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Relevant bibliographies by topics / Respiratory Tract Diseases

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Academic literature on the topic 'Respiratory Tract Diseases'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 June 2025

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Journal articles on the topic "Respiratory Tract Diseases"

1

Isobe, Takeshi. "Guidelines for Respiratory Tract Diseases." Nihon Naika Gakkai Zasshi 98, no. 8 (2009): 2014–22. http://dx.doi.org/10.2169/naika.98.2014.

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ISHIOKA, SHIN'ICHI. "Respiratory tract diseases and cytokine." Nihon Naika Gakkai Zasshi 84, no. 2 (1995): 301–6. http://dx.doi.org/10.2169/naika.84.301.

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Barson, A. J. "Respiratory and Alimentary Tract Diseases." Archives of Disease in Childhood 62, no. 12 (1987): 1295. http://dx.doi.org/10.1136/adc.62.12.1295.

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Meuer, Stefan. "Probiotics and Respiratory Tract Diseases." Annals of Nutrition and Metabolism 57, no. 1 (2010): 24–26. http://dx.doi.org/10.1159/000317350.

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Rees, H. C. "Respiratory and Alimentary Tract Diseases." Journal of Clinical Pathology 41, no. 1 (1988): 119. http://dx.doi.org/10.1136/jcp.41.1.119-c.

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Trifilieff, A., A. Da Silva, and JP Gies. "Kinins and respiratory tract diseases." European Respiratory Journal 6, no. 4 (1993): 576–87. http://dx.doi.org/10.1183/09031936.93.06040576.

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Bradykinin and related kinins are peptidic hormones, formed in tissues and fluids during inflammation. Various functional sites have been proposed as mediators of the biological effects of kinins, including the B1, B2 and B3 receptors. The existence of the B1 and the B2 receptor has largely been confirmed, whilst that of the B3 receptor is controversial and needs further confirmation. The role of bradykinin in the pathophysiology of asthma is not well understood, but bradykinin was proposed as a putative mediator of asthma, since asthmatic subjects are hyperresponsive to bradykinin, and since immunoreactive kinins are increased in the bronchoalveolar lavage fluids of asthmatic patients. Kinins could provoke bronchoconstriction by acting directly on smooth muscle and/or indirectly by their inflammatory properties. They may also contribute to the symptomatology of allergic and viral rhinitis, since they are the only mediators detected to date that are generated in nasal secretion during experimental and natural rhinovirus colds. Moreover, they can induce relevant symptoms when applied to airway mucosa. It has also been proposed that coughing during treatment with angiotensin-converting enzyme (ACE) inhibitors is linked to the action of kinins, since ACE is able to degrade kinins, and since the effects of ACE inhibitors are reduced by kinin antagonists. Due to their mitogenic properties, kinins have been proposed to regulate lung carcinoma growth. Their action remains speculative, but some findings are of great interest in order to define their role in these pathologies. Despite many studies in animals and in humans, the mode of action of kinins in airways is still poorly understood.(ABSTRACT TRUNCATED AT 250 WORDS)

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Neelam, Meena Surendra Singh Shekhawat Subhash Chand Meena Vipin Chand Bairwa and Anupriya. "Caprine Respiratory Diseases." Science World a monthly e magazine 3, no. 8 (2023): 1986–90. https://doi.org/10.5281/zenodo.8248219.

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Small ruminants are valuable assets for the Mediterranean, African, and Southeast Asian countries with the potential for providing meat, milk, and wool. These animals are highly susceptible to respiratory diseases, which account for almost 50% mortality amongst them. Irrespective of the etiology, the infectious respiratory diseases of sheep and goats contribute to 5.6 percent of the total diseases of small ruminants. Respiratory diseases can affect goats of all ages.  The infectious respiratory disorders are classified into two groups: the diseases of upper respiratory tract including sinusitis caused by the larvae of parasites, nasal foreign bodies, gaseous irritation & enzootic nasal tumors and the diseases of lower respiratory tract comprising mainly pneumonia, often these are of infectious origin (bacterial, viral, or fungal). In kids, respiratory diseases are usually from infectious agents. Respiratory problems due to trachea injury can arise from improper use of balling and drenching guns.

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Zakharova, I. N., I. V. Berezhnaya, L. Ya Klimov, A. N. Kasyanova, O. V. Dedikova, and K. A. Koltsov. "Probiotics in the management of respiratory diseases: ways of interaction and therapeutic perspectives." Medical Council, no. 2 (February 16, 2019): 173–82. http://dx.doi.org/10.21518/2079-701x-2019-2-173-182.

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Today, the composition of the gut microbiota has been studied in sufficient detail. Increasing number of studies show that the respiratory tract, both the upper and lower respiratory tract, have their own microbiota. The article presents the main today’s data about the species diversity of microorganisms in the respiratory and gastrointestinal tracts, describes the role of a healthy microbiota in providing local and general immunity. The authors specify the role of probiotic strains of microorganisms and their effect on various parts of the immune response and present the data of studies on the effect of probiotic products on the immunological resistance of humans, especially the respiratory tract with high viral load. Restoration of a healthy microbiota in the human tract using probiotic products administered through the gastrointestinal tract can reduce the risk and severity of manifestation of the respiratory infections.

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ITOH, Takashi. "Kampo Treatment for Respiratory Tract Diseases." Kampo Medicine 54, no. 1 (2003): 29–46. http://dx.doi.org/10.3937/kampomed.54.29.

