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Books on the topic 'Respiratory Gas Analysis'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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1

A, Paulus David, Hayes Thomas J, and Gravenstein J. S, eds. Gas monitoring in clinical practice. 2nd ed. Butterworth-Heinemann, 1994.

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2

Shapiro, Barry A. Clinical application of blood gases. 5th ed. Mosby-Year Book, 1993.

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3

Shapiro, Barry A. Clinical application of blood gases. 5th ed. Mosby, 1994.

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4

Stacey, Victoria. Respiratory. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199592777.003.0010.

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Asthma - Chronic obstructive pulmonary disease (COPD) - Non-invasive ventilation - Venous thromboembolism - Pneumonia - Spontaneous pneumothorax - Respiratory failure and oxygen therapy - Arterial blood gas analysis - SAQs

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5

Garby, Lars. The Respiratory Functions of Blood. Springer, 2012.

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6

Joynt, Gavin M., and Gordon Y. S. Choi. Blood gas analysis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0072.

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Arterial blood gases allow the assessment of patient oxygenation, ventilation, and acid-base status. Blood gas machines directly measure pH, and the partial pressures of carbon dioxide (PaCO2) and oxygen (PaO2) dissolved in arterial blood. Oxygenation is assessed by measuring PaO2 and arterial blood oxygen saturation (SaO2) in the context of the inspired oxygen and haemoglobin concentration, and the oxyhaemoglobin dissociation curve. Causes of arterial hypoxaemia may often be elucidated by determining the alveolar–arterial oxygen gradient. Ventilation is assessed by measuring the PaCO2 in the

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7

Banerjee, Ashis, and Clara Oliver. Respiratory emergencies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198786870.003.0010.

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Difficulty in breathing is both a common presenting complaint and a major acute presentation in the emergency department (ED). This chapter covers the common causes of breathlessness. It focuses on the management and diagnosis of asthma and chronic obstructive pulmonary disease (COPD) in line with the British Thoracic Society guidelines, which may commonly appear as a short-answer question (SAQ). In addition, this chapter covers the pathophysiology of T2RF and its management, including the indications and contraindications for non-invasive ventilation. Another common topic examined in the SAQ

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8

Paul, Berghuis, ed. Respiration. SpaceLabs, Inc., 1992.

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9

Gattinon, Luciano, and Eleonora Carlesso. Acute respiratory failure and acute respiratory distress syndrome. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0064.

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Respiratory failure (RF) is defined as the acute or chronic impairment of respiratory system function to maintain normal oxygen and CO2 values when breathing room air. ‘Oxygenation failure’ occurs when O2 partial pressure (PaO2) value is lower than the normal predicted values for age and altitude and may be due to ventilation/perfusion mismatch or low oxygen concentration in the inspired air. In contrast, ‘ventilatory failure’ primarily involves CO2 elimination, with arterial CO2 partial pressure (PaCO2) higher than 45 mmHg. The most common causes are exacerbation of chronic obstructive pulmon

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10

Gattinon, Luciano, and Eleonora Carlesso. Acute respiratory failure and acute respiratory distress syndrome. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0064_update_001.

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Respiratory failure (RF) is defined as the acute or chronic impairment of respiratory system function to maintain normal oxygen and CO2 values when breathing room air. ‘Oxygenation failure’ occurs when O2 partial pressure (PaO2) value is lower than the normal predicted values for age and altitude and may be due to ventilation/perfusion mismatch or low oxygen concentration in the inspired air. In contrast, ‘ventilatory failure’ primarily involves CO2 elimination, with arterial CO2 partial pressure (PaCO2) higher than 45 mmHg. The most common causes are exacerbation of chronic obstructive pulmon

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11

Determination of the lactate threshold by respiratory gas exchange measures and blood lactate levels during incremental-load work. 1987.

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12

Determination of the lactate threshold by respiratory gas exchange measures and blood lactate levels during incremental-load work. 1989.

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13

Determination of the lactate threshold by respiratory gas exchange measures and blood lactate levels during incremental-load work. 1989.

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14

Lee, Jan Hau, and Ira M. Cheifetz. Respiratory Failure and Mechanical Ventilation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199918027.003.0006.

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This chapter on respiratory failure and mechanical ventilation provides essential information about how to support children with severe respiratory disorders. The authors discuss multiple modes of respiratory support, including high-flow nasal cannula oxygen, noninvasive ventilation with continuous positive airway pressure and bilevel positive airway pressure, as well as conventional, high-frequency, and alternative modes of invasive ventilation. The section on invasive mechanical ventilation includes key information regarding gas exchange goals, modes of ventilation, patient–ventilator intera

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15

Respiratory gas exchange, heart rate and blood lactate during running at submaximal workloads established by the lactate threshold and % VOb2s max in highly trained and less trained runners. 1987.

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16

Respiratory gas exchange, heart rate and blood lactate during running at submaximal workloads established by the lactate threshold and % VO₂ max in highly trained and less trained runners. 1989.

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17

Respiratory gas exchange, heart rate and blood lactate during running at submaximal workloads established by the lactate threshold and % VOb2s max in highly trained and less trained runners. 1989.

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18

Magee, Patrick, and Mark Tooley. Intraoperative monitoring. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0043.

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Chapter 25 introduced some basic generic principles applicable to many measurement and monitoring techniques. Chapter 43 introduces those principles not covered in Chapter 25 and discusses in detail the clinical applications and limitations of the many monitoring techniques available to the modern clinical anaesthetist. It starts with non-invasive blood pressure measurement, including clinical and automated techniques. This is followed by techniques of direct blood pressure measurement, noting that transducers and calibration have been discussed in Chapter 25. This is followed by electrocardio

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19

Making Sense of Lung Function Tests: A Hands-On Guide (Arnold Publication). A Hodder Arnold Publication, 2003.

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20

Frew, Anthony. Air pollution. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0341.

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Any public debate about air pollution starts with the premise that air pollution cannot be good for you, so we should have less of it. However, it is much more difficult to determine how much is dangerous, and even more difficult to decide how much we are willing to pay for improvements in measured air pollution. Recent UK estimates suggest that fine particulate pollution causes about 6500 deaths per year, although it is not clear how many years of life are lost as a result. Some deaths may just be brought forward by a few days or weeks, while others may be truly premature. Globally, household

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