Les vibrations de complexes de cuivre ont été étudiées en moyen et lointain infrarouge. Les spectres des complexes Cu- poly-L- histidine ont été enregistrés en fonction du pH en moyen et lointain infrarouge. De même, les vibrations métal- ligand ont été observées dans le lointain infrarouge. La coordination du Cu par l'amyloïde- beta16 est une étape déterminante dans l'apparition de la maladie d'Alzheimer. Les complexes cuivre- amyloïde- beta16 ont été étudiés dans le moyen in
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45

Ozeir, Mohammad. "Etude de la voie du coenzyme Q¦ chez la levure Saccharomyces cerevisiae." Phd thesis, Université de Grenoble, 2012. http://tel.archives-ouvertes.fr/tel-00859892.

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Le coenzyme Q (ubiquinone ou Q) est une molécule organique lipophile composée d'une benzoquinone substituée et d'une chaîne polyisoprényle contenant 6 unités chez Saccharomyces cerevisiae (Q6), 8 chez Escherichia coli (Q8) et 10 chez l'homme (Q10). Q a un rôle bien connu de transporteur d'électrons dans les chaînes respiratoires et fonctionne également comme un antioxydant membranaire. La déficience primaire en Q10 a maintenant été attribuée à des mutations dans 6 gènes de la biosynthèse de Q10 et cause des pathologies sévères. La biosynthèse de Q6 est mitochondriale et nécessite au moins 9 pr
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46

劉永棠 and Wing-tong Ricky Lau. "Multiplex RT-PCR for typing and subtyping influenza and respiratory syncytial viruses." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31970667.

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47

Lau, Wing-tong Ricky. "Multiplex RT-PCR for typing and subtyping influenza and respiratory syncytial viruses." Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25151599.

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48

Brewer, Judy. "Metabolic Modeling of Inborn Errors of Metabolism: Carnitine Palmitoyltransferase II Deficiency and Respiratory Chain Complex I Deficiency." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:24078365.

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The research goal was to assess the current capabilities of a metabolic modeling environment to support exploration of inborn errors of metabolism (IEMs); and to assess whether, drawing on evidence from published studies of EMs, the current capabilities of this modeling environment correlate with clinical measures of energy production, fatty acid oxidation, accumulation of toxic by-products of defective metabolism, and mitigation via therapeutic agents. IEMs comprise several hundred disorders of energy production, often with significant impact on morbidity and mortality. Despite advances in g
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49

Bursby, Timothy Patrick. "Investigations of the mitochondrial #beta#-oxidation trifunctional protein and its association with complex 1 of the respiratory chain." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364807.

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50

Schimpf, Johannes [Verfasser], and Thorsten [Akademischer Betreuer] Friedrich. "Towards the structure of complex I and bo3 oxidase from Escherichia coli and exploring its aerobic respiratory chain." Freiburg : Universität, 2020. http://d-nb.info/1222436450/34.

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