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Chintu, Chifumbe, and Peter Mwaba. "Infectious diseases of the respiratory tract." Journal of Infection 38, no. 2 (1999): 137. http://dx.doi.org/10.1016/s0163-4453(99)90091-9.

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Dissertations / Theses on the topic "Respiratory Tract Diseases"

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Kharitonov, Sergei Alexandrovich. "Exhaled nitric oxide in airway diseases." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266411.

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Petersson, Christer. "Preschool children day-care, diseases and drugs : studies of risk factors for respiratory tract infections /." Lund : Dept. of Community Health Sciences, Lund University, 1994. http://books.google.com/books?id=Vs9sAAAAMAAJ.

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Almond, Elizabeth Jennifer Philippa. "Epstein-Barr virus infection of the lower respiratory tract." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1989. http://hub.hku.hk/bib/B31208484.

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Wrightson, John M. "Pathogen identification in lower respiratory tract infection." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:30c757ec-99b7-492e-a12e-ff996581863a.

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Treatment of lower respiratory tract infection (pneumonia and pleural infection) relies on the use of empirical broad spectrum antibiotics, primarily because reliable pathogen identification occurs infrequently. Another consequence of poor rates of pathogen identification is that our understanding of the microbiology of these infections is incomplete. This thesis addresses some of these issues by combining the acquisition of high quality lower respiratory tract samples, free from nasooropharyngeal contamination, with novel molecular microbiological techniques in an attempt to increase rates of pathogen identification. Four main areas are examined: (i) The role of so-called ‘atypical pneumonia’ bacteria in causing pleural infection. These pathogens have been previously identified in the pleural space infrequently and routine culture usually fails to isolate such bacteria. High sensitivity nested polymerase chain reaction (PCR) is a culture-independent technique which is used to undertake a systematic evaluation for these pathogens in pleural infection samples. (ii) The role of Pneumocystis jirovecii in pleural infection, either as a co-infecting pathogen or in monomicrobial infection. This fungus causes severe pneumonia, particularly in the immunosuppressed, but is increasingly recognised as a co-pathogen in community-acquired pneumonia, and is frequently isolated in the upper and lower respiratory tract in health. A high sensitivity real-time PCR assay is used to examine for this fungus. (iii) Ultra-deep sequencing of the 16S rRNA gene is used to perform a comprehensive microbial survey in samples taken from the multi-centre MIST2 study of pleural infection. The techniques employed allow analysis of polymicrobial samples and give very high taxonomic resolution, whilst incorporating methods to control for potential contamination. Further, these techniques provide confirmation of the results from the ‘atypical’ bacteria nested PCR study. (iv) Bedside ultrasound-guided percutaneous transthoracic needle aspiration (TNA) of consolidated lung is undertaken in patients with pneumonia, as part of the PIPAP study. An evaluation is undertaken of the efficacy and acceptability of TNA. Aspirate samples acquired are also processed using ultra-deep sequencing of the 16S rRNA gene. Other samples obtained as part of the PIPAP study, such as ‘control’ lung aspirates and ‘control’ pleural fluid samples, are similarly processed to enable calculation of sensitivity and specificity of the sequencing methodology.

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Wong, Chun-nin Adam, and 黃春年. "Analyses of influenza viral cytopathic effect in human lower respiratory tract." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41290860.

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Enright, Mark Charles. "Molecular characterization of Moraxella catarrhalis." Thesis, University of Aberdeen, 1994. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=158242.

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<I>Moraxella catarrhalis</I> is a gram-negative diplococcus which until recently was thought to be a harmless commensal. Increasing awareness has established the pathogenic nature of this organism and it is now recognised as a major cause of otitis media in children, exacerbations of chronic bronchitis in elderly patients and an occasional cause of invasive disease. <I>M. catarrhalis</I> is spread nosocomially especially in respiratory wards containing elderly patients. This study evaluated four methods for typing nosocomially spread isolates:- immunoblotting with normal human serum (NHS), and three DNA fingerprinting methods. The most discriminatory method found was restriction endonuclease analysis (REA) using <I>Taq</I> I, although immunoblotting with NHS and pulsed-field gel electrophoresis (PFGE) using <I>Sma</I> I sub-divided isolates grouped together by the other methods. PFGE using <I>Not</I> I only confirmed groupings made by other methods. A study of <I>M. catarrhalis</I> and phenotypically similar organisms was performed using comparisons of partial 16S rDNA sequence. 16S rDNA of <I>M. catarrhalis</I> strains from disparate geographical locations was found to be extremely conserved <I>M. catarrhalis</I> 16S rDNA was very similar to that of other <I>Moraxella</I> species whilst <I>Moraxella</I> species were found to be generally distinct from the <I>Neisseria</I> and <I>Kingella</I> species studied. These results confirm <I>M. catarrhalis</I> as a genuine member of the <I>Moraxellae</I>.

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Carstens, Ann. "Radiological tracheal dimensions of the normal Thoroughbred horse." Diss., University of Pretoria, 2008. http://hdl.handle.net/2263/30220.

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Respiratory conditions causing poor performance in horses are usually as result of upper respiratory tract diseases or are of pulmonary origin. The tracheal is rarely a cause of primary respiratory problems in the horse, but tracheal dimensions, particularly height, may be useful in evaluating upper repiratory tract conditions cranial to the trachea and lung pathology, due to resultant change in differential pressures between these areas. The normal radiological equine tracheal height along its length has as yet not been reported. Standing lateral radiographs of the cervical and thoracic trachea of 15 clinically normal sedated Thoroughbred horses, 3-6 years old, were made at peak inspiration and end expiration. Maximum height of the larynx, and trachea at the level of the third and fifth cervical vertebra, at the level of the first thoracic vertebra, carina and the left and right primary bronchi were measured. Ratios of laryngeal height relative to the third cervical vertebral body length and tracheal heights relative to the vertebral body lengths of adjacent third and fifth cervical vertebrae and first thoracic vertebra, and carina heights relative to a mid-thoracic vertebra, respectively were made, as well as tracheal height at the fist thoracic vertebra ratio with the thoracic inlet height. Known size metallic markers were used to determine magnification corrected tracheal heights in the sagittal plane and effect of body mass and height at the withers on tracheal height was determined. The magnification corrected radiological airway heights at end expiration and peak inspiration were measured and respectively the mean values were found to be: laryngeal height: 5.89 cm and 5.86 cm, tracheal height at the third cervical vertebra: 4.17 cm and 4.04 cm, tracheal height at the fifth cervical vertebra: 3.62 cm and 3.59 cm, tracheal height at the first thoracic vertebra: 3.4 cm and 3.23 cm and carina height: 3.85 cm and 4.12 cm. The ratios of these measurements to nearby vertebral body lengths were respectively: laryngeal height at the third cervical vertebra: 0.56 and 0.56, tracheal height at the third cervical vertebra: 0.4 and 0.39, tracheal height at the fifth cervical vertebra: 0.37 and 0.37, tracheal height at the first thoracic vertebra: 0.59 and 0.59, and carina height: 0.91 and 0.94. The ratio tracheal height at the first thoracic vertebra to the thoracic inlet respectively 0.15 and 0.15. Although there was no statistical difference in the data, there was a trend towards a higher tracheal height at expiration. No correlation was found between tracheal height and body mass or tracheal height and height at the withers, and measured tracheal height was generally lower than predicted tracheal height, possibly as result of sedation used. The small range of body mass and height in this study as well as the relatively small number of horses evaluated may account for the lack of correlation to predicted tracheal height. This study in normal horses may serve as a reference when radiologically evaluating cases of upper respiratory tract and lung pathology, where the tracheal dimensions may differ significantly due to differences in airway resistance and biomechanics. Radiographs to evaluate tracheal height can be made independent of respiratory phase in sedated horses, and it is recommended that ratios of tracheal height to an adjacent vertebral body length are more reliable values to compare within and between horses. It is recommended to take tracheal height measured at the fifth cervical vertebra since this measurement showed a slightly smaller standard deviation than at other sites measured as well as a medium amount of clinical effect. If only thoracic radiographs are made, measurements of tracheal height at the thoracic inlet is the alternative (the standard cranioventral view), but it is recommended to include the distal aspect of the first rib if the thoracic inlet is to be measured.<br>Dissertation (MMedVet)--University of Pretoria, 2008.<br>Companion Animal Clinical Studies<br>unrestricted

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Bello, Ortí Bernardo. "Haemophilus parasuis host-pathogen interactions in the respiratory tract." Doctoral thesis, Universitat Autònoma de Barcelona, 2015. http://hdl.handle.net/10803/312855.

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En el sector veterinario, la enfermedad de Glässer es un proceso patogénico frecuente que conduce a pérdidas económicas considerables. Esta enfermedad es causada por Haemophilus parasuis. Aunque se ha llevado a cabo un esfuerzo importante hacia la comprensión de los factores que intervienen en la evolución de la enfermedad, la falta de completa protección de las vacunas comerciales sugieren que debe dirigirse más trabajo hacia el estudio de este proceso patogénico. Para aumentar el conocimiento en patogénesis desarrollamos una serie de experimentos. Es bien sabido que existen diferentes cepas de H. parasuis, desde no virulentas a altamente virulentas. Ciertos mecanismos patogénicos se atribuyen a la virulencia de algunas cepas, mientras que las cepas no virulentas solamente colonizan el tracto respiratorio superior y no son capaces de causar enfermedad. Estos diferentes grados de virulencia podrían ser apreciados durante los primeros pasos de la infección. De este modo, usando muestras de las vías respiratorias de lechones infectados con dos cepas virulentas (Nagasaki y IT29755) y dos cepas no virulentas (SW114 y F9), se desarrollaron métodos de inmunohistoquímica e inmunofluorescencia, así como una doble tinción de H. parasuis y macrófagos/neutrófilos. Nuestros resultados revelaron que las cepas virulentas de H. parasuis estaban presentes en cornete nasal, tráquea y pulmón. Detalles adicionales mostraron que las cepas virulentas de H. parasuis no solo se asociaron a macrófagos y neutrófilos del pulmón, sino también a células tipo neumocitos. Por lo tanto, las cepas virulentas de H. parasuis fueron capaces de adherirse al epitelio de las vías respiratorias, invadir y diseminarse en el huésped. Por el contrario, las cepas no virulentas apenas se detectan en el tracto respiratorio. La cepa virulenta Nagasaki mostró patrones de biofilm en tráquea, que nos hizo cuestionar el papel de la formación de biofilm en la infección. Dado que la literatura publicada anteriormente indicaba que la formación de biofilm se presentaba principalmente en cepas no virulentas, se realizó una investigación adicional en esta dirección para comparar la formación de biofilm en cepas virulentas y no virulentas de H. parasuis. Nuestros resultados confirmaron que la capacidad de formar biofilm in vitro se presenta principalmente en cepas no virulentas. Por tanto, se secuenció el transcriptoma de la cepa no virulenta F9 durante su crecimiento en biofilm utilizando un modelo in vitro. Los resultados sugieren que bajo condiciones de biofilm, H. parasuis muestra un metabolismo reducido, demostrado por el perfil de expresión génica. Además, algunos de los genes inducidos en condiciones de biofilm eran específicos de las cepas no virulentas, como la hemaglutinina filamentosa fhaB, previamente asociada a la formación de biofilm en otras bacterias. Finalmente, la observación de cepas virulentas de H. parasuis en pulmón durante la infección motivó la secuenciación del transcriptoma de una cepa patógena en esta ubicación. Se determinó la expresión génica después de una infección corta in vivo y tras la inoculación de pulmón ex vivo. Los resultados mostraron tendencias comunes en la expresión génica de H. parasuis bajo infección pulmonar in vivo y ex vivo, como la reducción del metabolismo y la expresión de genes implicados en la adquisición de nutrientes, lo que podría indicar una estrategia de supervivencia en estas condiciones. Durante la infección pulmonar también se detectaron genes únicos de cepas virulentas de H. parasuis que codifican para proteínas de membrana externa. Estos genes requerirán una mayor caracterización como factores de virulencia, pudiendo ser también útiles para desarrollar nuevos antibióticos y vacunas. Nuestros resultados también apoyan el uso de explantes de pulmón como modelo para estudios de patogenicidad de otras bacterias respiratorias.<br>In the veterinary field, Glässer’s disease is a common pathogenic process that leads to considerable economic losses. This disease is caused by Haemophilus parasuis. Although considerable effort has been focused towards understanding the factors involved in disease outcome, evidences of lack of complete protection of commercial vaccine formulations suggest that more work should be addressed towards understanding this pathogenic process. To fill this gap in pathogenesis knowledge we developed a series of experiments. It is well known that different H. parasuis strains exist, ranging from non-virulent to highly virulent. Particular pathogenic mechanisms are attributed to virulence strains, while non-virulent strains only colonize the upper respiratory tract and are unable to cause disease. It is expected that these different virulence degrees can be appreciated also during the early steps of infection. Using samples from the respiratory tract of piglets infected with two virulent strains (Nagasaki and IT29755) and two non-virulent strains (SW114 and F9), immunohistochemistry and immunofluorescence methods were developed, as well as a double staining targeting H. parasuis and macrophage/neutrophil cells. Our results revealed that H. parasuis virulent strains were present in nasal, trachea and lung locations. Additional details showed that virulent H. parasuis was associated to macrophages and neutrophils in lung, but also to pneumocyte-like cells. Thus, virulent H. parasuis was able to attach to respiratory tract epithelia, invade and disseminate into the host. On the contrary, non-virulent strains were barely detected in the respiratory tract. Biofilm-like patterns were displayed by virulent Nagasaki strain in trachea and this made us question the role of biofilm formation in infection. Since previously published reports indicated that biofilm formation was mainly present in non-virulent strains, we performed additional research in this direction to compare biofilm formation with virulent and non-virulent H. parasuis strains. Our results confirmed that the capacity to form biofilm in vitro was mainly presented by non-virulent strains. Thus, we sequenced the transcriptome of non-virulent F9 strain under biofilm growth using an in vitro model. Results suggested that under biofilm conditions H. parasuis showed a low metabolic state, as indicated by the gene expression profile. Some of the genes induced under biofilm conditions were specific of non-virulent strains, as the filamentous hemagglutinin fhaB, which has been associated to biofilm formation in other bacteria. Additionally, the observation of virulent H. parasuis strains in the lung during infection inspired us to sequence the transcriptome of a pathogenic strain in this location. Gene expression was determined after a short in vivo infection and after ex vivo lung inoculation. Results showed common trends in H. parasuis gene expression under in vivo and ex vivo lung infection, such as reduced metabolism and higher expression of genes involved in nutrient acquisition, which could indicate a survival strategy under these conditions. Genes unique of virulent H. parasuis strains coding for outer membrane proteins were also detected during lung infection. These genes would require further characterization as virulent factors and could be also useful to develop new antimicrobials and vaccines. Our results also support the use of lung explants as models for pathogenicity studies of respiratory bacteria.

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Nathell, Lennart. "Some determinants of sick leave for respiratory disease : occupation, asthma, obesity, smoking, and rehabilitation /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-301-5/.

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Paudyal, Priyamvada. "Respiratory symptoms and lung function in relation to cotton dust and endotoxin exposure in textile workers in Nepal." Thesis, University of Aberdeen, 2011. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=166944.

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Background: Cotton workers are highly exposed to organic dust. Inhalation of cotton based particulate has been associated with various respiratory symptoms and impaired lung function. This study investigates the respiratory health profile of textile mill workers in Nepal in relation to dust and endotoxin exposure. Methods: This study was conducted in four sectors (garment, carpet, weaving and recycling) of the textile industry in Kathmandu, Nepal. A total of 938 individuals completed a health questionnaire and performed spirometry. A subset of 384 workers performed cross-shift spirometry. Personal exposure to inhalable dust and airborne endotoxin was measured during a full-shift for a 114 workers. Results: Geometric mean concentrations of personal exposure to cotton dust and endotoxin were 0.81 mg/m3 and 2160 EU/m3 respectively. Overall prevalence of persistent cough, persistent phlegm, wheeze, breathlessness and chest tightness were 8.5%, 12.5%, 3.2%, 6.5%and 3.6% respectively. Symptoms were most common among the recyclers and less in the garment sector. Exposure to inhalable dust significantly predicted the symptoms of persistent cough and chest tightness. Significant cross-shift reduction in FEV1, FVC, and FEF25_75 were measured in the textile workers (p<0.001 for all); reductions being greater in the recyclers (-143 ml) and smallest in the garment workers (-38 ml) (p=0.012). Cross-shift reduction in FEV1 was significantly predicated by exposure to inhalable dust. Exposure to endotoxin did not correlate with any of the respiratory symptoms nor to lung function. Conclusion: The measured association between exposure to inhalable dust and reporting of respiratory symptoms and lung function suggests that despite high levels of endotoxin exposures, inhalable dust is the driver for these effects and attention should turn to what might be the toxic component in this dust other than endotoxin.

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Books on the topic "Respiratory Tract Diseases"

1

Jariwalla, G. Respiratory diseases. MTP Press, 1985.

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A, Merchant James, National Institute for Occupational Safety and Health. Division of Respiratory Disease Studies., National Institute for Occupational Safety and Health., and United States. Dept. of Health and Human Services., eds. Occupational respiratory diseases. U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, National Institute for Occupational Safety and Health, 1986.

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Tattersfield, Anne E. Respiratory disease. Springer-Verlag, 1987.

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Rush, Bonnie. Equine respiratory diseases. Blackwell Science, 2004.

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Westra, Bonnie. Respiratory problems. Springhouse Corp., 1987.

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B, Athavale V. Diseases of respiratory tract: Ayurvedic concept. Chaukhamba Sanskrit Pratishthan, 2001.

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Farzan, Sattar. A concise handbook of respiratory diseases. 3rd ed. Appleton & Lange, 1992.

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1927-, Murray John F., and Nadel J. A. 1929-, eds. Textbook of respiratory medicine. 2nd ed. Saunders, 1994.

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1927-, Murray John F., and Nadel Jay A. 1929-, eds. Textbook of respiratory medicine. Saunders, 1988.

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Johnson, Norman McI. Respiratory medicine. 2nd ed. Blackwell Scientific, 1990.

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Book chapters on the topic "Respiratory Tract Diseases"

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Brooks, D., and E. M. Dunbar. "Respiratory Tract Infections." In Infectious Diseases. Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4133-5_4.

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Venner, Christopher P., Jennifer H. Pinney, and Helen J. Lachmann. "Amyloidosis and the Respiratory Tract." In Orphan Lung Diseases. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-2401-6_7.

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Arslan, Nevra Güllü, Füsun Öner Eyüboğlu, and Raquel Duarte. "Fungal Infections of the Lower Respiratory Tract." In Airway diseases. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-22483-6_41-1.

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Berrino, Franco. "Occupational Factors of Upper Respiratory Tract Cancers." In Prevention of Respiratory Diseases. CRC Press, 2024. http://dx.doi.org/10.1201/9781003573869-5.

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Mittal, Piyush, Manjari Mittal, Ujjawal Rawat, and Ambika. "Microbiome in Upper Respiratory Tract Infections." In Microbiome in Inflammatory Lung Diseases. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-8957-4_17.

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Mehta, Renuka, Suriyanarayana P. Hariprakash, Peter N. Cox, and Derek S. Wheeler. "Diseases of the Upper Respiratory Tract." In The Respiratory Tract in Pediatric Critical Illness and Injury. Springer London, 2008. http://dx.doi.org/10.1007/978-1-84800-925-7_13.

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Primhak, Sarah, Evangelia Myttaraki, and Elizabeth Whittaker. "Congenital infections of the respiratory tract." In Respiratory Diseases of the Newborn Infant. European Respiratory Society, 2021. http://dx.doi.org/10.1183/2312508x.10014720.

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"Respiratory Tract." In Pathology of Non-Helminth Infectious Diseases. American Registry of PathologyArlington, Virginia, 2024. http://dx.doi.org/10.55418/9781933477435-12.

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"Respiratory Tract." In Pathology of Non-Helminth Infectious Diseases. American Registry of PathologyArlington, Virginia, 2024. http://dx.doi.org/10.55418/9781933477435-04.

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"Respiratory Tract." In Pathology of Non-Helminth Infectious Diseases. American Registry of PathologyArlington, Virginia, 2024. http://dx.doi.org/10.55418/9781933477435-21.

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Conference papers on the topic "Respiratory Tract Diseases"

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Marangoni, Igor Parada. "Hospital morbidity and mortality profile due to pneumonia in Brazil between 2010 and 2020." In II INTERNATIONAL SEVEN MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/homeinternationalanais-080.

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Abstract Pneumonia is a lower respiratory tract infection that affected 489 million people worldwide in the year 2019 according to data from the Global Burden of Diseases (GBD) 2019 study,1,2. The disease was still responsible for over 2.49 million deaths in the year of the survey, surpassing other diseases such as tuberculosis and HIV, making pneumonia the leading infectious cause of death worldwide

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2

Sheenkova, M. V. "ON THE PATHOGENESIS OF GASTRODUODENAL ZONE LESIONS IN VARIOUS VARIANTS OF DRUG THERAPY FOR OCCUPATIONAL RESPIRATORY DISEASES." In The 16th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2021). FSBSI “IRIOH”, 2021. http://dx.doi.org/10.31089/978-5-6042929-2-1-2021-1-575-578.

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Abstract. A survey of workers of large industrial enterprises with occupational diseases of the respiratory system was conducted to assess the dependence of the risk of damage to the upper gastrointestinal tract on the features of pharmacological therapy of respiratory pathology. The specificity of the pathogenesis of gastric and duodenal lesions associated with the properties of drug treatment of occupational respiratory diseases was revealed.

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Dragun, K. V., and E. P. Zhivitskaya. "EPIDEMIOLOGICAL ANALYSIS OF THE MORBIDITY OF THE POPULATION OF KRUPKI DISTRICT WITH RESPIRATORY DISEASES." In SAKHAROV READINGS 2022: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2022. http://dx.doi.org/10.46646/sakh-2022-2-61-64.

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Respiratory diseases are one of the most common pathologies in the structure of general and primary morbidity. The analysis of the primary morbidity of respiratory diseases of the adult population of Krupki district for the period 2014-2020 was carried out and the trends of morbidity were determined. In the structure of the primary morbidity of the adult population, respiratory diseases occupy the first rank place. There is a decrease in the primary incidence of respiratory diseases of the adult population of Krupki district. Acute respiratory infections of the respiratory tract, pneumonia, chronic rhinitis, nasopharyngitis, pharyngitis, sinusitis, asthma and asthmatic status, vasomotor and allergic rhinitis made the main contribution to the structure of the primary incidence of respiratory diseases by nosological forms of the adult population in 2020. Acute respiratory viral infections, vasomotor and allergic rhinitis are characterized by a decrease in primary morbidity, and for pneumonia, chronic rhinitis, nasopharyngitis, pharyngitis and sinusitis - an increase in indicators.

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4

Leon-Abarca, J. A., and R. A. Accinelli. "Hypoxia Predicts Lower Respiratory Tract Diseases in Adults Living at High Altitudes." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4921.

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5

Mofakham, Amir A., Brian T. Helenbrook, Tanvir Ahmed, et al. "Significance of Vocal Tract Geometrical Variations and Loudness on Airflow and Droplet Dispersion in a Two-Dimensional Representation of [F]." In ASME 2021 Fluids Engineering Division Summer Meeting. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/fedsm2021-65485.

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Abstract The significance of respiratory droplet transmission in spreading respiratory diseases such as COVID-19 has been identified by researchers. Although one cough or sneeze generates a large number of respiratory droplets, they are usually infrequent. In comparison, speaking and singing generate fewer droplets, but occur much more often, highlighting their potential as a vector for airborne transmission. However, the flow dynamics of speech and the transmission of speech droplets have not been fully investigated. To shed light on this topic, two-dimensional geometries of a vocal tract for a labiodental fricative [f] were generated based on real-time MRI of a subject during pronouncing [f]. In these models, two different curvatures were considered for the tip tongue shape and the lower lip to highlight the effects of the articulator geometries on transmission dynamics. The commercial ANSYS-Fluent CFD software was used to solve the complex expiratory speech airflow trajectories. Simultaneously, the discrete phase model of the software was used to track submicron and large size respiratory droplets exhaled during [f] utterance. The simulations were performed for high, normal, and low lung pressures to explore the influence of loud, normal, and soft utterances, respectively, on the airflow dynamics. The presented results demonstrate the variability of the airflow and droplet propagation as a function of the vocal tract geometrical characteristics and loudness.

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6

Mdivnishvili, Nino, Ketevan Barabadze, and Nino Adamia. "INF-y and Neopterin as a diagnostic markers in recurrent respiratory tract diseases in children." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa1317.

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7

Akyuzluer Gunes, Setenay, Gizem Ozcan, Fazilcan Zirek, and Nazan Cobanoglu. "Evaluation of smoking habits of parents of patients with chronic lung and respiratory tract diseases." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1378.

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8

Wu, Xiaofang, Remy Mimms, Maureen Banigan, et al. "3D Glandular In Vitro Models To Investigate Mechanisms Of Glandular Hyperplasia In Respiratory Tract Diseases." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6320.

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Карпин, Владимир Александрович, and Ольга Ивановна Шувалова. "THE PROBLEM OF CHRONICLING THE INFLAMMATORY PROCESS." In Научные исследования в современном мире. Теория и практика: сборник статей XXVI всероссийской (национальной) научной конференции (Санкт-Петербург, Май 2024). Crossref, 2024. http://dx.doi.org/10.37539/240503.2024.55.68.002.

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В статье обсуждается проблема хронизации острых патологических процессов. Показаны существенные различия острых и хронических заболеваний на примере воспалительных процессов дыхательных путей. Постулируется необходимость существенного пересмотра проблемы хронизации острого воспалительного процесса. The article discusses the problem of chronicling acute pathological processes. Significant differences between acute and chronic diseases are shown by the example of inflammatory processes of the respiratory tract. The necessity of a significant revision of the problem of chronicling the acute inflammatory process is postulated.

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Kovalenko, N. I., O. O. Vovk, and I. V. Novikova. "Sensitivity to cephalosporins of opportunistic bacteria, excreted in patients with infectious diseases of upper and lower respiratory tract." In SCIENTIFIC PROGRESS OF MEDICINE AND PHARMACY OF THE EU COUNTRIES. Baltija Publishing, 2021. http://dx.doi.org/10.30525/978-9934-26-075-9-15.

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Reports on the topic "Respiratory Tract Diseases"

1

Wang, Xiaoyu. Pediatric tuina in treating recurrent respiratory tract infection in children: a systematic review and meta‑analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.4.0075.

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Review question / Objective: Is pediatric tuina an effective treatment for recurrent respiratory tract infection in children? Condition being studied: Recurrent respiratory tract infection (RRTI) is a common disease in children, which refers to the recurrence of upper and lower respiratory tract infections within a year, exceeding the prescribed number of times. It is more common in infants under 3 years old. The disease is easy to relapse and lasts for a long time, affecting the normal growth and development of children and physical and mental health, easily causing other diseases, leading to a variety of chronic wasting diseases, and damaging the function of organs and the immune system. Immunotherapy and nutritional therapy are commonly used in Western medicine. At present, the treatment of RRTI in children with traditional Chinese medicine has achieved a certain effect, and the treatment mainly includes internal treatment and external treatment. Tuina therapy is one of the common therapies for the treatment of RRTI in children with traditional Chinese medicine. Because of its advantages, there are many literature reports on tuina treatment of this disease, with a good total effective rate, but whether its therapeutic effect is higher than other therapies has not been determined as a whole. This study used the method of systematic review to collect the published clinical research literature on the treatment of RRTI in children at home and abroad for systematic review, so as to provide a reference for clinical research.

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Lin, Hongwei, Yanjun Gao, Kang Sun, and Faguang Jin. Association between PM2.5 pollution and outpatient visits for respiratory diseases in China: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0144.

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Review question / Objective: Previous epidemiological studies on the association between PM2.5 pollution and outpatient visits for respiratory diseases in China were mostly limited to one region， and the different papers have no coherent results. Our objective is to perform a systematic review and meta-analysis of the relevant literature in order to summarize the association between PM2.5 pollution and outpatient visits for respiratory diseases in multiple cities in China. Condition being studied: As an important component of air pollutants, particulate matter 2.5 (PM2.5) can float in the atmosphere for a long time with a small aerodynamic size (≤2.5μm) and large specific surface area which is attached to a variety of toxic and harmful substances . PM2.5 can deposite under the trachea of the respiratory tract, reaching deep into the alveolar area, damaging alveolar macrophages and type Ⅱ alveolar epithelial cells, inducing alveolar inflammation, resulting in decreased immunity of the respiratory tract and interfering with normal physiological functions of the lungs.

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3

Mao, Hui, YueHui Wei, Huimin Su, and Xun Li. Pediatric Tui Na for cough in children: A protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.2.0076.

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Review question / Objective: The aim of this systematic review is to evaluate the effectiveness and safety of pediatric Tui Na in the treatment of cough in children under seven years of age. Condition being studied: Cough is essentially a protective reflex of respiratory tract to various stimuli, typically in order to clear the lung airways of fluids, mucus, or other material. Cough not only has a negative impact on children’s daily activities and sleep, but is associated with parental stress and worries. Pediatric Tui Na, a therapeutic massage based on the Chinese traditional theory of Yin and Yang, Qi and blood, acupoints and meridians, enjoys a long history and has been widely applied to the treatment of common diseases like fever, diarrhea, cough and asthma. This study aims to evaluate the effectiveness and safety of pediatric Tui Na in the treatment of cough in children.

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4

Rahai, Hamid, and Jeremy Bonifacio. Numerical Investigations of Virus Transport Aboard a Commuter Bus. Mineta Transportation Institute, 2021. http://dx.doi.org/10.31979/mti.2021.2048.

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The authors performed unsteady numerical simulations of virus/particle transport released from a hypothetical passenger aboard a commuter bus. The bus model was sized according to a typical city bus used to transport passengers within the city of Long Beach in California. The simulations were performed for the bus in transit and when the bus was at a bus stop opening the middle doors for 30 seconds for passenger boarding and drop off. The infected passenger was sitting in an aisle seat in the middle of the bus, releasing 1267 particles (viruses)/min. The bus ventilation system released air from two linear slots in the ceiling at 2097 cubic feet per minute (CFM) and the air was exhausted at the back of the bus. Results indicated high exposure for passengers sitting behind the infectious during the bus transit. With air exchange outside during the bus stop, particles were spread to seats in front of the infectious passenger, thus increasing the risk of infection for the passengers sitting in front of the infectious person. With higher exposure time, the risk of infection is increased. One of the most important factors in assessing infection risk of respiratory diseases is the spatial distribution of the airborne pathogens. The deposition of the particles/viruses within the human respiratory system depends on the size, shape, and weight of the virus, the morphology of the respiratory tract, as well as the subject’s breathing pattern. For the current investigation, the viruses are modeled as solid particles of fixed size. While the results provide details of particles transport within a bus along with the probable risk of infection for a short duration, however, these results should be taken as preliminary as there are other significant factors such as the virus’s survival rate, the size distribution of the virus, and the space ventilation rate and mixing that contribute to the risk of infection and have not been taken into account in this investigation.

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Bacharach, Eran, and Sagar Goyal. Generation of Avian Pneumovirus Modified Clones for the Development of Attenuated Vaccines. United States Department of Agriculture, 2008. http://dx.doi.org/10.32747/2008.7696541.bard.

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Abstract (one page maximum, single spaced), include: List the original objectives, as defined in the approved proposal, and any revisions made at the beginning or during the course of project: The main goal described in our original proposal has been the development of a molecular infectious clone of the avian metapneumovirus subtype B (aMPV-B) and the modification of this clone to create mutated viruses for the development of attenuated vaccines. The Achievements and Appendix/Part I sections of this report describes the accomplishments in creating such a molecular clone. These sections also contain the results of a longitudinal study that we made in Israel, demonstrating the infiltration of field strains of aMPV into vaccinated flocks and emphasizing the need for the development of better vaccines. We also describe our unexpected findings regarding the ability of aMPV to establish persistent infection in cell cultures. Although this direction of research was not described in the original proposal we feel that it is highly important for the understanding of aMPV pathogenesis. For example, this direction has provided us with evidence showing that aMPV replication can augment influenza replication. Moreover, we observed that viruses that were produced from chronically-infected cells show reduced ciliostasis. Accordingly, we carried vaccination trials using such viruses. In the original grant proposal we also offered that the American lab will clone and express immunomodulators in the context of an aMPV -based replicon that the Israeli lab has generated. However, as we reported in our annual reports, further analysis of this replicon by the Israeli lab has revealed that the level of expression achieved by this vehicle is relatively poor; thus, the American lab has focused on sequencing the genomes of different aMPV-C isolates that differ in their virulence (including vaccine strains). Achievements and Appendix/Part II sections of this report include the summary of this effort. Background to the topic: The aMPVs belong to the paramyxoviridae family and cause mild to severe respiratory tract diseases mainly in turkeys and also in chickens. Four aMPV subgroups, A, B, C and D, have been characterized; in Israel aMPV-A and B are the common subtypes while in the USA type C is the prevalent one. Although vaccine strains do exist for aMPVs, they do not always provide full protection against virulent strains and the vaccines themselves may induce disease to some extent. Improved vaccines against aMPV are needed, to achieve better protection of the poultry industry against this pathogen. Major conclusions, solutions, achievements: We isolated aMPV-B from a diseased flock and accomplished the sequencing and cloning of its full-genome. In addition, we cloned the four genes encoding the viral replicase. These should serve as the platform for generation of modified aMPV-Bs from molecular clones. We also identified aMPVs that are attenuated in respect to their ciliostatic activity and accordingly showed the potential of such viruses as vaccine strains. For aMPV-C, the different mutations scattered along the genome of different isolates with varied virulence have been determined. Implications, both scientific and agricultural: The newly identified pattern of mutations in attenuated strains will allow better understanding of the pathogenicity of aMPV and the generation of aMPV molecular clones, together with isolation of strains with attenuated ciliostatic activity should generate improved vaccine strains Abstract (one page maximum, single spaced), include: List the original objectives, as defined in the approved proposal, and any revisions made at the beginning or during the course of project: The main goal described in our original proposal has been the development of a molecular infectious clone of the avian metapneumovirus subtype B (aMPV-B) and the modification of this clone to create mutated viruses for the development of attenuated vaccines. The Achievements and Appendix/Part I sections of this report describes the accomplishments in creating such a molecular clone. These sections also contain the results of a longitudinal study that we made in Israel, demonstrating the infiltration of field strains of aMPV into vaccinated flocks and emphasizing the need for the development of better vaccines. We also describe our unexpected findings regarding the ability of aMPV to establish persistent infection in cell cultures. Although this direction of research was not described in the original proposal we feel that it is highly important for the understanding of aMPV pathogenesis. For example, this direction has provided us with evidence showing that aMPV replication can augment influenza replication. Moreover, we observed that viruses that were produced from chronically-infected cells show reduced ciliostasis. Accordingly, we carried vaccination trials using such viruses. In the original grant proposal we also offered that the American lab will clone and express immunomodulators in the context of an aMPV -based replicon that the Israeli lab has generated. However, as we reported in our annual reports, further analysis of this replicon by the Israeli lab has revealed that the level of expression achieved by this vehicle is relatively poor; thus, the American lab has focused on sequencing the genomes of different aMPV-C isolates that differ in their virulence (including vaccine strains). Achievements and Appendix/Part II sections of this report include the summary of this effort. Background to the topic: The aMPVs belong to the paramyxoviridae family and cause mild to severe respiratory tract diseases mainly in turkeys and also in chickens. Four aMPV subgroups, A, B, C and D, have been characterized; in Israel aMPV-A and B are the common subtypes while in the USA type C is the prevalent one. Although vaccine strains do exist for aMPVs, they do not always provide full protection against virulent strains and the vaccines themselves may induce disease to some extent. Improved vaccines against aMPV are needed, to achieve better protection of the poultry industry against this pathogen. Major conclusions, solutions, achievements: We isolated aMPV-B from a diseased flock and accomplished the sequencing and cloning of its full-genome. In addition, we cloned the four genes encoding the viral replicase. These should serve as the platform for generation of modified aMPV-Bs from molecular clones. We also identified aMPVs that are attenuated in respect to their ciliostatic activity and accordingly showed the potential of such viruses as vaccine strains. For aMPV-C, the different mutations scattered along the genome of different isolates with varied virulence have been determined. Implications, both scientific and agricultural: The newly identified pattern of mutations in attenuated strains will allow better understanding of the pathogenicity of aMPV and the generation of aMPV molecular clones, together with isolation of strains with attenuated ciliostatic activity should generate improved vaccine strains.

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